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Artículos de revistas sobre el tema "Oxidative Aging"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 26 de julio de 2025

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1

Abouelsaad, Ahmed, and Greg White. "Comparing the Effect of Thermal-Oxidation and Photo-Oxidation of Asphalt Mixtures on the Rheological and Chemical Properties of Extracted Bituminous Binder." Materials 15, no. 19 (2022): 6793. http://dx.doi.org/10.3390/ma15196793.

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The reliable and representative ageing of asphalt samples in the laboratory is critically important to research on asphalt durability, waste material recycling and rejuvenation treatments. However, standard laboratory ageing protocols omit ultraviolet radiation and moisture, and are also based on a universally applied oven temperature and ageing duration. The aim of this research was to demonstrate the importance of ultraviolet radiation in laboratory asphalt aging, motivated by the need for more realistic ageing protocols. Asphalt cores were thermally aged in a standard laboratory oven for 98

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2

Xiao, Ling, Yu Li, Xuan Wang, and Chao Yu. "Study on aging mechanism of special neoprene in hot air." IOP Conference Series: Earth and Environmental Science 1171, no. 1 (2023): 012038. http://dx.doi.org/10.1088/1755-1315/1171/1/012038.

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Abstract The accelerated aging tests of the special neoprene material in hot air were carried in laboratory. The aging mechanism was analyzed by using FTIR, DSC and TG spectra. The results showed that the thermal oxidative aging in air was a self-catalyzed oxidating reaction, the cross-linking reaction firstly in the process of thermo-oxidation, and the degradation reaction occurs at the later stage of aging.

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3

Neki, N. S. "Oxidative Stress and Aging." Bangladesh Journal of Medical Science 14, no. 3 (2015): 221–27. http://dx.doi.org/10.3329/bjms.v14i3.23468.

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Ageing is an inevitable life process characterized by a gradual functional decline of all organ systems occurring at the cellular, tissue, organ and whole body levels further leading to the development of diseases and finally death. Although aging is a normal physiological process, it can be accelerated during oxidative stress or during chronic inflammatory conditions. An appropriate theory must explain four main characteristics of ageing: it is endogenous, progressive, irreversible and deleterious for the individual. Oxidative stress is caused by imbalance between oxidants and antioxidants. R

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4

Rad, Farhad Yousefi, Michael D. Elwardany, Cassie Castorena, and Y. Richard Kim. "Evaluation of Chemical and Rheological Aging Indices to Track Oxidative Aging of Asphalt Mixtures." Transportation Research Record: Journal of the Transportation Research Board 2672, no. 28 (2018): 349–58. http://dx.doi.org/10.1177/0361198118784138.

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Oxidative age hardening in asphalt binder leads to embrittlement. Embrittled asphalt is prone to fatigue and thermal cracking. Therefore, the ability to predict asphalt binder oxidative age hardening within a pavement throughout its service life could inform improved pavement material selection, design, and maintenance practices. Studying the evolution of oxidative aging requires the use of key properties to track oxidation levels, termed aging index properties (AIPs) here. The objective of this study is to identify suitable rheological and chemical AIPs to track oxidation levels in asphalt ma

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5

Szarka, András, Gábor Bánhegyi, and Balázs Sümegi. "Mitochondria, oxidative stress and aging." Orvosi Hetilap 155, no. 12 (2014): 447–52. http://dx.doi.org/10.1556/oh.2014.29852.

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The free radical theory of aging was defined in the 1950s. On the base of this theory, the reactive oxygen species formed in the metabolic pathways can play pivotal role in ageing. The theory was modified by defining the mitochondrial respiration as the major cellular source of reactive oxygen species and got the new name mitochondrial theory of aging. Later on the existence of a “vicious cycle” was proposed, in which the reactive oxygen species formed in the mitochondrial respiration impair the mitochondrial DNA and its functions. The formation of reactive oxygen species are elevated due to m

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6

Li, Yilin, Yonggang Liu, Wei Hao, et al. "Mechanical properties and quantum mechanical simulations of natural rubber composites with cerium complexes under aging conditions." Materials Science-Poland 42, no. 1 (2024): 158–70. http://dx.doi.org/10.2478/msp-2024-0013.

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Abstract Enhancing the service life of natural rubber (NR) products, including antioxidants, is crucial to prevent rubber degradation and enhance its oxidation resistance. Phenolic antioxidant 2246 and cerium complex of p-amino salicylic acid (PAS-Ce) are utilized as NR antioxidants. Numerous studies have qualitatively analyzed the antioxidant mechanisms of these compounds. Building upon this perspective, this study quantitatively assessed the protective mechanisms of these antioxidants by combining experimental data with molecular simulations. Additionally, it compared their impacts on the th

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7

Kregel, Kevin C., and Hannah J. Zhang. "An integrated view of oxidative stress in aging: basic mechanisms, functional effects, and pathological considerations." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 292, no. 1 (2007): R18—R36. http://dx.doi.org/10.1152/ajpregu.00327.2006.

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Aging is an inherently complex process that is manifested within an organism at genetic, molecular, cellular, organ, and system levels. Although the fundamental mechanisms are still poorly understood, a growing body of evidence points toward reactive oxygen species (ROS) as one of the primary determinants of aging. The “oxidative stress theory” holds that a progressive and irreversible accumulation of oxidative damage caused by ROS impacts on critical aspects of the aging process and contributes to impaired physiological function, increased incidence of disease, and a reduction in life span. W

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8

Zhuang, Tao, Ti Kun Shan, and Li Ling Zhou. "Research on the Thermal Oxidation and Ultraviolet Radiation Aging of Nano-ZnO/NR Composite." Advanced Materials Research 87-88 (December 2009): 462–67. http://dx.doi.org/10.4028/www.scientific.net/amr.87-88.462.

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To study the effection of nano-ZnO on thermal oxidative aging and ultraviolet radiation aging of NR, and the mechanisms of thermal oxidation aging and ultraviolet radiation aging of nano ZnO in NR were explained by TG and stress relaxation and ultraviolet analysis. The results revealed that nano-ZnO can improve the anti-thermal oxidation aging and anti-ultraviolet radiation aging of NR, and the protective effect improves with the increase dose of nano-ZnO.

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9

Petersen, J. Claine, and P. Michael Harnsberger. "Asphalt Aging: Dual Oxidation Mechanism and Its Interrelationships with Asphalt Composition and Oxidative Age Hardening." Transportation Research Record: Journal of the Transportation Research Board 1638, no. 1 (1998): 47–55. http://dx.doi.org/10.3141/1638-06.

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The kinetic data and chemistry of asphalt oxidative age hardening suggested a sequential, dual mechanism for asphalt oxidation. The dual mechanism rationalizes conflicts between earlier mechanistic investigations and explains the hyperbolic-like, time-versus-property plots characteristic of asphalt oxidative aging. The oxidation kinetics provide further confirmation of the asphalt microstructural model. It is proposed that the rapid initial oxidation rate of asphalt results from reaction of oxygen with limited amounts of highly reactive hydrocarbons. Final oxidation products of this initial re

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10

Moreira, Priscila Lucelia, Paulo Jose Fortes Villas Boas, and Ana Lucia Anjos Ferreira. "Association between oxidative stress and nutritional status in the elderly." Revista da Associação Médica Brasileira 60, no. 1 (2014): 75–83. http://dx.doi.org/10.1590/1806-9282.60.01.016.

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Ageing is a dynamic and progressive process that is characterized by the occurrence of morphological, biochemical, functional and psychological changes in the organism. The aim of the present article is to provide updated concepts on oxidative stress, covering its importance in aging, as well as nutritional status and supplementation with antioxidants (substances that prevent or attenuate oxidation of oxidizable substrates, such as lipids, proteins, carbohydrates and deoxyribonucleic acid) in the geriatric population. Evidence suggests that there is an inverse relationship between oxidative st

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11

Zhang, Xuemei, and Inge Hoff. "Comparative Study of Thermal-Oxidative Aging and Salt Solution Aging on Bitumen Performance." Materials 14, no. 5 (2021): 1174. http://dx.doi.org/10.3390/ma14051174.

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The aging of bitumen is detrimental to the durability and service life of asphalt pavement. Previous studies found that bitumen was suspected to be aged by not only thermal oxidation but also solution immersion. This research aims to compare the effect of thermal-oxidative aging and salt solution aging on bitumen performance. For this purpose, a thin film oven test (TFOT) and pressure aging vessel aging (PAV) were selected as thermal-oxidative aging, and 10% NaCl aging and 10% CaCl2 aging were selected as salt solution aging. The morphology, oxygen content, physical properties, low-temperature

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12

Breusing, Nicolle, and Tilman Grune. "Regulation of proteasome-mediated protein degradation during oxidative stress and aging." Biological Chemistry 389, no. 3 (2008): 203–9. http://dx.doi.org/10.1515/bc.2008.029.

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Abstract Protein degradation is a physiological process required to maintain cellular functions. There are distinct proteolytic systems for different physiological tasks under changing environmental and pathophysiological conditions. The proteasome is responsible for the removal of oxidatively damaged proteins in the cytosol and nucleus. It has been demonstrated that proteasomal degradation increases due to mild oxidation, whereas at higher oxidant levels proteasomal degradation decreases. Moreover, the proteasome itself is affected by oxidative stress to varying degrees. The ATP-stimulated 26

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13

Huh, Jung-Do, and Raymond E. Robertson. "Modeling of Oxidative Aging Behavior of Asphalts from Short-Term, High-Temperature Data as a Step toward Prediction of Pavement Aging." Transportation Research Record: Journal of the Transportation Research Board 1535, no. 1 (1996): 91–97. http://dx.doi.org/10.1177/0361198196153500112.

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The oxidative aging data collected during the Strategic Highway Research Program have been analyzed in terms of kinetics of viscosity change with time and temperature. Changes in viscosity have been used as the measure of the progress of aging. The objective is to model viscosity increases accurately enough to be able to predict aging (in terms of viscosity changes) at pavement temperatures from short-term test data acquired at high temperature. This involved constituting a mathematical model, based on oxidative reactions, and a nonlinear regression of the data to test predictability of the pr

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14

Chhunchha, Bhavana, Eri Kubo, and Dhirendra P. Singh. "Switching of Redox Signaling by Prdx6 Expression Decides Cellular Fate by Hormetic Phenomena Involving Nrf2 and Reactive Oxygen Species." Cells 11, no. 8 (2022): 1266. http://dx.doi.org/10.3390/cells11081266.

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Changes in intracellular reactive oxygen species (ROS) levels due to remodeling of antioxidant defense can affect the status of biological homeostasis in aging/oxidative stress. Peroxiredoxin 6 (Prdx6), an antioxidant gene downstream target for the Nrf2 pathway, plays a role in regulating ROS homeostasis. Using aging human (h) lens epithelial cells (LECs) or Prdx6-deficient (Prdx6−/−) mouse (m) LECs, here we showed that dichlorofluorescein (DCF) oxidation or H2O2 were strictly controlled by Prdx6. We observed that a moderate degree of oxidative stress augmented Nrf2-mediated Prdx6 expression,

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15

Haenold, Ronny, D. Mokhtar Wassef, Stefan H. Heinemann, and Toshinori Hoshi. "Oxidative damage, aging and anti-aging strategies." AGE 27, no. 3 (2005): 183–99. http://dx.doi.org/10.1007/s11357-005-2915-0.

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16

Forte, Lucrezia, Pasquale De Palo, Alessandra Aloia, and Aristide Maggiolino. "Could colour and volatile compounds be measurements of oxidation in horse meat?" Acta IMEKO 13, no. 1 (2024): 1–6. http://dx.doi.org/10.21014/actaimeko.v13i1.1625.

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Predicting and, consequently, controlling oxidative phenomena is a big challenge for meat industry. Since color modifications and volatile compounds synthesis are processes closely linked to the oxidative reactions occurring in meat, oxidation could be measured through colorimetric parameters or some volatile compounds. A Spearman’s correlation analysis of oxidative parameters and enzymatic activity with colorimetric parameters and volatile profile was performed on horse steaks subjected to short (14 days) and long aging time (56 days). In long aging, TBARS were negatively correlated with redn

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17

Tanakol, Ali, Tuğba Kevser Uzunçakmak, and Zekayi Kutlubay. "Oxidative Stress and Aging." Dermatoz 11, no. 3 (2021): 31–35. http://dx.doi.org/10.4274/dermatoz.galenos.2021.68077.

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18

NIKI, Etsuo. "Oxidative Stress and Aging." Internal Medicine 39, no. 4 (2000): 324–26. http://dx.doi.org/10.2169/internalmedicine.39.324.

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19

Зенков, Н. К., П. М. Кожин, А. В. Чечушков, Н. В. Кандалинцева, Г. Г. Мартинович, and Е. Б. Меньщикова. "OXIDATIVE STRESS IN AGING." Успехи геронтологии, no. 1 (April 12, 2020): 10–22. http://dx.doi.org/10.34922/ae.2020.33.1.001.

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Выдвинутая более 50 лет назад Д. Харманом свободнорадикальная теория старения остается популярной и сегодня. В обзоре проведен анализ возрастных изменений основных эндогенных механизмов продукции активированных кислородных метаболитов (АКМ) и механизмов антиоксидантной защиты. С возрастом генерация АКМ митохондриями, пероксисомами и NAD(P) H -оксидазами усиливается, в то время как транскрипционная активность важной системы поддержания редокс-баланса Keap 1/ Nrf 2/ ARE уменьшается. У старых животных отмечается также низкая активность аутофагии, удаляющей из клеток поврежденные органеллы и агрег

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20

Rattan, Suresh I. S. "Oxidative stress and aging." FEBS Letters 381, no. 3 (1996): 262. http://dx.doi.org/10.1016/s0014-5793(96)90651-1.

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21

Chen, Danica. "OXIDATIVE STRESS AND AGING." Free Radical Biology and Medicine 53 (November 2012): S3. http://dx.doi.org/10.1016/j.freeradbiomed.2012.10.006.

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22

Junqueira, Virginia B. C., Silvia B. M. Barros, Sandra S. Chan, et al. "Aging and oxidative stress." Molecular Aspects of Medicine 25, no. 1-2 (2004): 5–16. http://dx.doi.org/10.1016/j.mam.2004.02.003.

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23

Abdollahi, Mohammad, Majid Y. Moridani, Okezie I. Aruoma, and Sara Mostafalou. "Oxidative Stress in Aging." Oxidative Medicine and Cellular Longevity 2014 (2014): 1–2. http://dx.doi.org/10.1155/2014/876834.

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24

Warner, Huber R. "Oxidative stress and aging." Experimental Gerontology 31, no. 3 (1996): 436–38. http://dx.doi.org/10.1016/0531-5565(95)02036-5.

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25

Lesnefsky, Edward J., and Charles L. Hoppel. "Oxidative phosphorylation and aging." Ageing Research Reviews 5, no. 4 (2006): 402–33. http://dx.doi.org/10.1016/j.arr.2006.04.001.

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26

Berlett, Barbara S., and Earl R. Stadtman. "Protein Oxidation in Aging, Disease, and Oxidative Stress." Journal of Biological Chemistry 272, no. 33 (1997): 20313–16. http://dx.doi.org/10.1074/jbc.272.33.20313.

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27

Maxe, Charlotte, Rémy Romanet, Michel Parisot, Régis D. Gougeon, and Maria Nikolantonaki. "The Oxidative Stability of Champagne Base Wines Aged on Lees in Barrels: A 2-Year Study." Antioxidants 13, no. 3 (2024): 364. http://dx.doi.org/10.3390/antiox13030364.

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In contrast with the elaboration of still wines, the impact of barrel aging before the “prise de mousse” on the aging potential of Champagne base wines has not been studied so far. In the present study, the oxidative stability and related molecular fingerprints of Chardonnay Champagne base wines were reported after 1 year of on lees ageing in new oak barrels for two consecutive vintages. Regardless of the vintage, on lees ageing in new oak barrels improved the wines’ oxidative stability estimated by DPPH assay at 1 year, while UHPLC-Q-ToF-MS molecular profiling showed clear chemical modificati

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28

Lee, Yun Haeng, Myeong Uk Kuk, Moon Kyoung So, et al. "Targeting Mitochondrial Oxidative Stress as a Strategy to Treat Aging and Age-Related Diseases." Antioxidants 12, no. 4 (2023): 934. http://dx.doi.org/10.3390/antiox12040934.

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Mitochondria are one of the organelles undergoing rapid alteration during the senescence process. Senescent cells show an increase in mitochondrial size, which is attributed to the accumulation of defective mitochondria, which causes mitochondrial oxidative stress. Defective mitochondria are also targets of mitochondrial oxidative stress, and the vicious cycle between defective mitochondria and mitochondrial oxidative stress contributes to the onset and development of aging and age-related diseases. Based on the findings, strategies to reduce mitochondrial oxidative stress have been suggested

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29

Barcelos, Isabella Peixoto de, and Richard H. Haas. "CoQ10 and Aging." Biology 8, no. 2 (2019): 28. http://dx.doi.org/10.3390/biology8020028.

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The aging process includes impairment in mitochondrial function, a reduction in anti-oxidant activity, and an increase in oxidative stress, marked by an increase in reactive oxygen species (ROS) production. Oxidative damage to macromolecules including DNA and electron transport proteins likely increases ROS production resulting in further damage. This oxidative theory of cell aging is supported by the fact that diseases associated with the aging process are marked by increased oxidative stress. Coenzyme Q10 (CoQ10) levels fall with aging in the human but this is not seen in all species or all

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30

Sun, Zhuo Jun, Jian Gao, Hui Liu, et al. "Migration Characteristics and Degradation Kinetics of Bisphenol A." Materials Science Forum 850 (March 2016): 128–36. http://dx.doi.org/10.4028/www.scientific.net/msf.850.128.

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This paper study the migration characteristics and degradation kinetics of bisphenol A using TGA - gas chromatography - mass spectrometry and found that bisphenol A polycarbonate in the thermal oxidative aging conditions of 130 °C de-gradated to bisphenol A. At the range of 0 h to 120 h, the bisphenol A content of environmental hormones increased with time. When it reached 120 h, bisphenol A environmental hormone content decreased slightly with aging time. The content of bisphenol A reached 495mg/kg when the thermal oxidative aging time was 168 h, which was decreased compared to the content of

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31

Bolotina, D. I., A. I. Kononenko, A. L. Pomerantsev, and A. G. Tsikanin. "Modeling of thermal aging of cable polymer composition based on ethylene vinyl acetate." All the Materials. Encyclopedic Reference Book, no. 6 (June 2023): 30–37. http://dx.doi.org/10.31044/1994-6260-2023-0-6-30-37.

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The aging of a cable polymer shell composition based on ethylene vinyl acetate has been studied. During accelerated aging tests in the temperature range from 110 to 150 °С, the change in the mechanical and thermal properties of the elastomeric samples was monitored. The sample properties were controlled by measuring the following characteristics: the relative elongation at break (REB), the indentation modulus of elasticity (IME), the oxidative induction temperature, and the oxidative induction time. To simulate a REB change during the aging, an empirical kinetic equation was used, which reflec

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32

Pandey, Kanti Bhooshan, Mohd Murtaza Mehdi, Pawan Kumar Maurya, and Syed Ibrahim Rizvi. "Plasma Protein Oxidation and Its Correlation with Antioxidant Potential During Human Aging." Disease Markers 29, no. 1 (2010): 31–36. http://dx.doi.org/10.1155/2010/964630.

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Previous studies have indicated that the main molecular characteristic of aging is the progressive accumulation of oxidative damages in cellular macromolecules. Proteins are one of the main molecular targets of age-related oxidative stress, which have been observed during aging process in cellular systems. Reactive oxygen species (ROS) can lead to oxidation of amino acid side chains, formation of protein-protein cross-linkages, and oxidation of the peptide backbones. In the present study, we report the age-dependent oxidative alterations in biomarkers of plasma protein oxidation: protein carbo

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33

Chen, Ge, and Jia Lu Li. "Accelerated Test Design of Thermal-Oxidative Aging of Nylon 66 Airbag Material." Advanced Materials Research 332-334 (September 2011): 1202–5. http://dx.doi.org/10.4028/www.scientific.net/amr.332-334.1202.

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Accelerated thermal-oxidative aging tests on Nylon 66 automotive airbag material are designed and performed in this paper. After the environmental profile of airbag was analyzed, Nylon 66 airbag yarns were reasonably selected as the test samples, and its tensile strength was chosen as the significant character of their aging stability. By heat aging、humidity aging、cycle ageing and comprehensive process aging experiments, 350dtex Nylon 66 filament were sampled and the tensile strength loss of samples was measured, to determine the mechanisms and factors that influence aging stability of the Nyl

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34

Doria, Enrico, Daniela Buonocore, Angela Focarelli, and Fulvio Marzatico. "Relationship between Human Aging Muscle and Oxidative System Pathway." Oxidative Medicine and Cellular Longevity 2012 (2012): 1–13. http://dx.doi.org/10.1155/2012/830257.

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Ageing is a complex process that in muscle is usually associated with a decrease in mass, strength, and velocity of contraction. One of the most striking effects of ageing on muscle is known as sarcopenia. This inevitable biological process is characterized by a general decline in the physiological and biochemical functions of the major systems. At the cellular level, aging is caused by a progressive decline in mitochondrial function that results in the accumulation of reactive oxygen species (ROS) generated by the addition of a single electron to the oxygen molecule. The aging process is char

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35

Olinski, Ryszard, Agnieszka Siomek, Rafal Rozalski, et al. "Oxidative damage to DNA and antioxidant status in aging and age-related diseases." Acta Biochimica Polonica 54, no. 1 (2007): 11–26. http://dx.doi.org/10.18388/abp.2007_3265.

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Aging is a complex process involving morphologic and biochemical changes in single cells and in the whole organism. One of the most popular explanations of how aging occurs at the molecular level is the oxidative stress hypothesis. Oxidative stress leads in many cases to an age-dependent increase in the cellular level of oxidatively modified macromolecules including DNA, and it is this increase which has been linked to various pathological conditions, such as aging, carcinogenesis, neurodegenerative and cardiovascular diseases. It is, however, possible that a number of short-comings associated

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Zou, Ling, and Nan Xiang Zheng. "Applied Research of the Composite Antiager in Asphalt." Advanced Materials Research 919-921 (April 2014): 1191–94. http://dx.doi.org/10.4028/www.scientific.net/amr.919-921.1191.

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The simulation test system was established to study the performance of the thermo-oxidative aging and ultraviolet aging of 90# matrix asphalt and SBSI-C modified asphalt which were added composite antiager. Mechanism of thermo-oxidative aging and ultraviolet aging of asphalt was also analyzed. The results proved that : indexes of thermo-oxidative aging and ultraviolet aging of 90# matrix asphalt and SBSI-C modified asphalt were improved after adding the composite antiager,and composite antiager had positive effects to resist the aging of asphalt.

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37

Pochiraju, K. V., and G. P. Tandon. "Modeling Thermo-Oxidative Layer Growth in High-Temperature Resins." Journal of Engineering Materials and Technology 128, no. 1 (2005): 107–16. http://dx.doi.org/10.1115/1.2128427.

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This paper describes modeling of degradation behavior of high-temperature polymers under thermo-oxidative aging conditions. Thermo-oxidative aging is simulated with a diffusion-reaction model in which temperature, oxygen concentration, and weight-loss effects are considered. A parametric reaction model based on a mechanistic view of the reaction is used for simulating reaction-rate dependence on the oxygen availability in the polymer. Macroscopic weight-loss measurements are used to determine the reaction and polymer consumption parameters. The diffusion-reaction partial differential equation

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38

Golbidi, Saeid, and Ismail Laher. "Exercise and the Aging Endothelium." Journal of Diabetes Research 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/789607.

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The endothelium plays a critical role in the maintenance of cardiovascular health by producing nitric oxide and other vasoactive materials. Aging is associated with a gradual decline in this functional aspect of endothelial regulation of cardiovascular homeostasis. Indeed, age is an independent risk factor for cardiovascular diseases and is in part an important factor in the increased exponential mortality rates from vascular disease such as myocardial infarction and stroke that occurs in the ageing population. There are a number of mechanisms suggested to explain age-related endothelial dysfu

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39

Stojiljković, Dragana, Dušica Pavlović, and Ivana Arsić. "Oxidative Stress, Skin Aging and Antioxidant Therapy / Oksidacioni Stres, Starenje Kože I Antioksidaciona Terapija." Acta Facultatis Medicae Naissensis 31, no. 4 (2014): 207–17. http://dx.doi.org/10.2478/afmnai-2014-0026.

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Summary Skin aging is a dynamic process that occurs due to chronological and photo-aging caused by the influence of external factors, especially ultraviolet-UV radiation. Cumulative oxidative stress, formation of free radicals and their adverse effects on biological systems in the skin cells is a common mechanism of both skin aging processes. The formed reactive oxygen species-ROS can lead to the oxidation of cell`s building molecules. Lipid peroxidation, membrane protein damage and DNA mutation may lead to many structural, functional and aesthetic changes in the skin and can also initiate dev

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40

Sanna, Daniele, and Angela Fadda. "Oxidative Stability of Sunflower Oil: Effect of Blending with an Oil Extracted from Myrtle Liqueur By-Product." Antioxidants 14, no. 3 (2025): 300. https://doi.org/10.3390/antiox14030300.

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Myrtle oil extracted from the spent berries of myrtle liqueur production, using 2-methyltetrahydrofuran, was used to increase the oxidative stability of sunflower oil (SFO). Three blending ratios (5%, 10%, and 15% w/w) and the SFO without any addition were subjected to forced aging conditions at 70 °C for 21 days. The changes in peroxide value (PV), p-anisidine value (AV), total oxidation value (totox), and conjugated dienes and trienes were evaluated during forced aging. The oxidative stability of the blends was also assessed by the spin trapping method coupled with Electron Paramagnetic Reso

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Di Carlo, Emma, and Carlo Sorrentino. "Oxidative Stress and Age-Related Tumors." Antioxidants 13, no. 9 (2024): 1109. http://dx.doi.org/10.3390/antiox13091109.

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Oxidative stress is the result of the imbalance between reactive oxygen and nitrogen species (RONS), which are produced by several endogenous and exogenous processes, and antioxidant defenses consisting of exogenous and endogenous molecules that protect biological systems from free radical toxicity. Oxidative stress is a major factor in the aging process, contributing to the accumulation of cellular damage over time. Oxidative damage to cellular biomolecules, leads to DNA alterations, lipid peroxidation, protein oxidation, and mitochondrial dysfunction resulting in cellular senescence, immune

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42

Baba, Burcu, Dilek Yonar, Hikmet Taner Teker, and Taha Ceylani. "Protective Effects of Intermittent Fasting and Probiotics Usage on Oxidative Stress and Molecular Alterations in Aging Lung." Meandros Medical And Dental Journal 26, no. 1 (2025): 1–11. https://doi.org/10.69601/meandrosmdj.1583374.

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Objective: Aging is associated with increased oxidative stress and diminished cellular repair mechanisms, particularly in the lungs. This study investigates the protective effects of intermittent fasting (IF), SCD Probiotics, and their combination on oxidative stress and molecular alterations in the lungs of aging rats. Materials and Methods: Sprague-Dawley rats (24 months old) were divided into four groups: control, intermittent fasting, probiotics, and a combination of both treatments. Oxidative stress markers, including malondialdehyde (MDA), advanced oxidation protein products (AOPP), and

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43

Starke-Reed, Pamela E., and Cynthia N. Oliver. "Protein oxidation and proteolysis during aging and oxidative stress." Archives of Biochemistry and Biophysics 275, no. 2 (1989): 559–67. http://dx.doi.org/10.1016/0003-9861(89)90402-5.

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44

ZHANG, JING, XIANKUN DAI, MENGWEI WANG, et al. "Salidroside Enhances Aerobic Swimming to Improve Oxidative Aging Induced by D-Galactose in Mice." Acta Poloniae Pharmaceutica - Drug Research 80, no. 5 (2023): 831–40. http://dx.doi.org/10.32383/appdr/172999.

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This study observed the effect of salidroside on improving oxidative aging induced by D-galactose in mice by enhancing aerobic swimming. In this study, the oxidative aging model of mice was established by D-galactose, and the changes of organ index, histopathological changes, and mRNA expression in serum and tissue of oxidative aging mice after aerobic swimming and aerobic swimming+salidroside were detected. The results showed that aerobic swimming and aerobic swimming+salidroside inhibited the decline of heart index, liver index, spleen index and kidney index caused by oxidative aging in mice

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45

Thonusin, Chanisa, Patcharapong Pantiya, Aphisek Kongkaew, et al. "Exercise and Caloric Restriction Exert Different Benefits on Skeletal Muscle Metabolism in Aging Condition." Nutrients 15, no. 23 (2023): 5004. http://dx.doi.org/10.3390/nu15235004.

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Exercise and caloric restriction improve skeletal muscle metabolism. However, the benefits of exercise and caloric restriction on skeletal muscle metabolism in aging have never been compared. Seven-week-old male Wistar rats (n = 24) were divided into 4 groups (n = 6 per group) to receive either normal saline solution for 28 weeks, 150 mg/kg/day of D-galactose for 28 weeks to induce premature aging, 150 mg/kg/day of D-galactose for 28 weeks plus exercise for 16 weeks (week 13–28), or 150 mg/kg/day of D-galactose for 28 weeks plus 30% caloric restriction for 16 weeks (week 13–28). The 17-month-o

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46

Izzo, Carmine, Paolo Vitillo, Paola Di Pietro, et al. "The Role of Oxidative Stress in Cardiovascular Aging and Cardiovascular Diseases." Life 11, no. 1 (2021): 60. http://dx.doi.org/10.3390/life11010060.

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Aging can be seen as process characterized by accumulation of oxidative stress induced damage. Oxidative stress derives from different endogenous and exogenous processes, all of which ultimately lead to progressive loss in tissue and organ structure and functions. The oxidative stress theory of aging expresses itself in age-related diseases. Aging is in fact a primary risk factor for many diseases and in particular for cardiovascular diseases and its derived morbidity and mortality. Here we highlight the role of oxidative stress in age-related cardiovascular aging and diseases. We take into co

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47

Pandey, Kanti Bhooshan, and Syed Ibrahim Rizvi. "Resveratrol in vitro amelioratestert-butyl hydroperoxide-induced alterations in erythrocyte membranes from young and older humans." Applied Physiology, Nutrition, and Metabolism 39, no. 10 (2014): 1093–97. http://dx.doi.org/10.1139/apnm-2014-0064.

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Implication of reactive oxygen species/oxidative stress has been readily reported in etiology of aging and related manifestations. Plasma membrane as a regulator of numerous aspects of cell physiology including cell–cell interaction, solute transport, and signal transduction, provides structural integrity to the cells. The aim of the present study was to determine the effect of resveratrol administration in vitro, to evaluate the biological effect of this phytoalexin in oxidatively injured erythrocytes during aging. This study, carried out on 91 normal healthy subjects, provides experimental e

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Ahsanuddin, Sayeeda, Minh Lam, and Elma D. Baron. "Skin aging and oxidative stress." AIMS Molecular Science 3, no. 2 (2016): 187–95. http://dx.doi.org/10.3934/molsci.2016.2.187.

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Shaw, Peter X., Geoff Werstuck, and Yan Chen. "Oxidative Stress and Aging Diseases." Oxidative Medicine and Cellular Longevity 2014 (2014): 1–2. http://dx.doi.org/10.1155/2014/569146.

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50

Liu, Dongping, and Yang Xu. "p53, Oxidative Stress, and Aging." Antioxidants & Redox Signaling 15, no. 6 (2011): 1669–78. http://dx.doi.org/10.1089/ars.2010.3644.

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