Bibliografías temáticas / Partial-onset epilepsy

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Literatura académica sobre el tema "Partial-onset epilepsy"

Autor: Grafiati
Publicado: 7 de junio de 2025
Última modificación: 2 de agosto de 2025

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Artículos de revistas sobre el tema "Partial-onset epilepsy"

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Liu, Lynn, and J. Craig Henry. "New-onset partial epilepsy in adults." Current Treatment Options in Neurology 11, no. 4 (2009): 242–52. http://dx.doi.org/10.1007/s11940-009-0028-2.

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Lavania, Sagar, Mohd Aleem Siddiqui, Shantanu Bharti, and Abhishek Kumar. "Obsessive compulsive symptoms in patients with primary generalized and partial onset epilepsy." International Journal of Research in Medical Sciences 6, no. 4 (2018): 1183. http://dx.doi.org/10.18203/2320-6012.ijrms20181019.

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Background: To find out and compare the obsessive-compulsive symptoms / disorder among patients of primary generalized and partial onset epilepsy.Methods: Patients with epilepsy diagnosed clinically at psychiatric out patient’s department were selected for the study and categorized as primary generalized onset tonic clonic seizure type and partial onset seizure. Yale-Brown obsessive-compulsive symptoms check list and scale was applied to find out the obsessive-compulsive symptoms.Results: A total of 110 patients were categorized as primary generalized (GE) 49 and partial onset epilepsy (PE) 61
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Loring Levine, Reed, and David Y. Ko. "New Add-on Therapy for Partial-onset Epilepsy." US Neurology 04, no. 01 (2008): 48. http://dx.doi.org/10.17925/usn.2008.04.01.48.

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Sperling, Michael R. "More on the Timing and Meaning of FDG-PET Abnormalities in Partial Epilepsy." Epilepsy Currents 2, no. 6 (2002): 188. http://dx.doi.org/10.1111/j.1535-7597.2002.00071.x.

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Low Incidence of Abnormal FDG-PET in Children with New-onset Partial Epilepsy: A Prospective Study Gaillard W, Kopylev L, Weinstein S, Conry J, Pearl P, Spanaki M, Fazilat S, Venzina L, Dubovsky E, Theodore W Neurology 2002;58:717–722 Objective Patients with refractory partial epilepsy often exhibit regional hypometabolism. It is unknown whether the metabolic abnormalities are present at seizure onset or develop over time. Methods The authors studied 40 children within 1 year of their third unprovoked partial seizure with EEG, magnetic resonance imaging (MRI), and [18F]-fluorodeoxyglucose (18F
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Aungaroon, Gewalin, Katherine D. Holland, Paul S. Horn, Shannon M. Standridge, and Christina Mackell Imming. "Drug-resistant epilepsy in children with partial onset epilepsy treated with carbamazepine." International Journal of Neuroscience 127, no. 10 (2016): 849–53. http://dx.doi.org/10.1080/00207454.2016.1269089.

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Kuzmanova, Rumyana, та I. Vaneva. "Advantages of eslicarbazepine in the treatment of patients with fоcalepilepsy". Bulgarian Neurology 25, № 1 (2024): 1–4. https://doi.org/10.5281/zenodo.15369230.

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Epilepsies are among the most common chronic disorders of the brain and affect approximately 70 million people worldwide. The management of epilepsy is mainly symptomatic, aimed at reducing the risk of seizure recurrence. Despite the availability of many therapeutic options, more than 50% of patients do not achieve seizure freedom with initial monotherapy and despite combination therapy seizures remain uncontrolled in approximately one third of patients.The most frequent type of seizure is the partial-onset (focal) seizure.Eslicarbazepine acetate is a new antiepileptic drug, third-generation r
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Faulkner, Michele A. "Ezogabine for the Adjunctive Treatment of Partial Onset Seizures in Adults with Epilepsy." Clinical Medicine Insights: Therapeutics 3 (January 2011): CMT.S7241. http://dx.doi.org/10.4137/cmt.s7241.

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Partial seizures are frequently resistant to pharmacologic treatment. There are a plethora of medications currently approved for use in partial epilepsy. However, despite the large number of medications available, seizure control often remains elusive. A new medication with a unique mechanism of action has recently been approved for the adjunctive treatment of partial seizures in adults. Ezogabine (retigabine) exerts its actions at the level of voltage-gated potassium channels. In clinical trials it has demonstrated efficacy similar to that of other agents approved for resistant partial epilep
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Reddy, Samba. "Clinical Pharmacology and Therapeutics of Antiepileptic Drugs." International Journal of Pharmaceutical Sciences and Nanotechnology 13, no. 6 (2020): 5165–80. http://dx.doi.org/10.37285/ijpsn.2020.13.6.1.

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This article describes clinical antiepileptic drugs (AEDs) that are available for treatment of epilepsy. Epilepsy is characterized by repeated occurrence of seizures. Epileptic seizures are classified into focal onset (partial) and generalized onset (generalized) types. Around two-dozen AEDs are available for treating epilepsy. AEDs act on diverse molecular targets to selectively modify the abnormal excitability of neurons by reducing the focal seizure discharges or preventing spread of excitation. AEDs suppress seizures by blocking the voltage-gated sodium channels (phenytoin, carbamazepine,
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Lattanzi, S., C. Cagnetti, N. Foschi, A. Lorusso, L. Provinciali, and M. Silvestrini. "Eslicarbazepine acetate as adjunctive treatment in partial-onset epilepsy." Acta Neurologica Scandinavica 137, no. 1 (2017): 29–32. http://dx.doi.org/10.1111/ane.12803.

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Lambrecq, Virginie, Cécile Marchal, Véronique Michel, Dominique Guehl, Pierre Burbaud, and Alain Rougier. "Clinical features of late-onset partial cryptogenic epilepsy: Toward an idiopathic temporal epilepsy?" Epilepsy & Behavior 28, no. 2 (2013): 168–71. http://dx.doi.org/10.1016/j.yebeh.2013.05.001.

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Tesis sobre el tema "Partial-onset epilepsy"

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Van, Zyl Tiaan. "A longitudinal analysis of the prescribing patterns of anti–epileptic medicine by using a medicine claims database / T. van Zyl." Thesis, North-West University, 2010. http://hdl.handle.net/10394/4918.

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The prevalence of epilepsy in society is general knowledge; however the impact on social activity as well as other daily factors are not always fully recognised. Epilepsy frequently poses a problem with regard to work–related activities (Heaney, 1999:44). Moran et al. (2004:425) indicated that the major impacts of epilepsy on life were work and school difficulties, driving prohibition, psychological and social life of which restriction of work or schooling has the greatest impact on epileptic’s life. In all cases the type, severity, and frequency of the seizures as well as the age would be rel
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Libros sobre el tema "Partial-onset epilepsy"

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Krishnan, Vaishnav, Bernard S. Chang, and Donald L. Schomer. The Application of EEG to Epilepsy in Adults and the Elderly. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0019.

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Surface or scalp electroencephalography (EEG) has become an indispensable tool for the diagnosis, classification, and care of patients with epilepsy across the age spectrum. This chapter provides an overview of interictal and corresponding ictal scalp EEG patterns observed in adults with certain classical epilepsy syndromes. In patients with one or more new-onset seizures, the value of EEG testing begins with a close examination of the interictal record. The morphology, frequency, and topography of interictal epileptiform discharges (when present) are typically sufficient to broadly distinguis
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Haldeman, Clayton L., and John S. Kuo. Dysembryoplastic Neuroepithelial Tumors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0005.

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Dysembryoplastic neuroepithelial tumors (DNETs) are World Health Organization grade I tumors and have a low potential for malignant transformation. However, DNETs often affect a patient’s quality of life and can cause medically intractable seizures. The estimated prevalence of DNET is 0.6% to .0.8%; however, they are found in approximately 20% of patients undergoing surgery for epilepsy. Greater than 90% of patients with DNET have onset of epilepsy prior to the age of 20. The most common presentations of DNET are complex partial seizures and simple partial seizures. Generalized tonic–clonic se
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Noebels, Jeffrey L., Massimo Avoli, Michael A. Rogawski, Annamaria Vezzani, and Antonio V. Delgado-Escueta, eds. Jasper's Basic Mechanisms of the Epilepsies. 5th ed. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197549469.001.0001.

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Abstract Unverricht-Lundborg disease (ULD; EPM1) is an inherited neurodegenerative disorder characterized by onset at 6–15 years, stimulus-sensitive, action-activated myoclonus, epilepsy, and progressive neurological deterioration. It is caused by biallelic pathogenic variants in the CSTB gene, encoding a cystatin B. The most common of these is an unstable expansion of a dodecamer repeat element in the promoter region of the gene, leading to marked downregulation of CSTB expression. Total loss of CSTB is associated with severe neonatal-onset encephalopathy. A cystatin B–deficient mouse models
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Capítulos de libros sobre el tema "Partial-onset epilepsy"

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McGoldrick, Patty, Steven M. Wolf, Mary L. Zupanc, and C. L. Harden. "New Onset Partial Epilepsy in Adolescence." In Controversies in Caring for Women with Epilepsy. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29170-3_2.

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Seaborg, K. "My epilepsy story." In Injured Brains of Medical Minds II. Oxford University PressOxford, 2024. https://doi.org/10.1093/oso/9780198890034.003.0068.

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Abstract This chapter assembles four articles that delve into the personal experiences of those with epilepsy. Article 1 presents a family doctor’s narrative, highlighting the role of compassion and hope in confronting epilepsy-related stigma and low mood. Article 2 offers the reflections of a medical student on the stigma surrounding epilepsy and the complexities of disclosing such a diagnosis. Article 3 chronicles a paediatrician’s journey with epilepsy, from the early onset of complex partial seizures, to the diagnosis of hippocampal sclerosis, to surgical intervention, and then to dealing
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3

Simoens, Steven. "Health Technology Assessment of Lacosamide as Adjunctive Therapy for Partial-Onset Epileptic Seizures." In Novel Treatment of Epilepsy. InTech, 2011. http://dx.doi.org/10.5772/19425.

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Laura, A. "Epilepsy, my experience. The impact of compassion and hope amidst stigma and suicide." In Injured Brains of Medical Minds II. Oxford University PressOxford, 2024. https://doi.org/10.1093/oso/9780198890034.003.0066.

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Abstract This chapter assembles four articles that delve into the personal experiences of those with epilepsy. Article 1 presents a family doctor’s narrative, highlighting the role of compassion and hope in confronting epilepsy-related stigma and low mood. Article 2 offers the reflections of a medical student on the stigma surrounding epilepsy and the complexities of disclosing such a diagnosis. Article 3 chronicles a paediatrician’s journey with epilepsy, from the early onset of complex partial seizures, to the diagnosis of hippocampal sclerosis, to surgical intervention, and then to dealing
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5

Kapur, Narinder, Vaughan Bell, Daniel Friedland, and Steven Kemp. "Books by doctors/scientists recounting their personal experience of epilepsy." In Injured Brains of Medical Minds II. Oxford University PressOxford, 2024. https://doi.org/10.1093/oso/9780198890034.003.0069.

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Abstract This chapter assembles four articles that delve into the personal experiences of those with epilepsy. Article 1 presents a family doctor’s narrative, highlighting the role of compassion and hope in confronting epilepsy-related stigma and low mood. Article 2 offers the reflections of a medical student on the stigma surrounding epilepsy and the complexities of disclosing such a diagnosis. Article 3 chronicles a paediatrician’s journey with epilepsy, from the early onset of complex partial seizures, to the diagnosis of hippocampal sclerosis, to surgical intervention, and then to dealing
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6

Scott, C. "Reflections of a medical student with epilepsy. A case report." In Injured Brains of Medical Minds II. Oxford University PressOxford, 2024. https://doi.org/10.1093/oso/9780198890034.003.0067.

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Abstract This chapter assembles four articles that delve into the personal experiences of those with epilepsy. Article 1 presents a family doctor’s narrative, highlighting the role of compassion and hope in confronting epilepsy-related stigma and low mood. Article 2 offers the reflections of a medical student on the stigma surrounding epilepsy and the complexities of disclosing such a diagnosis. Article 3 chronicles a paediatrician’s journey with epilepsy, from the early onset of complex partial seizures, to the diagnosis of hippocampal sclerosis, to surgical intervention, and then to dealing
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7

Tegelberg, Saara, Tarja Joensuu, and Anna-Elina Lehesjoki. "Progressive Myoclonus Epilepsy." In Jasper's Basic Mechanisms of the Epilepsies, 5th ed., edited by Antonio V. Delgado-Escueta, José M. Serratosa, and Berge A. Minassian. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197549469.003.0051.

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Abstract Unverricht-Lundborg disease (ULD; EPM1) is an inherited neurodegenerative disorder characterized by onset at 6–15 years, stimulus-sensitive, action-activated myoclonus, epilepsy, and progressive neurological deterioration. It is caused by biallelic pathogenic variants in the CSTB gene, encoding a cystatin B. The most common of these is an unstable expansion of a dodecamer repeat element in the promoter region of the gene, leading to marked downregulation of CSTB expression. Total loss of CSTB is associated with severe neonatal-onset encephalopathy. A cystatin B–deficient mouse models
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8

Chassoux, Francine, Elisabeth Landré, and Bertrand Devaux. "Invasive EEG in Tumoural Epilepsy." In Invasive Studies of the Human Epileptic Brain, edited by Samden D. Lhatoo, Philippe Kahane, and Hans O. Lüders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198714668.003.0016.

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Long-term epilepsy-associated tumours (LEATs) are usually dysembryoplastic neuroepithelial tumours (DNTs) and gangliogliomas. Both usually show childhood-onset intractable partial epilepsy, concordant focal EEG abnormalities, and predominant temporal location. Imaging showing typical pseudocystic pattern is suggestive of DNT. Gangliogliomas are characterized by association of a true cyst and nodular contrast enhancement. However, less typical patterns may also be seen, rendering differentiation between the two difficult. Stereo-EEG has demonstrated intrinsic epileptogenicity of both, the typic
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Olson, Donald, Tuomas Westermarck,, and Shirley W. Ekvall. "Seizures And Epilepsy." In Pediatric Nutrition In Chronic Diseases And Developmental Disorders. Oxford University PressNew York, NY, 2005. http://dx.doi.org/10.1093/oso/9780195165647.003.0010.

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Abstract Seizures occur in 10% of people at some time during their lives. Seizures are sudden, discrete episodes of abnormal behavior caused by abnormal hypersynchronous firing of neurons in the cerebral cortex. They are classified as generalized when both hemispheres are involved or partial ( focal) when limited to an area in one hemisphere. Commonly recognized generalized seizure types are generalized tonic-clonic seizures (formerly called grand mal) and absence seizures (formerly called petit mal). Other types of generalized seizures are tonic, atonic, and myoclonic seizures as well as most
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Prince, David A., and Feng Gu. "Prophylaxis of Epileptogenesis in Injury and Genetic Epilepsy Models." In Jasper's Basic Mechanisms of the Epilepsies, 5th ed., edited by Michael A. Rogawski. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197549469.003.0068.

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Abstract The final common path for epileptogenesis involves pathological processes that enhance excitatory and/or decrease inhibitory synaptic transmission. In models involving traumatic- or status epilepticus–induced injury, both mechanisms underlie the epileptogenic process. Past results of the posttraumatic chronic neocortical isolation (UC) model suggested that limiting excessive excitatory connectivity with gabapentin and providing trophic support for defective GABAergic interneurons may serve as potential antiepileptogenic approaches following brain trauma. Results prompted further exper
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Actas de conferencias sobre el tema "Partial-onset epilepsy"

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Dias, Aline de Fátima, Lucas Scárdua Silva, Rafael Batista João, et al. "Enzyme-inducing antiseizure drugs associate with low bone mineral density in men with epilepsy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.155.

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Introduction: Little is known about the impact of enzyme-inducing antiseizure drugs (EI-ASD) on the reduction of Bone Mineral Density (BMD) in men with epilepsy (MWE). Objectives: To evaluate the BMD in MWE exposed to EI-ASDs (phenytoin, carbamazepine and phenobarbital) and its relationship with the duration of epilepsy. Methods: We evaluated BMD from 74 consecutive MWE (median age (range), 52.5 (25- 74) years) exposed to previous or current EI-ASDs, followed at UNICAMP-Brazil. Individuals were split into two groups (young-group, 31 individuals [25-49 years]; older group, 43 subjects, [50-74 y
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