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Artículos de revistas sobre el tema "Pathogenic"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 9 de junio de 2025

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1

Gvozdyak, R. I. "«Pathogen-1» Experiment Aggression of pathogenic bacteria in microgravity." Kosmìčna nauka ì tehnologìâ 6, no. 4 (July 30, 2000): 111. http://dx.doi.org/10.15407/knit2000.04.119.

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2

Ruchel, Reinhard. "Proteinasen pathogener Pilze: Proteinases of pathogenic fungi." Mycoses 42, S1 (April 1999): 48–52. http://dx.doi.org/10.1111/j.1439-0507.1999.tb04527.x.

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3

Luzzatto, Lucio, Caterina Nannelli, and Rosario Notaro. "Potentially pathogenic and pathogenic G6PD variants." American Journal of Human Genetics 110, no. 11 (November 2023): 1983–85. http://dx.doi.org/10.1016/j.ajhg.2023.10.003.

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4

Gharwalova, Lucia, Marketa Kulisova, Anastasiia Vasyliuk, Helena Maresova, Andrea Palyzova, Linda Nedbalova, and Irena Kolouchova. "Sphingolipids of plant pathogenic fungi." Plant Protection Science 57, No. 2 (March 1, 2021): 134–39. http://dx.doi.org/10.17221/131/2020-pps.

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Glycosphingolipids in filamentous fungi are significant components of the plasma membrane and are vital for different cellular processes, such as growth, morphological transition or signal transduction. Fungal growth inhibitors targeting glycosylinositolphosphoceramide (GIPCs) biosynthesis or antifungal compounds binding to GIPCs present in membranes could present a safe way of preventing fungal growth on crops since GIPCs are not present in mammalian cells. Mass spectrometry-based shotgun lipidomics was used to analyze sphingolipids of 11 fungal strains isolated from plant material. Molecular species with inositol ceramides containing zero to five carbohydrates were identified. Differences in the amount of individual molecular species were influenced by the taxonomic affiliation. All tested strains exhibited a relatively high content (more than 40 mol.%) of GIPCs with three and more saccharides attached to the polar head. It could be assumed that the sphingolipid profiles of the tested plant pathogens would be an adaptation mechanism to antifungal plant defensins.
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5

Finn, Albert F., and Peter D. Gorevic. "Pathogenic paraproteins." Current Opinion in Rheumatology 2, no. 4 (August 1990): 652–60. http://dx.doi.org/10.1097/00002281-199002040-00017.

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6

Horvat, Rebecca T. "PATHOGENIC FUNGI." Shock 30, no. 6 (December 2008): 753. http://dx.doi.org/10.1097/01.shk.0000336210.36795.86.

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7

Gould, EA, and T. Solomon. "Pathogenic flaviviruses." Lancet 371, no. 9611 (February 2008): 500–509. http://dx.doi.org/10.1016/s0140-6736(08)60238-x.

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8

DIPERRI, G. "PATHOGENIC ENTAMOEBA." Lancet 331, no. 8595 (May 1988): 1166. http://dx.doi.org/10.1016/s0140-6736(88)91980-0.

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9

Gao, Shou-Jiang. "Pathogenic procedures." Trends in Microbiology 5, no. 3 (March 1997): 125–26. http://dx.doi.org/10.1016/s0966-842x(97)87506-3.

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10

Tran Van Nhieu, Guy. "Pathogenic paradox?" Trends in Microbiology 7, no. 3 (March 1999): 102. http://dx.doi.org/10.1016/s0966-842x(99)01473-0.

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11

Casci, Tanita. "Pathogenic conversions." Nature Reviews Genetics 13, no. 1 (December 16, 2011): 2. http://dx.doi.org/10.1038/nrg3143.

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12

Dempsey, Laurie A. "Pathogenic antibodies." Nature Immunology 20, no. 11 (October 22, 2019): 1414. http://dx.doi.org/10.1038/s41590-019-0535-6.

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13

POOLMAN, J. "Pathogenic neisseriae." Lancet 336, no. 8722 (October 1990): 1061. http://dx.doi.org/10.1016/0140-6736(90)92518-m.

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14

Prasanna, Arun N., and Sarika Mehra. "Comparative Phylogenomics of Pathogenic and Non-Pathogenic Mycobacterium." PLoS ONE 8, no. 8 (August 28, 2013): e71248. http://dx.doi.org/10.1371/journal.pone.0071248.

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15

Foley, J. F. "Detecting a Pathogenic Activity, Not a Pathogenic Molecule." Science Signaling 7, no. 343 (September 16, 2014): ec252-ec252. http://dx.doi.org/10.1126/scisignal.2005897.

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16

Thongboonkerd, Visith, Wararat Chiangjong, Putita Saetun, Supachok Sinchaikul, Shui-Tein Chen, and Uraiwan Kositanont. "Analysis of differential proteomes in pathogenic and non-pathogenic Leptospira : Potential pathogenic and virulence factors." PROTEOMICS 9, no. 13 (July 2009): 3522–34. http://dx.doi.org/10.1002/pmic.200700855.

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17

Bøe, Johs. "On the Distinction Between Pathogenic and Non-Pathogenic Staphylococci." Acta Pathologica Microbiologica Scandinavica 21, no. 5 (August 14, 2009): 721–30. http://dx.doi.org/10.1111/j.1699-0463.1944.tb04972.x.

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18

Wurtzel, Omri, Nina Sesto, J. R. Mellin, Iris Karunker, Sarit Edelheit, Christophe Bécavin, Cristel Archambaud, Pascale Cossart, and Rotem Sorek. "Comparative transcriptomics of pathogenic and non‐pathogenic Listeria species." Molecular Systems Biology 8, no. 1 (January 2012): 583. http://dx.doi.org/10.1038/msb.2012.11.

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19

DiMiceli, Lauren. "Distinguishing Between Pathogenic and Non-Pathogenic Species of Entamoeba." Laboratory Medicine 35, no. 10 (October 1, 2004): 613–15. http://dx.doi.org/10.1309/b81npvaw8y4bgy11.

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20

Roberts, Glenn D. "Medical Mycology: The Pathogenic Fungi and the Pathogenic Actinomycetes." Mayo Clinic Proceedings 63, no. 10 (October 1988): 1061–62. http://dx.doi.org/10.1016/s0025-6196(12)64931-3.

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21

Prasad, Rajendra, Frédéric Devaux, Sanjiveeni Dhamgaye, and Dibyendu Banerjee. "Response of pathogenic and non-pathogenic yeasts to steroids." Journal of Steroid Biochemistry and Molecular Biology 129, no. 1-2 (March 2012): 61–69. http://dx.doi.org/10.1016/j.jsbmb.2010.11.011.

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22

Hopsu-Havu, V. K., C. E. Sonck, and Elvi Tunnela. "Production of Elastase by Pathogenic and Non-Pathogenic Fungi." Mycoses 15, no. 3 (April 24, 2009): 105–10. http://dx.doi.org/10.1111/j.1439-0507.1972.tb01357.x.

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23

Ahearn, Donald G. "Medical Mycology: The Pathogenic Fungi and the Pathogenic Actinomycetes." JAMA: The Journal of the American Medical Association 260, no. 12 (September 23, 1988): 1794. http://dx.doi.org/10.1001/jama.1988.03410120140051.

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24

Gaminda, K. A. P. "DEOXYRIBOZYMES IN DETECTION OF PATHOGENIC BACTERIA." Biotechnologia Acta 14, no. 5 (October 2021): 5–20. http://dx.doi.org/10.15407/biotech14.05.005.

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Aim. The purpose of the review was to analyze the use of DNAzyme biosensors for the detection of pathogens. In the recent years, deoxyribozymes (DNAzymes) have a significant impact as biosensors in diverse fields, from detection of metal ions in the environment to theranostic applications and detection of microorganisms. Although routinely used sophisticated instrumental methods are available to detect pathogenic bacterial contamination, they involve time-consuming, complicated sample pre-treatment and expensive instruments. As an alternative, pathogen-specific DNAzymes have demonstrated a series of advantages: a non-destructive rapid analysis technique with in situ and real-time detection of bacteria with high sensitivity and selectivity. A wide range of pathogen-specific DNAzymes has been developed using colorimetric and fluorescence-based detections for pathogenic bacterial contamination in various samples. The current review summarizes the in vitro selection of pathogen-specific DNAzymes, various strategies utilized in the sensor designs, and their potential use in theranostic applications.
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25

Orlyankin, B. G., and T. I. Aliper. "Porcine pathogenic viruses." "Veterinary Medicine" Journal 23, no. 01 (January 2020): 03–08. http://dx.doi.org/10.30896/0042-4846.2020.23.1.03-08.

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26

Kaper, James B., James P. Nataro, and Harry L. T. Mobley. "Pathogenic Escherichia coli." Nature Reviews Microbiology 2, no. 2 (February 2004): 123–40. http://dx.doi.org/10.1038/nrmicro818.

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27

Parkhill, Julian, and Colin Berry. "Relative pathogenic values." Nature 423, no. 6935 (May 2003): 23–24. http://dx.doi.org/10.1038/423023a.

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28

Suerbaum, Sebastian, and Torkel Wadström. "Bacterial pathogenic factors." Current Opinion in Gastroenterology 11 (1995): 11–15. http://dx.doi.org/10.1097/00001574-199501001-00003.

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29

Figura, Natale, and Soad Tabaqchali. "Bacterial pathogenic factors." Current Opinion in Gastroenterology 12 (January 1996): 11–15. http://dx.doi.org/10.1097/00001574-199601001-00003.

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30

Bisel, Ryan S., and Debra J. Ford. "Diagnosing Pathogenic Eschatology." Communication Studies 59, no. 4 (November 21, 2008): 340–54. http://dx.doi.org/10.1080/10510970802467395.

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31

Charlotte, Harrison. "Identifying pathogenic pathways." Nature Reviews Drug Discovery 12, no. 7 (July 2013): 506. http://dx.doi.org/10.1038/nrd4061.

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32

Paredes-Sabja, Daniel, Marjorie Pizarro-Guajardo, and Joseph A. Sorg. "Editorial: Pathogenic clostridia." Anaerobe 90 (December 2024): 102911. http://dx.doi.org/10.1016/j.anaerobe.2024.102911.

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33

Barešić, Anja, and Andrew C. R. Martin. "Compensated pathogenic deviations." BioMolecular Concepts 2, no. 4 (August 1, 2011): 281–92. http://dx.doi.org/10.1515/bmc.2011.025.

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AbstractDeleterious or ‘disease-associated’ mutations are mutations that lead to disease with high phenotype penetrance: they are inherited in a simple Mendelian manner, or, in the case of cancer, accumulate in somatic cells leading directly to disease. However, in some cases, the amino acid that is substituted resulting in disease is the wild-type native residue in the functionally equivalent protein in another species. Such examples are known as ‘compensated pathogenic deviations’ (CPDs) because, somewhere in the second species, there must be compensatory mutations that allow the protein to function normally despite having a residue which would cause disease in the first species. Depending on the nature of the mutations, compensation can occur in the same protein, or in a different protein with which it interacts. In principle, compensation can be achieved by a single mutation (most probably structurally close to the CPD), or by the cumulative effect of several mutations. Although it is clear that these effects occur in proteins, compensatory mutations are also important in RNA potentially having an impact on disease. As a much simpler molecule, RNA provides an interesting model for understanding mechanisms of compensatory effects, both by looking at naturally occurring RNA molecules and as a means of computational simulation. This review surveys the rather limited literature that has explored these effects. Understanding the nature of CPDs is important in understanding traversal along fitness landscape valleys in evolution. It could also have applications in treating diseases that result from such mutations.
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34

Jauniaux, E. "I158 PATHOGENIC MECHANISMS." International Journal of Gynecology & Obstetrics 119 (October 2012): S199—S200. http://dx.doi.org/10.1016/s0020-7292(12)60188-x.

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35

Angell, E. "Pathogenic waste treatment." Environment International 23, no. 3 (1997): IX—X. http://dx.doi.org/10.1016/s0160-4120(97)88023-3.

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36

Kaper, James B. "Pathogenic Escherichia coli." International Journal of Medical Microbiology 295, no. 6-7 (October 2005): 355–56. http://dx.doi.org/10.1016/j.ijmm.2005.06.008.

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37

Fattakhov, R. "Genesis pathogenic ecosystems." Parasitology International 47 (August 1998): 329. http://dx.doi.org/10.1016/s1383-5769(98)80967-7.

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38

Bahcall, Orli G. "Classifying pathogenic variation." Nature Reviews Genetics 16, no. 3 (February 18, 2015): 131. http://dx.doi.org/10.1038/nrg3915.

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39

Holmes, Edward C. "Virology: Pathogenic passengers." Nature 478, no. 7369 (October 2011): 319–20. http://dx.doi.org/10.1038/478319a.

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40

Shoenfeld, Yehuda. "Pathogenic natural autoantibodies." Clinical Immunology Newsletter 13, no. 2-3 (February 1993): 13–19. http://dx.doi.org/10.1016/0197-1859(93)90020-k.

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41

Wackett, Lawrence P. "Plant pathogenic microorganisms." Environmental Microbiology 17, no. 10 (October 2015): 4143–44. http://dx.doi.org/10.1111/1462-2920.13067.

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42

Kiberstis, P. A. "NEUROSCIENCE: Pathogenic Tangles." Science 287, no. 5462 (March 31, 2000): 2377f—2377. http://dx.doi.org/10.1126/science.287.5462.2377f.

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43

Voelkner, Nadine. "Managing pathogenic circulation." Security Dialogue 42, no. 3 (June 2011): 239–59. http://dx.doi.org/10.1177/0967010611405393.

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44

Broder, Samuel. "Pathogenic Human Retroviruses." New England Journal of Medicine 318, no. 4 (January 28, 1988): 243–45. http://dx.doi.org/10.1056/nejm198801283180409.

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45

H., G. C., and J. A. von Arx. "Plant Pathogenic Fungi." Mycologia 79, no. 6 (November 1987): 919. http://dx.doi.org/10.2307/3807701.

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46

Calam, John. "4 Pathogenic mechanisms." Baillière's Clinical Gastroenterology 9, no. 3 (September 1995): 487–506. http://dx.doi.org/10.1016/0950-3528(95)90044-6.

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47

Solbrig, Marylou V. "Human pathogenic arenaviruses." Annals of Neurology 64, no. 3 (February 25, 2008): 355–56. http://dx.doi.org/10.1002/ana.21350.

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48

van West, Pieter, and Gordon W. Beakes. "Animal pathogenic Oomycetes." Fungal Biology 118, no. 7 (July 2014): 525–26. http://dx.doi.org/10.1016/j.funbio.2014.05.004.

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49

Harborne, Jeffrey B. "Plant pathogenic bacteria." Phytochemistry 27, no. 5 (January 1988): 1569–70. http://dx.doi.org/10.1016/0031-9422(88)80251-6.

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50

Filip, Z., D. Kaddu-Mulindwa, and G. Milde. "Survival of Some Pathogenic and Facultative Pathogenic Bacteria in Groundwater." Water Science and Technology 20, no. 3 (March 1, 1988): 227–31. http://dx.doi.org/10.2166/wst.1988.0105.

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In model experiments carried out in the laboratory the survival of bacteria in groundwater kept at 10±l °C was tested. Only two of the tested bacteria species did not survive longer than 10 - 30 days. Escherichia coli, Salmonella typhimurium, Pseudomonas aeruginosa and other pathogenic or facultative pathogenic bacteria survived up to 100 days or even more in ground-water with or without the addition of sand from an aquifer. These results can be of importance for determining groundwater protection zones.
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