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Artículos de revistas sobre el tema "Post-streptococcal glomerulonephritis"

Autor: Grafiati
Publicado: 7 de junio de 2025
Última modificación: 2 de agosto de 2025

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1

Odalovic, A., N. Katanic, A. Milic, and A. Arsovic. "Post-streptococcal glomerulonephritis." Praxis medica 43, no. 1 (2014): 57–59. http://dx.doi.org/10.5937/pramed1401057o.

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2

Upton, C. J., and A. R. Watson. "POST-STREPTOCOCCAL GLOMERULONEPHRITIS." Lancet 330, no. 8570 (1987): 1269. http://dx.doi.org/10.1016/s0140-6736(87)91875-7.

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3

Ludwigsen, Erik, and Finn Hanberg SØrensen. "Post-Streptococcal Glomerulonephritis." Acta Pathologica Microbiologica Scandinavica Section A Pathology 86A, no. 1-6 (2009): 319–24. http://dx.doi.org/10.1111/j.1699-0463.1978.tb02050.x.

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4

Gracia, K. Falcones, R. Molina Gasset, and E. Ricart Alvarez. "Post-streptococcal glomerulonephritis." Clinica Chimica Acta 493 (June 2019): S463—S464. http://dx.doi.org/10.1016/j.cca.2019.03.979.

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5

Hsu, Yung-Hsiang. "Acute post-streptococcal glomerulonephritis." Tzu Chi Medical Journal 26, no. 3 (2014): 144. http://dx.doi.org/10.1016/j.tcmj.2013.09.012.

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6

Nand, N., N. B. Argent, A. R. Morley, and M. K. Ward. "Garland pattern post-streptococcal glomerulonephritis." Nephrology Dialysis Transplantation 7, no. 2 (1992): 155–57. http://dx.doi.org/10.1093/oxfordjournals.ndt.a092087.

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7

Savill, John, and Prof J. Savill. "Apoptosis in post-streptococcal glomerulonephritis." Kidney International 60, no. 3 (2001): 1203–14. http://dx.doi.org/10.1046/j.1523-1755.2001.0600031203.x.

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8

Berhane Alema, Haileselasie, Amanuel Alem, Meresa Gebremedhin, Tadis Birhane, and Abraham Mengstu. "Magnitude and associated factors of acute post streptococcal glomerulonepheritis among pediatric patients in aksum public hospitals, tigray, Ethiopia." MOJ Public Health 8, no. 6 (2019): 230–35. http://dx.doi.org/10.15406/mojph.2019.08.00312.

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Objective: Acute post streptococcus glomerulonephritis is reported from all over the world with higher rates among children than in adults. The objective of this study was to assess the magnitude and associate factors of acute post streptococcal glomerulonephritis among pediatric patients in Aksum city public hospitals, Tigray, Ethiopia. Methods: Institution based cross sectional study was conducted to retrieve data from 384 medical records of pediatric patients admitted to pediatric wards of Aksum city public hospitals between 2016 and 2018. The collected data were entered in to SPSS version
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9

Atadjanova, Shaira Khalilovna, Umida Abdukadirovna Arzibekova, and Gulrukh Shukhratjon kizi Abdukhalilova. "GLOMERULONEPHRITIS IN CHILDREN." Multidisciplinary Journal of Science and Technology 5, no. 2 (2025): 73–75. https://doi.org/10.5281/zenodo.14833447.

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10

Payne, D., P. Houtman, and M. Browning. "Acute post-streptococcal glomerulonephritis associated with prolonged hypocomplementaemia." Journal of Clinical Pathology 61, no. 10 (2008): 1133–35. http://dx.doi.org/10.1136/jcp.2008.056705.

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The case of a 6-year-old boy who presented with acute post-streptococcal glomerulonephritis is reported. C3 levels and complement alternative pathway activity remained low for at least 10 months after presentation, before returning to normal. There was no evidence of other renal disease. This case highlights that hypocomplementaemia in acute post-streptococcal glomerulonephritis may persist for several months, and that prolonged hypocomplementaemia does not exclude this diagnosis.
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11

Kagan, M. Yu. "Acute Post-Streptococcal Glomerulonephritis in Children." Current pediatrics 15, no. 1 (2016): 25–32. http://dx.doi.org/10.15690/vsp.v15i1.1497.

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12

Hart, D. H. L., C. Scheinkestel, J. A. Whitworth, and P. Kincaid-Smith. "Acute Post-Streptococcal Glomerulonephritis without Proteinuria." Journal of the Royal Society of Medicine 78, no. 10 (1985): 842–43. http://dx.doi.org/10.1177/014107688507801010.

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Two cases of histologically proven acute post-streptococcal glomerulonephritis presenting as congestive cardiac failure with normal blood pressure are reported. Proteinuria was not a finding. These features are discussed.
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13

Leung, D. T., R. Y. Tseng, S. H. Go, G. L. French, and C. W. Lam. "Post-streptococcal glomerulonephritis in Hong Kong." Archives of Disease in Childhood 62, no. 10 (1987): 1075–76. http://dx.doi.org/10.1136/adc.62.10.1075.

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14

Velibor, T., D. Kuzmanovska, and A. Sajkovski. "Post-streptococcal Glomerulonephritis in Hong Kong." Archives of Disease in Childhood 63, no. 3 (1988): 342. http://dx.doi.org/10.1136/adc.63.3.342.

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15

Fux, Christoph A., Mario G. Bianchetti, Stephan M. Jakob, and Luca Remonda. "REVERSIBLE ENCEPHALOPATHY COMPLICATING POST-STREPTOCOCCAL GLOMERULONEPHRITIS." Pediatric Infectious Disease Journal 25, no. 1 (2006): 85–87. http://dx.doi.org/10.1097/01.inf.0000195614.66987.2f.

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16

Oner, A., G. Demircin, and M. Bulbul. "Post-streptococcal acute glomerulonephritis in Turkey." Acta Paediatrica 84, no. 7 (1995): 817–19. http://dx.doi.org/10.1111/j.1651-2227.1995.tb13766.x.

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17

Butani, Lavjay. "Prolonged hypocomplementaemia after post‐streptococcal glomerulonephritis." Nephrology Dialysis Transplantation 16, no. 4 (2001): 869. http://dx.doi.org/10.1093/ndt/16.4.869.

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18

Zaffanello, Marco, and Michela Fedrizzi. "Delayed diagnosis of post-streptococcal glomerulonephritis." Journal of Pediatrics 154, no. 5 (2009): 777. http://dx.doi.org/10.1016/j.jpeds.2008.12.040.

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19

Reid, H. F. M., D. C. J. Bassett, E. Gaworzewska, G. Colman, and T. Poon-King. "Streptococcal serotypes newly associated with epidemic post-streptococcal acute glomerulonephritis." Journal of Medical Microbiology 32, no. 2 (1990): 111–14. http://dx.doi.org/10.1099/00222615-32-2-111.

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20

Demuynck, M., E. Lerut, D. Kuypers, et al. "POST-STREPTOCOCCAL GLOMERULONEPHRITIS: NOT AN EXTINCT DISEASE!" Acta Clinica Belgica 68, no. 3 (2013): 215–17. http://dx.doi.org/10.2143/acb.3292.

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21

Dowler, James, and Angela Wilson. "Acute post‐streptococcal glomerulonephritis in Central Australia." Australian Journal of Rural Health 28, no. 1 (2020): 74–80. http://dx.doi.org/10.1111/ajr.12568.

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22

Begolli, M., and I. Begolli. "986 Acute Post Streptococcal Glomerulonephritis in Children." Pediatric Research 68 (November 2010): 491. http://dx.doi.org/10.1203/00006450-201011001-00986.

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23

Annika Nordstrand, Mari Norgren, St. "Pathogenic Mechanism of Acute Post-Streptococcal Glomerulonephritis." Scandinavian Journal of Infectious Diseases 31, no. 6 (1999): 523–37. http://dx.doi.org/10.1080/00365549950164382.

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24

Wyatt, Robert J., Judith Forristal, Clark D. West, Sandra Sugimoto, and John G. Curd. "Complement profiles in acute post-streptococcal glomerulonephritis." Pediatric Nephrology 2, no. 2 (1988): 219–23. http://dx.doi.org/10.1007/bf00862594.

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25

Kaplan, Richard A., David L. Zwick, Stanley Hellerstein, Bradley A. Warady, and Uri Alon. "Cerebral vasculitis in acute post-streptococcal glomerulonephritis." Pediatric Nephrology 7, no. 2 (1993): 194–95. http://dx.doi.org/10.1007/bf00864396.

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26

Burova, Larisa A., Alexander N. Suvorov, Peter V. Pigarevsky, and Artem A. Totolian. "Autoimmune streptococcal glomerulonephritis: the problem of nephritogenicity of <i>Streptococcus pyogenes</i>." Russian Journal of Infection and Immunity 13, no. 3 (2023): 409–29. http://dx.doi.org/10.15789/2220-7619-asg-8491.

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Acute post-streptococcal glomerulonephritis usually occurs as a complication after a streptococcal infection due to untimely or inadequate antibiotic therapy. The etiology of post-streptococcal glomerulonephritis has been studied rather comprehensively. Today, both clinicians and microbiologists do not deny the dominant role of Streptococcus pyogenes (streptococcus attributed to serological group A, GAS). Usually, emergence of acute post-streptococcal glomerulonephritis (APSGN) is associated with the so-called GAS-related "nephritogenicity" often judged by appearance and accumulation of antibo
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27

Ali, Derakhshan. "Persistent hypocomplementemia in a 9 year old boy following acute post-streptococcal glomerulonephritis." Archives of Renal Diseases and Management 3, no. 2 (2017): 049–50. https://doi.org/10.17352/2455-5495.000030.

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Acute&nbsp; post-streptococcal&nbsp; glomerulonephritis&nbsp; (APSGN)&nbsp; is&nbsp; a common&nbsp; form&nbsp; of&nbsp; acute glomerulonephritis&nbsp; in&nbsp; children&nbsp; [1].&nbsp; It is&nbsp; presented&nbsp; with&nbsp; edema,&nbsp; hematuria,&nbsp; acute&nbsp; renal&nbsp; failure&nbsp; and hypertension and hypocomplementemia 7-10days following agroup&nbsp; a -hemolytic&nbsp; streptococcal&nbsp; pharyngitis&nbsp; or&nbsp; 2-4&nbsp; weeks following an impetigo [2-4]. Subclinical forms are common in close&nbsp; contacts&nbsp; [5-7].&nbsp; APSGN&nbsp; usually&nbsp; is&nbsp; a&nbsp; self-lim
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28

Azmiyatie, Mumtaza A., Adrian Umboh, and Valentine Umboh. "Gambaran Klinik dan Laboratorium Glomerulonefritis Akut Pasca Streptokokus pada Anak." e-CliniC 12, no. 1 (2023): 47–56. http://dx.doi.org/10.35790/ecl.v12i1.45235.

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Abstract: Acute post-streptococcal glomerulonephritis (APSGN) is the most common form of acute glomerulonephritis (AGN) that often occurs in children caused by group A β-hemolytic streptococcal infection. This disease is a common cause of child morbidity and mortality in low and middle-income countries. APSGN has a typical clinical features such as nephritic symptoms and the diagnosis is confirmed by laboratory tests. This study aimed to determine the clinical features and laboratory findings of acute post-streptococcal glomerulonephritis in children. This was a literature review study with jo
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29

Roy, Ranjit Ranjan, and Kamrul Laila. "Acute Post-Streptococcal Glomerulonephritis in Children – A Review." Bangladesh Journal of Child Health 38, no. 1 (2014): 32–39. http://dx.doi.org/10.3329/bjch.v38i1.20025.

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Acute post-streptococcal glomerulonephritis (APSGN) is characterized by abrupt onset of hematuria, edema, hypertension, oliguria and impaired renal function following streptococcal group A â hemolytic streptococcal throat and skin infection. There is a declining incidence of APSGN worldwide, particularly in industrialized nations because of easier and earlier access to competent medical treatment of streptococcal infections and the widespread use of fluorination of water since virulence factors in streptococcus pyogens are reduced with fluoride exposure. But in the underdeveloped world, global
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30

Alves, Giordano Rafael Tronco, Isadora Cristina Olesiak Cordenonsi, Régis Vinícius de Andrade Silva, and Carlos Jesus Pereira Haygert. "Posterior reversible encephalopathy syndrome following post-streptococcal glomerulonephritis." Bangladesh Journal of Medical Science 12, no. 3 (2013): 341–43. http://dx.doi.org/10.3329/bjms.v12i3.13323.

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Posterior reversible encephalopathy syndrome (PRES) comprises a unique pattern of brain vasogenic edema that is seen in the setting of a neurotoxic status. Besides many etiologies have been already associated with PRES development, such as chronic renal disease, use of chemotherapy agents and inflammatory conditions, the imaging features are very suggestive and helpful for an appropriate diagnosis. We report here a case of PRES secondary to post-streptococcal glomerulonephritis (PSGN), which evolved successfully after clinical management. An 11-year-old boy was admitted with a typical history
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31

Hammad, Ayman, Ashraf Bakr, Gihan Fathy, Manal Fathy, and Rizk El Baz. "Serum Nitric Oxide in Post-Streptococcal Acute Glomerulonephritis." GEGET 2, no. 1 (2002): 33–36. http://dx.doi.org/10.21608/geget.2002.48370.

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32

El-Morshedy, Salah, and Magy Fawzy. "Outcome of Post-Streptococcal Crescentic Glomerulonephritis in Children." GEGET 10, no. 1 (2010): 15–29. http://dx.doi.org/10.21608/geget.2010.16897.

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33

Turner, Neil. "Acute nephritis in 1875: recognising post-streptococcal glomerulonephritis." Journal of Renal Nursing 7, no. 5 (2015): 258. http://dx.doi.org/10.12968/jorn.2015.7.5.258.

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34

Usui, J., I. Ebihara, S. Kaneko, M. Kobayashi, and K. Yamagata. "Peritubular capillary lesions in post-streptococcal acute glomerulonephritis." Clinical Kidney Journal 3, no. 1 (2009): 91–92. http://dx.doi.org/10.1093/ndtplus/sfp130.

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35

Gupta, Shalu, Vishnu Kumar Goyal, and B. Talukdar. "Reversible posterior leucoencephalopathy syndrome in post streptococcal glomerulonephritis." Indian Pediatrics 47, no. 3 (2010): 274–76. http://dx.doi.org/10.1007/s13312-010-0037-y.

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36

Rovang, Ronald D., Edward T. Zawada, Jr., Robert N. Santella, Richard A. Jaqua, John L. Boice, and Randal L. Welter. "Cerebral Vasculitis Associated with Acute Post-Streptococcal Glomerulonephritis." American Journal of Nephrology 17, no. 1 (1997): 89–92. http://dx.doi.org/10.1159/000169077.

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37

Muguruma, T., T. Koyama, T. Kanadani, M. Furujo, H. Shiraga, and Y. Ichiba. "Acute thrombocytopenia associated with post-streptococcal acute glomerulonephritis." Journal of Paediatrics and Child Health 36, no. 4 (2000): 401–2. http://dx.doi.org/10.1046/j.1440-1754.2000.00521.x.

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38

Hibino, Satoshi, Akihiro Hoshino, Takanari Fujii, et al. "Post-streptococcal acute glomerulonephritis associated with pneumococcal infection." Pediatrics International 55, no. 5 (2013): e136-e138. http://dx.doi.org/10.1111/ped.12150.

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39

Demircioglu Kılıc, Beltinge, Mehtap Akbalık Kara, Mithat Buyukcelik, and Ayse Balat. "Pediatric post-streptococcal glomerulonephritis: Clinical and laboratory data." Pediatrics International 60, no. 7 (2018): 645–50. http://dx.doi.org/10.1111/ped.13587.

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40

Kari, JameelaA, Ahmed Bamagai, and SawsanM Jalalah. "Severe acute post-streptococcal glomerulonephritis in an infant." Saudi Journal of Kidney Diseases and Transplantation 24, no. 3 (2013): 546. http://dx.doi.org/10.4103/1319-2442.111061.

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41

Feldon, Michal, Lev Dorfman, Tsivia Tauber, Yair Morad, Tzvy Bistritzer, and Michael Goldman. "Post-streptococcal glomerulonephritis and uveitis—a case report." Pediatric Nephrology 25, no. 11 (2010): 2351–53. http://dx.doi.org/10.1007/s00467-010-1550-x.

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42

Sorof, Jonathan M., Noel Weidner, Donald Potter, and Anthony A. Portalel. "Acute post-streptococcal glomerulonephritis in a renal allograft." Pediatric Nephrology 9, no. 3 (1995): 317–19. http://dx.doi.org/10.1007/bf02254197.

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43

Suresh, Jayasurya, and Nidheesh Chandran R. "Simultaneous presentation of post infectious glomerulonephritis and acute rheumatic fever in an eight year old girl child." International Journal of Contemporary Pediatrics 7, no. 8 (2020): 1810. http://dx.doi.org/10.18203/2349-3291.ijcp20203184.

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An 8-year-old female child presented with simultaneous symptoms of post infectious glomerulonephritis and acute rheumatic fever. The child was treated with penicillin V, Aspirin and she responded well to the treatment. However, this co presentation of acute rheumatic fever and post infectious glomerulonephritis in a child is rare and hence authors report the case. This sequela of streptococcal autoimmunity are considered to be pathogenetically distinct.
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44

Barnham, M. "Streptococcal infection in general practice." Epidemiology and Infection 109, no. 2 (1992): 177–80. http://dx.doi.org/10.1017/s0950268800050135.

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The last 30 years have been changes in emphasis in the study of streptococci and streptococcal diseases. Earlier work concentrated mainly on the sources and methods of cross-infection and descriptive epidemiology of Streptococcus pyogenes in its major manifestations of respiratory, cutaneous and invasive infection and in the complications of rheumatic fever (RF), scarlet fever (SF) and post-streptococcal glomerulonephritis (PSGN).
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45

Pedro do Valle Varela, João, Matheus Alves Ribeiro, Layane Aiala de Sousa Lopes, et al. "SCARLET FEVER IN PEDIATRICS." Health and Society 5, no. 01 (2025): 125–33. https://doi.org/10.51249/hs.v5i01.2417.

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Scarlet fever is a bacterial infection caused by *Streptococcus pyogenes* which predominantly affects school-age children. Although its prevalence has decreased over the years due to the advance of antibiotics, outbreaks still occur, requiring special attention regarding early diagnosis and the prevention of post-streptococcal complications, such as rheumatic fever and glomerulonephritis. The aim of this study is to review updates in the diagnosis of scarlet fever in pediatrics, emphasizing clinical and laboratory methods, as well as assessing the impact of post-streptococcal complications on
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46

Talal, Mohammed Aldossary Abdulrahman Mohammed Alrowis Abdulaziz Thamer Alkhouzaie Saleh Mohammed Alsaikhan Nourah Ebrahim Alamri Rakan Ridha Alanazi Mohammed Salim Batarfi Mohammed Abdulrahman Aldahoos Faisal Hamad Alharbi Gousay Abdullah Alkhazmari. "CLINICAL APPROACHES TOWARD ACUTE PROLIFERATIVE GLOMERULONEPHRITIS, CAUSES, AND TREATMENT." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 11 (2018): 12855–60. https://doi.org/10.5281/zenodo.1493250.

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<em>Acute glomerular inflammation results from a preceding infection with nephritogenic strains of streptococci.</em> <em>The prevention of further complications is essential, the background and treatment method of adults and specifically children, due to its prevalence in that age, is reviewed. </em><em>We performed narrative review of all relevant papers found through searches of Medline, Embase and Science Direct published in English language through 2018.</em><em> Acute proliferative glomerulonephritis (post-streptococcal glomerulonephritisis) is caused by an infection with streptococcus m
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47

Suarta, I. Ketut. "Erythrocyturia and proteinuria conversion in post-streptococcal acute glomerulonephritis." Paediatrica Indonesiana 46, no. 2 (2016): 71. http://dx.doi.org/10.14238/pi46.2.2006.71-6.

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Background Acute glomerulonephritis (AGN) is a sudden onsetof macroscopic hematuria and edema. The chronic post-strepto-coccal acute glomerulonephritis (PSAGN) can be predicted if mi-croscopic hematuria, proteinuria, and low serum complement C3level are present for a period exceeding six months after initial onsetof illness. It is prudent to follow the course of PSAGN until proteinurianormalizes and microhematuria disappears in urinalysis.Objective To acquire the time of erythrocyturia and proteinuriaconversion in post-streptococcal acute glomerulonephritis (PSAGN)among children.Methods A retr
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48

Rastaldi, M. P., F. Ferrario, L. Yang, et al. "Adhesion molecules expression in noncrescentic acute post-streptococcal glomerulonephritis." Journal of the American Society of Nephrology 7, no. 11 (1996): 2419–27. http://dx.doi.org/10.1681/asn.v7112419.

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Clinicomorphological features of 11 cases of non-crescentic acute post-streptococcal glomerulonephritis (APSGN) were reviewed. Intraglomerular and interstitial leukocytes and their possible correlation with the adhesion molecules intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), and endothelial-leukocyte adhesion molecule-1 (ELAM-1/E-selectin) were investigated by an immunohistochemical method. Intraglomerular leukocytes were primarily granulocytes (11.4 +/- 10 cells/glomerular cross-section) and monocytes-macrophages (13.4 +/- 19.4 cells/glomerular cross-
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49

Burova, L. A., E. A. Gavrilov, P. V. Pigarevsky, and Artem A. Totolian. "A role of streptokinase in experimental post-streptococcal glomerulonephritis." Russian Journal of Infection and Immunity 11, no. 5 (2021): 853–64. http://dx.doi.org/10.15789/2220-7619-aro-1594.

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Post-streptococcal glomerulonephritis (PSGN) refers to the sequela of the acute infection, caused by Streptococcus pyogenes (group A streptococcus, GAS). This pathology has been studied for a long time, and today attempts are being made to identify the products of their life activity, able to initiate an immunopathological process in kidneys. Most attention has been paid to streptokinase, the enzyme transforming blood plasminogen into plasmin, capable, together with the plasmin receptor (NAPlr), of damaging the glomerular tissue, as well as activating the complement system. The aim of the stud
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50

Kalainesan, K., L. M. Safeek, and S. Azher. "Post-streptococcal glomerulonephritis complicated with reversible posterior leukoencephalopathy syndrome." Sri Lanka Journal of Medicine 30, no. 2 (2021): 121. http://dx.doi.org/10.4038/sljm.v30i2.288.

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