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Artículos de revistas sobre el tema "Prostaglandin-f2-alpha"

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Publicado: 4 de junio de 2021

Última modificación: 1 de febrero de 2022

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1

Peters, A. "Effects of prostaglandin F2 alpha." Veterinary Record 118, no. 16 (1986): 466–67. http://dx.doi.org/10.1136/vr.118.16.466-b.

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2

Young, I. "Effects of prostaglandin F2-alpha." Veterinary Record 118, no. 19 (1986): 543–44. http://dx.doi.org/10.1136/vr.118.19.543-a.

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3

Barnard, J. W., R. A. Ward, and A. E. Taylor. "Evaluation of prostaglandin F2 alpha and prostacyclin interactions in the isolated perfused rat lung." Journal of Applied Physiology 72, no. 6 (1992): 2469–74. http://dx.doi.org/10.1152/jappl.1992.72.6.2469.

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To characterize the interactions between prostaglandin F2 alpha and prostacyclin in controlling tone in the pulmonary circulation, isolated rat lungs were ventilated, perfused with blood, and subjected to challenge by prostaglandin F2 alpha in increasing doses. The pulmonary resistance was evaluated using occlusion techniques that separate the resistance into segments of large and small arteries and veins. The total vascular compliance was evaluated using outflow occlusion. Resistance increased after prostaglandin F2 alpha, and this resistance change was primarily in the small artery segment.

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4

Bedrick, A. D., M. A. Wells, D. L. Ford, and O. Koldovsky. "Intact biliary excretion of gastrically administered prostaglandin F2 alpha in rats: developmental differences." American Journal of Physiology-Gastrointestinal and Liver Physiology 253, no. 6 (1987): G787—G792. http://dx.doi.org/10.1152/ajpgi.1987.253.6.g787.

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Tritium-labeled prostaglandin F2 alpha was administered via orogastric tube to bile duct-cannulated suckling and weanling rats to determine if maturational differences were present in the biliary excretion of prostaglandin F2 alpha and metabolites. Animals were killed 2 h after radioactivity administration. Characterization of radioactivity present in bile revealed age-related differences in biliary prostaglandin F2 alpha excretion. Suckling rats had a greater proportion of radioactivity migrating in chromatographic regions of greater polarity than prostaglandin F2 alpha. Compared with the wea

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5

Ramagopal, M. V., and S. J. Mustafa. "Effect of adenosine and its analogues on calcium influx in coronary artery." American Journal of Physiology-Heart and Circulatory Physiology 255, no. 6 (1988): H1492—H1498. http://dx.doi.org/10.1152/ajpheart.1988.255.6.h1492.

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In the present study, we have investigated the changes in calcium influx during the relaxing responses to adenosine and its analogues. Calcium-45 influx was measured in bovine coronary artery rings in the presence of prostaglandin F2 alpha (10(-5) M) and KCl (50 and 100 mM). Prostaglandin F2 alpha and KCl caused increases in calcium influx. Prostaglandin F2 alpha produced a further contraction when added to rings maximally contracted with KCl (100 mM or higher), suggesting two different mechanisms for prostaglandin F2 alpha- and KCl-induced contractions. Similarly, a greater calcium influx was

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6

Cocnata, Christina L. "Use of Prostaglandin F2 Alpha for Cyclophosphamide-Induced Hemorrhagic Cystitis." Journal of Pharmacy Technology 10, no. 5 (1994): 204–6. http://dx.doi.org/10.1177/875512259401000504.

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Objective: To examine the use of prostaglandin F2 alpha in treating cyclophosphamide-induced hemorrhagic cystitis. Data Sources: An English language literature search using MEDLINE 1982–1993 and bibliographic reviews of related textbooks and review articles. Study Selection: Articles containing pertinent information regarding the therapeutic use and effects of prostaglandin F2 alpha as a treatment for cyclophosphamide-induced hemorrhagic cystitis in humans. Data Extraction: Resources were evaluated and information was extracted independently. Data Synthesis: A review of human cases suggests th

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7

Diczfalusy, U., and S. E. Alexson. "Peroxisomal chain-shortening of prostaglandin F2 alpha." Journal of Lipid Research 29, no. 12 (1988): 1629–36. http://dx.doi.org/10.1016/s0022-2275(20)38412-1.

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8

Shigematsu, S., H. Niwa, and T. Saikawa. "Vasospastic angina induced by prostaglandin F2 alpha." Heart 69, no. 4 (1993): 364–65. http://dx.doi.org/10.1136/hrt.69.4.364.

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9

Norman, RJ, and K. Reddi. "Prostaglandins in dysfunctional labour; evidence for altered production of prostaglandin F2 alpha." Reproduction, Fertility and Development 2, no. 5 (1990): 563. http://dx.doi.org/10.1071/rd9900563.

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Dysfunctional labour was studied in relation to prostaglandin concentrations in amniotic fluid and production by fetal membranes. Initial clinical validation of the model established the presence of hypokinetic labour with no evidence of obstruction to the fetal progress. Prostaglandin F2 alpha (PGF2 alpha) and 13, 14 dihydro-15-keto-prostaglandin-F2 alpha concentrations in the amniotic fluid were low despite relatively normal concentrations of prostaglandin E2. Membranes removed from patients with the condition released very low concentrations of PGF2 alpha from the amniotic side with no alte

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10

Flint, AP, HN Jabbour, and AS Loudon. "Oxytocin stimulates uterine prostaglandin F2 alpha secretion in red deer Cervus elaphus." Reproduction, Fertility and Development 6, no. 2 (1994): 269. http://dx.doi.org/10.1071/rd9940269.

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The prostaglandin F2 alpha analogue cloprostenol stimulates ovarian secretion of oxytocin in red deer hinds and Pere David's deer hinds, as in cattle and sheep, but the response of the uterus to administered oxytocin has not been studied in deer. In the present experiment, oxytocin administered intravenously caused an increase in circulating concentrations of 13,14-dihydro-15-keto prostaglandin F2 alpha from 186 +/- 35 to 404 +/- 34 pmol L-1 within 5 min; concentrations in saline-treated hinds were unchanged (150 +/- 12 and 164 +/- 12 pmol L-1 before and after treatment respectively). This sug

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11

Douglas, M. J., D. F. Farquharson, P. L. E. Ross, and J. E. Renwick. "Cardiovascular Collapse Following an Overdose of Prostaglandin F2 Alpha." Obstetric Anesthesia Digest 9, no. 4 (1990): 237. http://dx.doi.org/10.1097/00132582-199001000-00057.

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12

Douglas, M. J., D. F. Farquharson, P. L. E. Ross, and J. E. Renwick. "Cardiovascular Collapse Following an Overdose of Prostaglandin F2 Alpha." Obstetric Anesthesia Digest 10, no. 2 (1990): 115. http://dx.doi.org/10.1097/00132582-199007000-00075.

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13

Liang, Z., S. R. Sooranna, N. Engineer, et al. "Prostaglandin F2-alpha receptor regulation in human uterine myocytes." Molecular Human Reproduction 14, no. 4 (2008): 215–23. http://dx.doi.org/10.1093/molehr/gan008.

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14

Collins, Danielle, Aisling M. Hogan, Alan W. Baird, and Des C. Winter. "Prostaglandin F2-alpha modulates fluid secretion in human colon." Journal of the American College of Surgeons 209, no. 3 (2009): S21. http://dx.doi.org/10.1016/j.jamcollsurg.2009.06.038.

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15

Lepak, N. M., and G. Serrero. "Prostaglandin F2 alpha stimulates transforming growth factor-alpha expression in adipocyte precursors." Endocrinology 136, no. 8 (1995): 3222–29. http://dx.doi.org/10.1210/endo.136.8.7628355.

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16

Négrel, R., D. Gaillard, and G. Ailhaud. "Prostacyclin as a potent effector of adipose-cell differentiation." Biochemical Journal 257, no. 2 (1989): 399–405. http://dx.doi.org/10.1042/bj2570399.

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The terminal differentiation of Ob1771 pre-adipose cells induced by arachidonic acid in serum-free hormone-supplemented medium containing insulin, transferrin, growth hormone, tri-iodothyronine and fetuin (5F medium) was strongly diminished in the presence of inhibitors of prostaglandin synthesis, namely aspirin or indomethacin. Carbaprostacyclin, a stable analogue of prostacyclin (prostaglandin I2) known to be synthesized by pre-adipocytes and adipocytes, behaved as an efficient activator of cyclic AMP production and was able, when added to 5F medium, to mimic the adipogenic effect of arachid

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17

Eiler, H., C. S. Armstrong-Backus, and W. A. Lyke. "Desensitization of rabbit myometrium to oxytocin and prostaglandin F2 alpha." American Journal of Physiology-Endocrinology and Metabolism 257, no. 1 (1989): E20—E26. http://dx.doi.org/10.1152/ajpendo.1989.257.1.e20.

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The desensitizing effects of oxytocin (OT) and prostaglandin F2 alpha (PGF2 alpha) were investigated in the uteri of rabbits. Uterine motility was measured in anesthetized rabbits infused intravenously with either PGF2 alpha (50 micrograms/min) or OT (100 mU/min) alternately. The treatment sequence was saline (30 min), first drug (OT or PGF2 alpha, 90 min), second drug (OT if PGF2 alpha was first drug and vice versa, 90 min), and first drug (repeated) 60 min. Both OT and PGF2 alpha infusions increased motility approximately 200% within 5–10 min. Thereafter, motility decreased linearly to base-

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18

YAMADA, Yutaka. "Effects of Prostaglandin F2.ALPHA. in Early Pregnancy in Swine." Nihon Yoton Gakkaishi 35, no. 2 (1998): 43–46. http://dx.doi.org/10.5938/youton.35.43.

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19

Ocklind, A., S. Lake, K. Krook, I. Hallin, M. Nistér, and B. Westermark. "Localization of the prostaglandin F2 alpha receptor in rat tissues." Prostaglandins, Leukotrienes and Essential Fatty Acids 57, no. 6 (1997): 527–32. http://dx.doi.org/10.1016/s0952-3278(97)90555-x.

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20

van Dam, P. A., I. B. Vergote, G. M. Laekeman, G. H. Keersmaeckers, F. L. Uyttenbroeck, and A. G. Herman. "Prognostic value of prostaglandin F2 alpha concentrations in breast carcinoma." Journal of Clinical Pathology 42, no. 10 (1989): 1046–48. http://dx.doi.org/10.1136/jcp.42.10.1046.

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21

Tasaki, Yukari, Hwa-Yong Lee, Jian-Ping Cui, Tomas J. Acosta, and Kiyoshi Okuda. "Local Autoamplification System for Prostaglandin F2 Alpha in Bovine Endometrium." Biology of Reproduction 83, Suppl_1 (2010): 419. http://dx.doi.org/10.1093/biolreprod/83.s1.419.

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22

Pinto, C. R. F., C. Rubio, B. E. Holland, et al. "Antiluteogenesis effects of prostaglandin F2-alpha (PGF) in cyclic mares." Journal of Equine Veterinary Science 34, no. 1 (2014): 163. http://dx.doi.org/10.1016/j.jevs.2013.10.116.

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23

Watanabe, K., Y. Iguchi, S. Iguchi, Y. Arai, O. Hayaishi, and L. J. Roberts. "Stereospecific conversion of prostaglandin D2 to (5Z,13E)-(15S)-9 alpha-11 beta,15-trihydroxyprosta-5,13-dien-1-oic acid (9 alpha,11 beta-prostaglandin F2) and of prostaglandin H2 to prostaglandin F2 alpha by bovine lung prostaglandin F synthase." Proceedings of the National Academy of Sciences 83, no. 6 (1986): 1583–87. http://dx.doi.org/10.1073/pnas.83.6.1583.

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24

Suematsu, E., M. Resnick, and K. G. Morgan. "Change of Ca2+ requirement for myosin phosphorylation by prostaglandin F2 alpha." American Journal of Physiology-Cell Physiology 261, no. 2 (1991): C253—C258. http://dx.doi.org/10.1152/ajpcell.1991.261.2.c253.

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The mechanism of contraction of vascular smooth muscle by prostaglandin F2 alpha (PGF2 alpha) was examined by simultaneous measurement of the intracellular Ca2+ concentration [( Ca2+]i), force, and myosin light-chain (MLC) phosphorylation in ferret aorta. In the presence of 2.5 mM extracellular Ca2+, PGF2 alpha (10(-5)M) produced a tonic contraction with a transient spike in [Ca2+]i, followed by a relatively small sustained increase in [Ca2+]i (from a basal level of 2.32 +/- 0.07 x 10(-7) to 2.72 +/- 0.05 x 10(-7) M). In Ca(2+)-free bathing media, PGF2 alpha also produced a tonic contraction w

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25

Fairclough, RJ, and TM Lau. "Hormonal control of concentrations of endometrial oxytocin receptors in the ewe." Reproduction, Fertility and Development 4, no. 3 (1992): 313. http://dx.doi.org/10.1071/rd9920313.

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Uterine oxytocin receptors have been shown to play a major role in the regulation of uterine prostaglandin F2 alpha release during the oestrous cycle and early pregnancy in sheep. The concentration of endometrial oxytocin receptors increases sharply from around Day 13 of the oestrous cycle to reach a maximum between Days 15 and 16. The high concentration of endometrial oxytocin receptors at this time coincides with the release of endogenous uterine prostaglandin F2 alpha during luteal regression and the maximum uterine prostaglandin F2 alpha response to an oxytocin stimulus. The concentration

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26

Erkiliç, Kuddusi, Faruk Ekinciler, Ertuğrul Mirza, Hakki Doğan, and Nurullah Çağil. "Effects of Topically Applied Prostaglandin F2 alpha on Normotensive Human Eyes." Ophthalmic Research 28, no. 6 (1996): 351–55. http://dx.doi.org/10.1159/000267927.

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27

Villumsen, J., and A. Alm. "Prostaglandin F2 alpha-isopropylester eye drops: effects in normal human eyes." British Journal of Ophthalmology 73, no. 6 (1989): 419–26. http://dx.doi.org/10.1136/bjo.73.6.419.

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28

Rib�ri, O., I. Sziklai, and J. G. Kiss. "Cyclic nucleotide and prostaglandin F2 alpha contents of otosclerotic auditory ossicles." Archives of Oto-Rhino-Laryngology 242, no. 1 (1985): 63–66. http://dx.doi.org/10.1007/bf00464408.

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29

YL, D. "Nitric oxide prevents prostaglandin F2$alpha;-induced preterm labor in rats." Journal of the Society for Gynecologic Investigation 3, no. 2 (1996): 331A. http://dx.doi.org/10.1016/1071-5576(96)82993-5.

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30

Dhaliwal, G. S., R. D. Sharma, and G. Singh. "Efficacy of prostaglandin F2-alpha administration for inducing estrus in buffalo." Theriogenology 29, no. 6 (1988): 1401–6. http://dx.doi.org/10.1016/0093-691x(88)90021-0.

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31

Leighton, B., L. Budohoski, F. J. Lozeman, R. A. J. Challiss та E. A. Newsholme. "The effect of prostaglandins E1, E2 and F2α and indomethacin on the sensitivity of glycolysis and glycogen synthesis to insulin in stripped soleus muscles of the rat". Biochemical Journal 227, № 1 (1985): 337–40. http://dx.doi.org/10.1042/bj2270337.

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Prostaglandins E1 and E2 increased the sensitivity of glycolysis to insulin in the isolated stripped soleus muscle of the rat, but prostaglandin F2 alpha had no effect. Indomethacin, which inhibits prostaglandin formation, markedly decreased the sensitivity of glycolysis to insulin. These findings suggest that prostaglandins of the E series increase the sensitivity of muscle glycolysis to insulin in vivo.

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32

Kawai, Y., and T. Ohhashi. "Prostaglandin F2 alpha-induced endothelium-dependent relaxation in isolated monkey cerebral arteries." American Journal of Physiology-Heart and Circulatory Physiology 260, no. 5 (1991): H1538—H1543. http://dx.doi.org/10.1152/ajpheart.1991.260.5.h1538.

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Effects of prostaglandin F2 alpha (PGF2 alpha) on isolated monkey and dog cerebral arteries were investigated to reevaluate PGF2 alpha's possible action on the endothelium. Low concentrations of PGF2 alpha ranging from 10(-11) to 10(-8) M produced a dose-dependent relaxation in the monkey arteries. PGF2 alpha (10(-7) M) produced a transient contraction followed by a small relaxation, whereas higher concentrations (greater than 10(-6) M) of PGF2 alpha induced only contractions. The PGF2 alpha-induced relaxation was not observed in the canine cerebral arteries. The PGF2 alpha-induced relaxation

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33

Gilbert, CL, TH Burne, JA Goode, PJ Murfitt, and SL Walton. "Indomethacin blocks pre-partum nest building behaviour in the pig (Sus scrofa): effects on plasma prostaglandin F metabolite, oxytocin, cortisol and progesterone." Journal of Endocrinology 172, no. 3 (2002): 507–17. http://dx.doi.org/10.1677/joe.0.1720507.

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In the pig, nest building occurs in the day preceding parturition (gestation=114--116 days). Nest building behaviour can be induced in pregnant, pseudopregnant and cyclic female pigs following injection of prostaglandin F2alpha. Here we investigated behaviour and endocrine changes after the administration of indomethacin, which inhibits cyclo-oxygenase enzymes and thus prostaglandin synthesis. In experiment 1, pregnant primiparous pigs (gilts) were blood sampled through jugular vein catheters every 20 min from 1000 h on day 113 of pregnancy and behaviour was recorded until birth. Two hours aft

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34

Yasmin, Shakila, Rukhsana Aziz, Muhammad Hassan, and Mehak Fatima. "TERMINATION OF PREGNANCY." Professional Medical Journal 25, no. 06 (2018): 952–58. http://dx.doi.org/10.29309/tpmj/2018.25.06.287.

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Objectives: To compare efficacy of extra-amniotic Foley’s catheter balloon aloneversus combined use of Foley’s catheter balloon and extra-amniotic instillation of prostaglandinF2-alpha in therapeutic termination of second trimester pregnancy. Study Design: Randomizedcontrolled trial. Setting: Department of Obstetrics & Gynecology, Bahawal Victoria Hospital,Bahawalpur. Period: Two years. July 2014 to June 2016. Sample Technique: Non-probability,consecutive sampling technique. Patients & Methods: A total of 256 patients, 16 to 38 years ofage with fetal death or missed abortion on ultraso

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35

Sugatani, J., Y. Masu, M. Nishizawa, et al. "Hormonal regulation of prostaglandin F2 alpha receptor gene expression in mouse ovary." American Journal of Physiology-Endocrinology and Metabolism 271, no. 4 (1996): E686—E693. http://dx.doi.org/10.1152/ajpendo.1996.271.4.e686.

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In this study we examined regulation by pituitary gonadotropins of the prostaglandin F2 alpha (PGF2 alpha) receptor gene expression in the mouse ovary. Administration of pregnant mare serum gonadotropin (PMSG) to 35-day-old mice in the diestrus phase stimulated the ovary and enhanced the production of progesterone at 1 h PMSG also increased the ovarian PGF2 alpha receptor mRNA level in a time-dependent manner, reaching a sixfold maximum at 1 h. These actions of PMSG were mimicked by human chorionic gonadotropin (hCG), follicle-stimulating hormone (FSH), and cholera toxin, all of which elevate

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36

Graser, T., and P. M. Vanhoutte. "Hypoxic contraction of canine coronary arteries: role of endothelium and cGMP." American Journal of Physiology-Heart and Circulatory Physiology 261, no. 6 (1991): H1769—H1777. http://dx.doi.org/10.1152/ajpheart.1991.261.6.h1769.

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The effect of severe hypoxia in quiescent or contracted (prostaglandin F2 alpha) canine coronary artery rings with and without endothelium was studied. Hypoxia induced an initial transient relaxation followed by a sustained contraction. The hypoxic contraction in quiescent rings was comparable in rings with and without endothelium. The facilitation of the contraction to prostaglandin F2 alpha was more pronounced in rings with endothelium. Increasing the level of contractions by augmenting the contraction of prostaglandin F2 alpha potentiated the hypoxic contraction in rings with endothelium on

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37

Taniguchi, Ken, Fumie Kizuka, Isao Tamura, and Norihiro Sugino. "Prostaglandin F2-alpha Stimulates Cyclooxygenase-2 Expression and Prostaglandin F2-alpha Synthesis Through NF-kappaB Activation via Reactive Oxygen Species in the Corpus Luteum of Pseudopregnant Rats." Biology of Reproduction 83, Suppl_1 (2010): 124. http://dx.doi.org/10.1093/biolreprod/83.s1.124.

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38

Thore, C. R., M. Nam, and D. Busija. "Phorbol ester-induced prostaglandin production in piglet cortical astroglia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 267, no. 1 (1994): R34—R37. http://dx.doi.org/10.1152/ajpregu.1994.267.1.r34.

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We examined effects of phorbol 12,13-dibutyrate (PDB), which activates protein kinase C (PKC), on prostaglandin and leukotriene production in piglet cultured glia derived from cerebral cortex and white matter. Levels of prostaglandins were determined using enzyme immunoassay. Baseline levels in media for prostaglandin F2 alpha (PGF2 alpha) were 730 +/- 116 pg/ml and increased to 1,551 +/- 196 pg/ml at 10(-8) M PDB (P < 0.05) and to 2,182 +/- 190 pg/ml at 10(-6) M PDB (P < 0.05) (n = 16). Little or no 6-keto-prostaglandin F1 alpha, prostaglandin E2, or leukotrienes C4/D4 were produced. PG

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39

Harker, C. T., P. J. Ousley, C. J. Bowman, and J. M. Porter. "Cooling augments alpha 2-adrenoceptor-mediated contractions in rat tail artery." American Journal of Physiology-Heart and Circulatory Physiology 260, no. 4 (1991): H1166—H1171. http://dx.doi.org/10.1152/ajpheart.1991.260.4.h1166.

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Experiments were performed to assess the effects of acute moderate cooling on postjunctional alpha 1- and alpha 2-adrenoceptors in isolated rings of tail arteries from male Sprague-Dawley rats. Rings were contracted with norepinephrine (NE; 10(-9) to 10(-4) M) alone or in the presence of prazosin (Pz; 3 x 10(-7) M) or rauwolscine (Rw; 10(-7) M). NE concentration-response curves were inhibited by alpha 1-blockade (Pz) but not significantly affected by alpha 2-blockade (Rw). In all rings, cooling caused an increase in the slope of the dose-response curve and a significant increase in the concent

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40

Bode, C., G. Maute, and J. C. Bode. "Prostaglandin E2 and prostaglandin F2 alpha biosynthesis in human gastric mucosa: effect of chronic alcohol misuse." Gut 39, no. 3 (1996): 348–52. http://dx.doi.org/10.1136/gut.39.3.348.

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41

McLean, M. P., J. T. Billheimer, K. J. Warden, and R. B. Irby. "Prostaglandin F2 alpha mediates ovarian sterol carrier protein-2 expression during luteolysis." Endocrinology 136, no. 11 (1995): 4963–72. http://dx.doi.org/10.1210/endo.136.11.7588230.

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42

Bhattacharaya, Prabha R., Deepak P. Wakharia, Malati M. Andankar, and Puspha Gurtu. "Menstrual Regulation by(15S)-15 Methyl Prostaglandin F2 Alpha by Intramuscular Route." Asia-Oceania Journal of Obstetrics and Gynaecology 10, no. 4 (2010): 435–37. http://dx.doi.org/10.1111/j.1447-0756.1984.tb00708.x.

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43

Hankins, G. D. V., G. K. Berryman, R. T. Scott, and D. Hood. "Maternal Arterial Desaturation With 15-Methyl Prostaglandin F2 Alpha for Uterine Atony." Obstetric Anesthesia Digest 9, no. 1 (1989): 16. http://dx.doi.org/10.1097/00132582-198904000-00018.

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44

Uchiyama, M., and K. Sakai. "Increased main urinary metabolite of prostaglandin F2 alpha excretion in childhood migraine." Archives of Disease in Childhood 63, no. 3 (1988): 342. http://dx.doi.org/10.1136/adc.63.3.342-a.

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45

Douglas, M. Joanne, Duncan F. Farquharson, Peggy L. E. Ross, and Jamie E. Renwick. "Cardiovascular collapse following an overdose of prostaglandin F2 alpha: a case report." Canadian Journal of Anaesthesia 36, no. 4 (1989): 466–69. http://dx.doi.org/10.1007/bf03005350.

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46

Graves, R. L., R. G. Lutz, J. W. Riesen, T. A. Hoagland, and C. O. Woody. "Factors influencing estrus and conception in dairy heifers after prostaglandin F2-alpha." Theriogenology 23, no. 5 (1985): 733–42. http://dx.doi.org/10.1016/0093-691x(85)90148-7.

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47

Archer, D. F., J. E. Taylor, and T. A. Jacot. "Prostaglandin F2 alpha is a product of the decidualized human stromal cell." Fertility and Sterility 100, no. 3 (2013): S298. http://dx.doi.org/10.1016/j.fertnstert.2013.07.1053.

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48

Scott, I. M., R. H. Fertel, and J. A. Boulant. "Leukocytic pyrogen effects on prostaglandins in hypothalamic tissue slices." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 253, no. 1 (1987): R71—R76. http://dx.doi.org/10.1152/ajpregu.1987.253.1.r71.

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Some studies suggest that leukocytic pyrogen (LP) increase hypothalamic prostaglandins which, in turn, affect hypothalamic thermoregulatory neurons to produce fever. The present study used radioimmunoassays to quantitate the ability of guinea pig hypothalamic tissue slices to produce prostaglandin E2 (PGE2), prostaglandin F2 alpha (PGF2 alpha), 6-keto-prostaglandin F1 alpha (6-keto-PGF1 alpha), and thromboxane B2 (TxB2). Dose- and time-dependent prostaglandin increases occurred when these slices were perfused with LP media. Steady-state levels of tissue release were reached at 0-3 min for 6-ke

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49

Sada, K., M. Shirai, and I. Ninomiya. "Effects of prostaglandin F2 alpha and prostacyclin on pulmonary microcirculation in the cat." Journal of Applied Physiology 62, no. 3 (1987): 1124–32. http://dx.doi.org/10.1152/jappl.1987.62.3.1124.

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In pulmonary microcirculation, using a new X-ray television system, we measured the effects of prostaglandin F2 alpha (PGF2 alpha) and prostacyclin on the internal diameter (ID), flow velocity, volume flow, and transit times of a contrast medium in small arteries (Ta) and veins (Tv) in anesthetized cats. The ID of the arteries and veins ranged from 100 to 500 micron. PGF2 alpha, 0.3, 1, and 3 micrograms/kg, predominantly decreased ID on the arterial side in a dose-dependent manner but increased flow velocity 27–62%. Consequently, volume flow was kept relatively constant. With PGF2 alpha, Ta an

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50

Hoeffner, U., C. Boulanger, and P. M. Vanhoutte. "Proximal and distal dog coronary arteries respond differently to basal EDRF but not to NO." American Journal of Physiology-Heart and Circulatory Physiology 256, no. 3 (1989): H828—H831. http://dx.doi.org/10.1152/ajpheart.1989.256.3.h828.

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Experiments were designed to analyze the effects of endothelium-derived relaxing factor(s) (EDRF; released basally or on stimulation with acetylcholine) and nitric oxide (NO) on smooth muscle of coronary arteries of different diameter. During contractions of the bioassay ring evoked with prostaglandin F2 alpha, the relaxations caused by basal EDRF were greater in the distal than in the proximal coronary arteries, whereas there was no difference in response to the EDRF released by acetylcholine. During direct superfusion, NO caused similar relaxations in proximal and distal coronary artery ring

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