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Libros sobre el tema "Staphylococcal disease"

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Publicado: 4 de junio de 2021
Última modificación: 16 de febrero de 2022

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1

Mittag, Hans-Christian. Toxic shock syndrome and the other staphylococcal toxicoses. Stuttgart: New York, 1988.

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2

Mittag, Hans-Christian. Toxic shock syndrome and the other staphylococcal toxicoses. Stuttgart: Schattauer, 1988.

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3

Crossley, Kent B., Kimberly K. Jefferson, Gordon L. Archer y Vance G. Fowler, eds. Staphylococci in Human Disease. Oxford, UK: Wiley-Blackwell, 2009. http://dx.doi.org/10.1002/9781444308464.

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4

Honeyman, Allen L., Herman Friedman y Mauro Bendinelli, eds. Staphylococcus aureus Infection and Disease. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/b111097.

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5

Freeman-Cook, Lisa. Staphylococcus aureus infections. Editado por Freeman-Cook Kevin D, Alcamo I. Edward y Heymann David L. Philadelphia: Chelsea House Publishers, 2006.

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6

Georg, Peters. Role of clindamycin in the therapy of staphylococcal diseases. Erlangen: Pharmacia & Upjohn, 1996.

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7

Ireland. Food Safety Advisory Committee. Cryptosporidiosis staphylococcal food poisoning food borne illness enterohaemorrhagic E Coli (EHEC/VTEC). Dublin: Stationery Office, 1992.

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8

Parker, James N. y Philip M. Parker. Staphylococcus aureus: A medical dictionary, bibliography, and annotated research guide to internet references. San Diego, CA: ICON Health Publications, 2004.

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9

European Congress of Clinical Microbiology and Infectious Diseases (7th 1995 Vienna). Infections by multiresistant staphylococci: 7th European Congress of Clinical Microbiology and Infectious Diseases, Vienna, March 26-30, 1995. Editado por Kayser F. H y Carbon C. Basel: Karger, 1996.

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10

B, Crossley Kent y Archer Gordon 1943-, eds. The staphylococci in human disease. New York: Churchill Livingstone, 1997.

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11

B, Crossley Kent, ed. Staphylococci in human disease. 2a ed. Chichester, West Sussex: Wiley-Blackwell, 2010.

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12

Allen, Honeyman, Friedman Herman 1931- y Bendinelli Mauro, eds. Staphylococcus aureus infection and disease. New York: Kluwer Academic/Plenum, 2001.

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13

Abramson, Carl. Immunoserology of Staphylococcal Disease. Amer Society for Microbiology, 1987.

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14

Friedman, Herman, Allen Honeyman y Mauro Bendinelli. Staphylococcus aureus Infection and Disease. Springer, 2001.

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15

Proctor, Richard A. Staphylococcal Infectious Disease and Therapy. Informa Healthcare, 2010.

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16

(Editor), Allen Honeyman, Herman Friedman (Editor) y Mauro Bendinelli (Editor), eds. Staphylococcus Aureus : Infection and Disease (Infectious Agents and Pathogenesis) (Infectious Agents and Pathogenesis). 2a ed. Springer, 2001.

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17

Gorwitz, Rachel y John Jernigan. Staphylococcal Infections, an Issue of Infectious Disease Clinics. Elsevier - Health Sciences Division, 2009.

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18

(Editor), D. Ala'Aldeen y K. Hiramatsu (Editor), eds. Staphyloccus Aureus: Molecular And Clinical Aspects (Infectious Disease & Microbiology). Horwood Publishing Limited, 2004.

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19

Cuttle, Lisa. Dermatologic Manifestations of Infectious Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0044.

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Toxic infectious exfoliative conditions include staphylococcal toxic shock syndrome (TSS), streptococcal toxic shock syndrome (STSS), and staphylococcal scalded skin syndrome (SSSS). All three are mediated by bacterial toxin production and are considerations in the differential diagnosis of a febrile, hypotensive patient with a rash. Meningococcemia is potentially fatal and extremely contagious with a short incubation period. Disseminated gonococcal infection (DGI) presents with tenosynovitis, dermatitis, and polyarthralgias without purulent arthritis or with purulent arthritis but without skin lesions. Ecthyma gangrenosum (EG) is a cutaneous manifestation of Pseudomonas aeruginosa infection. Rocky Mountain Spotted Fever (RMSF) is caused by Rickettsia rickettsii, most commonly transmitted by the American dog tick. Patients present with nonspecific symptoms, such as fever, headache, myalgias, arthralgias, nausea, vomiting, and abdominal pain. Finally, vibrio vulnificus is a gram-negative bacterium that causes serious wound infections, sepsis, and diarrhea in patients exposed to shellfish or marine water.
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20

Goyal, Saurabh, Allon Barsam y Stephen Tuft. External eye disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199237593.003.0001.

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This chapter covers corneal and conjunctival basic science, before covering clinical skills (history taking for anterior segment disease and examination of the anterior segment). The chapter then covers blepharitis, staphylococcal hypersensitivity disorders, dry eye disease, conjunctivitis, cicatrizing conjunctival disease, conjunctival degeneration, conjunctival neoplasia, corneal degeneration, infectious keratitis, interstitial keratitis, peripheral ulcerative keratitis, metabolic and drug-induced keratopathies, corneal dystrophies, contact lenses, corneal ectasia, keratoplasty, complications of keratoplasty and graft rejection, anterior uveal tumours, anterior segment trauma, chemical injury, and refractive surgery. Practical skills are then covered, including corneal glue, removal of corneal sutures, removal of corneal foreign bodies, corneal topography, and corneal pachymetry. The chapter concludes with three case-based discussions, on chemical injury, Herpes zoster keratitis, and bacterial keratitis.
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21

Bunker, Professor Christopher y Dr Arani Chandrakumar. Dermatological diseases and emergencies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00017.

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Chapter 17 covers dermatological diseases and emergencies including a general introduction to the subject, followed by information on erythroderma, drug eruptions, angio-oedema, Kawasaki disease, staphylococcal toxic shock syndrome, Streptococcal toxic shock syndrome (streptococcal TSS), staphylococcal scalded skin syndrome, necrotizing fasciitis, psoriasis, eczema and dermatitis, cutaneous vasculitis, immunobullous disorders, pyoderma gangrenosum, scarring alopecia, herpes simplex viruses 1 and 2, varicella zoster virus infection, bacterial infections affecting the skin, fungal infections affecting the skin, ectoparasitic disease, HIV infection and the skin, malignant melanoma, non-melanoma skin cancer, and cutaneous T cell lymphoma.
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22

Crossley, Kent B., Kimberly K. Jefferson, Gordon L. Archer, Vance G. Fowler y Vance G. Jr Fowler. Staphylococci in Human Disease. Wiley & Sons, Incorporated, John, 2009.

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23

Dawson, Susan. Other bacterial diseasesStaphylococcal zoonoses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0026.

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Staphylococcal species are common commensals of the skin and mucous membranes of humans and animals but only in very recent years has zoonotic infections been recognised. They can also be associated with infection and disease, especially coagulase positive organisms. Staphylococcus aureus is relatively frequently carried by humans in the nasal passages and is a cause of infections in people including bacteraemias in hospitalised patients. More recently some strains of Staphylococcus aureus have acquired a resistance gene (mecA) which renders them resistant to meticillin (meticillin-resistant Staphylococcus aureus, MRSA). MRSA isolates are of major importance in healthcare situations as well as increasingly in the community. Animals can also be carriers of Staphylococcus aureus although less frequently than humans and MRSA can be carried or infect several different host species. For companion animals such as dogs and cats, the most frequently isolated MRSA strains are similar to the common local human healthcare strains; thus for the UK, EMRSA-15 and -16. This suggests a reverse zoonosis with spill over from the human population into their companion animals. In horses the situation is different, with some horses carrying or infected with human epidemic strains but others infected with strains less frequently seen in people. For food-producing animals the picture is different again with a particular strain, ST398, which appears to circulate endemically in animal populations, such as pigs, and can spill over into the human population where it can cause carriage as well as infection and disease. The transmission appears to be by direct contact with animals rather than through the food-chain.Where risk factors for infection with MRSA have been studied in animals they appear similar to some of the risks for human infection. Therefore, for control of MRSA in animals measures such as improved hygiene and good antibacterial stewardship are important.
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24

Autobiography of a Disease. Taylor & Francis Group, 2017.

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25

Autobiography of a Disease. Taylor & Francis Group, 2017.

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26

Staphylococcus aureus Infection and Disease. Cleveland: Kluwer Academic Publishers, 2002.

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27

Cleary, Georgia, Allon Barsam y Stephen Tuft. Cornea and conjunctiva. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199672516.003.0002.

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This chapter focuses on the cornea and conjunctiva. It first discusses corneal anatomy and physiology, corneal nutrition, and conjunctival anatomy and physiology. Next, it outlines history taking for anterior segment disease. It then discusses clinical knowledge areas, including blepharitis, staphylococcal hypersensitivity disorders, dry eye disease, conjunctivitis, cicatrizing conjunctival disease, conjunctival degeneration, conjunctival neoplasia, corneal degeneration, infectious keratitis (bacterial, fungal, and viral), interstitial keratitis, peripheral ulcerative keratitis, metabolic and drug induced keratopathies, corneal dystrophies, contact lenses, corneal ectasia, keratoplasty, anterior uveal tumours, anterior segment trauma, chemical injury, and refractive surgery. Practical skills, such as the removal of a corneal foreign body, and corneal sutures, are also discussed.
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28

Rodriguez-Iturbe, Bernardo y Mark Haas. Post-infectious glomerulonephritis. Editado por Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0076.

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Post-infectious glomerulonephritis (GN) defines an inflammatory lesion involving exclusively or predominantly the glomeruli that is a consequence of an infectious disease. There are numerous bacterial, viral, and fungal infections associated with GN. This chapter acts as an overview of the following chapters that discuss only post-streptococcal GN, immunoglobulin A-dominant GN associated with staphylococcal infections, GN associated with bacterial endocarditis, with infected ventriculoatrial shunts (‘shunt nephritis’), and GN associated with deep-seated infections (osteomyelitis, visceral abscesses, pleural suppuration, pneumonia).
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29

Fetsch, Alexandra. Staphylococcus Aureus. Elsevier Science & Technology Books, 2017.

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30

Lydyard, Peter, Michael Cole, John Holton, Will Irving, Nino Porakishvili, Pradhib Venkatesan y Kate Ward. Case Studies in Infectious Disease: Staphylococcus aureus. Garland Science, 2009. http://dx.doi.org/10.4324/9780203854051.

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31

Grisoli, Dominique y Didier Raoult. Prevention and treatment of endocarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0161.

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Initially always lethal, the prognosis of infective endocarditis (IE) has been revolutionized by antibacterial therapy and valve surgery. Nevertheless, it remains one of the deadliest infectious diseases, with ≥30% of patients dying within a year of diagnosis. Its incidence has also remained stable at 25–50 cases per million per year, and results predominantly from a combination of bacteraemia and a predisposing cardiac condition, including endocardial lesions and/or intracardiac foreign material. While antibiotic prophylaxis is recommended by various learned societies to cover healthcare procedures with the potential of causing bacteraemia in at-risk patients, there is no evidence to support this strategy. Even though the benefits are hypothetical, national guidelines should still be followed to avoid medico-legal issues. General preventive measures, such as education of clinicians and at-risk patients appear to be more crucial. Invasive procedures, especially intravenous catheterization, should be kept to the minimum possible. The severity of IE mandates a multidisciplinary and standardized approach to treatment, with involvement of dedicated surgeons within specialist centres. Standardized antibiotic protocols have produced dramatic reductions in hospital and 1-year mortality in reference centres. Most deaths now result from complications that constitute definite surgical indications, so optimization of surgical management and avoidance of delay will clearly improve prognosis. This disease has now entered an ‘early surgery’ era, with a more aggressive surgical approach showing promising results. Conditions such as septic shock, sudden death, and vancomycin-resistant staphylococcal endocarditis still constitute therapeutic and research challenges, and justify an important role for specialist centres.
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32

Govan, John y Andrew Jones. Microbiology of CF lung disease. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198702948.003.0003.

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This chapter presents the microbiology of CF and describes the classical bacterial pathogens including Staphylococcus aureus, Haemophilus influenza, Pseudomonas aeruginosa and organisms of the Burkholderia cepacia complex. The dominant of these is P. aeruginosa. Infections with other opportunistic pathogens including non-tuberculous mycobacteria, Stenotrophomonas maltophila, and Achromobacter (Alcaligenes) xylosoxidans are also encountered. This chapter details measures to prevent the onset of chronic infection with these organisms include regular screening of respiratory tract samples for bacterial pathogens and the use of aggressive antibiotic therapy to eradicate initial infection before the pathogen can adapt to the environment of the CF lung. Patient-to-patient spread of transmissible strains of bacterial pathogens has led to the implementation of strict infection control measures at CF centres, including patient segregation. In addition to bacterial pathogens, the contribution of fungal infection in CF lung disease is increasingly recognized.
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33

Tennant, Sally y Peter Calder. Miscellaneous orthopaedic infections. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.011002.

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♦ Clinical features and treatment of: leprosy; brucellosis; syphilis of bone and joints; hydatid disease of the bone; mycetoma; salmonella infection; staphylococcus aureus infections associated with Panton-Valentine leucocidin; Poliomyelitis; Tuberculosis.
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34

Iung, Bernard. Epidemiology and physiopathology. Editado por Gilbert Habib. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0389.

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The incidence of infective endocarditis (IE) is estimated at between 15 and 60 cases per million inhabitants per year from population-based studies in industrialized countries. The presentation of IE has changed since patients are getting older and Staphylococcus is now becoming the microorganism most frequently responsible, which is partly attributable to healthcare-associated infections. The incidence of IE is higher in patients with heart valve prosthesis, previous endocarditis, and complex congenital heart disease. In developing countries, IE occurs in younger patients with a majority of rheumatic valve disease and is most frequently due to streptococci. IE is the consequence of bacteraemia on a diseased native valve or foreign material, leading to vegetation or tissue destruction, or both of these. The main consequences of these lesions are heart failure due to valvular regurgitation, embolic events due to vegetation migration, and perivalvular abscesses.
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35

A, Weigelt John, ed. MRSA. 2a ed. New York: Informa Healthcare, 2009.

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36

Bowker, Lesley K., James D. Price, Ku Shah y Sarah C. Smith. Infection and immunity. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0024.

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This chapter provides information on the ageing immune system, an overview of infection in older people, antibiotic use in older patients, meticillin-resistant Staphylococcus aureus (MRSA), disease caused by MRSA, Clostridium difficile-associated diarrhoea, near-patient urine tests, asymptomatic bacteriuria, urinary tract infection, treatment of urinary tract infection, recurrent urinary tract infection, and varicella-zoster infection.
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37

Virk, Abinash. Specific Microorganisms. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0410.

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This chapter approaches the field of infectious diseases from 3 perspectives. This first section reviews the characteristics of specific pathogenic organisms (gram-positive cocci, staphylococci, gram-negative bacilli, gram-positive bacilli, gram-negative cocci, anaerobic bacteria, actinomycetes, mycobacteria, spirochetes, fungi, viruses, etc). Symptoms, diagnosis, and treatment of conditions caused by these organisms are discussed.
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38

Help! What's Eating My Flesh?: Runaway Staph and Strep Infections! (24/7: Science Behind the Scenes). Franklin Watts, 2007.

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39

Tilden, Thomasine E. Lewis. Help! Whats Eating My Flesh?: Runaway Staph and Strep Infections! (24/7: Science Behind the Scenes). Franklin Watts, 2007.

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40

McGregor, Laura, Monica N. Gupta y Max Field. Septic arthritis in adults. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0098.

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Septic arthritis (SA) is a medical emergency with mortality of around 15%. Presentation is usually monoarticular but in more than 10% SA affects two or more joints. Symptoms include rapid-onset joint inflammation with systemic inflammatory responses but fever and leucocytosis may be absent at presentation. Treatment according to British Society of Rheumatology/British Orthopaedic Association (BSR/BOA) guidelines should be commenced if there is a suspicion of SA. At-risk patients include those with primary joint disease, previous SA, recent intra-articular surgery, exogenous sources of infection (leg ulceration, respiratory and urinary tract), and immunosupression because of medical disorders, intravenous drug use or therapy including tumour necrosis factor (TNF) inhibitors. Synovial fluid should be examined for organisms and crystals with repeat aspiration as required. Most SA results from haematogenous spread-sources of infection should be sought and blood and appropriate cultures taken prior to antibiotic treatment. Causative organisms include staphylococcus (including meticillin-resistant Staphylococcus aureus, MRSA), streptococcus, and Gram-negative organisms (in elderly patients), but no organism is identified in 43%, often after antibiotic use before diagnosis. Antibiotics should be prescribed according to local protocols, but BSR/BOA guidelines suggest initial intravenous and subsequent oral therapy. Medical treatment may be as effective as surgical in uncomplicated native SA, and can be cost-effective, but orthopaedic advice should be sought if necessary and always in cases of infected joint prostheses. In addition to high mortality, around 40% of survivors following SA develop limitation of joint function. Guidelines provide physicians with treatment advice aiming to limit mortality and morbidity and assist future research.
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41

Ramsdale, David R. Color Atlas of Infective Endocarditis. Springer, 2005.

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42

Thuny, Franck y Didier Raoult. Pathophysiology and causes of endocarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0160.

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Endocarditis is defined as an inflammation of the endocardial surface of the heart. This may include heart valves, mural endocardium or the endocardium that covers implanted material, such as prosthetic valves, pacemaker/defibrillator leads and catheters. Infective and non-infective-related causes must be distinguished. In most cases, the inflammation is related to a bacterial or fungal infection with oral streptococci, group D streptococci, staphylococci and enterococci accounting for 85% of episodes. Infective endocarditis (IE) is a serious disease with an incidence ranging from 30 to 100 episodes/million patient-years. From various portals of entry (e.g. oral, digestive, cutaneous) and a subsequent bacteraemia, pathogens can adhere and colonize intracardiac foreign material or onto previously damaged endocardium due to numerous complex processes based on a unique host–pathogen interaction. Rarely, endocarditis can be related to non-infective causes, such as immunological or neoplastic. Mortality is high, with more than one-third dying within a year of diagnosis from complications such as acute heart failure or emboli. This disease still remains a diagnostic challenge with many cases being identified and subsequently treated too late. Diagnosis of IE usually relies on the association between an infectious syndrome and recent endocardial involvement. Blood cultures and echocardiography are the main diagnostic procedures, but are negative in almost 30% of cases, requiring the use of more sophisticated techniques. Computed tomography, magnetic resonance imaging and positron emission tomography are promising imaging modalities. Improved understanding of its pathophysiology and the development of relevant diagnostic strategies enables accelerated identification and treatment, and thus an improved prognosis.
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