Literatura académica sobre el tema "Thermoprotection"

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Artículos de revistas sobre el tema "Thermoprotection"

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Malayer, Jerry R., John W. Pollard, and Peter J. Hansen. "Modulation of thermal killing of bovine lymphocytes and preimplantation mouse embryos by alanine and taurine." American Journal of Veterinary Research 53, no. 5 (1992): 689–94. http://dx.doi.org/10.2460/ajvr.1992.53.05.689.

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Summary Addition of alanine and taurine blocked killing of lymphocytes caused by culture at 45 C. The optimal concentration for thermoprotection was achieved at 12.5 mM for l-alanine and 5 mM for taurine. Both d and l forms of alanine provided thermoprotection. The effect of these agents was not simply to increase osmolarity of the culture medium, because NaCl did not provide thermoprotection at comparable concentrations. Alanine and taurine were each tested at concentration of 50 mM for ability to block heat shock-induced killing and developmental retardation of 8- to 16-cell mouse embryos. B
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Barclay, J. W., and R. M. Robertson. "Heat-shock-induced thermoprotection of hindleg motor control in the locust." Journal of Experimental Biology 203, no. 5 (2000): 941–50. http://dx.doi.org/10.1242/jeb.203.5.941.

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Functional neuromuscular connections are critical for appropriate behavioural responses, but can be negatively affected by increases in temperature. We investigated the effects of heat shock on the thermosensitivity of a neuromuscular pathway to the hindleg tibial extensor muscle of Locusta migratoria. We found that exposure to heat shock induced thermoprotection of both neuromuscular transmission and extensor muscle contraction by (i) increasing the upper temperature limit for failure, (ii) improving recovery following heat-induced failure and (iii) stabilizing excitatory junction potential a
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Klose, M. K. "Stress-Induced Thermoprotection of Neuromuscular Transmission." Integrative and Comparative Biology 44, no. 1 (2004): 14–20. http://dx.doi.org/10.1093/icb/44.1.14.

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Caldas, Teresa, Nathalie Demont-Caulet, Alexandre Ghazi, and Gilbert Richarme. "Thermoprotection by glycine betaine and choline." Microbiology 145, no. 9 (1999): 2543–48. http://dx.doi.org/10.1099/00221287-145-9-2543.

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Neal, Scott J., Shanker Karunanithi, Adrienne Best, et al. "Thermoprotection of synaptic transmission in a Drosophila heat shock factor mutant is accompanied by increased expression of Hsp83 and DnaJ-1." Physiological Genomics 25, no. 3 (2006): 493–501. http://dx.doi.org/10.1152/physiolgenomics.00195.2005.

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In Drosophila larvae, acquired synaptic thermotolerance after heat shock has previously been shown to correlate with the induction of heat shock proteins (Hsps) including HSP70. We tested the hypothesis that synaptic thermotolerance would be significantly diminished in a temperature-sensitive strain ( Drosophila heat shock factor mutant hsf4), which has been reported not to be able to produce inducible Hsps in response to heat shock. Contrary to our hypothesis, considerable thermoprotection was still observed at hsf4 larval synapses after heat shock. To investigate the cause of this thermoprot
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PLATT, MARK W., MICHAEL D. RICH, and JAMES C. MCLAUGHLIN. "The Role of Chitin in the Thermoprotection of Vibrio cholerae." Journal of Food Protection 58, no. 5 (1995): 513–14. http://dx.doi.org/10.4315/0362-028x-58.5.513.

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Vibrio cholerae has been shown to be able to survive short periods of boiling. The potential role of chitin in the thermoprotection of Vibrio cholerae was determined. When V. cholerae 6706 (OI E1 Tor) cells were incubated at 37°C in the presence of chitin, the bacteria were able to grow in the absence of any other energy source. In phosphate-buffered saline, growth was not supported. The ability of six pathogenic strains to survive at 60°C was investigated. None of the isolates tested was able to survive at 60°C for 10 min regardless of whether chitin was present. Further investigation reveale
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Rodriguez, P. X., Y. Ono, Y. Sun, P. Hamet, S. N. Orlov, and J. Tremblay. "THERMOPROTECTION OF VASCULAR SMOOTH MUSCLE ION TRANSPORTERS." Journal of Hypertension 18 (June 2000): S183. http://dx.doi.org/10.1097/00004872-200006001-00634.

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Newman, Amy E. M., Chengfeng Xiao, and R. Meldrum Robertson. "Synaptic thermoprotection in a desert-dwellingDrosophila species." Journal of Neurobiology 64, no. 2 (2005): 170–80. http://dx.doi.org/10.1002/neu.20132.

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Kraus, K. W., E. M. Hallberg, and R. Hallberg. "Characterization of a Tetrahymena thermophila mutant strain unable to develop normal thermotolerance." Molecular and Cellular Biology 6, no. 11 (1986): 3854–61. http://dx.doi.org/10.1128/mcb.6.11.3854-3861.1986.

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For Tetrahymena thermophila cells to survive extended periods of time at 43 degrees C, they must continuously synthesize heat shock proteins. For its translational machinery to function at 43 degrees C, T. thermophila requires either prior nonlethal heat shock treatment or brief treatment with partially inhibiting doses of cycloheximide or emetine. We have identified and characterized a mutant strain of T. thermophila (MC-3) in which prior nonlethal heat shock does not prevent protein synthesis inactivation at 43 degrees C. In addition, treatment of MC-3 cells with either of the antibiotics th
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Kraus, K. W., E. M. Hallberg, and R. Hallberg. "Characterization of a Tetrahymena thermophila mutant strain unable to develop normal thermotolerance." Molecular and Cellular Biology 6, no. 11 (1986): 3854–61. http://dx.doi.org/10.1128/mcb.6.11.3854.

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For Tetrahymena thermophila cells to survive extended periods of time at 43 degrees C, they must continuously synthesize heat shock proteins. For its translational machinery to function at 43 degrees C, T. thermophila requires either prior nonlethal heat shock treatment or brief treatment with partially inhibiting doses of cycloheximide or emetine. We have identified and characterized a mutant strain of T. thermophila (MC-3) in which prior nonlethal heat shock does not prevent protein synthesis inactivation at 43 degrees C. In addition, treatment of MC-3 cells with either of the antibiotics th
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Tesis sobre el tema "Thermoprotection"

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Barclay, Jeffrey William. "Environmentally-induced thermoprotection of insect motor control." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ63403.pdf.

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Wu, Bernhard Shiaoyuen. "Environmental stress-induced thermoprotection of action potential signalling in Locusta migratoria." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ63472.pdf.

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Koch, Guillaume. "Optimisation de la protection du nerf sciatique lors des ablathermies du bassin." Electronic Thesis or Diss., Strasbourg, 2024. http://www.theses.fr/2024STRAD041.

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La thermoablation est une technique de destruction tumorale percutanée. Cette technique mini-invasive utilise des imageurs pour guider durant l’intervention les sondes de thermoablation jusqu’à la zone cible et pour guider la mise en place d’outils de protection des structures anatomiques sensibles. La thermoablation de lésion osseuse du bassin est une technique à risque par proximité du nerf sciatique. Sa protection est essentielle pour éviter une lésion nerveuse thermique iatrogène. L’hydrodissection du nerf sciatique est une technique de protection active par éloignement du nerf de la zone
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Armstrong, GARY. "Cellular signalling pathways involved in thermoprotection of neural ciruit function in the locust." Thesis, 2009. http://hdl.handle.net/1974/5102.

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Environmental temperature is arguably one of the most important abiotic physical factors affecting insect behaviour. Temperature affects virtually all physiological processes including those that regulate nervous system function. It is therefore not surprising that animals have evolved adaptations that confer tolerance to heat stress and allow for continued behaviour as ambient temperature fluctuates. Most animals have central nervous system (CNS) responses to heat shock (HS) preconditioning which extend the thermal operating range of neural circuits during exposure to extreme heat. It is uncl
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Capítulos de libros sobre el tema "Thermoprotection"

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Lin, Chu-Yung, Tsung-Luo Jinn, Ming-Hsiun Hsieh, and Yih-Ming Chen. "Class I low molecular weight heat shock proteins in plants: immunological study and thermoprotection against heat denaturation of soluble proteins." In Biochemical and Cellular Mechanisms of Stress Tolerance in Plants. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79133-8_7.

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Actas de conferencias sobre el tema "Thermoprotection"

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Zhestkov, B. Eu, I. V. Yegorov, W. P. P. Fischer, and J. Antonenko. "Windtunnel Catalyticity Evaluation for Thermoprotective Elements." In 31st International Conference On Environmental Systems. SAE International, 2001. http://dx.doi.org/10.4271/2001-01-2384.

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Wei, Yanhong, and Chang Xu. "Automatic testing system for parameters of high-current thermoprotector." In Instruments (ICEMI). IEEE, 2009. http://dx.doi.org/10.1109/icemi.2009.5274812.

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Informes sobre el tema "Thermoprotection"

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Hansen, Peter J., and Zvi Roth. Use of Oocyte and Embryo Survival Factors to Enhance Fertility of Heat-stressed Dairy Cattle. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7697105.bard.

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The overall goal was to identify survival factors that can improve pregnancy success following insemination or embryo transfer in lactating dairy cows exposed to heat stress. First, we demonstrated that oocytes are actually damaged by elevated temperature in the summer. Then we tested two thermoprotective molecules for their effect on oocyte damage caused by heat shock. One molecule, ceramide was not thermoprptective. Another, insulin-like growth factor-1 (IGF) reduced the effects of heat shock on oocyte apoptosis and oocyte cleavage when added during maturation. We also used lactating cows ex
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