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Tesis sobre el tema "Translocation renal cell carcinoma"

Autor: Grafiati

Publicado: 11 de marzo de 2023

Última modificación: 31 de julio de 2025

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1

Wake, Naomi Catherine. "Identification and functional analysis of a novel renal cell carcinoma (RCC) susceptibility gene from an RCC associated constitutional chromosomal translocation." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/3964/.

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Familial renal cell carcinoma (RCC) only accounts for 3% of all RCC, yet the study of these inherited forms has provided important insights into the more common sporadic RCC. Somatic VHL inactivation is found in 70% of sporadic clear cell RCC (ccRCC) though is rarely found in other forms of RCC including papillary and chromophobe types. VHL-independent RCC tumourigenesis is poorly understood and current research involves identifying novel RCC candidate genes to further understand the mechanisms involved. In this study a constitutional balanced translocation, t(5;19)(p15.3;q12), associated with

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2

Malouf, Gabriel. "Décryptage des changements épigénétiques impliqués dans la transition épithélio-mésenchymateuse et le cancer." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T037.

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La transition épithélio-Mésenchymateuse (TEM) est un processus de plasticité cellulaire qui existe dans le développement embryonnaire et qui permet la formation des tissus et organes. Dans la cancérogénèse, ce processus est réactivé par des facteurs de transcription dont l’action implique très probablement un remodelage de la chromatine. La cartographie exacte de ces changements épigénétiques est peu connue à l’échelle du génome entier, même si il y a eu quelques études antérieures explorant les changements de quelques loci de façon bien ciblée. Ce mémoire traite du remodelage épigénétique méd

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3

Rashidkhani, Bahram. "Diet and renal cell carcinoma /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-163-6/.

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4

Fallah, Abdul Karim. "Genomic studies in renal cell carcinoma." Thesis, Manchester Metropolitan University, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.528380.

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5

Al-Sharhan, Mouza Abdulla. "Prognostic factors in renal cell carcinoma." Thesis, University of Newcastle Upon Tyne, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285788.

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6

Chagnon, Fanny. "A dendritic cell vaccine for murine renal cell carcinoma." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=19400.

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Renal Cell Carcinoma (RCC) has a very high rate of mortality since it does not respond to conventional therapies such as chemotherapy and radiation therapy. Furthermore, in the majority of cases, metastases are already present at the time of diagnosis. The objective of our study is to develop a noval treatment for RCC, using a dendritic cell (DC) vaccine. An animal model of RCC, RENCA, was used to develop the vaccine.

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7

Giraldo-Castillo, Nicolas. "The Immune Microenvironment in Clear Cell Renal Cell Carcinoma : The heterogeneous immune contextures accompanying CD8+ T cell infiltration in clear cell Renal Cell Carcinoma." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066321/document.

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Dans cette étude, nous avons tenté de décrypter les mécanismes reliant l’augmentation de lymphocytes infiltrant les tumeurs (LIT) T CD8+ et un pronostic clinique défavorable dans le cancer du rein à cellules claires (ccRCC). Pour cela, nous avons déterminé 1) la relation entre le pronostic associé à l'expression d’immune checkpoints et l’infiltrat de cellules dendritiques (DC) et de LT CD8+ et 2) les caractéristiques phénotypiques des LIT T CD8+. L’expression des immune checkpoints a été déterminée par immunohistochimie dans une cohorte de 135 ccRCC. Nous avons constaté que les densités des ce

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8

Ronkainen, H. L. (Hanna-Leena). "Novel prognostic biomarkers for renal cell carcinoma." Doctoral thesis, Oulun yliopisto, 2012. http://urn.fi/urn:isbn:9789514297731.

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Abstract Background and aims: Stage and grade are the most widely used prognostic parameters for renal cell carcinoma (RCC). The clinical course of this disease is not, however, always predictable by traditional prognostic factors. In the era of new molecular targeted therapies a more accurate prognostication of RCC patient survival is important for the individualization of treatment and follow-up of patients. Despite exhaustive research there are still no prognostic biomarkers for RCC in clinical practice. In order to find novel prognostic tissue markers for RCC, we examined the expression of

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9

Lawrentschuk, Nathan Leo. "Hypoxia and angiogenesis in renal cell carcinoma." Connect to thesis, 2009. http://repository.unimelb.edu.au/10187/6790.

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Hypoxia is one of the hallmarks of cancer. It was first postulated to occur in solid tumours by Thomlinson and Gray in 1955.1 The presence of hypoxia has been demonstrated in different types of solid tumours.2 Intratumoral hypoxia is caused by the lack of functional blood vessels in proliferating tumour tissue, resulting in low intratumoral oxygen concentrations. If hypoxia is severe or prolonged, cell death occurs.3 Malignant cells can undergo genetic and adaptive changes that allow them to escape from dying of oxygen deprivation. These changes are associated with a more aggressive malignant

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10

Morrissey, Catherine. "The molecular pathology of renal cell carcinoma." Thesis, University of Birmingham, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.420407.

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11

Laird, Alexander. "Molecular prognostic markers in renal cell carcinoma." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/17873.

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Renal cell carcinoma (RCC) is the most deadly of urological malignancies. While metastatic disease affects one third of patients at diagnosis, a further third of patients who undergo extirpative surgery with curative intent subsequently develop metastatic disease. Inconsistency in the clinical course ensures predicting subsequent metastasis is notoriously difficult, despite the routine use of prognostic clinico-pathological parameters in risk stratification. With greater understanding of pathways involved in disease pathogenesis, a number of biomarkers have been proposed to be of prognostic si

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12

Griffiths, Richard Wyn. "Strategies for the adoptive cell therapy of renal cell carcinoma." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503835.

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13

Lidgren, Anders. "Hypoxia inducible factor-1α in renal cell carcinoma". Doctoral thesis, Umeå universitet, Kirurgisk och perioperativ vetenskap, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1462.

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Hypoxia Inducible Factor-1α in Renal Cell Carcinoma Departments of Surgical and Perioperative Sciences, Urology and Andrology; Radiation Sciences, Oncology; Medical Biosciences, Pathology; and Medical Biosciences, Clinical Chemistry, Umeå University, Umeå, Sweden Background: Renal cell carcinoma (RCC) accounts for approximately 2-3% of all human cancers. A distinguished feature of RCC is vascularisation and among the three dominating RCC types conventional RCC (cRCC) generally is more vascularised than papillary RCC (pRCC) and chromophobe RCC (chRCC). Angiogenesis is a critical step in tumour

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14

Menezes, Ravi. "Physical activity and risk of renal cell carcinoma." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0020/MQ53351.pdf.

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15

Jacobsen, Jan. "Vascular endothelial growth factor in renal cell carcinoma." Doctoral thesis, Umeå : Kirurgisk och perioperativ vetenskap, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-713.

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16

Alimov, Andrei. "Molecular genetic aspects of renal cell carcinoma development /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-559-X/.

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17

Ibraheem, K. "The role of CD40 in regulating renal cell carcinoma cell fate." Thesis, University of Huddersfield, 2018. http://eprints.hud.ac.uk/id/eprint/34515/.

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CD40 is a member of the TNF receptor (TNFR) superfamily and its expression by a variety of cell types including tumour cells has suggested a possible role for CD40 in epithelial homeostasis and potentially in the pathogenesis of cancer. CD40 ligation by membrane-presented CD40L (mCD40L), but not soluble agonists, causes extensive apoptosis in malignant epithelial cells, including bladder and colorectal cancer cells, while sparing their normal counterparts. However, the role of CD40 in renal cell carcinoma (RCC) is relatively unknown and the effect of CD40 ligation in RCC cells has not been stu

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18

Handa, Kiren. "Dietary patterns and the risk of renal cell carcinoma." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq45404.pdf.

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19

Young, Alison Claire. "Significance of VHL changes in sporadic renal cell carcinoma." Thesis, University of Leeds, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581867.

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The von Hippel-Lindau (VHL) tumour suppressor gene is central to the development of sporadic conventional clear cell renal cell carcinoma (ccRCC). The role VHL plays as part of a ubiquitin ligase targeting HIF-a for proteasomal degradation underpins many changes seen in ccRCC but the importance of other VHL functions and the clinical significance of specific genetic/epigenetic changes are not clear. Genetic and epigenetic analysis of VHL gene in 86 tumours from patients with ccRCC was carried out, adding to the 96 tumours already analysed in an ongoing study within the group. Overall, loss of

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20

Chen, Dong. "Identification of new prognostic markers in renal cell carcinoma." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-168397.

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The clinical course of renal cell carcinoma (RCC) shows a high variability. Prognostic markers are essential to enable an individualized therapeutic strategy. The objective of this study was the identification of novel independent prognostic markers and potential therapeutic targets in RCC. The focus was on genes involved in epithelial-mesenchymal transition (EMT) and cancer stem cell biology. EMT enhances tumor cell motility and hence plays a critical role in invasion and metastasis in various carcinomas. A set of transcription factors acts as master regulators of EMT. Whether EMT is import

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21

Volk, Andreas, Stephan Kersting, Ralf Konopke, et al. "Surgical Therapy of Intrapancreatic Metastasis from Renal Cell Carcinoma." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-136489.

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Background: Pancreatic métastases from renal cell carcinoma (RCC) are clinically rare but highly resectable. The aim of this article is to identify patients who profit from pancreatic resection of RCC despite the invasiveness of the surgery. Methods: Between January 1996 and December 2007, data from 744 patients were collected in a prospective pancreatic surgery database, and patients with metastasis into the pancreas from RCC were identified. Results: Resective surgery was performed in 14 patients with metastasis to the pancreas from RCC. Most patients were clinically asymptomatic. The median

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22

Fröhner, Michael, Andreas Manseck, Arndt Lossnitzer, and Manfred P. Wirth. "Late Local and Pulmonary Recurrence of Renal Cell Carcinoma." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-133953.

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Locally recurrent renal cell carcinoma and multiple pulmonary metastases were successfully resected in a patient 20 years after nephrectomy<br>Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich

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23

Foster, Keith. "Molecular genetic analysis of non-familial renal cell carcinoma." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289811.

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24

Jafri, Mariam. "Molecular characterisation of renal cell carcinoma and related disorders." Thesis, University of Birmingham, 2016. http://etheses.bham.ac.uk//id/eprint/6996/.

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Over the last two decades genetic advances have provided novel insights into the molecular basis of familial and sporadic cancers and provided the basis for the development of novel therapeutic approaches. For example, the identification of the gene for von Hippel Lindau disease provided seminal insights into its role in most clear cell renal carcinomas (RCC) and led to new treatments for RCC. In this thesis I investigated three related genetic aspects of neoplasia. Firstly, I analyzed the results of genetic testing for inherited phaeochromocytoma and investigated how clinical features could b

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25

Qayyum, Tahir. "The role of Src kinase in renal cell carcinoma." Thesis, University of Glasgow, 2014. http://theses.gla.ac.uk/5600/.

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Renal cancer is a malignancy which is not only increasing in incidence but there has also been an increase in mortality rates. There are various prognostic factors in renal cell cancer. We have demonstrated that some of these such as nuclear grading, tumour necrosis and systemic inflammatory response can be further refined to aid in prognosis but cannot be utilised at present to assess which would benefit from therapeutic agents when recurrence occurs. We investigated if SFK members are expressed in renal cancer. Eight SFK members were found to be expressed in renal cancer and were present to

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26

Fröhner, Michael, Andreas Manseck, Arndt Lossnitzer, and Manfred P. Wirth. "Late Local and Pulmonary Recurrence of Renal Cell Carcinoma." Karger, 1998. https://tud.qucosa.de/id/qucosa%3A27552.

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Locally recurrent renal cell carcinoma and multiple pulmonary metastases were successfully resected in a patient 20 years after nephrectomy.<br>Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich.

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27

Volk, Andreas, Stephan Kersting, Ralf Konopke, et al. "Surgical Therapy of Intrapancreatic Metastasis from Renal Cell Carcinoma." Karger, 2009. https://tud.qucosa.de/id/qucosa%3A27708.

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Background: Pancreatic métastases from renal cell carcinoma (RCC) are clinically rare but highly resectable. The aim of this article is to identify patients who profit from pancreatic resection of RCC despite the invasiveness of the surgery. Methods: Between January 1996 and December 2007, data from 744 patients were collected in a prospective pancreatic surgery database, and patients with metastasis into the pancreas from RCC were identified. Results: Resective surgery was performed in 14 patients with metastasis to the pancreas from RCC. Most patients were clinically asymptomatic. The median

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28

Mastrandrea, Nicholas Joseph. "Pentoxifylline As An Adjuvant Treatment In Renal Cell Carcinoma." Diss., The University of Arizona, 2014. http://hdl.handle.net/10150/337293.

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Cyclin D1, a proto-oncogene, is required for progression from the G1 phase into the S phase of the cell cycle. Over-expression of cyclin D1 causes an increase in cell cycle progression and cell proliferation, implicating it in a variety of cancers including renal cell carcinoma (RCC). The rodent RCC cell model, QTRRE, and human RCC cell models, ACHN, 786-O and Caki-2, exhibit elevated levels of cyclin D1. Pentoxifylline (PTX), a non-specific phosphodiesterase inhibitor, is an FDA-approved hemorheologic agent used to treat intermittent claudication, stemming from peripheral vascular diseases, a

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29

Winegard, Billie. "Renal Cell Carcinoma in Arizona American Indians/Alaska Natives." Thesis, The University of Arizona, 2012. http://hdl.handle.net/10150/221595.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>OBJECTIVE – This study assesses trends in the incidence of cancers of the kidney and renal pelvis (K&RP) with focus on renal cell carcinoma (RCC) from 1995-2009 among American Indian/Alaska Natives (AI/AN) residing in Arizona. RESEARCH DESIGN AND METHODS – Using the Arizona Cancer Registry (ACR), we obtained the total number of new cases of cancers of the K&RP from 1995 through 2009. The incidence rates of these cancers, as well as t

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30

Bulmer, Bronwyn. "Prostate specific antigen-like expression in renal cell carcinoma." Thesis, Queensland University of Technology, 2002.

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31

Vemuri, Bhargav R. "Identification of prognostic metabolic classifier in localized clear cell renal cell carcinoma." University of Cincinnati / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin161710619577556.

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32

Kanno, Toru. "JunB promotes cell invasion and angiogenesis in VHL-defective renal cell carcinoma." Kyoto University, 2012. http://hdl.handle.net/2433/152497.

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33

Shibasaki, Noboru. "Role of IL13RA2 in Sunitinib Resistance in Clear Cell Renal Cell Carcinoma." Kyoto University, 2016. http://hdl.handle.net/2433/215954.

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34

Arakaki, Ryuichiro. "CCL2 as a potential therapeutic target for clear cell renal cell carcinoma." 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225490.

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35

Lee, Wing-sang, and 李榮生. "The prognostic significance of DJ-1 in patients with renal cell carcinoma of clear cell type." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B42925095.

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36

Lee, Wing-sang. "The prognostic significance of DJ-1 in patients with renal cell carcinoma of clear cell type." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B42925095.

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37

Bartrolí, Comellas Mariona. "Prognostic markers and therapeutic targets for metastatic renal cell carcinoma." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/664198.

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Targeting cancer metastasis has gained considerable importance in the recent years when aiming to increase patients’ overall survival. In Renal Cell Carcinoma (RCC), the discovery of metastatic biomarkers and targets is still required, as most patients present metastatic disease at the time of diagnosis. Therefore, the aim of this thesis is the discovery of new biomarkers and targets of metastatic RCC using two variants of a patient-derived orthoxenograft (PDOX) animal model of clear cell RCC (ccRCC). Indeed, PDOX models have recently gained significant relevance for studying the progressio

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38

Carter, Jessica. "Epigenetic basis for Tensin3 dysregulation in human renal cell carcinoma." Thesis, University of Portsmouth, 2013. https://researchportal.port.ac.uk/portal/en/theses/epigenetic-basis-for-tensin3-dysregulation-in-human-renal-cell-carcinoma(e39a962d-9c23-4265-8318-bf5fd296148c).html.

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The Tensins are a family of intracellular cytoskeleton interacting proteins that are involved in the regulation of cell motility and migration. Downregulation of Tensins has been observed in several cancers, indicating that it may be advantageous for tumour progression. In this study, an epigenetic basis for the observed downregulation of Tensin3 in human renal cell carcinoma (RCC) has been investigated, specifically the methylation state of the TNS3 gene promoter. The aims of this study were to: 1. Identify and validate a functional promoter for the human TNS3 gene that contains a CpG island

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39

Sandlund, Johanna. "Angiogenesis in human renal cell carcinoma : hypoxia, vascularity and prognosis." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1331.

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40

Fonseca, Cátia Isabel Correia dos Reis. "Vaccine Targets in a Murine Model of Renal Cell Carcinoma." Doctoral thesis, Instituto de Ciências Biomédicas Abel Salazar, 2007. http://hdl.handle.net/10216/7210.

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41

Fonseca, Cátia Isabel Correia dos Reis. "Vaccine Targets in a Murine Model of Renal Cell Carcinoma." Tese, Instituto de Ciências Biomédicas Abel Salazar, 2007. http://hdl.handle.net/10216/7210.

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42

Rae, Fiona Karen. "Identification and characterisation of genes expressed in renal cell carcinoma." Thesis, Queensland University of Technology, 2001.

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43

Arévalo, Bautista Jazmine Paola. "The role of stat3 phosphorylation state in clear cell renal cell carcinoma (ccRCC)." Doctoral thesis, Universitat Autònoma de Barcelona, 2019. http://hdl.handle.net/10803/669570.

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STAT3 (signal transducer and activator of transcription 3) és un factor de transcripció latent que regula la transcripció de gens relacionats amb processos biològics essencials tals com: la diferenciació cel·lular, la proliferació, la migració, la inhibició de l’apoptosi i la supervivència. L’activació anormal d’STAT3 s’ha relacionat amb el desenvolupament d’aproximadament el 50% dels càncers humans, incloent el carcinoma renal de cèl·lula clara (ccRCC). Actualment, les propietats oncogèniques d’STAT3 s’atribueixen a la fosforilació del seu residu Tyr705; no obstant, recentment, la fosforilaci

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44

Weber, Thomas, Matthias Meinhardt, Stefan Zastrow, et al. "Stage-dependent prognostic impact of molecular signatures in clear cell renal cell carcinoma." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-147292.

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Purpose: To enhance prognostic information of protein biomarkers for clear cell renal cell carcinomas (ccRCCs), we analyzed them within prognostic groups of ccRCC harboring different tumor characteristics of this clinically and molecularly heterogeneous tumor entity. Methods: Tissue microarrays from 145 patients with primary ccRCC were immunohistochemically analyzed for VHL (von Hippel-Lindau tumor suppressor), Ki67 (marker of proliferation 1), p53 (tumor protein p53), p21 (cyclin-dependent kinase inhibitor 1A), survivin (baculoviral IAP repeat containing 5), and UEA-1 (ulex europaeus agglutin

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45

Polanski, Radoslaw. "Identification of MDM2-interacting proteins in renal cell carcinoma and characterization of phenotypic properties acquired by renal cell carcinoma cells which over-express MDM2 and p53." Thesis, University of Liverpool, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.533936.

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46

Reid, Janet Louise. "The mechanism of action of captopril in human renal cell carcinoma /." [St. Lucia, Qld.], 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17182.pdf.

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47

Gutteridge, Robert. "Caveolin-1 is a modulator of clonogenicity in renal cell carcinoma." Thesis, Cardiff University, 2015. http://orca.cf.ac.uk/91911/.

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Renal cell carcinoma (RCC) represents a group of aggressive tumours of the kidney. These tumours have a high propensity for metastasis and are extremely treatment refractive after disease relapse. Therefore, the identification of new therapeutic targets is of great importance. One such potential target for therapy are cancer stem cells (CSCs). CSCs are populations of cells imbued with a stem cell-like phenotype capable of driving tumour formation, metastasis and chemoresistance. As such, reliable methods for the identification of CSC populations and defining targets important to their function

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48

Iranparvar, Alamdari Farhood. "Renal cell carcinoma : factors of importance for follow-up and survival." Doctoral thesis, Umeå : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1418.

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49

Aggelis, Vassilis. "Proteomic identification of differentially expressed membrane proteins in renal cell carcinoma." Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.530852.

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50

Wolfe, Kara. "The Role of Purine Nucleotide Metabolism in Renal Cell Carcinoma Migration." University of Cincinnati / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563872926565308.

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