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Artículos de revistas sobre el tema "Trauma shock"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 12 de febrero de 2022

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1

Scherer, Priscilla. "Shock Trauma". American Journal of Nursing 89, n.º 11 (noviembre de 1989): 1440. http://dx.doi.org/10.2307/3426144.

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2

Willard, Louise A. "Shock Trauma!" Journal of Christian Nursing 10, n.º 3 (1993): 26–29. http://dx.doi.org/10.1097/00005217-199310030-00009.

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3

SCHERER, PRISCILLA. "SHOCK TRAUMA". AJN, American Journal of Nursing 89, n.º 11 (noviembre de 1989): 1440–45. http://dx.doi.org/10.1097/00000446-198911000-00016.

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4

Bhubaneswari, T. S. y Ajeet Singh. "Irony as Trauma & Trauma as Irony in Claude Lanzmann’s Shoah". Contemporary Research: An Interdisciplinary Academic Journal 3, n.º 1 (31 de diciembre de 2019): 67–71. http://dx.doi.org/10.3126/craiaj.v3i1.27492.

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Claude Lanzmann makes use of what this essay posits as metairony which dramatizes the shocks of the acting out of the trauma of the Holocaust. The film director makes the survivors and witnesses and the viewers to become retraumatized and to relive the past. By so doing, the traumatized mind can cope with the trauma because acting out helps the reflective consciousness to prevent itself from being overwhelmed by shock, in Walter Benjamin’s assumption, by reproducing shock, that is, by seizing upon each traumatic moment and parrying it - in effect, by responding to violence with violence. Testimonies in Shoah break the boundary between the experience of shock and experience as shock.
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5

Armstrong, Bruce y Julia Carpenter. "Shock trauma Baltimore, USA". Intensive and Critical Care Nursing 11, n.º 3 (junio de 1995): 151–56. http://dx.doi.org/10.1016/s0964-3397(95)80657-1.

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6

Dillworth, Judy y Julie Mill Strange. "Shock Trauma Care Plans". American Journal of Nursing 89, n.º 1 (enero de 1989): 140. http://dx.doi.org/10.2307/3471034.

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7

Dutton, Richard P. "SHOCK AND TRAUMA ANESTHESIA". Anesthesiology Clinics of North America 17, n.º 1 (marzo de 1999): 83–95. http://dx.doi.org/10.1016/s0889-8537(05)70080-8.

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8

Norris, H. Thomas. "Trauma, Sepsis, and Shock". American Journal of Surgical Pathology 13, n.º 6 (junio de 1989): 530. http://dx.doi.org/10.1097/00000478-198906000-00019.

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9

Ohnishi, Mitsuo. "Shock Wave Trauma Research". Impact 2019, n.º 3 (22 de marzo de 2019): 76–78. http://dx.doi.org/10.21820/23987073.2019.3.76.

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10

DILLWORTH, JUDY. "Shock Trauma Care Plans". AJN, American Journal of Nursing 89, n.º 1 (enero de 1989): 140. http://dx.doi.org/10.1097/00000446-198901000-00036.

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11

Strange, J. M. "Shock Trauma Care Plans". Dimensions Of Critical Care Nursing 7, n.º 2 (marzo de 1988): 101. http://dx.doi.org/10.1097/00003465-198803000-00006.

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12

Robson, Martin C. "Trauma, Sepsis, and Shock". Plastic and Reconstructive Surgery 84, n.º 1 (julio de 1989): 166. http://dx.doi.org/10.1097/00006534-198907000-00033.

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13

Staniforth, Paul. "Shock trauma care plans". Injury 19, n.º 3 (mayo de 1988): 222. http://dx.doi.org/10.1016/0020-1383(88)90024-1.

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14

Bedreag, Ovidiu Horea, Alexandru Florin Rogobete, Mirela Sarandan, Alina Carmen Cradigati, Radu Nartita, Dorel Sandesc y Marius Papurica. "Volemic Resuscitation in a Patient with Multiple Traumas and Haemorrhagic Shock. Anti-oxidative Therapy Management in Critical Patients. A Case Report". Acta Medica Marisiensis 62, n.º 1 (1 de marzo de 2016): 152–54. http://dx.doi.org/10.1515/amma-2016-0002.

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Abstract A patient with multiple traumas is usually found in severe haemorrhagic shock. In 40% of the cases, the patient with multiple traumas and haemorrhagic shock cannot recover due to secondary injuries and complications associated with the shock. In this paper we present the case of a male patient 30 years old, who suffered a car accident. The patient is admitted in our hospital with haemorrhagic shock due to femur fracture, acute cranial-cerebral trauma and severe thoracic trauma with bleeding scalp wound, associated with lethal triad of trauma. The clinical and biological parameters demand massive transfusion with packed red blood cells (PRBCs), fresh frozen plasma (FFP), cryoprecipitate (CRY) and colloidal solution (CO) sustained with vassopresor for the haemodynamic stabilisation. During his stay in the ICU, the patient benefits from anti-oxidative therapy with Vitamin C, Vitamin E and Vitamin B1. After 14 days the clinical state of the patient improves and he is transferred in Polytrauma Department.
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15

Albaran, RG, LN Diebel, DM Liberati, SA Dulchavsky, TA Myers y PC Montgomery. "SECRETORY IgA RESPONSES FOLLOWING TRAUMA AND SHOCK TRAUMA." Shock 5 (junio de 1996): 12. http://dx.doi.org/10.1097/00024382-199606002-00037.

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16

Greeson, S. Douglas. "Shock Trauma/Critical Care Handbook". Critical Care Medicine 15, n.º 10 (octubre de 1987): 991. http://dx.doi.org/10.1097/00003246-198710000-00026.

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17

Iserson, Kenneth V. "Shock Trauma/Critical Care Manual". Critical Care Medicine 15, n.º 12 (diciembre de 1987): 1166. http://dx.doi.org/10.1097/00003246-198712000-00027.

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18

Griffith, Richard. "Negligence, trauma and nervous shock". British Journal of Nursing 29, n.º 11 (11 de junio de 2020): 642–43. http://dx.doi.org/10.12968/bjon.2020.29.11.642.

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Richard Griffith, Senior Lecturer in Health Law at Swansea University, considers whether nurses who have suffered psychological trauma because of the negligent handling of the COVID-19 outbreak can claim compensation
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19

Berry, Robin D. "Management of shock in trauma". Anaesthesia & Intensive Care Medicine 9, n.º 9 (septiembre de 2008): 390–93. http://dx.doi.org/10.1016/j.mpaic.2008.07.006.

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20

Pearson, Jonathan D., Jonathan A. Round y Michael Ingram. "Management of shock in trauma". Anaesthesia & Intensive Care Medicine 12, n.º 9 (septiembre de 2011): 387–89. http://dx.doi.org/10.1016/j.mpaic.2011.06.005.

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21

Pearson, Jonathan D., Jonathan A. Round y Michael Ingram. "Management of shock in trauma". Anaesthesia & Intensive Care Medicine 15, n.º 9 (septiembre de 2014): 408–10. http://dx.doi.org/10.1016/j.mpaic.2014.06.007.

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22

Boyd, Matthew y Damian D. Keene. "Management of shock in trauma". Anaesthesia & Intensive Care Medicine 18, n.º 8 (agosto de 2017): 386–89. http://dx.doi.org/10.1016/j.mpaic.2017.05.002.

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23

O'Sullivan, Fin y Rob O'Donnell. "Management of shock in trauma". Anaesthesia & Intensive Care Medicine 21, n.º 8 (agosto de 2020): 393–96. http://dx.doi.org/10.1016/j.mpaic.2020.05.004.

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24

Stewart-Amidei, Christina. "The “Shock” of Trauma Care". Journal of Neuroscience Nursing 22, n.º 2 (abril de 1990): 63. http://dx.doi.org/10.1097/01376517-199004000-00001.

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25

Berry, Robin D. "Management of shock in trauma". Anaesthesia & Intensive Care Medicine 6, n.º 9 (septiembre de 2005): 308–10. http://dx.doi.org/10.1383/anes.2005.6.9.308.

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26

Goris, R. J. A. "Pathophysiology of Shock in Trauma". European Journal of Surgery 166, n.º 2 (24 de enero de 2000): 100–111. http://dx.doi.org/10.1080/110241500750009438.

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27

Tubbs, N. "Shock trauma/critical care manual". Injury 16, n.º 8 (septiembre de 1985): 576. http://dx.doi.org/10.1016/0020-1383(85)90122-6.

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28

Kim, Seung-Ho. "Treatment of Shock in Trauma Patients". Journal of the Korean Medical Association 42, n.º 5 (1999): 429. http://dx.doi.org/10.5124/jkma.1999.42.5.429.

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29

Rodríguez-González, Fayna y Efrén Martínez-Quintana. "Cardiogenic shock following blunt chest trauma". Journal of Emergencies, Trauma, and Shock 3, n.º 4 (2010): 398. http://dx.doi.org/10.4103/0974-2700.70772.

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30

Kirkman, E. y R. A. Little. "The pathophysiology of trauma and shock". Baillière's Clinical Anaesthesiology 2, n.º 3 (septiembre de 1988): 467–82. http://dx.doi.org/10.1016/s0950-3501(88)80002-3.

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31

Traber, Daniel L. "Arginine in shock, trauma, and sepsis*". Critical Care Medicine 30, n.º 3 (marzo de 2002): 705–6. http://dx.doi.org/10.1097/00003246-200203000-00038.

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32

Gando, Satoshi, Atsushi Sawamura y Mineji Hayakawa. "Trauma, Shock, and Disseminated Intravascular Coagulation". Annals of Surgery 254, n.º 1 (julio de 2011): 10–19. http://dx.doi.org/10.1097/sla.0b013e31821221b1.

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33

Napolitano, Lena M. "Resuscitation following trauma and hemorrhagic shock". Critical Care Medicine 23, n.º 5 (mayo de 1995): 795–97. http://dx.doi.org/10.1097/00003246-199505000-00001.

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34

Korshnyak, Volodymyr y Viktor Sukhorukov. "CLINICONEUROLOGICAL AND NEUROPSYCHOLOGICAL ASPECTS OF ACUTE PERIOD OF MILD CRANIAL BRAIN TRAUMA CAUSED BY SHOCK WAVE". EUREKA: Health Sciences 1 (29 de enero de 2016): 14–18. http://dx.doi.org/10.21303/2504-5679.2016.00035.

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Purpose: to study the clinical, neurological, and psychological status of patients at acute period of mild cranial brain trauma caused by shock wave. Material: patients participated in fighting actions in the East of Ukraine, which were treated in neurological department of Military medical clinical centre of Northern region in 2015. Results: authors have revealed some symptoms of sensory disorders in the form of nonspecific sensomotor hemisyndrome and general analgesia. Conclusions: Mild closed cranial brain trauma, caused by shock wave, in its acute period has more severe course than in civilian traumas. The identified symptoms are the result of complex influence of shock wave on the central nervous system and nonspecific structures of brain that subsequently can lead to the vegetative nervous system’s disorders and impairment of higher cortical (mental) functions, and also to adjustment disorders and social maladjustment.
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35

Özçete, Enver, Selahattin Kiyan, İlhan Uz, Meltem Songür Kodik y Yusuf Ali Altuncı. "The role of whole-body computed tomography in determining risky patient group with regard to polytrauma patients in the emergency department". Hong Kong Journal of Emergency Medicine 25, n.º 3 (14 de febrero de 2018): 123–29. http://dx.doi.org/10.1177/1024907918755174.

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Backround: High rates in trauma-related mortality pose a major health problem and increase every day. Early diagnosis and treatment can be lifesavers for this patient group in the emergency departments, which serve as the first place to admit trauma patients in a hospital. Objectives: We aim to determine high-risk criteria to indicate trauma patients getting the most use from whole-body tomography in patients with multiple traumas and reduce unnecessary computed tomography. Methods: We examined retrospectively all electronic files and computed tomography results of patients, who had been admitted to emergency department due to trauma, and who had undergone whole-body computed tomography. Results: We found that possibility of multiple injuries increased by 5.9 times in patients requiring mechanical ventilation. Possibility of multiple injuries in patients with free fluid in the Focused Assessment with Sonography for Trauma increased by 5.6 times. We also observed that possibility of multiple injuries in patients with Glasgow Coma Score < 13 increased by 4.3 times. Possibility of multiple injuries in hypoxic patients increased by 3.2 times. Possibility of multiple injuries in patients with a pulse ≥ 120/min increased by 1.8 times. Possibility of multiple injuries in patients with shock index ≥ 0.9 increased by 1.7 times. Conclusion: High-risk group in terms of multiple traumas involves mechanical ventilation need in trauma patients, positive Focused Assessment with Sonography for Trauma, Glasgow Coma Score being under 13, hypoxia, tachycardia, positive shock index, and extravehicular traffic accidents. Whole-body computed tomography should be performed in this patient group.
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36

Gando, Satoshi. "Hemostasis and Thrombosis in Trauma Patients". Seminars in Thrombosis and Hemostasis 41, n.º 01 (20 de enero de 2015): 026–34. http://dx.doi.org/10.1055/s-0034-1398378.

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Hemostasis and thrombosis in trauma patients consist of physiological hemostasis for wound healing and the pathological reaction of disseminated intravascular coagulation (DIC). Whole body trauma, isolated brain injury, and fat embolism syndrome, if extremely severe, can cause DIC and affect a patient's prognosis. Shock-induced hyperfibrinolysis causes DIC with the fibrinolytic phenotype, contributing to oozing-type severe bleeding. If uncontrolled, this phenotype progresses to thrombotic phenotype at the late stage of trauma, followed by microvascular thrombosis, leading to organ dysfunction. Another type of pathological hemostatic change is acute coagulopathy of trauma shock (ACOTS), which gives rise to activated protein C–mediated systemic hypocoagulation, resulting in bleeding. ACOTS occurs only in trauma associated with shock-induced hypoperfusion and there is nothing to suggest DIC in this phenomenon. This review will provide information about the recent advances in hemostasis and thrombosis in trauma and will clarify the pathogeneses of the pathological processes observed in trauma patients.
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37

Grodzka, Martyna. "Trauma I wojny światowej (shell shock) w poezji brytyjskich żołnierzy oraz w modernistycznej prozie kobiecej". Annales Universitatis Paedagogicae Cracoviensis | Studia Historicolitteraria 15 (12 de diciembre de 2017): 45–54. http://dx.doi.org/10.24917/3912.

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First World War shell shock in British poetry of soldier-poets and modernist prose of women writers The purpose of the following article is to compare and contrast the literary modes of representation of the First World War shell shock in the poetry of soldier-poets and in the prose of women writers. The war trauma called “shell shock” had a profound impact on British literature and the common memory of the Great World. At the time, the poets who were soldiers expressed their traumatic experiences in their works. Meanwhile, women authors who observed veterans suffering from trauma explored the causes and effects of shell shock in their prose.Keywords: trauma; shell shock; war; soldier;
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38

Xu, Ying Xin, Alfred Ayala y Irshad H. Chaudry. "Prolonged Immunodepression after Trauma and Hemorrhagic Shock". Journal of Trauma: Injury, Infection, and Critical Care 44, n.º 2 (febrero de 1998): 335–41. http://dx.doi.org/10.1097/00005373-199802000-00018.

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39

Heckbert, Susan R., Nicholas B. Vedder, Wilma Hoffman, Robert K. Winn, Leonard D. Hudson, Gregory J. Jurkovich, Michael K. Copass, John M. Harlan, Charles L. Rice y Ronald V. Maier. "Outcome after Hemorrhagic Shock in Trauma Patients". Journal of Trauma: Injury, Infection, and Critical Care 45, n.º 3 (septiembre de 1998): 545–49. http://dx.doi.org/10.1097/00005373-199809000-00022.

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40

Beloeil, Helene, Jean-Xavier Mazoit y Jacques Duranteau. "Norepinephrine Kinetics in Shock and Trauma Patients". Anesthesiology 96, Sup 2 (septiembre de 2002): A375. http://dx.doi.org/10.1097/00000542-200209002-00375.

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41

Stevenson, Judy. "Shock in a box: A trauma puzzle". Journal of PeriAnesthesia Nursing 19, n.º 4 (agosto de 2004): 268. http://dx.doi.org/10.1016/j.jopan.2004.06.010.

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42

Povidaylo, L., E. Tkachenko, Ya Aleksevich y V. Kovalyshyn. "TRAUMATIC SHOCK IN TRANSPORT TRAUMA (PATHOLOGY STUDY)". Shock 12, Supplement (noviembre de 1999): 54. http://dx.doi.org/10.1097/00024382-199911001-00167.

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43

Lee, Raphael C., Dajun Zhang y Jurgen Hannig. "Biophysical Injury Mechanisms in Electrical Shock Trauma". Annual Review of Biomedical Engineering 2, n.º 1 (agosto de 2000): 477–509. http://dx.doi.org/10.1146/annurev.bioeng.2.1.477.

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44

Sparkes, Brian. "Immune consequences of trauma, shock and sepsis". Burns 16, n.º 6 (diciembre de 1990): 482. http://dx.doi.org/10.1016/0305-4179(90)90099-i.

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45

Wells, John Paul, Arif Razzak y Sankar Ananth. "Facial Imaging in Shock Trauma – Who cares?" British Journal of Oral and Maxillofacial Surgery 55, n.º 10 (diciembre de 2017): e71-e72. http://dx.doi.org/10.1016/j.bjoms.2017.08.321.

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46

Zeckey, Christian y Christian Kammerlander. "Whats new in emergencies trauma and shock? Age and trauma: Geriatric trauma patients and geriatric trauma ward services". Journal of Emergencies, Trauma, and Shock 10, n.º 3 (2017): 89. http://dx.doi.org/10.4103/0974-2700.212495.

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47

van Veelen, Michiel J. y Monika Brodmann Maeder. "Hypothermia in Trauma". International Journal of Environmental Research and Public Health 18, n.º 16 (18 de agosto de 2021): 8719. http://dx.doi.org/10.3390/ijerph18168719.

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Hypothermia in trauma patients is a common condition. It is aggravated by traumatic hemorrhage, which leads to hypovolemic shock. This hypovolemic shock results in a lethal triad of hypothermia, coagulopathy, and acidosis, leading to ongoing bleeding. Additionally, hypothermia in trauma patients can deepen through environmental exposure on the scene or during transport and medical procedures such as infusions and airway management. This vicious circle has a detrimental effect on the outcome of major trauma patients. This narrative review describes the main factors to consider in the co-existing condition of trauma and hypothermia from a prehospital and emergency medical perspective. Early prehospital recognition and staging of hypothermia are crucial to triage to proper care to improve survival. Treatment of hypothermia should start in an early stage, especially the prevention of further cooling in the prehospital setting and during the primary assessment. On the one hand, active rewarming is the treatment of choice of hypothermia-induced coagulation disorder in trauma patients; on the other hand, accidental or clinically induced hypothermia might improve outcomes by protecting against the effects of hypoperfusion and hypoxic injury in selected cases such as patients suffering from traumatic brain injury (TBI) or traumatic cardiac arrest.
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48

Shiryazdi, SeyedMostafa, Mohammad Mirshamsi, HamidReza Piri Ardakani y SeyedAli Shiryazdi. "Relationship Between Shock Index and Clinical Outcome in Patients with Multiple Traumas". Internal Medicine and Medical Investigation Journal 2, n.º 3 (11 de septiembre de 2017): 94. http://dx.doi.org/10.24200/imminv.v2i3.90.

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Background: Initial assessment of hemodynamic parameters and timely management of patients regarding hypovolemic shock occurrence is the most essential clinical action in trauma patients and shock index (SI) has considerable accuracy associated with wide application. Therefore, this study is planned to evaluate the relationship of the shock index and clinical outcome in patients with multiple trauma referring to Shahid Sadoughi Hospital of Yazd in 2011.Methods: The present study was a descriptive cross-sectional study carried out on 334 patients with multiple trauma referring to Emergency Center of Shahid Sadoughi Hospital of Yazd in 2011. Patients were divided into two separate groups based on Shock index score (≥ 0.9 as abnormal SI and < 0.9 as normal SI).Finally, data were analyzed using Chi-square and independent sample t-test in SPSS ver.19.Results: There was significant difference between the two groups in terms of mean of and gender distribution (P= 0.001). There was also a significant difference between patients with head and neck trauma and pelvic injuries in terms of frequency distribution (P< 0.05). Hemodynamic parameters were also significantly different in the two studied groups (P< 0.001). Also, with regard to the frequency distribution of intensive care unit admission (ICU) and mortality rate, there was significant difference in the two groups.Conclusion: Shock index has considerable predictive value in patients with multiple trauma and can be used in initial management and assessment of patients with multiple trauma before any other diagnostic procedures since it is easily calculated. Shock index can also rapidly diagnose the real condition of trauma patient in primary hours and prevent secondary unpleasant clinical outcomes.
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49

Liu, Fu-Chao, Chih-Wen Zheng y Huang-Ping Yu. "Maraviroc-Mediated Lung Protection following Trauma-Hemorrhagic Shock". BioMed Research International 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/5302069.

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Objectives.The peroxisome proliferator-activated receptor gamma (PPARγ) pathway exerts anti-inflammatory effects in response to injury. Maraviroc has been shown to have potent anti-inflammatory effects. The aim of this study was to investigate whether PPARγplays an important role in maraviroc-mediated lung protection following trauma-hemorrhage.Methods.Male Sprague-Dawley rats underwent trauma-hemorrhage (mean blood pressure maintained at approximately 35–40 mmHg for 90 minutes), followed by fluid resuscitation. During resuscitation, a single dose of maraviroc (3 mg/kg, intravenously) with and without a PPARγinhibitor GW9662 (1 mg/kg, intravenously), GW9662, or vehicle was administered. Lung water content, tissue histology, and other various parameters were measured (n=8rats/group) 24 hours after resuscitation. One-way ANOVA and Tukey’s testing were used for statistical analysis.Results.Trauma-hemorrhage significantly increased lung water content, myeloperoxidase activity, intercellular adhesion molecule-1, interleukin-6, and interleukin-1βlevels. These parameters significantly improved in the maraviroc-treated rats subjected to trauma-hemorrhage. Maraviroc treatment also decreased lung tissue damage as compared to the vehicle-treated trauma-hemorrhaged rats. Coadministration of GW9662 with maraviroc abolished the maraviroc-induced beneficial effects on these parameters and lung injury.Conclusion.These results suggest that PPARγmight play a key role in maraviroc-mediated lung protection following trauma-hemorrhage.
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50

Fishman, Jordan E., Gal Levy, Vamsi Alli, Sharvil Sheth, Qu Lu y Edwin A. Deitch. "Oxidative modification of the intestinal mucus layer is a critical but unrecognized component of trauma hemorrhagic shock-induced gut barrier failure". American Journal of Physiology-Gastrointestinal and Liver Physiology 304, n.º 1 (1 de enero de 2013): G57—G63. http://dx.doi.org/10.1152/ajpgi.00170.2012.

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Recent studies demonstrate that mechanisms underlying gut barrier failure include systemic processes and less studied luminal processes. We thus tested the hypothesis that mucus layer oxidation is a component of trauma/hemorrhagic shock-induced gut injury and dysfunction. Male Sprague-Dawley rats underwent trauma/hemorrhagic shock. Controls underwent trauma only. Mucus from the terminal 30 cm of the ileum was collected, processed, and analyzed for reactive nitrogen intermediates (RNI)-mediated damage, reactive oxygen species (ROS)-induced damage, and total antioxidant capacity. The distal ileum was stained to quantify the mucus layer; gut permeability was assessed physiologically. A time course study was conducted to determine the temporal sequence of mucus layer damage. The role of free radical-mediated damage to the gut barrier was investigated by the effect of the free radical scavenger dimethyl sulfoxide on trauma/hemorrhagic shock-induced changes on the mucus and on gut permeability. Trauma/hemorrhagic shock increased intestinal permeability, which was associated with evidence of loss of the unstirred mucus layer. These changes correlated with increased ROS- and RNI-mediated mucus damage and loss of mucus total antioxidant capacity. Based on the time course study, ROS-mediated mucus damage and loss of total antioxidant capacity were present immediately following shock, whereas RNI-mediated damage was delayed for 3 h. Dimethyl sulfoxide ameliorated gut barrier loss, ROS-mediated changes to the mucus layer, and loss of total antioxidant capacity. There was no change in RNI-induced changes to the mucus layer. These results support the hypothesis that trauma/hemorrhagic shock leads to mucus damage and gut dysfunction through the generation of free radical species.
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