Bibliografías temáticas / Vascular resistance Measurement

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Literatura académica sobre el tema "Vascular resistance Measurement"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 29 de enero de 2023

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Artículos de revistas sobre el tema "Vascular resistance Measurement"

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O'DWYER, J. P., J. E. KING, C. E. WOOD, B. L. TAYLOR, and G. B. SMITH. "Continuous measurement of systemic vascular resistance." Anaesthesia 49, no. 7 (February 22, 2007): 587–90. http://dx.doi.org/10.1111/j.1365-2044.1994.tb14225.x.

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Abbas, Amr E., F. David Fortuin, Bhavesh Patel, Carlos A. Moreno, Nelson B. Schiller, and Steven J. Lester. "Noninvasive measurement of systemic vascular resistance using Doppler echocardiography." Journal of the American Society of Echocardiography 17, no. 8 (August 2004): 834–38. http://dx.doi.org/10.1016/j.echo.2004.04.008.

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Schrijen, F., C. Saunier, and F. Chabot. "Peripheral pulmonary vascular resistance." Journal of Applied Physiology 74, no. 2 (February 1, 1993): 613–16. http://dx.doi.org/10.1152/jappl.1993.74.2.613.

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The pressure-flow relationship has been studied in a peripheral portion of the lung vasculature in anesthetized dogs with use of a double-lumen catheter wedged in a distal pulmonary artery. One lumen was used to infuse mixed venous blood in the wedged area and the other to measure the corresponding perfusion pressure. Flow ranged from 0 to 9.2 ml/min, and the mean volume of the wedged area (n = 59) was 0.75 +/- 0.05 (SE) ml. In the areas where the distal pulmonary artery was in the same direction as the catheter ("coaxial"), the mean pressure-flow curve showed a negligible gamma-intercept and
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Moriyasu, Fuminori, Osamu Nishida, Nobuyuki Ban, Takefumi Nakamura, Kensuke Miura, Masahiko Sakai, Takeo Miyake, and Haruto Uchino. "Measurement of Portal Vascular Resistance in Patients With Portal Hypertension." Gastroenterology 90, no. 3 (March 1986): 710–17. http://dx.doi.org/10.1016/0016-5085(86)91127-3.

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Lautt, W. W., C. V. Greenway, D. J. Legare, and H. Weisman. "Localization of intrahepatic portal vascular resistance." American Journal of Physiology-Gastrointestinal and Liver Physiology 251, no. 3 (September 1, 1986): G375—G381. http://dx.doi.org/10.1152/ajpgi.1986.251.3.g375.

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The pressure drop from the portal vein to the vena cava occurs primarily across a postsinusoidal site localized to a narrow segment (less than 0.5 cm) of hepatic veins (roughly 1.5 mm diam) in the anesthetized cat. Portal venous pressure (PVP = 8.9 +/- 0.3 mmHg) and lobar hepatic venous pressure (LVP = 8.7 +/- 0.4 mmHg) are insignificantly different, and pressure changes imposed from the presinusoidal or postsinusoidal side are equally transmitted to both pressure sites. Several types of experiments were done to validate the LVP measurement. The portal vein, hepatic sinusoids, and hepatic vein
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Allison, R. C., B. Rippe, V. R. Prasad, J. C. Parker, and A. E. Taylor. "Pulmonary vascular permeability and resistance measurements in control and ANTU-injured dog lungs." American Journal of Physiology-Heart and Circulatory Physiology 256, no. 6 (June 1, 1989): H1711—H1718. http://dx.doi.org/10.1152/ajpheart.1989.256.6.h1711.

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Because questions have arisen regarding pulmonary vascular permeability and resistance measurements in isolated, perfused lungs, we sought to determine the 1) stability of repeated measurements of permeability and resistance in control lungs; and 2) magnitude of change in these measurements when permeability was greatly increased. Using blood-perfused dog lungs, we measured filtration coefficient (Kf) and isogravimetric capillary pressure (Pci) as indexes of vascular permeability, and we also determined total vascular resistance (Rt) as well as the segmental resistances using the double-occlus
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Parker, James C., Mark N. Gillespie, Aubrey E. Taylor, and Sherri L. Martin. "Capillary filtration coefficient, vascular resistance, and compliance in isolated mouse lungs." Journal of Applied Physiology 87, no. 4 (October 1, 1999): 1421–27. http://dx.doi.org/10.1152/jappl.1999.87.4.1421.

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Although many recently produced transgenic mice possess gene alterations affecting pulmonary vascular function, there are few baseline measurements of vascular resistance and permeability. Therefore, we excised the lungs of C57/BL6 mice and perfused them with 5% bovine serum albumin in RPMI-1640 culture medium at a nominal flow of 0.5 ml/min and ventilated them with 20% O2-5% CO2-75% N2. The capillary filtration coefficient, a sensitive measurement of hydraulic conductivity, was unchanged over 2 h (0.33 ± 0.03 ml ⋅ min−1 ⋅ cmH2O−1 ⋅ 100 g−1) in a control group ventilated with low peak inflatio
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Giesen, Leonie A., Michelle White, and Robert M. R. Tulloh. "Comparison of the effect of inhaled anaesthetic with intravenous anaesthetic on pulmonary vascular resistance measurement during cardiac catheterisation." Cardiology in the Young 25, no. 2 (February 19, 2014): 368–72. http://dx.doi.org/10.1017/s1047951114000195.

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AbstractBackground: Children with pulmonary hypertension routinely undergo pulmonary vascular resistance studies to assess the disease severity and vasodilator responsiveness. It is vital that results are accurate and reliable and are not influenced by the choice of anaesthetic agent. However, there are anecdotal data to suggest that propofol and inhalational agents have different effects on pulmonary vascular resistance. Methods: A total of 10 children with pulmonary hypertension were selected sequentially to be included in the study. To avoid confounding because of baseline anatomic or demog
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Parvin, S. D., D. H. Evans, and P. R. F. Bell. "Peripheral resistance measurement in the assessment of severe peripheral vascular disease." British Journal of Surgery 72, no. 9 (September 1985): 751–53. http://dx.doi.org/10.1002/bjs.1800720928.

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Rothe, C. F., and R. Maass-Moreno. "Gastrointestinal hemodynamics during compensation for hemorrhage and measurement of Pmcf." American Journal of Physiology-Heart and Circulatory Physiology 266, no. 3 (March 1, 1994): H1242—H1250. http://dx.doi.org/10.1152/ajpheart.1994.266.3.h1242.

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To quantify the degree of autonomic reflex control of the gastrointestinal vasculature, we studied the responses to a 10-ml/kg hemorrhage or transfusion and autonomic blockade in fentanyl- and pentobarbital-anesthetized dogs. The active total blood volume was estimated by indocyanine green dilution. Transfusion and hemorrhage did not significantly change gastrointestinal vascular compliance [1.82 +/- 0.68 (SD) ml/mmHg], but autonomic blockade with hexamethonium and atropine increased it by 0.57 +/- 0.37 ml/mmHg. Neither hemorrhage nor autonomic blockade significantly changed gastrointestinal v
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Tesis sobre el tema "Vascular resistance Measurement"

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Duncan, Henry J. (Henry John). "An isotope washout technique to study skin perfusion pressure and vascular resistance in diabetes, hypertension and peripheral vascular disease." 1986. http://web4.library.adelaide.edu.au/theses/09MD/09mdd911.pdf.

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Duncan, Henry J. (Henry John). "An isotope washout technique to study skin perfusion pressure and vascular resistance in diabetes, hypertension and peripheral vascular disease / by Henry J. Duncan." Thesis, 1986. http://hdl.handle.net/2440/38294.

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Libros sobre el tema "Vascular resistance Measurement"

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Comparison of total peripheral resistance and blood velocity as obtained from Doppler ultrasound waveforms during rest, exercise and recovery. 1991.

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Comparison of total peripheral resistance and blood velocity as obtained from Doppler ultrasound waveforms during rest, exercise and recovery. 1991.

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Comparison of total peripheral resistance and blood velocity as obtained from Doppler ultrasound waveforms during rest, exercise and recovery. 1992.

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4

Prout, Jeremy, Tanya Jones, and Daniel Martin. Respiratory system. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199609956.003.0002.

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This chapter includes a summary of respiratory physiology, respiratory mechanics (pressure-volume relationships and compliance, airway resistance and the work of breathing) and the pulmonary circulation (pulmonary vascular resistance, shunt and lung zones). Measurement of respiratory flow, lung volumes and diffusion capacity is summarized, as well as measurement and interpretation of arterial blood gases. The physics behind capnography and pulse oximetry are explained with abnormalities related to clinical contexts. The common clinical scenarios of respiratory failure and asthma are discussed
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Dussaule, Jean-Claude, Martin Flamant, and Christos Chatziantoniou. Function of the normal glomerulus. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0044_update_001.

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Glomerular filtration, the first step leading to the formation of primitive urine, is a passive phenomenon. The composition of this primitive urine is the consequence of the ultrafiltration of plasma depending on renal blood flow, on hydrostatic pressure of glomerular capillary, and on glomerular coefficient of ultrafiltration. Glomerular filtration rate (GFR) can be precisely measured by the calculation of the clearance of freely filtrated exogenous substances that are neither metabolized nor reabsorbed nor secreted by tubules: its mean value is 125 mL/min/1.73 m² in men and 110 mL/min/1.73 m
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Magder, Sheldon. Central venous pressure monitoring in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0132.

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Central venous pressure (CVP) is at the crucial intersection of the force returning blood to the heart and the force produced by cardiac function, which drives the blood back to the systemic circulation. The normal range of CVP is small so that before using it one must ensure proper measurement, specifically the reference level. A useful approach to hypotension is to first determine if arterial pressure is low because of a decrease in vascular resistance or a decrease in cardiac output. This is done by either measuring cardiac output or making a clinical assessment blood flow. If the cardiac o
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Capítulos de libros sobre el tema "Vascular resistance Measurement"

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L. Hungerford, Sara, Dhruv Nayya, Peter S. Hansen, Ravinay Bhindi, and Christopher Choong. "Perspective Chapter: Evolution of Techniques to Assess Vascular Impedance in Patients with Aortic Stenosis." In Aortic Stenosis - Recent Advances, New Perspectives and Applications [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104795.

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Aortic stenosis (AS) once was conceptualized as a mechanical problem with a fixed left ventricular (LV) afterload because of an obstructive valve. With time, there has been growing recognition that AS functions more like a series circuit, with important contributions from the ventricle through to the vasculature. Emerging evidence suggests that higher blood pressure and increased arterial stiffness, synonymous with vascular aging, increases global LV afterload in patients with AS. This in turn, has adverse consequences on quality-of-life measures and survival. Although traditional methods have emphasized measurement of the transvalvular pressure gradient, focusing on valvular hemodynamics alone may be inadequate. By definition, total vascular load of the human circulation includes both steady and pulsatile components. Steady load is best represented by the systemic vascular resistance whereas pulsatile load occurs because of wave reflections and vascular stiffness, and is often referred to as the valvulo-arterial impedance. In the following Review, we evaluate existing and upcoming methods to assess vascular load in patients with AS in order to better understand the effects of vascular aging on this insidious disease process.
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Heyndrickx, Guy, and Carlo Di Mario. "Haemodynamic data." In ESC CardioMed, 613–17. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0130.

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Direct catheter-based measurements of right and left heart saturations and pressures allow full characterization of patient haemodynamics, including the presence of cardiac shunts, valve gradients and valve areas, and pulmonary and vascular resistances.
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Actas de conferencias sobre el tema "Vascular resistance Measurement"

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Mori, Nobuhito, Yuya Morimoto, and Shoji Takeuchi. "Transendothelial electrical resistance (TEER) measurement system of 3D tubular vascular channel." In 2018 IEEE Micro Electro Mechanical Systems (MEMS). IEEE, 2018. http://dx.doi.org/10.1109/memsys.2018.8346551.

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Hunter, Kendall S., Craig J. Lanning, Joseph A. Albietz, Masahiko Oka, Karen A. Fagan, Kurt R. Stenmark, and Robin Shandas. "Measurement of In-Vivo Pulmonary Vascular Impedance in Two Animal Models of Pulmonary Hypertension." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175993.

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Pulmonary vascular input impedance has been increasingly promoted as an important diagnostic for pulmonary arterial hypertension (PAH) [1,2]. The gold-standard clinical diagnostic for the disease, pulmonary vascular resistance (PVR), quantifies only the mean resistance to flow but ignores the impact of vascular stiffness and flow pulsatility, which in PAH can represent up to 40% of the total load presented to the right ventricle. PVR has also been found to be only a moderate predictor of PAH outcomes [3]. The first of these deficiencies is not present in impedance; clinical studies have found
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Su, Zhenbi, Kendall Hunter, Wei Tan, and Robin Shandas. "Influence of Distal Resistance and Proximal Vascular Stiffness on Vascular Impedance and Hemodynamics in Pulmonary Hypertension: A Computational Study With Validation Using an Animal Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204759.

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Clinically, invasive measurement of pulmonary vascular flow and pressure provides the hemodynamic status of the pulmonary circulation with pulmonary arterial hypertension (PAH). Current diagnostics and therapeutics for PAH revolve around pulmonary vascular resistance (PVR), which is determined by the mean pressure divided by mean flow [1]. Though PVR correlates well with right ventricular (RV) afterload, failure of which is the primary determinant of mortality [2–4], PVR does not provide the complete measure of RV afterload since it neglects dynamic impedance effects [4, 5]. Although we have s
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Su, Zhenbi, Kendall Hunter, and Robin Shandas. "Effect of Vascular Stiffness on Pulmonary Flow in Normotensive and Hypertensive States: Numerical Study With Fluid Structure Interaction." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192831.

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Invasive measurement of pulmonary vascular flow and pressure provides the hemodynamic status of the pulmonary circulation for children with pulmonary arterial hypertension (PAH). Clinicians are primarily interested in pulmonary vascular resistance, which is the mean pressure of the circuit divided by the mean flow through it [1], in that it is believed to well-quantify the right ventricular (RV) afterload, the primary determinant of mortality. However, previous and current investigations on the pulmonary vascular stiffness (PVS), input impedance and RV power [2–4] have found PVS to be an impor
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Lee, Qim Y., Gregory S. H. Chan, Stephen J. Redmond, Paul M. Middleton, E. Steel, P. Malouf, C. Critoph, G. Flynn, E. O'Lone, and Nigel H. Lovell. "Classification of low systemic vascular resistance using photoplethysmogram and routine cardiovascular measurements." In 2010 32nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC 2010). IEEE, 2010. http://dx.doi.org/10.1109/iembs.2010.5628062.

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Reich, Alton, and Jason Heym. "Application of Model-Based Condition Monitoring to the Human Cardiovascular System." In ASME 2017 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/pvp2017-65500.

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The techniques applied by mechanical engineers to problems of machinery condition monitoring can also be applied in other fields. This paper discusses applying techniques that are regularly applied to machinery and system condition monitoring to the human cardiovascular system. Techniques such as physics-based system modeling coupled with limited measurements can be used to infer the condition of a system or specific component. In this case, a mathematical model of the flow through the cardiovascular system was implemented and can be used independently to simulate system performance. Given bas
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Ooi, Chen Yen, and Naomi C. Chesler. "The Role of Collagen in Pulmonary Hypertension-Induced Large Artery Stiffening." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192951.

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Hypoxic pulmonary hypertension (HPH) leads to stiffening of large pulmonary arteries, which affects right ventricular afterload. We hypothesized that vascular collagen accumulation is the major cause of large pulmonary artery (PA) stiffening in HPH. We tested this hypothesis with transgenic mice that produce collagen type I resistant to degradation (Col1a1R/R) and wild type littermate controls (Col1a1+/+) exposed to hypoxia and allowed to recover. Pressure-diameter testing on left PAs demonstrated that stiffness in control mice increased with hypoxia and decreased with recovery (p < 0.05).
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Burgett Graves, Shawna L., Craig Lanning, K. S. kirby, Michelle Calderbank, Jennifer Geppner, Shawn J. Nolan, Dunbar D. Ivy, Robin Shandas, and Kendall Hunter. "In-Vivo Pulmonary Vascular Stiffness Obtained From Color M-Mode Tissue Doppler Imaging And Pressure Measurements Predicts Clinical Outcomes Better Than Indexed Pulmonary Vascular Resistance In Pediatric Patients With Pulmonary Arterial Hypertension." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5748.

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Kato, Mitsuaki, Kenji Hirohata, Akira Kano, Shinya Higashi, Akihiro Goryu, Takuya Hongo, Shigeo Kaminaga, and Yasuko Fujisawa. "Fast CT-FFR Analysis Method for the Coronary Artery Based on 4D-CT Image Analysis and Structural and Fluid Analysis." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-51124.

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Non invasive fractional flow reserve derived from CT coronary angiography (CT-FFR) has to date been typically performed using the principles of computational fluid analysis in which a lumped parameter coronary vascular bed model is assigned to represent the impedance of the downstream coronary vascular networks absent in the computational domain for each coronary outlet. This approach may have a number of limitations. It may not account for the impact of the myocardial contraction and relaxation during the cardiac cycle, patient-specific boundary conditions for coronary artery outlets and vess
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