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Articles de revues sur le sujet « Anti-septic »

Auteur : Grafiati

Publié le 7 juin 2025

Mis à jour le 2 août 2025

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1

Ezzell, Carol. "(Anti-) Septic Strategies." Science News 142, no. 7 (1992): 104. http://dx.doi.org/10.2307/3976671.

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Wang, R., Q. Fang, L. Zhang, et al. "CD28 ligation prevents bacterial toxin-induced septic shock in mice by inducing IL-10 expression." Journal of Immunology 158, no. 6 (1997): 2856–61. http://dx.doi.org/10.4049/jimmunol.158.6.2856.

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Abstract The pathogenesis of septic shock is due mainly to bacterial toxin stimulation of the immune system, resulting in an excessive production of proinflammatory cytokines. TNF-alpha has been implicated as a major mediator in septic shock. Coinjection of D-galactosamine and LPS or staphylococcal enterotoxin B induced a rapid-onset, low-dose form of septic shock syndrome and ultimately led to death. We found that both the septic shock syndrome and death could be prevented by administration of anti-CD28 Ab. The protection induced by anti-CD28 Ab was associated with a decrease in TNF-alpha lev

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Shen, Yuchao, and Ying Cui. "Cholinergic anti-inflammatory pathway confers protective effects in septic shock rats." STEMedicine 5, no. 2 (2024): e191. http://dx.doi.org/10.37175/stemedicine.v5i2.191.

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Background: Septic shock can lead to multiple organ dysfunction. The cholinergic anti-inflammatory pathway is known to prevent organ damage from infection by modulating the inflammatory response. However, the modulatory capacity of cholinergic anti-inflammatory pathways in septic shock remains unclear. Methods: Cecal ligation and puncture (CLP) was used to establish a rat model of septic shock. Bilateral cervical vagal nerve isolation, dual cervical vagotomy, and vagus nerve stimulation (VNS) were used to inhibit or activate cholinergic anti-inflammatory pathways. Tacrine and α-bungarotoxin gr

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Yang, Sumin, Bong-Seon Lee, Changhun Lee, Sae-Kwang Ku, and Jong-Sup Bae. "Anti-Septic Effects of Cudratricusxanthone A Against HMGB1-Mediated Septic Responses." Natural Product Communications 14, no. 6 (2019): 1934578X1985679. http://dx.doi.org/10.1177/1934578x19856793.

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COHEN, J. "Anti-endotoxin immunotherapy in septic shock." Journal of Antimicrobial Chemotherapy 18, no. 4 (1986): 436–39. http://dx.doi.org/10.1093/jac/18.4.436.

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BAUMGARTNER, J. D., and M. P. GLAUSER. "Anti-endotoxin immunotherapy in septic shock." Journal of Antimicrobial Chemotherapy 19, no. 4 (1987): 551. http://dx.doi.org/10.1093/jac/19.4.551.

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Wu, Ji, Zhenhua Wang, Shanling Xu, et al. "Analysis of the role and mechanism of EGCG in septic cardiomyopathy based on network pharmacology." PeerJ 10 (March 9, 2022): e12994. http://dx.doi.org/10.7717/peerj.12994.

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Background Septic cardiomyopathy (SC) is a common complication of sepsis that leads to an increase in mortality. The pathogenesis of septic cardiomyopathy is unclear, and there is currently no effective treatment. EGCG (epigallocatechin gallate) is a polyphenol that has anti-inflammatory, antiapoptotic, and antioxidative stress effects. However, the role of EGCG in septic cardiomyopathy is unknown. Methods Network pharmacology was used to predict the potential targets and molecular mechanisms of EGCG in the treatment of septic cardiomyopathy, including the construction and analysis of protein-

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Gore, David, Lorena Preciado-Llanes, Gary Mills, Andrew Heath, and Robert Read. "In vitro hyporesponsiveness of CD4+ and CD8+ T cells in septic patients with faecal peritonitis (HUM8P.326)." Journal of Immunology 192, no. 1_Supplement (2014): 185.1. http://dx.doi.org/10.4049/jimmunol.192.supp.185.1.

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Abstract The immunological processes behind sepsis are ill defined. Predicting or diagnosing sepsis, based on cellular immune parameters is a research priority. This study quantified differences in peripheral blood mononuclear cell (PBMC) and cytokine levels between septic patients with fecal peritonitis, matched surgical patients (no sepsis) and healthy controls. To assess function, PBMC were stimulated in vitro with anti-CD3±anti-CD28 antibodies. Four days later T and B cell proliferation were measured by CFSE and activation by expression of CD25 and CD69 on T cells and CD86 on B cells. Viab

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Kim, Kyeong-Min, So-Yeon Kim, Tamanna Jahan Mony та ін. "Dracocephalum moldavica Ethanol Extract Suppresses LPS-Induced Inflammatory Responses through Inhibition of the JNK/ERK/NF-κB Signaling Pathway and IL-6 Production in RAW 264.7 Macrophages and in Endotoxic-Treated Mice". Nutrients 13, № 12 (2021): 4501. http://dx.doi.org/10.3390/nu13124501.

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The excessive synthesis of interleukin-6 (IL-6) is related to cytokine storm in COVID-19 patients. Moreover, blocking IL-6 has been suggested as a treatment strategy for inflammatory diseases such as sepsis. Sepsis is a severe systemic inflammatory response syndrome with high mortality. In the present study, we investigated the anti-inflammatory and anti-septic effects and the underlying mechanisms of Dracocephalum moldavica ethanol extract (DMEE) on lipopolysaccharide (LPS)-induced inflammatory stimulation in RAW 264.7 macrophages along with septic mouse models. We found that DMEE suppressed

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González, Celso, Ariadna Luna, Manuel Morales, Janet Sanchez, Claudia Guzman, and Juvenal Granillo. "Statin anti-inflammatory therapy in septic patients." Critical Care 12, Suppl 5 (2008): P26. http://dx.doi.org/10.1186/cc7059.

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Eichacker, P. Q., and C. Natanson. "The anti-inflammatory approach to septic shock." Intensivmedizin und Notfallmedizin 34, no. 2 (1997): 101–9. http://dx.doi.org/10.1007/s003900050026.

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Yan, Xue-Tao, Xiang-Hu He, Yan-Lin Wang, Zong-Ze Zhang, and Jun-Jiao Tang. "Transduced PEP-1-Heme Oxygenase-1 Fusion Protein Attenuates Lung Injury in Septic Shock Rats." Oxidative Medicine and Cellular Longevity 2018 (2018): 1–12. http://dx.doi.org/10.1155/2018/6403861.

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Oxidative stress and inflammation have been identified to play a vital role in the pathogenesis of lung injury induced by septic shock. Heme oxygenase-1 (HO-1), an effective antioxidant and anti-inflammatory and antiapoptotic substance, has been used for the treatment of heart, lung, and liver diseases. Thus, we postulated that administration of exogenous HO-1 protein transduced by cell-penetrating peptide PEP-1 has a protective role against septic shock-induced lung injury. Septic shock produced by cecal ligation and puncture caused severe lung damage, manifested in the increase in the lung w

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Guo, Yin, Liming Luan, Julia Bohannon, et al. "The lack of NK and mCD8+ T cells in IL-15KO mice confers resistance to septic shock." Journal of Immunology 196, no. 1_Supplement (2016): 60.9. http://dx.doi.org/10.4049/jimmunol.196.supp.60.9.

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Abstract Septic shock remains one of the leading causes of death in the USA. However, the immunological mechanisms of septic shock are still not well understood. Interleukin (IL)-15 is an essential mediator for natural killer (NK) and memory (m) CD8+T cell survival, as indicated by the deficits of these cells in IL-15 knockout (KO) mice. As NK and CD8+T cells are implicated in the pathogenesis of septic shock, we hypothesized that IL-15KO mice will be resistant to septic shock due to their lack of NK and mCD8+T cells. IL-15KO mice showed a significant survival advantage over wild type (WT) con

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Andreu-Ballester, Juan Carlos, Amparo Navarro, Miguel Angel Arribas, et al. "Increased Levels of Anti-Anisakis Antibodies During Hospital Admission in Septic Patients." Antibodies 13, no. 4 (2024): 96. http://dx.doi.org/10.3390/antib13040096.

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Background/Objectives: In a previous study, we described elevated anti-Anisakis IgG levels in septic patients in relation to disease severity. In this study, our objective was to analyze the evolution of anti-Anisakis immunoglobulins in septic patients during hospital admission and their association with αβ and γδ T cell subsets. Methods: We recruited 80 subjects: 40 patients with sepsis and 40 controls. αβ and γδ T cells were analyzed using flow cytometry. Apoptosis was also assessed, and anti-Anisakis antibodies were measured by ELISA in the sera of patients with sepsis and controls. Results

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&NA;. "Anti-TNF?? mAb has potential in septic shock." Inpharma Weekly &NA;, no. 980 (1995): 11. http://dx.doi.org/10.2165/00128413-199509800-00018.

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Sfeir, Tacla, Dhanonjoy Saha, Mark Astiz, and Eric Rackow. "PERSISTENT ANTI-INFLAMMATORY CYTOKINE RELEASE IN SEPTIC SHOCK." Critical Care Medicine 27, Supplement (1999): 48A. http://dx.doi.org/10.1097/00003246-199901001-00075.

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NAPOLI, DENISE. "Septic Arthritis Risk Raised With Anti-TNF Use." Internal Medicine News 42, no. 20 (2009): 42–43. http://dx.doi.org/10.1016/s1097-8690(09)70832-7.

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Freeman, Bradley D., and Charles Natanson. "Anti-inflammatory therapies in sepsis and septic shock." Expert Opinion on Investigational Drugs 9, no. 7 (2000): 1651–63. http://dx.doi.org/10.1517/13543784.9.7.1651.

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NAPOLI, DENISE. "Septic Arthritis Rates Rose With Anti-TNF Therapy." Rheumatology News 8, no. 11 (2009): 21. https://doi.org/10.1016/s1541-9800(09)70359-x.

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Chen, Kai-Lee, Ruey-Hsing Chou, Chun-Chin Chang, et al. "The high-density lipoprotein cholesterol (HDL-C)-concentration-dependent association between anti-inflammatory capacity and sepsis: A single-center cross-sectional study." PLOS ONE 19, no. 4 (2024): e0296863. http://dx.doi.org/10.1371/journal.pone.0296863.

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Introduction Known to have pleiotropic functions, high-density lipoprotein (HDL) helps to regulate systemic inflammation during sepsis. As preserving HDL-C level is a promising therapeutic strategy for sepsis, the interaction between HDL and sepsis worth further investigation. This study aimed to determine the impact of sepsis on HDL’s anti-inflammatory capacity and explore its correlations with disease severity and laboratory parameters. Methods and materials We enrolled 80 septic subjects admitted to the intensive care unit and 50 controls admitted for scheduled coronary angiography in this

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TIGHE, D., R. MOSS та D. BENNETT. "Porcine hepatic response to sepsis and its amplification by an adrenergic receptor α1 agonist and a β2 antagonist". Clinical Science 95, № 4 (1998): 467–78. http://dx.doi.org/10.1042/cs0950467.

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1.We investigated the effect of adrenergic receptor stimulation or inhibition on the hepatic ultrastructural changes in a porcine faecal peritonitis model of multi-organ failure. We infused either the α1 adrenergic receptor agonist methoxamine or the β2 adrenergic receptor antagonist ICI 118551 during 8 ;h of the study. 2.Anaesthetized pigs (25–30 ;kg) were divided into four non-septic groups (control, non-septic, non-septic methoxamine and non-septic ICI 118551) and three septic groups (septic, septic methoxamine and septic ICI 118551). 3.Changes in hepatic ultrastructure were measured by mor

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Sanghi, ShailBala. "ALOE VERA: A MEDICINAL HERB." International Journal of Research -GRANTHAALAYAH 3, no. 11 (2015): 32–34. http://dx.doi.org/10.29121/granthaalayah.v3.i11.2015.2913.

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Aloe vera is a herb distributed all over the world. The herb is used internally to counter most of the problems of digestion, diabetes, immune system, and peptic ulcers. It also contains anti-bacterial, anti-septic, anti-viral, anti-inflammatory, anti-tumor, anti -oxidant, moisturizing, anti-aging effects. This review highlights the importance of using Aloe vera as a medicinal herb.

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Bukhari, Hassan, and Anand Kumar. "Antiplatelet administration Improves Outcome in Patient with Septic Shock Secondary to Ischemic Bowel." Teikyo Medical Journal 44, no. 5 (2021): 1697–705. https://doi.org/10.5281/zenodo.7779079.

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  Ischemic bowel disease is a life-threatening condition, and mortality will worsen if the patient presented in septic shock. Several factors affect the outcome of this disease. Recent data suggests, however, that patients who develop shock secondary to ischemic bowel and have a history of taking anti-platelet medications within two weeks of presentation may have a higher chance of survival when compared to a patient who did not. The study aims to determine the impaction of prophylactic anti-platelet (Aspirin and clopidogrel) administration in improving outcomes among patients who present

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Kim, Jung-Yeon, Hyo-Lim Hong, Gyun Moo Kim, Jaechan Leem, and Hyun Hee Kwon. "Protective Effects of Carnosic Acid on Lipopolysaccharide-Induced Acute Kidney Injury in Mice." Molecules 26, no. 24 (2021): 7589. http://dx.doi.org/10.3390/molecules26247589.

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Septic acute kidney injury (AKI) is an important medical problem worldwide, but current treatments are limited. During sepsis, lipopolysaccharide (LPS) activates various signaling pathways involved in multiorgan failure. Carnosic acid is a natural phenolic diterpene and has multiple bioactivities, such as anti-tumor, anti-inflammatory, and anti-oxidative effects. However, the effect of carnosic acid on septic AKI has not been explored. Therefore, this study aimed to determine whether carnosic acid has a therapeutic effect on LPS-induced kidney injury. Administration of carnosic acid after LPS

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Dr., Shail Bala Sanghi. "ALOE VERA: A MEDICINAL HERB." International Journal of Research – Granthaalayah 3, no. 11 (2017): 32–34. https://doi.org/10.5281/zenodo.848987.

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Aloe vera is a herb distributed all over the world. The herb is used internally to counter most of the problems of digestion, diabetes, immune system, and peptic ulcers. It also contains antibacterial, anti-septic, anti-viral, anti-inflammatory, anti-tumor, anti -oxidant, moisturizing, anti-aging effects. This review highlights the importance of using Aloe vera as a medicinal herb.

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Bian, Jinghao, Xiaocheng Wei, Peizhen Chen, et al. "Effectiveness and Risk Assessment Septic Tank Treatment Systems in Rural Cold Regions of China." Processes 12, no. 11 (2024): 2385. http://dx.doi.org/10.3390/pr12112385.

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Rural toilet reform in cold areas is the focus and difficulty of China’s toilet revolution. In this paper, by carrying out the septic tank gradient burial depth test and regular seasonal monitoring of the operation effect of rural septic tanks in cold areas, the frost protection effect and treatment effect of rural toilets in cold areas are clarified, which provide important references for the rural toilet revolution and its practical significance in cold areas of China. Based on controlled tests and correlation analysis methods, different gradient burial depths of septic tanks (40, 60, and 80

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Voss, Oliver H., Konrad Krzewski, and John E. Coligan. "Lipopolysaccharide-induced CD300b receptor binding to Toll-like Receptor 4 alters signaling to drive cytokine responses that enhance septic shock." Journal of Immunology 198, no. 1_Supplement (2017): 75.12. http://dx.doi.org/10.4049/jimmunol.198.supp.75.12.

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Abstract LPS, present in gram-negative bacteria membranes, causes strong immune responses following detection by TLR4 on immune cells. Excess immune cell activation resulting from severe infection initiates a pro-inflammatory cytokine storm that leads to a more severe immunopathology, like septic shock, and subsequently, death. Intriguingly, Cd300b−/− mice were observed to be more resistant to septic shock than WT mice, however the mechanism responsible for this phenomenon remained elusive. Here, we identify LPS as a ligand for CD300b, and demonstrate that CD300b-expressing macrophages (Mφ) ar

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&NA;. "Anti-E-selectin antibody shows promise in septic shock." Inpharma Weekly &NA;, no. 1027 (1996): 8. http://dx.doi.org/10.2165/00128413-199610270-00012.

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Zeni, Fabrice, Bradley Freeman, and Charles Natanson. "Anti-inflammatory therapies to treat sepsis and septic shock." Critical Care Medicine 25, no. 7 (1997): 1095–100. http://dx.doi.org/10.1097/00003246-199707000-00001.

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Lepanto, Maria Stefania, Luigi Rosa, Rosalba Paesano, Piera Valenti, and Antimo Cutone. "Lactoferrin in Aseptic and Septic Inflammation." Molecules 24, no. 7 (2019): 1323. http://dx.doi.org/10.3390/molecules24071323.

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Lactoferrin (Lf), a cationic glycoprotein able to chelate two ferric irons per molecule, is synthesized by exocrine glands and neutrophils. Since the first anti-microbial function attributed to Lf, several activities have been discovered, including the relevant anti-inflammatory one, especially associated to the down-regulation of pro-inflammatory cytokines, as IL-6. As high levels of IL-6 are involved in iron homeostasis disorders, Lf is emerging as a potent regulator of iron and inflammatory homeostasis. Here, the role of Lf against aseptic and septic inflammation has been reviewed. In parti

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Xu, Qigang, Junjian Li, Zhe Chen, Yefan Mao та Chonglin Tao. "Protective effects of β-eudesmol against septic liver injury via inhibition of NF-κB signaling". Tropical Journal of Pharmaceutical Research 21, № 6 (2022): 1183–88. http://dx.doi.org/10.4314/tjpr.v21i6.7.

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Purpose: To investigate the role of β-eudesmol in septic liver injury in mice.Methods: Mice were intraperitoneally injected with 50 or 100 mg/kg β-eudesmol, and then subjected to cecal ligation and puncture for the establishment of a septic model 2 h later. Haematoxylin and eosin staining was used to evaluate histopathological changes in the liver tissues. Terminal deoxynucleotidyl transferase (TdT) dUTP Nick-End Labeling (TUNEL) staining and enzyme-linked immunosorbent assay (ELISA) were employed to determine liver damage, while inflammation and oxidative stress were evaluated by ELISA.Result

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Moriyama, Naoki, Masafumi Saito, Yuko Ono, Kimihiro Yamashita, Takashi Aoi та Joji Kotani. "Increased Interleukin-17-Producing γδT Cells in the Brain Exacerbate the Pathogenesis of Sepsis-Associated Encephalopathy and Sepsis-Induced Anxiety in Mice". Journal of Clinical Medicine 12, № 13 (2023): 4309. http://dx.doi.org/10.3390/jcm12134309.

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Overactivated microglia play a key role in sepsis-associated encephalopathy (SAE), although the involvement of T cells is unclear. γδT cells in the brain and meninges regulate normal fear responses via interleukin (IL)-17 in healthy mice. In our sepsis model, the mice showed exacerbated anxious behavior at 10 days post-induction (dpi). At 8 dpi, IL-17 mRNA was significantly upregulated in the brains of septic mice compared with those of control mice. Simultaneously, the number of γδT cells increased in the brains of septic mice in a severity-dependent manner. Additionally, IL-17-producing γδT

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Li, Jing, Ruijing Xuan, Weidong Wu, Yang Han, Jiani Guo, and Meixia Yang. "CTRP6 suppresses neutrophil extracellular traps formation to ameliorate sepsis-induced lung injury through inactivation of ERK pathway." Allergologia et Immunopathologia 50, no. 6 (2022): 53–59. http://dx.doi.org/10.15586/aei.v50i6.677.

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Background: Septic lung injury is associated with excessive neutrophil activation, while neu-trophil extracellular traps formation contributes to inflammatory lung injury in sepsis. C1q/ tumor necrosis factor–related protein-6 (CTRP6) is a paralog of adiponectin and exerts anti-inflammatory and antioxidant properties. The role of CTRP6 in sepsis-associated inflammatory lung injury was investigated in this study. Methods: Mice were injected with lipopolysaccharides (LPS) intraperitoneally to establish the mouse sepsis model. They were first tail-vein injected with adenovirus-mediated overexpres

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Nakamura, Tsukasa, Eiichi Sato, Nobuharu Fujiwara, et al. "Statins and hemoperfusion improve 28-day survival in septic shock patients." Open Medicine 7, no. 4 (2012): 475–80. http://dx.doi.org/10.2478/s11536-011-0169-z.

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AbstractUncontrolled inflammation and endotoxin play a central role in septic shock. Statins may possess anti-inflammatory properties, and removal of endotoxin by hemoperfusion with polymyxin B-immobilized fiber (PMX-F) could have favorable effects on sepsis. We examined retrospectively whether pre-existing statin and hemoperfusion with PMX-F at the time of admission were separately and independently associated with decreased overall 28-day mortality in septic shock patients. Consecutive 173 patients with septic shock (71.2±10.7 years old, 115 male and 58 female) were included in the present s

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Mierzchała-Pasierb, Magdalena, Małgorzata Lipińska-Gediga, Łukasz Lewandowski, and Małgorzata Krzystek-Korpacka. "Alterations in Serum Concentration of Soluble CD163 within Five Study Days from ICU Admission Are Associated with In-Hospital Mortality of Septic Patients—A Preliminary Study." International Journal of Environmental Research and Public Health 20, no. 3 (2023): 2263. http://dx.doi.org/10.3390/ijerph20032263.

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Background: CD163, a cell membrane surface molecule specifically expressed by macrophages with an anti-inflammatory phenotype, participates in innate immunity. The purpose of the study was to evaluate the clinical utility of sCD163 in septic patients in comparison to other parameters associated with infections, mainly PCT, CRP and IL-18. Methods: Serum samples were obtained from 40 septic patients on the ICU admission day, 3rd and 5th study days. The control group consisted of 30 healthy volunteers from whom the specimen was collected once. An enzyme-linked immunosorbent assay (ELISA) was used

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Servillo, Giuseppe, Maria Vargas, Antonio Pastore, et al. "Immunomodulatory Effect of Continuous Venovenous Hemofiltration during Sepsis: Preliminary Data." BioMed Research International 2013 (2013): 1–6. http://dx.doi.org/10.1155/2013/108951.

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Introduction. Severe sepsis and septic shock are the primary causes of multiple organ dysfunction syndrome (MODS), which is the most frequent cause of death in intensive care unit patients. Many pro- and anti-inflammatory mediators, such as interleukin-6 (IL-6), play a strategic role in septic syndrome. Continuous renal replacement therapy (CRRT) removes in a nonselective way pro- and anti-inflammatory mediators.Objective. To investigate the effects of continuous venovenous hemofiltration (CVVH) as an immunomodulatory treatment of sepsis in a prospective clinical study.Methods. High flux hemof

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Marseglia, Lucia, Russel Reiter, Giuseppe Buonocore, Eloisa Gitto, and Gabriella D'Angelo. "Melatonin and Neonatal Sepsis: A Promising Antioxidant Adjuvant Agent." American Journal of Perinatology 34, no. 14 (2017): 1382–88. http://dx.doi.org/10.1055/s-0037-1604244.

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AbstractSepsis represents a major clinical problem in neonatal setting with elevated mortality rate related to multiple organ failure. Despite decades of research, the exact mechanism of organ failure in sepsis is still not completely understood. Oxidative stress (OS), derived from an imbalance between pro-oxidant and antioxidant factors, is involved in the pathogenesis of several neonatal diseases, including sepsis, and plays a particular role in systemic organ failure. Recently, it has been recognized that administration of antioxidants could be useful in septic patients. Among all antioxida

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Kook, Minsoo, Sung Kyun Lee, Sang Doo Kim та ін. "Anti-septic activity of α-cubebenoate isolated from Schisandra chinensis". BMB Reports 48, № 6 (2015): 336–41. http://dx.doi.org/10.5483/bmbrep.2015.48.6.202.

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Jung, Byeongjin, Hyejin Kang, Wonhwa Lee, et al. "Anti-septic effects of dabrafenib on HMGB1-mediated inflammatory responses." BMB Reports 49, no. 4 (2016): 214–19. http://dx.doi.org/10.5483/bmbrep.2016.49.4.220.

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Antonelli, M. "Sepsis and Septic Shock: Pro-Inflammatory or Anti-Inflammatory State?" Journal of Chemotherapy 11, no. 6 (1999): 536–40. http://dx.doi.org/10.1179/joc.1999.11.6.536.

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Zhou, Xiang, Long-Xiang Su, Jia-Hui Zhang, Da-Wei Liu, and Yun Long. "Rules of anti-infection therapy for sepsis and septic shock." Chinese Medical Journal 132, no. 5 (2019): 589–96. http://dx.doi.org/10.1097/cm9.0000000000000101.

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Hangen, Donald H., John H. Stevens, Paul S. Satoh, Elaine W. Hall, Peter T. O'Hanley, and Thomas A. Raffin. "Complement levels in septic primates treated with anti-C5a antibodies." Journal of Surgical Research 46, no. 3 (1989): 195–99. http://dx.doi.org/10.1016/0022-4804(89)90055-3.

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Yoo, Hayoung, Sae-Kwang Ku, Min-Su Han, Kyung-Min Kim, and Jong-Sup Bae. "Anti-septic Effects of Fisetin In Vitro and In Vivo." Inflammation 37, no. 5 (2014): 1560–74. http://dx.doi.org/10.1007/s10753-014-9883-4.

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Al-Obaidy, Abdulameer Kareem Leelo, Saafa Ressan Abdullah, Rana Saleh Al-Difaie, and Hawraa Abdulameer. "Immunological Aspect of Septic Shock." International Journal of Research and Scientific Innovation XI, no. XII (2025): 560–74. https://doi.org/10.51244/ijrsi.2024.11120051.

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Septic shock is clinically characterized by hypotension necessitating vasoactive support that fails to react to fluid resuscitation in patients with suspected or microbiologically confirmed infections leading to severe organ failure without an identified etiology. Predisposing factors for septic shock arises when the body’s reaction to an infection impairs tissue perfusion and oxygen utilization, resulting in end-organ malfunction and heightened mortality risk. The etiologies of septic shock differ between adults and children. The mechanisms contributing to the predisposition for severe sepsis

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van der Poll, T., A. Marchant, W. A. Buurman, et al. "Endogenous IL-10 protects mice from death during septic peritonitis." Journal of Immunology 155, no. 11 (1995): 5397–401. http://dx.doi.org/10.4049/jimmunol.155.11.5397.

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Abstract IL-10 production during endotoxic shock is part of a protective mechanism that involves IL-10-induced inhibition of TNF synthesis. We sought to determine the role of IL-10 in septic peritonitis induced by cecal ligation and puncture (CLP). CLP led to a rapid induction of IL-10 mRNA in various organs of C57BI/6 mice. In liver, IL-10 mRNA was detectable within 1 h following CLP, while in spleen and lungs, IL-10 mRNA was detected from 2 to 4 h and onward. IL-10 protein became detectable in plasma 2 h after CLP, reaching peak concentrations after 12 h (12.7 +/- 5.7 ng/ml). Pretreatment (-

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Lvov, I. M. "On the treatment of septic postpartum disease with Marmoreck's anti-chain serum. (From the Kazan Provincial Zemsky Hospital.)." Journal of obstetrics and women's diseases 11, no. 12 (2020): 1643–47. http://dx.doi.org/10.17816/jowd11121643-1647.

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In view of the conflicting data in the literature on the therapeutic value of anti-chain serum in the treatment of septic postpartum diseases, on the one hand, and in view of the statement of the Kiev Obstetric and Gynecological Society that as many cases of treatment of postpartum diseases with this serum as possible should be published, on the other hand, I will allow Here are two cases of treatment of severe postpartum diseases of a septic nature with this serum.

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Huang, Ruizhi, Xiaoxiao He, Qiuqing Jin, Jingyan Huang, and Jialin Ye. "Emergency Nursing Care for a Pediatric Patient with Severe Scrub Typhus Complicated by Hemophagocytic Syndrome and Septic Shock." Journal of Clinical and Nursing Research 9, no. 6 (2025): 67–72. https://doi.org/10.26689/jcnr.v9i6.10987.

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This article summarizes the nursing management of a pediatric patient with severe scrub typhus complicated by hemophagocytic syndrome (HPS) and septic shock. Key nursing interventions included early recognition and management of septic shock, rational oxygen therapy, fluid resuscitation, administration of vasoactive agents, early and targeted anti-infective therapy, management of hyperpyrexia, care of eschar and edematous skin, strict infection prevention and isolation protocols, and family health education. After 11 days of intensive treatment and nursing care, the patient recovered and was d

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Pugin, Jérôme, Sabine Stern-Voeffray, Bruno Daubeuf, Michael A. Matthay, Greg Elson, and Irène Dunn-Siegrist. "Soluble MD-2 activity in plasma from patients with severe sepsis and septic shock." Blood 104, no. 13 (2004): 4071–79. http://dx.doi.org/10.1182/blood-2003-04-1290.

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Abstract In this paper, we show that plasma from patients with severe sepsis and septic shock but not normal plasma supports lipopolysaccharide (LPS) activation of epithelial cells expressing Toll-like receptor 4 (TLR4). Recombinant soluble myeloid differentiation protein-2 (MD-2) complemented normal plasma and allowed LPS activation of epithelial cells to levels measured with “septic” plasma, whereas soluble MD-2-depleted plasma lost its effects. The same “MD-2 activity” was found in urine from a patient with septic shock and in lung edema fluids from patients with adult respiratory distress

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Li, Jiang, Jeffrey Bonenfant, Luqman Nasouf, et al. "Resistin immunomodulation in healthy and septic patients." Journal of Immunology 202, no. 1_Supplement (2019): 120.22. http://dx.doi.org/10.4049/jimmunol.202.supp.120.22.

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Abstract Sepsis is a major healthcare challenge worldwide with no effective therapies. Sepsis pathogenesis involves immune dysregulation, however, the underlying mechanisms of inflammation that are protective or pathogenic in sepsis remain unclear. Human resistin is an immune cell-derived protein that is elevated in sepsis. Employing human resistin-expressing transgenic mouse we identified a protective role for resistin in preventing fatal LPS endotoxic shock. Resistin binds to the LPS receptor, Toll-like receptor 4, through its N-terminal helix and triggers a switch from pro-inflammatory to a

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Lee, In-Chul, and Jong-Sup Bae. "Suppressive Effects of Sulforaphane on TGFBIp-mediated Sepsis." Natural Product Communications 12, no. 10 (2017): 1934578X1701201. http://dx.doi.org/10.1177/1934578x1701201026.

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Sulforaphane (SFN) is produced when the enzyme myrosinase transforms glucoraphanin upon damage to the plant such as from chewing and effective in preventing carcinogenesis, diabetes, and inflammatory responses. Transforming growth factor β-induced protein (TGFBIp) is an extracellular matrix protein whose expression in several cell types is greatly increased by TGF-β. TGFBIp is released by human umbilical vein endothelial cells (HUVECs) and functions as a mediator of experimental sepsis. We hypothesized that SFN could reduce TGFBIp-mediated severe inflammatory responses in human endothelial cel

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