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Littérature scientifique sur le sujet « Bone resorption »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 25 juillet 2025

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Articles de revues sur le sujet "Bone resorption"

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Carron, CP, DM Meyer, VW Engleman, et al. "Peptidomimetic antagonists of alphavbeta3 inhibit bone resorption by inhibiting osteoclast bone resorptive activity, not osteoclast adhesion to bone." Journal of Endocrinology 165, no. 3 (2000): 587–98. http://dx.doi.org/10.1677/joe.0.1650587.

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Osteoclasts are actively motile on bone surfaces and undergo alternating cycles of migration and resorption. Osteoclast interaction with the extracellular matrix plays a key role in the osteoclast resorptive process and a substantial body of evidence suggests that integrin receptors are important in osteoclast function. These integrin receptors bind to the Arg-Gly-Asp (RGD) sequence found in a variety of extracellular matrix proteins and it is well established that the interaction of osteoclast alpha v beta 3 integrin with the RGD motif within bone matrix proteins is important in osteoclast-me

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Flatten, Jana, Thomasz Gedrange, Christoph Bourauel, Ludger Keilig, and Anna Konermann. "The Role of Bone and Root Resorption on the Biomechanical Behavior of Mandibular Anterior Teeth Subjected to Orthodontic Forces: A Finite Element Approach." Biomedicines 12, no. 9 (2024): 1959. http://dx.doi.org/10.3390/biomedicines12091959.

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Aims: This study was conducted to systematically evaluate the biomechanical impact of varying degrees of root and bone resorption resulting from periodontitis and orthodontic tooth movement (OTM) on the mandibular anterior teeth. The objective was to determine whether these distinct resorption patterns exert a specific influence on tooth displacement and strain patterns. Methods: A finite element (FE) model of an idealized anterior mandible from the first premolar in the third to the fourth quadrant was developed without bone or root resorption and a constant periodontal ligament (PDL) thickne

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Chu, Pei-Wen, Yu-Hsu Chen, Chien-Hui Chen, and Shau-Kwaun Chen. "Inflammatory environments disrupt both bone formation and bone resorption." Journal of Immunology 204, no. 1_Supplement (2020): 224.46. http://dx.doi.org/10.4049/jimmunol.204.supp.224.46.

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Abstract Inflammation has been associated with bone diseases such as osteoporosis and osteoarthritis. Bone loss were reported in the patients of several inflammatory diseases, such as rheumatoid arthritis, systemic lupus erythematosus, inflammatory bowel disease. However, how inflammation influence bone metabolism remains elusive. The bone loss in inflammatory environments are widely considered as the results of osteoclast overactivation which leads to excessive bone resorption. We previously discovered that osteoclasts induced from RAW macrophage treated with RANKL exhibited different cell pr

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Towhidul Alam, A. S. M., Christopher L. H. Huang, David R. Blake, and Mone Zaidi. "A hypothesis for the local control of osteoclast function by Ca2+, nitric oxide and free radicals." Bioscience Reports 12, no. 5 (1992): 369–80. http://dx.doi.org/10.1007/bf01121500.

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Several important conclusions have recently emerged from in vitro studies on the resorptive cell of bone, the osteoclast. First, it has been established that osteoclast function is modulated locally, by changes in the local concentration of Ca2+ caused by hydroxyapatite dissolution. It is thought that activation by Ca2+ of a surface membrane Ca2+ receptor mediates these effects, hence providing a feedback control. Second, a number of molecules produced locally by the endothelial cell, with which the osteoclast is in intimate contact, have been found to affect bone resorption profoundly. For in

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Borggaard, Xenia G., Dinisha C. Pirapaharan, Jean-Marie Delaissé, and Kent Søe. "Osteoclasts’ Ability to Generate Trenches Rather Than Pits Depends on High Levels of Active Cathepsin K and Efficient Clearance of Resorption Products." International Journal of Molecular Sciences 21, no. 16 (2020): 5924. http://dx.doi.org/10.3390/ijms21165924.

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Until recently, it was well-accepted that osteoclasts resorb bone according to the resorption cycle model. This model is based on the assumption that osteoclasts are immobile during bone erosion, allowing the actin ring to be firmly attached and thereby provide an effective seal encircling the resorptive compartment. However, through time-lapse, it was recently documented that osteoclasts making elongated resorption cavities and trenches move across the bone surface while efficiently resorbing bone. However, it was also shown that osteoclasts making rounded cavities and pits indeed resorb bone

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Datta, Harish K., Iain MacIntyre, and Mone Zaidi. "The effect of extracellular calcium elevation on morphology and function of isolated rat osteoclasts." Bioscience Reports 9, no. 6 (1989): 747–51. http://dx.doi.org/10.1007/bf01114813.

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Osteoclasts are large multinucleate cells unique in their capacity to resorb bone. These cells are exposed locally to high levels of ionised calcium during the process of resorption. We have therefore examined the effect of elevated extracellular calcium on the morphology and function of freshly disaggregated rat osteoclasts. Cell size and motility were quantitated by time-lapse video recording together with digitisation and computer-centred image analysis. In order to assess the resorptive capacity of isolated osteoclasts, we measured the total area of resorption of devitalised cortical bone

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Fuller, Karen, Barrie Kirstein, and Timothy J. Chambers. "Regulation and enzymatic basis of bone resorption by human osteoclasts." Clinical Science 112, no. 11 (2007): 567–75. http://dx.doi.org/10.1042/cs20060274.

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Although much has been learned recently of the mechanisms that regulate osteoclastic differentiation, much less is known of the means through which their resorptive activity is controlled. This is especially so for human osteoclasts. We have recently developed an assay that allows us to measure resorptive activity while minimizing confounding effects on differentiation by optimizing osteoclastogenesis, so that measurable resorption occurs over a short period, and by relating resorption in each culture during the test period to the resorption that had occurred in the same culture in a prior con

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Feng, Shi, Zhiyong Zhang, Lei Shi, et al. "Temporal Bone Resorption." Journal of Craniofacial Surgery 26, no. 2 (2015): e185-e187. http://dx.doi.org/10.1097/scs.0000000000001452.

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Taguchi, Takafumi, and Yoshio Terada. "Subperiosteal Bone Resorption." New England Journal of Medicine 370, no. 21 (2014): e32. http://dx.doi.org/10.1056/nejmicm1308814.

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Aspenberg, P., and P. Herbertsson. "PERIPROSTHETIC BONE RESORPTION." Journal of Bone and Joint Surgery. British volume 78-B, no. 4 (1996): 641–46. http://dx.doi.org/10.1302/0301-620x.78b4.0780641.

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Thèses sur le sujet "Bone resorption"

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Pierce, Angela Mary. "Cellular mechanisms in bone and tooth resorption morphological studies in rats and monkeys /." Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1988. http://books.google.com/books?id=usBpAAAAMAAJ.

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Heath, J. K. "Studies on cellular interactions in bone resorption." Thesis, Anglia Ruskin University, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.354876.

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McCauley, Laurie Kay. "Cellular mechanisms of lymphocyte-mediated bone resorption /." The Ohio State University, 1991. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487759055156174.

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Stutzer, Andre. "Retinoid induced bone resorption, model and application /." [S.l.] : [s.n.], 1987. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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Ransjö, Maria. "Regulation of bone resorption by the adenylate cyclase-cyclic AMP system a biochemical study on mouse calvarial bones and isolated bone cells /." Umeå, Sweden : University of Umeå, 1988. http://catalog.hathitrust.org/api/volumes/oclc/18171035.html.

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Bernhold, Brechter Anna. "Kinins : important regulators in inflammation induced bone resorption." Doctoral thesis, Umeå : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-959.

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Moroz, Adam. "Reduced order modelling of bone resorption and formation." Thesis, De Montfort University, 2011. http://hdl.handle.net/2086/5409.

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The bone remodelling process, performed by the Bone Multicellular Unit (BMU) is a key multi-hierarchically regulated process, which provides and supports various functionality of bone tissue. It is also plays a critical role in bone disorders, as well as bone tissue healing following damage. Improved modelling of bone turnover processes could play a significant role in helping to understand the underlying cause of bone disorders and thus develop more effective treatment methods. Moreover, despite extensive research in the field of bone tissue engineering, bonescaffold development is still very

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Ljunggren, Östen. "Involvement of bradykinin in inflammation induced bone resorption." Umeå : University of Umeå, 1991. http://catalog.hathitrust.org/api/volumes/oclc/24493228.html.

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Korhonen, T. (Tommi). "Bone flap survival and resorption after autologous cranioplasty." Doctoral thesis, Oulun yliopisto, 2019. http://urn.fi/urn:isbn:9789526222530.

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Abstract This thesis evaluated the factors affecting bone flap survival and bone flap resorption after primary cranial reparation surgery, cranioplasty, conducted to repair a cranial bone defect with an autologous cryopreserved bone flap. Emphasis is put on the predictors, progression, and definition of an important yet poorly understood postoperative complication, bone flap resorption. Study I assessed the rates and predictors of bone flap removal and bone flap resorption in a Finnish retrospective multicentre setting. 40% of patients developed complications of whom half required bone flap re

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Neale, Susan Dorothy. "The role of macrophages in pathological bone resorption /." Title page, table of contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09MSM/09msmn348.pdf.

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Livres sur le sujet "Bone resorption"

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Bronner, Felix, Mary C. Farach-Carson, and Janet Rubin, eds. Bone Resorption. Springer-Verlag, 2005. http://dx.doi.org/10.1007/b136184.

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Felix, Bronner, Farach-Carson Mary C. 1958-, and Rubin Janet, eds. Bone resorption. Springer, 2005.

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Harvey, Wilson. Prostaglandins in bone resorption. CRC Press, 1988.

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Pierce, Angela Mary. Cellular mechanisms in bone and tooth resorption: Morphological studies in rats and monkeys. Kongl. Carolinska Medico Chirurgiska Institutet, 1988.

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Brown, R. James. Histological and compositional responses of bone to immobilization and other experimental conditions: Semi-annual report. Institute of Chemical Biology, University of San Francisco, 1985.

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Thamsborg, Gorm. Effect of nasal salmon calcitonin on calcium and bone metabolism. Lægeforeningens Forlag, 1999.

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Maria, Bijvoet Olav Leonardus, ed. Bisphosphonate on bones. Elsevier, 1995.

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Arun, Shanbhag, Rubash Harry E, and Jacobs Joshua J. 1956-, eds. Joint replacement and bone resorption: Pathology, biomaterials, and clinical practice. Taylor & Francis, 2005.

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Åkesson, Louise. Panoramic radiography in the assessment of the marginal bone level. Department of Oral Radiology, Faculty of Odontology, Lund University, 1991.

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Ljunggren, Östen. Experimental studies on bone resorption: Effects of parathyroid hormone and vitamin D. Univ., 1993.

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Chapitres de livres sur le sujet "Bone resorption"

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Griffon, Dominique. "Bone Resorption." In Complications in Small Animal Surgery. John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781119421344.ch97.

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Alcin, Adem, and Halil Lutfi Canat. "Bone Health and Bone Targeted Therapies for Metastatic Prostate Cancer." In Current Management of Metastatic Prostate Cancer. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359142.15.

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This section provides information about bone health and bone-targeted therapies in metastatic prostate cancer. The most common site of metastasis for prostate cancer is the bone. Treatments used in cancer management can also have adverse effects on bones. Bone-related conditions in metastatic prostate cancer are one of the most important causes of morbidity and mortality. In bone-targeting treatments, resorption modulators and radiotherapy are the main components. Radionuclide agents are innovative treatments for targeting bones.

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Kumar, Vinay V., Supriya Ebenezer, and Andreas Thor. "Bone Augmentation Procedures in Implantology." In Oral and Maxillofacial Surgery for the Clinician. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-1346-6_19.

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AbstractSuccessful implant dentistry mandates implants to be placed in an appropriate three-dimensional manner that supports the prosthesis adequately. Due to the resorption patterns of edentulous jaws, the ideal position of implants required varying amounts of bone augmentation. Commonly carried out bone-augmentation procedures are Guided Bone Regeneration, onlay bone grafting and sinus floor elevation. This chapter discusses the resorption pattern of edentulous jaws, the biology of alveolar bone of relevance to the maxillofacial surgeon, the biomaterials used for augmentation and the commonl

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Robins, Simon P. "Pyridinium cross-links as bone resorption markers." In Bone Markers. CRC Press, 2024. http://dx.doi.org/10.1201/9781003579472-5.

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Zambonin Zallone, A., and G. Zambonin. "Cellular Basis of Bone Resorption." In Bone Densitometry and Osteoporosis. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80440-3_5.

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Caniggia, Angelo. "Pathophysiology of Bone Formation and Resorption." In Bone Regulatory Factors. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-1508-8_14.

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Ross, F. Patrick. "Osteoclast Biology and Bone Resorption." In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism. John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch3.

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Gowen, M., B. R. MacDonald, D. E. Hughes, H. Skjodt, and R. G. G. Russell. "Immune Cells and Bone Resorption." In Phosphate and Mineral Homeostasis. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5206-8_33.

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Russell, Graham, Gabrielle Mueller, Claire Shipman, and Peter Croucher. "Clinical Disorders of Bone Resorption." In The Molecular Basis of Skeletogenesis. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470846658.ch17.

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Délaissé, J. M., P. Ledent, Y. Eeckhout, and G. Vaes. "Cysteine proteinases and bone resorption." In Cysteine Proteinases and their Inhibitors, edited by Vito Turk. De Gruyter, 1986. http://dx.doi.org/10.1515/9783110846836-029.

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Actes de conférences sur le sujet "Bone resorption"

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Johansson, Lars, Ulf Edlund, Anna Fahlgren, and Per Aspenberg. "A Model for Bone Resorption." In ASME 8th Biennial Conference on Engineering Systems Design and Analysis. ASMEDC, 2006. http://dx.doi.org/10.1115/esda2006-95401.

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In the present paper a model for the resorption of bone, such as that observed at the interface between surgical implants and bone tissue, is developed. While there are many previous studies where models for bone remodelling calculations are proposed, these have been based on the stress or strain state of the bone tissue itself as the driving force for bone remodelling. We, instead, develop a constitutive model based on observations in recent experiments where it seems that fluid pressure, or possibly fluid flow velocity, is a major factor in the bone resorption process.

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Deguchi, Takahiro, Sami V. Koho, Tuomas Näreoja, Juha Peltonen, and Pekka Hänninen. "Tomographic STED microscopy to study bone resorption." In SPIE BiOS, edited by Thomas G. Brown, Carol J. Cogswell, and Tony Wilson. SPIE, 2015. http://dx.doi.org/10.1117/12.2079157.

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Altman, Allison R., Beom Kang Huh, Abhishek Chandra, Wei-Ju Tseng, Ling Qin, and X. Sherry Liu. "3D In Vivo Bone Dynamic Imaging of PTH’s Anabolic Action." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14671.

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Aging shifts bone remodeling toward a negative balance between bone formation and resorption, causing bone loss and increased fracture risk. Anti-resorptive agents are commonly used to inhibit bone resorption and stabilize bone mass. While they are effective to prevent further bone loss, there is also a great need for anabolic agents which can reverse bone deterioration and regain lost skeletal integrity. Intermittent parathyroid hormone (PTH) treatment is the only FDA-approved anabolic treatment for osteoporosis, which greatly stimulates bone formation. Combined therapy of anti-resorptive dru

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Fujita, Hiroshi, Kazuhiro Ido, Weam Farid Mousa, et al. "FEMORAL BONE RESORPTION OBSERVED IN CANINE THA USING BIOACTIVE BONE CEMENT." In Proceedings of the 12th International Symposium on Ceramics in Medicine. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814291064_0124.

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Chong, Desmond Y. R., Ulrich N. Hansen, and Andrew A. Amis. "Computational Biomechanical Analysis of Fixation Performance and Bone Resorption of Tibial Prosthesis Implantation." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205205.

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Long-term survivorship of total knee replacement (TKR) relies on the periprosthetic bone strength and its initial fixation stability. Aseptic loosening caused by mechanical factors is a recognised failure mode for knee prostheses. Bone resorption due to “stress-shielding” of the stiff stemmed implants will potentially lead to weakened bone strength, and also presents a challenge for revision surgery. While the bone cement is commonly used to provide mechanical attachment of the prosthesis to the bone, cement fatigue and bone-cement interface failures would eventually lead to component migratio

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Dong, X. Neil, Y. Young Huang, and X. Edward Guo. "Transversely Isotropic Model of Osteonal Cortical Bone: Contribution of Haversian and Resorptive Porosity." In ASME 1999 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1999. http://dx.doi.org/10.1115/imece1999-0438.

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Abstract Age related changes in porosity of cortical bone have been previously reported. The cortical porosity increases with age in both men and women, from 4.6% in men and 4% in women at age 40 to 10% and more at age 80 (Laval-Jeantet et al., 1983). The porosity is defined as the percentage of cortical bone occupied by vascular and resorption cavities. There are a few quantitative data regarding the influences of Haversian canal and resorption space on porosity. Age related increases in Haversian canal size and Haversian canal number contribute to the increasing porosity of cortical bone for

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Chen, George, Joachim Loo, Margasanti Wijaya, and Yik Thai Hoe. "Determination of Resorption in Bone using Phase Shifting Interferometry." In Bio-Optics: Design and Application. OSA, 2011. http://dx.doi.org/10.1364/boda.2011.jtua2.

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Timchenko, Elena V., Pavel E. Timchenko, Elena V. Pisareva, et al. "Optical analysis of cortical bone tissue in modelling bone resorption in microgravity environment." In XLIV ACADEMIC SPACE CONFERENCE: dedicated to the memory of academician S.P. Korolev and other outstanding Russian scientists – Pioneers of space exploration. AIP Publishing, 2021. http://dx.doi.org/10.1063/5.0036909.

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Penninger, Charles L., Andre´s Tovar, Glen L. Niebur, and John E. Renaud. "Signaling Pathways for Bone Resorption Predicted as a Hybrid Cellular Automaton Process." In ASME 2010 International Mechanical Engineering Congress and Exposition. ASMEDC, 2010. http://dx.doi.org/10.1115/imece2010-39358.

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The bone remodeling process provides for various functions such as mineral homeostasis, damage repair, and adaptation to mechanical loading. At present, a clear link between the mechanical stimulation of bones and the biochemical response is not fully understood. Computational simulations can provide a means to test hypotheses and gain insight into processes that are difficult to examine experimentally. The objective of this work is to predict the effect of damage and strain as the stimulus for regulating the cellular signaling activity of remodeling. In this study, potential signaling pathway

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Kolb, Alexus D., and Karen M. Bussard. "Abstract 3993: 'Educated' osteoblasts suppress osteoclastogenesis and bone resorption in a bone mimetic microenvironment." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-3993.

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Rapports d'organisations sur le sujet "Bone resorption"

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Guan, Junwen, and J. Guan. Risk factors for bone flap resorption after autologous bone cranioplasty: protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.5.0063.

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M, Gilbert. Case Report: An Extreme Case of Alveolar Bone Resorption in an Edentulous Mandible. Science Repository, 2019. http://dx.doi.org/10.31487/j.dobcr.2019.02.03.

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Pavasant, Prasit, and Tussanee Yongchaitrakul. Influence of mechanical stress on the expression of osteopontin in human periodontal ligament cells. Chulalongkorn University, 2008. https://doi.org/10.58837/chula.res.2008.14.

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Background: Mechanical stress such as orthodontic forces can produce mechanical damage and inflammatory reaction in periodontium. Osteopontin (OPN) is a multifunctional cytokine that found to be correlated with periodontal disease progression. As periodontal ligaments (PDL) can be affected by stress and PDL cells are involved in periodontal destruction and remodeling, we aimed to investigate the influence of mechanical stress on the expression and regulation of OPN in human PDL (HPDL) cells. Methods: The mechanical stress was generated by continuous compressive force and the expression of OPN

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Ampornaramveth, Ruchanee. IL-1β mediate cementoblasts and osteoclast precursors interaction. Chulalongkorn University, 2016. https://doi.org/10.58837/chula.res.2016.17.

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An injury of the periodontium followed by an inflammatory response often leads to root resorption. Resorption is accomplished by osteoclasts and their generation may depend on an interaction with the cells in direct contact with the root, the cementoblasts. Our study aimed to investigate the role of human cementoblasts in the formation of osteoclasts and the effect of IL-1β hereupon. Extracted teeth from healthy volunteers were subjected to sequential digestion by type I collagenase and trypsin. The effect of enzymatic digestion on the presence of cells on the root surface was analyzed by hist

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Alshadidi, Abdulkhaliq. Comparative Analysis of Allografts and Xenografts as Bone Substitute Materials In Dentistry: A Systematic Review and Meta-Analysis of Integration And Resorption Rates. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.2.0113.

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Aleksandrov, V. A., A. V. Aleksandrov, L. N. Shilova, and N. V. Aleksandrova. Diagnostic role of angiopoietin-like protein type 3 in assessing the activity of resorptive processes in bone tissue in women with rheumatoid arthritis. Ljournal, 2020. http://dx.doi.org/10.18411/wco-iof-esceo-2020-309.

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