Bibliographies thématiques / Calcification, Physiologic

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Littérature scientifique sur le sujet « Calcification, Physiologic »

Auteur : Grafiati
Publié le 4 juin 2021
Mis à jour le 3 février 2022

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Articles de revues sur le sujet "Calcification, Physiologic"

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Rutsch, Frank, and Robert Terkeltaub. "Deficiencies of physiologic calcification inhibitors and low-grade inflammation in arterial calcification: lessons for cartilage calcification." Joint Bone Spine 72, no. 2 (March 2005): 110–18. http://dx.doi.org/10.1016/j.jbspin.2004.05.014.

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Herrmann, Jaqueline, Milen Babic, Markus Tölle, Markus van der Giet, and Mirjam Schuchardt. "Research Models for Studying Vascular Calcification." International Journal of Molecular Sciences 21, no. 6 (March 23, 2020): 2204. http://dx.doi.org/10.3390/ijms21062204.

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Calcification of the vessel wall contributes to high cardiovascular morbidity and mortality. Vascular calcification (VC) is a systemic disease with multifaceted contributing and inhibiting factors in an actively regulated process. The exact underlying mechanisms are not fully elucidated and reliable treatment options are lacking. Due to the complex pathophysiology, various research models exist evaluating different aspects of VC. This review aims to give an overview of the cell and animal models used so far to study the molecular processes of VC. Here, in vitro cell culture models of different
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Scott, J. E., and M. Haigh. "Is dermatan sulfate proteoglycan the physiologic inhibitor of type I collagen calcification?" Bone 7, no. 2 (January 1986): 154. http://dx.doi.org/10.1016/8756-3282(86)90717-9.

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Doyle, Anthony James, and Graeme D. Anderson. "Physiologic Calcification of the Pineal Gland in Children on Computed Tomography: Prevalence, Observer Reliability and Association with Choroid Plexus Calcification." Academic Radiology 13, no. 7 (July 2006): 822–26. http://dx.doi.org/10.1016/j.acra.2006.04.004.

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Al-Zaghal, Abdullah, Siavash Mehdizadeh Seraj, Thomas J. Werner, Oke Gerke, Poul F. Høilund-Carlsen, and Abass Alavi. "Assessment of Physiologic Intracranial Calcification in Healthy Adults Using 18F-NaF PET/CT." Journal of Nuclear Medicine 60, no. 2 (July 12, 2018): 267–71. http://dx.doi.org/10.2967/jnumed.118.213678.

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Caliskan, Emine, and Mehmet Ozturk. "Evaluation of physiologic pineal gland calcification via computed tomography in the pediatric population." Annals of Medical Research 26, no. 10 (2019): 2391. http://dx.doi.org/10.5455/annalsmedres.2019.06.338.

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Nemes, Attila, and Tamás Forster. "Functional vascular alterations associated with aortic valve stenosis." Orvosi Hetilap 152, no. 25 (June 2011): 993–99. http://dx.doi.org/10.1556/oh.2011.29145.

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Degenerative changes, atherosclerotic process and calcification of valvular leaflets are mostly responsible for valvular aortic valve stenosis, but congenital bicuspid aortic valve and rheumatic fever in history are also known predisposing factors. Aortic valve stenosis is frequently associated with different functional vascular alterations. The aim of this review is to demonstrate these vascular alterations evaluated by non-invasive methods and underlying physiologic and pathophysiologic processes. Orv. Hetil., 2011, 152,993–999.
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Rezvova, M. A., E. A. Ovcharenko, T. V. Glushkova, Yu A. Kudryavtseva, and L. S. Barbarash. "Evaluation of calcification resistance of xenopericardium treated with polyhydroxy compounds." Russian Journal of Transplantology and Artificial Organs 23, no. 1 (April 10, 2021): 75–83. http://dx.doi.org/10.15825/1995-1191-2021-1-75-83.

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Calcification of biomaterials used in prosthetic heart valves has been a challenging issue in cardiovascular surgery. The objective of this work is to compare the efficiency of polyvinyl alcohol (PVA) and tannic acid (TA) modification of xenomaterials, pre-stabilized with glutaraldehyde (GA) and ethylene glycol diglycidyl ether (EGDE), in reducing calcification. Analysis of mechanical properties evaluated under uniaxial tension, showed a significant increase in the tensile strength of the test samples compared to the control (unmodified) samples (p < 0.05). Additional treatment of GA-fixed
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Fukai, Atsushi, Naohiro Kawamura, Taku Saito, Yasushi Oshima, Toshiyuki Ikeda, Fumitaka Kugimiya, Akiro Higashikawa, et al. "Akt1 in murine chondrocytes controls cartilage calcification during endochondral ossification under physiologic and pathologic conditions." Arthritis & Rheumatism 62, no. 3 (February 25, 2010): 826–36. http://dx.doi.org/10.1002/art.27296.

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Terkeltaub, Robert. "Physiologic and pathologic functions of the NPP nucleotide pyrophosphatase/phosphodiesterase family focusing on NPP1 in calcification." Purinergic Signalling 2, no. 2 (June 2006): 371–77. http://dx.doi.org/10.1007/s11302-005-5304-3.

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Thèses sur le sujet "Calcification, Physiologic"

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Barragan-Adjemian, Maria del Cielo Bonewald Lynda F. "Mechanisms of mineralization in bone." Diss., UMK access, 2006.

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Thesis (Ph. D.)--School of Dentistry. University of Missouri--Kansas City, 2006.<br>"A dissertation in oral biology and cell biology and biophysics." Advisor: Lynda F. Bonewald. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed Nov. 12, 2007. Includes bibliographical references (leaves 121-139). Online version of the print edition.
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Bennett, Brian J. "Chondroplastic conversion and calcification of advanced atherosclerotic lesions : the impact of bone regulatory proteins and diet /." Thesis, Connect to this title online; UW restricted, 2006. http://hdl.handle.net/1773/6602.

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Curinga, Gabrielle Mercedes. "The role of runt-related transcription factor 2 in arterial smooth muscle cell mineralization /." Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/6353.

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Clark, Ruti H. "A model system for investigating biomineralization : elucidating protein G/calcium oxalate monohydrate interactions /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/8067.

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Bertucci, Daniela Vendrame. "Estudo sobre o efeito do atenolol na mineralização de dentes e ossos de filhotes de ratas espontaneamente hipertensas (SHR) e normotensas /." Araçatuba : [s.n.], 2009. http://hdl.handle.net/11449/95467.

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Orientador: Cristina Antoniali Silva<br>Banca: Alberto Carlos Botazzo Delbem<br>Banca: Carlos Ferreira dos Santos<br>Resumo: O tratamento da hipertensão durante a gravidez visa diminuir os riscos maternos e fetais. Entre os diferentes tipos de anti-hipertensivos que podem ser utilizados durante este período, estão os antagonistas dos receptores β-adrenérgicos. O atenolol é um antagonista seletivo de receptores 1-adrenérgicos que atravessa a barreira placentária e é excretado no leite materno chegando com facilidade ao feto de mães tratadas e aos recém-nascidos amamentados. Embora vários estudo
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Adragão, Maria Teresa Pulido. "Calcificações vasculares nos doentes em diálise : elo de ligação entre doença óssea e doença vascular." Doctoral thesis, Faculdade de Ciências Médicas. Universidade Nova de Lisboa, 2011. http://hdl.handle.net/10362/6298.

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RESUMO: A presente dissertação para tese de doutoramento apresenta o desenvolvimento e a validação de um método simples e original para o diagnóstico de calcificações vasculares em doentes em diálise, utilizando um score semiquantitativo criado por nós e obtido em RX simples da bacia e das mãos, denominado score de calcifi cação vascular simples. Demonstramos que este score vascular simples é preditor de risco cardiovascular nos doentes em diálise. O score de calcificação vascular simples associou-se ainda à baixa densidade mineral óssea avaliada por dual energy X -ray absortiometry (DXA) no
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Gilbert, Michele. "Design of synthetic peptides that display cell binding and signaling sequences on calcium phosphate surfaces /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/8063.

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Somogyi-Ganss, Eszter. "Novel non-collagenous modulators of biomineralization in bone and dentin /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-101-6/.

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Bertucci, Daniela Vendrame [UNESP]. "Estudo sobre o efeito do atenolol na mineralização de dentes e ossos de filhotes de ratas espontaneamente hipertensas (SHR) e normotensas." Universidade Estadual Paulista (UNESP), 2009. http://hdl.handle.net/11449/95467.

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Made available in DSpace on 2014-06-11T19:27:47Z (GMT). No. of bitstreams: 0 Previous issue date: 2009-11-26Bitstream added on 2014-06-13T19:15:13Z : No. of bitstreams: 1 bertucci_dv_me_araca.pdf: 382588 bytes, checksum: 00700240bf69abe838e21d8e0b8b4fac (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>O tratamento da hipertensão durante a gravidez visa diminuir os riscos maternos e fetais. Entre os diferentes tipos de anti-hipertensivos que podem ser utilizados durante este período, estão os antagonistas dos receptores β-adrenérgicos. O atenolol é um antagonist
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Anderson, Paul Hamill. "The regulation of Vitamin D metabolism in the kidney and bone." Title page, contents and abstract only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09pha5486.pdf.

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Includes bibliographical references (leaves 226-273.) Investigates the regulation of the expression of CYP27B1, CYP24 and vitamin D receptor (VDR) mRNA, both in the bone and in the kidney, with the aim to determine whether the regulation of the vitamin D metabolism in the bone is independent from that in the kidney. The effects of age, dietary calcium and vitamin D status on the expression of these genes in both the kidney and the bone, as well as on a number of biochemical factors known to regulate the renal metabolism of 1,25D, such as PTH, calcium and 1,25D itself, were examined. CYP27B1 mR
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Livres sur le sujet "Calcification, Physiologic"

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Yousuf, Ali S., ed. Cell mediated calcification and matrix vesicles: Proceedings of the IV International Conference on Matrix Vesicles, Cambridge, 1-5 July 1985. Amsterdam: Excerpta Medica, 1986.

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C, Slavkin Harold, and Price Paul A, eds. Chemistry and biology of mineralized tissues: Proceedings of the Fourth International Conference on the Chemistry and Biology of Mineralized Tissues held in Coronado, California on February 5-9, 1992. Amsterdam: Excerpta Medica, 1992.

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International, Workshop on Calcified Tissues (6th 1984 Kiryat ʻAnavim Israel). Current advances in skeletogenesis: Induction, biomineralization, bone seeking hormones, congenital and metabolic bone diseases : proceedings of the Sixth International Workshop on Calcified Tissues, Kiryat-Anavim, Israel, 18-23 March 1984. Amsterdam: Excerpta Medica, 1985.

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International Conference on the Chemistry and Biology of Mineralized Tissues (3rd 1988 Chatham, Mass.). The chemistry and biology of mineralized tissues: Proceedings of the Third International Conference on the Chemistry and Biology of Mineralized Tissues, held in Chatham, Massachusetts on October 16-21, 1988. New York: Gordon and Breach, 1989.

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European Symposium on Calcified Tissues (20th 1987 Sirmione, Italy). XX European Symposium on Calcified Tissues, Sirmione, Italy, October 4-8, 1987: Abstracts, including Satellite Workshop on Molecular and Cell Biology and Satellite Workshop on Biology and Regulation of Bone Metabolism : Clinical Significance. New York: Springer International, 1987.

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Calcium ions in nerve cell function. Oxford: Oxford University Press, 1992.

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M, Rabie A. Bakr, and Urist Marshall R, eds. Bone formation and repair: Proceedings of the International Symposium on Formation and Repair of Mineralized Extracellular Matrix, Hong Kong, 18-19 October, 1996. Amsterdam [Netherlands]: Elsevier, 1997.

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L, Hukins David W., ed. Calcified tissue. Boca Raton, Fla: CRC Press, 1989.

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(Editor), Erich Konigsberger, and LanChi Konigsberger (Editor), eds. Biomineralization: Medical Aspects of Solubility. Wiley, 2006.

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Erich, Königsberger, and Königsberger LanChi, eds. Biomineralization: Medical aspects of solubility. Chichester, West Sussex, England: John Wiley & Sons, 2006.

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Chapitres de livres sur le sujet "Calcification, Physiologic"

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Taylor, Alison R., and Colin Brownlee. "Calcification." In The Physiology of Microalgae, 301–18. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-24945-2_14.

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McKinney, Alexander M. "Basal Ganglia: Physiologic Calcifications." In Atlas of Normal Imaging Variations of the Brain, Skull, and Craniocervical Vasculature, 427–40. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-39790-0_19.

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Xu, Kai, Kunshan Gao, and David A. Hutchins. "Measurements of Calcification and Silicification." In Research Methods of Environmental Physiology in Aquatic Sciences, 269–76. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-5354-7_32.

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Kimura, Yasuko, Shigeshi Kikunaga, Ichiro Takahashi, Yuji Hatakeyama, Satoshi Fukumoto, and Yasuyuki Sasano. "The physiological calcification process is replicated in a rat embryonic calvarial culture." In Interface Oral Health Science 2009, 179–80. Tokyo: Springer Japan, 2010. http://dx.doi.org/10.1007/978-4-431-99644-6_40.

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Dupont, Sam. "Use of the Fluorochrome Calcein to Measure Growth and Calcification in Marine Organisms." In Research Methods of Environmental Physiology in Aquatic Sciences, 277–84. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-5354-7_33.

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Grosell, Martin. "CO2 and calcification processes in fish." In Fish Physiology, 133–59. Elsevier, 2019. http://dx.doi.org/10.1016/bs.fp.2019.07.002.

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Argulian, Edgar. "Degenerative cardiovascular disease in the elderly." In ESC CardioMed, 2960–64. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0716.

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The ageing population represents a unique challenge for cardiovascular care. Ageing causes remodelling of the cardiovascular system and commonly results in distinct degenerative changes. Most of these degenerative changes have significant interactions with cardiovascular risk factors by either being a marker of disease burden or being exaggerated by co-morbid conditions. Also, age-related degenerative conditions have physiological and prognostic implications. This chapter discusses several common degenerative cardiovascular conditions in the elderly such as cardiac fibrosis, amyloidosis, mitral annular calcification, and aortic valve sclerosis. Some of these conditions (such as cardiac fibrosis and amyloidosis) are implicated in pathogenesis of heart failure with preserved ejection fraction. Others (such as mitral annular calcification and aortic valve sclerosis) do not typically cause any measurable physiological abnormality but have prognostic significance.
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Argulian, Edgar. "Degenerative cardiovascular disease in the elderly." In ESC CardioMed, 2960–64. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0716_update_001.

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The ageing population represents a unique challenge for cardiovascular care. Ageing causes remodelling of the cardiovascular system and commonly results in distinct degenerative changes. Most of these degenerative changes have significant interactions with cardiovascular risk factors by either being a marker of disease burden or being exaggerated by co-morbid conditions. Also, age-related degenerative conditions have physiological and prognostic implications. This chapter discusses several common degenerative cardiovascular conditions in the elderly such as cardiac fibrosis, amyloidosis, mitral annular calcification, and aortic valve sclerosis. Some of these conditions (such as cardiac fibrosis and amyloidosis) are implicated in pathogenesis of heart failure with preserved ejection fraction. Others (such as mitral annular calcification and aortic valve sclerosis) do not typically cause any measurable physiological abnormality but have prognostic significance.
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Williams, Daniel C., and Charles A. Frolik†. "Physiological and Pharmacological Regulation of Biological Calcification." In International Review of Cytology, 195–292. Elsevier, 1991. http://dx.doi.org/10.1016/s0074-7696(08)60685-3.

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A. Aljafary, Meneerah, Hussah Alshwyeh, Nada Alahmadi, Adeeb Shehzad, Huseyin Tombuloglu, Zagit Gaymalov, Abdelqader Homieda, and Ebtesam Al-Suhaimi. "Physiological and Cellular Functions of Vitamin K on Cardiovascular Function." In Vitamin K - Recent Advances, New Perspectives and Applications for Human Health [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99344.

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This chapter reviews the physiological and cellular functions of vitamin K in the cardiovascular system based on the latest pre-clinical and clinical evidence. Vitamin K belongs to a family of structurally similar fat-soluble vitamins, actively required by the body for the synthesis of essential proteins as well as regulate blood clotting, bone metabolism and calcium level. The authors emphasize the quintessential association between dietary vitamin K2 and cardiovascular diseases shown in various studies. The association, through the vitamin K - dependent hormones, plays a primary role in regulating calcification of different cell types, especially their role in calcification of the vascular endothelial cells. The consequences of vitamin K deficiency in the vascular system are unfavorable, shown in various clinical studies on statins - well-known inhibitors of vitamin K production in the body. New clinical insights suggest that vitamin K levels in the body and its dietary supplementation play a crucial role in cardiovascular disease prevention. There is negative influence of these antagonist’s pate in vascular composition and functions. Therefore, there is a need for prospective studies to make more in-depth exploration and increase the current understanding of this critical relationship to confidently apply such knowledge to prevent cardiovascular diseases and improve their outcomes.
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Actes de conférences sur le sujet "Calcification, Physiologic"

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Govindarajan, V., J. Mousel, S. C. Vigmostad, H. S. Udaykumar, M. M. Levack, J. H. Gorman, B. M. Jackson, R. C. Gorman, and K. B. Chandran. "Patient-Specific Valve Dynamics Using 3D Fluid-Structure Interaction Modeling: Comparison Between Bicuspid and Tricuspid Aortic Valves." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14563.

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Aortic valve diseases such as congenital bicuspid aortic valve (BAV) and progressive calcification in tricuspid valves affect the hemodynamics in the aortic arch. In addition to leaflet calcification, BAVs are associated with other ailments such as aortic coarctation, aneurysm and dissection [1]. It has also been observed that progressive calcification is accelerated in the case of BAVs compared to normal tricuspid valves. While it is not yet known whether the geometric distortion in BAVs is the main cause of calcification [2] in these valves, the distortion in the leaflets may give rise to al
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Govindarajan, V., H. S. Udaykumar, S. Vigmostad, M. M. Levack, J. H. Gorman, B. M. Jackson, R. C. Gorman, and K. B. Chandran. "Fluid Structural Interaction of a Patient Specific Congenital Bicuspid Aortic Valve." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80196.

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Congenital Bicuspid Aortic Valve (BAV) is a valvular anomaly where a patient is born with a valve with two leaflets instead of a normal tri-leaflet valve. It has also been reported that BAVs are prone to progressive calcification and also other complications such as ascending aortic dilatation, dissection and rupture [1]. The geometrical variations with the BAV may be a factor in altering the deformation and stresses on the leaflets resulting in calcification of the leaflets earlier than with normal tri-leaflet aortic valves. Altered flow patterns past BAV into the ascending aorta can also be
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Kapnisis, Konstantinos, Polyvios Eleftheriou, George Lapathitis, Christos Karaiskos, Preston Beck, Jack Lemons, David Connolly, Costas Pitsillides, and Andreas Anayiotos. "Surface Modified Nitinol Stents Release Metal Ions in Blood." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14244.

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Intravascular nitinol stents are used in the treatment of atherosclerosis and intracranial aneurysms. Despite the unique physical properties of shape memory and superelasticity, the chemical composition of NiTi has raised concerns due to the presence of nickel ions within the alloy which can have adverse effects on human health. Currently, stents are manufactured from corrosion resistant alloys which form protective titanium oxide films, insulating the bulk material from the corrosive physiologic fluid. However, nanometer thick regions of oxides are lost at locations of high strain due to sign
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Boekhoven, Renate W., Richard G. P. Lopata, Marcel C. M. Rutten, Marc R. H. M. van Sambeek, and Frans N. van de Vosse. "Novel Strategy of the Determination of Mechanical Properties of Human Carotid Atherosclerotic Plaques." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80669.

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Carotid endarterectomy is the procedure of choice in patients with a recent symptomatic stenosis of 70–99%. Currently, the selection of candidates eligible for carotid endarterectomy is based on stenosis size only. However, the treatment is only beneficial for patients with unstable plaques, which comprises only 16% of the patient population [1]. Hence, identifying plaque stability at an early stage would permit timely intervention, while substantially reducing overtreatment of stable plaques. The objective of this study is to distinguish between stable and unstable carotid atherosclerotic pla
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