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Littérature scientifique sur le sujet « Chemical Induced Toxicity »

Auteur : Grafiati

Publié le 2 juin 2025

Mis à jour le 31 juillet 2025

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Articles de revues sur le sujet "Chemical Induced Toxicity"

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Fariss, Marc W., Marda K. Brown, John A. Schmitz, and Donald J. Reed. "Mechanism of chemical-induced toxicity." Toxicology and Applied Pharmacology 79, no. 2 (1985): 283–95. http://dx.doi.org/10.1016/0041-008x(85)90350-3.

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Fariss, Marc W., and Donald J. Reed. "Mechanism of chemical-induced toxicity." Toxicology and Applied Pharmacology 79, no. 2 (1985): 296–306. http://dx.doi.org/10.1016/0041-008x(85)90351-5.

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Souza Monteiro de Araújo, Daniel, Rafael Brito, Danniel Pereira-Figueiredo, et al. "Retinal Toxicity Induced by Chemical Agents." International Journal of Molecular Sciences 23, no. 15 (2022): 8182. http://dx.doi.org/10.3390/ijms23158182.

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Vision is an important sense for humans, and visual impairment/blindness has a huge impact in daily life. The retina is a nervous tissue that is essential for visual processing since it possesses light sensors (photoreceptors) and performs a pre-processing of visual information. Thus, retinal cell dysfunction or degeneration affects visual ability and several general aspects of the day-to-day of a person’s lives. The retina has a blood–retinal barrier, which protects the tissue from a wide range of molecules or microorganisms. However, several agents, coming from systemic pathways, reach the r

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Martin, WJ. "Pulmonary toxicity induced by chemical agents." European Respiratory Journal 3, no. 3 (1990): 375–76. http://dx.doi.org/10.1183/09031936.93.03030375.

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Kennedy, Katherine A., William N. Hait, and John S. Lazo. "Chemical modulation of bleomycin induced toxicity." International Journal of Radiation Oncology*Biology*Physics 12, no. 8 (1986): 1367–70. http://dx.doi.org/10.1016/0360-3016(86)90173-2.

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Colby, H. D. "Adrenal Gland Toxicity: Chemically Induced Dysfunction." Journal of the American College of Toxicology 7, no. 1 (1988): 45–69. http://dx.doi.org/10.3109/10915818809078702.

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Among the endocrine organs, the adrenal cortex appears to be the most vulnerable to chemically induced injury. A wide variety of chemicals has been found to cause morphological or functional lesions in the gland. Some of the lesions are highly localized to specific anatomical zones of the adrenal cortex, and the resulting functional deficits depend on the physiological role(s) of the zone affected. In addition, metabolic activation is an important factor contributing to the gland's vulnerability to chemical injury. For example, carbon tetrachloride (CCl4) causes adrenocortical necrosis, but on

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Rokad, Dharmin, Huajun Jin, Vellareddy Anantharam, Arthi Kanthasamy, and Anumantha G. Kanthasamy. "Exosomes as Mediators of Chemical-Induced Toxicity." Current Environmental Health Reports 6, no. 3 (2019): 73–79. http://dx.doi.org/10.1007/s40572-019-00233-9.

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Jaganathan, Keerthana, Mobeen Ur Rehman, Hilal Tayara, and Kil To Chong. "XML-CIMT: Explainable Machine Learning (XML) Model for Predicting Chemical-Induced Mitochondrial Toxicity." International Journal of Molecular Sciences 23, no. 24 (2022): 15655. http://dx.doi.org/10.3390/ijms232415655.

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Organ toxicity caused by chemicals is a serious problem in the creation and usage of chemicals such as medications, insecticides, chemical products, and cosmetics. In recent decades, the initiation and development of chemical-induced organ damage have been related to mitochondrial dysfunction, among several adverse effects. Recently, many drugs, for example, troglitazone, have been removed from the marketplace because of significant mitochondrial toxicity. As a result, it is an urgent requirement to develop in silico models that can reliably anticipate chemical-induced mitochondrial toxicity.

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Batt, A. M., and L. Ferrari. "Manifestations of chemically induced liver damage." Clinical Chemistry 41, no. 12 (1995): 1882–87. http://dx.doi.org/10.1093/clinchem/41.12.1882.

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Abstract Possible liver damage induced by chemicals or drugs must be detected early during drug development or industrial exposure, although damage is still difficult to predict, especially when immunotoxicity is involved. Liver toxicity may result from cytolysis, steatosis, cholestasis, phospholipidosis, or vascular lesions, most the outcome of a disadvantageous balance between chemicals or metabolites vs protective mechanisms, resulting from chemical dosage, genetic factors, or the immunoallergic status of the patient. Drug metabolism, lipid peroxidation, and thiol oxidation are frequently i

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Stavrakeva, K., M. Popova, M. Esad, et al. "Drug-Induced Liver Toxicity." Acta Medica Bulgarica 51, no. 4 (2024): 77–85. http://dx.doi.org/10.2478/amb-2024-0083.

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Abstract The liver performs many vital functions such as regulating homeostasis, bile production, storage of vitamins, and more. Another important function of the liver is to neutralize toxic substances entering the body. Substances entering the human body can be eliminated unchanged, retained unchanged, or undergo chemical transformation. Drugs are one of the most important and common causes of hepatotoxicity. It can manifest in various forms, ranging from elevated serum levels of transaminases to acute liver failure. The mechanisms of drug-induced liver damage may include the formation of a

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Thèses sur le sujet "Chemical Induced Toxicity"

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Lovatt, Cerys Ann. "The role of glutathione S-transferase Pi in chemical-induced toxicity." Thesis, University of Liverpool, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412639.

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Kang, SooYeon. "HIGH-THROUGHPUT METABOLISM-INDUCED TOXICITY ASSAYS ON A 384-PILLAR PLATE." Cleveland State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=csu1530883608111352.

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Mehta, Darshan. "New Fragmentation Method to Enhance Structure-Based In Silico Modeling of Chemically-Induced Toxicity." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1449759891.

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Rajanahalli, Krishnamurthy Pavan. "Toxicity Of Silver Nanoparticles In Mouse Embryonic Stem Cells And Chemical Based Reprogramming Of Somatic Cells To Sphere Cells." University of Dayton / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=dayton1327115925.

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Yang, Jianhua. "Protective action of metallothionein against chemically induced toxicity." HKBU Institutional Repository, 1999. http://repository.hkbu.edu.hk/etd_ra/185.

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Sauer, John-Michael. "All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity." Diss., The University of Arizona, 1996. http://hdl.handle.net/10150/187501.

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It has been previously reported that acute hypervitaminosis A in rats dramatically increases the hepatotoxicity of a number of chemicals. This potentiation appears to be mediated by the enhanced release of reactive oxygen species from retinol-primed Kupffer cells. However, in the lung it has been shown that retinol can protect against many of the inflammatory effects caused by bleomycin. Whether or not retinol pretreatment can modulate chemical-induced injury of compounds that cause both liver and lung toxicity is unknown. Therefore, the studies presented here were designed to test the hypothe

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Smith, Bill J. "The role of epoxidation in 4-vinylcyclohexene-induced ovarian toxicity." Diss., The University of Arizona, 1990. http://hdl.handle.net/10150/185059.

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The basis for the species difference between B6C3F1 mice (susceptible) and Fischer 344 rats (resistant) to 4- vinylcyclohexene (VcH)-induced ovarian tumorigenicity was investigated. Greater than 95% of a single oral 400 mg/kg dose of [¹⁴C]VCH was eliminated in 48 hr by mice and rats. Approximately 50-60% of the administered dose was excreted in the urine, while the remaining 30-40% of the dose was expired as organically soluble radioactivity. VCH-treated mice had dramatically higher blood concentrations of the VCH metabolite VCH-1,2-epoxide compared to VCH-treated rats. Furthermore, mouse hepa

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Nahle, Sara. "Réponse macrophagique aux nanomatériaux carbonés : effets de leur caractéristiques physiques et chimiques sur le transcriptome Carbon-based nanomaterials induce inflammation and autophagy in rat alveolar macrophages Single wall and multiwall carbon nanotubes induce different toxicological responses in rat alveolar macrophages Gene expression profiling of alveolar macrophages exposed to non-functionalized, anionic or cationic multi-walled carbon nanotubes shows three different mechanisms of toxicity Cytotoxicity and global transcriptional responses induced by zinc oxide nanoparticles NM 110 in PMA-differentiated THP-1 cells Protein and lipid homeostasis altered in rat macrophages after exposure to metallic oxide nanoparticles." Thesis, Université de Lorraine, 2019. http://www.theses.fr/2019LORR0142.

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Les nanomatériaux carbonés (NMC) sont très utilisés dans le monde industriel et leurs applications, nombreuses, sont en plein développement. L’absence de réglementation pour leur préparation et leur emploi fait qu’il est nécessaire comme pour tous les nano-objets, de déterminer le risque qu’une exposition fait courir à l’Homme et d’adapter la législation en conséquence. Une meilleure connaissance de leur potentiel toxique est donc nécessaire. Les difficultés de plus en plus grandes pour utiliser les modèles animaux, rend nécessaire le développement d’études avec des lignées cellulaires au sein

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Galdones, Eugene. "The role of retinoic acid receptor gamma in retinoid-induced limb dysmorphogenesis /." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=115711.

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Retinol (vitamin A) and its active metabolite, all-trans retinoic acid, signal through nuclear retinoic acid and retinoid X receptor (RAR/RXR) heterodimers. These complexes regulate the expression of genes involved in developmental processes such as limb development. In excess, retinoids are potent teratogens and cause marked reductive effects on the developing limb. The goal of this thesis was to elucidate the molecular mechanisms underlying retinoid-induced limb dysmorphogenesis. Specifically, using an in vitro limb culture system, I examined the involvement of one RAR isoform, RARgamma, in

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Campbell, Holly R. 1976. "Chlorine-induced lung injury and the role of iNOS." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111574.

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Reactive airways dysfunction syndrome (RADS), a form of irritant-induced asthma (IIA) has been observed in humans following acute chlorine (Cl 2) gas exposure in occupational and domestic settings. Following Cl 2 injury, subepithelial fibrosis, mucous hyperplasia, and non-specific airway hyperresponsiveness have been reported. Based on the disease profile, we hypothesized that pulmonary damage may be oxidative in nature.<br>The aim of this work was to develop a murine model of irritant-induced asthma in order to investigate the pathogenic processes and potential oxidative mechanisms involved i

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Livres sur le sujet "Chemical Induced Toxicity"

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David, Warshawsky, and Landolph Joseph R, eds. Molecular carcinogenesis and the molecular biology of human cancer. CRC, Taylor and Francis, 2005.

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Ching-Hung, Hsu, and Stedeford Todd, eds. Cancer risk assessment: Chemical carcinogenesis, hazard evaluation, and risk quantification. Wiley, 2010.

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Wilson, Cynthia. Chemical exposure and human health: A reference to 314 chemicals with a guide to symptoms and a directory of organizations. McFarland & Co., 1993.

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Richard, Cothern C., Mehlman Myron A, and Marcus William L, eds. Risk assessment and risk management of industrial and environmental chemicals. Princeton Scientific Pub. Co., 1988.

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1925-, Papirmeister Bruno, ed. Medical defense against mustard gas: Toxic mechanisms and pharmacological implications. CRC Press, 1991.

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Andreas, Luch, ed. The carcinogenic effects of polycyclic aromatic hydrocarbons. Imperial College Press, 2004.

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Miloň, Tichý, Společnost pracovního lékařství (Czechoslovakia), and European Symposium on Quantitative Structure-Activity Relationships (5th : 1984 : Bad Segeberg, Germany), eds. QSAR in toxicology and xenobiochemistry: Proceedings of a symposium, Prague, Czechoslovakia, September 12-14, 1984 (a satellite symposium to the 5th European Symposium on Chemical Structure-Biological Activity, Bad Segeberg, F.R.G., September 17-21, 1984). Elsevier, 1985.

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Project, Depleted Uranium Education. Metal of dishonor, depleted uranium: How the Pentagon radiates soldiers and civilians with DU weapons. International Action Center, 1997.

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IARC Working Group on the Evaluation of Carcinogenic Risks to Humans. Dry cleaning, some chlorinated solvents and other industrial chemicals. IARC, 1995.

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Imperial Cancer Research Fund (Great Britain). Nitrate, nitrite and nitroso compounds in human cancer. Edited by Forman David, Shuker David, and Shuker David E. G. Published for the Imperial Cancer Research Fund by Oxford University Press, 1989.

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Chapitres de livres sur le sujet "Chemical Induced Toxicity"

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Tiwari, Abhishek, Varsha Tiwari, and Bimal Krishna Banik. "The Role of Tempol in NRTI-Induced Mitochondrial toxicity." In Chemical and Clinical Applications of Tempol. CRC Press, 2024. http://dx.doi.org/10.1201/9781003426820-6.

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Wagner, Bridget K. "Chemical Biology Approach to Suppression of Statin-Induced Muscle Toxicity." In Concepts and Case Studies in Chemical Biology. Wiley-VCH Verlag GmbH & Co. KGaA, 2014. http://dx.doi.org/10.1002/9783527687503.ch10.

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Uniyal, Navendra, and Sattiraju Krishna Sundari. "Chemical Pesticides Induced Toxicity on Biotic Systems and Biopesticides as the Way Forward." In Advances in Science, Technology & Innovation. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-80189-1_4.

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Gatica-Arias, Andrés, and César Vargas-Segura. "Chemical Mutagenesis of Coffee Seeds (Coffea arabica L. var. Catuaí) Using NaN3." In Mutation Breeding in Coffee with Special Reference to Leaf Rust. Springer Berlin Heidelberg, 2023. http://dx.doi.org/10.1007/978-3-662-67273-0_13.

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AbstractCoffee (Coffea arabica L.) is one of the most important crops in the world and one of the main export products in several developing countries. Coffee is a perennial crop threatened by multiple, serious diseases and pests. Induced mutagenesis of seeds is widely used for increasing the genetic diversity and improvement of annual seed crops and could equally be applied to Arabica coffee breeding and genetic studies. Here we describe protocols to induce genetic variability in Arabica coffee seeds through mutagenesis using sodium azide (NaN3). Methods for NaN3 chemical toxicity testing and

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Masood, Muhammad Arslan, Samuel Kaski, Hugo Ceulemans, Dorota Herman, and Markus Heinonen. "Balancing Imbalanced Toxicity Models: Using MolBERT with Focal Loss." In Lecture Notes in Computer Science. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-72381-0_8.

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AbstractDrug-induced liver injury (DILI) presents a multifaceted challenge, influenced by interconnected biological mechanisms. Current DILI datasets are characterized by small sizes and high imbalance, posing difficulties in learning robust representations and accurate modeling. To address these challenges, we trained a multi-modal multi-task model integrating preclinical histopathologies, biochemistry (blood markers), and clinical DILI-related adverse drug reactions (ADRs). Leveraging pretrained BERT models, we extracted representations covering a broad chemical space, facilitating robust le

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Gatica-Arias, Andrés, and Jorge Rodríguez-Matamoros. "Chemical Mutagenesis of Zygotic Embryos of Coffea arabica L. var. Catuaí Using EMS and NaN3." In Mutation Breeding in Coffee with Special Reference to Leaf Rust. Springer Berlin Heidelberg, 2023. http://dx.doi.org/10.1007/978-3-662-67273-0_9.

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AbstractThe genetic improvement of C. arabica L. is challenged by its low genetic diversity and autogamous reproductive biology. Induced mutagenesis offers an alternative approach to conventional cross-breeding to increase genetic variability in wild and cultivated Arabica coffee germplasm for further use in breeding programs and genetic studies. Here protocols are described for the preparation of zygotic embryos from C. arabica seed and for toxicity testing of zygotic embryos using two chemical mutagens, sodium azide (NaN3) and ethyl methanesulfonate (EMS). Zygotic embryos were immersed for 1

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Yih, T. D., A. J. M. Degen, J. Kamerman, and H. F. P. Joosten. "The Dutch Rabbit in Toxicity Testing Chemical — Induced Creatine Kinase Release; a Species- and Strain-Specific Response?" In New Developments in Biosciences: Their Implications for Laboratory Animal Science. Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-3281-4_36.

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Smith, Martyn T. "Role of Reactive Oxygen Species and Lipid Peroxidation in Chemically Induced Toxicity and Carcinogenesis." In Reactive Oxygen Species in Chemistry, Biology, and Medicine. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-0417-4_11.

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Heena, Mrs, Satyandra Kumar, and Yeshvandra Verma. "ZINC INDUCED OCCUPATIONAL TOXICITY." In Advances in Chemical Science: Exploring New Frontiers. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/nbennurch153.

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With five stable isotopes, zinc is the 24th most abundant element in the crust of the earth. Zinc is a vital trace element that is required for both prenatal and postnatal development in humans, animals, plants, and microorganisms. It is the only metal that is present in every class of enzyme and is the second most common trace metal in humans, after irons. It is a co-factor for the folding of proteins and necessary for cellular metabolism. It has pleiotropic effects on the physiology of cells, and either an excess or a deficiency can lead to pathologies such as stroke and diabetes that have d

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Polaka, Suryanarayana, Muktika Tekade, Nupur Vasdev, et al. "Exosomes as mediators of chemical-induced toxicity." In Essentials of Pharmatoxicology in Drug Research, Volume 1. Elsevier, 2023. http://dx.doi.org/10.1016/b978-0-443-15840-7.00007-5.

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Actes de conférences sur le sujet "Chemical Induced Toxicity"

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Lauenstein, Lan, Simone Switalla, Frauke Prenzler, et al. "Assessment Of Lung Toxicity Induced By Chemical Allergens In Human Precision-Cut Lung Slices." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4662.

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"The Protective Effect of Withania somnifera Root Extract Against Acetaminophen Induced Liver Toxicity in Cyprinus carpio Compared to Silymaryn." In International Conference on Chemical, Environment & Biological Sciences. International Institute of Chemical, Biological & Environmental Engineering, 2014. http://dx.doi.org/10.15242/iicbe.c914122.

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Pineda-Medina, Alfredo. "In Vitro Evaluation of Adjuvant Therapy With Hyperthermia Induced by Irradiation (Nir) of AuNPs and Icg in Tumor Cells." In 5th World Conference on Chemistry and Chemical Engineering and 5th World Conference on Advanced Materials, Nanoscience and Nanotechnology. Eurasia Conferences, 2024. https://doi.org/10.62422/978-81-970328-7-5-012.

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Hyperthermia (HT) is a novel alternative to cancer treatment based on the increase of temperature between 40-43°C inducing selective death in tumor cells, facing cancer treatment limitations such as drug resistance. HT employs nanomaterials such as gold nanoparticles (AuNPs) with the capacity to absorb light at a wavelength of 600-800nm by near-infrared region (NIR) producing heat, allowing efficient penetration of light into the tumor without damaging near tissue and improving cellular absorption. This work aimed to evaluate the effect on cell viability of HT combined with 5-FU on SW620-GFP c

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Zedan, Randa Hisham, Zahoor Ul-Hassan, Roda Al-Thani, Quirico Migheli, and Samir Jaoua. "Biological Control of Mycotoxigenic Fungi and Ochratoxin by the In-Vitro Application of a Qatari Burkholderia cepacia Strain (QBC03)." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0091.

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Mycotoxins are secondary metabolites synthesized by mycotoxigenic fungi belonging mainly to three major fungal genera that are Aspergillus, Fusarium and Penicillium. The latter mycotoxigenic fungi contaminate plants and different food commodities and cause various health concerns (carcinogenicity, mutagenicity, nephrotoxicity, etc...) due to their corresponding mycotoxins. One of the most studied mycotoxins is ochratoxin A (OTA) owing to its toxicity level (classified in 2B group as possible human carcinogenic). To remediate the mycotoxins’ contamination, physical and chemical techniques can b

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Li, Jiang, J. W. Ma, J. Y. Tu, L. Tian, and G. Ahmadi. "Detailed Analysis of Fiber Motion in Human Nasal Airways." In ASME 2021 Fluids Engineering Division Summer Meeting. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/fedsm2021-65576.

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Abstract Information on the fiber particle transport and deposition in human nasal airways is of great importance in inhalation toxicology study. Due to the complex interactions between the inhaled aerosol particles and human respiratory airways, the particles’ toxicity varies with their chemical composition, size, and shape. In the earlier computational study of fiber particle motion in human nasal cavities, overall deposition efficiency curves were evaluated and compared with the available experimental data. The majority of investigations were on micron-scale fiber particles, and the observe

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Grubbs, Clinton J., Yian Wang, Ming You, Vernon Steele, Daniel Boring, and Ronald Lubet. "Abstract 962: Similar preventive effects of naproxen and NO-naproxen in a chemically-induced model of urinary bladder cancer: Dose dependency, gene expression and toxicity." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-962.

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Rapports d'organisations sur le sujet "Chemical Induced Toxicity"

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Lawanprasert, Somsong, Chaiyo Chaichantipyuth, Supatra Srichairat, Nuansri Niwattisaiwong, and Laddawal Phivthong-ngam. Subchronic exposure of Pueraria mirifica in normal - and high cholesterol diet fed rats : influence on hepatic cytochrome P450, lipid profile and toxicity. Chulalongkorn University, 2004. https://doi.org/10.58837/chula.res.2004.28.

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Pueraria mirifica airy shaw and suvatabandhu, know locally as Kwao Keur, is a plant in family Leguminosae. In this study, effects of P.mirifica on hepatic cytochrome P450 (CYP), serum lipid profile and subchronic toxicity were investigated in male Wistar rats. Rats were randomly divided into four treatment groups as following: normal diet-fed group; normal diet-fed supplemented with P.mirifica group; high cholesterol diet-fed group; high cholesterol diet-fed supplemented with P.mirifica group. Each group comprised 10 rats. P.mirifica was administered orally at a dosage of 100 mg/kg/day for 90

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