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Livres sur le sujet « Chromatic system »

Auteur : Grafiati

Publié le 10 mai 2025

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1

Emili, Andrew, Jack Greenblatt et Shoshana Wodak, dir. Systems Analysis of Chromatin-Related Protein Complexes in Cancer. New York, NY : Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-7931-4.

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2

Arthur, M. Sackler Colloquia of the National Academy of Sciences (2002 Washington D. C. ). Self-perpetuating structural states in biology, disease, and genetics. Washington, D.C : National Academy of Sciences, 2002.

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3

E, Vance Dennis, et Vance Jean E, dir. Biochemistry of lipids, lipoproteins, and membranes. Amsterdam : Elsevier, 1991.

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4

Pinna, Baingio. On the Watercolor Illusion. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780199794607.003.0057.

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The watercolor illusion is a long-range assimilative spread of color emanating from a thin colored line running contiguous to a darker chromatic contour and imparting a figure-ground effect across a large area. The watercolored figure appears evenly colored by an opaque light veil of chromatic tint (coloration effect), with a clear surface color property spreading from the lighter edges. At the same time, the watercolored figure manifests a strong figure-ground organization and a solid figural appearance comparable to a rounded surface segregated in depth which extends out from the flat surface. The complementary region appears as a hole or empty space. The phenomenal properties of coloration and figure-ground effects and their relationship are described and demonstrated. The watercolor illusion and its main effects are discussed in the light of parallel mechanisms. Boundary and surface dynamics are processed by the boundary contour system and by the feature contour system.

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Hamburger, Kai, Thorsten Hansen et Karl R. Gegenfurtner. Geometric-Optical Illusions Under Isoluminance ? Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780199794607.003.0018.

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This chapter briefly introduces nine classical geometric-optical illusions. These include the Delboeuf illusion, the Ebbinghaus illusion, the Judd illusion, the Müller-Lyer illusion, the Ponzo illusion, the vertical illusion, the Hering illusion, the Poggendorff illusion, and the Zoellner illusion. It then demonstrates that they persist under different luminance conditions and under isoluminance. The empirical findings show that our conscious percept is similarly affected by luminance conditions and isoluminance, suggesting that joint contour processing (chromatic and luminance) may extend well beyond early visual areas. The chapter further discusses these concepts in terms of the magnocellular system, the parvocellular system, and the koniocellular system.

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6

Binda, Olivier. Chromatin Signaling and Neurological Disorders. Elsevier Science & Technology Books, 2019.

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7

Binda, Olivier. Chromatin Signaling and Neurological Disorders. Elsevier Science & Technology, 2019.

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8

Anstis, Stuart. Color and Luminance. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780199794607.003.0038.

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Color and luminance interact in many ways in the human visual system. For instance, the colors in an afterimage, which are due to adaptation of retinal cones, are especially vivid when test contours, presented after the adapting image, coincide with the blurred edges of the afterimage. A single colored adapting pattern can give rise to two differently colored afterimages, according to the position of black lines in the test field. This shows that colors seen by the low-acuity chromatic pathways will diffuse outward along, but not across, luminance contours. This is also true for real colors. Finally, flicker-augmented contrast shows that the visual system, when given a choice, will select the most salient color/luminance borders in a stimulus.

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9

Pattenden, Samantha. Analysis of chromatin remodeling in an IFN-[gamma] responsive system. 2003.

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10

Emili, Andrew, Jack Greenblatt et Shoshana Wodak. Systems Analysis of Chromatin-Related Protein Complexes in Cancer. Springer London, Limited, 2013.

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11

Emili, Andrew, Jack Greenblatt et Shoshana Wodak. Systems Analysis of Chromatin-Related Protein Complexes in Cancer. Springer, 2015.

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12

Emili, Andrew, Jack Greenblatt et Shoshana Wodak. Systems Analysis of Chromatin-Related Protein Complexes in Cancer. Springer, 2013.

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13

Georgel, Philippe. Effects of nucleosomes on transcription by polymerase I in a reconstituted system. 1993.

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14

van der Vlag, Johan, et Jo H. M. Berden. The patient with systemic lupus erythematosus. Sous la direction de Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient is diagnosed with SLE. Apoptotic debris is present in the extracellular matrix and circulation of patients with SLE due to an aberrant process of apoptosis and/or insufficient clearance of apoptotic cells and apoptotic debris. The non-cleared apoptotic debris in patients with SLE may lead to activation of both the innate (myeloid and plasmacytoid dendritic cells) and adaptive (T and B cells) immune system. In addition to the activation by apoptotic debris and immune complexes, the immune system in SLE may be deregulated at the level of (a) presentation of self-peptides by antigen-presenting cells, (b) selection processes for both B and T cells, and (c) regulatory processes of B- and T-cell responses. Lupus nephritis may be classified in different classes based on histological findings in renal biopsies. The chromatin-containing immune complexes deposit in the capillary filter, most likely due to the interaction of chromatin with the polysaccharide heparan sulphate. A decreased renal expression of the endonuclease DNaseI further contributes to the glomerular persistence of chromatin and the development of glomerulonephritis.Current treatment of lupus nephritis is not specific and aims to reduce the inflammatory response with general immunosuppressive therapies. However, research has revealed novel potential therapeutic candidates at the level of dendritic cells, B cells, and T cells.

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15

Systems Analysis Of Chromatinrelated Protein Complexes In Cancer. Springer-Verlag New York Inc., 2013.

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16

Sacks, Oliver W. to nisi ton tyflon sta chromata. Agra, 2013.

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17

Gay, Steffen, et Michel Neidhart. Epigenetics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0039.

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In higher eukaryotic organisms epigenetic modifications are crucial for proper chromatin folding and thereby proper regulation of gene expression. Epigenetics include DNA methylation, histone modifications, and microRNAs. First described in tumors, the involvement of aberrant epigenetic modifications has been reported also in other diseases, i.e. metabolic, psychiatric, inflammatory, and autoimmune. Deregulation of epigenetic mechanisms occurred in patients with rheumatoid arthritis, systemic lupus erythematosus, and scleroderma. Many questions remain: e.g. what is the cause of these epigenetic changes and how can we interfere in the pathological process? Here we discuss whether supplementation with methyl donors could represent a novel therapeutic concept for such diseases.

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