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Littérature scientifique sur le sujet « CXCR2 receptor »

Auteur : Grafiati

Publié le 9 mars 2023

Mis à jour le 31 juillet 2025

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Articles de revues sur le sujet "CXCR2 receptor"

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Hou, Zhi-Shuai, Hong-Kui Zhao, Pedro Perdiguero, et al. "Pleiotropic Role of Rainbow Trout CXCRs in Response to Disease and Environment: Insights from Transcriptional Signatures and Structure Analysis." Biomolecules 14, no. 3 (2024): 337. http://dx.doi.org/10.3390/biom14030337.

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Chemokines are cytokines with chemoattractant capacities that exert their physiological functions through the binding of chemokine receptors. Thus, chemokine and receptor complexes exert important roles in regulating development and homeostasis during routine immune surveillance and inflammation. Compared to mammals, the physiology and structure of chemokine receptors in fish have not been systematically studied. Furthermore, the salmonid-specific whole genome duplication has significantly increased the number of functional paralogs of chemokine receptors. In this context, in the current study

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Zhang, Jing, Shouguo Huang, Lini Quan, et al. "Determination of Potential Therapeutic Targets and Prognostic Markers of Ovarian Cancer by Bioinformatics Analysis." BioMed Research International 2021 (March 19, 2021): 1–13. http://dx.doi.org/10.1155/2021/8883800.

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This study is to study the expression of CXCRs in ovarian cancer tissues and their value in prognosis. The expressions of CXCR1-CXCR7 mRNA between ovarian tumor tissues and normal tissues and in different pathological types of ovarian tumor tissues were compared by ONCOMINE online tool. The relationship between the expression of CXCRs and clinical pathological staging was studied by GEPIA. Kaplan-Meier plotter online tool was used to analyze prognosis. Finally, GO and KEGG analyses and protein interaction network analysis were performed for CXCRs by the DAVID software to predict their function

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Korbecki, Jan, Klaudyna Kojder, Patrycja Kapczuk, et al. "The Effect of Hypoxia on the Expression of CXC Chemokines and CXC Chemokine Receptors—A Review of Literature." International Journal of Molecular Sciences 22, no. 2 (2021): 843. http://dx.doi.org/10.3390/ijms22020843.

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Hypoxia is an integral component of the tumor microenvironment. Either as chronic or cycling hypoxia, it exerts a similar effect on cancer processes by activating hypoxia-inducible factor-1 (HIF-1) and nuclear factor (NF-κB), with cycling hypoxia showing a stronger proinflammatory influence. One of the systems affected by hypoxia is the CXC chemokine system. This paper reviews all available information on hypoxia-induced changes in the expression of all CXC chemokines (CXCL1, CXCL2, CXCL3, CXCL4, CXCL5, CXCL6, CXCL7, CXCL8 (IL-8), CXCL9, CXCL10, CXCL11, CXCL12 (SDF-1), CXCL13, CXCL14, CXCL15,

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Konrad, F. M., and J. Reutershan. "CXCR2 in Acute Lung Injury." Mediators of Inflammation 2012 (2012): 1–8. http://dx.doi.org/10.1155/2012/740987.

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In pulmonary inflammation, recruitment of circulating polymorphonuclear leukocytes is essential for host defense and initiates the following specific immune response. One pathological hallmark of acute lung injury and acute respiratory distress syndrome is the uncontrolled transmigration of neutrophils into the lung interstitium and alveolar space. Thereby, the extravasation of leukocytes from the vascular system into the tissue is induced by chemokines that are released from the site of inflammation. The most relevant chemokine receptors of neutrophils are CXC chemokine receptor (CXCR) 1 and

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Daniele, Simona, Simona Saporiti, Stefano Capaldi, et al. "Functional Heterodimerization between the G Protein-Coupled Receptor GPR17 and the Chemokine Receptors 2 and 4: New Evidence." International Journal of Molecular Sciences 24, no. 1 (2022): 261. http://dx.doi.org/10.3390/ijms24010261.

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GPR17, a G protein-coupled receptor, is a pivotal regulator of myelination. Its endogenous ligands trigger receptor desensitization and downregulation allowing oligodendrocyte terminal maturation. In addition to its endogenous agonists, GPR17 could be promiscuously activated by pro-inflammatory oxysterols and chemokines released at demyelinating lesions. Herein, the chemokine receptors CXCR2 and CXCR4 were selected to perform both in silico modelling and in vitro experiments to establish their structural and functional interactions with GPR17. The relative propensity of GPR17 and CXCR2 or CXCR

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Uhl, Barbara, Katharina T. Prochazka, Katrin Pansy, et al. "Distinct Chemokine Receptor Expression Profiles in De Novo DLBCL, Transformed Follicular Lymphoma, Richter’s Trans-Formed DLBCL and Germinal Center B-Cells." International Journal of Molecular Sciences 23, no. 14 (2022): 7874. http://dx.doi.org/10.3390/ijms23147874.

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Chemokine receptors and their ligands have been identified as playing an important role in the development of diffuse large B-cell lymphoma (DLBCL), follicular lymphoma, and Richter syndrome (RS). Our aim was to investigate the different expression profiles in de novo DLBCL, transformed follicular lymphoma (tFL), and RS. Here, we profiled the mRNA expression levels of 18 chemokine receptors (CCR1–CCR9, CXCR1–CXCR7, CX3CR1 and XCR1) using RQ-PCR, as well as immunohistochemistry of seven chemokine receptors (CCR1, CCR4–CCR8 and CXCR2) in RS, de novo DLBCL, and tFL biopsy-derived tissues. Tonsil-

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Korbecki, Jan, Patrycja Kupnicka, Katarzyna Barczak, et al. "The Role of CXCR1, CXCR2, CXCR3, CXCR5, and CXCR6 Ligands in Molecular Cancer Processes and Clinical Aspects of Acute Myeloid Leukemia (AML)." Cancers 15, no. 18 (2023): 4555. http://dx.doi.org/10.3390/cancers15184555.

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Acute myeloid leukemia (AML) is a type of leukemia known for its unfavorable prognoses, prompting research efforts to discover new therapeutic targets. One area of investigation involves examining extracellular factors, particularly CXC chemokines. While CXCL12 (SDF-1) and its receptor CXCR4 have been extensively studied, research on other CXC chemokine axes in AML is less developed. This study aims to bridge that gap by providing an overview of the significance of CXC chemokines other than CXCL12 (CXCR1, CXCR2, CXCR3, CXCR5, and CXCR6 ligands and CXCL14 and CXCL17) in AML’s oncogenic processe

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Coperchini, Francesca, Laura Croce, Michele Marinò, Luca Chiovato, and Mario Rotondi. "Role of chemokine receptors in thyroid cancer and immunotherapy." Endocrine-Related Cancer 26, no. 8 (2019): R465—R478. http://dx.doi.org/10.1530/erc-19-0163.

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Inflammation is currently regarded as an essential component of malignancies. It is now known that the tumor microenvironment may profoundly influence the biological behavior of cancer cells and ultimately the patient’s outcome. Chemokine and their receptor play a major role in determining the immune phenotype of the cells infiltrating the thyroid tumor microenvironment. Experimental evidence shows that both normal and cancer thyroid cells express specific chemokine receptors. The expression of at least some of these receptors exerts several biological effects, which influence the course of th

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Richardson, Micheler, Timothy Adekoya, Nikia Smith, and Parag Kothari. "Opposite effects of CXCR1 and CXCR2 overexpression in prostate tumorigenesis." Journal of Immunology 208, no. 1_Supplement (2022): 178.12. http://dx.doi.org/10.4049/jimmunol.208.supp.178.12.

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Abstract Chemokines and their receptors play important role in tumor progression and metastasis. In prostate cancer (PCa), some members of the CXC chemokine receptor have been shown to enhance angiogenesis, proliferation and metastasis. In this study we assess the roles of the interleukin-8 (IL-8/CXCL8) receptors CXCR1 and CXCR2 in prostate tumorigenesis, using MDA PCa 2b and LNCaP cell lines. Our results show that overexpression of CXCR2 enhanced in-vitro cell proliferation, soft agar growth and in-vivo tumor xenograft in nude mice, whereas overexpression of CXCR1 inhibited cell proliferation

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Yildirim, Sedat, Frank Bautz, Andreas M. Boehmler, Lothar Kanz, and Robert Möhle. "Regulation of CXCR1, CXCR2 and CXCR4 in Human Neutrophils: Potential Role in the Release from the Bone Marrow, Clearance of Senescent Cells, and Cell Function at Sites of Inflammation." Blood 106, no. 11 (2005): 3068. http://dx.doi.org/10.1182/blood.v106.11.3068.3068.

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Abstract In the mouse model, it has been shown that the interleukin-8 (IL-8) receptor CXCR2 is involved in the release of mature neutrophils from the bone marrow into the circulation. When neutrophils age, upregulation of CXCR4 and downmodulation of CXCR2 result in homing and subsequent sequestration of senescent cells in the bone marrow. In our study, we observed a similar time-dependent (starting at 3 hrs., maximum at 12–18 hrs.) downregulation of CXCR2 in human neutrophils during aging in ex vivo culture, while expression of the second IL-8 receptor CXCR1, which is mainly responsible for th

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Thèses sur le sujet "CXCR2 receptor"

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Williams, Mark Anthony. "DISPARATE REGULATION OF NEUTROPHIL PRO-INFLAMMATORY FUNCTIONING BY CXCR2-SELECTIVE CHEMOKINES." University of Cincinnati / OhioLINK, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=ucin971879221.

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RACCOSTA, LAURA. "Tumour-released Liver X Receptor ligands attract tumour promoting neutrophils in a CXCR2 dependent manner." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2012. http://hdl.handle.net/10281/28479.

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Tumour formation is the result of molecular alterations involving cellular regulators as well as the ability of tumor cells to affect the tumor microenvironment through smoldering inflammation, or even taking advantage of inflammation to grow and metastasize. Tumour microenvironment is composed of various cell types, among them neutrophils are recognized as playing an important pro-tumorigenic role, by promoting neoangiogenesis and/or by suppressing antitumor immune responses. We have recently shown that ligands of liver X receptors (LXRs), which are involved in cholesterol homeostasis and in

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Padovani-Claudio, Dolly Ann. "Functional analysis of the chemokine receptor Cxcr2 in the normal and demyelinated adult central nervous system." Connect to text online, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1152193193.

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Padovani-Claudio, Dolly Ann. "FUNCTIONAL ANALYSES OF THE CHEMOKINE RECEPTOR CXCR2 IN THE NORMAL AND DEMYELINATED ADULT CENTRAL NERVOUS SYSTEM." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1152193193.

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Russo, Remo de Castro. "Efeito da administração do antagonista de receptor CXCR2 no modelo de fibrose pulmonar induzida por bleomicina." Universidade Federal de Minas Gerais, 2005. http://hdl.handle.net/1843/UCSD-8FTN2Z.

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Pulmonary fibrosis is a disease characterized by progressive interstitial collagen deposition, which causes changes in the normal lung architecture and loss of function, which could lead to death. Acute pulmonary inflammatory processes and their chronification are associated with fibrotic phenomena acting as triggering events. The inflammation that precedes the emergence of pulmonary fibrosis is characterized by an increased cell influx, which culminates in the liberation of inflammatory mediators, which perpetuate the initial lesion. Therefore it is likely that the inhibition of the inflammat

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Wilson, Shirley Risk. "Oligomerisation of chemokine receptors CXCR1 and CXCR2." Thesis, University of Glasgow, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418346.

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Trevelin, Silvia Cellone. "Papel do receptor toll-like 9 na falência de migração dos neutrófilos na sepse." Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/17/17133/tde-14082013-055722/.

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O recrutamento de neutrófilos para o sítio da infecção é um evento crucial para o combate aos microrganismos e sobrevivência na sepse. A migração destes polimorfonucleares é dirigida através de um gradiente quimiotático por meio do reconhecimento de quimiocinas por receptores acoplados a proteína G (GPCRs), os quais são regulados por quinases específicas (GRKs). Estudos prévios demonstraram que na sepse ocorre uma falência na migração de neutrófilos para o foco infeccioso em função da dessensibilização de receptores quimiotáticos via GRKs induzida pela ativação de receptores toll-like (TLRs),

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Kiss, Debra Lois. "Regulation of the Chemokine Receptors CXCR4, CXCR7 , and the Androgen Receptor in Prostate Cancer." Thesis, Griffith University, 2013. http://hdl.handle.net/10072/367690.

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The chemokine receptor CXCR4 contributes to tumour cell migration and invasion during the progression of prostate cancer. In particular, this pathway is central to the metastasis of prostate cancer to the bone marrow. Limited therapeutic options exist for prostate cancer patients who have progressed to advanced metastatic disease, and pharmacological interference of the chemokine network may serve to control tumour cell dissemination and the establishment of metastasis. A more detailed knowledge of the mechanisms regulating chemokine receptors is required, in order to further characterise and

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Bento, Allisson Freire. "Efeito do SB225002, antagonista seletivo do receptor para quimiocinas CXCR2, no modelo de colite induzida pelo ácido 2,4,6-trinitrobenzeno sulfônico (TNBS) em camundongos." reponame:Repositório Institucional da UFSC, 2012. http://repositorio.ufsc.br/xmlui/handle/123456789/92097.

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Dissertação (mestrado) - Universidade Federal de Santa Catarina, Centro de Ciências Biológicas, Programa de Pos-Graduação em Farmacologia, Florianópolis, 2008.<br>Made available in DSpace on 2012-10-24T05:44:12Z (GMT). No. of bitstreams: 1 271599.pdf: 9768628 bytes, checksum: ac3442843a34970b9d196ca07b9b8959 (MD5)<br>Os neutrófilos são células importantes para a eliminação de patógenos, no entanto, o recrutamento excessivo dessas células pode levar a lesão tecidual. Essa migração é mediada pelas quimiocinas CXC, e seus receptores, CXCR1 e CXCR2 presentes nos neutrófilos. Dessa forma, a redução

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Khurram, Syed Ali. "The chemokine receptors XCR1, CXCR1 and CXCR2 regulate oral epithelial cell behaviour." Thesis, University of Sheffield, 2008. http://etheses.whiterose.ac.uk/10311/.

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Chemokines are chemoattractant cytokines which act on specific receptors and play an important role in tumour biology. The aim of this project was to determine whether the chemokine receptors XCRl, CXCRl and CXCR2 and their respective ligands lymphotactin, IL-8 (CXCRl&2) and GRO-a regulate the behaviour of normal and malignant oral epithelial cells. XCRl, CXCRl and CXCR2 mRNA and surface protein expression was detected in normal and oral cancer cell lines. Lymphotactin, IL-8 and GRO-a facilitated intracellular activation of ERK1/2 signaling pathway and stimulated migration, invasion and prolif

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Livres sur le sujet "CXCR2 receptor"

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Fruehauf, Stefan, W. Jens Zeller, and Gary Calandra. Novel Developments in Stem Cell Mobilization: Focus on CXCR4. Springer, 2014.

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Fruehauf, Stefan, W. Jens Zeller, and Gary Calandra. Novel Developments in Stem Cell Mobilization: Focus on CXCR4. Springer, 2012.

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Livingston, Schuyler, Benjamin Young, Martin Markowitz, Poonam Mathur, and Bruce L. Gilliam. HIV Virology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0017.

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HIV is a member of the lentivirus subfamily of retroviruses. Two distinct groups of viruses are pathogenic in humans: HIV-1 and HIV-2. Both are transmitted sexually and known to cause immunodeficiency disease. HIV enters the cell through use of the CD4 receptor and chemokine co-receptors, primarily CCR5 and CXCR4. The viral genome is transcribed from RNA to DNA by reverse transcriptase and integrated into the host genome by integrase. The HIV genome encodes 15 proteins, comprising three categories: structural, regulatory, and accessory. After budding from the host cell, the virus matures into

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Noels, Heidi, and Jürgen Bernhagen, eds. The CXCR4 Ligand/Receptor Family and the DPP4 Protease in High-Risk Cardiovascular Patients. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-858-0.

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Novel Developments In Stem Cell Mobilization Focus On Cxcr4. Springer, 2012.

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Chapitres de livres sur le sujet "CXCR2 receptor"

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Sarau, Henry M., Katherine L. Widdowson, Michael R. Palovich, John R. White, David C. Underwood/surname, and Don E. Griswold. "Interleukin-8 Receptor (CXCR2) Antagonists." In New Drugs for Asthma, Allergy and COPD. KARGER, 2001. http://dx.doi.org/10.1159/000062157.

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Lam, Clarissa, Mahmud Arif Pavel, Parul Kashyap, Zahra Salehi-Najafabadi, Victoria Valentino, and Yong Yu. "Detection of CXCR2 Cytokine Receptor Surface Expression Using Immunofluorescence." In Cytokine Bioassays. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0928-5_17.

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Blay, Jonathan. "Chemokine Receptor CXCR4." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_1067-3.

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Blay, Jonathan. "Chemokine Receptor CXCR4." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-46875-3_1067.

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Furusato, Bungo, and Johng S. Rhim. "CXCR4 and Cancer." In Chemokine Receptors in Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-267-4_2.

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Baljinder, Singh, Watts Ankit, Amit Singh Shekhawat, et al. "CXCR4 Theranostics: A Potential Game Changer in Solid Tumors and Hematological Malignancies." In Beyond Becquerel and Biology to Precision Radiomolecular Oncology: Festschrift in Honor of Richard P. Baum. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-33533-4_31.

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AbstractAn overexpression of CXCR4 receptors is reported in at least 30 different human solid tumors and hematological malignancies. This overexpression is often associated with tumor aggressiveness, increased risk of metastasis, and a higher probability of recurrence, which in turn leads to a poor prognosis. No in vivo method suitable for whole-body CXCR4 disease quantification has been described and this unmet clinical need or the scientific question has been reported recently. 68Ga-Pentixafor which is a CXCR4 targeting high-affinity PET imaging probe and the tracer has been evaluated in mul

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Li, Yanchun, and Amy M. Fulton. "The CXCR3/CXCL3 Axis in Cancer." In Chemokine Receptors in Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-267-4_5.

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Wijtmans, Maikel, Iwan J. P. de Esch, and Rob Leurs. "Therapeutic Targeting of the CXCR3 Receptor." In Methods and Principles in Medicinal Chemistry. Wiley-VCH Verlag GmbH & Co. KGaA, 2011. http://dx.doi.org/10.1002/9783527631995.ch13.

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Woodard, Lauren E., and Sridhar Nimmagadda. "Molecular Imaging of CXCR4 Receptor Expression in Tumors." In Novel Developments in Stem Cell Mobilization. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-1960-0_21.

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Calì, Corrado, Julie Marchaland, Osvaldo Mirante, and Paola Bezzi. "Chemokines as Neuromodulators: Regulation of Glutamatergic Transmission by CXCR4-Mediated Glutamate Release From Astrocytes." In Chemokine Receptors and NeuroAIDS. Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0793-6_12.

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Actes de conférences sur le sujet "CXCR2 receptor"

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Sharma, Bhawna, Dhananjay M. Nawandar, Michelle L. Varney, and Rakesh K. Singh. "Abstract 693: Evaluating the role of CXCR2 receptor and its ligand in breast cancer therapy resistance." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-693.

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Wade, R. C., D. Xing, V. Lin, et al. "Inflammatory Ligands of CXC Chemokine Receptor 2 (CXCR2) Are Associated with Coronary Artery Calcification in COPD." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2401.

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Sharma, Bhawna, Dhananjay Nawandar, Michelle L. Varney, Kalyan C. Nannuru, and Rakesh K. Singh. "Abstract 5462: Enhancing efficacy of drugs by targeting CXCR2 receptor signaling for the treatment of malignant breast cancer." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5462.

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Dong, Yuanlin, Syeda M. Kabir, Eunsook Lee, and Deok-Soo Son. "Abstract 527: Proinflammatory chemokine receptor CXCR2 promotes cellular proliferation through suppression of cell cycle inhibitor p21 protein in ovarian cancer." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-527.

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Ustach, Carolyn V., Aprill Watanabe, Meraj Aziz, et al. "Abstract 393: The chemokine receptor, CXCR2/IL8RB, contributes to the survival of pancreatic adenocarcinoma, and may play a role in stroma-tumor communication." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-393.

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Adekoya, Timothy O., Nikia Smith, Parag Kothari, and Ricardo M. Richardson. "Abstract PO-134: Differential effects of CXCR1 and CXCR2 receptors on prostate tumorigenesis." In Abstracts: AACR Virtual Conference: 14th AACR Conference on the Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; October 6-8, 2021. American Association for Cancer Research, 2022. http://dx.doi.org/10.1158/1538-7755.disp21-po-134.

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Silva, Mariane Ricciardi da, Nádia Calvo Martins Okuyama, and Karen Brajão De Oliveira. "PAPEL DAS VARIANTES GENÉTICAS DE CXCL12 (RS1801157) E DE CXCR4 (RS2228014) NA EXPRESSÃO PROTEICA DO RECEPTOR E EM PARÂMETROS CLINICOPATOLÓGICOS DO CÂNCER DE COLO DE ÚTERO." In II Congresso Brasileiro de Saúde On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1519.

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Introdução: O câncer cervical é o terceiro câncer mais comum em mulheres no mundo e a inflamação é um componente crucial na progressão do tumor, mas outros cofatores devem estar presentes para o desenvolvimento da malignidade, como fatores genéticos individuais. Nesse contexto, os genes CXCL12 e CXCR4 podem ter uma variação de nucleotídeo único (SNV) rs1801157 e rs2228014, respectivamente, que estão envolvidas na sobrevivência, angiogênese e invasão de células malignas. Objetivos: O objetivo do presente estudo foi verificar uma possível associação entre SNVs de CXCL12 e CXCR4 e sua influência

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Pham, Kien, Che Liu, Defang Luo, Brent A. Reynolds, and Jeffrey K. Harrison. "Abstract 5194: Heterogenous expression of chemokine receptors in primary patient-derived GBM lines; association of CXCR3, CXCR4, and CXCR7 with a slow cycling sub-population." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5194.

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Miekus, Katarzyna, Danuta Jarocha, Elzbieta Trzyna, and Marcin Majka. "Abstract B113: Role of I‐TAC‐binding receptors CXCR3 and CXCR7 in biology of various tumor cell lines." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-b113.

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Costa, Leonardo, Jürgen Haas, Henriette Rudolph, et al. "The Choroid Plexus Is Permissive for a Preactivated Antigen-Experienced Memory B Cell Subset in Multiple Sclerosis." In Building Bridges in Medical Science 2021. Cambridge Medicine Journal, 2021. http://dx.doi.org/10.7244/cmj.2021.03.001.2.

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Background: The role of B cells in multiple sclerosis (MS) is increasingly recognized. B cells undergo compartmentalized redistribution in blood and cerebrospinal fluid (CSF) during active MS, whereby memory B cells accumulate in the CSF. While B-cell trafficking across the blood– brain barrier has been intensely investigated, cellular diapedesis through the blood–CSF barrier (BCSFB) is incompletely understood. Objectives: To investigate how B cells interact with the choroid plexus to transmigrate into the CSF, we isolated circulating B cells from healthy donors (HC) and MS patients, utilized

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