Se connecter

Bibliographies thématiques / Cytoplazma / Livres

Pour voir les autres types de publications sur ce sujet consultez le lien suivant : Cytoplazma.

Livres sur le sujet « Cytoplazma »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 1 février 2022

Créez une référence correcte selon les styles APA, MLA, Chicago, Harvard et plusieurs autres

Choisissez une source :

Consultez les 42 meilleurs livres pour votre recherche sur le sujet « Cytoplazma ».

À côté de chaque source dans la liste de références il y a un bouton « Ajouter à la bibliographie ». Cliquez sur ce bouton, et nous générerons automatiquement la référence bibliographique pour la source choisie selon votre style de citation préféré : APA, MLA, Harvard, Vancouver, Chicago, etc.

Vous pouvez aussi télécharger le texte intégral de la publication scolaire au format pdf et consulter son résumé en ligne lorsque ces informations sont inclues dans les métadonnées.

Parcourez les livres sur diverses disciplines et organisez correctement votre bibliographie.

1

Agutter, Paul S. Between nucleus and cytoplasm. London : Chapman and Hall, 1991.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

2

Milani, M. Differential two colour x-ray radiobiology of membrane / cytoplasm yeast cells. Chilton : Rutherford Appleton Laboratory, 1998.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

3

Schmid, Hans-Peter. Identifizierung und Charakterisierung einer neuen Klasse von Ribonukleoprotein-Partikeln aus dem Cytoplasma der eukaryontischen Zelle : Prosomen. Stuttgart : Biologisches Institut der Universität Stuttgart, 1987.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

4

Ed, Stadelmann, Bushnell William R et Bushnell Ann H, dir. Pathology of the plant cell. Karachi, Pakistan : Saad Publications, 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

5

The secretory and endocytic paths : Mechanism and specificity of vesicular traffic in the cell cytoplasm. New York : Wiley, 1987.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

6

Conrad, Gary W. Effects of silver and other metals on the cytoskeleton : Summary of research : 02/15/95-06/30/97. [Washington, DC : National Aeronautics and Space Administration, 1997.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

7

Tse, Wai Yee. Neutrophil Fc[gamma] receptor polymorphisms and pathogenetic mechanisms of neutrophil activation in anti-neutrophil cytoplasm (ANCA)-associated vasculitis. Birmingham : University of Birmingham, 1999.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

8

Hargrove, T. R. Changes in rice breeding in 10 Asian countries : 1965-84 : diffusion of genetic materials, breeding objectives, and cytoplasm. Manila : International Rice Research Institute, 1985.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

9

Schründer, Jürgen. Cytoplasmatische Vektoren zur heterologen Genexpression bei Hefen : Konstruktion und Analyse linearer Hybridplasmide basierend auf dem Killer-System der Hefe Kluyveromyces lactis. Berlin : J. Cramer, 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

10

Bacterial growth and division : Biochemistry and regulation of prokaryotic and eukaryotic division cycles. San Diego : Academic Press, 1991.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

11

M, Malacinski George, dir. Cytoplasmic organization systems. New York : McGraw-Hill, 1990.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

12

The Functioning Cytoplasm. Springer, 2012.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

13

Frey-Wyssling, Albert. Comparative Organellography of the Cytoplasm. Springer, 2012.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

14

Harry, Walter, Brooks Donald E et Srere Paul A, dir. Microcompartmentation and phase separation in cytoplasm. San Diego, Calif : Academic Press, 2000.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

15

Microcompartmentation and Phase Separation in Cytoplasm. Elsevier, 1999. http://dx.doi.org/10.1016/s0074-7696(08)x6044-9.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

16

Allen, Geoffrey. Proteins : Soluble Proteins of the Cytoplasm (Proteins). JAI Press, 1997.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

17

Ph, Jeanteur, dir. Cytoplasmic fate of messenger RNA. Berlin : Springer, 1997.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

18

1943-, Peters R., dir. Nucleo-Cytoplasmic transport. London : Academic Press, 1988.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

19

1943-, Peters R., et Trendelenburg M, dir. Nucleocytoplasmic transport. Berlin : Springer-Verlag, 1986.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

20

Dejesus, Carl, et Lourdes Trask. Chloroplasts and Cytoplasm : Structure and Functions : Life Sciences / Biology / Cell Biology. Nova Science Publishers, Incorporated, 2018.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

21

Carton, James. Infectious diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0002.

Texte intégral

Résumé :

Microbes 16Antimicrobial agents 17Human immunodeficiency virus 18Tuberculosis 20Infectious mononucleosis 21Malaria 22Syphilis 24Lyme disease 25Leishmaniasis 26• Single-celled organisms with their double-stranded DNA lying free in cytoplasm surrounded by a cell membrane and cell wall.• Most grow in air (aerobes), but can grow without it (facultative anaerobes). Some only grow in the absence of oxygen (strict anaerobes)....

Styles APA, Harvard, Vancouver, ISO, etc.

22

Amos, Linda A., et Jan Löwe. Prokaryotic Cytoskeletons : Filamentous Protein Polymers Active in the Cytoplasm of Bacterial and Archaeal Cells. Springer, 2017.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

23

Physiology of rubber tree latex : The laticiferous cell and latex : a model of cytoplasm. Boca Raton, Fla : CRC Press, 1989.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

24

Ekiz, Hasan. Nuclear X cytoplasm genetic interactions controlling anther culture response in wheat (Triticum aestivum L.). 1990.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

25

Physiology of Rubber Tree Latex The Laticiferous Cell and Latex- A Modell of Cytoplasm. CRC, 1988.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

26

Amos, Linda A., et Jan Löwe. Prokaryotic Cytoskeletons : Filamentous Protein Polymers Active in the Cytoplasm of Bacterial and Archaeal Cells. Springer, 2018.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

27

Bagni, Claudia, et Eric Klann. Molecular Functions of the Mammalian Fragile X Mental Retardation Protein : Insights Into Mental Retardation and Synaptic Plasticity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0008.

Texte intégral

Résumé :

Chapter 8 discusses how Fragile X syndrome (FXS) is caused by the absence of the RNA-binding protein fragile X mental retardation protein (FMRP). FMRP is highly expressed in the brain and gonads, the two organs mainly affected in patients with the syndrome. Functionally, FMRP belongs to the family of RNA-binding proteins, shuttling from the nucleus to the cytoplasm, and, as shown for other RNA-binding proteins, forms large messenger ribonucleoparticles.

Styles APA, Harvard, Vancouver, ISO, etc.

28

Leeds, Janet M. DNA precursor compartmentation in mammalian cells : Distribution and rates of equilibration between nucleus and cytoplasm. 1986.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

29

(Editor), Harry Walter, Donald E. Brooks (Editor), Paul A. Srere (Editor) et Kwang W. Jeon (Series Editor), dir. Microcompartmentation and Phase Separation in Cytoplasm (International Review of Cytology, Volume 192) (International Review of Cytology). Academic Press, 1999.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

30

Salama, Alan D. The patient with vasculitis. Sous la direction de Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0159_update_001.

Texte intégral

Résumé :

Primary systemic vasculitis frequently leads to renal involvement and is responsible for significant numbers of patients progressing to end-stage renal disease. Frequently this is due to small vessel vasculitis, in association with antineutrophil cytoplasm antibody, which requires prompt recognition and timely therapeutic intervention to optimize renal and patient outcomes. Other organ systems are often affected. Relapses occur in about 50%.Less commonly medium or larger vessel vasculitis may involve the kidneys and through ischaemia lead to impaired renal function and renovascular hypertension, as in Takayasu’s or Kawasaki disease, and polyarteritis nodosa (PAN).

Styles APA, Harvard, Vancouver, ISO, etc.

31

Xue, Feiyu. MOLECULAR ANALYSIS OF KELCH, A GENE NECESSARY FOR NURSE CELL CYTOPLASM TRANSPORT IN DROSOPHILA OOGENESIS ( KELCH GENE). 1992.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

32

M, Tazawa, dir. Cell dynamics. Wien : Springer-Verlag, 1989.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

33

Peter, Satir, Condeelis John S, Lazarides Elias, University of California, Los Angeles., Genentech Inc et UCLA Symposium on Signal Tranduction in Cytoplasmic Organization and Cell Motility (1987 : Lake Tahoe, Calif.), dir. Signal transduction in cytoplasmic organization and cell motility : Proceedings of a UCLA Symposium held in Lake Tahoe, California, February 15-21, 1987. New York : Liss, 1988.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

34

Mason, Peggy. Synthesis, Packaging, and Termination of Neurotransmitters. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0012.

Texte intégral

Résumé :

The synthesis, packaging, and termination of action of neurotransmitters are detailed. There are far more varieties of peptide neurotransmitters than there are of low-molecular-weight neurotransmitters. Yet low-molecular-weight neurotransmitters are the ubiquitous workhorses of the nervous system. Acetylcholine, the catecholamines norepinephrine and dopamine, serotonin, glutamate, and GABA are examined in some depth. The vesicular transporters that carry low-molecular-weight neurotransmitters from the cytoplasm into synaptic vesicles are covered. The role of monoamines in affect and mood and the psychotropic effects of monoaminergic drugs are discussed. Principles of catecholamine synthesis are applied to understand phenylketonuria. Uptake of monoamines into neurons is discussed in the context of amphetamine, cocaine, and other drugs of abuse. Stiff-person syndrome, which results from an impairment of GABA synthesis, is introduced. The modes of action for peptide and gaseous neurotransmitters are briefly covered.

Styles APA, Harvard, Vancouver, ISO, etc.

35

Sedel, Frédéric, et Carla E. M. Hollak. Disorders of Thiamine Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0028.

Texte intégral

Résumé :

Thiamine is a water-soluble vitamin acting in the mitochondria as a cofactor for energy metabolism and, in the cytoplasm, in the pentose phosphate biosynthetic pathway. Its transport through the plasma membrane requires two transporters with overlapping functions: THTR1 encoded by SLC19A2, and THTR2 encoded by SLC19A3. Thiamine is transformed into its active form, thiamine pyrophosphate (TPP) by a kinase encoded by the TPK1 gene. Then it may enter the mitochondria through a TPP transporter encoded by SLC25A19. Mutations in SLC19A2 cause thiamine-responsive megaloblastic anemia (TRMA). Mutations in SLC19A3 cause biotin/thiamine–responsive basal ganglia disease. Mutations in SLC25A19 may cause early microcephaly with death in infancy (also called Amish microcephaly) or a later-onset bilateral striatal necrosis with progressive peripheral neuropathy. Recently, mutations in the TPK1 gene have been associated with recurrent encephalopathy with mild lactic acidosis.

Styles APA, Harvard, Vancouver, ISO, etc.

36

E, Steele Vernon, dir. Cellular and molecular targets for chemoprevention. Boca Raton, Fla : CRC Press, 1992.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

37

Cytoplasmatische Vektoren Zur Heterologen Genexpression Bei Hefen : Konstruktion Und Analyse Linearer Hybridplasmide Basierend Auf Dem Killer - System (Bibliotheca Mycologica). Gebruder Borntraeger Verlagsbuchhandlung, 1996.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

38

Watts, Richard A., et David G. I. Scott. Vasculitis—classification and diagnosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0130.

Texte intégral

Résumé :

The vasculitides are a group of conditions characterized by inflammation and necrosis of blood vessels; they are generally of unknown aetiology. The classification of vasculitides is based on the size of vessel involved and whether there is a known cause (secondary) or not (primary). This approach has stood the test of time. The American College of Rheumatology (ACR) in 1990 produced classification criteria for the major types of vasculitis and in 1994 definitions were promulgated by the Chapel Hill Consensus Conference. These did not include anti-neutrophil cytoplasm antibodies (ANCA) and the ACR scheme did not include microscopic polyangiitis. The definitions have recently been updated to include modern concepts of pathogenesis including ANCA. No validated diagnostic criteria are available for routine clinical practice. The diagnosis of vasculitis requires a high index of suspicion, especially in the systemically unwell patient with multiorgan involvement. The key to diagnosis is a detailed and systematic approach to patient assessment involving all potentially involved organs. In a patient with suspected vasculitis immediate urinalysis is mandatory as the severity of renal involvement at presentation is a major determinant of outcome. Each potentially involved organ should be comprehensively evaluated. Tissue biopsy should be obtained whenever possible, as treatment is potentially toxic using glucocorticoids combined with cytotoxic agents. Biopsy should not, however, delay initiation of treatment. Potential alternative diagnosis should be considered, especially infection and malignancy, and excluded whenever possible.

Styles APA, Harvard, Vancouver, ISO, etc.

39

Gross, Wolfgang L., et Julia U. Holle. Clinical features of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0131.

Texte intégral

Résumé :

The primary ANCA-associated vasculitides are granulomatosis with polyangiitis (Wegener's, GPA), microscopic polyangiitis (MPA), and eosinophilic granulomatosis with polyangiitis (EGPA, Churg-Strauss syndrome, CSS). They predominantly affect small (and medium-sized) vessels and share a variable association with ANCA (anti-neutrophil cytoplasm antibody) directed against neutrophil proteinase 3 (PR3, mainly in GPA) and myeloperoxidase (MPO, mainly in MPA and CSS). Crescentic necrotizing glomerulonephritis and alveolar haemorrhage due to pulmonary capillaritis represent classical (vasculitic) organ manifestations of the ANCA-associated vasculitides (AAV). MPA occurs as a 'pure' small (to medium-size) vessel vasculitis, whereas GPA and CSS are characterized by additional distinct clinical and pathological features. In GPA, granulomatous lesions of the upper and/or lower respiratory tract are a hallmark of the disease. Granulomatous lesions may be large in appearance and occur as space-consuming, infiltrating, and destructive inflammatory masses. GPA is believed to follow a stagewise course with an initial localized form, restricted granulomatous lesions of the upper and/or lower respiratory tract without clinical signs of vasculitis, and a consecutive generalization to systemic vasculitis which may be either non-organ-threatening (early systemic) or organ- and life- threatening (generalized GPA). Rarely, patients arrest in the localized stage and do not progress to systemic disease. In EGPA asthma, hypereosinophilia and eosinophilic organ infiltration (e.g. eosinophilic myocarditis) are typical features of the disease apart from vasculitis. Similarly to GPA, EGPA follows a stagewise course: asthma and eosinophilia may precede full-blown disease for several months or years. Recent cohort studies suggest different phenotypes in EGPA (predominantly vasculitic and MPO-ANCA-positive and predominantly with eosinophilic organ infiltration, usually ANCA-negative). This chapter focuses on the clinical features of the primary AAV and their outcome.

Styles APA, Harvard, Vancouver, ISO, etc.

40

1941-, Bradshaw Ralph A., McAlister-Henn Lee, Douglas Michael G, E.I. du Pont de Nemours & Company. et University of California, Los Angeles., dir. Molecular biology of intracellular protein sorting and organelle assembly : Proceedings of a DuPont-UCLA symposium held in Taos, New Mexico, January 30-February 5, 1987. New York : Liss, 1988.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

41

Krecic, Annette Marie. Nuclear and cytoplasmic functions of a yeast heterogeneous nuclear ribonucleoprotein. 1998.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

42

Jayne, David. Treatment of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0132.

Texte intégral

Résumé :

The goals of treatment in anti-neutrophil cytoplasm antibody (ANCA) vasculitis are to stop vasculitic activity, to prevent vasculitis returning, and to address longer-term comorbidities caused by tissue damage, drug toxicity, and increased cardiovascular and malignancy risk. Cyclophosphamide and high-dose glucocorticoids remain the standard induction therapy with alternative immunosuppressives, such as methotrexate or azathioprine, to prevent relapse. Refractory disease resulting from a failure of induction or remission maintenance therapy requires alternative agents and rituximab has been particularly effective. Replacement of cyclophosphamide by rituximab for remission induction is supported by recent evidence. Additional therapy with intravenous methylprednisolone and plasma exchange is employed in severe presentations with failing vital organ function. Drug toxicity contributes to comorbidity and mortality and has led to newer regimens with reduced cyclophosphamide exposure. Glucocorticoid toxicity remains a major problem, with controversy over the rapidity with which glucocorticoids can be reduced or withdrawn. Disease relapse occurs in 50% and requires early detection at a stage when it will not adversely affect outcomes. Rates of cardiovascular disease and malignancy are higher than in control populations but strategies to reduce their risk, apart from cyclophosphamide-sparing regimens, have not been developed. Thromboembolic events occur in 10% and may be linked to the recently identified autoantibodies to plasminogen and tissue plasminogen activator. Outcomes of vasculitis depend heavily on the level of tissue damage at diagnosis, especially renal dysfunction, but are also influenced by patient age, ANCA subtype, disease extent, and response to therapy. Eosinophilic granulomatosis with polyangiitis (Churg-Strauss)is treated along similar principles to granulomatosis with polyangiitis (GPA) and microscopic polyangiitis but the persistence of steroid-dependent asthma in over one-third and differences in pathogenesis has suggested alternative treatment approaches. Chronic morbidity results from tissue damage and is especially common in the upper and lower respiratory tract and kidneys. Tracheobronchial disease is a severe late complication of GPA, while deafness, nasal obstruction, and chronic sinusitis are sequelae of nasal and ear vasculitis. Chronic infection of damaged epithelial surfaces acts as a drive for vasculitic activity and adequate infection control is necessary for stable remission. Chronic kidney disease can stabilize for many years but the risks of endstage renal disease (ESRD) are increased by acute kidney injury at presentation or renal relapse. Renal transplantation is successful, with similar outcomes to other causes of ESRD.

Styles APA, Harvard, Vancouver, ISO, etc.

Nous offrons des réductions sur tous les plans premium pour les auteurs dont les œuvres sont incluses dans des sélections littéraires thématiques. Contactez-nous pour obtenir un code promo unique!