Bibliographies thématiques / Experimental Autoimmune Encephalomyelitis, Progesterone

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Littérature scientifique sur le sujet « Experimental Autoimmune Encephalomyelitis, Progesterone »

Auteur : Grafiati
Publié le 9 mars 2023
Mis à jour le 31 juillet 2025

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Articles de revues sur le sujet "Experimental Autoimmune Encephalomyelitis, Progesterone"

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Giatti, S., D. Caruso, M. Boraso, et al. "Neuroprotective Effects of Progesterone in Chronic Experimental Autoimmune Encephalomyelitis." Journal of Neuroendocrinology 24, no. 6 (2012): 851–61. http://dx.doi.org/10.1111/j.1365-2826.2012.02284.x.

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Milosevic, Ana, Irena Lavrnja, Danijela Savic, et al. "Rat Ovarian Function Is Impaired during Experimental Autoimmune Encephalomyelitis." Cells 12, no. 7 (2023): 1045. http://dx.doi.org/10.3390/cells12071045.

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Multiple sclerosis (MS) is an autoimmune disease affecting the CNS and occurring far more prevalently in women than in men. In both MS and its animal models, sex hormones play important immunomodulatory roles. We have previously shown that experimental autoimmune encephalomyelitis (EAE) affects the hypothalamic–pituitary–gonadal axis in rats of both sexes and induces an arrest in the estrous cycle in females. To investigate the gonadal status in female rats with EAE, we explored ovarian morphometric parameters, circulating and intraovarian sex steroid levels, and the expression of steroidogeni
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Garay, Laura, Maria Claudia Gonzalez Deniselle, Regine Sitruk-Ware, Rachida Guennoun, Michael Schumacher, and Alejandro F. De Nicola. "Efficacy of the selective progesterone receptor agonist Nestorone for chronic experimental autoimmune encephalomyelitis." Journal of Neuroimmunology 276, no. 1-2 (2014): 89–97. http://dx.doi.org/10.1016/j.jneuroim.2014.08.619.

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Yates, M. A., Y. Li, P. Chlebeck, T. Proctor, A. A. Vandenbark, and H. Offner. "Progesterone treatment reduces disease severity and increases IL-10 in experimental autoimmune encephalomyelitis." Journal of Neuroimmunology 220, no. 1-2 (2010): 136–39. http://dx.doi.org/10.1016/j.jneuroim.2010.01.013.

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Garay, L. I., M. C. González Deniselle, M. E. Brocca, A. Lima, P. Roig, and A. F. De Nicola. "Progesterone down-regulates spinal cord inflammatory mediators and increases myelination in experimental autoimmune encephalomyelitis." Neuroscience 226 (December 2012): 40–50. http://dx.doi.org/10.1016/j.neuroscience.2012.09.032.

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Garay, Laura, Maria Claudia Gonzalez Deniselle, Maria Meyer, et al. "Protective effects of progesterone administration on axonal pathology in mice with experimental autoimmune encephalomyelitis." Brain Research 1283 (August 2009): 177–85. http://dx.doi.org/10.1016/j.brainres.2009.04.057.

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Utevska, S. V. "Experimental autoimmune encephalomyelitis (EAE) course in prenatally stressed rat males, the offspring of mothers with different sensitivity to EAE." Faktori eksperimental'noi evolucii organizmiv 24 (August 30, 2019): 244–48. http://dx.doi.org/10.7124/feeo.v24.1109.

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Aim. The research is aimed at investigating the effect of prenatal stress on the incidence and course of experimental autoimmune encephalomyelitis (EAE) as well as the level of sex hormones in 200-days-old male rats, offspring of females with different sensitivity to EAE induction. Methods. The incidence and severity of EAE including duration of latent period, duration of the period from the first to the maximum manifestation of motor disfunction, mean clinical scores, maximum level of motor disfunction (maximum clinical scores) were analyzed in rats with induced EAE. Serum testosterone, estra
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Milosevic, Ana, Katarina Milosevic, Anica Zivkovic, et al. "Alterations in the Hypothalamic–Pituitary–Adrenal Axis as a Response to Experimental Autoimmune Encephalomyelitis in Dark Agouti Rats of Both Sexes." Biomolecules 14, no. 8 (2024): 1020. http://dx.doi.org/10.3390/biom14081020.

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Multiple sclerosis (MS) is a chronic inflammatory disease that affects the central nervous system, usually diagnosed during the reproductive period. Both MS and its commonly used animal model, experimental autoimmune encephalomyelitis (EAE), exhibit sex-specific features regarding disease progression and disturbances in the neuroendocrine and endocrine systems. This study investigates the hypothalamic–pituitary–adrenal (HPA) axis response of male and female Dark Agouti rats during EAE. At the onset of EAE, Crh expression in the hypothalamus of both sexes is decreased, while males show reduced
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Yu, Hong-jun, Jun Fei, Xing-shu Chen, et al. "Progesterone attenuates neurological behavioral deficits of experimental autoimmune encephalomyelitis through remyelination with nucleus-sublocalized Olig1 protein." Neuroscience Letters 476, no. 1 (2010): 42–45. http://dx.doi.org/10.1016/j.neulet.2010.03.079.

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Ghoumari, Abdel Mouman, Charly Abi Ghanem, Narimène Asbelaoui, Michael Schumacher, and Rashad Hussain. "Roles of Progesterone, Testosterone and Their Nuclear Receptors in Central Nervous System Myelination and Remyelination." International Journal of Molecular Sciences 21, no. 9 (2020): 3163. http://dx.doi.org/10.3390/ijms21093163.

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Progesterone and testosterone, beyond their roles as sex hormones, are neuroactive steroids, playing crucial regulatory functions within the nervous system. Among these, neuroprotection and myelin regeneration are important ones. The present review aims to discuss the stimulatory effects of progesterone and testosterone on the process of myelination and remyelination. These effects have been demonstrated in vitro (i.e., organotypic cultures) and in vivo (cuprizone- or lysolecithin-induced demyelination and experimental autoimmune encephalomyelitis (EAE)). Both steroids stimulate myelin formati
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Thèses sur le sujet "Experimental Autoimmune Encephalomyelitis, Progesterone"

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BALLARINI, ELISA. "Caratterizzazione di un modello di encefalomielite autoimmune sperimentale e ruolo neuroprotettivo del progesterone." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2012. http://hdl.handle.net/10281/39833.

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Studies concerning the role of neuroactive steroids in chronic models of Experimental Autoimmune Encephalomyelitis (EAE) are still scarce. First we considered different pathological targets in Dark Agouti (DA) rats affected by EAE in order to well characterize this chronic model of Multiple Sclerosis (MS) which well reflects the relapsing-remitting form of MS. We analyzed neuroinflammatory profile, assonopathy and neuroactive steroid levels in the spinal cord of DA rats affected by EAE. Data obtained at 14 dpi (i.e. days post induction) showed that acute neurological signs were associated wi
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Harness, Jacqueline. "Immunoregulation of experimental autoimmune encephalomyelitis /." St. Lucia, Qld, 2002. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17375.pdf.

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Isaksson, Magnus. "Initiation of Autoimmunity in Experimental Autoimmune Encephalomyelitis." Doctoral thesis, Uppsala universitet, Molekylär medicin, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-173427.

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The events that trigger an autoimmune disease remain largely unknown. To study these events animal models are necessary because symptoms of autoimmune diseases are preceded by a long asymptomatic period in humans. Experimental autoimmune encephalomyelitis (EAE) is the best characterized model for cell mediated autoimmunity and an animal model for the human disease multiple sclerosis. EAE is induced in rodents by immunization with myelin antigens (Ags) together with adjuvants. After immunization, T cells are primed in the periphery by Ag presenting cells and subsequently invade the central nerv
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Ruppova, Klara. "Role of eosinophils in experimental autoimmune encephalomyelitis." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-231835.

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Experimental autoimmune encephalomyelitis (EAE) is the rodent model of multiple sclerosis (MS), a chronic autoimmune neuroinflammatory disease that has a devastating impact on various neurological functions of the patients. The hallmarks of both, MS and EAE, are neuroinflammation, demyelination and neuroaxonal degeneration. Various types of lymphoid and myeloid cells were shown to infiltrate the central nervous system and to participate in disease pathology. However, the role of eosinophil granulocytes has been less explored thus far. An early study showed that eosinophils infiltrate into the
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Weissert, Robert. "Immunogenetics and treatment of experimental autoimmune encephalomyelitis /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3645-5/.

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Lobell, Anna. "Suppressive DNA vaccination in experimental autoimmune encephalomyelitis /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3782-6/.

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Dowdell, Kennichi C. "Neuroendocrine regulation of relapsing Experimental Autoimmune Encephalomyelitis /." The Ohio State University, 1999. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488191124569455.

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Wefer, Judit. "Studies of cellular pathogenesis in experimental autoimmune encephalomyelitis /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-023-0/.

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Wållberg, Maja. "Modulation of immune responses in experimental autoimmune encephalomyelitis /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-335-3/.

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Berl, Sabina [Verfasser]. "Neuronal Response to Experimental Autoimmune Encephalomyelitis / Sabina Berl." Mainz : Universitätsbibliothek Mainz, 2020. http://d-nb.info/1203322933/34.

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Livres sur le sujet "Experimental Autoimmune Encephalomyelitis, Progesterone"

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Weissert, Robert, ed. Experimental Autoimmune Encephalomyelitis - Models, Disease Biology and Experimental Therapy. InTech, 2012. http://dx.doi.org/10.5772/1190.

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Stern, Joel N. H. Mechanisms of suppression of experimental autoimmune encephalomyelitis (EAE) by synthetic compounds and fusion antibodies. 2008.

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Chiba, Kenji. Therapeutic Effects of the Sphingosine 1-Phosphate Receptor Modulator, Fingolimod (FTY720), on Experimental Autoimmune Encephalomyelitis. INTECH Open Access Publisher, 2012.

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Elucidating the migration of Th17 cells to the central nervous system in experimental autoimmune encephalomyelitis. 2012.

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Chapitres de livres sur le sujet "Experimental Autoimmune Encephalomyelitis, Progesterone"

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Rao, Praveen, and Benjamin M. Segal. "Experimental Autoimmune Encephalomyelitis." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60761-720-4_18.

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Gran, B., K. O'Brien, D. Fitzgerald, and A. Rostami. "Experimental Autoimmune Encephalomyelitis." In Handbook of Neurochemistry and Molecular Neurobiology. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-30398-7_16.

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Ballerini, Clara. "Experimental Autoimmune Encephalomyelitis." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1311-5_27.

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Hartung, Hans-Peter, and Bernd C. Kieseier. "Experimental Autoimmune Encephalomyelitis." In Neuroinflammation. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-297-5_18.

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Jagessar, S. Anwar, Karin Dijkman, Jordon Dunham, Bert A. ‘t Hart, and Yolanda S. Kap. "Experimental Autoimmune Encephalomyelitis in Marmosets." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/7651_2014_113.

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Terry, Rachael L., Igal Ifergan, and Stephen D. Miller. "Experimental Autoimmune Encephalomyelitis in Mice." In Methods in Molecular Biology. Springer New York, 2014. http://dx.doi.org/10.1007/7651_2014_88.

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Encinas, J. A., and V. K. Kuchroo. "Genetics of Experimental Autoimmune Encephalomyelitis." In Genes and Genetics of Autoimmunity. KARGER, 1999. http://dx.doi.org/10.1159/000060485.

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Constantinescu, Cris S. "Environmental Influences in Experimental Autoimmune Encephalomyelitis." In Experimental Models of Multiple Sclerosis. Springer US, 2005. http://dx.doi.org/10.1007/0-387-25518-4_25.

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Kieseier, Bernd C., and Hans-Peter Hartung. "Matrix Metalloproteinases in Experimental Autoimmune Encephalomyelitis." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9613-1_39.

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Bettelli, Estelle, and Lindsay B. Nicholson. "Pathogenic and Regulatory Cytokines in Experimental Autoimmune Encephalomyelitis." In Cytokines and Autoimmune Diseases. Humana Press, 2002. https://doi.org/10.1007/978-1-59259-129-9_10.

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Actes de conférences sur le sujet "Experimental Autoimmune Encephalomyelitis, Progesterone"

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dos Santos Farias, Alessandro, and Fernanda Garcia Fossa. "B-lymphocytes characterization in experimental autoimmune encephalomyelitis." In XXIII Congresso de Iniciação Científica da Unicamp. Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-38297.

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Kasheke, Gracious, Scott P. Holman, Kaitlyn Fraser, et al. "IRX4204 enhances gait recovery in mice subjected to experimental autoimmune encephalomyelitis." In ASPET 2023 Annual Meeting Abstracts. American Society for Pharmacology and Experimental Therapeutics, 2023. http://dx.doi.org/10.1124/jpet.122.146780.

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Borin Pereira, Alexandre, Tabata R. Costa, Fernanda Garcia Fossa, et al. "Alterations in thymocyte subpopulations during the course of experimental autoimmune encephalomyelitis." In XXIII Congresso de Iniciação Científica da Unicamp. Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-38275.

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Maria Barbosa Dos Santos, Leonilda, Vanessa Cristina De Barros Mariano, Stephani Oliveira Alves, Vitor Almeida Da Silva, and Fabiana Ferreira Aquino. "The effect of vitamin D in the evolution of experimental autoimmune encephalomyelitis." In XXIII Congresso de Iniciação Científica da Unicamp. Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-37964.

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RODRIGUES COSTA, TABATA, Alessandro Farias, Carolina Francelin Rovarotto, et al. "Analysis of CD4+RUNX3+ T lymphocytes during clinical course of experimental autoimmune encephalomyelitis." In XXIV Congresso de Iniciação Científica da UNICAMP - 2016. Galoa, 2016. http://dx.doi.org/10.19146/pibic-2016-51128.

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Aulova, K. S., and G. S. Nevinsky. "CATALYTIC ANTIBODIES DURING THE SPONTANEOUS DEVELOPMENT OF EXPERIMENTAL ENCEPHALOMYELITIS IN MICE OF 2D2, TH LINES AND FIRST-GENERATION HYBRIDS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-299.

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The parameters of the spontaneous experimental autoimmune encephalomyelitis development in mice, a model of multiple sclerosis, were investigated in first-generation hybrids obtained by crossing transgenic lines 2D2 and Th. The data were compared with similar parameters for mice of the parental lines 2D2 and Th.
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Maria Barbosa Dos Santos, Leonilda, and Amanda De Barros Piffer. "The effect of vitamin D in the evolution of experimental autoimmune encephalomyelitis. Effect on B-lymphocytes function." In XXIII Congresso de Iniciação Científica da Unicamp. Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-38190.

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Li, Chung-Hsien, Ming-Hong Lin, Yen-Fu Chen, et al. "Abstract 1295: Role of glycine N-methyltransferase in the regulation of T cell responses in experimental autoimmune encephalomyelitis." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1295.

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Gruber, Ross, Anna Blazier, Lan Lee, et al. "Evaluating the Effect of a Bruton’s Tyrosine Kinase Inhibitor in a Murine Experimental Autoimmune Encephalomyelitis Model of Multiple Sclerosis (P2-3.012)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000202142.

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Alassiri, Mohammed. "THE THERAPEUTIC TREATMENT OF PEPITEM INCREASES THE PROTEIN EXPRESSION OF SIRT1 IN A MOUSE MODEL OF EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS (EAE) AS A MODEL FOR HUMAN MS." In Dubai International Conference on Research in Life-Science & Healthcare, 22-23 February 2024. Global Research & Development Services, 2024. http://dx.doi.org/10.20319/icrlsh.2024.1625.

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Objective: To investigate the effect of the prophylactic and therapeutic treatment of the immunopeptide PEPITEM on the protein expression of SIRT1 in a mouse model of experimental autoimmune encephalomyelitis (EAE) as a model for human MS. Methods: Using C57BL/6 female mice, we dosed the PEPITEM in the EAE model via intraperitoneal injections either prophylactically or therapeutically. The disease was induced using MOG35-55 and complete Freund's adjuvants augmented with pertussis toxin. The EAE score was recorded daily until the end of the experiment (21 days). A Western blot analysis was perf
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