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Littérature scientifique sur le sujet « Folic acid Metabolism »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 25 juillet 2025

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Articles de revues sur le sujet "Folic acid Metabolism"

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Khaitovich, M. V. "Folates: Modern Pregnant Health Support." HEALTH OF WOMAN, no. 4(150) (May 30, 2020): 37–42. http://dx.doi.org/10.15574/hw.2020.150.37.

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Folates (folic acid-based chemical compounds) got their name from the Latin “folio” - “leaf”, since they were first synthesized from spinach leaves, in which vitamin B9 is found in maximum quantities. As an important cofactor in carbon metabolism, folates are involved in the most important metabolic processes in the body, in particular, they play a key role in the synthesis of nucleotides and DNA replication. The article provides information on the physiological role of folates, their metabolism and its genetic aspects. The clinical significance of folate deficiency is examined, their sources

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Lucock, Mark. "Folic Acid: Beyond Metabolism." Journal of Evidence-Based Complementary & Alternative Medicine 16, no. 2 (2011): 102–13. http://dx.doi.org/10.1177/1533210110392950.

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Mandatory and discretionary fortification with folic acid is eliminating deficiency of this vitamin. Blood levels of the vitamin have never been higher, with hematologic folate values commonly exceeding the upper range of calibration. The synthetic analog (pteroylmonoglutamic acid) prevents neural tube defects and lowers homocysteine, both positive attributes, yet negative correlates of pteroylmonoglutamic acid are increasingly reported. These involve increased risk for common cancers (ie, colon, breast, prostate) and antimetabolite effects on natural killer cells and at dihydrofolate reductas

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Metz, Jack. "Folic Acid Metabolism and Malaria." Food and Nutrition Bulletin 28, no. 4_suppl4 (2007): S540—S549. http://dx.doi.org/10.1177/15648265070284s407.

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Pellis, Linette, Yvonne Dommels, Dini Venema, et al. "High folic acid increases cell turnover and lowers differentiation and iron content in human HT29 colon cancer cells." British Journal of Nutrition 99, no. 4 (2007): 703–8. http://dx.doi.org/10.1017/s0007114507824147.

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Folate, a water-soluble B vitamin, is a cofactor in one-carbon metabolism and is essential for DNA synthesis, amino acid interconversion, methylation and, consequently, normal cell growth. In animals with existing pre-neoplastic and neoplastic lesions, folic acid supplementation increases the tumour burden. To identify processes that are affected by increased folic acid levels, we compared HT29 human colon cancer cells exposed to a chronic supplemental (100 ng/ml) level of folic acid to cells exposed to a normal (10 ng/ml) level of folic acid, in the presence of vitamin B12and other micronutri

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Sahar, Saniya. "Role of Folate and Folic Acid During Pregnancy." International Journal for Research in Applied Science and Engineering Technology 9, no. 12 (2021): 1488–92. http://dx.doi.org/10.22214/ijraset.2021.39295.

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Abstract: Pregnancy represents a period of fast tissue growth of maternal and foetal tissues that's related to enhanced energy and nutrient needs. Maternal nutrition throughout gestation period, has being essential for best offspring development, reducing long unwellness burden and for general health throughout life. Maternal Folate throughout pregnancy might have numerous roles in offspring health, as well as neurodevelopment and psychological feature performance in childhood. Folate is crucial for C1 metabolism, a network of pathways concerned in many biological processes as well as nucleoti

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Han, Xuhui, Bingqi Wang, Dongxu Jin, et al. "Precise Dose of Folic Acid Supplementation Is Essential for Embryonic Heart Development in Zebrafish." Biology 11, no. 1 (2021): 28. http://dx.doi.org/10.3390/biology11010028.

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Folic acid, one of the 13 essential vitamins, plays an important role in cardiovascular development. Mutations in folic acid synthesis gene 5,10-methylenetetrahydrofolate reductase (MTHFR) is associated with the occurrence of congenital heart disease. However, the mechanisms underlying the regulation of cardiac development by mthfr gene are poorly understood. Here, we exposed zebrafish embryos to excessive folate or folate metabolism inhibitors. Moreover, we established a knock-out mutant of mthfr gene in zebrafish by using CRISPR/Cas9. The zebrafish embryos of insufficient or excessive folic

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Ledowsky, Carolyn, Abela Mahimbo, Vanessa Scarf, and Amie Steel. "Women Taking a Folic Acid Supplement in Countries with Mandatory Food Fortification Programs May Be Exceeding the Upper Tolerable Limit of Folic Acid: A Systematic Review." Nutrients 14, no. 13 (2022): 2715. http://dx.doi.org/10.3390/nu14132715.

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Background: In preconception and pregnancy, women are encouraged to take folic acid-based supplements over and above food intake. The upper tolerable limit of folic acid is 1000 mcg per day; however, this level was determined to avoid masking a vitamin B12 deficiency and not based on folic acid bioavailability and metabolism. This review’s aim is to assess the total all-source intake of folate in women of childbearing age and in pregnancy in high-income countries with folate food fortification programs. Methods: A systematic search was conducted in five databases to find studies published sinc

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James, A. Greenberg, J. Bell Stacey, Guan Yong, and Yu Yan-Hong. "Folic acid supplementation and pregnancy: more than just neural tube defect prevention." REPRODUCTIVE ENDOCRINOLOGY, no. 34 (April 30, 2017): 57–63. https://doi.org/10.18370/2309-4117.2017.34.57-63.

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Folate (vitamin B9) is an essential nutrient that is required for DNA replication and as a substrate for a range of enzymatic reactions involved in amino acid synthesis and vitamin metabolism. Demands for folate increase during pregnancy because it is also required for growth and development of the fetus. Folate deficiency has been associated with abnormalities in both mothers (anemia, peripheral neuropathy) and fetuses (congenital abnormalities). This article reviews the metabolism of folic acid, the appropriate use of folic acid supplementation in pregnancy, and the potential benefits of fol

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Gunnala, Siddarth, Lori M. Buhlman, and Nafisa M. Jadavji. "How Increased Dietary Folic Acid Intake Impacts Health Outcomes Through Changes in Inflammation, Angiogenesis, and Neurotoxicity." Nutrients 17, no. 7 (2025): 1286. https://doi.org/10.3390/nu17071286.

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Dietary folic acid supplementation is well known for playing a crucial role in the closure of the neural tube. Individuals have continued to increase dietary intake of folic acid in counties with mandatory fortication laws in place. Some studies have demonstrated adverse health effects in individuals with high dietary intake of folic acid. Nutrition is a modifiable risk factor for ischemic stroke. Specifically, elevated levels of homocysteine, they can be reduced by increasing intake of vitamins, such as folic acid, a B-vitamin. Hypoxia, when levels of oxygen are reduced, is a major component

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James A. Greenberg, Stacey J. Bell, Yong Guan, and Yan-Hong Yu. "Folic acid supplementation and pregnancy: more than just neural tube defect prevention." REPRODUCTIVE ENDOCRINOLOGY, no. 34 (April 30, 2017): 57–63. http://dx.doi.org/10.18370/2309-4117.2017.34.57-63.

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Folate (vitamin B9) is an essential nutrient that is required for DNA replication and as a substrate for a range of enzymatic reactions involved in amino acid synthesis and vitamin metabolism. Demands for folate increase during pregnancy because it is also required for growth and development of the fetus. Folate deficiency has been associated with abnormalities in both mothers (anemia, peripheral neuropathy) and fetuses (congenital abnormalities).This article reviews the metabolism of folic acid, the appropriate use of folic acid supplementation in pregnancy, and the potential benefits of foli

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Thèses sur le sujet "Folic acid Metabolism"

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Padmanabhan, Nisha. "The biological and molecular effects of abnormal folate metabolism." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708242.

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Bufalino, Andreia 1983. "Analise da suplementação vitaminica e de polimorfismos em genes da via metabolica do acido folico em mães de individuos com fissuras labio-palatinas não-sindromicas." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/288724.

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Orientador: Ricardo Della Coletta<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba<br>Made available in DSpace on 2018-08-15T05:46:12Z (GMT). No. of bitstreams: 1 Bufalino_Andreia_M.pdf: 1848465 bytes, checksum: e05d7021dc64d36ed2556bd7fc4b97fd (MD5) Previous issue date: 2010<br>Resumo: A fissura labial e/ou palatina (FL/P) não-sindrômica é uma malformação congênita do lábio e/ou palato com alta frequência na população brasileira. A etiologia das fissuras é complexa e conta com a participação de fatores genéticos e ambientais. Inúmeros est

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Chan, Manuel. "Characterization of the 5' region of the human methylenetetrahydrofolate reductase, MTHFR, gene." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0026/MQ50734.pdf.

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Kapil, Aditya. "Transport and metabolism of pyridoxine and folic acid in the rat small intestine." Thesis, University of York, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284168.

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Weisberg, Ilan S. "Evaluation of common polymorphisms in methylenetetrahydrofolate reductase (MTHFR) and betaine-homocysteine methyltransferase (BHMT)." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0032/MQ64479.pdf.

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Crott, Jimmy. "The effects of folic acid deficiency and defects in folate metabolism on chromosome damage in vitro." Title page, table of contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phc9515.pdf.

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Reprints of the author's previously published articles included as an appendix. Bibliography: leaves 165-188. "This thesis describes a series of experiments that aimed to investigate the effects of folic acid deficiency and defects in folate metabolism on chromosome damage rates in human lymphocytes. The accumulation of chromosome damage over time is an important issue because it is thought to contribute to the mechanism of ageing and the aetiology of diseases of age such as cancer and Alzheimer's disease."

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Nimchuk, Anastasia Katherine. "Homocysteine metabolism as a response to aging and folic acid intake in Fisher 344 rats." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0009/MQ59858.pdf.

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Sibani, Sahar. "Genetic and nutritional folate deficiency : implications for homocystinuria and intestinal neoplasia." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31539.

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Folate deficiency, a prevalent vitamin deficiency in America, can stem from environmental and/or genetic causes. The most common inborn error of folate metabolism is deficiency of methylenetetrahydrofolate reductase (MTHFR), which catalyzes the reduction of 5,10-methylenetetrahydrofolate to 5-methyltetrahydrofolate. Severe MTHFR deficiency results in hyperhomocysteinemia and homocystinuria; patients present with developmental delay, and various neurological and vascular disorders. This thesis describes three mutations identified in the MTHFR locus in patients with severe deficiency: 1025T&rarr

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Öhrvik, Veronica. "Folate bioavailability in vitro experiments and human trials /." Uppsala : Dept. of Food Science, Swedish University of Agricultural Sciences, 2009. http://epsilon.slu.se/200963.pdf.

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Lawrance, Andrea Karin. "The impact of genetic and nutritional disturbances of folate metabolism on tumourigenesis in a mouse model of colorectal cancer /." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111885.

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The relationship between colorectal cancer (CRC) and folate metabolism is complex. Dietary folate, depending on the timing and dose, may either prevent or enhance tumour initiation and/or growth, and polymorphisms in the genes encoding folate-metabolising enzymes may also modulate risk. In this thesis, the Apcmin/+ mouse model of CRC was used to investigate the effect of nutritional and genetic disturbances in folate metabolism on tumourigenesis and to examine various mechanisms.<br>The reduced folate carrier I (RFC1) is responsible for the cellular uptake and intestinal absorption of folate,

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Livres sur le sujet "Folic acid Metabolism"

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Frances, Picciano Mary, Stokstad E. L. Robert, Gregory Jesse F, and American Chemical Society. Food and Nutritional Biochemistry Subdivision., eds. Folic acid metabolism in health and disease. Wiley-Liss, 1990.

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Bailey, Lynn B. Folate in health and disease. 2nd ed. Taylor & Francis, 2010.

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J, Massaro Edward, and Rogers John M, eds. Folate and human development. Humana Press, 2002.

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Amouzou, Kou'santa Sabiba. Evaluation des marqueurs nutritionnels et génétiques du statut en coenzymes B (cobalamines et folates) et de l'homocystéinemie plasmatique dans une population d'Afrique de l'Ouest (Benin-Togo). [s.n., 2003.

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Pristoupilova, Kamila. Role of folates in metabolic pathways (Studie AV CR). Academia, 1997.

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Folate in health and disease. 2nd ed. Taylor & Francis, 2010.

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Hitchings, George H. Inhibition of Folate Metabolism in Chemotherapy: The Origins and Uses of Co-trimoxazole. Brand: Springer, 2011.

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Folate in Health and Disease. 2nd ed. CRC, 2009.

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Bailey, Lynn B. Folate in Health and Disease. Taylor & Francis Group, 1994.

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Bailey, Lynn B. Folate in Health and Disease. Taylor & Francis Group, 2009.

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Chapitres de livres sur le sujet "Folic acid Metabolism"

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Tiwari, Deeksha, Annu Rani, and Hem Chandra Jha. "Homocysteine and Folic Acid Metabolism." In Homocysteine Metabolism in Health and Disease. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-6867-8_1.

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Huennekens, F. M., and M. J. Osborn. "Folic Acid Coenzymes and One-Carbon Metabolism." In Advances in Enzymology - and Related Areas of Molecular Biology. John Wiley & Sons, Inc., 2006. http://dx.doi.org/10.1002/9780470122662.ch8.

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Lorenzini, L., A. De Martino, W. Testi, et al. "Radioimmunoassay of Folic Acid and its Correlation with Age." In Purine and Pyrimidine Metabolism in Man VIII. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-2584-4_166.

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Scott, J. M., D. G. Weir, A. Molloy, J. Mcpartlin, L. Daly, and P. Kirke. "Folic Acid Metabolism and Mechanisms of Neural Tube Defects." In Ciba Foundation Symposium 181 - Neural Tube Defects. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514559.ch11.

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Girdwood, Ronald H. "Some Aspects of Disordered Folic Acid Metabolism in Man." In Ciba Foundation Symposium - Chemistry and Biology of Pteridines. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470718919.ch28.

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Clifford, Andrew J., Ali Arjomand, Stephen R. Dueker, Philip D. Schneider, Bruce A. Buchholz, and John S. Vogel. "The Dynamics of Folic Acid Metabolism in an Adult Given a Small Tracer Dose of 14C-Folic Acid." In Advances in Experimental Medicine and Biology. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4899-1959-5_15.

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Laloglu, Esra. "Water Soluble Vitamins." In Vitamins, Minerals and Trace Elements. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359173.2.

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Vitamins are organic molecules that cannot be synthesized in sufficient quantities by humans and therefore must be obtained through diet. Nine vitamins (folic acid, cobalamin, ascorbic acid, pyridoxine, thiamine, niacin, riboflavin, biotin and pantothenic acid) are classified as water-soluble. When taken in excess of the body’s needs, they are easily excreted in the urine and therefore must be taken continuously through the diet. Toxicity is rare. However, deficiencies can develop rapidly. Vitamins are necessary for the performance of certain cellular functions. For example, many water-soluble

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Castro, Gerardo Daniel, and José Alberto Castro. "Metabolism of Ethanol to Acetaldehyde in the Rat Mammary Tissue: Inhibitory Effects of Plant Polyphenols and Folic Acid." In Alcohol, Nutrition, and Health Consequences. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-047-2_12.

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Zhu, T., R. Koepsel, M. M. Domach, and M. M. Ataai. "Metabolic Engineering of Folic Acid Production." In ACS Symposium Series. American Chemical Society, 2003. http://dx.doi.org/10.1021/bk-2003-0862.ch013.

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Woods, D. D. "Metabolic Relations Between P-Aminobenzoic Acid and Folic Acid in Micro-Organisms." In Ciba Foundation Symposium - Chemistry and Biology of Pteridines. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470718919.ch18.

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Actes de conférences sur le sujet "Folic acid Metabolism"

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Bayer, A., E. Zrenner, H. J. Thiel, S. Ried, and D. Schmidt. "Early detection of anticonvulsant drug toxicity by colour vision tests." In Noninvasive Assessment of the Visual System. Optica Publishing Group, 1991. http://dx.doi.org/10.1364/navs.1991.md10.

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Phenytoin and Carbamazepine, especially in combination or after repeated exposure to toxic blood concentrations, can produce neurological and mental syndroms. Possible mechanisms of impaired neurological and mental function include neuronal damage, or disturbance of folic acid, monoamine or hormonal metabolism. Phenytoin and Carbamazepine may also impair the ocular system. The most common symptoms are diplopia, nystagmus and blurred vision. Cerebellar or vestibulo-ocular dysfunction is suspected as mechanism of these ocular adverse effects.

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Akca, Emine Erdağ, Özlem Çağındı, and Ergun Köse. "The Importance of Cereal Based Foods to Prevention of Iron Deficiency." In 6th International Students Science Congress. Izmir International Guest Student Association, 2022. http://dx.doi.org/10.52460/issc.2022.025.

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Iron is vital for almost every organism, due to its role in a wide range of metabolic processes. For the human body, it contributes to crucial activities, especially haemoglobin synthesis and oxygen transport. Iron deficiency anaemia occurs when iron stores release inadequate amount of iron [1; 2; 3]. However, basic strategies can be used effectively in the prevention of iron deficiency and related anaemia. Although the applicability of these strategies mostly depends on the health infrastructure, the economy of the countries and people's access to resources, it is stated that food fortificati

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O'Flanagan, Ciara H., Xuewen Chen, Zahra Ashkavand, Sergey A. Krupenko, and Stephen D. Hursting. "Abstract 247: Nutrient stress via folic acid modulation causes systemic and cancer-specific metabolic reprogramming and differential effects on primary and metastatic mammary tumor growth in lean and obese mice." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-247.

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