Bibliographies thématiques / Glycosaminoglycans Metabolism

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Littérature scientifique sur le sujet « Glycosaminoglycans Metabolism »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 30 janvier 2023

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Articles de revues sur le sujet "Glycosaminoglycans Metabolism"

Viola, Manuela, Timothy E. L. Douglas, Laura Alaniz, and Barbara Bartolini. "Glycosaminoglycans Metabolism." Biochemistry Research International 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/245792.

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Mironov, S. P., A. M. Gerasimov, L. N. Furtseva, A. G. Tikhomirov, D. O. Vasiliev, and R. V. Merkurieva. "Oxyprolinuria and Glycosaminoglycansuria in Achilles Tendon Ruptures." N.N. Priorov Journal of Traumatology and Orthopedics 5, no. 2 (June 15, 1998): 51–53. http://dx.doi.org/10.17816/vto104492.

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In 17 athlets and ballet dancers with Achilles tendon ruptures oxyprolinuria and urine content of hexuronic acid were studied. Considerable increase of collagen and glycosaminoglycans decay products was detected. The results of differential spectrophotometry showed that urine glycosaminoglycanes presented by proteoglycans. It was assumed that Achilles tendon ruptures could be caused by excessive mechanical load and/or lack of the connective tissue metabolism. The authors consider that further study of oxyprolinuria as well as enzyme-substrate systems of glycosaminoglycans are perspective for t

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Koźma, Ewa M., Kornelia Kuźnik-Trocha, Katarzyna Winsz-Szczotka, Grzegorz Wisowski, Paweł Olczyk, Katarzyna Komosińska-Vassev, Mariusz Kasperczyk, and Krystyna Olczyk. "Significant Remodeling Affects the Circulating Glycosaminoglycan Profile in Adult Patients with both Severe and Mild Forms of Acute Pancreatitis." Journal of Clinical Medicine 9, no. 5 (May 1, 2020): 1308. http://dx.doi.org/10.3390/jcm9051308.

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Acute pancreatitis (AP) manifests itself either as a mild, self-limiting inflammation or a severe, systemic inflammatory process that is associated with various complications and a high mortality rate. It is unknown whether these two forms of the disease can differ in the profile of circulating glycosaminoglycans, which are molecules with huge biological reactivity due to a high density of negative electric charge. Plasma glycosaminoglycans were characterized/quantified in 23 healthy controls, 32 patients with mild AP, and 15 individuals with severe disease using electrophoresis with enzymatic

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Wolf, Hanna, Andrea Graßmann, Romina Bester, André Hossinger, Christoph Möhl, Lydia Paulsen, Martin H. Groschup, Hermann Schätzl, and Ina Vorberg. "Modulation of Glycosaminoglycans Affects PrPScMetabolism but Does Not Block PrPScUptake." Journal of Virology 89, no. 19 (July 22, 2015): 9853–64. http://dx.doi.org/10.1128/jvi.01276-15.

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ABSTRACTMammalian prions are unconventional infectious agents composed primarily of the misfolded aggregated host prion protein PrP, termed PrPSc. Prions propagate by the recruitment and conformational conversion of cellular prion protein into abnormal prion aggregates on the cell surface or along the endocytic pathway. Cellular glycosaminoglycans have been implicated as the first attachment sites for prions and cofactors for cellular prion replication. Glycosaminoglycan mimetics and obstruction of glycosaminoglycan sulfation affect prion replication, but the inhibitory effects on different st

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Kittlick, P. D. "Inflammation, glycolytic metabolism, and glycosaminoglycans." Experimental pathology 30, no. 1 (January 1986): 1–19. http://dx.doi.org/10.1016/s0232-1513(86)80051-2.

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Bower, L., C. Warren, and G. Manley. "Human Serum and Urine Glycosaminoglycans in Health and in Patients with Chronic Renal Failure." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 29, no. 2 (March 1992): 190–95. http://dx.doi.org/10.1177/000456329202900212.

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Quantitation of uronic acid precipitable by cetylpyridinium chloride (CPC) and electrophoretic separation of glycosaminoglycans were performed on sera from patients with chronic renal failure and compared to normal controls. Serum CPC-precipitable uronic acid (CpUA) levels in patients with renal failure were significantly higher (mean 13·7 mg/L, range 7·1–23·6 mg/L) than normal controls (mean 9·6 mg/L, range 5·1–13·9 mg/L) due to increased concentrations of low sulphated chondroitin sulphate. A positive correlation between serum CpUA and creatinine was found in renal failure patients. Urine Cp

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Kahaly, G., C. Stover, J. Beyer, and E. Otto. "In vitro synthesis of glycosaminoglycans in endocrine ophthalmopathy." Acta Endocrinologica 127, no. 5 (November 1992): 397–402. http://dx.doi.org/10.1530/acta.0.1270397.

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The effects of humoral and cell-mediated immunity on the glycosaminoglycan synthesis of retrobulbar fibroblasts was evaluated in patients with endocrine ophthalmopathy. After incubation with IgG and sera, secreted glycosaminoglycans, radiolabeled with D-6-3H-glucosamine and 35sulfate, were precipitated with cetylpyridinium chloride and ethanol. Hyaluronic acid synthesis of human retrobulbar fibroblasts after incubation with sera and IgG and after co-culture with lymphocytes was assessed by means of a radiometric test. Patients' IgG, compared to controls', accounted for a higher secretory stimu

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Chang, Chih-Cheng, Tien-Chun Chang, Shine CS Kao, Yea-Fhey Kuo, and Li-Fei Chien. "Pentoxifylline inhibits the proliferation and glycosaminoglycan synthesis of cultured fibroblasts derived from patients with Graves' ophthalmopathy and pretibial myxoedema." Acta Endocrinologica 129, no. 4 (October 1993): 322–27. http://dx.doi.org/10.1530/acta.0.1290322.

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Excessive amounts of glycosaminoglycans accumulate in the extraocular muscles of patients with Graves' ophthalmopathy and in the affected skin of patients with pretibial myxoedema. It is widely accepted that fibroblasts are the sources of glycosaminoglycan synthesis. Pentoxifylline, an analogue of the methylxanthine theobromine, inhibits the proliferation and certain biosynthetic activities of fibroblasts derived from normal human skin and from skin of patients with some fibrotic disorders. Our objective was to determine whether pentoxifylline has similar effects on fibroblasts derived from pa

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Bondar', I. A., and V. V. Klimontov. "Glycosaminoglycans and diabetic nephropathy." Problems of Endocrinology 50, no. 2 (April 15, 2004): 29–34. http://dx.doi.org/10.14341/probl11392.

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Diabetic nephropathy (DN) is one of the leading places in the structure of mortality of patients with diabetes mellitus (DM) in Russia and abroad. Despite intensive study, the causes and development mechanisms of this complication are not finally clear. Most often, diabetic kidney damage is seen as the result of a complex interaction of metabolic, hemodynamic, genetic and other mechanisms. At the same time, the majority of researchers give the leading role to hyperglycemia and the metabolic disorders triggered by it. The latter include intensification of non-enzymatic glycation processes, acti

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KAHALY, G., M. SCHULER, A. C. SEWELL, G. BERNHARD, J. BEYER, and U. KRAUSE. "URINARY GLYCOSAMINOGLYCANS IN Graves'OPHTHALMOPATHY." Clinical Endocrinology 33, no. 1 (July 1990): 35–44. http://dx.doi.org/10.1111/j.1365-2265.1990.tb00463.x.

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Thèses sur le sujet "Glycosaminoglycans Metabolism"

Lewis, Martin David. "Human lysosomal sulphate transport." Title page, contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phl6752.pdf.

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Addendum inserted at back Includes bibliographical references (leaves 266-287). 1. Introduction -- 2. Materials and general methods -- 3. Characterisation and partial purification of the lysosomal sulphate transporter -- 4. Identification of proteins involved in lysosomal sulphate transport -- 5. The relationship between a sulphate anion transporter family and the lysosomal sulphate transporter -- 6. Investigation of sulphate transport in human skin fibroblasts -- 7. Concluding remarks

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Freeman, Craig. "The lysosomal degradation of heparan sulphate : a comparative study of the physical and catalytic properties of the heparan sulphate degradative enzymes /." Title page, contents and abstract only, 1991. http://web4.library.adelaide.edu.au/theses/09PH/09phf855.pdf.

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Nigro, Julie. "The role of PPAR-α ligands (fibrates) in the regulation of vascular smooth muscle proteoglycan synthesis and structure as a contributor to reduced lipoprotein binding and the development of atherosclerosis". Monash University, Dept. of Medicine, 2004. http://arrow.monash.edu.au/hdl/1959.1/5464.

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Leroux, Mélanie. "Production de glycosaminoglycanes par voie microbiologique et enzymatique." Thesis, Université Grenoble Alpes (ComUE), 2019. http://www.theses.fr/2019GREAV024.

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Les glycosaminoglycanes (GAGs) sont des polymères de sucres linéaires, présents chez tous les animaux. Certaines bactéries pathogènes synthétisent également des polysaccharides identiques ou très similaires aux GAGs humains. Cette thèse a porté en particulier sur la synthèse de la chondroïtine sulfate et de l’héparosan qui font partie de cette famille de polysaccharides. L’intérêt pour ces deux GAGs est grandissant dans l’industrie pharmaceutique du fait des nombreuses applications médicales qu’ils pourraient permettre. La chondroïtine sulfate est d’ores et déjà extraite de tissus animaux ce q

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Lucon, Marcos. "Avaliação do metabolismo de glicosaminoglicanos em pacientes portadores de cistite intersticial." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5153/tde-06022013-164806/.

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Introdução: a cistite intersticial é doença crônica do trato urinário inferior cujos sintomas são: aumento da freqüência urinária, nictúria, dor pélvica ou perineal que piora com a repleção vesical e melhora com a micção. A etiopatogenia não é totalmente conhecida, mas há indícios de que os glicosaminoglicanos e proteoglicanos que revestem o urotélio vesical possam participar da sua gênese. A perda destes componentes protetores facilitaria o contato de íons e solutos presentes na urina com as porções mais profundas do urotélio desencadeando e perpetuando um processo inflamatório local. Para te

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Robert, Joe͏̈lle. "Influence de divers constituants de la matrice extracellulaire sur le comportement de cellules dermiques d'embryon de poulet cultivées in vitro." Grenoble 1, 1988. http://www.theses.fr/1988GRE10110.

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Donida, Bruna. "Investigação dos biomarcadores de estresse oxidativo e inflamação em pacientes portadores de mucopolissacaridose tipo IVA submetidos à terapia de reposição enzimática." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2015. http://hdl.handle.net/10183/158766.

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A Mucopolissacaridose tipo IVA (MPS IVA), é uma doença lisossômica de depósito ocasionada pela degradação deficiente dos glicosaminoglicanos (GAG) queratan sulfato e condroitin-6-sulfato devido à deficiência da enzima N-acetilgalactosamina 6-sulfatase. Como a fisiopatologia desta doença ainda não está totalmente elucidada e muitos estudos vêm demonstrando o envolvimento do estresse oxidativo e inflamação na patogênese de outros tipos de mucopolissacaridoses, o objetivo principal deste trabalho foi investigar os parâmetros de estresse oxidativo e mediadores inflamatórios em pacientes MPS IVA so

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Franco, Renata Nogueron. "Biomarcadores do metabolismo da cartilagem e sua relação com as alterações morfológicas, inflamatórias e funcionais: um estudo sobre a lesão condral secundária em joelhos humanos." Universidade Federal de São Carlos, 2011. https://repositorio.ufscar.br/handle/ufscar/5131.

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Made available in DSpace on 2016-06-02T20:18:15Z (GMT). No. of bitstreams: 1 3931.pdf: 5591864 bytes, checksum: b4873ba462675328d3fc2005b8032afb (MD5) Previous issue date: 2011-02-28 Osteoarthritis (OA), a degenerative joint disease, is one of the most frequent causes of pain in the musculoskeletal system and of the inability to work in Brazil and the world. It is a multifactor, chronic disease, leading to progressive functional inability. It can arise as a result of injuries to structures such as the anterior crossed ligament and/or meniscus (post-traumatic OA), which, in this case, can

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Lewis, Martin D. "Human lysosomal sulphate transport / Martin David Lewis." 2001. http://hdl.handle.net/2440/21672.

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Addendum inserted at back Includes bibliographical references (leaves 266-287). xxiv, 289 leaves, [2] leaves of plates : ill. ; 30 cm. Title page, contents and abstract only. The complete thesis in print form is available from the University Library. Thesis (Ph.D.)--University of Adelaide, Dept. of Paediatrics, 2001

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Freeman, Craig. "The lysosomal degradation of heparan sulphate : a comparative study of the physical and catalytic properties of the heparan sulphate degradative enzymes / by Craig Freeman." Thesis, 1991. http://hdl.handle.net/2440/19747.

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Copies of author's previously published articles inserted Includes bibliographic references 2 v. (various foliations) : ill ; 30 cm. Summary: Studies the enzymology of some of the nine lysosomal exo-enzyme activities which act together to degrade the more highly sulphated regions of the glycosaminoglycans heparin and heparan sulphate. A deficiency of any one of these enzyme activities can result in one of the lysosomal storage disorders collectively known as the Mucopolysaccharidoses (MPS) Thesis (Ph.D.)--University of Adelaide, Dept. of Paediatrics, 1991

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Livres sur le sujet "Glycosaminoglycans Metabolism"

The role of proteoglycans and glycosaminolglycans in aging. Basel: Karger, 1994.

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(Editor), Vittorio Zambotti, ed. Glycolipids, Glycoproteins, and Mucopolysaccharides of the Nervous System. Springer, 1995.

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Frawley, Geoff. Mucopolysaccharidoses. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0064.

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The mucopolysaccharidoses (MPS) are a group of seven chronic progressive diseases caused by deficiencies of 11 different lysosomal enzymes required for the catabolism of glycosaminoglycans (GAGs). Hurler syndrome (MPS IH) is an autosomal recessive storage disorder caused by a deficiency of α‎-L-iduronidase. Hunter syndrome (MPS II) is an X-linked recessive disorder of metabolism involving the enzyme iduronate-2-sulfatase. Many of the MPS clinical manifestations have potential anesthetic implications. Significant airway issues are particularly common due to thickening of the soft tissues, enlar

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Chapitres de livres sur le sujet "Glycosaminoglycans Metabolism"

Linhardt, R. J., D. Loganathan, A. Al-Hakim, and S. A. Ampofo. "Structure and Metabolism of Glycosaminoglycans." In New Trends in Haemostasis, 12–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-84318-1_2.

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Kreysel, H. W., and H. P. Nissen. "Glycosaminoglycan Metabolism." In Hair and Hair Diseases, 255–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74612-3_11.

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Jones, Simon, and Frits A. Wijburg. "Glycosaminoglycans and Oligosaccharides Disorders: Glycosaminoglycans Synthesis Defects, Mucopolysaccharidoses, Oligosaccharidoses and Sialic Acid Disorders." In Inborn Metabolic Diseases, 765–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-63123-2_41.

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Fluharty, Arvan L. "Diseases of Glycosaminoglycan and Proteoglycan Metabolism." In Connective Tissue Disease, 491–521. Boca Raton: CRC Press, 2021. http://dx.doi.org/10.1201/9781003210016-25.

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Kopper, László, József Timár, András Jeney, and Károly Lapis. "Glycosaminoglycan (GAG) Metabolism as a Potential Target to Prevent Metastasis Formation." In Advances in Experimental Medicine and Biology, 367–75. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-5037-6_40.

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HARDINGHAM, TIM. "Proteoglycans and Glycosaminoglycans." In Dynamics of Bone and Cartilage Metabolism, 85–98. Elsevier, 2006. http://dx.doi.org/10.1016/b978-012088562-6/50006-6.

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HARDINGHAM, T. "Proteoglycans and Glycosaminoglycans." In Dynamics of Bone and Cartilage Metabolism, 85–98. Elsevier, 2006. http://dx.doi.org/10.1016/b9-78-012088-5/62650-0066.

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Lachmann, Robin H. "Disorders of Carbohydrate Metabolism." In Oxford Textbook of Endocrinology and Diabetes 3e, edited by John A. H. Wass, Wiebke Arlt, and Robert K. Semple, 1893–901. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198870197.003.0234.

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Sugar molecules play many roles in metabolism. Glucose is an essential source of energy in the body, but carbohydrates also have important structural and signalling functions as constituents of glycoproteins, glycolipids, and glycosaminoglycans. Disorders of carbohydrate metabolism, although caused be defects in individual enzymes, are best viewed as disorders of metabolic pathways. Their tissue pathology can be due to deficiency of a product of metabolism, but just as often it is due to accumulation of toxic molecules which cannot be metabolized. In this chapter, a number of monogenic diseases will be described which involve the monosaccharides glucose, galactose, and fructose, and their roles in intermediary metabolism. The many other inherited metabolic diseases which affect the formation of glycosylated macromolecules (the congenital disorders of glycosylation) or their breakdown (lysosomal storage disorders) will not be discussed.

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Cervós-Navarro, Jorge, and Henry Urich. "Disorders of Glycosaminoglycan Metabolism (Mucopolysaccharidoses)." In Metabolic and Degenerative Diseases of the Central Nervous System, 110–36. Elsevier, 1995. http://dx.doi.org/10.1016/b978-012165250-0/50004-0.

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Actes de conférences sur le sujet "Glycosaminoglycans Metabolism"

Vannucchi, S., F. Pasquali, P. Bianchi-ni, and M. Ruggiero. "BINDING AND METABOLISM OF HEPARIN BY ENDOTHELIAL CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644187.

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In this study we show that bovineadrenal capillary endothelial cells(BACE) contain heparin (HP); this HP has been found associated with the cell surface (i.e; trypsin-removable^and intracellularly. How-ever, experiments with [ sjsodium sulfate labelling, demonstrate that BACE cells donot synthesize HP de novo, but they uptake it from serum. We have studied binding, uptake, and metabolism odifferent molecular weight-HPs: 13 Kd-HP from bovine source, 14 Kd-HP from porcine source, 4.5 Kd, and 2.5-HP fragments. Comparison among different HPs, was carried out by calculating the IC from competition

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Smith, Robert Lane. "Mechanical Loading and Articular Cartilage Metabolism." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2520.

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Abstract Articular cartilage provides diarthrodial joints with a loading-bearing surface that ensures functional motility. The physical characteristics of articular cartilage originate with the highly organized matrix of extracellular macromolecules that provide structural elements to the tissue. The matrix specialization rests with specific proteins produced by the cartilage cells, the chondrocytes that undergo extensive post-translational modification through addition of sulfated glycosaminoglycan and oligosaccharides. The matrix proteins fall into three major categories, the collagens, the

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