Bibliographies thématiques / Green tea

Littérature scientifique sur le sujet « Green tea »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 9 mars 2023

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Articles de revues sur le sujet "Green tea":

Rahman, M., I. A. Jahan, S. Ahmed, K. S. Ahmed, M. Roy, W. Zzaman et I. Ahmad. « Bioactive compounds and antioxidant activity of black and green tea available in Bangladesh ». Food Research 5, n^o 3 (16 mai 2021) : 107–11. http://dx.doi.org/10.26656/fr.2017.5(3).491.

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People in Bangladesh are traditionally used to consume mainly black tea. However, some tea manufacturing companies are now producing green tea, though in a small scale. To create new knowledge as well as awareness about the consumption of green tea, the present study was carried out to compare the black and green tea available in Bangladesh based on their bioactive compounds and antioxidant activity. A total of eight brands of black tea and two brands of green tea were bought from the supermarkets. Total phenolics, total tannin, total flavonoids, and caffeine content were measured as bioactive compounds, and antioxidant activity was evaluated by using two different methods such as DPPH (1,1- diphenyl-2-picrylhydrazyl) radical-scavenging activity and ABTS+ radical scavenging activity of methanol extracts of black and green tea. Every bioactive compound in black and green tea was found to be significantly different (P < 0.05). The total phenolic content, on average, was measured at 242.46 mg GAE/g dry extract and 763.41 mg GAE/ g dry extract in black and green tea, respectively. Black tea contained 6.47 mg TAE/g dry extract tannin, whereas green tea had much more tannin content, 14.51 mg TAE/g dry extract, which is more than double in amount. On the other hand, the total flavonoid content was almost double in black tea (61.82 mg QE/g dry extract) compared to green tea (31.85 mg QE/g dry extract). Antioxidant activities were determined at different concentrations of tea samples. At every concentration, green tea presented higher ABTS+ and DPPH radical scavenging activity than black tea. The highest percentage of inhibition was observed at 20 ppm both in black and green tea, finding 98.50 % and 99.07 % inhibition, respectively. Overall, significantly (P < 0.05) higher amount of phenolic compounds as well as antioxidant activity were observed in green tea.

&NA;. « Green tea ». Reactions Weekly &NA;, n^o 1163 (août 2007) : 14. http://dx.doi.org/10.2165/00128415-200711630-00043.

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&NA;. « Green tea ». Reactions Weekly &NA;, n^o 1132 (décembre 2006) : 10–11. http://dx.doi.org/10.2165/00128415-200611320-00028.

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&NA;. « Green tea ». Reactions Weekly &NA;, n^o 1135 (janvier 2007) : 17. http://dx.doi.org/10.2165/00128415-200711350-00065.

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&NA;. « Green tea ». Reactions Weekly &NA;, n^o 1269 (septembre 2009) : 22. http://dx.doi.org/10.2165/00128415-200912690-00061.

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&NA;. « Green tea ». Reactions Weekly &NA;, n^o 1249 (avril 2009) : 23. http://dx.doi.org/10.2165/00128415-200912490-00074.

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&NA;. « Green tea ». Reactions Weekly &NA;, n^o 1308 (juillet 2010) : 20. http://dx.doi.org/10.2165/00128415-201013080-00055.

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Hutcheon, Deborah A., et Jane Ziegler. « Green Tea ». Topics in Clinical Nutrition 29, n^o 3 (2014) : 268–77. http://dx.doi.org/10.1097/tin.0000000000000004.

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Hume, Anne L. « Green tea ». Pharmacy Today 25, n^o 9 (septembre 2019) : 16. http://dx.doi.org/10.1016/j.ptdy.2019.08.007.

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Guslandi, Mario. « Green tea ». Alimentary Pharmacology & ; Therapeutics 19, n^o 10 (29 avril 2004) : 1135. http://dx.doi.org/10.1111/j.1365-2036.2004.01953.x.

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Thèses sur le sujet "Green tea":

Yu, Sze-tak. « Effects of Chinese green tea and tea catechins on lipolysis ». Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21106137.

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余詩德 et Sze-tak Yu. « Effects of Chinese green tea and tea catechins on lipolysis ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31969677.

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Cheng, Tak-him Terence. « Neuroprotective effect of green tea extracts ». Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/b40203517.

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何禮昌 et Lai-cheong Ho. « Effects of green tea on ovariectomized rats ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31970540.

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Ho, Lai-cheong. « Effects of green tea on ovariectomized rats ». Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp ?

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Mudau, Fhatuwani N. « Growth, development and chemical composition of bush tea (Athrixia phylicoides L.) as affected by seasonal nitrogen, phosphorus and potassium nutrition ». Pretoria : [s.n.], 2006. http://upetd.up.ac.za/thesis/available/etd-08242006-133149.

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McArdle, Nicholas J. « The antigenotoxic effect of tea ». Thesis, University of Surrey, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390574.

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Avila, Alejandra. « Experimental study of the effects of green tea on improving the outcomes of BALB/c mice infected with Leishmania Mexicana ». To access this resource online via ProQuest Dissertations and Theses @ UTEP, 2009. http://0-proquest.umi.com.lib.utep.edu/login?COPT=REJTPTU0YmImSU5UPTAmVkVSPTI=&clientId=2515.

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Charoenchon, Nisamanee. « Can green tea catechin supplement protect against photoageing ? » Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/can-green-tea-catechin-supplement-protect-against-photoageing(64eefb5f-ef37-4900-9c03-3477c8a74e50).html.

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Photoaged skin caused by chronic ultraviolet radiation (UVR) is characterised clinically with hyperpigmentation, coarse skin texture and deep wrinkles; the worst outcome is skin cancer. Histological investigation of the alteration within major extracellular matrices (ECM; elastic fibres, fibrillar collagens) is essential study to understand the cellular effect on skin structure from UVR. This thesis used an acute dose of radiation to examine in humans in vivo the effect of UVR on ECM components before assessing whether a dietary intervention could protect skin from UVR damage. Green tea catechins (GTCs) have anti-oxidant properties and may be an interesting option as a systemic photoprotection agent. Hence this thesis assesses: 1) the effect of acute irradiation of skin on dermal ECM damage to see whether it mimics the changes observed in photoageing and; 2) whether dietary supplementation with GTC will provide dermal ECM protection. UV-induced change in elastic fibre network. Initially, the effect of two different UV light sources on elastic fibre protein (elastic fibres, fibrillin-rich microfibrils and fibulin-2 and -5 microfibrils) remodelling was performed. The effect of ultraviolet B vs full-spectrum solar simulated radiation (SSR) were investigated in a small sample of healthy Caucasian volunteers (n = 6 per group). At 24 hour after 3× MED irradiation, Weigert's resorcin–fuchsin stained elastic fibres showed a significant reduction regardless of irradiation protocol (UVB, P<0.01; SSR P<0.05). Specific components were identified by immunohistochemistry; a significant reduction in fibrillin-rich microfibrils (FRM) was observed in UVB-irradiated skin (P<0.05), whilst fibulin-5-positive microfibrils were only affected by SSR (P<0.05). The data revealed, therefore, differential effects on UV wavelength on ECM remodelling. SSR, the more physiologically relevant light source was used in subsequent studies Supplement effect in SSR-induced damage in elastic fibre. Fifty healthy volunteers were recruited to this randomised control trial to investigate whether GTC can protect skin from photodamage. Volunteers were randomized to GTC (1080 mg plus 100 mg vitamin C; n=25) or placebo (maltodextrin; n = 25) daily for 12-weeks with compliance assessed biochemically in urine samples. Of the n = 50 recruited, 44 volunteers completed the study. In baseline, UVR challenge resulted in a significant remodeling of the cutaneous elastic fiber system (P<0.001), particularly fibulin-2 and fibulin-5-positive microfibrils at 24-hr after 3×MED irradiation. In post-supplementation, fibulin-5 positive microfibrils were protected from UVR remodeling (% staining, mean ± SE; no UV, 18.1±0.89; UVR, 17.1±0.61; P=0.30) whilst no protection was seen in the placebo group (no UVR, 19.41±0.79; UVR, 17.69±0.61; P<0.05). Supplement effect in SSR-induced damage in collagenous matrix. In the identical experiment, collagenous matrices including synthesis of procollagen I was also examined as fibrillar collagens are the major ECM components providing strength within dermis. The fibrillar collagen and newly synthesised procollagen I were stained by Picrosirius red and immunohistochemistry respectively. At baseline, acute irradiation significantly reduced papillary dermal fibrillar collagens (P<0.001) and induced deposition of newly synthesised pro-collagen I (P=0.02). In post-supplementation, GTC enhanced the deposition of thin collagen fibres in the dermis. Whilst placebo showed no effect on the altered organisation of fibrillar collagens or deposition of pro-collagen I following the irradiation challenge, GTC protected the organisation of fibrillar collagens in the papillary dermis (P=0.97).This novel in vivo human study may be used to recapitulate elastic fibre and collagen changes associated with photoageing and may be useful for dissecting out the mechanisms underlying extracellular matrix damage in response to chronic sunlight exposure. Furthermore, in a randomized control trial, dietary GTC protected fibulin-5 microfibrils and collagen fibres in the papillary dermis from UV-mediated degradation. The mechanism by which this protection occurs requires further study.

Zheng, Yuanyuan. « PROTECTION AGAINST ENDOTHELIAL INFLAMMATION BY GREEN TEA FLAVONOIDS ». UKnowledge, 2010. http://uknowledge.uky.edu/gradschool_diss/64.

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Endothelial inflammation is a pivotal early event in the development of atherosclerosis. Long term exposure to cardiovascular risk factors will ultimately exhaust those protective anti-inflammatory factors such as the heme oxygenase (HO) system. The HO system plays a critical role in cellular and tissue self-defense against oxidative stress and inflammation. Caveolae are membrane domains and are particularly abundant in endothelial cells, where they are believed to play a major role in the regulation of endothelial vesicular trafficking as well as the uptake of lipids and related lipophilic compounds, possibly including bioactive food components such as flavonoids. Research in this dissertation addresses the role of HO-1 and caveolae on dietary flavonoid epigallocatechin gallate (EGCG) mediated protection against pro-inflammatory cytokine tumor necrosis factor-α (TNF-α) and linoleic acid-induced activation of endothelial cells. The data support the hypothesis that EGCG protects against TNF-α-induced monocyte recruitment and adhesion partially through the induction of HO-1 and bilirubin. The observed anti-inflammatory effects of EGCG are mimicked by the HO-1 inducer cobalt protoporphyrin (CoPP) and abolished by HO-1 gene silencing. Nrf2 is the major transcription factor of phase II antioxidant enzymes including HO-1. Results clearly show that EGCG-induced HO-1 expression and subsequent bilirubin productions are dependent on functional Nrf2. EGCG also can down-regulate the base-line level of caveolin-1. Furthermore, silencing of the caveolin-1 gene can markedly down-regulate linoleic acid-induced COX-2 and MCP-1, indicating that caveolae may be a critical platform regulating inflammatory signaling pathways. Similar to EGCG treatment, silencing of caveolin-1 can also result in the activation of Nrf2, up-regulation of HO-1 and bilirubin. This may be one of the mechanisms to explain the protection effect of caveolin-1 gene silencing against endothelial inflammation. Moreover, EGCG rapidly accumulates in caveolae, which is associated with caveolin-1 displacement from the plasma membrane towards the cytosol. Caveolin-1 gene silencing can significantly reduce the uptake of EGCG in endothelial cells within 30 min. These data suggest that caveolae may play a role in the uptake and transport of EGCG in endothelial cells. These studies provide a novel target through which EGCG functions to protect against inflammatory diseases such as atherosclerosis.

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Livres sur le sujet "Green tea":

Taylor, Nadine. Green tea. New York : Kensington Books, 1998.

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Williams, Marie. Green vanilla tea. Sydney : Finch Publishing, 2013.

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Zhang, Yuan. Lü cha = : Green tea. 8^e éd. Beijing : Hua yi chu ban she, 2003.

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Mitscher, Lester A. The Green Tea Book. New York : Penguin Group USA, Inc., 2008.

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Toews, Victoria Dolby. All about green tea. Garden City Park, N.Y : Avery, 1998.

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Rosen, Diana. The book of green tea. Pownal, Vt : Storey Books, 1998.

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Canadian Breast Cancer Research Initiative., dir. Green tea : An information package. Toronto : Canadian Breast Cancer Research Initiative, 1996.

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Allāh, Yaḥyá al-Ṭāhir ʻAbd. The mountain of green tea. Cairo, Egypt : American University in Cairo Press, 1991.

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Fanu, Joseph Sheridan Le. Green Tea. Independently Published, 2017.

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Fanu, Joseph Sheridan Le. Green Tea. Independently Published, 2020.

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Chapitres de livres sur le sujet "Green tea":

Takeo, T. « Green and semi-fermented teas ». Dans Tea, 413–57. Dordrecht : Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2326-6_13.

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Houston, Neil, et Alexa Boer Kimball. « Green Tea Extract ». Dans Cosmeceuticals and Cosmetic Practice, 122–32. Chichester, UK : John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118384824.ch12.

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Gilinsky, Armand, et Wakako Kusumoto. « Koots Green Tea ». Dans Comparative Entrepreneurship Initiatives, 276–99. London : Palgrave Macmillan UK, 2011. http://dx.doi.org/10.1057/9780230314368_11.

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Rashidinejad, Ali. « Green Tea Catechins ». Dans Tea as a Food Ingredient, 33–69. Boca Raton : CRC Press, 2022. http://dx.doi.org/10.1201/9781003152828-3.

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Koide, Shoji, et Ken-ichi Kimura. « Japanese Green Tea ». Dans Tea as a Food Ingredient, 81–100. Boca Raton : CRC Press, 2022. http://dx.doi.org/10.1201/9781003152828-5.

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Fujiki, Hirota. « Green Tea Cancer Prevention ». Dans Encyclopedia of Cancer, 1–5. Berlin, Heidelberg : Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_6592-7.

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Kuroda, Yukiaki, et Yukihiko Hara. « Green Tea in Japan ». Dans Health Effects of Tea and Its Catechins, 1–10. Boston, MA : Springer US, 2004. http://dx.doi.org/10.1007/978-1-4757-5390-5_1.

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Lunder, T. L. « Catechins of Green Tea ». Dans ACS Symposium Series, 114–20. Washington, DC : American Chemical Society, 1992. http://dx.doi.org/10.1021/bk-1992-0507.ch009.

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Anastassakis, Konstantinos. « Green Tea (Camellia sinensis) ». Dans Androgenetic Alopecia From A to Z, 451–57. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-08057-9_52.

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Fujiki, Hirota. « Green Tea Cancer Prevention ». Dans Encyclopedia of Cancer, 1960–65. Berlin, Heidelberg : Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_6592.

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Actes de conférences sur le sujet "Green tea":

Wibisono, W., Yufitri Mayasari, D. Putra et I. Ariesta. « Black Tea and Green Tea in Reducing Children Dental Caries ». Dans International Conference on Environmental Awareness for Sustainable Development in conjunction with International Conference on Challenge and Opportunities Sustainable Environmental Development, ICEASD & ICCOSED 2019, 1-2 April 2019, Kendari, Indonesia. EAI, 2019. http://dx.doi.org/10.4108/eai.1-4-2019.2287267.

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Rong-Xiang Zhang, Wen-Li Wang, Guang Li, Xiao-Hui Zhao, Lian-Shui Zhang et Xiao-Wei Li. « The grade recognition of green tea ». Dans 2008 International Conference on Machine Learning and Cybernetics (ICMLC). IEEE, 2008. http://dx.doi.org/10.1109/icmlc.2008.4620639.

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Tai, Melvin Jia-Yong, Wei-Wen Liu, Cheng-Seong Khe, N. M. S. Hidayah, Yi-Peng Teoh, C. H. Voon, H. Cheun Lee et P. Y. P. Adelyn. « Green synthesis of reduced graphene oxide using green tea extract ». Dans 4TH ELECTRONIC AND GREEN MATERIALS INTERNATIONAL CONFERENCE 2018 (EGM 2018). Author(s), 2018. http://dx.doi.org/10.1063/1.5080845.

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Megawati, Teni Ernawati, Lia Meilawati, Indah D. Dewijanti et Edi Supriadi. « Formulation of herbal tea drinks by adding green tea to improve antioxidant activities ». Dans PROCEEDINGS OF THE 5TH INTERNATIONAL SYMPOSIUM ON APPLIED CHEMISTRY 2019. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5134577.

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Zulliati, Zulliati, et Nurul Hidayah. « Green Tea and The Preeclampsia in Intra Partum ». Dans Proceedings of the First National Seminar Universitas Sari Mulia, NS-UNISM 2019, 23rd November 2019, Banjarmasin, South Kalimantan, Indonesia. EAI, 2020. http://dx.doi.org/10.4108/eai.23-11-2019.2298376.

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Priyandoko, Didik, Wahyu Widowati, Hanna Sari Widya Kusuma, Ervi Afifah, Cahyaning Riski Wijayanti, Cintani Dewi Wahyuni, Amannah Mutmainnah Idris, Rizka Amelia Putdayani et Rizal Rizal. « Antioxidant Activity of Green Tea Extract and Myricetin ». Dans 2021 IEEE International Conference on Health, Instrumentation & Measurement, and Natural Sciences (InHeNce). IEEE, 2021. http://dx.doi.org/10.1109/inhence52833.2021.9537285.

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Mystrioti, Christiana, Anthimos Xenidis, Nymphodora Papassiopi, Dimitris Dermatas et Mariza Chrysochoou. « Fate of Green Tea Iron Nanoparticles in Calcareous Soils ». Dans Geo-Congress 2014. Reston, VA : American Society of Civil Engineers, 2014. http://dx.doi.org/10.1061/9780784413272.213.

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Mali, Suraj N., et Anima Pandey. « Naturally Occurring Green Tea Polyphenols as Anti-Mycobacterial Agents ». Dans ECMS 2021. Basel Switzerland : MDPI, 2021. http://dx.doi.org/10.3390/ecms2021-10844.

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Srimanothip, Anjaly, et Phakkharawat Sittiprapaporn. « Effect of Drinking Green Tea to Stress Reduction : Electroencephalographic Investigation ». Dans 2019 16th International Conference on Electrical Engineering/Electronics, Computer, Telecommunications and Information Technology (ECTI-CON). IEEE, 2019. http://dx.doi.org/10.1109/ecti-con47248.2019.8955191.

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Rahmawati, Sela, Jadi Suprijadi et Zulhanif. « Text mining factor analysis (TFA) in green tea patent data ». Dans STATISTICS AND ITS APPLICATIONS : Proceedings of the 2nd International Conference on Applied Statistics (ICAS II), 2016. Author(s), 2017. http://dx.doi.org/10.1063/1.4979456.

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Rapports d'organisations sur le sujet "Green tea":

Mukhtar, Hasan. Green Tea in Prevention and Therapy of Prostate Cancer. Fort Belvoir, VA : Defense Technical Information Center, juillet 2001. http://dx.doi.org/10.21236/ada398205.

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Cobrinik, David E. Effect on Green Tea Polyphenols on Breast Cancer Signaling. Fort Belvoir, VA : Defense Technical Information Center, avril 1999. http://dx.doi.org/10.21236/ada367380.

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Gupta, Sanjay. Green Tea in Prevention and Therapy of Prostate Cancer. Fort Belvoir, VA : Defense Technical Information Center, septembre 2002. http://dx.doi.org/10.21236/ada410754.

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Samavat, Hamed. Green Tea Modulation of Obesity and Breast Cancer Risk. Fort Belvoir, VA : Defense Technical Information Center, avril 2013. http://dx.doi.org/10.21236/ada581017.

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Cobrinik, David. Effect of Green Tea Polyphenols on Breast Cancer Signaling. Fort Belvoir, VA : Defense Technical Information Center, avril 2000. http://dx.doi.org/10.21236/ada392163.

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Mukhtar, Hasan. Sustained Release Oral Nanoformulated Green Tea for Prostate Cancer. Fort Belvoir, VA : Defense Technical Information Center, mai 2011. http://dx.doi.org/10.21236/ada545577.

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Mukhtar, Hasan. Sustained Release Oral Nanoformulated Green Tea for Prostate Cancer Prevention. Fort Belvoir, VA : Defense Technical Information Center, mai 2013. http://dx.doi.org/10.21236/ada585226.

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Mukhtar, Hasan, Nihal Ahmad, Vaqar M. Adhami et Naghma Khan. Sustained Release Oral Nanoformulated Green Tea for Prostate Cancer Prevention. Fort Belvoir, VA : Defense Technical Information Center, mai 2012. http://dx.doi.org/10.21236/ada589659.

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bu, peili. Effect of green tea supplementation on blood pressure：a systematic review and dose-response meta-analysis of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review Protocols, avril 2020. http://dx.doi.org/10.37766/inplasy2020.4.0021.

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Bigaud, Mariah A., et Anna Kam-Ha Yeung-Cheung. The in vitro Studies of the Inhibitory Effect of Green Tea (Camellia sinensis) on Pseudomonas aeruginosa Treated Contact Lenses. Journal of Young Investigators, avril 2017. http://dx.doi.org/10.22186/jyi.32.4.25-29.

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