Se connecter

Bibliographies thématiques / Gwashu / Livres

Pour voir les autres types de publications sur ce sujet consultez le lien suivant : Gwashu.

Livres sur le sujet « Gwashu »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 16 février 2022

Créez une référence correcte selon les styles APA, MLA, Chicago, Harvard et plusieurs autres

Choisissez une source :

Consultez les 42 meilleurs livres pour votre recherche sur le sujet « Gwashu ».

À côté de chaque source dans la liste de références il y a un bouton « Ajouter à la bibliographie ». Cliquez sur ce bouton, et nous générerons automatiquement la référence bibliographique pour la source choisie selon votre style de citation préféré : APA, MLA, Harvard, Vancouver, Chicago, etc.

Vous pouvez aussi télécharger le texte intégral de la publication scolaire au format pdf et consulter son résumé en ligne lorsque ces informations sont inclues dans les métadonnées.

Parcourez les livres sur diverses disciplines et organisez correctement votre bibliographie.

1

Dowd, Anthony. The special bindings of Gwasg Gregynog : An illustrated catalogue of the special bindings produced at Gregynog between 1977 and 2002. Newtown, Powys : Gwasg Gregynog, 2004.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

2

Dowd, Anthony. The special bindings of Gwasg Gregynog : An illustrated catalogue of the special bindings produced at Gregynog between 1977 and 2002. Newtown : Gwasg Gregynog, 2004.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

3

Creu argraff : Atgofion teulu Gwasg Gomer. Llandysul, Ceredigion : Gomer, 2012.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

4

Hughes, Mair Wynn. Gwas y stabl. Llandysul : Gomer, 2004.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

5

Gwas y gwaredwr. Abertawe : T^y John Penry, 1991.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

6

Nicieja, Stanisław Sławomir. Z Kijowa na Piccadilly : Wokół biografii Tadeusza Zabłockiego-Gwasza. Opole : Wydawn. Helionia, 1993.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

7

Z kijowa na Picadilly : Wokół biografii Tadeusza Zabłockiego-Gwasza. Opole : Helionia, 1994.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

8

The archaeology of Rutland Water : Excavations at Empingham in Gwash Valley, Rutland, 1967-73 and 1990. Leicester : University of Leicester Archaeological Services, 2000.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

9

Harrop, Dorothy A. The Gregynog Press Gwasg Gregynog : Three essays Tair ysgrif. [Newtown, Powys] : [Gwasg Gregynog], 2003.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

10

Glaz, Kazimir. From the Essence Series : Istota rzeczy : works on paper, gwasze, 1985-2005. Toronto : Toronto Center for Contemporary Art, 2005.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

11

Davies, Ieuan. Emlyn G. Jenkins : Gwas i Grist. Abertawe : Ieuan Davies, 2004.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

12

Esslemont, David. Gwasg Gregynog : A descriptive catalogue of printing at Gregynog 1970-1990. Newtown, Powys, Wales : Gwasg Gregynog, 1990.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

13

Williams, Meirion Llewelyn. Gwas yr achos mawr : Cofiant Huw Llewelyn Williams. Dinbych : Gee, 1991.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

14

Evans, Jayne. Gregynog Press : Catalogue of an exhibition of Gregynog books from the Collection of Manchester Polytechnic Library. Manchester : Manchester Polytechnic Library, 1990.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

15

Providing for certain lands to be held in trust for the Utu Utu Gwaitu Paiute Tribe : Report (to accompany H.R. 854). [Washington, D.C : U.S. G.P.O., 2006.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

16

To provide for certain lands to be held in trust for the Utu Utu Gwaitu Paiute Tribe : Report (to accompany H.R. 854) (including cost estimate of the Congressional Budget Office). [Washington, D.C : U.S. G.P.O., 2006.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

17

United States. Congress. House. Committee on Resources. To provide for certain lands to be held in trust for the Utu Utu Gwaitu Paiute Tribe : Report (to accompany H.R. 854) (including cost estimate of the Congressional Budget Office). [Washington, D.C : U.S. G.P.O., 2006.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

18

Kaduna State (Nigeria). Judicial Commission of Inquiry into the Disturbances of 2nd November, 2001 in Gwantu Town of Sanga Local Government and the Affairs of the Local Government Councils Indicted by the Kaduna State House of Assembly Committee. White paper on the interim report of the Kaduna State Judicial Commission of Inquiry into the Disturbances of 2nd November, 2001 in Gwantu Town of Sanga Local Government and the Affairs of the Local Government Councils Indicted by the Kaduna State House of Assembly Committee. [Kaduna : s.n.], 2002.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

19

Pastoral Fulbe Family in Gwandu. Taylor & Francis Group, 2018.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

20

Hopen, C. Edward. Pastoral Fulbe Family in Gwandu. Taylor & Francis Group, 2020.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

21

Hopen, C. Edward. The Pastoral Fulbe Family in Gwandu. Routledge, 2018. http://dx.doi.org/10.4324/9780429488955.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

22

Petro, Pamela. Gwasg Gregynog : The reincarnation of a press. 1987.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

23

Oldham, Mary. Gwasg Gregynog : The next ten years. 1989.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

24

Moore. Six Dinner Sid - Gwasg Y Dre. Hodder & Stoughton Childrens Division, 1992.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

25

A, Harrop Dorothy, Hughes Glyn Tegai 1923-, Vickers David 1969- et Gregynog Press, dir. The Gregynog Press : Three essays = Gwasg Gregynog : tair ysgrif. Powys : Gwasg Gregynog, 2003.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

26

From the Essence Series = : Istota Rzeczy : Works on Paper/Gwasze, 1985-2005. Not Avail, 2005.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

27

Erdmann, Jeanette, et Tanja Zeller, dir. From GWAS Hits to Treatment Targets. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-982-7.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

28

Cyfaill Pwy O'r Hen Wlad ? : Gwasg Gyfnodol Gymraeg America, 1838-1866. Gwasg Prifysgol Cymru / University of Wales Press, 2017.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

29

Ebstein, Richard P., Songfa Zhong, Robin Chark, Poh San Lai et Soo Hong Chew. Modeling the Genetics of Social Cognition in the Laboratory. Sous la direction de Turhan Canli. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.017.

Texte intégral

Résumé :

This chapter examines recent advances in the genetics of social cognition, discussing evidence from twin studies that confirm the relevancy of genetic hard wiring in understanding many social phenotypes, with important implications for the social sciences and for genome-wide association studies (GWAS) that may identify specific genes contributing to a wide range of social phenotypes, genoeconomics, and individual and social decision making. Stressing the importance of phenotype definition and precise measurement as key to success in GWAS, the authors argue that laboratory-based behavioral economic paradigms using ethnically homogenous student populations generate the best prospects for successful GWAS. Also discussed are the neurochemical/neurogenetic architecture of behavioral economic games that measure individual and social decision making and the considerable progress made in unraveling the neurogenetics of human parenting and the beginning of a political attitudes neuroscience. The authors’ own GWAS is used to present a set of guidelines for future research directions.

Styles APA, Harvard, Vancouver, ISO, etc.

30

Hedd Wyn 1887-1917 : Herdenking = coffâd = commemoration : Langemark-Ieper 31 vii 1992 : persoverzicht = adolygiad gwasg = press review. [Ieper] : [s.n.], 1992.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

31

Levinson, Douglas F., et Walter E. Nichols. Genetics of Depression. Sous la direction de Dennis S. Charney, Eric J. Nestler, Pamela Sklar et Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0024.

Texte intégral

Résumé :

Major depressive disorder (MDD) is a common and heterogeneous complex trait. Twin heritability is 35%–40%, perhaps higher in severe/recurrent cases. Adverse life events (particularly during childhood) increase risk. Current evidence suggests some overlap in genetic factors among MDD, bipolar disorder, and schizophrenia. Large genome-wide association studies (GWAS) are now proving successful. Polygenic effects of common SNPs are substantial. Findings implicate genes with effects on synaptic development and function, including two obesity-associated genes (NEGR1 and OLFM4), but not previous “candidate genes.” It can now be expected that larger GWAS samples will produce additional associations that shed new light on MDD genetics.

Styles APA, Harvard, Vancouver, ISO, etc.

32

Zhang, Yuan-Ming, Zhenyu Jia et Jim M. Dunwell, dir. The Applications of New Multi-Locus GWAS Methodologies in the Genetic Dissection of Complex Traits. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-834-9.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

33

Merriman, Tony R. The genetic basis of gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0040.

Texte intégral

Résumé :

An individual’s risk of gout is determined by a complex relationship between inherited genetic variants and environmental exposures. Genetic variants that control hyperuricaemia and subsequent progression to clinical gout specify pathogenic pathways that could be therapeutically targeted. Genome-wide association studies (GWAS) have provided novel insights into the pathways leading to hyperuricaemia. GWAS have identified the renal uric acid transporter SLC2A9/GLUT9 and the gut excretory molecule ABCG2, which each have very strong genetic effects in the control of urate levels and risk of gout. Histone deacetylase inhibitors are able to correct the genetically-determined ABCG2 dysfunction. Other renal uric acid transporters, such as SLC22A11/OAT4 and SLC22A12/URAT1 have been confirmed to be genetically associated with urate and the risk of gout. Genes that generate urate during glycolysis (e.g. GCKR) are also implicated. In contrast very little is known about genetic variants that control the progression from hyperuricaemia to gout with the toll-like receptor 4 gene being the only gene with replicated evidence of association.

Styles APA, Harvard, Vancouver, ISO, etc.

34

GWAS : the Rise of Hypothesis-Free Biomedical Science : Could Genome-Wide Association Studies Transform Modern Medicine ? Elsevier Science & Technology Books, 2016.

Trouver le texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

35

Perovic, Dragan, Hikmet Budak, Kazuhiro Sato et Pierre Sourdille, dir. Use of Barley and Wheat Reference Sequences : Downstream Applications in Breeding, Gene Isolation, GWAS and Evolution. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-963-2.

Texte intégral

Styles APA, Harvard, Vancouver, ISO, etc.

36

Lewis, Myles, et Tim Vyse. Genetics of connective tissue diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0042.

Texte intégral

Résumé :

The advent of genome-wide association studies (GWAS) has been an exciting breakthrough in our understanding of the genetic aetiology of autoimmune diseases. Substantial overlap has been found in susceptibility genes across multiple diseases, from connective tissue diseases and rheumatoid arthritis (RA) to inflammatory bowel disease, coeliac disease, and psoriasis. Major technological advances now permit genotyping of millions of single nucleotide polymorphisms (SNPs). Group analysis of SNPs by haplotypes, aided by completion of the Hapmap project, has improved our ability to pinpoint causal genetic variants. International collaboration to pool large-scale cohorts of patients has enabled GWAS in systemic lupus erythematosus (SLE), systemic sclerosis and Behçet's disease, with studies in progress for ANCA-associated vasculitis. These 'hypothesis-free' studies have revealed many novel disease-associated genes. In both SLE and systemic sclerosis, identified genes map to known pathways including antigen presentation (MHC, TNFSF4), autoreactivity of B and T lymphocytes (BLK, BANK1), type I interferon production (STAT4, IRF5) and the NFκ‎B pathway (TNIP1). In SLE alone, additional genes appear to be involved in dysregulated apoptotic cell clearance (ITGAM, TREX1, C1q, C4) and recognition of immune complexes (FCGR2A, FCGR3B). Future developments include whole-genome sequencing to identify rare variants, and efforts to understand functional consequences of susceptibility genes. Putative environmental triggers for connective tissue diseases include infectious agents, especially Epstein-Barr virus; cigarette smoking; occupational exposure to toxins including silica; and low vitamin D, due to its immunomodulatory effects. Despite numerous studies looking at toxin exposure and connective tissue diseases, conclusive evidence is lacking, due to either rarity of exposure or rarity of disease.

Styles APA, Harvard, Vancouver, ISO, etc.

37

Walsh, Bruce, et Michael Lynch. The Neutral Divergence of Quantitative Traits. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198830870.003.0012.

Texte intégral

Résumé :

The joint action of genetic drift and mutation results in the divergence of trait means over time. This chapter examines the expected amount of divergence, which forms the basis for a number of tests on whether an observed pattern is either too large relative to drift (suggesting directional selection) or two small (suggesting stabilizing selection). It then applies these results to examine tests for selection over a very diverse range of data sets, ranging from a stratophenetic series of fossils to divergence in gene expression over time. It also examines a number of trait-augmented marked-based tests (such as using the QTLs or GWAS hits for a trait) for departures from neutrality.

Styles APA, Harvard, Vancouver, ISO, etc.

38

Poretti, Andrea, et Michael V. Johnston. Genetic Disorders and Stroke. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0110.

Texte intégral

Résumé :

A variety of monogenic and polygenic genetic disorders have been linked to stroke, making it important for the clinician to keep up with the new discoveries and the potential to provide new gene-based therapies. Hematologic disorders such as sickle cell disease and thrombophilia due to mutations in prothrombin, factor V Leiden, and homocysteine metabolism are fairly well known, but mutations in mitochondrial metabolism and matrix metalloproteinases are less recognized. In addition, results of genome-wide association studies (GWAS) in stroke populations are revealing mutations that could predispose to stroke in specific ethnic populations. These studies are also revealing some crossover in mutations between stroke and familial hemiplegic migraine as well as mutations in growth factors such as brain derived neurotrophic factor (BDNF) that appear to influence the recovery from stroke by altering cortical plasticity.

Styles APA, Harvard, Vancouver, ISO, etc.

39

Purcell, Shaun M. Genetic Methodologies and Applications. Sous la direction de Dennis S. Charney, Eric J. Nestler, Pamela Sklar et Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0001.

Texte intégral

Résumé :

Mental illness is highly heritable, yet it has been difficult historically to identify the specific genes that comprise that risk. This difficulty resides in the fact that the genetic risk for all common mental disorders is polygenic, with perhaps hundreds of genetic variations, each of small effect, contributing to the overall risk. Despite these challenges, the field has made dramatic advances over the past decade in beginning to understand the genetic basis of mental illness. This chapter provides an overview of the experimental approaches used, beginning with epidemiology and population genetics to define the heritability of an illness, to classic studies of large families and linkage disequilibrium analysis, to genome-wide investigations including genome-wide association studies (GWAS), exome sequencing, and whole genome sequencing. Increasingly, these genetic advances are being understood within the biological context of disease pathophysiology.

Styles APA, Harvard, Vancouver, ISO, etc.

40

Langley, Kate. ADHD genetics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198739258.003.0003.

Texte intégral

Résumé :

This chapter reviews the evidence suggesting that there is a strong genetic component to ADHD and the efforts to identify the specific genetic factors that might be involved. It discusses the different types of genetic contributions, from common to rare variants, and the evidence that these are involved in the aetiology of the disorder. An overview of the methodological strategies employed, including genome-wide association studies (GWAS), polygenic risk score, and copy number variant (CNV) analyses, is undertaken, as well as discussion of the strengths and pitfalls of such work. The contradictory findings in the field and controversies that arise as a result are also explored. Finally, this chapter considers how the heritability of ADHD and specific genetic factors involved need to be examined in the context of clinical factors such as comorbidity and how these factors affect investigations into the genetics of ADHD.

Styles APA, Harvard, Vancouver, ISO, etc.

41

Hyde, Luke W., Ryan Bogdan et Ahmad Hariri. Neurogenetics of Individual Differences in Brain, Behavior, and Risk for Psychopathology. Sous la direction de Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.007.

Texte intégral

Résumé :

Neurogenetics research is advancing understanding of how genetic variation gives rise to individual differences in brain function, which, in turn, shapes behavior and risk for psychopathology. Despite these advancements, neurogenetics research is currently confronted by three major challenges: 1) conducting research on individual variables with small effects, 2) absence of detailed mechanisms, and 3) a need to translate findings towards greater clinical relevance. This essay showcases techniques and developments that address these challenges and highlights the benefits of a neurogenetics approach to understanding brain, behavior, and risk for psychopathology. To address the challenge of small effects, we explore approaches including incorporating the environment, modeling epistatic relationships, and using multilocus profiles. To address the challenge of mechanism, we explore how nonhuman animal research, epigenetics research, and GWAS can inform our mechanistic understanding of behaviorally relevant brain function. Finally, to address the challenge of clinical relevance, the essay examines how neurogenetics research can identify novel therapeutic targets and identify for whom treatments work best. By addressing these challenges neurogenetics research is poised to exponentially increase our understanding of how genetic variation interacts with the environment to shape the brain, behavior and risk for psychopathology.

Styles APA, Harvard, Vancouver, ISO, etc.

42

Thun, Michael J., Martha S. Linet, James R. Cerhan, Christopher A. Haiman et David Schottenfeld. Introduction. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0001.

Texte intégral

Résumé :

This Introduction provides a broad overview of the scientific advances and crosscutting developments that increasingly influence epidemiologic research on the causes and prevention of cancer. High-throughput technologies have identified the molecular “driver” events in tumor tissue that underlie the multistage development of many types of cancer. These somatic (largely acquired) alterations disrupt normal genetic and epigenetic control over cell maintenance, division and survival. Tumor classification is also changing to reflect the genetic and molecular alterations in tumor tissue, as well as the anatomic, morphologic, and histologic phenotype of the cancer. Genome-wide association studies (GWAS) have identified more than 700 germline (inherited) genetic loci associated with susceptibility to various forms of cancer, although the risk estimates for almost all of these are small to modest and their exact location and function remain to identified. Advances in genomic and other “OMIC” technologies are identifying biomarkers that reflect internal exposures, biological processes and intermediate outcomes in large population studies. While research in many of these areas is still in its infancy, mechanistic and molecular assays are increasingly incorporated into etiologic studies and inferences about causation. Other sections of the book discuss the global public health impact of cancer, the growing list of exposures known to affect cancer risk, the epidemiology of over 30 types of cancer by tissue of origin, and preventive interventions that have dramatically reduced the incidence rates of several major cancers.

Styles APA, Harvard, Vancouver, ISO, etc.

Nous offrons des réductions sur tous les plans premium pour les auteurs dont les œuvres sont incluses dans des sélections littéraires thématiques. Contactez-nous pour obtenir un code promo unique!