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Littérature scientifique sur le sujet « HIPPO-PATHWAY-YAP »

Auteur : Grafiati

Publié le 1 avril 2023

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Articles de revues sur le sujet "HIPPO-PATHWAY-YAP"

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Matthaios, Dimitrios, Maria Tolia, Davide Mauri, Konstantinos Kamposioras, and Michalis Karamouzis. "YAP/Hippo Pathway and Cancer Immunity: It Takes Two to Tango." Biomedicines 9, no. 12 (2021): 1949. http://dx.doi.org/10.3390/biomedicines9121949.

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Hippo pathway with its main molecule YAP is a crucial pathway for development, tissue homeostasis, wound healing, tissue regeneration, and cancer. In this review, we discuss the multiple effects of the YAP/Hippo pathway in the immune system and cancer. We analyzed a series of effects: extracellular vesicles enhanced immunity through inhibition of LATS1/2, ways of modulation of the tumor microenvironment, YAP- and TAZ-mediated upregulation of PDL1, high expression of YAP and PDL1 in EGFR-TKI-resistant cells, enhanced YAP activity in inflammation, and the effect of the Hippo pathway on T cells,

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Salem and Hansen. "The Hippo Pathway in Prostate Cancer." Cells 8, no. 4 (2019): 370. http://dx.doi.org/10.3390/cells8040370.

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Despite recent efforts, prostate cancer (PCa) remains one of the most common cancers in men. Currently, there is no effective treatment for castration-resistant prostate cancer (CRPC). There is, therefore, an urgent need to identify new therapeutic targets. The Hippo pathway and its downstream effectors—the transcriptional co-activators, Yes-associated protein (YAP) and its paralog, transcriptional co-activator with PDZ-binding motif (TAZ)—are foremost regulators of stem cells and cancer biology. Defective Hippo pathway signaling and YAP/TAZ hyperactivation are common across various cancers. H

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Agarinis, C., V. Orsini, P. Megel, et al. "Activation of Yap-Directed Transcription by Knockdown of Conserved Cellular Functions." Journal of Biomolecular Screening 21, no. 3 (2015): 269–76. http://dx.doi.org/10.1177/1087057115617906.

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The Yap-Hippo pathway has a significant role in regulating cell proliferation and growth, thus controlling organ size and regeneration. The Hippo pathway regulates two highly conserved, transcription coactivators, YAP and TAZ. The upstream regulators of the Yap-Hippo pathway have not been fully characterized. The aim of this study was to use a siRNA screen, in a liver biliary cell line, to identify regulators of the Yap-Hippo pathway that allow activation of the YAP transcription coactivator at high cell density. Activation of the YAP transcription coactivator was monitored using a high-conten

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Grijalva, James L., Megan Huizenga, Kaly Mueller, et al. "Dynamic alterations in Hippo signaling pathway and YAP activation during liver regeneration." American Journal of Physiology-Gastrointestinal and Liver Physiology 307, no. 2 (2014): G196—G204. http://dx.doi.org/10.1152/ajpgi.00077.2014.

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The Hippo signaling pathway has been implicated in mammalian organ size regulation and tumor suppression. Specifically, the Hippo pathway plays a critical role regulating the activity of transcriptional coactivator Yes-associated protein (YAP), which modulates a proliferative transcriptional program. Recent investigations have demonstrated that while this pathway is activated in quiescent livers, its inhibition leads to liver overgrowth and tumorigenesis. However, the role of the Hippo pathway during the natural process of liver regeneration remains unknown. Here we investigated alterations in

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Yang, Haitang, Sean R. R. Hall, Beibei Sun, et al. "NF2 and Canonical Hippo-YAP Pathway Define Distinct Tumor Subsets Characterized by Different Immune Deficiency and Treatment Implications in Human Pleural Mesothelioma." Cancers 13, no. 7 (2021): 1561. http://dx.doi.org/10.3390/cancers13071561.

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(1) Inactivation of the tumor suppressor NF2 is believed to play a major role in the pathogenesis of malignant pleural mesothelioma (MPM) by deregulating the Hippo-YAP signaling pathway. However, NF2 has functions beyond regulation of the Hippo pathway, raising the possibility that NF2 contributes to MPM via Hippo-independent mechanisms. (2) We performed weighted gene co-expression analysis (WGCNA) in transcriptomic and proteomic datasets obtained from The Cancer Gene Atlas (TCGA) MPM cohort to identify clusters of co-expressed genes highly correlated with NF2 and phospho (p)-YAP protein, surr

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Huang, Shiyuan, Xiaona Wang, Xinmei Wu, et al. "Yap regulates mitochondrial structural remodeling during myoblast differentiation." American Journal of Physiology-Cell Physiology 315, no. 4 (2018): C474—C484. http://dx.doi.org/10.1152/ajpcell.00112.2018.

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Yes-associated protein (Yap) is a core transcriptional coactivator in the downstream Hippo pathway that regulates cell proliferation and tissue growth. However, its role in the regulation of myoblast differentiation remains unclear. Regulation of mitochondrial networks by dynamin-related protein 1 (Drp1) and mitofusion 2 (Mfn2) is crucial for the activation of myoblast differentiation. In the present study, we investigated the interplay between the Hippo/Yap pathway and protein contents of Mfn2 and Drp1 during myoblast differentiation. The Hippo/Yap pathway was inactivated at the early stage o

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Jagannathan, Radhika, Gregory V. Schimizzi, Kun Zhang, et al. "AJUBA LIM Proteins Limit Hippo Activity in Proliferating Cells by Sequestering the Hippo Core Kinase Complex in the Cytosol." Molecular and Cellular Biology 36, no. 20 (2016): 2526–42. http://dx.doi.org/10.1128/mcb.00136-16.

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The Hippo pathway controls organ growth and is implicated in cancer development. Whether and how Hippo pathway activity is limited to sustain or initiate cell growth when needed is not understood. The members of the AJUBA family of LIM proteins are negative regulators of the Hippo pathway. In mammalian epithelial cells, we found that AJUBA LIM proteins limit Hippo regulation of YAP, in proliferating cells only, by sequestering a cytosolic Hippo kinase complex in which LATS kinase is inhibited. At the plasma membranes of growth-arrested cells, AJUBA LIM proteins do not inhibit or associate with

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Höffken, Verena, Anke Hermann, Hermann Pavenstädt, and Joachim Kremerskothen. "WWC Proteins: Important Regulators of Hippo Signaling in Cancer." Cancers 13, no. 2 (2021): 306. http://dx.doi.org/10.3390/cancers13020306.

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The Hippo signaling pathway is known to regulate cell differentiation, proliferation and apoptosis. Whereas activation of the Hippo signaling pathway leads to phosphorylation and cytoplasmic retention of the transcriptional coactivator YAP, decreased Hippo signaling results in nuclear import of YAP and subsequent transcription of pro-proliferative genes. Hence, a dynamic and precise regulation of the Hippo signaling pathway is crucial for organ size control and the prevention of tumor formation. The transcriptional activity of YAP is controlled by a growing number of upstream regulators includ

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Thaventhiran, James, Anja Hoffmann, and Douglas Fearon. "CTLA-4 activates the hippo pathway to regulate terminal differentiation of the CD8+ T cell. (46.17)." Journal of Immunology 186, no. 1_Supplement (2011): 46.17. http://dx.doi.org/10.4049/jimmunol.186.supp.46.17.

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Abstract Replicating, antigen-specific CD8+ T cells must not commit to terminal differentiation until there has been sufficient clonal expansion. The Hippo pathway of organ size control mediates this requirement by linking expression of the differentiation-inducing transcription factor, Blimp-1, to contact between replicating cells, which would be dependent on their frequency. TCR and IL-2R stimulation assemble the Hippo pathway in the CD8+ T cell by inducing expression of WW45, Mob1, Lats1, and YAP, the transcriptional co-activator that mediates organ growth. Contact between activated CD8+ T

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Casati, G., L. Giunti, A. Iorio, A. Marturano, and I. Sardi. "P04.20 The role of YAP in Glioblastoma cell lines." Neuro-Oncology 23, Supplement_2 (2021): ii22—ii23. http://dx.doi.org/10.1093/neuonc/noab180.074.

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Abstract BACKGROUND Glioblastoma (GBM) is a primary human malignant brain tumor, the most common in adults. Several studies have highlighted the Hippo-pathway as a cancer signalling network. The Hippo pathway is an evolutionarily conserved signal cascade, which is involved in the control of organ growth. Dysregulations among this pathway have been found in lung, ovarian, liver and colorectal cancer. The key downstream effector of the Hippo-pathway is the Yes-associated protein (YAP); in the nucleus, its function as transcription co-activator is to interact with transcription factors, resulting

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Thèses sur le sujet "HIPPO-PATHWAY-YAP"

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Bui, Duyen Amy. "The Hippo Pathway Effector YAP Regulates Cytokinesis." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17467231.

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Yes-associated protein (YAP) is a co-transcription factor that acts downstream of the evolutionarily conserved Hippo pathway. Canonically, this pathway regulates tissue growth in flies and mammals, by controlling the nuclear localization of YAP. Interestingly, in addition to the conserved functions of this pathway, some of the mammalian orthologs of pathway components (e.g. MST, RASSF1, WW45, and LATS) have been shown to localize to the nucleus and alterations in their expression induces alterations in mitotic processes, suggesting additional roles for these proteins in mitosis. In this thes

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Moleirinho, Susana. "Mammalian upstream Hippo signalling pathway proteins activate core pathway kinases and functionally antagonize oncogenic YAP." Thesis, University of St Andrews, 2013. http://hdl.handle.net/10023/3662.

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The mechanism of body and organ size control is an unsolved puzzle. Initially characterized in Drosophila melanogaster, the Salvador/Warts/Hippo (Hippo) signalling pathway, highly conserved throughout evolution, defines a novel signalling cascade regulating cell contact inhibition, organ size control, cell growth, proliferation, apoptosis, and cancer development in mammals. The upstream regulation of this pathway has been less well defined than the core kinase cassette. Previously Willin/FRMD6 has been proposed as the human orthologue of Expanded and, to date, little is known about the functio

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Toloczko, Aleksandra. "Deubiquitination and control of the Hippo pathway." Thesis, University of Manchester, 2017. https://www.research.manchester.ac.uk/portal/en/theses/deubiquitination-and-control-of-the-hippo-pathway(8afdf3df-8635-4116-99c8-57fbe423501e).html.

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The Hippo signalling pathway is an evolutionarily conserved kinase cascade responsible for the cell proliferation, tissue growth and apoptosis during development and its dysregulation contributes to tumourigenesis. This signalling pathway was initially discovered in Drosophila and soon after that, it was shown to be highly conserved in mammals. The core Lats kinases of this tumour suppressive pathway phosphorylate and inhibit the downstream transcriptional co-activators YAP and TAZ, which are implicated in various cancers. Latest reports revealed various E3 ubiquitin ligases to negatively regu

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Sidor, C. M. "Mask proteins are co-factors of Yorkie/YAP in the Hippo signaling pathway." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1352451/.

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One of the key questions in developmental biology is how tissue growth is controlled to give rise to organs of specific sizes and shapes. Although some genes and pathways involved in the genetic and environmental control of tissue growth have been uncovered, the understanding of this process remains incomplete. In order to find new regulators of growth we carried out an in vivo RNAi screen in the Drosophila wing. I participated in the validation of candidate genes from the screen and identified the mask gene as an essential regulator of tissue growth acting in the Hippo signaling pathway. This

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Astone, Matteo. "A novel Yap/Taz zebrafish reporter reveals a role of Hippo pathway transducers in angiogenesis." Doctoral thesis, Università degli studi di Padova, 2015. http://hdl.handle.net/11577/3424650.

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YAP and TAZ, by orchestrating cell proliferation, cell death and cell-fate decisions, are key players of a complex network of signaling pathways acting during development. Deregulation of YAP/TAZ signaling causes robust organ overgrowth during organogenesis, which translates to loss of tissue homeostasis in the adult and consequent cancer development. YAP/TAZ are transcriptional co-activators that interact with TEAD transcription factors to promote cell proliferation and survival. Their transcriptional activity is regulated by nucleocytoplasmic shuttling and nuclear accumulation, which are con

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Meléndez, García Rodrigo. "YAP as a Regulator of DNA Replication Timing." Thesis, université Paris-Saclay, 2020. http://www.theses.fr/2020UPASL014.

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Une cellule souche est capable de s’auto-renouveler et de générer des cellules différenciées après division cellulaire. La duplication complète de son génome doit être exempte d'erreurs afin d'éviter la propagation aux cellules filles de mutations délétères. Chez les eucaryotes, il a été montré que des segments d’ADN sur les chromosomes se répliquent de manière coordonnée et à des moments définis pendant la phase de synthèse, un processus appelé programme spatio-temporel de réplication de l'ADN (RT). Des changements majeurs dans le RT sont corrélés avec les changements de détermination des cel

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Cherrett, Claire. "Structural and functional studies of proteins from the Hippo signalling pathway." Thesis, University of Bath, 2011. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.548103.

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The paralogous multi-functional adaptor proteins YAP and TAZ are nuclear effectors of the Hippo pathway, a central regulator of developmental organ size control, tissue homeostasis and tumour suppression. YAP/TAZ target the TEAD transcription factor family to promote cell survival and inhibit apoptosis. TEAD proteins contain a DNAbinding domain and a YAP/TAZ interaction domain. PCR analysis of medaka fish TEAD cDNA revealed the presence of alternative TEAD splice-forms with variations at the C-terminus of the DNA-binding domain. Structural analysis indicated the YAPbinding domain of TEAD prote

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García-García, Diana. "Müller Cells and Retinal Regeneration : The Role of the Hippo/YAP Signaling Pathway Yap Haploinsufficiency Leads to Müller Cell Dysfunction and Late-Onset Cone Dystrophy Linking YAP to Müller Glia Quiescence Exit in the Degenerative Retina." Thesis, université Paris-Saclay, 2020. http://www.theses.fr/2020UPASL068.

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Les maladies dégénératives de la rétine sont une des causes principales de cécité. Parmi les différentes stratégies thérapeutiques actuellement étudiées, notre équipe s’intéresse au potentiel régénératif de la rétine. Une source cellulaire d'intérêt sont les cellules de Müller, principal type de cellules gliales de la rétine capables de se réactiver en cas de dégénérescence, un processus appelé gliose réactive, et dans certaines espèces d’adopter des caractéristiques de cellules souches. Si un tel processus confère la capacité de régénérer la rétine chez les téléostéens, il est cependant large

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Kimura, Masahiro. "Homeobox A4 Suppresses Vascular Remodeling as a Novel Regulator of YAP/TEAD Transcriptional Activity." Kyoto University, 2020. http://hdl.handle.net/2433/253486.

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Ruggeri, Naomi. "REGULATION OF YAP BY GLUCOCORTICOIDS." Doctoral thesis, Università degli studi di Trieste, 2015. http://hdl.handle.net/10077/11122.

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2013/2014<br>The Hippo signalling pathway is tumour suppressor cascade with a central role in the regulation of fundamental cellular biological processes, such as cell proliferation, apoptosis, organ size control and stem cell functions. The Hippo pathway transduces external signals that come to the cell into the nucleus, where it can control the expression of specific target genes, mainly involved in cell proliferation and differentiation. The Hippo pathway is an inhibitory pathway that control by phosphorylation and inhibition Yes-associated protein (YAP) coactivator, one of the two nuclear

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Chapitres de livres sur le sujet "HIPPO-PATHWAY-YAP"

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Reuven, Nina, and Yosef Shaul. "The c-Abl/YAP/p73 Apoptotic Module and the HIPPO Pathway." In The Hippo Signaling Pathway and Cancer. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6220-0_9.

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Yi, Chunling, and Joseph Kissil. "Merlin and Angiomotin in Hippo-Yap Signaling." In The Hippo Signaling Pathway and Cancer. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6220-0_2.

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Yu, Fa-Xing, Bin Zhao, and Kun-Liang Guan. "Regulation of YAP and TAZ Transcription Co-activators." In The Hippo Signaling Pathway and Cancer. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6220-0_5.

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Piccolo, Stefano, and Michelangelo Cordenonsi. "Regulation of YAP and TAZ by Epithelial Plasticity." In The Hippo Signaling Pathway and Cancer. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6220-0_6.

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Donzelli, Sara, Sabrina Strano, and Giovanni Blandino. "YAP and p73: A Matter of Mutual Specificity in Tumor Suppression." In The Hippo Signaling Pathway and Cancer. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6220-0_8.

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Nazir, Aqsa, Muhammad Aqib, and Muhammad Usman. "Liver Cancer-Genesis, Progression and Metastasis." In Liver Cancer - Genesis, Progression and Metastasis [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.106020.

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Liver cancer or hepatocellular carcinoma (HCC) is a malignant tumor in liver tissue and worldwide it is fourth leading death cause among all cancers. The most common causes of liver cancer are hepatitis B or C virus infections, alcoholic liver disease (ALD), nonalcoholic fatty liver disease (NAFLD) to non-alcoholic steatohepatitis (NASH), smoking and obesity. The development and metastasis of liver cancer is a multistage and branched process of morphological and genetic traits. Various corresponding signaling pathways such as Yes-Associated Protein-Hippo Pathway (YAP-HIPPO), Wnt/β-catenin and inflammation by interleukin-6 (IL-6), tumor necrosis factor (TNF), nuclear factor-Κb (NF-κB), biological pathways including epithelial–mesenchymal transition (EMT), tumor microenvironment, tumor-stromal interactions and cancer stem cells and gut microbial dysbiosis are allied to both origination, progression and metastasis of liver cancer. Numerous therapeutic approaches are classified into different categories such as pharmacological therapy including sorafenib, lenvatinib and ramuciruma, surgery of HCC patients includes surgical resection, adjuvant therapy after surgical resection and liver transplantation. Loco-regional ablative therapy includes cryotherapy, ethanol injection and radiofrequency ablation, cytotoxic chemotherapy, natural compounds such as piperine, as curcumin and oleocanthal, oncolytic virus therapy, immunotherapies and nanotechnology.

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Actes de conférences sur le sujet "HIPPO-PATHWAY-YAP"

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Schönbeck, K., A. Winkler, MJ Witthauer, et al. "Hippo-YAP pathway activation favors neuroblastoma progression." In 31. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1645011.

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Tang, Tracy T., Andrei W. Konradi, Ying Feng, Xiao Peng, Sofie Qiao, and Leonard Post. "Abstract PR07: Targeting the Hippo-YAP pathway with small-molecule compounds." In Abstracts: AACR Special Conference on the Hippo Pathway: Signaling, Cancer, and Beyond; May 8-11, 2019; San Diego, CA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3125.hippo19-pr07.

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Kulkarni, A., J. Vissers, and K. Harvey. "PO-100 Targeting YAP and TAZ to treat hippo pathway mutant malignant mesotheliomas." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.141.

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Isago, Hideaki, Akihisa Mitani, Shiho Kohno, et al. "The Hippo pathway effectors TAZ and YAP are sequentially required in lung development." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.oa3604.

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Tang, Tracy T., Andrei W. Konradi, Ying Feng, Xiao Peng, Sofie Qiao, and Leonard Post. "Abstract 2693: Targeting the Hippo-YAP pathway with novel small-molecule inhibitors of the YAP-TEAD transcription activity." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2693.

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Tang, Tracy T., Andrei W. Konradi, Ying Feng, Xiao Peng, Sofie Qiao, and Leonard Post. "Abstract 2693: Targeting the Hippo-YAP pathway with novel small-molecule inhibitors of the YAP-TEAD transcription activity." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2693.

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Liu, K., M. Tóth, T. Guo, et al. "Hippo pathway effectors YAP and TAZ are opponents in the regulation of hepatic fibrosis." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402120.

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Isfort, Ilka, Sandra Elges, Magdalene Cyra, et al. "Abstract 2139: Hippo pathway transcriptional coactivators YAP/TAZ in soft tissue and bone tumors." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2139.

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Isfort, Ilka, Sandra Elges, Magdalene Cyra, et al. "Abstract 2139: Hippo pathway transcriptional coactivators YAP/TAZ in soft tissue and bone tumors." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2139.

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Kim, Ji Su, Ei Yong Ahn, and Young Nyun Park. "Abstract C87: Regulation of stemness by Hippo-YAP pathway under hypoxia in hepatocellular carcinoma." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c87.

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