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Littérature scientifique sur le sujet « Hypervolemia »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 16 février 2022

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Articles de revues sur le sujet "Hypervolemia"

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Ulrich, Christof, Annegret Wilke, Nadja Schleicher, Matthias Girndt et Roman Fiedler. « Hypervolemia-Induced Immune Disturbances Do Not Involve IL-1ß but IL-6 and IL-10 Activation in Haemodialysis Patients ». Toxins 12, n^o 3 (3 mars 2020) : 159. http://dx.doi.org/10.3390/toxins12030159.

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Dysregulated fluid homeostasis is frequent in haemodialysis (HD) patients and is linked to inflammation which may be elicited by endotoxemia. The impact of hypervolemia on immune cells has not been studied in detail. Therefore, we analysed the hypervolemic activation of peripheral blood mononuclear cells (PBMCs) in HD with special focus on the NLRP3 inflammasome response. First, 45 HD were included in the observational study. Immune parameters including cell counts, caspase-1, oxidative stress, cytokine gene expression and serum analysis (IL-1ß, IL-6, IL-10) were all measured at two time points. Fluid status was evaluated by electrical bioimpedance vector analysis, defining hypervolemia (H) as >75 vector percentile. Then, 17 patients were classified as hypervolemic (H-HD), 19 as normovolemic (N-HD) and 9 failed to meet the inclusion criteria. Monocytes were elevated and lymphocytes were decreased by hypervolemia. NLRP3 inflammasome components, caspase-1 and IL-1ß expression were not statistically different between the two groups. Serum IL-6 levels were significantly elevated in H-HD. IL-10 mRNA transcripts were elevated by 2-fold in H-HD but were not efficiently translated. We conclude that the NLRP3 inflammasome is not activated by hypervolemia thus refuting the thesis that endotoxemia may be a main driver for inflammation in H-HD. Nevertheless, inflammation is generally higher in H-HD compared to N-HD patients and is not sufficiently balanced by anti-inflammatory mechanisms.

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Raabe, Andreas, Jügen Beck, Mike Keller, Hartmuth Vatter, Michael Zimmermann et Volker Seifert. « Relative importance of hypertension compared with hypervolemia for increasing cerebral oxygenation in patients with cerebral vasospasm after subarachnoid hemorrhage ». Journal of Neurosurgery 103, n^o 6 (décembre 2005) : 974–81. http://dx.doi.org/10.3171/jns.2005.103.6.0974.

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Object. Hypervolemia and hypertension therapy is routinely used for prophylaxis and treatment of symptomatic cerebral vasospasm at many institutions. Nevertheless, there is an ongoing debate about the preferred modality (hypervolemia, hypertension, or both), the degree of therapy (moderate or aggressive), and the risk or benefit of hypervolemia, moderate hypertension, and aggressive hypertension in patients following subarachnoid hemorrhage. Methods. Monitoring data and patient charts for 45 patients were retrospectively searched to identify periods of hypervolemia, moderate hypertension, or aggressive hypertension. Measurements of central venous pressure, fluid input, urine output, arterial blood pressure, intracranial pressure, and oxygen partial pressure (PO2) in the brain tissue were extracted from periods ranging from 1 hour to 24 hours. For these periods, the change in brain tissue PO2 and the incidence of complications were analyzed. During the 55 periods of moderate hypertension, an increase in brain tissue PO2 was found in 50 cases (90%), with complications occurring in three patients (8%). During the 25 periods of hypervolemia, an increase in brain oxygenation was found during three intervals (12%), with complications occurring in nine patients (53%). During the 10 periods of aggressive hypervolemic hypertension, an increase in brain oxygenation was found during six of the intervals (60%), with complications in five patients (50%). Conclusions. When hypervolemia treatment is applied as in this study, it may be associated with increased risks. Note, however, that further studies are needed to determine the role of this therapeutic modality in the care of patients with cerebral vasospasm. In poor-grade patients, moderate hypertension (cerebral perfusion pressure 80–120 mm Hg) in a normovolemic, hemodiluted patient is an effective method of improving cerebral oxygenation and is associated with a lower complication rate compared with hypervolemia or aggressive hypertension therapy.

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Hoh, Brian L., Bob S. Carter et Christopher S. Ogilvy. « Risk of Hemorrhage from Unsecured, Unruptured Aneurysms during and after Hypertensive Hypervolemic Therapy ». Neurosurgery 50, n^o 6 (1 juin 2002) : 1207–12. http://dx.doi.org/10.1097/00006123-200206000-00006.

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Abstract OBJECTIVE Hypertensive hypervolemic therapy for vasospasm is widely practiced. It is not clear, however, whether the use of hypertension and hypervolemia as a treatment for vasospasm risks hemorrhage from an unsecured, unruptured aneurysm. METHODS From 1991 to 2000, the neurovascular unit at the Massachusetts General Hospital treated 1908 aneurysms, of which 966 were ruptured. Forty patients with ruptured aneurysms had unsecured, unruptured aneurysms and underwent hypertensive hypervolemic therapy for vasospasm. Hypertension was induced by intravenously administered phenylephrine, norepinephrine, and/or dopamine, and hypervolemia was achieved by intravenously administered crystalloid and colloid solutions. The 24-hour mean arterial systolic blood pressure (SBP) and the 24-hour mean central venous pressure were calculated on the basis of hourly measurements during hypertensive hypervolemic treatment. RESULTS The 40 study patients harbored 124 aneurysms, of which 51 aneurysms were treated (clipping, 37; coiling, 14) by the time hypertensive hypervolemic therapy began, leaving 73 unsecured aneurysms at risk. The mean size of the unsecured aneurysms was 4.45 mm. Nineteen patients were treated with mild hypertension (SBP, 140–180 mm Hg), 12 patients were treated with moderate hypertension (SBP, 180–200 mm Hg), and 9 patients were treated with severe hypertension (SBP, >200 mm Hg). The 24-hour mean SBP readings were 166.81 ± 8.19, 187.57 ± 5.79, and 204.01 ± 3.75 mm Hg for the mild, moderate, and severe hypertension groups, respectively. The mean central venous pressure was 10.43 ± 3.89 mm Hg. The mean course of hypertensive hypervolemic therapy was 7.25 days, and therapy began on mean post-subarachnoid hemorrhage Day 6.73. Twenty-eight aneurysms were eventually treated in later procedures (clipping, 25; coiling, 3). The mean interval to treatment was 6.93 months. In a treatment and follow-up period of 121.75 aneurysm-years of risk, there was no instance of hemorrhage. CONCLUSION Hypertension and hypervolemia do not seem to increase the risk of hemorrhage from unsecured, unruptured aneurysms in the acute setting or in their short-term natural history.

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Fritsch-Yelle, Janice M., Victor A. Convertino et Todd T. Schlegel. « Acute manipulations of plasma volume alter arterial pressure responses during Valsalva maneuvers ». Journal of Applied Physiology 86, n^o 6 (1 juin 1999) : 1852–57. http://dx.doi.org/10.1152/jappl.1999.86.6.1852.

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The effects of changes in blood volume on arterial pressure patterns during the Valsalva maneuver are incompletely understood. In the present study we measured beat-to-beat arterial pressure and heart rate responses to supine Valsalva maneuvers during normovolemia, hypovolemia induced with intravenous furosemide, and hypervolemia induced with ingestion of isotonic saline. Valsalva responses were analyzed according to the four phases as previously described (W. F. Hamilton, R. A. Woodbury, and H. T. Harper, Jr. JAMA 107: 853–856, 1936; W. F. Hamilton, R. A. Woodbury, and H. T. Harper, Jr. Am. J. Physiol. 141: 42–50, 1944). Phase I is the initial onset of straining, which elicits a rise in arterial pressure; phase II is the period of straining, during which venous return is impeded and pressure falls (early) and then partially recovers (late); phase III is the initial release of straining; and phase IV consists of a rapid “overshoot” of arterial pressure after the release. During hypervolemia, early phase II arterial pressure decreases were significantly less than those during hypovolemia, thus making the response more “square.” Systolic pressure hypervolemic vs. hypovolemic falls were −7.4 ± 2.1 vs. −30.7 ± 7 mmHg ( P = 0.005). Diastolic pressure hypervolemic vs. hypovolemic falls were −2.4 ± 1.6 vs. −15.2 ± 2.6 mmHg ( P = 0.05). A significant direct correlation was found between plasma volume and phase II systolic pressure falls, and a significant inverse correlation was found between plasma volume and phase III-IV systolic pressure overshoots. Heart rate responses to systolic pressure falls during phase II were significantly less during hypovolemia than during hypervolemia (0.7 ± 0.2 vs. 2.82 ± 0.2 beats ⋅ min−1 ⋅ mmHg−1; P = 0.05) but were not different during phase III-IV overshoots. We conclude that acute changes in intravascular volume from hypovolemia to hypervolemia affect cardiovascular responses, particularly arterial pressure changes, to the Valsalva maneuver and should be considered in both clinical and research applications of this maneuver.

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Shimoda, Masami, Shinri Oda, Ryuichi Tsugane et Osamu Sato. « Intracranial complications of hypervolemic therapy in patients with a delayed ischemic deficit attributed to vasospasm ». Journal of Neurosurgery 78, n^o 3 (mars 1993) : 423–29. http://dx.doi.org/10.3171/jns.1993.78.3.0423.

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✓ This investigation has revealed the frequency of various intracranial complications that may result from hypervolemic therapy for a delayed ischemic deficit following subarachnoid hemorrhage (SAH). Among 323 patients with SAH, 112 patients developed a delayed ischemic deficit, 94 of whom underwent hypervolemic therapy. Infarction due to vasospasm was found ultimately in 43 of these 94 patients. Twenty-six patients (28%) developed an intracranial complication during hypervolemic therapy: cerebral edema was aggravated in 18, and a hemorrhagic infarction developed in eight. In 13 of 18 patients with aggravation of edema, delayed ischemic deficit developed within 6 days after the SAH; at that time, a massive new infarction was found in four and edema in 10 patients. After hypervolemic therapy, the 18 patients with aggravation of edema deteriorated rapidly, and 14 of them died. In every case in which hemorrhagic infarction followed hypervolemic therapy, a new infarct was found on computerized tomography (CT) when the delayed ischemic deficit became apparent. Hemorrhagic infarction developed as the delayed ischemic deficit resolved, with one exception. In patients who sustained no complication from hypervolemia, the incidence of both massive new infarction and edema at the time when the delayed ischemic deficit was manifested was only 1%. In 44 of 68 patients who sustained no complication from hypervolemia, the delayed ischemic deficit was manifested on or after the 7th day following the SAH. This study suggests that hypervolemic therapy is contraindicated in a patient who is found to have a massive abnormality on CT at the time when a delayed ischemic deficit is manifested, especially when it occurs within 6 days after the SAH. To avoid hemorrhagic infarction, it is important to discontinue hypervolemic therapy as soon as the delayed ischemic deficit resolves.

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Karpavičiūtė, Justina, Inga Skarupskienė, Vilma Balčiuvienė, Rūta Vaičiūnienė, Edita Žiginskienė et Inga Arūnė Bumblytė. « Assessment of Fluid Status by Bioimpedance Analysis and Central Venous Pressure Measurement and Their Association with the Outcomes of Severe Acute Kidney Injury ». Medicina 57, n^o 6 (22 mai 2021) : 518. http://dx.doi.org/10.3390/medicina57060518.

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Background and Objectives: Fluid disbalance is associated with adverse outcomes in critically ill patients with acute kidney injury (AKI). In this study, we intended to assess fluid status using bioimpedance analysis (BIA) and central venous pressure (CVP) measurement and to evaluate the association between hyperhydration and hypervolemia with the outcomes of severe AKI. Materials and Methods: A prospective study was conducted in the Hospital of the Lithuanian University of Health Sciences Kauno Klinikos. Forty-seven patients treated at the Intensive Care Unit (ICU) with severe AKI and a need for renal replacement therapy (RRT) were examined. The hydration level was evaluated according to the ratio of extracellular water to total body water (ECW/TBW) of bioimpedance analysis and volemia was measured according to CVP. All of the patients were tested before the first hemodialysis (HD) procedure. Hyperhydration was defined as ECW/TBW > 0.39 and hypervolemia as CVP > 12 cm H2O. Results: According to bioimpedance analysis, 72.3% (n = 34) of patients were hyperhydrated. According to CVP, only 51.1% (n = 24) of the patients were hypervolemic. Interestingly, 69.6% of hypovolemic/normovolemic patients were also hyperhydrated. Of all study patients, 57.4% (n = 27) died, in 29.8% (n = 14) the kidney function improved, and in 12.8% (n = 6) the demand for RRT remained after in-patient treatment. A tendency of higher mortality in hyperhydrated patients was observed, but no association between hypervolemia and outcomes of severe AKI was established. Conclusions: Three-fourths of the patients with severe AKI were hyperhydrated based on bioimpedance analysis. However, according to CVP, only half of these patients were hypervolemic. A tendency of higher mortality in hyperhydrated patients was observed.

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GREEN, HOWARD J., LAURIE L. JONES, RICHARD L. HUGHSON, DOUG C. PAINTER et BRIAN W. FARRANCE. « Training-induced hypervolemia ». Medicine & ; Science in Sports & ; Exercise 19, n^o 3 (juin 1987) : 202???206. http://dx.doi.org/10.1249/00005768-198706000-00003.

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Permutt, S., et H. E. Fessler. « CPAP with hypervolemia. » American Journal of Respiratory and Critical Care Medicine 153, n^o 3 (mars 1996) : 1187–88. http://dx.doi.org/10.1164/ajrccm.153.3.8630566.

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Sungkar, Ali, Fita Maulina et M. Adya F. Dilmy. « Hypervolemia and physiology changes in triplet pregnancy in a mother with permanent pacemakers due to bradicardia resulting from sinus node dysfunction due to AV block with secondary infertility for 19 years ». Majalah Obstetri & ; Ginekologi 28, n^o 3 (7 décembre 2020) : 128. http://dx.doi.org/10.20473/mog.v28i32020.128-134.

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The well-known hypervolemia associated with normal pregnancy averages 40 to 45 percent above blood volume in non-pregnant women after 32 to 34 weeks. The case was on Mrs. FN, 40 years old, a patient reffered due to sinus bradycardia before the insertion of permanent pacemaker. After 6 month-use of the permanent pacemaker, she became pregnant with triplet pregnancy. This case report evaluated the patient's condition from her hypervolemic condition to her cardiac function.

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Raabe, Andreas, et Bertil Romner. « Hypervolemia in Cerebral Vasospasm ». Journal of Neurosurgery 104, n^o 6 (juin 2006) : 994–95. http://dx.doi.org/10.3171/jns.2006.104.6.994.

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Thèses sur le sujet "Hypervolemia"

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Nelson, William Bradley. « Exercise Induced Hypervolemia : Role of Exercise Mode ». Diss., CLICK HERE for online access, 2007. http://contentdm.lib.byu.edu/ETD/image/etd2128.pdf.

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Harris, S. Kelly. « Short-term training-induced hypervolemia and diuresis, effects on oxygen uptake and left ventricular diastolic function in older men ». Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ58043.pdf.

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Parente, Cynara Carvalho. « Efeitos da hipervolemia crÃnica sobre a motilidade gastrintestinal e transporte intestinal de Ãgua e eletrÃlitos em ratos sob nefrectomia parcial ». Universidade Federal do CearÃ, 2010. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=5345.

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nÃo hÃ
EstÃ bem estabelecido que a regulaÃÃo dos lÃquidos corporais seja garantida pela interaÃÃo funcional entre os sistemas cardiovascular e renal. Atualmente, evidÃncias clÃnicas e experimentais sugerem que o trato gastrintestinal (TGI) ajusta seu padrÃo motor e absortivo apÃs variaÃÃes agudas da volemia. Embora a insuficiÃncia renal parcial ou total promova variaÃÃes dos volumes corporais circulantes, poucos estudos apontam a relaÃÃo entre a insuficiÃncia renal e o funcionamento do trato gastrintestinal. Neste trabalho, estudamos o efeito da dieta salina sobre a motilidade gastrintestinal [esvaziamento gÃstrico (EG) e o trÃnsito gastrintestinal (GI) de lÃquidos] e transporte intestinal (IT) de Ãgua e eletrÃlitos (Na+, K+ e Cl-) em ratos nefrectomizados parcialmente (nefre5/6) ou falso-operados (FO) em condiÃÃes de hidrataÃÃo e desidrataÃÃo. Para tanto, 138 ratos Wistar, machos (180-220g), submetidos Ã nefrectomia parcial (nefre5/6) em duas etapas (0 e 7 dias). ApÃs nefre 5/6 ou FO, os animais foram distribuÃdos em dois grupos diferentes, a saber: raÃÃo e salina 1% ou raÃÃo e Ãgua. ApÃs 3d e, sob jejum de 24h com livre acesso Ã salina ou Ãgua, 1,5ml da refeiÃÃo teste (vermelho fenol 0,5mg/ml e glicose 5%) foi administrada por gavagem em animais acordados. Decorridos 10, 20 ou 30min, os animais foram sacrificados por deslocamento cervical, seguidos da exÃrese das vÃsceras abdominais para determinaÃÃo da taxa de EG e trÃnsito GI. AlÃm disso, um experimento semelhante foi realizado utilizando 5ml de polietilenoglicol-PEG (30% - 20.000 DA) injetado por via subcutÃnea em ratos quatro horas antes do inÃcio do experimento, a fim de simular as condiÃÃes de desidrataÃÃo. Para os estudos do transito intestinal de Ãgua e eletrÃlitos, os animais anestesiados, foram submetidos Ã perfusÃo ileal com Ringer + vermelho fenol durante 60min. Para todos os experimentos, monitoramos os parÃmetros hemodinÃmicos (pressÃo arterial-PA, pressÃo venosa central-PVC, frequÃncia cardÃaca-FC e volume sanguÃneo-VS) e ainda as concentraÃÃes bioquÃmicas plasmÃticas de Ur, Cr, Na+, K+ e Cl- foram determinadas. Em relaÃÃo ao grupo FO a dieta salina nÃo modificou o EG ou transito GI, nem os parÃmetros hemodinÃmicos ou bioquÃmicos, porÃm promoveu secreÃÃo ileal de Ãgua e eletrÃlitos. Por outro lado, a dieta salina nos animais nefre5/6 promoveu: i) aumento da retenÃÃo gÃstrica de 47%, 26% e 38% (10, 20 e 30 minutos de tempo pÃs-prandial, respectivamente), ii aceleraÃÃo do trÃnsito GI, iii) aumento na secreÃÃo ileal de Ãgua e eletrÃlitos e, iv) aumento da PA, PVC, FC, BV. A desidrataÃÃo aguda com PEG preveniu as alteraÃÃes da motilidade, da secreÃÃo GI e dos parÃmetros hemodinÃmicos secundÃrios hipervolemia crÃnica Ã custa da nefrectomia 5/6 associada Ã dieta salina. A motilidade gastrintestinal e o ajuste da absorÃÃo, devido Ã dieta salina sobre os animais submetidos Ã nefrectomia parcial, estÃo relacionados aos nÃveis do volume do sangue e pode ser revertida por desidrataÃÃo aguda. Em conclusÃo, trato gastrointestinal pode ajustar tanto o seu motor, bem como atividades de absorÃÃo apÃs desequilÃbrios crÃnicos volume de sangue.
It is well established that the regulation of the corporal fluids is guaranteed by functional interaction between cardiovascular and renal systems. Currently, clinical and experimental evidences suggest that gastrointestinal (GI) tract (GIT) adjust their motor and absorptive activities due to acute changes in the blood volume. Although total or partial renal failure promotes corporal fluids changing. Several studies indicate a relationship among renal failure and GIT functions. In this work, we study the effect of a salt diet on GIT motility [gastric emptying (GE) and GI transit of liquids] and intestinal transport (IT) of H2O and electrolytes (Na+, K+ and Cl-) in partial nephrectomized (nefre5/6) or false-operated (FO) rats under hydrated and dehydrate conditions. For that, 138 male Wistar rats (180-220g) submitted to partial nephrectomy (nefre5/6) in two steps (0 and 7 days). After nefre5/6 or FO procedures, animals were distributed into 2 different experiments both containig two groups as follows: feed+1% saline or feed+water. For the hydrate conditions experiment, after 3d and under 24-hour fasting with free access to water or saline, 1.5ml of the test meal (phenol red 0.5 mg/mL containg 5% glucose) was gavaged in the awake animals. Next 10, 20 or 30 minutes, the animals were sacrificed by cervical dislocation. Following, excision of the abdominal viscera was performed in order to determine the GE rate and GI transit. Additionally, similar experiment were performed using 5mL of polyethylene glycol-PEG (30% - 20,000 DA) injected subcutaneous in the rats 4 hours before the beginning of the experiment in order to simulate the dehydrate conditions. For IT studies, anesthetized animals underwent ileal perfusion with Ringer+phenol red solution and were monitored along 60 min. For all experiments, mean arterial pressure - MAP, central venous pressure-CVP, heart rate-HR and blood volume-BV were monitored. Also, plasmatic concentrations of Ur, Cr, Na+, K+ and Cl- were determined. Compared with FO group, nefre5/6 did not change the GE or GI transit, neither hemodynamic or biochemical parameters, but promotes ileal secretion of water and electrolytes. On the other hand, comparing the salt diet and standard diet, the nefre5/6 animals caused: i) increases on the gastric retention of 47%, 26% and 38% (at 10, 20 and 30 minutes of postprandial time, respectively), ii) acceleration of the GI transit, iii) increases on the ileal secretion of water and electrolytes and, iv) increases BP, CVP, HR and BV. However, changes on the other plasmatic biochemical parameters were not observed in this study. The acute dehydration with PEG prevented gut motility and hemodynamic changes and the increase of gastrointestinal secretions. Gastrointestinal motility and absorptive adjustments due to salt diet on the partial nephrectomized animals, is related to blood volume levels and, can be reversed by acute dehydration. In conclusion, gastrointestinal tract can adjust both their motor as well as absorptive activities after chronic blood volume imbalances.

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Nelson, Michael Douglas. « Hypervolemia, thermoregulation, and exercise performance under severe heat stress ». Thesis, 2007. http://hdl.handle.net/1828/2268.

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The purpose of this study was to determine if the ingestion of sodium citrate (CIT), and the subsequent expansion of plasma volume, would have a direct effect on thermoregulation and physiological function during a simulated 1-hour time trial (TT) under sever heat stress. Eight subjects, aged 24.9 years (SD + 4.4), were studied under the following three conditions: (1) High Sodium Citrate (HCIT; 0.2 g/kg sodium citrate), (2) Low Sodium Citrate (LCIT; 0.1 g/kg sodium citrate), and (3) Control (Gatorade). Blood samples, taken before and during exercise, were analyzed for hematocrit (Het), haemoglobin (Hb), bicarbonate (HCO3-), base excess (BE), pH, sodium (Na), potassium (K), and glucose (GLU). Rectal temperature (Tr) and skin temperature (Tsk) was also recorded. Heart rate and psychophysical strain were also measured throughout each TT. Plasma volume significantly increased by 7.1% after ingesting the HCIT compared to the other two trials. There were no differences in mean body temperature, physiological strain, cardiovascular strain or psychophysical perception. The ingestion of HCIT appears to improve cycling performance despite no difference in physiological measurements. HCIT reduced split times, better maintained power output over the TT. and improved time total cycling time.

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Zhao, Heng-Li, et 趙恆立. « Hypervolemic hemodilution exhibits effective neuroprotection in striatal ischemia and cell injury during rat heatstroke ». Thesis, 1998. http://ndltd.ncl.edu.tw/handle/70689710487979279792.

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Livres sur le sujet "Hypervolemia"

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Elevated central venous pressure : A consequence of exercise training-induced hypervolemia ? [Washington DC : National Aeronautics and Space Administration, 1990.

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Center, Ames Research, dir. Hypervolemia in men from drinking hyperhydration fluids at rest and during exercise. Moffett Field, Calif : National Aeronautics and Space Administration, Ames Research Center, 1994.

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Wijdicks, Eelco F. M., et Sarah L. Clark. Drugs to Correct Electrolyte Disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0015.

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Critically ill patients develop electrolyte imbalances, and replacement orders are daily practice. Knowing how to manage sodium imbalances in both neurocritical care and neurosurgical practices is imperative. Changes in serum sodium values are very common in acute neurocritical illness, and these derangements are important not only because the patient’s level of consciousness may change but because they may signal a neurologic change. This chapter discusses how to replace common electrolyte replacements and offers more detailed information about the management of disorders of sodium and water homeostasis, including the use of vaptans, which are reserved for patients with difficult-to-manage euvolemic or hypervolemic hyponatremia.

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Chapitres de livres sur le sujet "Hypervolemia"

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Unterberg, A., J. Gethmann, A. von Helden, G. H. Schneider et W. Lanksch. « Treatment of Cerebral Vasospasm with Hypervolemia and Hypertension ». Dans Neurosurgical Standards Cerebral Aneurysms Malignant Gliomas, 198–201. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77109-5_34.

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Todd, Michael M. « Fluid Management in Neurosurgery : Crystalloids, Hypervolemia and Hemodilution ». Dans Neuroanesthesia, 75–86. Dordrecht : Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5774-2_7.

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Segizbaeva, M. O., Zh A. Donina, V. G. Aleksandrov et N. P. Aleksandrova. « The Mechanisms of Compensatory Responses of the Respiratory System to Simulated Central Hypervolemia in Normal Subjects ». Dans Advances in Experimental Medicine and Biology, 9–17. Cham : Springer International Publishing, 2014. http://dx.doi.org/10.1007/5584_2014_100.

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Koscielny, J., H. Förster, W. Kolepke et F. Jung. « Comparison of Iso- and Hypervolemic Hemodilution with Haes ». Dans Hemodilution, 115–84. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-662-07748-1_4.

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Xue, Yang, Pam R. Taub, Arrash Fard et Alan S. Maisel. « Hypervolemic and Optivolemic Natriuretic Peptides in Acute Heart Failure ». Dans Hemodialysis, 74–79. Basel : KARGER, 2011. http://dx.doi.org/10.1159/000327390.

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Adams, R. J., F. T. Nichols, D. Hughes et S. Hill. « Hemispheric Cerebral Blood Flow Changes with Hypervolemic Hemodilution Using Pentastarch After Acute Stroke ». Dans Cerebral Ischemia and Hemorheology, 502–7. Berlin, Heidelberg : Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1_60.

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Staedt, U., G. Schlierf, P. Oster, U. Mechtersheimer, G. Baessler, U. Seufzer, M. Hütt et H. Mörl. « Hypervolemic Hemodilution with 10% HES 200/0.5 and 10% Dextran 40 in Patients with Ischemic Stroke ». Dans Cerebral Ischemia and Hemorheology, 429–35. Berlin, Heidelberg : Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1_51.

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Krayenbühl, N., T. Hegner, Y. Yonekawa et E. Keller. « Cerebral Vasospasm after Subarachnoid Hemorrhage : Hypertensive Hypervolemic Hemodilution (Triple-H) Therapy According to new Systemic Hemodynamic Parameters ». Dans Cerebral Vasospasm, 247–50. Vienna : Springer Vienna, 2001. http://dx.doi.org/10.1007/978-3-7091-6232-3_53.

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Levy, Michael L., C. H. Rabb, V. Zelman et S. L. Giannotta. « The Use of Dobutamine to Enhance Cardiac Performance and Improve Outcome in Patients Refractory to Hypervolemic Therapy for Cerebral Vasospasm : a Preliminary Study ». Dans New Trends in Management of Cerebro-Vascular Malformations, 132–35. Vienna : Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9330-3_23.

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Jeyendran, Rajasingam S., et Milica Ivanovic. « Seminal Hypo- and Hypervolemia ». Dans Encyclopedia of Reproduction, 70–72. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-12-801238-3.64838-9.

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Actes de conférences sur le sujet "Hypervolemia"

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Ho, Kam S., Paaras Kohli, Yasmin Herrera, Archana Pattupara, Joseph Poon, Shabnam Nasserifar, Andre Sotelo et Raymonde Jean. « Go Easy with the Fluids ? Increased Mortality in Acute Respiratory Distress Syndrome with Hypervolemia ». Dans ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3442.

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Segizbaeva, Marina, et Nina Aleksandrova. « Physiological mechanisms of compensatory responses of the respiratory system to central hypervolemia : A modeling study ». Dans ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa2428.

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Ismail-Sayed, I., J. D. Foster et D. E. Kim. « Diffuse Alveolar Hemorrhage as a Manifestation of Hypervolemic State ». Dans American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1966.

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Anozie, O. O., G. Singh et S. Patel. « Use of Portal Vein Pulsatility to Differentiate Hypovolemic and Hypervolemic Hyponatremia ». Dans American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3446.

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