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Littérature scientifique sur le sujet « Hypoxia »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 25 juillet 2025

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Articles de revues sur le sujet "Hypoxia"

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Khaytsev, Nikolay Valentinovich, Andrey Glebovich Vasilyev, and Aleksandr Petrovich Trashkov. "THE EFFECT OF ADVANCE HYPOXIC TRAINING UPON TISSUE OXYGEN TENSION IN THE TUMOR DURING AQUTE HYPOXIA OF DIFFERENT TYPES." Pediatrician (St. Petersburg) 4, no. 1 (2013): 74–77. http://dx.doi.org/10.17816/ped4174-77.

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The tumors of rats who had not been acclimated to hypoxia subjected to acute hypoxic hypoxia (altitude chamber) as well as blood hypoxia (carbon monoxide) decreased tissue oxygen tension while histotoxic hypoxia (NaCN) on the contrary increased tissue oxygen tension. The tumor acclimated to hypoxia seems to select compensatory mechanisms mostly associated with increased tissue oxygen tension thus taking advantage of lower extent of tissue oxygen tension when subject to acute hypoxic hypoxia than in femoral muscle. All types of acute hypoxias caused a decrease of tissue oxygen tension in the ma

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Resta, T. C., J. M. Resta, and B. R. Walker. "Role of endogenous opioids and serotonin in the hemodynamic response to hemorrhage during hypoxia." American Journal of Physiology-Heart and Circulatory Physiology 269, no. 5 (1995): H1597—H1606. http://dx.doi.org/10.1152/ajpheart.1995.269.5.h1597.

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Previous studies from our laboratory indicate that acute but not chronic hypoxia decreases the hemorrhage volume required to elicit reflex hypotension. Furthermore, chronically hypoxic animals exhibit an elevated hypotensive threshold during both normoxia and hypoxia compared with control animals. Because reports suggest that opioid and serotonergic mechanisms may be involved in mediating the sympathoinhibition that occurs with hemorrhage, we hypothesized that opioid and/or serotonergic systems are stimulated during hemorrhage under conditions of acute hypoxia and suppressed after chronic expo

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Voronina, Tatiana A. "The role of hypoxia in stroke and convulsive states. Antihypoxants." Reviews on Clinical Pharmacology and Drug Therapy 14, no. 1 (2016): 63–70. http://dx.doi.org/10.17816/rcf14163-70.

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The review deals with the main types of hypoxia and the reasons leading to its development, discusses the development of mechanisms of hypoxiа. Particular attention is paid to brain hypoxia and its role in the development of strokes and convulsive states. The features of the application of antihypoxants and antioxidants at different hypoxic conditions including stroke and seizures are discussed.

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Lowry, T. F., H. V. Forster, M. J. Korducki, A. L. Forster, and M. A. Forster. "Comparison of ventilatory responses to sustained reduction in arterial oxygen tension vs. content in awake ponies." Journal of Applied Physiology 76, no. 5 (1994): 2147–53. http://dx.doi.org/10.1152/jappl.1994.76.5.2147.

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To gain insight into central and peripheral contributions to changes in breathing during hypoxia, we compared effects on breathing of reducing inspired PO2 (hypoxic hypoxia) with reducing arterial O2 content (CaO2) through elevation of carboxy-hemoglobin (COHb) (CO hypoxia). Twelve awake ponies were studied during 1 h of breathing room air followed by 6 h when COHb was increased to 25% and CaO2 was decreased by 17%. When COHb was increased, arterial PCO2 (PaCO2) increased gradually to 1.3 Torr above (P < 0.05) control level between 30 and 45 min of CO exposure. Pulmonary ventilation (VE) de

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Conde, S. V., E. C. Monteiro, R. Rigual, A. Obeso, and C. Gonzalez. "Hypoxic intensity: a determinant for the contribution of ATP and adenosine to the genesis of carotid body chemosensory activity." Journal of Applied Physiology 112, no. 12 (2012): 2002–10. http://dx.doi.org/10.1152/japplphysiol.01617.2011.

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Excitatory effects of adenosine and ATP on carotid body (CB) chemoreception have been previously described. Our hypothesis is that both ATP and adenosine are the key neurotransmitters responsible for the hypoxic chemotransmission in the CB sensory synapse, their relative contribution depending on the intensity of hypoxic challenge. To test this hypothesis we measured carotid sinus nerve (CSN) activity in response to moderate and intense hypoxic stimuli (7 and 0% O2) in the absence and in the presence of adenosine and ATP receptor antagonists. Additionally, we quantified the release of adenosin

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Yang, B. C., and J. L. Mehta. "Alterations in pulmonary artery tone during repeated episodes of hypoxia." American Journal of Physiology-Lung Cellular and Molecular Physiology 269, no. 3 (1995): L293—L298. http://dx.doi.org/10.1152/ajplung.1995.269.3.l293.

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To examine the basis of pulmonary constriction during chronic hypoxia, rat pulmonary artery rings were precontracted and exposed to multiple episodes of hypoxia. The first hypoxic episode resulted in a transient contraction, followed by potent relaxation, and then a slow sustained contraction. Repeated hypoxic exposure resulted in stronger initial contraction and attenuated relaxation. Prolongation of the normoxic interval between hypoxic episodes reversed the attenuation of hypoxic relaxation. Pulmonary artery rings that were deendothelialized or treated with the nitric oxide synthesis inhibi

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Dahan, A., D. Ward, M. van den Elsen, J. Temp, and A. Berkenbosch. "Influence of reduced carotid body drive during sustained hypoxia on hypoxic depression of ventilation in humans." Journal of Applied Physiology 81, no. 2 (1996): 565–72. http://dx.doi.org/10.1152/jappl.1996.81.2.565.

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To evaluate whether the intact hypoxic drive from the carotid bodies during sustained hypoxia is required for the generation of hypoxic depression of ventilation (VE), 16 volunteers were exposed to two consecutive periods of isocapnic hypoxia (first period 20 min; second period 5 min; end-tidal PO2 45 Torr) separated by 6 min of normoxia. In study A, saline was given. In study B, 3 micrograms.kg-1.min-1 i.v. dopamine (DA), a carotid body inhibitor, was given during the first hypoxic exposure followed by saline during normoxia and the second hypoxic exposure. In study C, 20 min of normoxia with

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Zonneveld, Marijke, Tom Keulers, and Kasper Rouschop. "Extracellular Vesicles as Transmitters of Hypoxia Tolerance in Solid Cancers." Cancers 11, no. 2 (2019): 154. http://dx.doi.org/10.3390/cancers11020154.

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Tumour hypoxia is a common feature of solid tumours that contributes to poor prognosis after treatment. This is mainly due to increased resistance of hypoxic cells to radio- and chemotherapy and the association of hypoxic cells with increased metastasis development. It is therefore not surprising that an increased hypoxic tumour fraction is associated with poor patient survival. The extent of hypoxia within a tumour is influenced by the tolerance of individual tumor cells to hypoxia, a feature that differs considerably between tumors. High numbers of hypoxic cells may, therefore, be a direct c

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Smith, Zachary M., Erin Krizay, Rui Carlos Sá, et al. "Evidence from high-altitude acclimatization for an integrated cerebrovascular and ventilatory hypercapnic response but different responses to hypoxia." Journal of Applied Physiology 123, no. 6 (2017): 1477–86. http://dx.doi.org/10.1152/japplphysiol.00341.2017.

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Ventilation and cerebral blood flow (CBF) are both sensitive to hypoxia and hypercapnia. To compare chemosensitivity in these two systems, we made simultaneous measurements of ventilatory and cerebrovascular responses to hypoxia and hypercapnia in 35 normal human subjects before and after acclimatization to hypoxia. Ventilation and CBF were measured during stepwise changes in isocapnic hypoxia and iso-oxic hypercapnia. We used MRI to quantify actual cerebral perfusion. Measurements were repeated after 2 days of acclimatization to hypoxia at 3,800 m altitude (partial pressure of inspired O2 = 9

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Sattiraju, Anirudh, Sangjo Kang, Valerie Marallano, et al. "TAMI-59. RECIPROCAL IMPACT OF CANCER IMMUNITY AND TUMOR HYPOXIA DURING GLIOBLASTOMA PROGRESSION." Neuro-Oncology 23, Supplement_6 (2021): vi210. http://dx.doi.org/10.1093/neuonc/noab196.841.

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Abstract Tumor hypoxia is linked to poor outcome for glioblastoma (GBM), a highly malignant brain cancer, but the underlying mechanisms and the environmental factors that initiate tumor hypoxia are poorly understood. We tracked tumor hypoxia in GBM in immunocompetent mice with a hypoxia sensitive fluorescent reporter combined with single cell transcriptomics. We found that hypoxic GBM cells are quiescent, immunosuppressive and display a mesenchymal transition, all of which are linked to malignant potency. We also captured in vivo hypoxia gene signature, which is more represented in recurrent G

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Thèses sur le sujet "Hypoxia"

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O'Dell, Adam David. "Hypoxia." Bowling Green State University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1490629987974442.

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Chacaroun, Samarmar. "Stratégies thérapeutiques par conditionnement hypoxique : modalités pratiques et effets sur la santé cardio-respiratoire et métabolique." Thesis, Université Grenoble Alpes (ComUE), 2018. http://www.theses.fr/2018GREAS020/document.

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L’hypoxie désigne une baisse de la biodisponibilité en oxygène au niveau tissulaire. La combinaison de l’hypoxie intermittente et de l’hypercapnie est identifiée dans le cadre de plusieurs maladies respiratoires comme un élément physiopathologique déterminant. Cependant, des travaux de recherche suggèrent qu’une exposition à l’hypoxie hypo- ou normocapnique à l’éveil peut améliorer la santé cardiovasculaire. La combinaison d’une exposition hypoxique et de l’entraînement à l’effort est utilisée par les athlètes pour améliorer la performance sportive aérobie. Des études pilotes récentes y compri

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Liang, Dinghua. "Progress towards hypoxia-activated SN-38: the potential to target hypoxic tumors." Bentham Science, 2015. http://hdl.handle.net/1993/31591.

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Solid tumors are commonly subject to hypoxia. Hypoxic cancer cells have undesirable properties such as a high tendency to metastasize and resistance to chemotherapy and radiotherapy. Hypoxia-inducible factors (HIFs) respond to the changes in oxygen levels, orchestrating the transcription of many proteins that are vital for the survival of hypoxic cancer cells. With their parent drug SN-38 as an inhibitor of both topoisomerase 1 and HIF-1, hypoxia-activated SN-38s may have a dual inhibitory effect on hypoxic tumor cells due to hypoxia-targeting and HIF-1 inhibition. To develop hypoxia-activa

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Peters, Caren Lorraine. "Hypoxia in inflammation." Thesis, University of Bath, 2003. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426151.

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Lau, Yue-huen Thomas. "Nuclear transcription factors and hypoxia-inducible genes in chronic liver hypoxia." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31939302.

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Lau, Yue-huen Thomas, and 劉汝這. "Nuclear transcription factors and hypoxia-inducible genes in chronic liver hypoxia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31939302.

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Fantacci, M. "In vivo distribution of Hypoxia-Inducible Factor-1α and DNA fragmentation in hypoxic tumoral and non-tumoral cells: correlation between chronic hypoxia and apoptosis". Doctoral thesis, Università degli Studi di Milano, 2004. http://hdl.handle.net/2434/154279.

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Querido, Jordan S. "Intermittent hypoxia : cardiorespiratory and cerebrovascular consequences to acute hypoxia and submaximal exercise." Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/32125.

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Intermittent hypoxia (IH) is broadly defined as repeatedly breathing decreased amounts of oxygen (hypoxia) interspersed with periods of room air breathing (normoxia). In animal, human diseased, and healthy human models, research has shown IH to negatively affect cerebrovascular vessel dilation. We have previously shown poikilocapnic (uncontrolled carbon dioxide (CO₂)) IH to blunt the vasodilatory response of a cerebral vessel during acute hypoxia. The purpose of this study was to measure the ventilatory, cardiovascular and cerebrovascular responses to: I) acute hypoxia and; II) to submaximal

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Smith, Quintina Denine. "From here to hypoxia." College Park, Md. : University of Maryland, 2006. http://hdl.handle.net/1903/3614.

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Thesis (M.F.A.) -- University of Maryland, College Park, 2006.<br>Thesis research directed by: Dept. of Art. Title from t.p. of PDF. Includes bibliographical references. Published by UMI Dissertation Services, Ann Arbor, Mich. Also available in paper.

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Holland, Jason P. "Hypoxia-Selective Copper Radiopharmaceuticals." Thesis, University of Oxford, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.491536.

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This thesis presents detailed experimental and computational investigations into the chemistry of copper(II) and zinc(II) bis(thiosemicarbazonato) complexes as potential' radiopharmaceutical agents for imaging and therapy. Chapter 1 provides an introduction to the field of molecular imaging in medicine with particular emphasis on the role of positron emission tomography in oncology. Key aspects of tumour physiology are discussed, and the effects of tumour hypoxia on radiotherapy and chemotherapy are considered. A range of hypoxia-selective radiopharmaceuticals are also introduced including a r

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Livres sur le sujet "Hypoxia"

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Roach, Robert C., Peter D. Wagner, and Peter H. Hackett, eds. Hypoxia. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-8997-0.

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Huang, L. Eric, ed. Hypoxia. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7665-2.

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Roach, Robert C., Peter D. Wagner, and Peter H. Hackett, eds. Hypoxia. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4757-3401-0.

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Roach, Robert C., Peter D. Wagner, and Peter H. Hackett, eds. Hypoxia. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4711-2.

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Roach, Robert C., Peter H. Hackett, and Peter D. Wagner, eds. Hypoxia. Springer US, 2016. http://dx.doi.org/10.1007/978-1-4899-7678-9.

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Gilkes, Daniele M., ed. Hypoxia. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3633-6.

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service), ScienceDirect (Online, ed. Hypoxia. Academic Press, 2009.

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Rosenthal, Myron, Peter L. Lutz, Thomas J. Sick, and Peter W. Hochachka. Surviving Hypoxia. CRC Press, 2022. http://dx.doi.org/10.1201/9780367813048.

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Justic, Dubravko, Kenneth A. Rose, Robert D. Hetland, and Katja Fennel, eds. Modeling Coastal Hypoxia. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54571-4.

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Machulla, Hans-Jürgen, ed. Imaging of Hypoxia. Springer Netherlands, 1999. http://dx.doi.org/10.1007/978-94-017-1828-8.

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Chapitres de livres sur le sujet "Hypoxia"

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Sainburg, Robert L., Andrew L. Clark, George E. Billman, et al. "Hypoxia, Focus Hypoxic Hypoxia." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_107.

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Walley, K. R. "Hypoxic Hypoxia." In Tissue Oxygenation in Acute Medicine. Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-58268-4_6.

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Harr, Jeffrey N., Philip F. Stahel, Phillip D. Levy, et al. "Hypoxic Hypoxia." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1729.

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Sainburg, Robert L., Andrew L. Clark, George E. Billman, et al. "Hypoxia, Focus Hypobaric Hypoxia." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_98.

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Brand, Marc. "Hypoxia." In Common Surgical Diseases. Springer New York, 1998. http://dx.doi.org/10.1007/978-1-4757-2945-0_101.

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Gibson, Gary E. "Hypoxia." In Cerebral Energy Metabolism and Metabolic Encephalopathy. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1209-3_3.

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Gardner, Lawrence B. "Hypoxia." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_2926-2.

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Jain, K. K. "Hypoxia." In Textbook of Hyperbaric Medicine. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-47140-2_5.

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Fischer, Bernd, Kewal K. Jain, Erwin Braun, and Siegfried Lehrl. "Hypoxia." In Handbook of Hyperbaric Oxygen Therapy. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-72990-4_3.

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Moses, Marsha A. "Hypoxia." In Encyclopedia of Systems Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_1546.

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Actes de conférences sur le sujet "Hypoxia"

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Lee, Jason S., Richard I. Ray, Brenda J. Little, and Edward J. Lemieux. "An Evaluation of Ballast Tank Corrosion in Hypoxic Seawater." In CORROSION 2006. NACE International, 2006. https://doi.org/10.5006/c2006-06300.

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Abstract Investigations were undertaken to evaluate the impact of hypoxia on corrosion within ballast tanks. Carbon steel coupons were exposed to seawater in alternating fill/drain conditions encountered in operational ballast tanks. The following alternating exposure conditions between fill/drain conditions were examined: 1) oxygenated seawater/air, 2) hypoxic seawater (0.2 mg/L O2)/hypoxic gas. The effect of oxygen on corrosion in a hypoxic environment was evaluated by introducing oxygenated air into the hypoxic chamber at the midpoint of the experiment. Differences in corrosion rate and mor

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Lee, Jason S., Richard I. Ray, Brenda J. Little, and Edward J. Lemieux. "Evaluation of Deoxygenation as a Corrosion Control Measure for Ballast Tanks." In CORROSION 2005. NACE International, 2005. https://doi.org/10.5006/c2005-05217.

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Abstract Field experiments designed to evaluate deoxygenation of natural seawater as a corrosion control measure for unprotected seawater ballast tanks demonstrated decreased corrosion in hypoxic (&lt; 0.2 ppm O2) seawater using weight loss and linear polarization measurements. The experiments also demonstrated the difficulty of maintaining hypoxic seawater. Using a gas mixture it was possible to displace dissolved oxygen in natural seawater. However, aerobic respiration and corrosion reactions consumed oxygen and produced totally anaerobic conditions within the first days of hypoxia. When

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Markov, Nikolay, and Vladimir Filatov. "TECHNOLOGIES OF AUTOMATED NOTIFICATION OF HUMAN HEALTH AND PERFORMANCE RISKS UNDER HYPOXIC HYPOXIA." In XIV International Scientific Conference "System Analysis in Medicine". Far Eastern Scientific Center of Physiology and Pathology of Respiration, 2020. http://dx.doi.org/10.12737/conferencearticle_5fe01d9d39e494.70316289.

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The technologies of automated notification of health risks and human performance under hypoxic hypoxia are described, which provide the calculation and presentation in real time of visual and acoustic information about the value of the assessment of the reserve time for maintaining human health and performance under hypoxic hypoxia

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Amorim, Ísis Salviano Soares de, Priscyanne Barreto Siqueira, Mariana Moreno de Sousa Rodrigues, and Andre Luiz Mencalha. "GENOMIC AND CLINICAL DATA ANALYSIS OF APE1 PROTEIN, BREAST CANCER STEM CELL PHENOTYPE, AND HYPOXIC TUMOR MICROENVIRONMENT." In Brazilian Breast Cancer Symposium 2022. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s2004.

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Introduction: Breast cancer (BC) is a heterogeneous disease at cellular and molecular levels. BC tumors present a cellular subpopulation of breast cancer stem cells (BCSCs) linked with tumor initiation and progression, recurrence, and therapeutic failure. The BCSC is preferentially found in hypoxic areas of the tumor, which are common features of BC and are significantly associated with worse prognosis. Although hypoxia activates an aggressive BCSC phenotype, the proteins that perform this molecular crossroad are still unknown. Therefore, finding proteins that performed this crossing would hel

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Кечерюкова, Тахмина Мажитовна, Анна Александровна Шульга, and Анна Сергеевна Гончарова. "IN VIVO STUDY THE ACTION OF HYPOXIC CONDITIONS ON THE PROLIFERATIVE POTENTIAL OF LIVER CANCER CELLS." In Фундаментальные и прикладные исследования. Актуальные проблемы и достижения: сборник статей XXII всероссийской (национальной) научной конференции (Санкт-Петербург, Октябрь 2023). Crossref, 2023. http://dx.doi.org/10.37539/231004.2023.23.56.004.

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Гипоксия играет важную роль во многих биологических процессах, включая пролиферацию клеток. В этом исследовании были определены уровни экспрессии маркера пролиферации Ki-67 в тканях опухоли печени с гипоксическими условиями и без гипоксии. Hypoxia play critical roles in various biological processes, icluding cell proliferation. This study expression of the proliferation marker Ki-67 was measured in liver tumor tissues with hypoxic conditions and without hypoxia.

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Tanishita, Kazuo, Kazuto Masamoto, Iwao Kanno, and Hirosuke Kobayashi. "Biotransport to the Cerebral Tissues Related to the Vascular Diseases." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192501.

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Brain is a highly oxidative organ and its consumption rate of oxygen accounts for 20 percent of that of the whole body. This large consumption rate must be met by continuous supply of oxygen, because lack of oxygen rapidly causes irreversible damage to central nervous system. Acute hypoxic episodes cause a certain pattern of regional damage. Cerebral cortex (e.g., layers III, V, and VI) is one of the most susceptible regions to hypoxia, and damage to sensorimotor function is particularly severe in humans that survive hypoxic/ischemic episodes. However, little is known about whether oxygen tran

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Klinnikova, A. A., G. A. Danilova та N. P. Aleksandrova. "The role of neuronal NO synthase in the respiratory effects of TNF-α". У VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-115-118.

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It was shown that an increase level of proinflammatory cytokines has a modulating effect on the reflex control of respiration. The aim of this study was to investigate the involvement of neuronal nitric oxide synthase (nNOS) in the mechanisms of the influence of an increased level of Tumor necrosis factor – α (TNF-α) on the hypoxic ventilatory response. To achieve this goal, experiments were carried out on urethane anesthetized rats with intravenous administration of TNF-α before and after pretreatment with 7-nitroindazole specific nNOS inhibitor. The hypoxic ventilation response was assessed

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Морякина, Светлана Васильевна, Хадижат Адамовна Гагаева, and Тамила Исаевна Матиева. "EFFECTS OF PHYSICAL STRESS ON VISUAL ACUITY IN THE PLAIN AND MOUNTAIN CONDITIONS." In Сборник избранных статей по материалам научных конференций ГНИИ «Нацразвитие» (Санкт-Петербург, Ноябрь 2021). Crossref, 2021. http://dx.doi.org/10.37539/nov322.2021.81.79.007.

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Научная статья посвящена установлению изменений со стороны зрительных функций у студенток старших курсов очно-заочной формы обучения в условиях гипоксической и динамической гипоксии. В период акклиматизации к гипоксической гипоксии в течение пятнадцати дней наблюдалась волнообразность или двухфазность изменения остроты зрения. Повышение с первого по пятый день и постепенное снижение с десятого по пятнадцатый день. Scientific article is devoted to establishing changes on the part of visual functions in students of senior courses of full-time education in conditions of hypoxic and dynamic hypoxi

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Kobs, Ryan W., Nidal E. Muvarak, and Naomi C. Chesler. "Hypoxia-Induced Changes in the Mechanical Properties of the Mouse Pulmonary Artery." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43086.

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Hypobaric hypoxia produces pulmonary hypertension in mice which causes pulmonary vascular remodeling. To study the biomechanics of this process, mice were exposed to hypoxia for 0-(control), 10-, and 15-days. Using a pressurized arteriograph system, mechanical properties of the main pulmonary artery were measured and compared to the biological changes in the vessel wall measured histologically. 10- and 15-day hypoxic vessels were significantly stiffer when compared to 0-day vessels. This stiffness correlated with greater elastin and collagen content in the vessel wall.

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Bocharov, M. I. "Temporary cardiointerval organization with varying degrees of acute normobaric hypoxia in a healthy person." In VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-36-40.

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The influence of acute normobaric hypoxia (ANH) on the male heart chronotropic effects was studied. Thus, a mild degree of ANH (14.5 % O2, 20 min), causing a decrease in blood oxygenation by 6.3 abs. %, accompanied by an initial (by 5 min) decrease in the RR and QT intervals. The average degree of ANH (12.3% O2) leads to a decrease in blood oxygenation by 19.7 abs. %. At the same time, in inverse relationship to the developing hypoxemia, RR and QT significantly decrease. Corrected values (Pc, PQc, QRSc, QTc) increase during the action period of an average degree of hypoxia, indicating an incre

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Rapports d'organisations sur le sujet "Hypoxia"

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Morgan, Thomas, Elizabeth Combs, Megan Clayton, et al. The Effects of Hypoxic Hypoxia on Cognitive Performance Decay. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ada621541.

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Negri, Cristina, Shyam Nair, Leslie Ovard, and Henriette Jager. Bioenergy Solutions to Gulf Hypoxia. Office of Scientific and Technical Information (OSTI), 2018. http://dx.doi.org/10.2172/1475550.

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Wang, Weigang. Hypoxia in Invasion and Metastasis. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada485743.

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Nielsen, T. B., and J. L. Kidwell. Cell Biology of Hypoxia, 1996. Defense Technical Information Center, 1996. http://dx.doi.org/10.21236/ada340589.

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Mahoney, Sean J. Hypoxia, Monitoring, and Mitigation System. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada590823.

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Mahoney, Sean J. Hypoxia, Monitoring, and Mitigation System. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada604355.

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Mahoney, S. J. Hypoxia, Monitoring, and Mitigation System. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ada623229.

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Hewett, Kate J., Ian P. Curry, Edna Rath, and Stephanie M. Collins. Subtle Cognitive Effects of Moderate Hypoxia. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada511511.

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Phillips, Jeffrey B., Leslie A. Drummond, F. E. Robinson, and Matthew E. Funke. Hypoxia: Exposure Time Until Significant Performance Effects. Defense Technical Information Center, 2016. http://dx.doi.org/10.21236/ad1006779.

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Marshall, Lynne. Effect of Hypoxia on Speech Recognition in Noise. Defense Technical Information Center, 1987. http://dx.doi.org/10.21236/ada193943.

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