Littérature scientifique sur le sujet « Immun javob »

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Articles de revues sur le sujet "Immun javob"

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Ahmedova, Saodat Toshboltayevna, Mohinur Normurod qizi Aminova, and Gulnora Karim qizi Mardonqulova. "INSON ORGANIZMIDA IMMUN SISTEMANING TUTGAN O'RNI, VAZIFASI VA O'ZIGA XOS XUSUSIYATLARI." GOLDEN BRAIN 1, no. 2 (2023): 101–5. https://doi.org/10.5281/zenodo.7555494.

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<em>Immun sistemasi&nbsp;</em><em>organizmni</em><em>&nbsp;</em><em>kasalliklardan</em><em>&nbsp;himoya qiluvchi biologik jarayonlar tarmogʻidir. U&nbsp;</em><em>viruslardan</em><em>&nbsp;tortib&nbsp;</em><em>parazit chuvalchanglarga</em><em>&nbsp;qadar turli xil patogenlar, shuningdek, saraton hujayralari, hatto yogʻoch qirindilarigacha taniydi, ularga immunologik javob qaytaradi va ularni organizm sogʻlom&nbsp;</em><em>toʻqimalaridan</em><em>&nbsp;ajratib turadi. Koʻpgina biologik turlarda immun sistemasi ikkita asosiy kichik guruhdan iborat. Tugʻma immun sistemasi xilma-xil holatlar va taʼs
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To'ychiyev, H.H, and S.Ch. Eshkaraev. "REVMATIZM KASALLIKLARINING KELIB CHIQISH SABABLARI VA DAVOLASH USULLARI." MEDICINE, PEDAGOGY AND TECHNOLOGY: THEORY AND PRACTICE 2, no. 8 (2024): 91–96. https://doi.org/10.5281/zenodo.14601186.

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Ushbu maqolada revmatizm kasalliklarining asosiy etiologik omillari, klinik ko&lsquo;rinishlari, diagnostika usullari va zamonaviy davolash strategiyalari haqida batafsil ma&rsquo;lumot beriladi. Revmatizm asosan autoimmun jarayonlar bilan bog&lsquo;liq bo&lsquo;lib, organizmning o&lsquo;z to&lsquo;qimalariga hujum qiluvchi immun javob natijasida rivojlanadi. Kasallik bo&lsquo;g&lsquo;imlar, yurak va boshqa muhim organlarning shikastlanishiga olib kelishi mumkin. Ushbu maqolada kasallikni samarali nazorat qilish usullari, oldini olish choralari va bemorlar uchun tavsiyalar muhokama qilingan.
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Abdullayeva, R.O, and N.I Tursunova. "ENDOMETRIAL STROMAL SARKOMALARNING GETEROGENLIGI VA O'SIMTA MIKRO-MUHITINING TERAPEVTIK JAVOBGA TA'SIRI." Multidisciplinary Journal of Science and Technology 5, no. 5 (2025): 1205–9. https://doi.org/10.5281/zenodo.15532041.

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Endometrial stromal sarkoma (ESS) bachadonning kam uchraydigan malign o&lsquo;smalaridan biri bo&lsquo;lib, uning molekulyar va gistologik xilma-xilligi davolash natijalariga sezilarli ta&rsquo;sir ko&lsquo;rsatadi. Ushbu kasallikning heterogen tabiati individual davolash strategiyalarini ishlab chiqishni qiyinlashtiradi. Tadqiqot ESSning molekulyar geterogenligi va o&lsquo;simta mikro-muhitining (TME) terapevtik javobga ta&rsquo;sirini o&lsquo;rganadi. JAZF1-SUZ12 va YWHAE-NUTM2B kabi genetik translokatsiyalar o&lsquo;simtaning klinik kechishi, o&lsquo;sish tezligi va davolashga chidamliligin
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Ziyodullayeva, Iroda. ""Shegellalar – dizenteriya qo'zg'atuvchisi"." MULTIDISCIPLINARY JOURNAL: FUNDAMENTAL RESEARCH SCIENTIFIC JOURNAL 1, no. 4 (2025): 106–11. https://doi.org/10.5281/zenodo.15344811.

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Ushbu maqolada dizenteriya kasalligining asosiy etiologik omili bo&lsquo;lgan Shigella turkumiga mansub bakteriyalar &mdash; shegellalarning morfologik, biologik va epidemiologik xususiyatlari tahlil qilingan. Maqolada shegellalarning inson organizmiga kirish yo&lsquo;llari, ularning patogenlik mexanizmlari, shuningdek, immunologik javob va organizmda yuzaga keladigan o&lsquo;zgarishlar ilmiy asosda yoritilgan. Shuningdek, dizenteriyaning klinik belgilari, diagnostika usullari va zamonaviy davolash choralari haqida ma&rsquo;lumot berilgan. Antibiotiklarga nisbatan rivojlanayotgan rezistentlik
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Bhandari, Ashish, Michael F. Cuccarese, Kevin Fales, et al. "Abstract 1888: Identification and optimization of novel small molecule modulators of immune checkpoint resistance with a unified representation space for genomic and chemical perturbations." Cancer Research 82, no. 12_Supplement (2022): 1888. http://dx.doi.org/10.1158/1538-7445.am2022-1888.

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Abstract Immune checkpoint inhibitors have revolutionized cancer treatment, producing a durable response consistent with immunologic memory in a subset of patients. However, the majority of patients demonstrate innate or acquired resistance that must be characterized and overcome to induce successful treatment. Advancements in human reverse translation and scaled in vivo CRISPR screening have uncovered novel molecular and genomic correlates of resistance, and promising druggable mechanisms - driven by highly complex interactions between tumor cells and the immune system. It is this core biolog
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Negandhi, Priyanka, Amol Andhale, Sourya Acharya, Faizan Khan, and Tushar Patil. "Creutzfeldt-Jakob disease − A case report." International Journal of Nutrition, Pharmacology, Neurological Diseases 15, no. 1 (2025): 112–17. https://doi.org/10.4103/ijnpnd.ijnpnd_154_24.

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Creutzfeldt-Jakob disease (CJD) is a brain disorder that causes dementia. It forms part of a set of animal and human diseases called prion disorders. Symptoms of Creutzfeldt-Jakob disease can resemble those of Alzheimer’s disease. This disease is always fatal, and most patients do not survive beyond a year. Myoclonus, vision problems, brain and pyramidal/extrapyramidal symptoms, and changes in cognitive function and cognitive abilities are seen as manifestations. We present a case of a 65-year-old woman complaining of rapidly progressive dementia, which was followed by the slowness of movement
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Bui, Esther, Eric Ehrensperger, Demetrios J. Sahlas, et al. "Inflammatory Cerebrospinal Fluid in Sporadic Creutzfeldt-Jakob Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 35, no. 5 (2008): 625–29. http://dx.doi.org/10.1017/s0317167100009422.

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Background:Sporadic Creutzfeldt-Jakob disease (CJD) is a fatal, transmissible spongiform encephalopathy characterized by rapidly progressive dementia, myoclonus, ataxia and akinetic mutism. The underlying mechanism is believed to be a conformational change of a native prion protein which characteristically fails to provoke an immune response. Commensurate with the latter, cerebrospinal fluid (CSF) classically exhibits a non-inflammatory profile.Cases:We report two patients with pathologically-proven sporadic CJD presenting with a significant CSF pleocytosis.Conclusion:Although uncommon, the pr
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Milroy, A. M., E. Bouzamondo, H. J. Ralston III, S. B. Prusiner, and S. J. DeArmond. "The Plain and the Ugly Prion Infected Neuronal Tissue in an Experimental Animal Model; An Electron Microscopic Study." Microscopy and Microanalysis 6, S2 (2000): 646–47. http://dx.doi.org/10.1017/s1431927600035728.

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Transmissible encephalopathy of animals (scrapie and bovine spongiform encephalopathy) and of man (Creutzfeldt-Jacob disease, new variant Creutzfeldt-Jacob disease, Kuru, Gerstmann-Straussler-Scheinker disease and familial fatal insomnia) have been well characterized as progressive neurodegenerative diseases, often associated with spongiform degeneration, neuronal loss, reactive astrocytic gliosis and variable amyloid plaques, without any sign of an immune response or inflammatory infiltrates. Prion proteins are elements that propagate variability through multiple biologically active conformer
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Patel, Bhavika, Stephanie Allen, Brenna Dennison, et al. "Abstract 6666: Multispectral imaging to detect immune phenotypes in pre and post therapy breast cancer patient specimens." Cancer Research 83, no. 7_Supplement (2023): 6666. http://dx.doi.org/10.1158/1538-7445.am2023-6666.

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Abstract The immune microenvironment is an important component in cancer therapy. Immune cells can encourage tumor growth, leading to disease progression. A high number of immune cells may be predictive of disease prognosis and response to therapies. Therefore, understanding the immune cell phenotypes present within cancerous tissue can be valuable in developing strategies that aid in the treatment of cancer. In this study, we analyzed pre-and post-therapy samples from breast cancer patients, treated with neoadjuvant chemotherapy, using a 6-plex immunofluorescence assay which included clinical
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Beretta, Simone, Andrea Stabile, Claudia Balducci, et al. "COVID‐19‐associated immune‐mediated encephalitis mimicking acute‐onset Creutzfeldt‐Jakob disease." Annals of Clinical and Translational Neurology 8, no. 12 (2021): 2314–18. http://dx.doi.org/10.1002/acn3.51479.

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Actes de conférences sur le sujet "Immun javob"

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Gouvêa, Sabrina Vechini, Victor Guimarães Correa, Giovana Barros e. Silva Ribeiro, et al. "A Creutzfeldt-Jakob disease mimics: case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.562.

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Introduction: Creutzfeld-Jakob Disease (CJD) is an important differential diagnoses in patients with rapidly progressive dementia (RPD). Several inflammatory, immune-mediated, vascular, granulomatous and neurodegenerative conditions can mimic this disease. Objectives: Report a curious case of CJD mimics that was a limbic encephalitis. Methods: A 71-year-old man, with diabetes mellitus. His initial symptoms included visual hallucinations and temporo-spatial disorientation. Over the course of a month, he lost his critical judgment, and began with aggressive behavior and disinhibition. He progres
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Guelli, Mariana Sandoval Terra Campos, Daniela Bastos de Almeida Zampier, Lorena Araújo Silva Dias, and Marina de Oliveira Nunes Ibrahim. "Creutzfeldt-Jakob Disease - a literature review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.126.

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Background: Creutzfeldt-Jakob disease (CJD) is a progressive, rare, fatal and rapid human neurodegenerative disease that occurs in the etiologies: sporadic (CJD), familial, iatrogenic (CJD) and CJD variant (CJV) in which cell prion protein (PrP) can be transmitted through animals. Objectives: Literature review about Creutzfeldt-Jakob diseaseDesign and setting: Literature review development in the Centro Universitário de Volta Redonda, Rio de Janeiro, Brazil. Methods: The Creutzfeldt-Jakob disease, infectious diseases and neuroinfection indexes were used in the PUBMED and Scielo databases. Resu
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