Littérature scientifique sur le sujet « Infectious endocarditis »

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Articles de revues sur le sujet "Infectious endocarditis"

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WOLFF, M. "Endocardites infectieuses gravesSerious infectious endocarditis." Réanimation 10, no. 3 (2001): 282–90. http://dx.doi.org/10.1016/s1164-6756(01)00116-5.

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Carceller, A. "Infectious endocarditis." Anales de Pediatría 63, no. 5 (2005): 383–88. http://dx.doi.org/10.1157/13080399.

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Maclagan, E. "Infectious Endocarditis." Canadian Journal of Cardiology 29, no. 10 (2013): S411. http://dx.doi.org/10.1016/j.cjca.2013.07.758.

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Porubčinová, Ingrid, Michal Hulman, and Jana Jevčáková. "Libman-Sacks endocarditis versus infectious endocarditis." Cor et Vasa 48, no. 7-8 (2006): 281–84. http://dx.doi.org/10.33678/cor.2006.087.

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Pesavento, P. A., B. B. Chomel, R. W. Kasten, K. A. McDonald, and F. C. Mohr. "Pathology of Bartonella Endocarditis in Six Dogs." Veterinary Pathology 42, no. 3 (2005): 370–73. http://dx.doi.org/10.1354/vp.42-3-370.

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In a 5-year retrospective study of dogs presenting to the Veterinary Medical Teaching Hospital at the University of California, Davis, there were 31 histologic diagnoses of valvular endocarditis. By polymerase chain reaction (PCR) amplification of embedded valvular tissue, Bartonella organisms were exclusively associated with 6 out of 31 cases (19%). Confirmed Bartonella cases involved the aortic valve alone (five out of six) or in combination with the mitral valve (one of six). Microscopic features of Bartonella endocarditis were compared with valves from non -Bartonella endocarditis and with
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Popov, D. A., R. A. Osokina, T. Yu Vostrikova, et al. "Infectious endocarditis caused by Cutibacterium avidum in a patient with endocardial pacemaker and multiple drug hypersensitivity reactions: a clinical case." Clinical Microbiology and Antimicrobial Chemotherapy 26, no. 2 (2024): 208–14. http://dx.doi.org/10.36488/cmac.2024.2.208-214.

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Cutibacterium avidum is a well-known skin commensal, that sometimes can cause superficial or invasive infections, including infectious endocarditis. Cases of C. avidum-induced infectious endocarditis of native heart valves, prosthetic valves or other cardiac implantable devices are described, which is associated with the biological feature of the pathogen with a tendency to form biofilms. The article presents a clinical case of infectious endocarditis, caused by C. avidum, associated with implantation of pacemaker in 42-yearold patient with a history of multiple drug hypersensitivity reactions
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Shonbin, Alexey N., Dmitry O. Bystrov, Boris O. Afonin, and Roman O. Sorokin. "SURGICAL TREATMENT OF INFECTIOUS ENDOCARDITIS." Acta medica Eurasica, no. 4 (December 26, 2022): 73–79. http://dx.doi.org/10.47026/2413-4864-2022-4-73-79.

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Infectious endocarditis is one of extremely dangerous diseases. Despite the opportunities of modern medicine, hospital mortality in infectious endocarditis remains high. Most patients are operated on for urgent and emergency indications. the article presents 11 years of experience in surgical treatment of infectious endocarditis at the State Budgetary Healthcare Institution JSC "PGKB named after E.E. Volosevich" in the town of Arkhangelsk. the study included 169 patients with infectious endocarditis, verified based on the presence of criteria corresponding to the modified Duke criteria. Infect
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&NA;. "Active Infectious Endocarditis." Survey of Anesthesiology 50, no. 4 (2006): 170–71. http://dx.doi.org/10.1097/01.sa.0000234681.56679.5a.

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Dobreva-Yatseva, B., F. Nikolov, R. Raycheva, and M. Tokmakova. "Infectious endocarditis – current profile." Bulgarian Cardiology 28, no. (4) (2022): 92–111. https://doi.org/10.3897/bgcardio.28.e90366.

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Infective endocarditis (IE) is a disease of the endocardium of the heart and the endocardium of the great vessels, with infection affecting heart valves (native or prosthetic) and subvalvular structures and, in the last few decades, indwelling intracardiac devices or catheters. It is a life-threatening disease with a wide distribution worldwide. IE was first described 350 years ago, but it continues to be a huge challenge for doctors for several reasons. First – IE is a changing disease. Nowadays, the profile of patients with IE has significantly changed in terms of age, predisposing fac
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Talipova, I. Zh, B. K. Zholdin, S. A. Seitmagambetova, G. L. Kurmanalina та D. E. Kushimova. "Infectious endocarditis сomplicated spondilodiscitis". Bulletin of Siberian Medicine 17, № 4 (2018): 287–93. http://dx.doi.org/10.20538/1682-0363-2018-4-287-293.

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This publication presents a clinical case of infectious endocarditis in an adult male aged 60, with manifestation of the disease in the form of spondilodiscitis. The presented observation demonstrates the development of onset of infectious endocarditis of bacterial spondylodiscitis in the adult patient. For a long time the patient’s intensive pain in the lumbar region could not be connected with infective endocarditis. According to the results of the examination the changes in the spine were not evaluated as an onset of IE. Dynamic observation and control MRT study allowed for the conclusion a
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Thèses sur le sujet "Infectious endocarditis"

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Thalme, Anders. "Infectious endocarditis, aspects on pathogenesis, diagnosis and prognosis /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-361-2/.

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MIKOLAJCZAK, NATHALIE. "Les endocardites fongiques : a propos de trois observations." Lille 2, 1994. http://www.theses.fr/1994LIL2M326.

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Leyral, Jérôme. "Endocardites à Neisseria Mucosa à propos d'un cas : revue de la littérature." Bordeaux 2, 1997. http://www.theses.fr/1997BOR2M060.

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Werner, Maria. "Blood culture negative endocarditis /." Göteborg : Department of Infectious Diseases, The Sahlgrenska Academy at Göteborg University, 2006. http://hdl.handle.net/2077/776.

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MONTAGNESE, YANNICK. "Les endocardites a salmonelles : revue de la litterature a propos d'une observation d'une endocardite tricuspidienne a salmonella paratyphi a chez l'adolescent." Lille 2, 1992. http://www.theses.fr/1992LIL2M043.

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Colnet, François-Xavier de. "Anevrysmes mycotiques compliquant une endocardite a neisseria : a propos d'un cas." Amiens, 1988. http://www.theses.fr/1988AMIEM012.

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Delafoulhouze, Gilles. "Les endocardites à Haemophilus aphrophilus." Montpellier 1, 1992. http://www.theses.fr/1992MON11110.

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Sauvage, Christine. "Corynebacterium multi-resistants aux antibiotiques : etude bacteriologique et clinique a propos d'un cas d'endocardite a corynebacterium du groupe d2 a point de depart urinaire." Université Louis Pasteur (Strasbourg) (1971-2008), 1990. http://www.theses.fr/1990STR1M062.

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Mohee, Amar Raj. "Infections in urological practice : bacteraemia and infective endocarditis." Thesis, University of Leeds, 2014. http://etheses.whiterose.ac.uk/6839/.

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Introduction. It is known that infections may occur after urological instrumentation, as some patients develop infective symptoms. The purpose of this study was to investigate bacteraemia in patients undergoing transurethral resection of the prostate (TURP) and catheter manipulation, using contemporary culture methods. Another aim was to explore the potential for molecular methods to detect, identify and quantify bacteraemia. We aim to evaluate the association between urological instrumentation and the development of infective endocarditis (IE). Methods. Microbiological molecular methods to id
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BLANC-VANNET, PHILIPPE. "Les manifestations rhumatologiques des endocardites infectieuses." Lyon 1, 1991. http://www.theses.fr/1991LYO1M392.

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Livres sur le sujet "Infectious endocarditis"

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Siniawski, Henryk. Active Infective Aortic Valve Endocarditis with Infection Extension. Steinkopff, 2006. http://dx.doi.org/10.1007/3-7985-1629-4.

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Kocher, Ajar. Infective Endocarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0018.

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Infectious endocarditis (IE) is an infection of the heart’s innermost layer, the endothelium. Most cases require a predisposing injury to the endocardium to serve as a nidus for thrombus development, which in turn acts as nidus for bloodstream microorganisms. These intravascular microorganisms can result from dental and other invasive procedures, infected vascular catheters, and skin lesions. However, most episodes of IE result from transient bacteremia during menial tasks, such as chewing and brushing one’s teeth. Blood cultures and echocardiograms are critical for IE diagnosis. Transesophage
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Wilson, John W., and Lynn L. Estes. Infectious Syndromes in Adults. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696924.003.0005.

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This section contains tables and text covering an exhaustive group of infectious syndromes including respiratory tract infections, infective endocarditis, intravascular catheter-related infections, central nervous system infections, urinary tract infections, soft-tissue infections, osteomyelitis, gastrointestinal infections, tick-borne infections, tuberculosis, sexually transmitted diseases, HIV, hepatitis, and fungal and zoonotic infections. Vaccination schedules, travel medicine, and bioterrorism are also reviewed.
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Orenstein, Robert. Clinical Syndromes in Infectious Diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0411.

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This chapter approaches the field of infectious diseases from 3 perspectives. This second part covers clinical syndromes associated with various infections, such as infective endocarditis, meningitis, sexually transmitted infections, urinary tract infections, gastrointestinal infections, and soft-tissue infections. Symptoms, diagnosis, and treatment of these conditions are reviewed.
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Rodriguez-Iturbe, Bernardo, and Mark Haas. Post-infectious glomerulonephritis. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0076.

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Post-infectious glomerulonephritis (GN) defines an inflammatory lesion involving exclusively or predominantly the glomeruli that is a consequence of an infectious disease. There are numerous bacterial, viral, and fungal infections associated with GN. This chapter acts as an overview of the following chapters that discuss only post-streptococcal GN, immunoglobulin A-dominant GN associated with staphylococcal infections, GN associated with bacterial endocarditis, with infected ventriculoatrial shunts (‘shunt nephritis’), and GN associated with deep-seated infections (osteomyelitis, visceral absc
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Rodriguez-Iturbe, Bernardo, and Mark Haas. Glomerulonephritis associated with endocarditis, deep-seated infections, and shunt nephritis. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0079_update_001.

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Endocarditis is a cause of glomerulonephritis. Healthcare interventions (prosthetic valves, indwelling catheters, pacemaker wires) and intravenous drug abuse are presently the most common causes of endocarditis and Staphylococcus aureus is frequently the infecting bacteria. Shunt nephritis is a form of glomerulonephritis associated with infection of ventriculoatrial shunts implanted to relieve hydrocephalus and, typically, are caused by prolonged infections of low-pathogenicity microorganisms. This complication led to the replacement of the technique by ventriculoperitoneal shunts. Deep-seated
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Euster, Caren. Infection in the Intravenous Drug User. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0058.

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Injection drug abuse has spread worldwide and is increasing among young adults and adolescents. This chapter focuses on the management of acute infectious consequences of injection drug use (IDU), including skin and soft tissue infections, endocarditis, and systemic infections. The approach to infection is determined based upon etiology: local (injection site) infections, infections distant to the injection site, systemic infections, complications of primary infections, modifying factors, and infections associated with the patient with IDU’s lifestyle. Infections in patients with a history of
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Thuny, Franck, and Didier Raoult. Pathophysiology and causes of endocarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0160.

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Endocarditis is defined as an inflammation of the endocardial surface of the heart. This may include heart valves, mural endocardium or the endocardium that covers implanted material, such as prosthetic valves, pacemaker/defibrillator leads and catheters. Infective and non-infective-related causes must be distinguished. In most cases, the inflammation is related to a bacterial or fungal infection with oral streptococci, group D streptococci, staphylococci and enterococci accounting for 85% of episodes. Infective endocarditis (IE) is a serious disease with an incidence ranging from 30 to 100 ep
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Infective Endocarditis: Management in the Era of Intravascular Devices (Infectious Disease and Therapy). Informa Healthcare, 2007.

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Grisoli, Dominique, and Didier Raoult. Prevention and treatment of endocarditis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0161.

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Initially always lethal, the prognosis of infective endocarditis (IE) has been revolutionized by antibacterial therapy and valve surgery. Nevertheless, it remains one of the deadliest infectious diseases, with ≥30% of patients dying within a year of diagnosis. Its incidence has also remained stable at 25–50 cases per million per year, and results predominantly from a combination of bacteraemia and a predisposing cardiac condition, including endocardial lesions and/or intracardiac foreign material. While antibiotic prophylaxis is recommended by various learned societies to cover healthcare proc
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Chapitres de livres sur le sujet "Infectious endocarditis"

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Ewy, Gordon A. "Infectious Endocarditis." In Cardiology for the primary care Physician. Current Medicine Group, 2001. http://dx.doi.org/10.1007/978-1-4615-6601-4_29.

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Hayley, Bradley, and Kwan Leung Chan. "Infectious Complications in Infective Endocarditis." In Infective Endocarditis. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32432-6_10.

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Matheson, Eric. "Endocarditis." In Management of Antimicrobials in Infectious Diseases. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-239-1_13.

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Daghestani, Lena. "Endocarditis." In Management of Antimicrobials in Infectious Diseases. Humana Press, 2001. http://dx.doi.org/10.1007/978-1-59259-036-0_13.

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Samson, Anda. "Endocarditis." In Practical Clinical Microbiology and Infectious Diseases. CRC Press, 2020. http://dx.doi.org/10.1201/9781315194080-4-19.

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Ghadaki, Bahareh, and Deborah Yamamura. "Infective Endocarditis." In Evidence-Based Infectious Diseases. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119260363.ch4.

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Norton, Laura E., and Mary Anne Jackson. "Infective Endocarditis." In Introduction to Clinical Infectious Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-91080-2_10.

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Dhawan, Vinod K. "Infective Endocarditis." In Infectious Disease in the Aging. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-534-7_9.

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Terpenning, Margaret S. "Infective Endocarditis." In Infectious Disease in the Aging. Humana Press, 2001. http://dx.doi.org/10.1007/978-1-59259-026-1_8.

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Domachowske, Joseph, and Manika Suryadevara. "Infective Endocarditis." In Clinical Infectious Diseases Study Guide. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50873-9_15.

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Actes de conférences sur le sujet "Infectious endocarditis"

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Rodriguez, W., C. Castillo-Latorre, O. J. Cantres, A. Torres-Palacios, M. M. Rivera Agosto, and D. Sanchez-Paredes. "Infectious Pulmonary Emboli: Not Endocarditis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3922.

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Kühn, C., E. Rubalskii, S. Rümke, et al. "Universal Automated Molecular Diagnosis of Infectious Endocarditis." In 48th Annual Meeting German Society for Thoracic, Cardiac, and Vascular Surgery. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678791.

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Kumar, D., M. Saliaj, Z. Khan, and M. Bachan. "An Unusual Case of Infectious Mycotic Aneurysm Rupture with Endocarditis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3461.

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Tomberlin, A., M. Khan, and K. Bird. "IV Drug Use and Infectious Endocarditis With Tricuspid Aortic Valve Infection and Acquired Gerbode Defect." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a1635.

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Fuqua, J. L., A. Roohollahi, and R. Leonard. "A Rare Cause for a Broken Heart: Right-Sided Invasive Infectious Endocarditis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6913.

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Beramendi, Y. Labeaga, R. Pampín, A. León Barbosa, et al. "4CPS-076 Evaluation of the treatment and morbimortality of infectious endocarditis by staphylococcus aureus." In Abstract Book, 23rd EAHP Congress, 21st–23rd March 2018, Gothenburg, Sweden. British Medical Journal Publishing Group, 2018. http://dx.doi.org/10.1136/ejhpharm-2018-eahpconf.167.

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Melo, Elyabe, João Paulo Peres Lima, Carolina Tiemi Tonholo Ikedo, et al. "ANTI-MDA5 POSITIVE DERMATOMYOSITIS COMPLICATED BY INFECTIOUS ENDOCARDITIS AND MULTISYSTEMIC MANIFESTATIONS: A CASE REPORT." In XLI Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2024. https://doi.org/10.47660/cbr.2024.1753.

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Xiao, Dua, Raeann Dalton, Adam Fineman, Michael Benz, and Antonios Tsompanidis. "The AngioVac® Device Limits Lethal Complications of Sepsis and Severe Infective Endocarditis with a Large Tricuspid Valve Vegetation: A Case Report." In 27th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2023. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.55_2023.

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Introduction: Infectious endocarditis of the tricuspid valve is a common complication of intravenous drug use. Endocarditis caused by viridans streptococci can lead to heart valve vegetations which may be life threatening due to the potential for embolism and obstruction. Management of large valvular vegetations is often difficult due to the risks involved with open heart surgery, especially in patients with comorbid conditions. The AngioVac® device has been shown in rare cases to be effective at debulking vegetations without the need for invasive surgery. Case Presentation: We present a patie
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Gonzaga, Bruno Pissolati Mattos, Gabriel Chung, Darah Fontes da Silva Assunção, et al. "Case report: ultrasound examination in central retinal artery occlusion in a patient with infective endocarditis." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.514.

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Introduction: Central retinal artery occlusion (CRAO) is an important cause of sudden monocular blindness. It can be divided into arteritic and non-arteritic forms, with non-arteritic being the most common, typically caused by emboli. Ultrasound is useful to detect embolic CRAO, with retrobulbar spot sign commonly found in atherosclerotic etiology. Case report of a patient with CRAO secondary to infective endocarditis, obtained by medical records review. Case report: A 65-year-old male patient presented to the emergency department with sudden monocular vision loss one month from admission with
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Norris, M. R., J. Purewal, L. Alam, and N. Sakul. "Concurrent ST-Elevation Myocardial Infarction and Stroke as Sentinel Symptoms for Infectious Endocarditis Secondary to Erythroderma." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6933.

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Rapports d'organisations sur le sujet "Infectious endocarditis"

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Splitter, Gary A., Menachem Banai, and Jerome S. Harms. Brucella second messenger coordinates stages of infection. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7699864.bard.

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Aim 1: To determine levels of this second messenger in: a) B. melitensiscyclic-dimericguanosinemonophosphate-regulating mutants (BMEI1448, BMEI1453, and BMEI1520), and b) B. melitensis16M (wild type) and mutant infections of macrophages and immune competent mice. (US lab primary) Aim 2: To determine proteomic differences between Brucelladeletion mutants BMEI1453 (high cyclic-dimericguanosinemonophosphate, chronic persistent state) and BMEI1520 (low cyclicdimericguanosinemonophosphate, acute virulent state) compared to wild type B. melitensisto identify the role of this second messenger in esta
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