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Littérature scientifique sur le sujet « Inflammation Mediators »

Auteur : Grafiati
Publié le 4 juin 2021
Mis à jour le 25 juillet 2025

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Articles de revues sur le sujet "Inflammation Mediators"

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Finsnes, Finn, Torstein Lyberg, Geir Christensen, and Ole H. Skjønsberg. "Effect of endothelin antagonism on the production of cytokines in eosinophilic airway inflammation." American Journal of Physiology-Lung Cellular and Molecular Physiology 280, no. 4 (2001): L659—L665. http://dx.doi.org/10.1152/ajplung.2001.280.4.l659.

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Endothelin (ET)-1 has been launched as an important mediator in bronchial asthma, which is an eosinophilic airway inflammation. However, the interplay between ET-1 and other proinflammatory mediators during the development of airway inflammation has not been elucidated. We wanted to study 1) whether the production of ET-1 precedes the production of other proinflammatory mediators and 2) whether ET-1 stimulates the production of these mediators within the airways. These hypotheses were studied during the development of an eosinophilic airway inflammation in rats. The increase in ET-1 mRNA level
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GREAVES, M. W. "Inflammation and mediators." British Journal of Dermatology 119, no. 4 (1988): 419–26. http://dx.doi.org/10.1111/j.1365-2133.1988.tb03245.x.

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Sacca, R. "Mediators of inflammation." Current Opinion in Immunology 9, no. 6 (1997): 851–57. http://dx.doi.org/10.1016/s0952-7915(97)80189-6.

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Moore, Terry L., and Terry D. Weiss. "Mediators of inflammation." Seminars in Arthritis and Rheumatism 14, no. 4 (1985): 247–62. http://dx.doi.org/10.1016/0049-0172(85)90044-7.

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Lazarus, Stephen C. "Inflammation, Inflammatory Mediators, and Mediator Antagonists in Asthma." Journal of Clinical Pharmacology 38, no. 7 (1998): 577–82. http://dx.doi.org/10.1002/j.1552-4604.1998.tb04463.x.

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Sudo, Maki, and Koichi Fujimoto. "Diffusive mediator feedbacks control the health-to-disease transition of skin inflammation." PLOS Computational Biology 20, no. 1 (2024): e1011693. http://dx.doi.org/10.1371/journal.pcbi.1011693.

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The spatiotemporal dynamics of inflammation provide vital insights into the understanding of skin inflammation. Skin inflammation primarily depends on the regulatory feedback between pro- and anti-inflammatory mediators. Healthy skin exhibits fading erythema. In contrast, diseased skin exhibits expanding erythema with diverse patterns, which are clinically classified into five types: circular, annular, arcuate, gyrate, and polycyclic. Inflammatory diseases with expanding erythema are speculated to result from the overproduction of pro-inflammatory mediators. However, the mechanism by which fee
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Alderton, Gemma. "Lipid mediators of inflammation." Science 371, no. 6526 (2021): 248.9–250. http://dx.doi.org/10.1126/science.371.6526.248-i.

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Robinson, Dwight R. "Lipid Mediators of Inflammation." Rheumatic Disease Clinics of North America 13, no. 2 (1987): 385–405. http://dx.doi.org/10.1016/s0889-857x(21)00854-1.

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Laurent, G. J. "Mediators of Pulmonary Inflammation." Thorax 47, no. 9 (1992): 764. http://dx.doi.org/10.1136/thx.47.9.764-a.

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Lasky, Laurence A. "Combinatorial mediators of inflammation?" Current Biology 3, no. 6 (1993): 366–68. http://dx.doi.org/10.1016/0960-9822(93)90203-z.

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Thèses sur le sujet "Inflammation Mediators"

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Farrell, Adrian J. "Mediators of synovial inflammation." Thesis, University of Bristol, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284326.

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Woollard, Kevin J. "Mediators of monocyte activity in inflammation." Thesis, Aston University, 2003. http://publications.aston.ac.uk/11001/.

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The effects of incubation of CRP with human primary and monocytic cell lines were examined using monocytic cytokine expression, adhesion molecule expression and adhesion to endothelial cells and intracellular peroxide formation, as end points. Monocytic intracellular signalling events were investigated after interaction of CRP with specific CRP receptors on monocytes. These initial signalling events were examined for their role in modulating monocyric adhesipn molecule and cytokine expression. Monocyte recruitment and retention in the vasculature is also influenced by oxidative stress. Therefo
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Robinson, Emily. "Mediators of inflammation in acute neurotoxicity." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/mediators-of-inflammation-in-acute-neurotoxicity(a657668f-f6de-4fb6-878d-65846fc18e66).html.

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Neuroinflammation is a major feature of most neurodegenerative conditions, and can leadto the exacerbation of neuronal injury. Inflammatory challenges in the central nervoussystem (CNS) have been shown to activate peripheral immune cells, which subsequentlyinfiltrate into the brain. Concurrently, resident inflammatory cells in the CNS, such asmicroglia, become activated and release inflammatory mediators, including cytokines.The pro-inflammatory cytokine interleukin-1 (IL-1) is a key mediator of neuronal injury.Although two IL-1 agonists exist, IL-1α and IL-1β, the majority of research has foc
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Wesseldijk, Feikje. ""Inflammatory Soup" mediators of inflammation in CRPS." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/13553.

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Wilson, Susan Jane. "Mucosal inflammation in allergic rhinitis." Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295233.

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McKay, Anne. "The role of immune mediators in airway inflammation." Thesis, University of Glasgow, 2004. http://theses.gla.ac.uk/4828/.

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Asthma is a chronic inflammatory condition of the airways characterised by reversible airflow obstruction, airway hyper-responsiveness and inflammatory infiltrates in the airway walls containing eosinophils, T lymphocytes and mast cells. T helper (Th) lymphocyte subsets, defined by the cytokines they secrete, are thought to play a key role in the in the initiation and perpetuation of chronic airway inflammation. Th2 cells, producing interleukin (IL)-4, IL-5, IL-9 and IL-13, are thought to be of particular importance. In contrast, Thl cells producing interferon (IFN)-y may counteract the develo
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李蕙琛 and Wai-sum Rachel Li. "Control of adenosine in human umbilical vein endothelial cells during inflammation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39557479.

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Airila-Månsson, Stella. "Progression of periodontitis and influence of periodontal bacteria on release of inflammatory markers in Swedish adults /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-622-0/.

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Khan, Sarah Basir. "Mediators of inflammation and fibrosis in experimental crescentic glomerulonephritis." Thesis, Imperial College London, 2005. http://hdl.handle.net/10044/1/11303.

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Porter, John Robert Stephen. "Microvesicles as mediators of inflammation in severe burn injury." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/43963.

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The host response to a severe burn injury is characterised by exaggerated systemic inflammation. Early clinical manifestations include shock, respiratory failure, renal failure and immunosuppression. The signalling pathways that propagate the inflammatory response are unclear but have traditionally been thought to involve overspill of proinflammatory cytokines. The importance of microvesicles, sub-cellular membrane-bound particles, is increasingly being recognised in the context of intercellular communication. Although circulating microvesicles are elevated in proinflammatory states such as se
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Livres sur le sujet "Inflammation Mediators"

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R, Ruffolo Robert, and Hollinger Mannfred A, eds. Inflammation--mediators and pathways. CRC Press, 1995.

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A, Willoughby D., ed. Inflammation: Mediators and mechanisms. Churchill Livingstone for the British Council, 1987.

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A, Bray Michael, and Anderson Wayne H, eds. Mediators of pulmonary inflammation. M. Dekker, 1991.

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1948-, Higgs G. A., Williams T. J. 1945-, International Union of Pharmacology, and International Congress of Pharmacology (9th : 1984 : London, England), eds. Inflammatory mediators. VCH, 1985.

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G, Bazán Nicolás, ed. Lipid mediators in eye inflammation. Karger, 1990.

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F, Henry J., ed. Mediators of inflammation: The local response. Libbey, 1988.

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1937-, Cohen Stanley, ed. Chemical mediators of inflammation and immunity. Academic Press, 1986.

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1951-, Morgan Douglas W., Forssmann Ulf 1967-, and Nakada Marian T. 1961-, eds. Cancer and inflammation. Birkhauser, 2004.

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Sharma, Jagdish N. Topics in mediator pharmacology. Nova Science Publishers, 2011.

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George, Poste, Crooke Stanley T, and Smith, Kline, and French Research Symposium on New Horizons in Therapeutics (5th : 1987 : Philadelphia, Pa.), eds. Cellular and molecular aspects of inflammation. Plenum Press, 1988.

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Chapitres de livres sur le sujet "Inflammation Mediators"

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Saria, A., and J. M. Lundberg. "Neurogenic Inflammation." In Inflammatory Mediators. Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07834-9_8.

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Galvão, Izabela, Michelle A. Sugimoto, Juliana P. Vago, Marina G. Machado, and Lirlândia P. Sousa. "Mediators of Inflammation." In Immunopharmacology and Inflammation. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-77658-3_1.

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Gonçalves, William Antonio, Alesandra Côrte Reis Melão, Mauro Martins Teixeira, Barbara Maximino Rezende, and Vanessa Pinho. "Pro-resolving Mediators." In Immunopharmacology and Inflammation. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-77658-3_6.

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Saldeen, T. "The Fibrinolytic System in Inflammation." In Inflammatory Mediators. Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07834-9_9.

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Foreman, J. C. "The Role of Histamine in Inflammation." In Inflammatory Mediators. Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07834-9_2.

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Glaser, K. B., and M. S. Barnette. "Lipid Mediators: Mechanisms." In Inflammation: Mechanisms and Therapeutics. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7343-7_21.

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Ait-Oufella, Hafid, Ziad Mallat, and Alain Tedgui. "Mediators of Vascular Inflammation." In Inflammation and Atherosclerosis. Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0338-8_10.

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Hedqvist, Per, and Lennart Lindbom. "Lipid Mediators of Inflammation." In Physiology of Inflammation. Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4614-7512-5_7.

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Parker, C. W. "Lipid Mediators and Inflammation." In New Trends in Allergy II. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71316-3_8.

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Krane, Stephen M. "Peptide Mediators of Inflammation." In Cellular and Molecular Aspects of Inflammation. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-5487-1_5.

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Actes de conférences sur le sujet "Inflammation Mediators"

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Dalli, Jesmond, Ana Rodriguez, Bernd Spur, and Charles Serhan. "Structure elucidation and biological evaluations of sulfido-conjugated specialized pro-resolving mediators." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/mqgv6628.

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Inflammatory diseases are characterized by unabated inflammation that leads tissue destruction resulting in malaise. Whilst much is known on the mechanism that perpetuate inflammation, less is known about the molecules and pathways that coordinate the termination of inflammation and facilitate the repair and regeneration of damaged tissues. To evaluate the potential contribution of essential fatty acid-derived mediators in coordinating this life saving response we interrogated inflammatory exudates obtained following self-limited inflammatory challenge. Using radio-isotope tracking we found th
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Jones, Caleb W. "Reactive Aldehydes As Mediators Of Pulmonary Inflammation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2732.

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Bevilacqua, M. A., and M. A. Gimbrone. "LEUKOCYTE-ENDOTHELIAL INTERACTIONS: IMPLICATIONS FOR INFLAMMATION AND COAGULATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642948.

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A rapidly increasing body of data indicates that the vascular endothelium plays an active role in the development of inflammatory and thrombotic processes. Our laboratory has focused on the modulation of certain endothelial cell functions by inflammatory/immune mediators. Initially, we demonstrated that human monocyte derived interleukin-1 (hmIL-1) can act directly on cultured human endothelial cells (HEC) to increase the expression of tissue factor procoagulant activity in a time- and protein-synthesis dependent fashion (J. Exp. Med. 160:618, 1984). Increased expression of HEC tissue factor w
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Jain, V., I. Abdelgawwad, A. Brooks, et al. "Mediators of Antenatal Inflammation Propagating Chronic Neonatal Lung Disease." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a7388.

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Lim, Henry W. "Role mediators of inflammation and cells in porphyrin-induced phototoxicity." In OE/LASE '89, edited by Thomas J. Dougherty. SPIE, 1989. http://dx.doi.org/10.1117/12.978000.

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Baraldo, Simonetta, Simone Agostini, Graziella Turato, et al. "Non-eosinophilic Asthma In Children: Mediators Of Airway Remodeling And Inflammation." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2507.

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De Paula Vieira, R., R. Moraes-Ferreira, M. A. R. Brandao-Rangel, et al. "Aerobic Training Reduces Chronic Airway Inflammation and Mediators of Remodelingin Asthma." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.74.

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Pidaparti, Ramana M., and Kevin R. Ward. "Airway Inflammation Induced by Mechanical Ventilation Through Multiscale Modeling." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80174.

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Inflammation has been recognized as a major integral component for most of the acute and chronic diseases. Inflammation can be initiated within the body as an innate process or by external factors such as infections and trauma. Inflammation is a complex and dynamic process, and involves nonlinearity and stochasticity. Without the inflammation, the harmful stimuli cannot be removed and the healing process cannot occur. However, an over-expression or under-expression of inflammatory responses can lead to severe consequences, such as Multiple Organ Dysfunction Syndrome (MODS), which is characteri
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Branković, Jovica, Vesna Milovanović, and Vladimir P. Petrović. "CYCLOOXYGENASE-2 AS „IN SILICO“ TARGET OF PHENOLIC HYDRAZONE- TYPE DERIVATIVES." In 1st INTERNATIONAL Conference on Chemo and BioInformatics. Institute for Information Technologies, University of Kragujevac, 2021. http://dx.doi.org/10.46793/iccbi21.324b.

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In the present work, a series of phenolic hydrazone analogs were investigated in silico for their potential inhibitory activity toward COX-2. These examinations were based on the capability of hydrazone-based compounds to interact with numerous enzymes, as well as on their versatile biological features and therapeutical applications. COX-2 was selected due to its involvement in the inflammation and carcinogenesis processes. Regarding this, COX-2 represents a valid target for the development of compounds that could block the formation of harmful inflammation mediators.
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Al-amodi, O., H. Jodeleit, F. Beigel, E. Wolf, M. Siebeck, and R. Gropp. "CD1a expressing monocytes as sensors and mediators of inflammation in ulcerative colitis." In 46. Jahrestagung der Gesellschaft für Gastroenterologie in Bayern e.V. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1648568.

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Rapports d'organisations sur le sujet "Inflammation Mediators"

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Liu, Yangjun, Wei Xie, Zbigniew Ossowski, et al. Physical activity, abdominal obesity and inflammatory response in the elderly: a systematic review and meta-analysis of randomized-controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.3.0051.

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Review question / Objective: The purpose of this study was to explore the effects of physical activity (i.e., type of exercise, FITT criteria, control group, other interventions) on abdominal obesity and inflammatory response in elderly? The study method was a randomized controlled trial. Condition being studied: An increasing number of studies have demonstrated that chronic inflammation is closely associated with the initiation and progression of a broad range of age-related diseases, such as cardiovascular disease, cancer, diabetes, Alzheimer’s disease, and other neurodegenerative diseases a
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DeMar, James C., Miya I. Hill, Robert B. Gharavi, et al. Evaluation of Novel Polyunsaturated Fatty Acid Derived Lipid Mediators of Inflammation to Ameliorate the Deleterious Effects of Blast Overpressure on Eye and Brain Visual Processing Centers in Rats. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada606425.

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DeMar, James. Evaluation of Novel Polyunsaturated Fatty Acid Derived Lipid Mediators of Inflammation to Ameliorate the Deleterious Effects of Blast Over Pressure on Eye and Brain Visual Processing Centers in Rats. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ada621266.

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