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Articles de revues sur le sujet « Kernicteru »

Auteur : Grafiati

Publié le 1 avril 2023

Mis à jour le 31 juillet 2025

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1

Huda, Walia Murshida, Preeti Sharma, Juhi Aggarwa, and Alka Agrawal. "A Comparative Study of Cord Blood Bilirubin and Albumin as a Predictor for Neonatal Jaundice in Term Newborns." Journal of Datta Meghe Institute of Medical Sciences University 16, no. 2 (2021): 295–302. http://dx.doi.org/10.4103/jdmimsu.jdmimsu_80_21.

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Background: Neonatal hyperbilirubinemia (NH) is the commonest abnormal physiological finding during the 1st week of life. More than two-third of newborn babies develop clinical jaundice. The clinical finding such as yellowish discoloration of the skin and sclera in newborns is because of accumulation of unconjugated bilirubin. In most infants, however, unconjugated hyperbilirubinemia is a normal physiological phenomenon. Aim of the Study: The aim of the study was to predict the development of NH at birth in term newborns using cord blood bilirubin and albumin as a risk predictor. Materials and

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2

Ebbesen, Finn. "Kernicterus." Acta Obstetricia et Gynecologica Scandinavica 89, no. 5 (2010): 726. http://dx.doi.org/10.3109/00016340903540653.

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Stephenson, John B. P. "Kernicterus." European Journal of Paediatric Neurology 15, no. 2 (2011): 181. http://dx.doi.org/10.1016/j.ejpn.2010.12.002.

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Simpson, Kathleen Rice. "Kernicterus Prevention." MCN, The American Journal of Maternal/Child Nursing 32, no. 2 (2007): 132. http://dx.doi.org/10.1097/01.nmc.0000264309.07661.c3.

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Watchko, Jon F., and Frank A. Oski. "Kernicterus in Preterm Newborns: Past, Present, and Future." Pediatrics 90, no. 5 (1992): 707–15. http://dx.doi.org/10.1542/peds.90.5.707.

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This historical overview of kernicterus in prematurity, from the 1950s to the present, provides a unique perspective on this clinical conundrum. Three separate periods of pediatric history are detailed in relationship to our understanding of kernicterus in the preterm newborn: (1) the pre-intensive care era (1950 to 1965); (2) the low bilirubin kernicterus era (1965 to 1982); and (3) the 1980s. Each period demonstrates selected insights regarding kernicterus in prematurity, and together with recent reports suggest that premature newborns are now at extremely low risk of developing kernicterus

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Kasirer, Yair, Michael Kaplan, and Cathy Hammerman. "Kernicterus on the Spectrum." NeoReviews 24, no. 6 (2023): e329-e342. http://dx.doi.org/10.1542/neo.24-6-e329.

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Kernicterus is the potential toxic sequela of extreme neonatal hyperbilirubinemia resulting from the passage of excess free, unconjugated bilirubin across the blood−brain barrier, irreversibly and selectively damaging vulnerable target brain cells including the basal ganglia, the cerebellum, and the auditory system. Kernicterus continues to plague the modern world. Not only does it continue to be uncontrolled in developing countries with underdeveloped medical systems, and health organizations rendered ineffective by the ravages of war, but it also remains prevalent in industrialized countries

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7

Das, Sumit, and Frank van Landeghem. "Clinicopathological Spectrum of Bilirubin Encephalopathy/Kernicterus." Diagnostics 9, no. 1 (2019): 24. http://dx.doi.org/10.3390/diagnostics9010024.

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Bilirubin encephalopathy/kernicterus is relatively rare, but continues to occur despite universal newborn screening. What is more interesting is the spectrum of clinical and even neuropathological findings that have been reported in the literature to be associated with bilirubin encephalopathy and kernicterus. In this review, the authors discuss the array of clinicopathological findings reported in the context of bilirubin encephalopathy and kernicterus, as well as the types of diagnostic testing used in patients suspected of having bilirubin encephalopathy or kernicterus. The authors aim to r

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8

Millichap, J. Gordon. "MRI in Kernicterus." Pediatric Neurology Briefs 9, no. 10 (1995): 75. http://dx.doi.org/10.15844/pedneurbriefs-9-10-5.

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Shah, Zarine, Ashish Chawla, Deepak Patkar, and Sona Pungaonkar. "MRI in kernicterus." Australasian Radiology 47, no. 1 (2003): 55–57. http://dx.doi.org/10.1046/j.1440-1673.2003.00973.x.

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Shapiro, Steven M., Vinod K. Bhutani, and Lois Johnson. "Hyperbilirubinemia and Kernicterus." Clinics in Perinatology 33, no. 2 (2006): 387–410. http://dx.doi.org/10.1016/j.clp.2006.03.010.

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Leite, Emanuelly Falcão de Sousa, and Gidelson Gabriel Gomes. "Diagnósticos e tratamentos de kernicterus / diagnostic and treatment of kernicterus." Brazilian Journal of Health Review 4, no. 2 (2021): 9046–55. http://dx.doi.org/10.34119/bjhrv4n2-411.

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12

Belz, Katie M., Andrew J. Specht, Victoria S. Johnson, and Julia A. Conway. "MRI Findings in a Dog with Kernicterus." Journal of the American Animal Hospital Association 49, no. 4 (2013): 286–92. http://dx.doi.org/10.5326/jaaha-ms-5881.

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A severe increase in total bilirubin coincided with a decline in neurologic status to comatose in a 9 yr old spayed female mixed-breed dog being treated for immune-mediated hemolytic anemia. MRI of the brain was performed to investigate potential causes for the neurologic signs. MRI revealed bilaterally symmetrical hyperintensities within the caudate nuclei, globus pallidus, thalamus, deep cerebellar nuclei, and cortical gray matter on T2-weighted and fluid-attenuated inversion recovery (FLAIR) sequences, which coincided with areas of bilirubin deposition and neuronal necrosis (kernicterus) id

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13

Defiyuliyanti Bazikho, Efori Buulolo, and Meryance V. Siagian. "Implementasi Metode Tsukamoto Pada Sistem Pakar Diagnosa Kernikterus." Journal of Computing and Informatics Research 3, no. 1 (2023): 153–63. http://dx.doi.org/10.47065/comforch.v3i1.1004.

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Kernicterus is brain damage in infants due to high levels of bilirubin in the blood. Bilirubin is the cause of jaundice, and if left untreated, it can accumulate in the brain. The issue at hand is that the community, especially parents, still struggle to find a solution to determine the diagnosis of Kernicterus in a baby's body. Expert systems are advanced technology that can be used to address diagnostic problems with relevant accuracy. One of the expert system methods that can be employed for diagnosing diseases is Tsukamoto. Therefore, in this research, a expert system is built to obtain a

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14

Wang, Nanqin, Yongzhu Jia, Xuanzi Zhou, Xia Wang, Huyao Zhou, and Nong Xiao. "Effects of Repetitive Transcranial Magnetic Stimulation on Pallidum GABAergic Neurons and Motor Function in Rat Models of Kernicterus." Brain Sciences 13, no. 9 (2023): 1252. http://dx.doi.org/10.3390/brainsci13091252.

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Kernicterus is a serious complication of hyperbilirubinemia, caused by neuronal injury due to excessive unconjugated bilirubin (UCB) in specific brain areas. This injury induced by this accumulation in the globus pallidus can induce severe motor dysfunction. Repetitive transcranial magnetic stimulation (rTMS) has shown neuroprotective effects in various neurological diseases. This study aimed to investigate the effects of rTMS on pallidal nerve damage and motor dysfunction in a rat model of kernicterus. Rats were divided into a sham group (n = 16), a model group (bilirubin with sham rTMS; n =

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15

Juretschke, Linda. "Kernicterus: Still a Concern." Neonatal Network 24, no. 2 (2005): 7–19. http://dx.doi.org/10.1891/0730-0832.24.2.7.

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The presence of yellow staining and damage to the brain caused by unconjugated bilirubin was first described by Hervieux in 1847. Kernicterus, the technical term used to describe the intense yellow staining in the basal ganglia of the brain, was first used by Schmorl in 1903. Perhaps as many as 60 percent of all babies born each year in the U.S. are diagnosed with clinical jaundice. Kernicterus is a preventable brain injury caused by severe jaundice, yet it remains a threat today. Because of this, anyone caring for newborns must be aware of the risks and treatment for hyperbilirubinemia and th

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16

Muralt, G. "The Prophylaxis of Kernicterus." Developmental Medicine & Child Neurology 4, no. 2 (2008): 133–46. http://dx.doi.org/10.1111/j.1469-8749.1962.tb03122.x.

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17

YEUNG, C. Y. "Kernicterus in term infants." Journal of Paediatrics and Child Health 21, no. 4 (1985): 273–74. http://dx.doi.org/10.1111/j.1440-1754.1985.tb00163.x.

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18

Poland, Ronald L. "Preventing kernicterus: Almost there." Journal of Pediatrics 140, no. 4 (2002): 385–86. http://dx.doi.org/10.1067/mpd.2002.124310.

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19

Newman, Thomas B., and M. Jeffrey Maisels. "How to avoid kernicterus." Journal of Pediatrics 142, no. 2 (2003): 212–13. http://dx.doi.org/10.1067/mpd.2003.70.

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20

Cashore, William. "Kernicterus and Bilirubin Encephalopathy." Seminars in Liver Disease 8, no. 02 (1988): 163–67. http://dx.doi.org/10.1055/s-2008-1040536.

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21

Roth, Philip, and Richard A. Polin. "Controversial Topics in Kernicterus." Clinics in Perinatology 15, no. 4 (1988): 965–90. http://dx.doi.org/10.1016/s0095-5108(18)30687-0.

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Hansen, Thor Willy Ruud. "Kernicterus: an international perspective." Seminars in Neonatology 7, no. 2 (2002): 103–9. http://dx.doi.org/10.1053/siny.2002.0118.

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23

AlOtaibi, Suad F., Susan Blaser, and Daune L. MacGregor. "Neurological Complications of Kernicterus." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 32, no. 3 (2005): 311–15. http://dx.doi.org/10.1017/s0317167100004182.

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ABSTRACT:Objective and Background:Prevention of bilirubin encephalopathy relies on the detection of newborns who are at risk of developing serious hyperbilirubinemia. The objective of this study was to reassess the clinical syndrome of kernicterus as neurodiagnostic studies have become more readily available and can be used to evaluate these infants.Methods:The study population was neonates born at term or near term admitted to The Hospital for Sick Children in Toronto, Ontario, Canada, between January 1990 and May 2000. During the study period, there were 9776 admissions (average number of ad

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Okumura, A., H. Kidokoro, H. Shoji, et al. "Kernicterus in Preterm Infants." PEDIATRICS 123, no. 6 (2009): e1052-e1058. http://dx.doi.org/10.1542/peds.2008-2791.

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Waser, Marco, Paul Kleihues, and Paul Frick. "Kernicterus in an adult." Annals of Neurology 19, no. 6 (1986): 595–98. http://dx.doi.org/10.1002/ana.410190614.

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26

Manchanda, Smita, Akhila Prasad, and Barindra Prasad Baruah. "MRI Findings in Kernicterus." Indian Journal of Pediatrics 80, no. 2 (2012): 181–82. http://dx.doi.org/10.1007/s12098-012-0878-6.

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27

Gourley, Glenn R. "Bilirubin Metabolism and Kernicterus." Advances in Pediatrics 44, no. 1 (1997): 173–229. http://dx.doi.org/10.1016/s0065-3101(24)00052-5.

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IVES, N. KEVIN. "Kernicterus in Preterm Infants; Lest We Forget (To Turn on the Lights)." Pediatrics 90, no. 5 (1992): 757–59. http://dx.doi.org/10.1542/peds.90.5.757.

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Watchko and Oski have a reputation for stimulating debate on the topic of neonatal jaundice. As scriptwriters of "Vigintiphobia: a one-act play,"1 they questioned the `standard practice' applied to the management of jaundice in otherwise healthy term infants. In the current issue of Pediatrics2 they again court controversy by turning their attention to treatment thresholds and the risk of kernicterus in jaundiced preterm infants. We are provided with a thoroughly researched historical review of the risk of kernicterus in the preterm infant from 1950 to the 1990s. The story is presented as a jo

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Soeetomenggolo, Taslim S. "Blood Bilirubin Content in Neonatal Tetanus Patient with Hyperbilirubinemia during Treatment with Intravenous Diazepam." Paediatrica Indonesiana 32, no. 3-4 (2019): 59–64. http://dx.doi.org/10.14238/pi32.3-4.1992.59-64.

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An evaluation on 128 neonatal tetanus patients with hyperbilirubinemia was done at the Department of Child Health, Dr. Cipto Mangunkusumo General Hospital, Jakarta. The patients were treated with high dosages of diazepam intravenously. This drug is potential to cause the increase of blood bilirubin, and in turn this latter condition is potential to cause kernicterus. Of the 128 patients there were 70 males and 58 females. The age of the patients were mostly (79.6 %) 4- 7 days. Fourteen of the 128 patients showed the increase of their blood bilirubin content during the second day of

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30

STERN, LEO, and ROLF BRODERSEN. "Kernicterus Research and the Basic Sciences: A Prospect for Future Development." Pediatrics 79, no. 1 (1987): 154–56. http://dx.doi.org/10.1542/peds.79.1.154.

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Kernicterus was a major problem of pediatrics in the first half of this century. Important progress has taken place since pediatricians have mastered rhesus disease, introduced exchange transfusion, avoided giving sulfonamides, and have placed at risk infants under blue light. These measures, and above all the general improvements in intensive care in modern neonatology units, have reduced the problem so that kernicterus is now mainly seen as secondary to prematurity, respiratory distress, and severe infections.1 There, it remains a challenge to both clinical and basic science. The mechanism (

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Gregol, Patrícia Janaína, Amanda Soares Rodrigues, Grazielle Mecabô, Bárbara Sackser Horvath, and Anderson Felipe Ferreira. "ENCEFALOPATIA NEONATAL DECORRENTE DE TRATAMENTOS TARDIOS EM CASOS DE HIPERBILIRRUBINEMIA NEONATAL: UM ESTUDO DE REVISÃO." Brazilian Journal of Implantology and Health Sciences 6, no. 10 (2024): 2647–63. http://dx.doi.org/10.36557/2674-8169.2024v6n10p2647-2663.

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Introdução: A icterícia neonatal é uma condição clínica associada a neonatos, é caracterizada pela elevação de níveis séricos de bilirrubina indireta, gerando um acúmulo de pigmento amarelado na pele e mucosas, é uma condição frequentemente benigna, contudo se não for devidamente monitorada e tratada poderá desenvolver uma condição de toxicidade no sistema nervoso central, o kernicterus, descrito como um dano neurológico decorrente da alta concentração de bilirrubina não conjugada através da barreira hematoencefálica. Objetivo: Elaborar uma revisão de literatura, buscando compreender acerca da

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Brown, A. K. "Kernicterus: Past, Present, and Future." NeoReviews 4, no. 2 (2003): 33e—40. http://dx.doi.org/10.1542/neo.4-2-e33.

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Millichap, J. Gordon. "Kernicterus Re-Emergence and Prevention." Pediatric Neurology Briefs 14, no. 11 (2000): 84. http://dx.doi.org/10.15844/pedneurbriefs-14-11-6.

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Newman, T. B., M. J. Maisels, R. A. Zimmerman, and M. C. Harris. "Magnetic Resonance Imaging and Kernicterus." PEDIATRICS 109, no. 3 (2002): 555. http://dx.doi.org/10.1542/peds.109.3.555-a.

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Newman, Thomas B., and M. Jeffrey Maisels. "Magnetic Resonance Imaging and Kernicterus." Pediatrics 109, no. 3 (2002): 555. http://dx.doi.org/10.1542/peds.109.3.555b.

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HERNÁNDEZ CH, MARTA, M. IGNACIA SCHMIDT C, and ISIDRO HUETE L. "Encefalopatía por Kernicterus: Serie clínica." Revista chilena de pediatría 84, no. 6 (2013): 659–66. http://dx.doi.org/10.4067/s0370-41062013000600009.

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Sangster, C. R., C. K. Stevenson, B. A. Kidney, D. L. Montgomery, and A. L. Allen. "Kernicterus in an Adult Dog." Veterinary Pathology 44, no. 3 (2007): 383–85. http://dx.doi.org/10.1354/vp.44-3-383.

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Martin, Gilbert I. "Editor's Perspective: Jaundice, Bilirubin, Kernicterus." Journal of Perinatology 21, S1 (2001): S2. http://dx.doi.org/10.1038/sj.jp.7210623.

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Garone, Giacomo, Federica Graziola, Federico Vigevano, and Alessandro Capuano. "Vertical Gaze Palsy in Kernicterus." Neuropediatrics 50, no. 04 (2019): 262–63. http://dx.doi.org/10.1055/s-0039-1685527.

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Schyve, Paul M. "Editorial: Microsystems, Macrosystems, and Kernicterus." Joint Commission Journal on Quality and Safety 30, no. 11 (2004): 591–92. http://dx.doi.org/10.1016/s1549-3741(04)30069-9.

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Loynachan, Alan T., N. M. Williams, and J. F. Freestone. "Kernicterus in a Neonatal Foal." Journal of Veterinary Diagnostic Investigation 19, no. 2 (2007): 209–12. http://dx.doi.org/10.1177/104063870701900215.

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Nakamura, Hajime. "Assessing the risk of kernicterus." Indian Journal of Pediatrics 54, no. 5 (1987): 625–31. http://dx.doi.org/10.1007/bf02751269.

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Sonne, Luciana, Djeison Lutier Raymundo, Bianca Santana De Cecco, et al. "Kernicterus in an Adult Dog." Acta Scientiae Veterinariae 46 (January 9, 2018): 3. http://dx.doi.org/10.22456/1679-9216.85116.

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Background: Kernicterus or bilirubin encephalopathy is a condition rarely observed in animal characterized by a yellowish discoloration of the central nervous system. It is a potentially fatal condition due to bilirubin neurotoxic effects caused by the increase of non-conjugated bilirubin pigment, which passes blood brain barrier and has been attributed to an imbalance between albumin and bilirubin levels. Intracellular bilirubin is toxic for cells and can cause decrease in protein synthesis, specially albumin, depression of cell respiration and cellular death. This paper describes kernicterus

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Campos, Francielle Santana, and Izailza Matos Dantas Lopes. "Kernicterus: relato de caso clínico." Brazilian Journal of Health Review 6, no. 2 (2023): 5396–406. http://dx.doi.org/10.34119/bjhrv6n2-073.

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Introdução: Kernicterus é uma enfermidade crônica, irreversível resultante da toxicidade ocasionada pelo acúmulo de elevada quantidade de bilirrubina indireta no sistema nervoso central, em especial, nos núcleos da base e do tronco cerebral. As principais complicações são atetose, paralisia cerebral distônica, surdez e disfunção intelectual. O presente relato de caso teve como objetivo descrever um caso de Kernicterus em maternidade filantrópica no nordeste do Brasil que teve seguimento no ambulatório de especialidades da referida maternidade. Nasceu a termo, sexo masculino, com boa vitalidade

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Watchko, Jon F., and Diana Claassen. "Kernicterus in Premature Infants: Current Prevalence and Relationship to NICHD Phototherapy Study Exchange Criteria." Pediatrics 93, no. 6 (1994): 996–99. http://dx.doi.org/10.1542/peds.93.6.996.

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Objective. This study was undertaken to determine the current prevalence of kernicterus in premature neonates and to relate the occurrence of kernicterus to 1) the categorization of the infant as "at risk" by National Institute of Child Health and Human Development (NICHD) Phototherapy Study exchange transfusion criteria, and 2) the clinical management of the infant's hyperbilirubinemia. Design. Retrospective review of postmortem and clinical records. Setting. Magee-Womens Hospital, a University of Pittsburgh Medical Center affiliated hospital with approximately 10 000 deliveries per year and

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Yang, Fu-Chen, Jay L. Vivian, Catherine Traxler, Steven M. Shapiro, and John A. Stanford. "MGE-Like Neural Progenitor Cell Survival and Expression of Parvalbumin and Proenkephalin in a Jaundiced Rat Model of Kernicterus." Cell Transplantation 31 (January 2022): 096368972211011. http://dx.doi.org/10.1177/09636897221101116.

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Kernicterus is a permanent condition caused by brain damage from bilirubin toxicity. Dystonia is one of the most debilitating symptoms of kernicterus and results from damage to the globus pallidus (GP). One potential therapeutic strategy to treat dystonia in kernicterus is to replace lost GP neurons and restore basal ganglia circuits through stem cell transplantation. Toward this end, we differentiated human embryonic stem cells (hESCs) into medial ganglion eminence (MGE; the embryological origin of most of the GP neurons)-like neural precursor cells (NPCs). We determined neurochemical phenoty

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47

Tsao, Pei-Chen, Hsin-Ling Yeh, Yen-Chen Chang, et al. "Outcomes of neonatal jaundice in Taiwan." Archives of Disease in Childhood 103, no. 10 (2018): 927–29. http://dx.doi.org/10.1136/archdischild-2017-314063.

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ObjectiveTo investigate the burden of clinically significant neonatal jaundice (SNJ) in Taiwan, 2000–2010.Study designThe nationwide, population-based health insurance database in Taiwan was used to investigate the incidence, kernicterus rate and mortality rates of SNJ cohort born between 2000 and 2010.ResultsFrom 2000 to 2010, up to 242 546 patients admitted with neonatal jaundice (NJ) were identified. The incidence of SNJ was 5.9% in 2000 and increased to 13.7% in 2010 (P<0.001). The mortality rate significantly decreased from 0.51% in 2000 to 0.26% in 2010 (P<0.001) and the average in

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48

Resaz, Martina, Alessia Pepe, Domenico Tortora, Andrea Rossi, Luca Antonio Ramenghi, and Andrea Calandrino. "The Importance of Neuroimaging Follow-Up in Bilirubin-Induced Encephalopathy: A Clinical Case Review." Brain Sciences 15, no. 6 (2025): 539. https://doi.org/10.3390/brainsci15060539.

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Introduction: Hyperbilirubinemia in newborns can lead to kernicterus, a severe form of neonatal encephalopathy caused by bilirubin toxicity. Despite timely interventions such as exchange transfusion, kernicterus can still develop, especially in high-risk infants. MRI is crucial for detecting early and evolving signs of bilirubin-induced brain damage. Case Report: We report a term newborn who developed severe hyperbilirubinemia and kernicterus despite receiving exchange transfusion. The infant presented on day 3 of life with jaundice, hypotonia, and feeding difficulties and had a bilirubin leve

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Ip, S. "Hyperbilirubinemia and Kernicterus: 50 Years Later." PEDIATRICS 114, no. 1 (2004): 263–64. http://dx.doi.org/10.1542/peds.114.1.263.

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Penn, Anna A., Dieter R. Enzmann, Jin S. Hahn, and David K. Stevenson. "Kernicterus in a Full Term Infant." Pediatrics 93, no. 6 (1994): 1003–6. http://dx.doi.org/10.1542/peds.93.6.1003.

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Neonatal jaundice can represent a benign physiologic process or be the harbinger of serious illness with associated severe neurotoxicity. The neurological manifestations of kernicterus, a condition resulting from the deposition of unconjugated bilirubin in the central nervous system, are rarely seen in modern neonatal care, but jaundice, which reflects elevated serum bilirubin levels, is one of the most common findings in the neonatal period.1 More than half of all term infants will develop some neonatal jaundice and at least 6% will have a serum bilirubin concentration above 12.9 mg/dL.2 The

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