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Littérature scientifique sur le sujet « Neurochemical networks »

Auteur : Grafiati
Publié le 29 mars 2025

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Articles de revues sur le sujet "Neurochemical networks"

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Amirah, Farah. "The neurochemical underpinnings of autism spectrum disorder." International Journal of Clinical Medical Research 3, no. 1 (2025): 16–17. https://doi.org/10.61466/ijcmr3010004.

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Autism spectrum disorder encompasses a range of neurobehavioral and neurodevelopmental conditions marked by deficits in social interaction and communication, as well as restricted and repetitive behaviors or interests, alongside atypical sensory processing. Environmental, immunological, genetic, and epigenetic factors contribute to the pathophysiology of autism, triggering neuroanatomical and neurochemical changes early in central nervous system development. Numerous neurochemical pathways contribute to the etiology of autism spectrum disorder; however, the interactions among these intricate n
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Khani, Abbas, and Gregor Rainer. "Neural and neurochemical basis of reinforcement-guided decision making." Journal of Neurophysiology 116, no. 2 (2016): 724–41. http://dx.doi.org/10.1152/jn.01113.2015.

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Decision making is an adaptive behavior that takes into account several internal and external input variables and leads to the choice of a course of action over other available and often competing alternatives. While it has been studied in diverse fields ranging from mathematics, economics, ecology, and ethology to psychology and neuroscience, recent cross talk among perspectives from different fields has yielded novel descriptions of decision processes. Reinforcement-guided decision making models are based on economic and reinforcement learning theories, and their focus is on the maximization
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Marotta, Rosa, Maria C. Risoleo, Giovanni Messina, et al. "The Neurochemistry of Autism." Brain Sciences 10, no. 3 (2020): 163. http://dx.doi.org/10.3390/brainsci10030163.

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Autism spectrum disorder (ASD) refers to complex neurobehavioral and neurodevelopmental conditions characterized by impaired social interaction and communication, restricted and repetitive patterns of behavior or interests, and altered sensory processing. Environmental, immunological, genetic, and epigenetic factors are implicated in the pathophysiology of autism and provoke the occurrence of neuroanatomical and neurochemical events relatively early in the development of the central nervous system. Many neurochemical pathways are involved in determining ASD; however, how these complex networks
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Jiménez-Jiménez, Félix, Hortensia Alonso-Navarro, Elena García-Martín, and José Agúndez. "Neurochemical Features of Rem Sleep Behaviour Disorder." Journal of Personalized Medicine 11, no. 9 (2021): 880. http://dx.doi.org/10.3390/jpm11090880.

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Dopaminergic deficiency, shown by many studies using functional neuroimaging with Single Photon Emission Computerized Tomography (SPECT) and Positron Emission Tomography (PET), is the most consistent neurochemical feature of rapid eye movement (REM) sleep behaviour disorder (RBD) and, together with transcranial ultrasonography, and determination of alpha-synuclein in certain tissues, should be considered as a reliable marker for the phenoconversion of idiopathic RBD (iRBD) to a synucleopathy (Parkinson’s disease –PD- or Lewy body dementia -LBD). The possible role in the pathogenesis of RBD of
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Schwarz, Adam J., Alessandro Gozzi, Alessandro Chessa, and Angelo Bifone. "Voxel Scale Complex Networks of Functional Connectivity in the Rat Brain: Neurochemical State Dependence of Global and Local Topological Properties." Computational and Mathematical Methods in Medicine 2012 (2012): 1–15. http://dx.doi.org/10.1155/2012/615709.

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Network analysis of functional imaging data reveals emergent features of the brain as a function of its topological properties. However, the brain is not a homogeneous network, and the dependence of functional connectivity parameters on neuroanatomical substrate and parcellation scale is a key issue. Moreover, the extent to which these topological properties depend on underlying neurochemical changes remains unclear. In the present study, we investigated both global statistical properties and the local, voxel-scale distribution of connectivity parameters of the rat brain. Different neurotransm
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Frisaldi, Elisa, Alessandro Piedimonte, and Fabrizio Benedetti. "Placebo and Nocebo Effects: A Complex Interplay Between Psychological Factors and Neurochemical Networks." American Journal of Clinical Hypnosis 57, no. 3 (2015): 267–84. http://dx.doi.org/10.1080/00029157.2014.976785.

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Brodowicz, Justyna, Edmund Przegaliński, Christian P. Müller, and Malgorzata Filip. "Ceramide and Its Related Neurochemical Networks as Targets for Some Brain Disorder Therapies." Neurotoxicity Research 33, no. 2 (2017): 474–84. http://dx.doi.org/10.1007/s12640-017-9798-6.

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Samardžija, Bobana, Milan Petrović, Beti Zaharija, et al. "Transgenic Drosophila melanogaster Carrying a Human Full-Length DISC1 Construct (UAS-hflDISC1) Showing Effects on Social Interaction Networks." Current Issues in Molecular Biology 46, no. 8 (2024): 8526–49. http://dx.doi.org/10.3390/cimb46080502.

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Disrupted in Schizophrenia 1 (DISC1) is a scaffold protein implicated in major mental illnesses including schizophrenia, with a significant negative impact on social life. To investigate if DISC1 affects social interactions in Drosophila melanogaster, we created transgenic flies with second or third chromosome insertions of the human full-length DISC1 (hflDISC1) gene fused to a UAS promotor (UAS-hflDISC1). Initial characterization of the insertion lines showed unexpected endogenous expression of the DISC1 protein that led to various behavioral and neurochemical phenotypes. Social interaction n
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Ziminski, Joseph J., Polytimi Frangou, Vasilis M. Karlaftis, Uzay Emir, and Zoe Kourtzi. "Microstructural and neurochemical plasticity mechanisms interact to enhance human perceptual decision-making." PLOS Biology 21, no. 3 (2023): e3002029. http://dx.doi.org/10.1371/journal.pbio.3002029.

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Experience and training are known to boost our skills and mold the brain’s organization and function. Yet, structural plasticity and functional neurotransmission are typically studied at different scales (large-scale networks, local circuits), limiting our understanding of the adaptive interactions that support learning of complex cognitive skills in the adult brain. Here, we employ multimodal brain imaging to investigate the link between microstructural (myelination) and neurochemical (GABAergic) plasticity for decision-making. We test (in males, due to potential confounding menstrual cycle e
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De Pascalis, Vilfredo. "Brain Functional Correlates of Resting Hypnosis and Hypnotizability: A Review." Brain Sciences 14, no. 2 (2024): 115. http://dx.doi.org/10.3390/brainsci14020115.

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This comprehensive review delves into the cognitive neuroscience of hypnosis and variations in hypnotizability by examining research employing functional magnetic resonance imaging (fMRI), positron emission tomography (PET), and electroencephalography (EEG) methods. Key focus areas include functional brain imaging correlations in hypnosis, EEG band oscillations as indicators of hypnotic states, alterations in EEG functional connectivity during hypnosis and wakefulness, drawing critical conclusions, and suggesting future research directions. The reviewed functional connectivity findings support
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Thèses sur le sujet "Neurochemical networks"

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Butler, Jasmine J. "Action of 5-HT2A receptors on neurotransmitter systems in the mouse brain : application to psychedelics." Electronic Thesis or Diss., Bordeaux, 2025. http://www.theses.fr/2025BORD0026.

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Le sous-type de récepteur de la sérotonine 2A (5-HT2AR) suscite un grand intérêt suite aux recherches clinique et préclinique sur les psychédéliques sérotoninergiques. Ces composés ont une action agoniste convergente sur le 5-HT2AR et ont des propriétés antidépressives et anxiolytiques. Cet intérêt s’ajoute à des données antérieures montrant que l'antagonisme du récepteur 5-HT2AR, qui fait partie du profil pharmacologique des antipsychotiques atypiques, pourrait avoir des effets bénéfiques dans plusieurs pathologies dont la schizophrénie. Néanmoins, l’impact des 5-HT2AR sur le fonctionnement c
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Livres sur le sujet "Neurochemical networks"

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Ashraf, Ghulam. Neurochemical Systems and Signaling: From Molecules to Networks. Taylor & Francis Group, 2023.

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Neurochemical Systems and Signaling: From Molecules to Networks. CRC Press LLC, 2023.

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Ashraf, Ghulam. Neurochemical Systems and Signaling: From Molecules to Networks. Taylor & Francis Group, 2023.

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Ashraf, Ghulam. Neurochemical Systems and Signaling: From Molecules to Networks. Taylor & Francis Group, 2023.

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Ashraf, Ghulam. Neurochemical Systems and Signaling: From Molecules to Networks. Taylor & Francis Group, 2023.

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Ashraf, Ghulam. Neurochemical Systems and Signaling: From Molecules to Networks. Taylor & Francis Group, 2023.

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Carrión, Victor G., John A. Turner, and Carl F. Weems. Sleep. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190201968.003.0005.

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The architecture of healthy sleep rests upon a network of several interacting neurochemical systems, an arrangement that is easily disrupted by the experience of traumatic stress. As a result, sleep may be among the most susceptible of behaviors to have a negative impact as a result of trauma. Sleep disturbances, or “parasomnias,” such as nightmares, sleepwalking, and insomnia are one of the most prominent hallmarks of PTSD, and the study of these sleep-specific symptoms can provide a window into the underlying pathology of the disorder. The current chapter reviews the preclinical animal liter
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Chapitres de livres sur le sujet "Neurochemical networks"

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Ezhov, Alexandr A., Andrei G. Khromov, and Svetlana S. Terentyeva. "On Neurochemical Aspects of Agent-Based Memory Model." In Advances in Neural Networks – ISNN 2016. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40663-3_43.

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KELLEY, ANN E. "Neurochemical Networks Encoding Emotion and Motivation." In Who Needs Emotions? Oxford University Press, 2005. http://dx.doi.org/10.1093/acprof:oso/9780195166194.003.0003.

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Devinsky, Orrin, and Mark D’esposito. "Memory and Memory Disorders." In Neurology Of Cognitive And Behavioral Disorders. Oxford University PressNew York, NY, 2003. http://dx.doi.org/10.1093/oso/9780195137644.003.0008.

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Abstract Understanding of the neural basis of memory function has advanced rapidly during the past few decades. Numerous neuropsychological and neurophysiological studies of memory function reveal a system of dissociable processes, not a unitary system. Anatomical studies in humans and other primates map these dissociable processes onto distributed neural networks. Neuro pharmacological studies reveal that different neurotransmitter systems play distinct roles in the various memory processes. Memory is not localized to one brain region or restricted to one neurotransmitter system. The theoretical distinctions of memory functions developed by psychologists and neuroscientists provide a meaningful framework for understanding the symptoms of memory disorders. New therapies will likely arise from advances in understanding the neuroanatomical and neurochemical underpinnings of memory function.
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Smith, Yoland, and J. P. Bolam. "Combined approaches to experimental neuroanatomy: combined tracing and immunocytochemical techniques for the study of neuronal microcircuits." In Experimental Neuroanatomy. Oxford University PressOxford, 1992. http://dx.doi.org/10.1093/oso/9780199633265.003.0011.

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Abstract Most of the chapters in this volume have so far dealt with individual techniques in experimental neuroanatomy at the electron microscopic level. From these techniques we can identify projection neurones and examine the morphology of their afferent terminals. Moreover, the structural characteristics of the post-synaptic target of a set of anterogradely labelled terminals can be analysed from material containing immunostained structures, or intracellularly labelled neurones. These approaches in themselves produce valuable information concerning neuronal elements within the microcircuits and networks of the brain. However, the amount of data obtained using such approaches is limited in the sense that the identified structure is examined ‘in isolation’. In order to establish the position of an identified neurone, or population of terminals within the neural network in relation to other neuronal elements, it is necessary to know the nature of the terminals afferent to a labelled neurone, or the nature of the post-synaptic targets of identified terminals. By this we mean that it is necessary to know the origin, neurochemical nature, and pattern of innervation of the synaptic terminals afferent to an identified neurone. Similarly, it is important to know the morphology, the chemical nature and the synaptic output of the neurones that are post-synaptic to a specific population of labelled terminals. Thus, to establish the microcircuitry or neuronal networks of a particular area of the brain it is necessary to combine the individual experimental approaches in a single experimental animal.
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Alexander, Rebecca, and Justine Megan Gatt. "Resilience." In Genes, brain, and emotions. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198793014.003.0020.

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Resilience refers to the process of adaptive recovery following adversity or trauma. It is likely to include an intertwined series of dynamic interactions between neural, developmental, environmental, genetic, and epigenetic factors over time. Neuroscientific research suggests the potential role of the brain’s threat and reward systems, as well as executive control networks. Developmental research provides insight into how the environment may affect these neural systems across the lifespan towards greater risk or resilience to stress. Genetic work has revealed numerous targets that alter key neurochemical systems in the brain to influence mental health. Current challenges include ambiguities in the definition and measurement of resilience and a simplified focus on resilience as the absence of psychopathology, irrespective of levels of positive mental functioning. Greater emphasis on understanding the protective aspects of resilience and related well-being outcomes are important to delineate the unique neurobiological factors that underpin this process, so that effective interventions can be developed to assist vulnerable populations and resilience promotion.
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Pradhan, Basant. "Social Psychiatry." In The WASP Textbook on Social Psychiatry, edited by Rama Rao Gogineni, Andres J. Pumariega, Roy A. Kallivayalil, Marianne Kastrup, and Eugenio M. Rothe. Oxford University PressNew York, 2023. http://dx.doi.org/10.1093/med/9780197521359.003.0022.

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Abstract Social cognitive neuroscience is an emerging discipline that bridges across several disciplines and includes the concepts of social brain and social psychiatry. Simply speaking, social brain is the biological substrate of what social psychiatry entails. Our sociality is the outcome of an intricate and multi-dimensional relationship between the size of the social brain, the richness as well as the selectivity of its neural networks and the behavioral complexities emanating from the bonded relationships that underpin our social coalitions. Although no specific brain area is specifically designated as the social brain, accumulating data from the neuroimaging and cognitive neuroscience research reveal that distinct neuronal networks do subserve our social functionings and also that the social information processing may actually be different non-social information processing. Our various social functioning and the related processes serve as the operational domains for both the social brain as well as social psychiatry. Their scopes are quite broad and range from epidemiological/anthropological research and an indistinct boundary with individual or group psychotherapy at one end, and quite precise brain mapping, genetic, neurochemical studies and targeted neuromodulation and integrative psychiatric interventions at the other end. This chapter embarks upon these fascinating aspects from social cognitive neuroscience, biological, developmental and spiritual perspectives and also discusses some therapeutic implications, both actual and potential.
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Yue, Guangxin. "Regulation of Oxytocin on Empathy and Its Neural Mechanism." In Oxytocin and Social Function [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.112743.

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Empathy is a multidimensional concept, including emotion and cognition. It plays a vital role in social communication, and it is very important for establishing harmonious relationships, trust, and mutual understanding. Empathy includes the ability to feel and understand the emotions of others, which can be learned and improved through various ways. Oxytocin is a neuropeptide, and its influence on social behavior and emotions has been widely studied. It is found that it can enhance emotional and cognitive empathy, as well as trust and cooperative behavior. Oxytocin acts on specific brain regions, such as the insula, amygdala, and reward circuitry, to modulate empathy-related neural processes. Oxytocin receptor gene polymorphisms are also related to empathy. Future research could explore the effect of oxytocin interventions on individuals with empathy deficiency, investigate the relationship between oxytocin receptor gene polymorphism and empathy neural networks, and study the neural mechanisms of the influence of other neurochemical substances (such as dopamine) affecting empathy. In addition, further study on empathy of typical developing individuals could provide valuable insights into the symptoms and causes of various diseases. Finally, promoting the practical application and value transformation of research results related to empathy is helpful to develop intelligent systems that can simulate human empathy and enhance human-computer interactions.
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Brommelhoff Jessica A. and Sultzer David L. "Brain Structure and Function Related to Depression in Alzheimer's Disease: Contributions from Neuroimaging Research." In Advances in Alzheimer’s Disease. IOS Press, 2015. https://doi.org/10.3233/978-1-61499-542-5-295.

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The development of minimally invasive in vivo methods for imaging the brain has allowed for unprecedented advancement in our understanding of brain-behavior relationships. Structural, functional, and multimodal neuroimaging techniques have become more sophisticated in detecting structural and physiological abnormalities that may underlie various affective disorders and neurological illnesses such as depression in Alzheimer's disease (AD). In general, neuroimaging studies of depression in AD investigate whether depression is associated with damage to structures in specific neural networks involving frontal and subcortical structures or with functional disruption of cortical neural systems. This review provides an overview of how various imaging modalities have contributed to our understanding of the neurobiology of depression in AD. At present, the literature does not conclusively support any specific pathogenesis for depression, and it is not clear whether patients with AD and depression have histopathological and neurochemical characteristics that contribute to mood symptoms that are different from cognitively intact individuals with depression. Neuroimaging studies suggest that atrophy of temporal or frontal structures, white matter lesions in frontal lobe or subcortical systems, reduced activity in dorsolateral frontal cortex, or small vessel cerebrovascular disease may be associated with depression in AD. Conceptual, clinical, and methodological challenges in studying this relationship are discussed. Further work is needed to understand the specific brain structures, relevant white matter tracts, and interactions among them that are most important. This review concludes with potential directions for future research.
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HENDRY, S. H. C. "A Neurochemically Distinct Third Channel in the Macaque Lateral Geniculate Nucleus." In Thalamic Networks for Relay and Modulation. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-08-042274-9.50027-4.

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Actes de conférences sur le sujet "Neurochemical networks"

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Roever, Paul, Khalid B. Mirza, Konstantin Nikolic, and Christofer Toumazou. "A Convolutional Neural Network for Classification of Nerve Activity Based on Action Potential Induced Neurochemical Signatures." In 2020 IEEE International Symposium on Circuits and Systems (ISCAS). IEEE, 2020. http://dx.doi.org/10.1109/iscas45731.2020.9180734.

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