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Littérature scientifique sur le sujet « SALT II »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 25 avril 2022

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Articles de revues sur le sujet "SALT II"

HULETT, LOUISA S. « Carter, Salt II and Detente II ». Australian Journal of Politics & ; History 28, n^o 2 (7 avril 2008) : 190–200. http://dx.doi.org/10.1111/j.1467-8497.1982.tb00177.x.

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Sartori, Leo. « Will SALT II Survive ? » International Security 10, n^o 3 (1985) : 147. http://dx.doi.org/10.2307/2538945.

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Earle, Ralph. « Don't abandon SALT II ». Bulletin of the Atomic Scientists 42, n^o 7 (août 1986) : 8–9. http://dx.doi.org/10.1080/00963402.1986.11459397.

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Mancini, Matthew. « Elvira Santamaria, Salt Cartographies II ». Public 30, n^o 59 (1 juin 2019) : 204–6. http://dx.doi.org/10.1386/public.30.59.204_5.

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Mancini, Matthew. « Elvira Santamaria, Salt Cartographies II ». Public 31, n^o 59 (1 juin 2019) : 204–6. http://dx.doi.org/10.1386/public.31.59.204_5.

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Rudd, M. Audrey, Maria Trolliet, Susan Hope, Anne Ward Scribner, Geraldine Daumerie, George Toolan, Timothy Cloutier et Joseph Loscalzo. « Salt-induced hypertension in Dahl salt-resistant and salt-sensitive rats with NOS II inhibition ». American Journal of Physiology-Heart and Circulatory Physiology 277, n^o 2 (1 août 1999) : H732—H739. http://dx.doi.org/10.1152/ajpheart.1999.277.2.h732.

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Although recent evidence suggests that reduced nitric oxide (NO) production may be involved in salt-induced hypertension, the specific NO synthase (NOS) responsible for the conveyance of salt sensitivity remains unknown. To determine the role of inducible NOS (NOS II) in salt-induced hypertension, we treated Dahl salt-resistant (DR) rats with the selective NOS II inhibitor 2-amino-5,6-dihydro-6-methyl-4H-1,3-thiazine (AMT) for 12 days. Tail-cuff systolic blood pressures rose 29 ± 6 and 42 ± 8 mmHg in DR rats given 150 and 300 nmol AMT/h, respectively ( P < 0.01, 2-way ANOVA) after 7 days of 8% NaCl diet. We observed similar results with two other potent selective NOS II inhibitors, S-ethylisourea (EIT) and N-[3-(aminomethyl)benzyl]acetamidine hydrochloride (1400W). Additionally, AMT effects were independent of alterations in endothelial function as assessed by diameter change of mesenteric arterioles in response to methacholine using videomicroscopy. We, therefore, conclude from these data that NOS II is important in salt-induced hypertension.

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Nugraha, Yuda Prasetya, Haruki Sugiyama et Hidehiro Uekusa. « Ciprofloxacin salt and salt co-crystal with dihydroxybenzoic acids ». Acta Crystallographica Section E Crystallographic Communications 78, n^o 3 (3 février 2022) : 259–63. http://dx.doi.org/10.1107/s2056989022001177.

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The crystal structure of two multi-component crystals of ciprofloxacin [systematic name: 1-cyclopropyl-6-fluoro-4-oxo-7-(piperazin-1-yl)quinoline-3-carboxylic acid], a fluoroquinolone antibiotic, namely, ciprofloxacin 2,6-dihydroxybenzoate salt, C17H19FN3O3 +·C7H5O4 −, (I), and ciprofloxacin hydrochloride–3,5-dihydroxybenzoic–water (1/1/1), C17H19FN3O3 +·Cl−·C7H6O4·H2O, (II), were determined. In (I) and (II), the ciprofloxacin cations are connected via head-to-tail N—H...O hydrogen bonding. Both structures show an alternating layered arrangement between ciprofloxacin and dihydroxybenzoic acid.

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Ando, K., Y. Sato et T. Fujita. « Salt sensitivity in hypertensive rats with angiotensin II administration ». American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 259, n^o 5 (1 novembre 1990) : R1012—R1016. http://dx.doi.org/10.1152/ajpregu.1990.259.5.r1012.

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We examined the salt sensitivity of blood pressure in angiotensin II (ANG II)-induced hypertension. Wistar rats, salt loaded (0.66, 2, or 8% salt-containing diet) for 4 or 12 days, were infused intravenously with 15 or 60 ng/min of ANG II. Systolic blood pressure (SBP) was not increased by long-term (12 days) salt loading, and SBP was unchanged with ANG II and normal-salt (0.66%) diet. However, when combined with salt loading, ANG II produced hypertension in a dose-dependent fashion; compared with control (120 +/- 2 mmHg), SBP was increased with 15 ng/min of ANG II and 8% salt diet (145 +/- 5 mmHg, P less than 0.05) and with 60 ng/min of ANG II and either 2 or 8% salt diet (149 +/- 8 and 174 +/- 8 mmHg, P less than 0.05, respectively). Na space (exchangeable Na) was increased in a roughly similar pattern and correlated significantly (r = 0.531, P less than 0.05) with SBP. However, with 15 ng/min of ANG II, Na space was not different among rats on either level of salt loading, although the 8% salt diet elevated SBP. Data obtained with short-term (4 days) treatment indicate that an elevated Na space preceded development of hypertension. With 15 ng/min of ANG II and 8% salt diet for 4 days, Na space was markedly (P less than 0.05) increased, but SBP was unchanged.(ABSTRACT TRUNCATED AT 250 WORDS)

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Sasser, Jennifer M., Jennifer S. Pollock et David M. Pollock. « Renal endothelin in chronic angiotensin II hypertension ». American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 283, n^o 1 (1 juillet 2002) : R243—R248. http://dx.doi.org/10.1152/ajpregu.00086.2002.

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To determine the influence of chronic ANG II infusion on urinary, plasma, and renal tissue levels of immunoreactive endothelin (ET), ANG II (65 ng/min) or saline vehicle was delivered via osmotic minipump in male Sprague-Dawley rats given either a high-salt diet (10% NaCl) or normal-salt diet (0.8% NaCl). High-salt diet alone caused a slight but not statistically significant increase (7 ± 1%) in mean arterial pressure (MAP). MAP was significantly increased in ANG II-infused rats (41 ± 10%), and the increase in MAP was significantly greater in ANG II rats given a high-salt diet (59 ± 1%) compared with the increase observed in rats given a high-salt diet alone or ANG II infusion and normal-salt diet. After a 2-wk treatment, urinary excretion of immunoreactive ET was significantly increased by ∼50% in ANG II-infused animals and by over 250% in rats on high-salt diet, with or without ANG II infusion. ANG II infusion combined with high-salt diet significantly increased immunoreactive ET content in the cortex and outer medulla, but this effect was not observed in other groups. In contrast, high-salt diet, with or without ANG II infusion, significantly decreased immunoreactive ET content within the inner medulla. These data indicate that chronic elevations in ANG II levels and sodium intake differentially affect ET levels within the kidney and provide further support for the hypothesis that the hypertensive effects of ANG II may be due to interaction with the renal ET system.

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SMITH, R. J. « U.S. Official Defends SALT II Decision ». Science 232, n^o 4756 (13 juin 1986) : 1334. http://dx.doi.org/10.1126/science.232.4756.1334.

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Thèses sur le sujet "SALT II"

Lu, Jiao. « Central Mechanisms Mediating Ang II-Salt Hypertension ». Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/34811.

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Abstract Statement of problem Plasma angiotensin II (Ang II) increases blood pressure (BP) through the activation of brain angiotensinergic pathways and the aldosterone-mineralocorticoid receptors (MR)- epithelial Na+ channel (ENaC)-endogenous ouabain (EO) pathway. The response of BP to circulating Ang II is enhanced by high salt intake, but the central mechanisms mediating this elevated response are not known. Methods of investigation Study 1) Male Wistar rats were divided into 4 groups and treated with regular salt diet (0.4% NaCl), high salt diet (2% NaCl), sc Ang II infusion (150 ng/kg/min), or sc Ang II infusion together with 2% salt diet for 14 days; plasma aldosterone and corticosterone levels, CYP11B2 mRNA in adrenal cortex and the mRNA levels of Ang II type 1 receptors (AT1R), CYP11B1 (11-β hydroxylase), CYP11B2 (aldosterone synthase), MR, 11βHSD2, ENaC α, ENaC β and ENaC γ in the subfornical organ (SFO), paraventricular nucleus (PVN), supraoptic nucleus (SON) and rostral ventrolateral medulla (RVLM) were measured. Study 2) MR blockers (eplerenone, spironolactone), ENaC blocker (benzamil), AT1R blocker (losartan) or vehicles were centrally infused in rats treated with Ang II plus high salt, and BP and heart rate (HR) were recorded by telemetry; plasma aldosterone and corticosterone levels and CYP11B2 mRNA expression in adrenal cortex were measured. Major findings Ang II alone caused a small increase in BP. Ang II together with 2% salt diet markedly increased the BP and plasma aldosterone level. Sc Ang II decreased 11βHSD2 and MR mRNA expression in the PVN, increased AT1R and ENaC γ expression in the PVN, and increased AT1R mRNA expression in the RVLM. Other genes tested in the four brain nuclei were not affected by sc Ang II or high salt diet. BP and plasma aldosterone increases in response to Ang II and salt, as well as CYP11B2 mRNA expression in adrenal cortex, were largely prevented by central infusion of eplerenone, spironolactone, benzamil or losartan. Main conclusion BP and plasma aldosterone responses to Ang II-salt are under the control of central mechanisms, and MR-AT1R activation in the brain plays a critical role in Ang II-salt induced hypertension.

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King, Andrew J. « Sympathetic mechanisms of salt-sensitive hypertension ». Diss., Connect to online resource - MSU authorized users, 2008.

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Thesis (Ph.D.)--Michigan State University. Dept. of Pharmacology and Toxicology, 2008.
Title from PDF t.p. (viewed on Mar. 30, 2009) Includes bibliographical references (p.177-201). Also issued in print.

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Veerasingham, Shereeni J. « Salt-induced hypertension, central regulation by ouabain-like compounds and angiotensin II ». Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ58297.pdf.

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Zenteno, Savin Tania. « Plasma arginine vasotocin and angiotensin II concentrations during saline acclimation in birds with salt glands ». Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/29892.

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Pekin ducks (Anas platyrhynchos), Canada geese (Branta canadensis) and Glaucous-winged gulls (Larus glaucescens) responded differently during acclimation to saline equivalent to full strength sea water. All species were in hydrosmotic balance until they were given 375 mM NaCl. When the birds drank 75 mM NaCl ducks transiently decreased plasma sodium concentration ([Na⁺][formula omitted]) and osmolality (Osm[formula omitted]) and increased cloacal Na⁺ excretion; geese transiently increased hematocrit (Hct), suggesting transient extracellular dehydration; and gulls increased Osm[formula omitted] (but not Hct), suggesting that their Osm[formula omitted] had been below normal when fresh water was offered. During acclimation to moderately concentrated saline, all species progressively increased plasma arginine vasotocin levels ([AVT][formula omitted]) (without concomitant increase in Osm[formula omitted]) and decreased plasma angiotensin II concentration ([Ang II][formula omitted]) without parallel changes in Hct. Following acclimation to 300 mM NaCl total body water (TBW) was increased in ducks and geese, but acclimation to 375 mM NaCl did not affect TBW in gulls. When they drank 450 mM NaCl, ducks and geese decreased body mass and increased Hct, plasma electrolyte and osmotic concentrations, [AVT][formula omitted] and [Ang II][formula omitted], indicating they were dehydrated. In contrast, saline acclimated gulls did not increase [Ang II][formula omitted] and increased [AVT][formula omitted] less than ducks or geese. Initial [AVT][formula omitted] was not significantly correlated with sex in either ducks or geese. Female ducks increased [Ang II[formula omitted] and Osm[formula omitted] less than males during exposure to 450 mM NaCl, while female geese increased [Ang II][formula omitted] more than males. Salt gland NaCl secretion and renal water retention counterbalanced NaCl ingested in low to moderately concentrated saline in ducks, geese and gulls, but not in ducks and geese drinking saline equivalent to sea water. Gulls likely maintained simultaneous, concerted function of kidneys and salt glands during high salt intake, while cloacal excretion may have decreased in the Anatidae. Based on their relative salt secreting efficiencies, plasma ionic concentrations, Osm[formula omitted] and Hct increased much more in ducks than in geese when they drank 450 mM NaCl, and remained unchanged in gulls drinking 375 mM NaCl. Release of AVT and Ang II in birds with salt glands appears to be controlled by a complicated interrelationship between volume and tonicity (threshold for release varies among species), and these (and possibly other) hormones may affect salt gland and kidney function to maintain salt and water balance.
Science, Faculty of
Zoology, Department of
Graduate

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Huang, Chunhua. « Impact of dietary salt intake during growth on cardiovascular homeostasis and neural control of the kidney : role of brain angiotensin II (Ang II) ». Thesis, University of Birmingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368519.

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Stansfield, Ron E. (Ron Earl) Carleton University Dissertation International Affairs. « Taming the technological beast : the failure of Salt II to introduce stability into superpower strategic nuclear forces structures ». Ottawa, 1992.

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Nurkiewicz, Timothy Robert. « Effects of hypertension and dietary salt on myogenic activity in the microcirculation possible roles of nitric oxide and angiotensin II / ». Morgantown, W. Va. : [West Virginia University Libraries], 1999. http://etd.wvu.edu/templates/showETD.cfm?recnum=1004.

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Thesis (Ph. D.)--West Virginia University, 1999.
Title from document title page. Document formatted into pages; contains xvi, 200 p. : ill. (some col.). Vita. Includes abstract. Includes bibliographical references.

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Gabor, Alexander. « Role of Angiotensin II, Glutamate, Nitric Oxide and an Aldosterone-ouabain Pathway in the PVN in Salt-induced Pressor Responses in Rats ». Thèse, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/22900.

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High salt intake contributes to the development of hypertension in salt-sensitive humans and animals and the mechanistic causes are poorly understood. In Dahl salt-sensitive (S) but not salt-resistant (R) rats, high salt diet increases cerebrospinal fluid (CSF) [Na+] and activates an aldosterone-mineralocorticoid receptor-epithelial sodium channel-endogenous ouabain (MR-ENaC-EO) neuromodulatory pathway in the brain that enhances the activity of sympatho-excitatory angiotensinergic and glutamatergic pathways, leading to an increase in sympathetic nerve activity (SNA) and blood pressure (BP). We hypothesize that high salt diet in Dahl S rats enhances Ang II release in the paraventricular nucleus (PVN), causing a decrease in local nitric oxide (NO) action and an increase in local glutamate release thereby elevating SNA, BP and heart rate (HR). The present study evaluated the effects of agonists or blockers of MR, ENaC, EO, nitric oxide synthase (NOS) or glutamate and AT1-receptors on the BP and HR responses to acute infusions of Na+ rich aCSF, intracerebroventricularly (icv), or in the PVN of Dahl S, R or Wistar rats or to high salt diet in Dahl S and R rats. In Wistar rats, aldosterone in the PVN enhanced the BP and HR responses to infusion of Na+ rich aCSF in the PVN, but not in the CSF, and only the enhancement was prevented by blockers of MR, ENaC and EO in the PVN. AT1-receptor blockers in the PVN fully blocked the enhancement by aldosterone and the responses to infusion of Na+ rich aCSF icv, or in the PVN. Na+ rich aCSF in the PVN caused larger increases in BP and HR in Dahl S vs. R rats and the responses to Na+ were fully blocked by an AT1-receptor blocker in the PVN. BP and HR responses to a NOS blocker in the PVN were the same, but L-NAME enhanced Na+ effects more in Dahl R than S rats. High salt diet attenuated increases in BP from L-NAME in the PVN of Dahl S but not R rats. AT1 and glutamate receptor blockers candesartan and kynurenate in the PVN decreased BP in Dahl S but not R rats on high salt diet. At the peak BP response to candesartan, kynurenate in the PVN further decreased BP whereas candesartan did not further decrease BP at the peak BP response to kynurenate. Our findings indicate that both an acute increase in CSF [Na+] and high salt intake in Dahl S rats increases AT1-receptor activation and decreases NO action in the PVN thereby contributing to the pressor responses to Na+ and presumably, to dietary salt-induced hypertension. The increased BP response to AT1-receptor activation in the PVN of Dahl S is mediated by enhanced local glutamate receptor activation. An MR-ENaC-EO pathway in the PVN can be functionally active and further studies need to assess its role in Dahl S rats on high salt intake.

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Ambrose, Matthew John. « The Limits of Control : A History of the SALT Process, 1969-1983 ». The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1417687511.

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Zandevakili, Roham. « Effects of ANG II and its receptor blockers on nasal salt gland secretion and arterial blood pressure in conscious perkin ducks (Anas plalytrhynchos) ». Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq40893.pdf.

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Livres sur le sujet "SALT II"

Talbott, Strobe. Endgame : The inside story of SALT II. New York : Harper and Row, 1985.

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Kaplan, Liza, dir. Salt to the Sea. New York, USA : Penguin Books, 2017.

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A, Calvo Michel, dir. The SALT agreements : Content, application, verification. Dordrecht : Martinus Nijhoff, 1987.

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Diem, Wolfgang. Petrographie, Feinstratigraphie und Petrofazies des Stassfurt Steinsalzes (Zechstein 2) im Aufschlussbereich des Salzbergwerkes Asse II bei Braunschweig. Neuherberg : Gesellschaft für Strahlen- und Umweltforschung München, 1985.

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Caldwell, Dan. The dynamics of domestic politics and arms control : The SALT II Treaty ratification debate. Columbia, S.C : University of South Carolina Press, 1991.

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Bennett, Paul R. Russian negotiating strategy : Analytic case studies from SALT and START. Commack, NY : Nova Science Publishers, 1997.

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Bennett, Paul R. Russian negotiating strategy : Analytic case studies from SALT to START. Commack, NY : Nova Science Publishers, 1997.

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Huang, Chunhua. Impact of dietary salt intake during growth on cardiovascular homeostasis and neural control of the kidney : Role of brain angiotensin II (Ang II). Birmingham : University of Birmingham, 2001.

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Meiers, Franz-Josef. Raketenschach : Von SALT II zu INF : der innen- und aussenpolitische Kontext der amerikanischen Rüstungskontrollpolitik, 1977-1988. Zürich : Rüegger, 1991.

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United States. Congress. Senate. Committee on Foreign Relations. The Salt II Treaty debate : The Cold War congressional hearings over nuclear weapons and Soviet-American arms control. St. Petersburg, Fla : Red and Black Publishers, 2011.

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Chapitres de livres sur le sujet "SALT II"

Earle, Ralph. « SALT II Compliance Controversies ». Dans Verification and Compliance, 166–84. London : Palgrave Macmillan UK, 1988. http://dx.doi.org/10.1007/978-1-349-10143-6_9.

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Caldwell, Dan. « The SALT II Treaty ». Dans The Politics of Arms Control Treaty Ratification, 279–353. New York : Palgrave Macmillan US, 1991. http://dx.doi.org/10.1007/978-1-137-04534-8_7.

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Walker, John E., David M. Howell, Richard J. Thompson et Billy C. Archibld. « Dichlorobis(hydroxylamine)zinc(II) (Crismer's Salt) ». Dans Inorganic Syntheses, 2–3. Hoboken, NJ, USA : John Wiley & Sons, Inc., 2007. http://dx.doi.org/10.1002/9780470132401.ch2.

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Moens, Alexander. « SALT II : The ‘Deep Cuts’ Proposals ». Dans Foreign Policy Under Carter, 63–89. New York : Routledge, 2021. http://dx.doi.org/10.4324/9780429045592-4.

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Chai, Li, Jiancang Xie, Jichao Liang, Rengui Jiang et Hao Han. « Simulation and visualization of salty soil water and salt migration ». Dans Advances in Energy Science and Equipment Engineering II, 429–33. Taylor & Francis Group, 6000 Broken Sound Parkway NW, Suite 300, Boca Raton, FL 33487-2742 : CRC Press, 2017. http://dx.doi.org/10.1201/9781315116167-85.

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McKinley, M. J., D. A. Denton, M. L. Mathai, B. J. Oldfield et R. S. Weisinger. « Angiotensin Actions on the Brain Influencing Salt and Water Balance ». Dans Angiotensin Vol. II, 115–39. Berlin, Heidelberg : Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18497-0_7.

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Sahyun, M. R. V., R. J. DeVoe et P. M. Olofson. « Photochemistry of Onium Salt Decomposition ». Dans Radiation Curing in Polymer Science and Technology—Volume II, 505–27. Dordrecht : Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1876-7_10.

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Chock, P. B., J. Halpern, F. E. Paulik, Saul I. Shupack et Thomas P. DeAngelis. « Potassium Trichloro(ethylene)platinate(II)(zeise's salt ) ». Dans Inorganic Syntheses, 90–92. Hoboken, NJ, USA : John Wiley & Sons, Inc., 2007. http://dx.doi.org/10.1002/9780470132456.ch17.

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Chock, P. B., J. Halpern, F. E. Paulik, Saul I. Shupack et Thomas P. Deangelis. « Potassium Trichloro(Ethene)Platinate(II) (Zeise's Salt) ». Dans Inorganic Syntheses, 349–51. Hoboken, NJ, USA : John Wiley & Sons, Inc., 2007. http://dx.doi.org/10.1002/9780470132593.ch90.

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Sovga, E. E., E. S. Eremina, L. V. Kharitonova et T. V. Khmara. « Current Water and Salt Regime of the Sivash Bay ». Dans Processes in GeoMedia - Volume II, 225–34. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-53521-6_26.

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Actes de conférences sur le sujet "SALT II"

Silva Junior, J. F., A. E. Klar, A. A. Tanaka, I. P. F. Silva et A. I. I. Cardoso. « Tomatoes Seeds Vigor under Water or Salt Stress ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a734.

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Hilton, Matt. « SALT spectroscopic observations of galaxy clusters detected by ACT and a Type II quasar hosted by a brightest cluster galaxy ». Dans SALT Science Conference 2015. Trieste, Italy : Sissa Medialab, 2016. http://dx.doi.org/10.22323/1.250.0042.

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Swain, Trilochan, et Gouri Sankhar Brahma. « Low temperature property of Metaphosphatecopper(II/I) salt ». Dans 5TH NATIONAL CONFERENCE ON THERMOPHYSICAL PROPERTIES : (NCTP‐09). American Institute of Physics, 2016. http://dx.doi.org/10.1063/1.4945220.

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Medeiros, M. J. L., M. M. A. Silva, M. M. C. Granja, G. S. Silva-Junior, T. R. Camara et L. Willadino. « Action of Exogenous Proline in Sugarcane Genotypes Grown in vitro under Salt Stress ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a464.

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Miranda, R. S., J. T. Prisco et E. Gomes-Filho. « Nitrogen Nutrition with NH4+ Instead of NO3- Confers Salt Tolerance in Sorghum Plants ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a513.

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Lyubovskava, R. N., M. Z. Aldoshina, V. A. Konovalikhin, O. A. Dyachenko et R. B. Lyubovskii. « ET salt with [Cu(I)Hg(II)] containing anion ». Dans International Conference on Science and Technology of Synthetic Metals. IEEE, 1994. http://dx.doi.org/10.1109/stsm.1994.834903.

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Silva, M. L. S., A. R. Feijão, E. C. Marques, M. L. S. Silva, E. Gomes Filho et J. T. Prisco. « Growth, Accumulation of Solutes and Nitrogen Metabolism in Plants of Sunflower Under Salt Stress ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a649.

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Brito, M. E. B., E. C. B. Silva, P. D. Fernandes, F. V. S. Sá, W. S. Soares Filho et F. A. Silva. « Salt Balance in Substrate and Dry Matter of Hybrids of Mandarin Sunk Under Saline Water ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a598.

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Bonifácio, A., M. O. Martins, M. C. Lima Neto, F. E. L. Carvalho, A. K. M. Lobo et J. A. G. Silveira. « Alleviation of Oxidative Stress by Photosynthetic Modulations in Rice Plants Simultaneously Stressed with Salt and Heat ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a739.

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Leite, M. C. B. S., A. R. B. Farias, F. J. Freire, F. D. Andreote, J. Kuklinsky-Sobral et M. B. G. S. Freire. « Isolation, Bioprospecting and Diversity of Salt-Tolerant Bacteria Associated with Sugarcane in Soils of Pernambuco, Brazil ». Dans II Inovagri International Meeting. Fortaleza, Ceará, Brasil : INOVAGRI/INCT-EI/INCTSal, 2014. http://dx.doi.org/10.12702/ii.inovagri.2014-a498.

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Rapports d'organisations sur le sujet "SALT II"

Piccolo, S. F. HLW Salt Disposition Alternatives Preconceptual Phase II Summary Report. Office of Scientific and Technical Information (OSTI), juillet 1999. http://dx.doi.org/10.2172/9178.

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Dojcsak, L., et J. Marriner. SDSS-II : Determination of shape and color parameter coefficients for SALT-II fit model. Office of Scientific and Technical Information (OSTI), août 2010. http://dx.doi.org/10.2172/1016874.

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Jain, V., H. Shah, C. J. Bannochie et W. R. Wilmarth. Mercury Phase II Study - Mercury Behavior in Salt Processing Flowsheet. Office of Scientific and Technical Information (OSTI), juillet 2016. http://dx.doi.org/10.2172/1281779.

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Author, Not Given. Molten Salt Thermal Energy Storage Subsystem Research Experiment. Volume II. Final technical report. Office of Scientific and Technical Information (OSTI), mai 1985. http://dx.doi.org/10.2172/5749327.

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Chavez, J., et D. Smith. A final report on the phase 1 testing of a molten-salt cavity receiver. Volume II, the main report. Office of Scientific and Technical Information (OSTI), mai 1992. http://dx.doi.org/10.2172/5042358.

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Barg, Rivka, Erich Grotewold et Yechiam Salts. Regulation of Tomato Fruit Development by Interacting MYB Proteins. United States Department of Agriculture, janvier 2012. http://dx.doi.org/10.32747/2012.7592647.bard.

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Résumé :

Background to the topic: Early tomato fruit development is executed via extensive cell divisions followed by cell expansion concomitantly with endoreduplication. The signals involved in activating the different modes of growth during fruit development are still inadequately understood. Addressing this developmental process, we identified SlFSM1 as a gene expressed specifically during the cell-division dependent stages of fruit development. SlFSM1 is the founder of a class of small plant specific proteins containing a divergent SANT/MYB domain (Barg et al 2005). Before initiating this project, we found that low ectopic over-expression (OEX) of SlFSM1 leads to a significant decrease in the final size of the cells in mature leaves and fruits, and the outer pericarp is substantially narrower, suggesting a role in determining cell size and shape. We also found the interacting partners of the Arabidopsis homologs of FSM1 (two, belonging to the same family), and cloned their tomato single homolog, which we named SlFSB1 (Fruit SANT/MYB–Binding1). SlFSB1 is a novel plant specific single MYB-like protein, which function was unknown. The present project aimed at elucidating the function and mode of action of these two single MYB proteins in regulating tomato fruit development. The specific objectives were: 1. Functional analysis of SlFSM1 and its interacting protein SlFSB1 in relation to fruit development. 2. Identification of the SlFSM1 and/or SlFSB1 cellular targets. The plan of work included: 1) Detailed phenotypic, histological and cellular analyses of plants ectopically expressing FSM1, and plants either ectopically over-expressing or silenced for FSB1. 2) Extensive SELEX analysis, which did not reveal any specific DNA target of SlFSM1 binding, hence the originally offered ChIP analysis was omitted. 3) Genome-wide transcriptional impact of gain- and loss- of SlFSM1 and SlFSB1 function by Affymetrix microarray analyses. This part is still in progress and therefore results are not reported, 4) Search for additional candidate partners of SlFSB1 revealed SlMYBI to be an alternative partner of FSB1, and 5) Study of the physical basis of the interaction between SlFSM1 and SlFSB1 and between FSB1 and MYBI. Major conclusions, solutions, achievements: We established that FSM1 negatively affects cell expansion, particularly of those cells with the highest potential to expand, such as the ones residing inner to the vascular bundles in the fruit pericarp. On the other hand, FSB1 which is expressed throughout fruit development acts as a positive regulator of cell expansion. It was also established that besides interacting with FSM1, FSB1 interacts also with the transcription factor MYBI, and that the formation of the FSB1-MYBI complex is competed by FSM1, which recognizes in FSB1 the same region as MYBI does. Based on these findings a model was developed explaining the role of this novel network of the three different MYB containing proteins FSM1/FSB1/MYBI in the control of tomato cell expansion, particularly during fruit development. In short, during early stages of fruit development (Phase II), the formation of the FSM1-FSB1 complex serves to restrict the expansion of the cells with the greatest expansion potential, those non-dividing cells residing in the inner mesocarp layers of the pericarp. Alternatively, during growth phase III, after transcription of FSM1 sharply declines, FSB1, possibly through complexing with the transcription factor MYBI serves as a positive regulator of the differential cell expansion which drives fruit enlargement during this phase. Additionally, a novel mechanism was revealed by which competing MYB-MYB interactions could participate in the control of gene expression. Implications, both scientific and agricultural: The demonstrated role of the FSM1/FSB1/MYBI complex in controlling differential cell growth in the developing tomato fruit highlights potential exploitations of these genes for improving fruit quality characteristics. Modulation of expression of these genes or their paralogs in other organs could serve to modify leaf and canopy architecture in various crops.

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Molten Salt Electric Experiment (MSEE) - Phase I report. Volume II. Office of Scientific and Technical Information (OSTI), août 1985. http://dx.doi.org/10.2172/5169553.

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Shallow ground water in the Whitney area, southeastern Las Vegas Valley, Clark County, Nevada ; Part II, Assessment of a proposed strategy to reduce the contribution of salts to Las Vegas Wash. US Geological Survey, 1993. http://dx.doi.org/10.3133/wri924051.

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