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Bibliographies thématiques / Tumor U87

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Littérature scientifique sur le sujet « Tumor U87 »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 17 février 2022

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Articles de revues sur le sujet "Tumor U87"

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Stakišaitis, Donatas, Eligija Damanskienė, Rūta Curkūnavičiūtė, et al. "The Effectiveness of Dichloroacetate on Human Glioblastoma Xenograft Growth Depends on Na+ and Mg2+ Cations." Dose-Response 19, no. 1 (2021): 155932582199016. http://dx.doi.org/10.1177/1559325821990166.

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The study’s aim was to investigate the effectiveness of sodium dichloroacetate (NaDCA) or magnesium dichloroacetate (MgDCA) on adult U87 MG and pediatric PBT24 cell lines glioblastoma (GB) xenografts in a chicken chorioallantoic membrane (CAM) model. The study groups were: treated with 10 mM, 5 mM of NaDCA, and 5 mM, 2.5 mM of MgDCA, and controls. The U87 MG and PBT24 xenografts growth, frequency of tumor invasion into CAM, CAM thickening, and the number of blood vessels in CAM differed depending on the dichloroacetate salt treatment. NaDCA impact on U87 MG and PBT24 tumor on proliferating cel

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Wei, Dan, JiaLi Hou, Ke Zheng, et al. "Suicide Gene Therapy Against Malignant Gliomas by the Local Delivery of Genetically Engineered Umbilical Cord Mesenchymal Stem Cells as Cellular Vehicles." Current Gene Therapy 19, no. 5 (2019): 330–41. http://dx.doi.org/10.2174/1566523219666191028103703.

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Background: Glioblastoma (GBM) is a malignant tumor that is difficult to eliminate, and new therapies are thus strongly desired. Mesenchymal stem cells (MSCs) have the ability to locate to injured tissues, inflammation sites and tumors and are thus good candidates for carrying antitumor genes for the treatment of tumors. Treating GBM with MSCs that have been transduced with the herpes simplex virus thymidine kinase (HSV-TK) gene has brought significant advances because MSCs can exert a bystander effect on tumor cells upon treatment with the prodrug ganciclovir (GCV). Objective: In this study,

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Herrera, Victoria L. M., Glaiza L. A. Tan, Ann Marie Moran, Kristine A. Pasion, Julius L. Decano, and Nelson Ruiz-Opazo. "Dual endothelin-1/VEGFsp receptor (DEspR): Common target on tumor vascular endothelial cells (TVECs), tumor cells (TCs), and cancer stem cells (CSCs) in glioblastoma and pancreatic cancer." Journal of Clinical Oncology 30, no. 15_suppl (2012): e13574-e13574. http://dx.doi.org/10.1200/jco.2012.30.15_suppl.e13574.

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e13574 Background: New therapies are needed for glioblastoma (GBM) and pancreatic adenocarcinoma (PCa), given their low survival rates. To address this mandate, we tested the hypothesis that inhibition of DEspR, as a common target on TVECs, TCs, and CSCs, can affect multiple key steps in metastasis: angiogenesis, invasiveness, anoikis resistance. Methods: Various analyses were performed: co-immunostaining of tumor tissue and cancer cell lines from PCa and GBM using anti-CD133 and anti-DEspR mAbs; in vitro anti-DEspR mAb inhibition of angiogenesis and TC invasiveness; isolation, functional vali

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Perez, Joshua, Javier Fierro, Rocio Aguilar, and Huanyu Dou. "OTME-18. Targeted CRISPR/Cas9 gene-editing regulates the brain tumor environment." Neuro-Oncology Advances 3, Supplement_2 (2021): ii17. http://dx.doi.org/10.1093/noajnl/vdab070.069.

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Abstract Glioblastoma multiform (GBM) is the most common malignant brain tumor. Recent immunotherapy has demonstrated potential to treat GBM. However, the immune suppressive tumor environment in the brain represents a significant barrier for the treatment of GBM. Overexpression of programmed death ligand-1 (PD-L1) in GBM tumor cells and macrophages plays a key role in GBM vitality, proliferation, and migration, while also suppressing the immune system. We developed a CRISPR/Cas9 gene-editing system to delete whole cell PD-L1. Human PD-L1 targeted sgRNA were cloned into CRISPR/Cas9 plasmids wit

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Zhang, Hanwen, Masatomo Maeda, Masahiro Shindo, et al. "Imaging CXCR4 Expression with Iodinated and Brominated Cyclam Derivatives." Molecular Imaging and Biology 22, no. 5 (2020): 1184–96. http://dx.doi.org/10.1007/s11307-020-01480-1.

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Abstract Purpose CXCR4 is one of several “chemokine” receptors expressed on malignant tumors (including GBM and PCNSL) and hematopoietic stem cells. Although 68Ga-pentixafor and 68Ga-NOTA-NFB have been shown to effectively image CXCR4 expression in myeloma and other systemic malignancies, imaging CXCR4 expression in brain tumors has been more limited due to the blood-brain barrier (BBB) and a considerable fraction of CXCR4 staining is intracellular. Methods We synthesized 6 iodinated and brominated cyclam derivatives with high affinity (low nM range) for CXCR4, since structure-based estimates

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Qu, Xiaochao, Xiaoxiao Li, Jingning Liang, Yanran Wang, Muhan Liu, and Jimin Liang. "Micro-CT Imaging of RGD-Conjugated Gold Nanorods Targeting TumorIn Vivo." Journal of Nanomaterials 2016 (2016): 1–13. http://dx.doi.org/10.1155/2016/8368154.

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Gold nanomaterials as computed tomography (CT) contrast agents at lower X-ray dosage to get a higher contrast have advantages of longer imaging time and lower toxic side effects compared to current contrast agents. As a receptor for Cyclo (Arg-Gly-Asp-D-Phe-Lys) (RGD) peptide, integrinαvβ3is overexpressed on some tumor cells and tumor neovasculature. In this paper, we conjugated the RGD peptide on the surface of gold nanorods (AuNRs), designated as RGD-AuNRs, a promising candidate in applications such as tumor targeting and imaging capability for micro-CT imaging. Integrinαvβ3-positive U87 cel

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Thokala, Radhika, Zev Binder, Yibo Yin, Michael Milone, and Donald M. O’Rourke. "IMMU-36. HIGH AFFINITY CHIMERIC ANTIGEN RECEPTOR WITH CROSS-REACTIVITY TO CLINICALLY-RELEVANT EGFR MUTATED PROTEINS." Neuro-Oncology 22, Supplement_2 (2020): ii112. http://dx.doi.org/10.1093/neuonc/noaa215.466.

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Abstract Tumor heterogeneity is one of the key reasons for therapeutic failure in Glioblastoma (GBM). Our chimeric antigen receptor (CAR) T cell clinical trial (NCT02209376) against Epidermal growth factor receptor (EGFR) variant III (EGFRvIII) demonstrated successful trafficking of T cells across the blood brain barrier into GBM active tumor sites. However, CART cell infiltration was associated with a selective loss of EGFRvIII+ tumor cells and upregulation of immunosuppressive molecules. Post-CAR T treated tumor specimens showed continued presence of EGFR amplification and oncogenic EGFR ext

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Safaee, Sahar, Masoumeh Fardi, Nima Hemmat, et al. "Silencing ZEB2 Induces Apoptosis and Reduces Viability in Glioblastoma Cell Lines." Molecules 26, no. 4 (2021): 901. http://dx.doi.org/10.3390/molecules26040901.

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Background: Glioma is an aggressive type of brain tumor that originated from neuroglia cells, accounts for about 80% of all malignant brain tumors. Glioma aggressiveness has been associated with extreme cell proliferation, invasion of malignant cells, and resistance to chemotherapies. Due to resistance to common therapies, glioma affected patients’ survival has not been remarkably improved. ZEB2 (SIP1) is a critical transcriptional regulator with various functions during embryonic development and wound healing that has abnormal expression in different malignancies, including brain tumors. ZEB2

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Kavaliauskaitė, Dovilė, Donatas Stakišaitis, Justė Martinkutė, et al. "The Effect of Sodium Valproate on the Glioblastoma U87 Cell Line Tumor Development on the Chicken Embryo Chorioallantoic Membrane and on EZH2 and p53 Expression." BioMed Research International 2017 (2017): 1–12. http://dx.doi.org/10.1155/2017/6326053.

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Literature data support evidences that glioblastoma (GBM) patients experience prolonged survival due to sodium valproate (NaVP) treatment. The study assessed the human GBM cell U87 xenograft studied in the chicken embryo chorioallantoic membrane (CAM) model evaluating NaVP effect on tumor. Three groups of tumors (eachn= 10) were studied: nontreated, treated with 4 mM, and treated with 8 mM of NaVP. The majority of tumors without NaVP treatment during tumor growth destroyed the chorionic epithelium, invaded the mesenchyme, and induced angiogenesis. Incidence of tumor formation on CAM without in

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Oliver, Katherine, Jarrod Longcor, and Irawati Kandela. "Efficacy of a repeat dose injection of CLR 131 (I-131-CLR1404) in U87-MG tumor-bearing athymic nude mice." Journal of Clinical Oncology 35, no. 15_suppl (2017): e14089-e14089. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e14089.

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e14089 Background: CLR 131 is a novel radioiodinated therapeutic that exploits the selective uptake and retention of phospholipid ethers (PLEs) by malignant cells. Study was to evaluate the therapeutic effect of CLR 131 when administered as a repeat dose intravenous injection in U87-MG tumor bearing mice. Malignant gliomas are associated with a low survival rate, and treatment with chemotherapy, external beam radiation, or surgery have shown limited effectiveness. In this study, therapeutic effect was evaluated by measurement of tumor volume increase and by survival of CLR 131 treated animals

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Thèses sur le sujet "Tumor U87"

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Flament, Julien. "Développement de l'imagerie RMN par agents CEST : application à un modèle rongeur de tumeur cérébrale." Phd thesis, Université Paris Sud - Paris XI, 2012. http://tel.archives-ouvertes.fr/tel-00720031.

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L'objectif de cette thèse est de développer l'imagerie de transfert de saturation des agents de contraste lipoCEST pour la détection de l'angiogenèse dans un modèle souris de tumeur cérébrale U87. Un lipoCEST offrant un seuil de sensibilité in vitro de 100 pM est optimisé afin de répondre aux contraintes de l'imagerie CEST in vivo. Grâce à la mise en place d'un dispositif expérimental dédié à l'imagerie CEST, nous évaluons les performances des lipoCEST pour détecter de façon spécifique l'angiogenèse tumorale. Nous montrons pour la première fois qu'il est possible de détecter un lipoCEST in viv

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Letzien, Ulrike. "Effects of Carnosine and L-histidine on Viability and Expression of Pyruvate Dehydrogenase Kinase 4 in Human Glioblastoma Cells." Doctoral thesis, Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-197285.

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Die Arbeit behandelt die Ergebnisse von Experimenten über die Wirkung des Dipeptides Carnosin (β Alanyl L Histidin) und der Aminosäuren L Histidin und β-Alanin auf Kulturen der humanen Zellreihen U87, T98G und LN405, welche von Zellen des malignen Hirntumors Glioblastoma multiforme abgeleitet sind. Die Vitalität der Zellen nach Inkubation mit Carnosin oder L Histidin wurde anhand der Adenosintriphosphatproduktion und der Dehydrohenaseaktivität für Inkubationszeiträume von 24, 48 und 72 Stunden bestimmt. Dabei zeigte sich eine signifikant niedrigere Vitalität der mit Carnosin oder L Histidin

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Hammerbacher, Katharina [Verfasser], and Ralf [Akademischer Betreuer] Kinscherf. "Effects of novelly synthesized nucleolipides on different tumor cell lines (HT29, HepG2, Panc-1, RenCa) with special respect to glioma cell lines (BT4Ca, GOS3, G28, G112, U251, U87) of human or other species / Katharina Hammerbacher ; Betreuer: Ralf Kinscherf." Marburg : Philipps-Universität Marburg, 2020. http://d-nb.info/120746967X/34.

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Letzien, Ulrike. "Effects of Carnosine and L-histidine on Viability and Expression of Pyruvate Dehydrogenase Kinase 4 in Human Glioblastoma Cells." Doctoral thesis, 2015. https://ul.qucosa.de/id/qucosa%3A14489.

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Die Arbeit behandelt die Ergebnisse von Experimenten über die Wirkung des Dipeptides Carnosin (β Alanyl L Histidin) und der Aminosäuren L Histidin und β-Alanin auf Kulturen der humanen Zellreihen U87, T98G und LN405, welche von Zellen des malignen Hirntumors Glioblastoma multiforme abgeleitet sind. Die Vitalität der Zellen nach Inkubation mit Carnosin oder L Histidin wurde anhand der Adenosintriphosphatproduktion und der Dehydrohenaseaktivität für Inkubationszeiträume von 24, 48 und 72 Stunden bestimmt. Dabei zeigte sich eine signifikant niedrigere Vitalität der mit Carnosin oder L Histidin

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Oettler, Manuela. "Vergleich der Strahlenwirkung auf Tumorzellkulturen und Tumorstammzellkulturen aus unterschiedlichen Glioblastomen." Doctoral thesis, 2014. http://hdl.handle.net/11858/00-1735-0000-0022-5E9B-4.

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Actes de conférences sur le sujet "Tumor U87"

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Dean, David, John E. George III, Yusra Ahmad, et al. "Pc 4 photodynamic therapy of U87 (human glioma) orthotopic tumor in nude rat brain." In Biomedical Optics 2005, edited by Kenneth E. Bartels, Lawrence S. Bass, Werner T. W. de Riese, et al. SPIE, 2005. http://dx.doi.org/10.1117/12.591213.

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Wachsberger, Phyllis Rachelle, Yaacov Lawrence, Yi Liu, Xia Xu, and Adam P. Dicker. "Abstract LB-310: Effects of epidermal growth factor receptor (EGFR) expression on anti-tumor efficacy of vandetanib or cediranib combined with radiotherapy (RT) in U87 human glioblastoma (GBM) xenografts." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-310.

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Scicinski, Jan, Bryan Oronsky, Shoucheng Ning, Andrew Minchinton, and Susan Knox. "Abstract 4371: RRx-001 modulates intratumor blood flow in SCCVII and U87 tumors." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4371.

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Varghai, Davood, Nathan Cross, Chandra Spring-Robinson, et al. "Monitoring Pc 4-mediated photodynamic therapy of U87 tumors with 18F- fluorodeoxy-glucose PET imaging in the Athymic Nude Rat." In Biomedical Optics (BiOS) 2007, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2007. http://dx.doi.org/10.1117/12.701517.

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Varghai, Davood, Kelly Covey, Rahul Sharma, et al. "Monitoring Pc 4-mediated photodynamic therapy of U87 tumors with dynamic contrast enhanced-magnetic resonance imaging (DCE-MRI) in the athymic nude rat." In Biomedical Optics (BiOS) 2008, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2008. http://dx.doi.org/10.1117/12.763983.

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Cross, Nathan, Davood Varghai, Chris A. Flask, Denise K. Feyes, Nancy L. Oleinick, and David Dean. "Optimal gadolinium dose level for magnetic resonance imaging (MRI) contrast enhancement of U87-derived tumors in athymic nude rats for the assessment of photodynamic therapy." In SPIE BiOS: Biomedical Optics, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2009. http://dx.doi.org/10.1117/12.809707.

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Cross, Nathan, Davood Varghai, Chandra Spring-Robinson, et al. "Analysis of 18F-fluorodeoxy-glucose PET imaging data captured before and after Pc 4-mediated photodynamic therapy of U87 tumors in the athymic nude rat." In Biomedical Optics (BiOS) 2007, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2007. http://dx.doi.org/10.1117/12.701672.

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