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Littérature scientifique sur le sujet « Type II Fc gamma receptor »

Auteur : Grafiati

Publié le 4 juin 2021

Mis à jour le 1 février 2022

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Articles de revues sur le sujet "Type II Fc gamma receptor"

Liang, L., та C. K. Huang. "Tyrosine phosphorylation induced by cross-linking of Fc γ-receptor type II in human neutrophils". Biochemical Journal 306, № 2 (1995): 489–95. http://dx.doi.org/10.1042/bj3060489.

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Neutrophils express several receptors for the Fc region of IgG molecules. Specific cross-linking of the type II receptor (Fc gamma RII) can be achieved by treating neutrophils with the Fab fragment of a specific monoclonal antibody IV.3 against the receptor followed by goat anti-mouse IgG F(ab′)2 fragment. Such treatment initiates a number of neutrophil responses including the release of O2-. and increased protein tyrosine phosphorylation. The increase in tyrosine phosphorylation is rapid and transient and correlates with O2-. release. Both responses are inhibited by pretreatment of neutrophil

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Benech, P. D., K. Sastry, R. R. Iyer, Q. G. Eichbaum, D. P. Raveh, and R. A. Ezekowitz. "Definition of interferon gamma-response elements in a novel human Fc gamma receptor gene (Fc gamma RIb) and characterization of the gene structure." Journal of Experimental Medicine 176, no. 4 (1992): 1115–23. http://dx.doi.org/10.1084/jem.176.4.1115.

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The human Fc gamma RI (CD64) is a high affinity receptor for the Fc portion of immunoglobulin (Ig), and its constitutively low expression on the cell surface of monocyte/macrophage and neutrophils is selectively upregulated by interferon gamma (IFN-gamma) treatment (Perussia, B., E. T. Dayton, R. Lazarus, V. Fanning, and G. Trinchieri. 1983. J. Exp. Med. 158:1092). Three distinct cDNAs have been cloned and code for proteins that predict three extracellular Ig-like domains (Allen, J.M., and B. Seed. 1989. Science [Wash. DC]. 243:378). Several differences in the coding region of these cDNAs sugg

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Kindt, G. C., J. G. van de Winkel, S. A. Moore, and C. L. Anderson. "Identification and structural characterization of Fc gamma-receptors on pulmonary alveolar macrophages." American Journal of Physiology-Lung Cellular and Molecular Physiology 260, no. 6 (1991): L403—L411. http://dx.doi.org/10.1152/ajplung.1991.260.6.l403.

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Little is known about the structure of the cell surface receptors for the Fc portion of immunoglobulin G (Fc gamma R) on tissue macrophages. Studies on leukocytes indicate the existence of three classes of Fc gamma R, denoted I, II, and III. The purpose of this study was to structurally characterize Fc gamma R on alveolar macrophages obtained by bronchoalveolar lavage, comparing them with Fc gamma R of monocytes, neutrophils, and U937 cells. Flow-cytometric evaluation, utilizing anti-Fc gamma R class-specific monoclonal antibodies, showed that alveolar macrophages expressed three Fc gamma R cl

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De Reys, S., C. Blom, B. Lepoudre, et al. "Human platelet aggregation by murine monoclonal antiplatelet antibodies is subtype-dependent." Blood 81, no. 7 (1993): 1792–800. http://dx.doi.org/10.1182/blood.v81.7.1792.1792.

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Abstract Twenty murine monoclonal antibodies (MoAbs) generated against different human platelet antigens induced clumping of human platelets in plasma and buffer. Whereas one MoAb could agglutinate platelets, clumping for 19 MoAbs was blocked by metabolic inhibitors, indicating that these induce platelet activation. Fifteen MoAbs were of IgG1, two of IgG2a, and two of IgG2b subtype. F(ab')2 fragments of these did not evoke an aggregatory response, but specifically inhibited aggregations by and binding of their respective intact MoAbs to platelets. Single-platelet counting technology indicated

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Twenty murine monoclonal antibodies (MoAbs) generated against different human platelet antigens induced clumping of human platelets in plasma and buffer. Whereas one MoAb could agglutinate platelets, clumping for 19 MoAbs was blocked by metabolic inhibitors, indicating that these induce platelet activation. Fifteen MoAbs were of IgG1, two of IgG2a, and two of IgG2b subtype. F(ab')2 fragments of these did not evoke an aggregatory response, but specifically inhibited aggregations by and binding of their respective intact MoAbs to platelets. Single-platelet counting technology indicated that the

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Sarmay, G., I. Pecht, and J. Gergely. "Protein-tyrosine kinase activity tightly associated with human type II Fc gamma receptors." Proceedings of the National Academy of Sciences 91, no. 10 (1994): 4140–44. http://dx.doi.org/10.1073/pnas.91.10.4140.

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Choquet, D., M. Partiseti, S. Amigorena, C. Bonnerot, W. H. Fridman, and H. Korn. "Cross-linking of IgG receptors inhibits membrane immunoglobulin-stimulated calcium influx in B lymphocytes." Journal of Cell Biology 121, no. 2 (1993): 355–63. http://dx.doi.org/10.1083/jcb.121.2.355.

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By cross-linking membrane immunoglobulins (mIg), the antigenic stimulation of B lymphocytes induces an increase in intracellular free calcium levels ([Ca2+]i) because of a combination of release from intracellular stores and transmembrane influx. It has been suggested that both events are linked, as in a number of other cases of receptor-induced increase in [Ca2+]i. Conversely, in B lymphocytes, type II receptors for the Fc fragment of IgG (Fc gamma RII) inhibit mIg-mediated signaling. Thus, we have investigated at the level of single cells if these receptors could act on specific phases of mI

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Král, Bohuslav, Jan Krejsek, Zdena Paráková, et al. "Some Serum Activity Markers of Airways Inflammation in Difficult-To-Control Asthma Patients." Acta Medica (Hradec Kralove, Czech Republic) 40, no. 3 (1997): 61–70. http://dx.doi.org/10.14712/18059694.2020.46.

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The main aim of the present study was a search for a characteristic serum marker of inflammatory activity in the airways of asthmatics with difficult-to-control disease. Therefore, serum levels of interleukin-4 (IL-4), serum low-affinity Fc Epsilon Receptor II (sFcER II), Interferon-gamma (INF-gamma) Immunoglobulin-E (IgE), Interleukin-2 (IL-2), serum Interleukin Receptor 2 (sIL-2R) and Intercellular Adhesion Molecule-1 (sICAM-1) were measured in 2 groups of asthmatics: 1-26 patients with difficult-to-control asthma (DTCA), 2-22 asthmatics, minimally symptomatic (MSA). Results: No significant

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Lebrazi, Jamal, Gérard Helft, Mustapha Abdelouahed, et al. "Human Anti-Streptokinase Antibodies Induce Platelet Aggregation in an Fc Receptor (CD32) Dependent Manner." Thrombosis and Haemostasis 74, no. 03 (1995): 938–42. http://dx.doi.org/10.1055/s-0038-1649851.

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SummaryExposure to streptokinase (SK) elicits anti-SK antibodies (Abs), which inhibit fibrinolysis and induce platelet aggregation. The mechanism of the latter is not fully understood, although it seems to involve platelet binding by a plasminogen streptokinase and anti-SK ternary complex. Anti-SK Abs were purified by affinity chromatography from serum of patients having received SK for acute myocardial infarction (AMI), and were shown to be of the IgG type. Their effects were studied with (i) human platelets in citrated plasma in the presence of SK or acetylated plasminogen-SK activator compl

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Garcia-Foncillas, Jesus, Javier Rodriguez, Valentina Boni, et al. "Role of polymorphic fc gamma receptor II/III in the efficacy of cetuximab in KRAS-NRAS-BRAF-PI3K mutated." Journal of Clinical Oncology 30, no. 4_suppl (2012): 477. http://dx.doi.org/10.1200/jco.2012.30.4_suppl.477.

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477 Background: The immunoglobulin G1 (IgG1) monoclonal antibody (MoAb) cetuximab is active in metastatic colorectal cancer (mCRC) as first or subsequent lines of therapy. Efficacy seems restricted to KRAS wild-type tumors. IgG1 may also induce antibody dependent cell mediated citotoxicity (ADCC) by recruitment of immune effector cells. ADCC is influenced by FcγR polymorphisms. We investigated the association of FcγR polymorphisms and disease control rate (DCR) in mCRC patients treated with chemotherapy plus cetuximab. Methods: Tumor tissues from 106 patients were screened for KRAS codon 12 an

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Thèses sur le sujet "Type II Fc gamma receptor"

Zhou, Xueyuan. "Follicular Dendritic Cells, Human Immunodeficency Virus Type 1, and Alpha 1 Antitrypsin." BYU ScholarsArchive, 2012. https://scholarsarchive.byu.edu/etd/3407.

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HIV/AIDS is raging and causing millions of deaths around the world. The major challenge in treating HIV/AIDS is the establishment of HIV reservoirs where the viruse escapes both drug and immune system attempts at eradication. Throughout the course of HIV/AIDS, productive HIV infection occurs primarily in the lymphoid follicles or germinal centers (GC) surrounding follicular dendritic cells (FDC). In the GCs, FDCs trap and maintain infectious HIV for years and provide these infectious viruses to the host cells. FDCs also attract B and T cells into the GCs and increase the ability of CD4+ T cell

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Actes de conférences sur le sujet "Type II Fc gamma receptor"

Blom, AB, PL Van Lent, AE Holthuysen, et al. "THU0111 Fc gamma receptor i and iii expression by macrophages of collagen type ii susceptible mice is higher and differently regulated compared to other strains." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.988.

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Bougherara, Houcine, Fariba Némati, Christophe De Romeuf, et al. "Abstract A01: Ex vivo evaluation of an anti-Müllerian hormone type II receptor humanized antibody with optimized Fc effector function in ovarian cancer." In Abstracts: AACR Special Conference: Tumor Immunology and Immunotherapy: A New Chapter; December 1-4, 2014; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/2326-6074.tumimm14-a01.

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