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Articles de revues sur le sujet « Vascular neuropathy »

Auteur : Grafiati

Publié le 7 juin 2025

Mis à jour le 2 août 2025

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1

Tesfaye, S., R. Malik, and J. D. Ward. "Vascular factors in diabetic neuropathy." Diabetologia 37, no. 9 (1994): 847–54. http://dx.doi.org/10.1007/s001250050187.

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Tesfaye, S., R. Malik, and J. D. Ward. "Vascular factors in diabetic neuropathy." Diabetologia 37, no. 9 (1994): 847–54. http://dx.doi.org/10.1007/bf00400938.

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Plummer, E. Sharon, and Stewart G. Albert. "Foot Care Assessment in Patients with Diabetes: A Screening Algorithm for Patient Education and Referral." Diabetes Educator 21, no. 1 (1995): 47–51. http://dx.doi.org/10.1177/014572179502100108.

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The foot care behaviors of patients with diabetes were assessed by medical history, and their feet were examined for peripheral neuropathy, peripheral vascular disease, foot ulcers, and deformities. The sample consisted of 136 patients (14 with insulin- dependent diabetes and 122 with non-insulin-dependent diabetes). Mean age was 61 years and mean duration of diabetes was 13 years. Peripheral vascular disease was found in 25% of the patients, peripheral neuropathy in 33%, and 13% had both peripheral vascular disease and peripheral neuropathy. Potentially unsafe nail and foot care practices wer

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Roy, Bhaskar, Avash Das, Kumar Ashish, et al. "Neuropathy with vascular endothelial growth factor receptor tyrosine kinase inhibitors." Neurology 93, no. 2 (2019): e143-e148. http://dx.doi.org/10.1212/wnl.0000000000007743.

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ObjectiveTo explore the association of peripheral neuropathy with vascular endothelial growth factor receptor tyrosine kinase inhibitors (VEGFR-TKIs) use in patients with cancer.MethodsPublished data search up to November 2018 reporting peripheral neuropathy in patients with cancer treated with VEGFR-TKIs was performed. The primary outcome was presence of peripheral neuropathy at the end of the trial. Random-effects meta-analysis was performed to estimate relative risk (RR) of individual treatment.ResultsThirty randomized clinical trials (RCTs) including 12,490 patients with cancer were includ

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Korada, H. Ya, E. Arora, Sh K. Rao, M. Hande, S. Shetty, and A. G. Maiya. "Plantar pressure distribution profile of type 2 diabetes mellitus with diabetic foot syndrome: A hospital-based observational study." Diabetes mellitus 24, no. 6 (2021): 548–52. http://dx.doi.org/10.14341/dm12723.

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INTRODUCTION: The prevalence of diabetic foot syndrome is increasing in the Indian population. It is a triad of neurological, vascular, and biomechanical changes due to long term diabetes mellitus (DM). Altered plantar pressure distribution is a risk factor for developing diabetic foot ulcers. The purpose of this study is to evaluate the altered plantar pressure distribution in diabetic peripheral neuropathy individuals with peripheral vascular disease in comparison with diabetic peripheral neuropathy and non-diabetic neuropathy. Therefore, the objective of the study is to evaluate the plantar

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Wahane, Madhugandha, Amit Agrawal, and Rajendra Kumar Sureka. "A Rare Case of an Inferior Gluteal Artery Causing Compressive Vascular Neuropathy of the Sciatic Nerve Presenting as Foot Drop." Journal of Modern Medicine 2, no. 2 (2024): 88–90. https://doi.org/10.4103/jomm.jomm_7_24.

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Abstract Foot drop is most frequently caused by peroneal or sciatic nerve injury. The most common causes of nerve injury are trauma, compression, tumor, or entrapment. Vascular compressive neuropathy was extensively studied by Hultborn and Kjellman, and they observed that gluteal artery aneurysm can present with sciatica-like symptoms. Saritha et al. have studied the anatomical variation in the bifurcation of the sciatic nerve and its presentation. So far, there are no case reports on a vascular anomaly in association with an anatomical variation leading to compressive vascular neuropathy. We

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Moneta, G. L. "Vascular Risk Factors and Diabetic Neuropathy." Yearbook of Vascular Surgery 2006 (January 2006): 117–18. http://dx.doi.org/10.1016/s0749-4041(08)70101-5.

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Tesfaye, Solomon, Nish Chaturvedi, Simon E. M. Eaton, et al. "Vascular Risk Factors and Diabetic Neuropathy." New England Journal of Medicine 352, no. 4 (2005): 341–50. http://dx.doi.org/10.1056/nejmoa032782.

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Verma, A. "Vascular Risk Factors and Diabetic Neuropathy." Yearbook of Neurology and Neurosurgery 2006 (January 2006): 47–48. http://dx.doi.org/10.1016/s0513-5117(08)70263-1.

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Tesfaye, S., N. Chaturvedi, and S. E. Eaton. "Vascular risk factors and diabetic neuropathy." Journal of Vascular Surgery 41, no. 6 (2005): 1079. http://dx.doi.org/10.1016/j.jvs.2005.03.051.

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Liew, Colin, M. Sharma, L. Barreto, B. M. W. Bailey, M. Manisali, and B. Visavadia. "Non-vascular considerations in trigeminal neuropathy." British Journal of Oral and Maxillofacial Surgery 45, no. 7 (2007): e19. http://dx.doi.org/10.1016/j.bjoms.2007.07.163.

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Inoue, M. "Vascular Optic Neuropathy in Diabetes Mellitus." Japanese Journal of Ophthalmology 41, no. 5 (1997): 328–31. http://dx.doi.org/10.1016/s0021-5155(97)00068-3.

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Simpson, L. O. "Vascular factors in diabetic neuropathy: comment." Diabetologia 38, no. 7 (1995): 872–73. http://dx.doi.org/10.1007/bf03035304.

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Leahy, J. L. "Vascular Risk Factors and Diabetic Neuropathy." Yearbook of Endocrinology 2006 (January 2006): 85–87. http://dx.doi.org/10.1016/s0084-3741(08)70298-1.

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Krendel, David A. "Vascular inflammation in proximal diabetic neuropathy." Journal of Neurology 245, no. 11 (1998): 748. http://dx.doi.org/10.1007/s004150050280.

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Durcanova, Beata, Alvin Y. Chan, Joeseph A. Osorio, et al. "Venous vascular malformations and compressive neuropathy." Interdisciplinary Neurosurgery 16 (June 2019): 113–16. http://dx.doi.org/10.1016/j.inat.2019.01.010.

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Tesfaye, S., N. Chaturvedi, and S. E. M. Eaton. "Vascular risk factors and diabetic neuropathy." ACC Current Journal Review 14, no. 4 (2005): 20–21. http://dx.doi.org/10.1016/j.accreview.2005.02.082.

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Ram, Z., M. Sadeh, R. Walden, and R. Adar. "Vascular Insufficiency Quantitatively Aggravates Diabetic Neuropathy." Archives of Neurology 48, no. 12 (1991): 1239–42. http://dx.doi.org/10.1001/archneur.1991.00530240043016.

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Teliti, Marsida, Giulia Cogni, Lucia Sacchi, et al. "Risk factors for the development of micro-vascular complications of type 2 diabetes in a single-centre cohort of patients." Diabetes and Vascular Disease Research 15, no. 5 (2018): 424–32. http://dx.doi.org/10.1177/1479164118780808.

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Aims: In type 2 diabetes, we aimed at clarifying the role of glycated haemoglobin variability and other risk factors in the development of the main micro-vascular complications: peripheral neuropathy, nephropathy and retinopathy. Methods: In a single-centre cohort of 900 patients, glycated haemoglobin variability was evaluated as intra-individual standard deviation, adjusted standard deviation and coefficient of variation of serially measured glycated haemoglobin in the 2-year period before a randomly selected index visit. We devised four models considering different aspects of glycated haemog

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Broadstone, Vasti L., Douglas A. Greene, Jahangir Cyrus, and Michael A. Pfeifer. "Diabetic Peripheral Neuropathy Part I: Sensorimotor Neuropathy." Diabetes Educator 13, no. 1 (1987): 30–35. http://dx.doi.org/10.1177/014572178701300107.

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Diabetic neuropathy, defined as objective evidence of neural im pairment accompanied by discern ible symptoms, is a common complication of diabetes. Pathologically it is considered a "dying back " neuropathy in which the distal portions of the neurons are first and more severely affected. Both vascular and metabolic hypotheses have been advanced for its patho genesis. Diabetic sensorimotor neuropathy has two subclasses: focal and distal symmetrical polyneuropathy. Signs and symp toms, complications, and guide lines for treatment are listed for the different syndromes. Current research includes

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Druzhinina, Evgeniya S., Igor V. Isaev, Fedor A. Kostylev, Anton G. Narbutov, and Nikolay N. Zavadenko. "Venous malformation as a rare cause of sciatic neuropathy." L.O. Badalyan Neurological Journal 4, no. 3 (2023): 175–80. http://dx.doi.org/10.46563/2686-8997-2023-4-3-175-180.

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Sciatic neuropathy is rare and difficult to diagnose in pediatrics and often associated with difficult to control neuropathic pain. The most common causes of sciatic nerve damage in children include trauma and iatrogenic lesions, less often the cause is tumor and vascular formations, among which intraneural venous malformations are the rarest. We present a clinical case of a 13-year boy suffered from right sciatic pain neuropathy with unexpressed neuropathic pain syndrome, asymmetry of the feet, scant impairment of motor and sensory function of the limb for five years, which changed the gait.

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Antoine-Frank, Chrystal D., Kaydeonne T. Ellis, Malcolm R. Antoine, Pars Daniel Annan, and Rimanatou Seyni-Boureima. "Diabetic peripheral neuropathy: pain management." International Journal of Advances in Medicine 8, no. 3 (2021): 466. http://dx.doi.org/10.18203/2349-3933.ijam20210610.

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As diabetes mellitus continues to be a global health issue, more and more cases of macro-vascular and micro-vascular complications are being commonly observed amongst this category of patients. One micro-vascular complication often seen in diabetic patients is diabetic neuropathy as at least 50% of diabetic patients will experience some form of neuropathy following a diagnosis of diabetes for about 25 years. Even though diabetic patients are at risk for developing various types of neuropathies, polyneuropathies are most commonly observed and develop mainly develop due to hyperglycaemia, dyslip

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Winston, Paul, and Dannika Bakker. "Ischemic Monomelic Neuropathy: The Case for Reintroducing a Little-Known Term." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 47, no. 5 (2020): 697–99. http://dx.doi.org/10.1017/cjn.2020.88.

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ABSTRACT:Ischemic monomelic neuropathy (IMN) is a little-known, painful axonal neuropathy, secondary to vascular occlusion or steal phenomenon. It typically occurs after vascular bypass, hemodialysis fistulization, or diabetic microvascular disease in the absence of significant clinical features of ischemia. There is limited literature to assist in the characterization and diagnosis of this condition. We describe three patients with IMN with no surgical or peripheral vascular disease history who exhibited spontaneous, persistent foot pain, edema numbness, and weakness with denervation on needl

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Chen, Zecai. "Prediction of vasculitic neuropathy using supervised machine learning approaches." Информатика. Экономика. Управление - Informatics. Economics. Management 3, no. 1 (2024): 0301–10. http://dx.doi.org/10.47813/2782-5280-2024-3-1-0301-0310.

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Vasculitic neuropathy is an inflammation-driven nerve condition that often goes undiagnosed until irreversible damage occurs. This study developed and validated a supervised machine learning model to predict future onset of vasculitic neuropathy using electronic health record data from 450 cases and 1,800 matched controls. The predictive algorithm analyzed 134 structured features related to diagnoses, medications, lab tests and clinical notes. Selected logistic regression model with L2 regularization achieved an AUC of 0.92 (0.89-0.94 CI) internally, and maintained an AUC of 0.90 (0.84-0.93 CI

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Zhelyazkova, Sashka, and Velina Guergueltcheva. "Multiple mononeuropathy in eosinophilic granulomatosis with polyangiitis – a case report." Bulgarian Neurology 25, no. 1 (2024): 37–40. https://doi.org/10.5281/zenodo.15379848.

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Eosinophilic granulomatosis with polyangiitis (EGPA), formerly known as Churg-Strauss syndrome, is a systemic disease characterised by asthma, pulmonary infiltrates, hypereosinophilia, and systemic vasculitis. Vasculitic neuropathy in EGPA commonly is presented as multiple mononeuropathy but rarely as polyradiculopathy.Central nervous system manifestations are uncommon compared to peripheral nervous system involvement. We present a 49-year-old patient in which EGPA was diagnosed based on the presence of asthma, nasal polyps, chronic pansinusitis, peripheral blood eosinophilia, positive antineu

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Greene, D. "Metabolic vs. Vascular Abnormalities In Diabetic Neuropathy." Journal of the Peripheral Nervous System 5, no. 3 (2000): 172–73. http://dx.doi.org/10.1046/j.1529-8027.2000abstracts-13.x.

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SCHMETTERER, L. "Vascular optic neuropathies versus glaucomatous optic neuropathy." Acta Ophthalmologica 92 (August 20, 2014): 0. http://dx.doi.org/10.1111/j.1755-3768.2014.4722.x.

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Ener, Behçet K., Handan Uçankale, and Reyhan Sürmeli. "Temporal Vascular Changes in Leg with Ulcer Due to Autonomic Neuropathy." International Journal of Angiology 28, no. 03 (2019): 167–72. http://dx.doi.org/10.1055/s-0039-1692662.

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Background Vascular dysfunction dominates the clinical picture of peripheral autonomic neuropathy in lower extremity. Patients and Methods We have studied functional changes of leg vasculature in 30 patients with chronic ulceration due to peripheral autonomic neuropathy between clinical stages 1 and 3. They suffered from lower extremity wounds. After sympathetic skin response test, pedal arterial blood flow analysis including peak systolic velocity (PSV) and pulsatility index (PI) was made by duplex ultrasonography (DUS) in involved legs. Vascular anatomy of leg was also examined by magnetic r

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Shashidhar, Mehta, Kumar Chanchal, Parvez Suhel, and Bhatnagar Suhasini. "Alcohol-Induced Neuropathy: Exploring the Impact on Molecular Channels and Pain Mechanisms." International Journal Of Ayurvedic And Herbal Medicine 15, no. 02 (2025): 4845–60. https://doi.org/10.5281/zenodo.15235359.

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<strong>ABSTRACT: </strong> Neuropathic pain is a severe form of pain that is brought on by dysfunction in the brain and is connected with a number of degenerative alterations in the nervous system. Hyperalgesia and allodynia are two of the main symptoms of pain. Many neurological diseases seen in chronic alcoholics are linked to dietary deficiencies, especially vitamin deficits that are crucial for healthy brain function. The brain may suffer from severe functional impairment and tissue damage, mostly neuronal and vascular, as a result of specific vitamin and nutritional shortages that d

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Pizova, N. V. "Diabetic neuropathy." Medical Council, no. 18 (November 17, 2018): 68–75. http://dx.doi.org/10.21518/2079-701x-2018-18-68-75.

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Diabetes mellitus is one of the emergency situations worldwide. DM is the most common cause of neuropathy throughout the world that create a wide range of conditions associated with the damage of various nerves and various pathological mechanisms. The article considers the main pathogenetic mechanisms of neuropathy - the role of metabolic, vascular and genetic factors. It presents the main clinical manifestations, depending on what type of nerve fibers are affected. A modern classification of diabetic neuropathy is provided. The most common form - diabetic distal symmetric polyneuropathy is co

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Bin Ghouth, Abdullah Salim, Adnan Abdullah Ba-Karman, Hassan Ahmed Alaidroos, Mohammed Hassan Alajely, and Mohsen Hussein Bin-Talib. "Hypertension and Micro-Vascular Complications Among Type 2 Dm Adult Patients in Mukalla, Yemen." Journal of Clinical Research and Reports 3, no. 5 (2020): 01–04. http://dx.doi.org/10.31579/2690-1919/056.

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The Background and Purpose: to assess the prevalence of hypertension and micro-vascular complications among type 2 diabetic patients registered in primary health care center in Mukalla city at eastern Yemen. Methods: A Cross-sectional study was conducted in a randomly selected eligible patients from the diabetic registry of the AL Noor Charity Center (ACC), Mukalla city in Yemen. Results: Prevalence of hypertension in the T2DM patients were 46.7% (56/120). Retinopathy is the most micro-vascular complications among the adults having T2DM (73/120, 60.8%) while nephropathy is the least prevalence

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Goss, James R. "The therapeutic potential of gene transfer for the treatment of peripheral neuropathies." Expert Reviews in Molecular Medicine 9, no. 8 (2007): 1–20. http://dx.doi.org/10.1017/s1462399407000270.

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AbstractPeripheral neuropathy is a common medical problem with numerous aetiologies. Unfortunately, for the majority of cases there is no available medical solution for the underlying cause, and the only option is to try to treat the resulting symptoms. Treatment options exist when neuropathy results in positive symptoms such as pain, but there is a significant lack of treatments for negative symptoms such as numbness and weakness. Systemic application of growth factor peptides has shown promise in protecting nerves from neuropathic insults in preclinical animal studies, but translation into h

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ÖCAL, Halit. "Autoimmune Optic Neuropathy." Güncel Retina Dergisi (Current Retina Journal) 9, no. 1 (2024): 47–53. http://dx.doi.org/10.37783/crj-0436.

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Autoimmune optic neuropathy represents a chronic, heterogeneous disease that is usually accompanied by bilateral vision loss and is caused by injury occurring through autoimmune mechanisms. Autoimmune optic neuropathy can occur as a component of autoimmune or collagen vascular diseases, as well as it can be seen in an isolated way. Plasmapheresis and monoclonal antibodies are used together with steroids in the treatment of autoimmune optic neuropathy, the pathophysiology of which is not yet fully understood despite advances in existing laboratory and imaging devices. This rare clinical picture

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Sanada, M., M. Terada, E. Suzuki, A. Kashiwagi, and H. Yasuda. "MR angiography for the evaluation of non-systemic vasculitic neuropathy: A case report." Acta Radiologica 44, no. 3 (2003): 316–18. http://dx.doi.org/10.1080/j.1600-0455.2003.00072.x.

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Peripheral neuropathy due to vasculitis without any complications of vasculitis in other organs was first reported in 1987. This condition was termed non-systemic vasculitic neuropathy (NSVN). Although vasculitis is believed to develop in small arteries and arterioles in this disease, the level of vascular involvement has not been fully established. We present a case of NSVN followed up by MR angiography, which was thought to be useful to assess the level as well as the state of vascular lesions in this condition.

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Mori, Hideki. "Early detection and intervention in diabetic gastroparesis: Role of body surface gastric mapping." World Journal of Gastroenterology 30, no. 45 (2024): 4836–38. http://dx.doi.org/10.3748/wjg.v30.i45.4836.

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Diabetic gastrointestinal neuropathy is a diabetes-related complication, associated with a complex interplay of hyperglycemic damage, autoimmune responses, oxidative stress, gastrointestinal hormones, and vascular insufficiency. Patients with diabetes should be monitored and therapeutic intervention introduced to prevent neuropathy due to diabetes prior to “the point of no return”. Determining gastric bioelectrical activity by body surface gastric mapping may be a promising option to monitor diabetic gastrointestinal neuropathy.

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Patel, Hershel R., and Curtis E. Margo. "Pathology of Ischemic Optic Neuropathy." Archives of Pathology & Laboratory Medicine 141, no. 1 (2017): 162–66. http://dx.doi.org/10.5858/arpa.2016-0027-rs.

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Ischemic optic neuropathy (ION) describes a state of hypoxic injury of the optic nerve. Clinically, ION is divided into anterior and posterior forms defined by the presence or absence of optic disc swelling, respectively. It is further classified as arteritic when secondary to vasculitis, and nonarteritic when not. The site of vascular occlusion for anterior ION from giant cell arteritis is the short posterior ciliary arteries, but mechanical vascular obstruction does not play a role in most nonarteritic cases. Histologically, ION is characterized by axon and glial necrosis, edema, and a spars

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ÜKE UZUN, Saniye, and Semra TİRYAKİ DEMİR. "Posterior Ischemic Optic Neuropathy." Güncel Retina Dergisi (Current Retina Journal) 9, no. 1 (2024): 24–32. http://dx.doi.org/10.37783/crj-0433.

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Posterior ischemic optic neuropathy (PION) is one of the rare optic neuropathies that occurs as a result of hypoperfusion of the optic nerve posterior to the optic nerve head. It usually causes acute, painless vision loss, and no features are detected at the optic nerve head during fundus examination. PION is a multifactorial disease that combines vascular and local risk factors, develops on the basis of various systemic diseases, and is accompanied by inadequate autoregulation in the optic nerve. Etiologically; it is divided into three subtypes: nonarteritic PION, which develops on the basis

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Dr., Ayesha Tahsin Shaikh Dr. Rabia Sajjad Dr. Ayesha Shahid. "ANALYSIS OF GLYCEMIC CONTROL AND MICRO-VASCULAR COMPLICATIONS IN TYPE 2 DIABETES AND CONTROL OF GLUCOSE." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 08 (2018): 7998–8001. https://doi.org/10.5281/zenodo.1404266.

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<strong><em>Introduction: </em></strong><em>Diabetes Mellitus (DM) is a chronic disorder characterized by impaired metabolism of glucose and other energy yielding fuels as well as by the late development of vascular and neuropathic complications. <strong>Objectives of the study: </strong>The main objective of this study is to analyze the role of glycemic control and micro-vascular complications in Type 2 diabetes and control of glucose.<strong> Methodology of the study: </strong>This study was conducted at Rafique Anwer Memorial Trust Hospital, Gujranwala. This study was conducted with the per

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Van Gompel, Jamie J., Christoph J. Griessenauer, Bernd W. Scheithauer, Kimberly K. Amrami, and Robert J. Spinner. "Vascular Malformations, Rare Causes of Sciatic Neuropathy: A Case Series." Neurosurgery 67, no. 4 (2010): 1133–42. http://dx.doi.org/10.1227/neu.0b013e3181ecc84e.

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Abstract BACKGROUND: Sciatica is typically a clear-cut symptom complex commonly related to an impingement at the spinal nerve level. Etiologies of sciatic neuropathy outside the neural foramina are uncommon. OBJECTIVE: To describe 4 patients presenting with radiating leg pain due to sciatic nerve involvement, all with a vascular etiology. METHODS: Four patients presenting with neuropathic pain were retrospectively reviewed. Preoperative 3 Tesla magnetic resonance imaging was used to identify these lesions, which most commonly showed diffuse T2 changes with nerve enhancement upon administration

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Thomas, P. K. "Diabetic Neuropathy: Models, Mechanisms and Mayhem." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 19, no. 1 (1992): 1–7. http://dx.doi.org/10.1017/s0317167100042463.

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ABSTRACT:Rational treatment of diabetic polyneuropathy depends upon establishing its cause, which is at present unknown. A number of animal models of diabetes have been examined and although abnormalities are detectable in the peripheral nervous system they do not duplicate the degenerative neuropathy encountered in the human. The relevance of these abnormalities is therefore uncertain, although they may reflect the earlier changes in man. For human neuropathy, it is likely that vascular lesions or an abnormal susceptibility to mechanical injury are responsible for focal neuropathies. The evid

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Musial, Diego Castro, Maria Eduarda Ajita, and Guilherme Henrique Souza Bomfim. "Benefits of Cilostazol’s Effect on Vascular and Neuropathic Complications Caused by Diabetes." Medical Sciences 13, no. 1 (2024): 1. https://doi.org/10.3390/medsci13010001.

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Diabetes mellitus (DM) is a global health concern with a rising incidence, particularly in aging populations and those with a genetic predisposition. Over time, DM contributes to various complications, including nephropathy, retinopathy, peripheral arterial disease (PAD), and neuropathy. Among these, diabetic neuropathy and PAD stand out due to their high prevalence and significant impact on patients’ quality of life. Diabetic distal symmetric polyneuropathy, the most common form of diabetic neuropathy, is driven by neuroinflammation stemming from prolonged hyperglycemia. Simultaneously, hyper

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Battula, Pradeep, S. Afreen, E. Meena, S. Siva Ram Reddy, and G. Sujatha. "Prevalence of sensory peripheral neuropathy in diabetic patients at diabetes care centre: a cross sectional study." International Journal of Research in Medical Sciences 5, no. 9 (2017): 4066. http://dx.doi.org/10.18203/2320-6012.ijrms20173984.

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Background: Diabetic neuropathy is a nerve damaging disorder associated with diabetes; result from micro vascular injury involving small blood vessels that supply to the nerve (vas nervorum) in addition to macro vascular condition that can culminate in diabetic neuropathy. By the year 2025 hundreds of people were estimated to become diabetic. The rates of prevalence of neuropathy increasing worldwide which is directly related to the nonmodifiable risks like age, duration of diabetes, obesity, alcoholism, gender.Methods: A cross sectional interventional study was conducted on diabetic patients.

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Kim, Mun Gyu, Youn Sil Jang, Keun Her, and Chang Bong Lee. "Ultrasound-Guided Nerve Hydrodissection for Neuropathic-Like Pain Arising from Hemodialysis: A Case Report." Soonchunhyang Medical Science 28, no. 2 (2022): 155–58. http://dx.doi.org/10.15746/sms.22.032.

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Peripheral neuropathy is very common in patients with chronic renal failure. The pain arising from hemodialysis can be caused by vascular problems (such as vascular stenosis and steal syndrome) and neuropathy. Hemodialysis patients who need to be dialyzed three times a week should not be told to endure worsening pain every time they are dialyzed. We report that the pain arising from hemodialysis in the lower arm was a concern due to the nerve entrapment in the axillary area, and it was successfully controlled with ultrasound-guided nerve hydrodissection.

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Jadhav, Ghanshyam B., Shubham J. Khairnar, Pavan B. Udavant, Rahul R. Sable, and Krishna N. Mundlod. "Evaluation of Protective Effect of Gemigliptin and Rizatriptan in Streptozotocin induced Diabetic Neuropathy in Rats." Biosciences Biotechnology Research Asia 21, no. 1 (2024): 147–56. http://dx.doi.org/10.13005/bbra/3210.

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ABSTRACT: A typical micro-vascular consequence of diabetes mellitus is diabetic neuropathy. The prevalence of diabetic neuropathy patients is rising in spite of strict glycemic control, blood pressure, and lipid lowering medication. New prevention and treatment methods are required because of the drawbacks and side effects of current medicines. Serotonin, a neurotransmitter implicated in the transmission of pain, is being investigated for its potential to process pain and to reduce inflammatory responses. Gemigliptin and rizatriptan are being studied for the treatment of hyperglycemic mortalit

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Taïb, Sonia, Juliette Durand, Vianney Dehais, et al. "Vascular dysfunction is at the onset of oxaliplatin-induced peripheral neuropathy symptoms in mice." Life Science Alliance 8, no. 2 (2024): e202402791. http://dx.doi.org/10.26508/lsa.202402791.

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Oxaliplatin-induced peripheral neuropathy (OIPN) is an adverse side effect of this chemotherapy used for gastrointestinal cancers. The continuous pain experienced by OIPN patients often results in the reduction or discontinuation of chemotherapy, thereby affecting patient survival. Several pathogenic mechanisms involving sensory neurons were shown to participate in the occurrence of OIPN symptoms. However, the dysfunction of the blood-nerve barrier as a source of nerve alteration had not been thoroughly explored. To characterise the vascular contribution to OIPN symptoms, we undertook two comp

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Cameron, N. E., and M. A. Cotter. "Vascular changes in animal models of diabetic neuropathy." Journal of Neurochemistry 85 (May 8, 2003): 14. http://dx.doi.org/10.1046/j.1471-4159.85.s2.14_3.x.

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Dotan, Gad, and Amos D. Korczyn. "Nonarteritic Ischemic Optic Neuropathy and Other Vascular Diseases." Neuroepidemiology 40, no. 3 (2013): 225–26. http://dx.doi.org/10.1159/000343960.

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Miyamoto, Wataru, Soichiro Yamamoto, Ryuta Kii, and Yuji Uchio. "Vascular Leiomyoma Resulting in Ulnar Neuropathy: Case Report." Journal of Hand Surgery 33, no. 10 (2008): 1868–70. http://dx.doi.org/10.1016/j.jhsa.2008.07.008.

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Thomas, P. K. "Vascular factors in the causation of diabetic neuropathy." Trends in Neurosciences 10, no. 1 (1987): 6–8. http://dx.doi.org/10.1016/0166-2236(87)90114-7.

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Eidelman, B. H., V. K. Nielsen, M. Moller, and P. J. Jannetta. "Vascular compression, hemifacial spasm, and multiple cranial neuropathy." Neurology 35, no. 5 (1985): 712. http://dx.doi.org/10.1212/wnl.35.5.712.

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