Bibliographies thématiques / Venous Outflow Obstruction

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Littérature scientifique sur le sujet « Venous Outflow Obstruction »

Auteur : Grafiati
Publié le 7 juin 2025
Mis à jour le 2 août 2025

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Articles de revues sur le sujet "Venous Outflow Obstruction"

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Åkesson, Henrik, Lars Brudin, Ragnar Jensen, Per Ohlin, and Gunnar Plate. "Pysiological Evaluation of Venous Obstruction in the Post-Thrombotic Leg." Phlebology: The Journal of Venous Disease 4, no. 1 (1989): 3–14. http://dx.doi.org/10.1177/026835558900400103.

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The accuracy and value of occlusion plethysmography (OP) in assessing post-thrombotic iliac and femoral vein obstruction was determined in 45 patients (85 legs) six months after an acute iliofemoral venous thromboses using contrast phlebography (CP) as reference method. The additional value of femoral venous pressure (FVP) measurements in assessing the physiological importance of iliac vein obstructions was determined in 34 of these patients (60 legs). The sensitivity and specificity of OP in detecting femoral and iliac vein obstructions was 79% and 84% respectively. OP was unable to distingui
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Neglén, Peter. "Chronic Venous Obstruction: Diagnostic Considerations and Therapeutic Role of Percutaneous Iliac Stenting." Vascular 15, no. 5 (2007): 273–80. http://dx.doi.org/10.2310/6670.2007.00071.

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Iliac venous outflow obstruction has an important role in the expression of symptomatic chronic venous insufficiency. This anatomic obstruction is frequently overlooked, owing in part to diagnostic difficulty. The combination of venous obstruction and reflux leads to more severe clinical disease. Current diagnostic modalities do not allow an definitive assessment of hemodynamically critical venous obstruction. No single invasive or noninvasive study can accurately detect borderline obstruction of potential hemodynamic significance. A high index of suspicion is critical in the initial recogniti
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Labovitz, Jonathan, Paul Gagne, Keith Penera, and Sandra Wainwright. "Nonhealing Venous Ulcers and Chronic Venous Outflow Obstruction." Journal of the American Podiatric Medical Association 105, no. 6 (2015): 541–49. http://dx.doi.org/10.7547/14-075.1.

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The etiology of chronic venous insufficiency is typically neglected or misunderstood when treating lower-extremity edema and venous ulcerations. Despite the high prevalence of venous compression syndromes, it is rarely considered when treating venous ulcers and unresolved venous disease. We report a case of bilateral iliac vein outflow obstruction that prohibited venous ulcer healing until properly treated. This case highlights the importance of properly identifying and treating venous compression syndromes to enhance ulcer healing and decrease the risk of venous ulcer recurrence.
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Lichtenberg, Michael, Rick de Graaf, and Christian Erbel. "Standards for recanalisation of chronic venous outflow obstructions." Vasa 47, no. 4 (2018): 259–66. http://dx.doi.org/10.1024/0301-1526/a000696.

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Abstract. Postthrombotic syndrome (PTS) is the most common complication after iliofemoral deep vein thrombosis. It reduces quality of life and increases deep vein thrombosis (DVT)-related costs. The clinical symptoms and severity of PTS may vary; the most common symptoms include edema, pain (venous claudication), hyperpigmentation, lipodermatosclerosis, and ulceration. PTS is based on the principle of outflow obstruction, which may be caused by venous hypertension and may lead to valvular damage and venous reflux or insufficiency. Recent technical developments and new stent techniques now allo
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Kohli, V., G. K. Pande, K. S. Reddy, S. Nundy, V. Dev, and U. Kaul. "Management of hepatic venous outflow obstruction." Lancet 342, no. 8873 (1993): 718–22. http://dx.doi.org/10.1016/0140-6736(93)91712-u.

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Mohammed Osman, Ashraf Mustafa, Mohammed Al Fadil Gar al naby, Asma Ibrahim Ahmed, and Babiker Abd Elwahab Awad alla. "SONOGRAPHIC EVALUATION OF OUTFLOW VEINS IN SUDANESE LIVER TRANSPLANTS." International Journal of Research -GRANTHAALAYAH 6, no. 12 (2018): 77–83. http://dx.doi.org/10.29121/granthaalayah.v6.i12.2018.1079.

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The purpose of this study was to identify the specific Doppler criteria for the outflow vein (hepatic veins and inferior vena cava) obstruction in liver transplants. A case control study was done after performing venous Doppler sonographic studies in 300 normal (control) and 45 liver transplant cases (4 whole liver, 41 lobar) with no vascular obstruction. The ultrasonic Doppler study were classified as normal, occluded, or stenosed on the basis of gray scale and color flow mapping appearances as well as elevated or absent waves. The following Doppler parameters were evaluated: the outflow vein
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Ashraf, Mustafa Mohammed Osman, Dr. Mohammed Al Fadil Gar al naby Professor., Asma Ibrahim Ahmed Dr., and Babiker Abd Elwahab Awad alla Dr. "SONOGRAPHIC EVALUATION OF OUTFLOW VEINS IN SUDANESE LIVER TRANSPLANTS." International Journal of Research - Granthaalayah 6, no. 12 (2018): 77–83. https://doi.org/10.5281/zenodo.2528461.

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The purpose of this study was to identify the specific Doppler criteria for the outflow vein (hepatic veins and inferior vena cava) obstruction in liver transplants. A case control study was done after performing venous Doppler sonographic studies in 300 normal (control) and 45 liver transplant cases (4 whole liver, 41 lobar) with no vascular obstruction. The ultrasonic Doppler study were classified as normal, occluded, or stenosed on the basis of gray scale and color flow mapping appearances as well as elevated or absent waves. The following Doppler parameters were evaluated: the outflow vein
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Dashti, Shervin R., Peter Nakaji, Yin C. Hu, et al. "Styloidogenic Jugular Venous Compression Syndrome: Diagnosis and Treatment: Case Report." Neurosurgery 70, no. 3 (2011): 783. http://dx.doi.org/10.1227/neu.0b013e3182333859.

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Abstract Background and Importance: Intracranial venous hypertension is known to be associated with venous outflow obstruction. We discuss the diagnosis and treatment of mechanical venous outflow obstruction causing pseudotumor cerebri. Clinical Presentation: We report 2 patients presenting with central venous outflow obstruction secondary to osseous compression of the internal jugular veins at the craniocervical junction. The point of jugular compression was between the lateral tubercle of C1 and a prominent, posteriorly located styloid process. In both cases, catheter venography showed high-
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Higgins, J. Nicholas P., Neil G. Burnet, Christian F. Schwindack, and Alan Waters. "Severe brain edema caused by a meningioma obstructing cerebral venous outflow and treated with venous sinus stenting." Journal of Neurosurgery 108, no. 2 (2008): 372–76. http://dx.doi.org/10.3171/jns/2008/108/2/0372.

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✓ Peritumoral edema is a common feature of cerebral meningiomas, but venous outflow obstruction is not normally considered as a cause even when a tumor involves the venous sinuses. The authors describe a patient with a ventricu-loperitoneal shunt in situ, who had undergone several debulking operations and conventional radiotherapy and in whom had developed progressive life-threatening edema from a tentorial meningioma. Radiological studies showed occlusion of the straight sinus and stenosis of the sagittal sinus at the confluence of sinuses. The sagittal sinus was stented, and thereafter the p
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Kollar, Christopher, Geoffrey Parker, and Ian Johnston. "The endovascular treatment of cranial venous sinus obstruction resulting in pseudotumor syndrome." Journal of Neurosurgery 94, no. 4 (2001): 646–51. http://dx.doi.org/10.3171/jns.2001.94.4.0646.

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✓ It is probable that a significant number of cases of pseudotumor syndrome (PTS) occur because of cranial venous outflow obstruction, yet reports of direct treatment of the obstruction are few and inconclusive. In this study the authors report three cases of PTS with angiographically confirmed venous sinus obstruction treated by direct, endovascular procedures; urokinase infusion in two and balloon venoplasty in one. Two patients suffered transient complications that resolved satisfactorily. All three showed initial resolution of the signs and symptoms of PTS but one relapsed after 8 months a
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Thèses sur le sujet "Venous Outflow Obstruction"

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Tanner, Daniel Edward. "Design, analysis, testing, and evaluation of a prosthetic venous valve." Thesis, Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/51758.

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Chronic Venous Insufficiency (CVI) is characterized by chronic venous hypertension from blood pooling in the lower limbs. The resulting symptoms include leg pain, varicose veins, fatigue, venous edema, skin pigmentation, inflammation, induration, and ulceration. Reflux from incompetent venous valves is a factor in up to 94% of individuals with CVI. Current treatments of CVI include compression stockings, drug therapy, vein disabling, venous stenting, and surgical correction with varying rates of success. However, a minimally invasive correction of deep venous reflux does not currently exist. A
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Yabuta, Minoru. "Long-term Outcome of Percutaneous Interventions for Hepatic Venous Outflow Obstruction after Pediatric Living Donor Liver Transplantation: Experience from a Single Institute." Kyoto University, 2015. http://hdl.handle.net/2433/199187.

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Livres sur le sujet "Venous Outflow Obstruction"

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Parikh, Roshni A., and David M. Williams. Managing Chronic Iliac Venous Occlusions That Extend Below the Inguinal Ligament. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0034.

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This chapter describes the management, applications, challenges, and potential complications when venous occlusions extend below the inguinal ligament. Recanalization of a chronic iliocaval occlusion in combination with anticoagulation can significantly improve a patient’s quality of life. The success of treating iliocaval venous obstruction, however, depends on good venous inflow. Without adequate venous inflow, the outflow stents will fail. Evaluation of the saphenofemoral junction, femoral vein confluence, and/or saphenous vein, recanalization of the occluded segments, and extension of the
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Chapitres de livres sur le sujet "Venous Outflow Obstruction"

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Bayraktar, Yusuf. "Hepatic Venous Outflow Obstruction." In Molecular Pathology Library. Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7107-4_48.

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Rathi, Sahaj, Akash Roy, and Virendra Singh. "Hepatic Venous Outflow Tract Obstruction." In Clinical Rounds in Hepatology. Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-8448-7_16.

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Owler, B. K., G. Parker, G. M. Halmagyi, et al. "Cranial Venous Outflow Obstruction and Pseudotumor Cerebri Syndrome." In Advances and Technical Standards in Neurosurgery. Springer Vienna, 2005. http://dx.doi.org/10.1007/3-211-27208-9_4.

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Valla, Dominique-Charles. "Budd-Chiari Syndrome - Hepatic Venous Outflow Tract Obstruction." In Portal Hypertension V. Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444393989.ch13.

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Hernández-Gea, Virginia, Anna Baiges, Fanny Turon, and Juan Carlos Garcia-Pagan. "Budd-Chiari Syndrome: Hepatic Venous Outflow Tract Obstruction." In Vascular Disorders of the Liver. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-82988-9_6.

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Valla, Dominique-Charles. "Budd-Chiari Syndrome/Hepatic Venous Outflow Tract Obstruction: East vs West." In Portal Hypertension VI. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23018-4_39.

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"Hepatic Venous Outflow Obstruction." In Diagnostic Pathology: Hepatobiliary and Pancreas. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-323-44307-4.50067-1.

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Kalra, Naveen, and Niranjan Khandelwal. "Hepatic Venous Outflow Tract Obstruction." In AIIMS-MAMC-PGI’s Comprehensive Textbook of Diagnostic Radiology (3 Volumes). Jaypee Brothers Medical Publishers (P) Ltd., 2017. http://dx.doi.org/10.5005/jp/books/12990_88.

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Kalra, Naveen, and Niranjan Khandelwal. "Hepatic Venous outflow tract Obstruction." In Diagnostic Radiology: Gastrointestinal and Hepatobiliary Imaging. Jaypee Brothers Medical Publishers (P) Ltd., 2009. http://dx.doi.org/10.5005/jp/books/10213_21.

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Khan, Mohammad Qasim, and Patrick S. Kamath. "Extrahepatic portal venous obstruction, idiopathic non-cirrhotic portal fibrosis, and hepatic venous outflow tract obstruction." In Liver, Gall Bladder, and Bile Ducts, edited by Mohamed Rela, Pierre-Alain Clavien, Samiran Nundy, and Dirk J. Gouma. Oxford University PressOxford, 2023. http://dx.doi.org/10.1093/med/9780192862457.003.0012.

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Abstract While portal hypertension commonly arises secondary to cirrhosis, it can also present in its absence, termed non-cirrhotic portal hypertension. This group of disorders is classified into prehepatic, intrahepatic and post-hepatic aetiologies based on the level of involvement. Prehepatic aetiologies involve disruption of the portal venous or splenic venous systems. Intrahepatic aetiologies may be presinusoidal, sinusoidal, or postsinusoidal. Post-hepatic aetiologies of portal hypertension comprise disruption of the hepatic venous outflow from the level of the hepatic venules to the hear
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Actes de conférences sur le sujet "Venous Outflow Obstruction"

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Hepworth, Edward J., Alison V. Love, and Rebekah L. Hartford. "Intracranial Hypertension Secondary to Venous Outflow Obstruction." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725347.

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Greeneway, G., J. West, R. Garner, et al. "O-034 Correlation between angiographic stenosis and physiologic venous sinus outflow obstruction in idiopathic intracranial hypertension." In SNIS 15TH ANNUAL MEETING, July 23–26, 2018, Hilton San Francisco Union Square San Francisco, CA. BMJ Publishing Group Ltd., 2018. http://dx.doi.org/10.1136/neurintsurg-2018-snis.34.

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Huisman, M. V., H. R. Buller, and J. W. ten Cate. "RATE OF NORMALIZATION OF ABNORMAL IMPEDANCE PLETHYSMOGRAPHY IN PATIENTS WITH PROVEN DEEP VEIN THROMBOSIS: SIGNIFICANCE IN THE MANAGEMENT OF RECURRENT SYMPTOMS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642894.

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The diagnosis of deep vein thrombosis (DVT) by clinical signs and symptoms is unreliable, but contrast venography is expensive and invasive. Therefore, the use of non-invasive methods to detect DVT have become en vogue, of which impedance plethysmography (IPG), either in combination with 125x fibrinogen leg scanning or performed serially as a single test, have been demonstrated to be a safe and effective alternative. Since the principle of IPG is based on the measurement of venous outflow obstruction due to intravascular thrombus and since the aim of anticoagulant treatment is to facilitate re
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