Bibliografie tematiche / AIDS dementia complex - Pathogenesis

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Letteratura scientifica selezionata sul tema "AIDS dementia complex - Pathogenesis"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 6 febbraio 2022

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Articoli di riviste sul tema "AIDS dementia complex - Pathogenesis"

1

Dal Canto, M. C. "AIDS-Dementia-Complex: pathology, pathogenesis and future directions." Italian Journal of Neurological Sciences 10, no. 3 (June 1989): 277–87. http://dx.doi.org/10.1007/bf02333773.

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2

Berman, N. E. J., J. K. Johnson, L. Raymond, E. B. Stephens, S. Joag, and O. Narayan. "Pathogenesis of the aids dementia complex in the SIV model." European Neuropsychopharmacology 6 (June 1996): 22. http://dx.doi.org/10.1016/0924-977x(96)87400-1.

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3

McCaddon, A., B. Regland, and C. F. Fear. "Trypsin inhibition: A potential cause of cobalamin deficiency common to the pathogenesis of Alzheimer-type dementia and AIDS dementia complex?" Medical Hypotheses 45, no. 2 (August 1995): 200–204. http://dx.doi.org/10.1016/0306-9877(95)90069-1.

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4

Krivine, Anne, Gilles Force, Jerome Servan, Anne-Elisabeth Cabée, Flore Rozenberg, Lucia Dighiero, Françoise Marguet та Pierre Lebon. "Measuring HIV-1 RNA and interferon-α in the cerebrospinal fluid of AIDS patients: insights into the pathogenesis of AIDS Dementia Complex". Journal of Neurovirology 5, № 5 (січень 1999): 500–506. http://dx.doi.org/10.3109/13550289909045379.

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5

Cunningham, A. L., H. Naif, N. Saksena, G. Lynch, J. Chang, S. Li, R. Jozwiak, et al. "HIV infection of macrophages and pathogenesis of AIDS dementia complex: interaction of the host cell and viral genotype." Journal of Leukocyte Biology 62, no. 1 (July 1997): 117–25. http://dx.doi.org/10.1002/jlb.62.1.117.

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6

Perrella, O., M. Guerriero, E. Izzo, M. Soscia, and P. B. Carrieri. "Interleukin-6 and granulocyte macrophage-csf in the cerebrospinal fluid from hiv infected subjects with involvement of the central nervous system." Arquivos de Neuro-Psiquiatria 50, no. 2 (June 1992): 180–82. http://dx.doi.org/10.1590/s0004-282x1992000200008.

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Abstract (sommario):
We detected the cytokines interleukin-6 (IL-6) and granulocyte macrophage-CSF (GM-CSF) by ELISA in the CSF and serum of 30 HIV-infected patients classified as AIDS dementia complex (ADC), and 20 subjects with other neurological diseases (OND). We have found a high incidence of detectable IL-6 and GM-CSF in the CSF of ADC patients compared with OND patients. No statistical differences were observed between both groups for serum IL-6 and GM-CSF levels. These results suggest an intrathecal synthesis of these cytokines and a possible involvement in the pathogenesis of ADC.
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7

Price, Richard W. "The AIDS dementia complex as a model for other neurodegenerative diseases: a pathogenetic conceit." Journal of the Neurological Sciences 127, no. 1 (December 1994): 6–7. http://dx.doi.org/10.1016/0022-510x(94)90120-1.

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8

A.L. Sidelkovsky, P.A. Fedorov, V.V. Marusichenko, and M.R. Ignatischev. "Damage to the nervous system associated with HIV infection (a clinical case)." INTERNATIONAL NEUROLOGICAL JOURNAL 16, no. 8 (March 10, 2021): 53–56. http://dx.doi.org/10.22141/2224-0713.16.8.2020.221963.

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Abstract (sommario):
In Eastern Europe, at least 130 thousand new cases of HIV infection have been registered, which undoubtedly reflects the urgency of this medical problem. In our country, the average rate of human immunodeficiency infection is 58 cases per 100 thousand people. It is known that the disease is caused by an RNA-containing human immunodeficiency virus. Two types of it have been studied — HIV-1 and HIV-2, which have many subtypes. An important clinical feature of this virus is its tropism to cells of the human nervous and immune systems. The main risk group for the disease is injecting drug users, b
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9

Lim, Chai K., Bruce J. Brew, Gayathri Sundaram, and Gilles J. Guillemin. "Understanding the Roles of the Kynurenine Pathway in Multiple Sclerosis Progression." International Journal of Tryptophan Research 3 (January 2010): IJTR.S4294. http://dx.doi.org/10.4137/ijtr.s4294.

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Abstract (sommario):
The kynurenine pathway (KP) is a major degradative pathway of tryptophan ultimately leading to the production of nicotinamide adenine dinucleotide (NAD+) and is also one of the major regulatory mechanisms of the immune response. The KP is known to be involved in several neuroinflammatory disorders including Alzheimer's disease, amyotrophic lateral sclerosis, AIDS dementia complex, Parkinson's disease, schizophrenia, Huntington's disease and brain tumours. However, the KP remains a relatively new topic for the field of multiple sclerosis (MS). Over the last 2–3 years, some evidence has progress
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10

Talley, A. K., S. Dewhurst, S. W. Perry, S. C. Dollard, S. Gummuluru, S. M. Fine, D. New, L. G. Epstein, H. E. Gendelman, and H. A. Gelbard. "Tumor necrosis factor alpha-induced apoptosis in human neuronal cells: protection by the antioxidant N-acetylcysteine and the genes bcl-2 and crmA." Molecular and Cellular Biology 15, no. 5 (May 1995): 2359–66. http://dx.doi.org/10.1128/mcb.15.5.2359.

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Abstract (sommario):
Tumor necrosis factor alpha (TNF-alpha) is a candidate human immunodeficiency virus type 1-induced neurotoxin that contributes to the pathogenesis of AIDS dementia complex. We report here on the effects of exogenous TNF-alpha on SK-N-MC human neuroblastoma cells differentiated to a neuronal phenotype with retinoic acid, TNF-alpha caused a dose-dependent loss of viability and a corresponding increase in apoptosis in differentiated SK-N-MC cells but not in undifferentiated cultures. Importantly, intracellular signalling via TNF receptors, as measured by activation of the transcription factor NF-
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Tesi sul tema "AIDS dementia complex - Pathogenesis"

1

Cysique, Lucette Adeline Juliette St Vincent's Hospital UNSW. "Aids dementia complex in the era of highly active antiretroviral therapy: a neuropsychological study." Awarded by:University of New South Wales. St. Vincent's Hospital, 2005. http://handle.unsw.edu.au/1959.4/22074.

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Abstract (sommario):
The aim of the thesis was to undertake an evaluation of the neuropsychological functioning of non-demented and demented patients with advanced HIV-infection who have been treated with Highly Active Antiretroviral Therapy (HAART) for several years. One hundred and one non-demented HIV-infected individuals and 23 patients with mild or moderate AIDS Dementia Complex (ADC), from the outpatient clinics and Neurology department at St. Vincent's Hospital, Sydney, Australia were randomly selected to participate in a prospective study of the neurological and neuropsychological complications of HIV dise
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2

Owe-Young, Robert School of Medicine UNSW. "Kynurenine pathway metabolism at the blood-brain barrier." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/26183.

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Abstract (sommario):
A major product of HIV-infected and cytokine-stimulated monocytic-lineage cells is quinolinic acid (QUIN), a neurotoxic metabolite of the kynurenine pathway (KP) of L-tryptophan (L-Trp) metabolism. Despite the large number of neurotoxins found in HIV patients with AIDS Dementia Complex (ADC), only QUIN correlates with both the presence and severity of ADC. With treatment, cerebrospinal fluid (CSF) QUIN concentrations decrease proportionate to the degree of clinical and neuropsychological improvement. As endothelial cells (EC) of the blood-brain barrier (BBB) are the first brain-associated cell
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3

Di, Stefano Mariantonietta. "Molecular dynamics in HIV-1 infection of the brain /." Stockholm : Karolinska institutet, 1997. http://diss.kib.ki.se/1997/91-85910-65-1.

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Lovec, Theobald Rhonda. "A review of pharmacological and psychosocial management of AIDS dementia complex." Honors in the Major Thesis, University of Central Florida, 1999. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/71.

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Abstract (sommario):
This item is only available in print in the UCF Libraries. If this is your Honors Thesis, you can help us make it available online for use by researchers around the world by following the instructions on the distribution consent form at http://library.ucf.edu/Systems/DigitalInitiatives/DigitalCollections/InternetDistributionConsentAgreementForm.pdf You may also contact the project coordinator, Kerri Bottorff, at kerri.bottorff@ucf.edu for more information.<br>Bachelors<br>Health and Public Affairs<br>Nursing
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Sabri, Farideh. "Astrocytes during HIV infection of the brain : relevance for neuropathogenesis /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4536-5/.

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Anderson, Deborah E. (Deborah Elaine) 1967. "HIV-Associated Dementia: Cofactors as Predictors of Severity of Neurocoenitive Deficits." Thesis, University of North Texas, 1996. https://digital.library.unt.edu/ark:/67531/metadc277756/.

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Abstract (sommario):
The objective of the present study was to evaluate the relationship between a set of cofactors and severity of cognitive impairment, to determine if there were any factors which significantly predicted more severe neurocognitive deficits in persons with AIDS. Twenty-four male volunteers recruited from community groups and physician referrals participated. Subjects completed several self-report questionnaires eliciting information regarding demographics and risk factor variables, in addition to a comprehensive battery of neuropsychological tests. A severity of cognitive impairment summary score
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7

Bam, Isabel M. S. "'N Ondersoekende kwalitatiewe studie na die siektenarratiewe van individue met VIGS-demensiekompleks." Diss., Pretoria : [s.n.], 2003. http://upetd.up.ac.za/thesis/available/etd-02092005-091416.

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Zheve, Georgina Teurai. "Neuroprotective mechanisms of nevirapine and efavirenz in a model of neurodegeneration." Thesis, Rhodes University, 2008. http://hdl.handle.net/10962/d1003285.

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Abstract (sommario):
AIDS Dementia Complex (ADC) is a neurodegenerative disorder implicated in HIV-1 infection that is associated with elevated levels of the neurotoxin, quinolinic acid (QA) which causes a cascade of events to occur, leading to the production of reactive oxygen species (ROS), these being ultimately responsible for oxidative neurotoxicity. In clinical studies, Non-nucleoside reverse transcriptase inhibitors (NNRTIs), efavirenz (EFV) and nevirapine (NVP) have been shown to potentially delay the progressive degeneration of neurons, thus reducing the frequency and neurological deficits associated with
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9

Jasmina, Boban. "Multivokselska magnetno-rezonantna spektroskopija mozga kod HIV+ pacijenata." Phd thesis, Univerzitet u Novom Sadu, Medicinski fakultet u Novom Sadu, 2016. http://www.cris.uns.ac.rs/record.jsf?recordId=101759&source=NDLTD&language=en.

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Abstract (sommario):
UVOD: Neurokognitivni poremećaj udružen sa HIVinfekcijom (HIV associated neurocognitive disorder- HAND) javlja se u oko polovine pacijenata sa HIV-om. HAND obuhvata spektar neurolo&scaron;kih poremećaja koji variraju od asimptomatskog neurokognitivnog poremećaja(ANI), preko blagog kognitivnog o&scaron;tećenja, koje se naziva blagi kognitivni poremećaj (MND) do demencije udružene sa HIV-om (HAD). U evaluaciji i dijagnostici ovog poremećaja koriste se razne laboratorijske, kliničke i metode imidžinga, među kojima magnetno-rezonantni imidžing (MRI) zauzima posebno mesto u pogledu dijagnostike str
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Fouche, Jean-Paul. "A diffusion tensor imaging study in HIV patients with and without apathy." Thesis, Stellenbosch : University of Stellenbosch, 2010. http://hdl.handle.net/10019.1/5146.

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Abstract (sommario):
Thesis (MScMedSc (Biomedical Sciences. Medical Physiology))--University of Stellenbosch, 2010.<br>ENGLISH ABSTRACT: HIV/AIDS is a global epidemic that accounts for a large percentage of the mortality in South Africa every year. Since the implementation of anti-retroviral treatment, HIV positive individuals have been living longer, and the cognitive impairment associated with the disease is becoming increasingly apparent. During the initial systemic infection of HIV, the virus migrates through the blood-brain barrier and inflicts axonal injury by causing upregulation of cytokines and neuro
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Libri sul tema "AIDS dementia complex - Pathogenesis"

1

C, McArthur Justin, ed. AIDS and neurology. Edinburgh: Churchill Livingstone, 1995.

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2

1955-, Leary Mark, and Boccellari Alicia A. 1955-, eds. AIDS and the impact of cognitive impairment: A treatment guide for mental health providers. San Francisco, CA: AIDS Health Project, University of California San Francisco, 1995.

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3

1941-, Price Richard W., and Sidtis John J, eds. The cellular basis of central nervous system HIV-1 infection and the AIDS dementia complex. New York: Haworth Medical Press, 1996.

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G, Van Gorp W., and Buckingham Stephan L, eds. Practitioner's guide to the neuropsychiatry of HIV/AIDS. New York: Guilford Press, 1998.

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5

1941-, Price Richard W., Perry Samuel 1940-, and Association for Research in Nervous and Mental Diseases. Meeting, eds. HIV, AIDS, and the brain. New York: Raven Press, 1994.

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6

Alireza, M.D. Minagar (Editor) and Paul Shapshak (Editor), eds. Neuro-AIDS. Nova Biomedical Books, 2006.

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7

Price, Richard W., and John J. Sidtis. The Cellular Basis of Central Nervous System Hiv-1 Infection and the AIDS Dementia Complex. Haworth Press, 1995.

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(Editor), Richard W. Price, and John J. Sidtis (Editor), eds. The Cellular Basis of Central Nervous System Hiv-1 Infection And the AIDS Dementia Complex. Haworth Pr Inc, 1995.

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9

Price, Richard W. The Cellular Basis of Central Nervous System HIV-1 Infection and the AIDS Dementia Complex. Routledge, 2014. http://dx.doi.org/10.4324/9781315061283.

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1954-, Gendelman Howard E., ed. The neurology of AIDS. New York: Chapman & Hall, 1997.

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Capitoli di libri sul tema "AIDS dementia complex - Pathogenesis"

1

Portegies, Peter, and Roelien H. Enting. "Aids Dementia Complex." In AIDS Pathogenesis, 209–20. Dordrecht: Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-017-0685-8_12.

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2

Benos, Dale J., James K. Bubien, Beatrice H. Hahn, George M. Shaw, and Etty N. Benveniste. "The Role of the Astrocyte in the Pathogenesis of the AIDS Dementia Complex." In Technical Advances in AIDS Research in the Human Nervous System, 223–33. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1949-2_17.

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3

Price, R. W. "AIDS Dementia Complex and HIV-1 Brain Infection: A Pathogenetic Framework for Treatment and Evaluation." In Current Topics in Microbiology and Immunology, 33–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79657-9_3.

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4

Toggas, S. M., and L. Mucke. "Transgenic Models in the Study of AIDS Dementia Complex." In Current Topics in Microbiology and Immunology, 223–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-85208-4_12.

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5

Gisslén, Magnus, Lars Hagberg, Paola Cinque, Bruce Brew, and Richard W. Price. "Cerebrospinal Fluid Markers in the Management of Central Nervous System HIV Infection and the AIDS Dementia Complex." In The Spectrum of Neuro-AIDS Disorders, 173–79. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815691.ch13.

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6

"AIDS Dementia Complex." In Encyclopedia of Behavioral Medicine, 74. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_300056.

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Brew, Bruce J. "AIDS dementia complex." In HIV/AIDS and the Nervous System, 79–91. Elsevier, 2007. http://dx.doi.org/10.1016/s0072-9752(07)85006-8.

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"Case 117 AIDS Dementia Complex." In Teaching Atlas of Nuclear Medicine, edited by Kevin J. Donohoe and Annick D. Van den Abbeele. Stuttgart: Georg Thieme Verlag, 2000. http://dx.doi.org/10.1055/b-0034-45326.

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Cinque, Paola, Bruce J. Brew, Magnus Gisslen, Lars Hagberg, and Richard W. Price. "Cerebrospinal fluid markers in central nervous system HIV infection and AIDS dementia complex." In HIV/AIDS and the Nervous System, 261–300. Elsevier, 2007. http://dx.doi.org/10.1016/s0072-9752(07)85017-2.

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Maj, Mario. "Dementia due to HIV disease." In New Oxford Textbook of Psychiatry, 384–86. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199696758.003.0049.

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Abstract (sommario):
The first description of a syndrome consisting of cognitive, motor, and behavioural disturbances in patients with AIDS was published in 1986. The syndrome was named ‘AIDS dementia complex’. In 1990, the World Health Organization (WHO) introduced the term ‘HIV-associated dementia’, pointing out that subclinical or mild cognitive and/or motor dysfunctions without impairment of performance in daily living activities cannot be subsumed under the term ‘dementia’. The expression ‘mild cognitive/motor disorder’ was proposed for those conditions. The same distinction was made in 1991 by the American Academy of Neurology, which identified an ‘HIV-associated dementia complex’ and an ‘HIV-associated minor cognitive/motor disorder’. The present chapter focuses on the dementia syndrome associated with HIV infection.
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