Letteratura scientifica selezionata sul tema "Bone cells"

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Articoli di riviste sul tema "Bone cells"

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Ahmed Elkammar, Hala. "Effect of human bone marrow derived mesenchymal stem cells on squamous cell carcinoma cell line." International Journal of Academic Research 6, no. 1 (January 30, 2014): 110–16. http://dx.doi.org/10.7813/2075-4124.2014/6-1/a.14.

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Zahran, Faten, Ahmed Abdel Zaher Ahmed Abdel.Zaher, Nermin Raafat, and Mohamed Ali Mohamed Ali. "Hepatocyte derived from Rat Bone Marrow Mesenchymal Stem Cells." Indian Journal of Applied Research 3, no. 10 (October 1, 2011): 1–5. http://dx.doi.org/10.15373/2249555x/oct2013/135.

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RemyaV, RemyaV, Naveen Kumar, and Kutty M. V. H. Kutty M.V.H. "A Method for Cell Culture and RNA Extraction of Rabbit Bone Marrow Derived Mesenchymal Stem Cells." International Journal of Scientific Research 3, no. 7 (June 1, 2012): 31–33. http://dx.doi.org/10.15373/22778179/july2014/11.

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Van Epps, Heather L. "Bone cells unite." Journal of Experimental Medicine 202, no. 3 (August 1, 2005): 335. http://dx.doi.org/10.1084/jem2023iti3.

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Aubin, Jane E. "Bone stem cells." Journal of Cellular Biochemistry 72, S30-31 (1998): 73–82. http://dx.doi.org/10.1002/(sici)1097-4644(1998)72:30/31+<73::aid-jcb11>3.0.co;2-l.

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Hashimoto, Futoshi, Kikuya Sugiura, Kyoichi Inoue, and Susumu Ikehara. "Major Histocompatibility Complex Restriction Between Hematopoietic Stem Cells and Stromal Cells In Vivo." Blood 89, no. 1 (January 1, 1997): 49–54. http://dx.doi.org/10.1182/blood.v89.1.49.

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Abstract Graft failure is a mortal complication in allogeneic bone marrow transplantation (BMT); T cells and natural killer cells are responsible for graft rejection. However, we have recently demonstrated that the recruitment of donor-derived stromal cells prevents graft failure in allogeneic BMT. This finding prompted us to examine whether a major histocompatibility complex (MHC) restriction exists between hematopoietic stem cells (HSCs) and stromal cells. We transplanted bone marrow cells (BMCs) and bones obtained from various mouse strains and analyzed the cells that accumulated in the eng
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Hashimoto, Futoshi, Kikuya Sugiura, Kyoichi Inoue, and Susumu Ikehara. "Major Histocompatibility Complex Restriction Between Hematopoietic Stem Cells and Stromal Cells In Vivo." Blood 89, no. 1 (January 1, 1997): 49–54. http://dx.doi.org/10.1182/blood.v89.1.49.49_49_54.

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Graft failure is a mortal complication in allogeneic bone marrow transplantation (BMT); T cells and natural killer cells are responsible for graft rejection. However, we have recently demonstrated that the recruitment of donor-derived stromal cells prevents graft failure in allogeneic BMT. This finding prompted us to examine whether a major histocompatibility complex (MHC) restriction exists between hematopoietic stem cells (HSCs) and stromal cells. We transplanted bone marrow cells (BMCs) and bones obtained from various mouse strains and analyzed the cells that accumulated in the engrafted bo
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Chambers, T. J., and K. Fuller. "Bone cells predispose bone surfaces to resorption by exposure of mineral to osteoclastic contact." Journal of Cell Science 76, no. 1 (June 1, 1985): 155–65. http://dx.doi.org/10.1242/jcs.76.1.155.

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The cell-free endocranial surface of young adult rat parietal bones was used as a substrate for osteoclastic bone resorption, either without prior treatment, or after incubation of the parietal bones with collagenase or neonatal rat calvarial cells. Untreated, the endocranial surface consisted of unmineralized organic fibres; incubation with calvarial cells or collagenase caused disruption and removal of these fibres, with extensive exposure of bone mineral on the endocranial surface, without morphologically detectable mineral dissolution. Neonatal rabbit osteoclasts resorbed bone to a greater
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INOUE, HIROMASA. "Cells phagocytizing bone. Bone metabolism and osteoclast." Kagaku To Seibutsu 23, no. 2 (1985): 99–102. http://dx.doi.org/10.1271/kagakutoseibutsu1962.23.99.

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Kelder, Cindy, Cornelis J. Kleverlaan, Marjolijn Gilijamse, Astrid D. Bakker, and Teun J. de Vries. "Cells Derived from Human Long Bone Appear More Differentiated and More Actively Stimulate Osteoclastogenesis Compared to Alveolar Bone-Derived Cells." International Journal of Molecular Sciences 21, no. 14 (July 17, 2020): 5072. http://dx.doi.org/10.3390/ijms21145072.

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Osteoblasts derived from mouse skulls have increased osteoclastogenic potential compared to long bone osteoblasts when stimulated with 1,25(OH)2 vitamin D3 (vitD3). This indicates that bone cells from specific sites can react differently to biochemical signals, e.g., during inflammation or as emitted by bioactive bone tissue-engineering constructs. Given the high turn-over of alveolar bone, we hypothesized that human alveolar bone-derived osteoblasts have an increased osteogenic and osteoclastogenic potential compared to the osteoblasts derived from long bone. The osteogenic and osteoclastogen
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Tesi sul tema "Bone cells"

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Fong, Jenna. "Breast cancer cells affect bone cell differentiation and the bone microenvironment." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104758.

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Breast carcinoma is the most commonly diagnosed cancer among women worldwide, with approximately 1 in 7 expected to be affected during her lifetime. The spread of breast cancer to secondary sites is generally incurable. Bone is the preferred site of metastasis, where the development of a secondary tumour causes severe osteolysis, hypercalcemia and a considerable pain burden. However, how breast cancer cells establish supportive interactions with bone cells is not well understood. We have examined the effects of factors released from MDA-MB-231 and 4T1 breast cancer cells on the differentiation
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Hoebertz, Astrid. "Purinergic signalling in bone cells." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249706.

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Laketic-Ljubojevic, Ira. "Glutamate signalling in bone cells." Thesis, University of York, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311080.

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Malone, Amanda Michelle Dolphin. "Mechanotransduction mechanisms in bone cells /." May be available electronically:, 2007. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Porter, Ryan Michael. "Examination of Glucocorticoid Treatment on Bone Marrow Stroma: Implications for Bone Disease and Applied Bone Regeneration." Thesis, Virginia Tech, 2002. http://hdl.handle.net/10919/36365.

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Long-term exposure to pharmacological doses of glucocorticoids has been associated with the development of osteopenia and avascular necrosis. Bone loss may be partially attributed to a steroid-induced decrease in the osteoblastic differentiation of multipotent progenitor cells found in the bone marrow. In order to determine if there is a change in the osteogenic potential of the bone marrow stroma following glucocorticoid treatment, Sprague-Dawley rats were administered methylprednisolone for up to six weeks, then sacrificed at 0, 2, 4, or 6 weeks during treatment or 4 weeks after cessation
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Bennett, Jonathan Hilary. "The differentiation of osteogenic cells from bone marrow." Thesis, University of Oxford, 1991. http://ora.ox.ac.uk/objects/uuid:3460f26e-a124-4605-8601-2e300241de14.

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Gronthos, Stan. "Stromal precursor cells : purification and the development of bone tissue." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phg8757.pdf.

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Bibliography: leaves 152-223. Experiments were designed to identify and purify human bone marrow stromal precursor cells by positive immunoselection, based on the cell surface expression of the VCAM-1 and STRO-1 antigens. The data presented demonstrates a hierarchy of bone cell development in vitro.
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Kandimalla, Yugandhar. "Study of Chitosan Microparticles with Bone Marrow Mesenchymal Stem Cells for Bone Tissue Regeneration." University of Toledo Health Science Campus / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=mco1250778129.

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Wigzell, Cathy. "Differentiation of bone cells in vitro." Thesis, University of St Andrews, 1990. http://hdl.handle.net/10023/14070.

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Osteoblastic differentiation was studied in vitro using primary cultures of bone cells derived from neonatal mouse calvaria. Using alkaline phosphatase as a marker, maintenance of the osteoblastic phenotype was found to be dependent upon the presence of ascorbic acid. No toxic effect due to ascorbic acid was seen. Insulin and dexamethasone were found to stimulate alkaline phosphatase expression, the former only in the absence of ascorbic acid. Two growth factors, epidermal growth factor and platelet-derived growth factor, were found to inhibit alkaline phosphatase expression in the presence of
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Weber, Matthew Charles. "Engineering human bone marrow stromal cells." Case Western Reserve University School of Graduate Studies / OhioLINK, 1991. http://rave.ohiolink.edu/etdc/view?acc_num=case1055867071.

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Libri sul tema "Bone cells"

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Bone research protocols. 2nd ed. New York: Humana Press, 2012.

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A manual for differentiation of bone marrow-derived stem cells to specific cell types. New Jersey: World Scientific, 2014.

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Pathology of bone marrow and blood cells. 2nd ed. Baltimore, Md: Lippincott William & Wilkins, 2009.

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Thomas, Gethin Penar. Load responsiveness of bone marrow stromal cells. Birmingham: University of Birmingham, 1994.

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Preston, Michael Robert. Signal transducing ion channels of bone cells. Birmingham: University of Birmingham, 1997.

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Gu, Yuchun. Investigation of ion channels on bone cells. Birmingham: University of Birmingham, 2000.

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Diggs, L. W. The morphology of human blood cells. 6th ed. Abbott Park, Ill: Abbott Laboratories, 2003.

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International Workshop on Cells and Cytokines in Bone and Cartilage (2nd 1988 Davos, Switzerland). Second International Workshop on Cells and Cytokines in Bone and Cartilage: 9-12 April 1988, Davos, Switzerland : abstracts. New York, N.Y: Springer International, 1988.

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International, Workshop on Cells and Cytokines in Bone and Cartilage (3rd 1990 Davos Switzerland). Third International Workshop on Cells and Cytokines in Bone and Cartilage: 8-11 April 1990, Davos, Switzerland : abstracts. New York, N.Y: Springer International, 1990.

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Antin, Joseph H. Manual of stem cell and bone marrow transplantation. New York: Cambridge University Press, 2009.

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Capitoli di libri sul tema "Bone cells"

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Oranger, Angela, Graziana Colaianni, and Maria Grano. "Bone Cells." In Imaging of Prosthetic Joints, 3–13. Milano: Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5483-7_1.

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Gooch, Keith J., and Christopher J. Tennant. "Bone Cells." In Mechanical Forces: Their Effects on Cells and Tissues, 55–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-03420-0_3.

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Reza Rezaie, Hamid, Mohammad Hossein Esnaashary, Masoud Karfarma, and Andreas Öchsner. "Productivity: Cells." In Bone Cement, 43–68. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39716-6_3.

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Oni, Olusola O. A., S. Dearing, and S. Pringle. "Endothelial Cells and Bone Cells." In Bone Circulation and Vascularization in Normal and Pathological Conditions, 43–48. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2838-8_5.

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Nakano, Toru, Takumi Era, Hiroaki Kodama, and Tasuku Honjo. "Development of Blood Cells from Mouse Embryonic Stem Cells in Culture." In Bone Marrow Transplantation, 9–19. Tokyo: Springer Japan, 1996. http://dx.doi.org/10.1007/978-4-431-68320-9_2.

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Gotfried, Y., J. Yaremchuk, M. A. Randolph, and A. J. Weiland. "The Target Cells in Vascularized Bone Allografts." In Bone Transplantation, 111–25. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83571-1_17.

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Boyce, B. F., D. E. Hughes, K. R. Wright, L. Xing, and A. Dai. "Apoptosis in Bone Cells." In Novel Approaches to Treatment of Osteoporosis, 61–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-09007-7_3.

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Barcellos-Hoff, Mary Helen. "Bone Marrow-derived Cells." In Encyclopedia of Systems Biology, 152–54. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_1395.

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Duong, Minh Ngoc, Yu-Ting Ma, and Ray C. J. Chiu. "Bone Marrow Stem Cells." In Methods in Molecular Biology, 33–46. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-511-8_3.

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Ho, A. D., and W. Wagner. "Bone Marrow Niche and Leukemia." In Cancer Stem Cells, 125–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/2789_2007_048.

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Atti di convegni sul tema "Bone cells"

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Ominsky, Michael S., Philippe K. Zysset, and Steven A. Goldstein. "Elastic Properties of 3D Cells for Trabecular Bone: Digital vs. Structural Finite Element Models." In ASME 1997 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-0201.

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Abstract Characterizing morphological and elastic properties of trabecular bone is a critical step towards understanding bone fragility. Idealized 3D cells for trabecular bone based on a tetrakaidecahedral geometry have been previously described to study morphology and its relationship to mechanical properties [1,2]. Two types of cells have been proposed: an open cell consisting of rectangular beams and a closed cell with hexagonal plates [Fig. 1]. The morphology of the cell is quantified by measures of structural density (SD) and mean intercept length (MIL). Structural density is mainly contr
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Shikata, Tetsuo, Toshihiko Shiraishi, Kumiko Tanaka, Shin Morishita, and Ryohei Takeuchi. "Effects of Amplitude and Frequency of Vibration Stimulation on Cultured Osteoblasts." In ASME 2007 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/detc2007-34949.

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Mechanical stimulation to bones affects osteogenesis such as decrease of bone mass of astronauts under zero gravity, walking rehabilitation to bone fracture and fracture repair with ultrasound devices. Bone cells have been reported to sense and response to mechanical stimulation at cellular level morphologically and metabolically. In the view of mechanical vibrations, bone cells are deformed according to mechanical stimulation and their mechanical characteristics. Recently, it was reported that viscoelasticity of cells was measured using tensile and creep tests and that there was likely natura
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Romito, Marilisa, Konstantina M. Stankovic, and Demetri Psaltis. "Imaging of cochlear cells through scattering bone." In Frontiers in Optics. Washington, D.C.: OSA, 2018. http://dx.doi.org/10.1364/fio.2018.jw3a.111.

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Ganji, Yasaman, and Mehran Kasra. "Comparison of Mechanosensitivity of Human Primary-Cultured Osteoblast Cells and Human Osteosarcoma Cell Line Under Hydrostatic Pressure." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80030.

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Regarding to the advances in mechanical stimulation of cells, this study aims to address important issues in bone generation and therapy at cellular level as it relates to understanding of bone cell response to hydrostatic pressure as well as choosing a proper cell model in studies of bone cell response to mechanical stimulation. G292 human osteosarcoma cell line and human primary osteoblast cells were tested under cyclic hydrostatic pressure. Monolayer culture of cells were divided into three groups of control without loading, static with the pressure of 0.5 MPa, and dynamic with the pressure
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Uddin, Sardar M. Zia, and Yi-Xian Qin. "Anabolic Effects of Ultrasound as Countermeasures of Simulated Microgravity in In-Vitro and In-Vivo Functional Disuse Models." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53796.

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Microgravity (MG) during space flight has been known to cause adverse effect on bone quality. Data collected from studies done on spaceflights show loss of 1–1.6% bone mineral density (BMD) per space-flight-month[1]. Most BMD has been recorded in load-bearing bones [2]. Some studies has considered using drugs and different growth factors to maintain bone mass in microgravity conditions but it can be too expensive to maintain over longer periods of time besides the systematic effects of such treatments [3]. Considering the effects of microgravity are partially attributed to lack of mechanical f
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Cowin, Stephen C. "The Search for Mechanism in Bone Adaptation Studies." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-1929.

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Abstract The mechanosensory mechanisms in bone include (i) the cell system that is stimulated by external mechanical loading applied to the bone; (ii) the system that transduces that mechanical loading to a communicable signal; and (iii) the systems that transmit that signal to the effector cells for the maintenance of bone homeostasis and for strain adaptation of the bone structure. The effector cells are the osteoblasts and the osteoclasts. These systems and the mechanisms that they employ have not yet been unambiguously identified. The candidate systems are reviewed here. The current theore
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Lan, Sheeny K., Daniel N. Prater, Russell D. Jamison, David A. Ingram, Mervin C. Yoder, and Amy J. Wagoner Johnson. "Vasculogenic Potential of Porcine Endothelial Colony Forming Cells." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192848.

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The natural healing process cannot restore form and function to critical size bone defects without the presence of a graft to support and guide tissue regeneration [1]. Critical size bone defects in humans are typically on the order of centimeters or larger [2]. Thus, a major limitation of synthetic grafts or bone tissue engineering constructs is the lack of vascularization to support cell viability after placement in vivo [3]. Cells that participate in bone regeneration, must reside within 150–200 microns of a blood supply in order to gain proper nutrients and to eliminate waste [4]. Conseque
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Jeon, Jong Heon, Tae Kyung Kim, So Hee Park, Jung Wook Shin, and Ok Chan Jeong. "Experimental Study on Cytoplasmic Calcium Oscillation in MG-63 Cells Induced by Pressure-Driven Fluid Flow." In ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-11121.

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This paper determined the optimal periodic mechanical stimulation of live bone cells from the intracellular calcium oscillation induced by shear stress. The shear stress-induced intracellular calcium responses of cells on a micro-cell chip were measured to study the mechanotransduction of bone cells. From the measured static and dynamic characteristics of the internal cellular signaling in cells, the optimum duration of the mechanical stimulation is determined.
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Srinivasan, Jayendran, Vincent Kish, Sydha Salihu, Madhavi Ayyalasomayajula, and Nilay Mukherjee. "Poking Cells in Cell-Gel Constructs: A Potential Way of Measuring Fluid Pressure in Cells." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61290.

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In this study, fluorescently labeled ATDC5 and bone marrow derived preosteoblastic cells were embedded in agarose gel and poked with a micropipette under a confocal microscope. Image stacks of cross sections of cells were taken before and after poking and the largest cross sectional areas were analyzed. After adjusting for photobleaching effects by equalizing the area of a nearby unpoked cell (by adjusting threshold values of the images) the area of the poked cell before and after poking was measured and the percentage change in area was calculated. The percentage change in cross sectional are
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Millon, Debra Chenet, Darren L. Hitt, and Stephan J. LaPointe. "Heat Generation in Bone Cutting-Implications for Thermal Necrosis." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/htd-24430.

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Abstract A bunion is a common foot disorder caused by an abnormal outward projection of the joint and inward turning of the toe. Surgery to correct the malformation involves cutting the first metatarsal head, repositioning and setting it; the bone is then left to heal itself over time. A potentially serious by-product of the bone cutting is the frictional heat generated. While the heat susceptibility of individual bone cells varies throughout bone and is difficult to quantify, studies have shown that when injured, bone may not always heal as bone but rather as a fibrous tissue of varying degre
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Rapporti di organizzazioni sul tema "Bone cells"

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Dooner, Mark, Jason M. Aliotta, Jeffrey Pimental, Gerri J. Dooner, Mehrdad Abedi, Gerald Colvin, Qin Liu, Heinz-Ulli Weier, Mark S. Dooner, and Peter J. Quesenberry. Cell Cycle Related Differentiation of Bone Marrow Cells into Lung Cells. Office of Scientific and Technical Information (OSTI), December 2007. http://dx.doi.org/10.2172/936517.

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Mastro, Andrea M. Trafficking of Metastatic Breast Cancer Cells in Bone. Fort Belvoir, VA: Defense Technical Information Center, August 2004. http://dx.doi.org/10.21236/ada433936.

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Mastro, Andrea M. Trafficking of Metastatic Breast Cancer Cells in Bone. Fort Belvoir, VA: Defense Technical Information Center, August 2006. http://dx.doi.org/10.21236/ada460748.

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Gay, Carol V. Directed Secretion by Bone Cells of a Factor that Attracts Breast Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, October 2001. http://dx.doi.org/10.21236/ada398984.

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Park, Serk I. Activation of Myeloid-Derived Suppressor Cells in Bone Marrow. Fort Belvoir, VA: Defense Technical Information Center, December 2013. http://dx.doi.org/10.21236/ada600504.

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Donohue, Henry J., Christopher Niyibizi, and Alayna Loiselle. Induced Pluripotent Stem Cell Derived Mesenchymal Stem Cells for Attenuating Age-Related Bone Loss. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada606237.

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Donahue, Henry J. Induced Pluripotent Stem Cell Derived Mesenchymal Stem Cells for Attenuating Age-Related Bone Loss. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada581680.

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Shevde-Samant, Lalita. Crosstalk Between Cancer Cells and Bones Via the Hedgehog Pathway Determines Bone Metastasis of Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2008. http://dx.doi.org/10.21236/ada487471.

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Donahue, Henry J. Fluid Flow Sensitivity of Bone Cells as a Function of Age. Fort Belvoir, VA: Defense Technical Information Center, October 2001. http://dx.doi.org/10.21236/ada401057.

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Majeska, Robert J., and Mitchell B. Schaffler. Role of Bone Remodeling in Skeletal Colonization by Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, August 2005. http://dx.doi.org/10.21236/ada444893.

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