Bibliografie tematiche / Cerebral ischemia

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Letteratura scientifica selezionata sul tema "Cerebral ischemia"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "Cerebral ischemia"

1

Zhao, Ling, Qiwei Liao, Yueting Zhang, Shufen Tan, Shuqing Li, and Tingyu Ke. "Ischemic Postconditioning Mitigates Retinopathy in Tree Shrews with Diabetic Cerebral Ischemia." Journal of Diabetes Research 2020 (February 12, 2020): 1–10. http://dx.doi.org/10.1155/2020/6286571.

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Ischemic postconditioning (PC) is proved to efficiently protect diabetic patients with acute myocardial infarction from ischemia-reperfusion injury. We aimed to explore the protective roles of ischemic PC on diabetic retinopathy in tree shrews with diabetic cerebral ischemia. A diabetic tree shrew model was established through high-fat diet feeding combined with streptozotocin (STZ) injection, while cortical thrombotic cerebral ischemia was induced photochemically. Tree shrews were divided into the normal control group, sham operation group, diabetes mellitus group, diabetes mellitus+cerebral
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Kanazawa, Masato, Tetsuya Takahashi, Masanori Ishikawa, Osamu Onodera, Takayoshi Shimohata, and Gregory J. del Zoppo. "Angiogenesis in the ischemic core: A potential treatment target?" Journal of Cerebral Blood Flow & Metabolism 39, no. 5 (2019): 753–69. http://dx.doi.org/10.1177/0271678x19834158.

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The ischemic penumbra is both a concept in understanding the evolution of cerebral tissue injury outcome of focal ischemia and a potential therapeutic target for ischemic stroke. In this review, we examine the evidence that angiogenesis can contribute to beneficial outcomes following focal ischemia in model systems. Several studies have shown that, following cerebral ischemia, endothelial proliferation and subsequent angiogenesis can be detected beginning four days after cerebral ischemia in the border of the ischemic core, or in the ischemic periphery, in rodent and non-human primate models,
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Wang, Lei, Xu Zhang, Xiaoxing Xiong, et al. "Nrf2 Regulates Oxidative Stress and Its Role in Cerebral Ischemic Stroke." Antioxidants 11, no. 12 (2022): 2377. http://dx.doi.org/10.3390/antiox11122377.

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Abstract (sommario):
Cerebral ischemic stroke is characterized by acute ischemia in a certain part of the brain, which leads to brain cells necrosis, apoptosis, ferroptosis, pyroptosis, etc. At present, there are limited effective clinical treatments for cerebral ischemic stroke, and the recovery of cerebral blood circulation will lead to cerebral ischemia-reperfusion injury (CIRI). Cerebral ischemic stroke involves many pathological processes such as oxidative stress, inflammation, and mitochondrial dysfunction. Nuclear factor erythroid 2-related factor 2 (Nrf2), as one of the most critical antioxidant transcript
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Danilova, T. V. "Features of epilepsy in acute and chronic cerebral ischemia." Kazan medical journal 98, no. 6 (2017): 877–83. http://dx.doi.org/10.17750/kmj2017-877.

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Aim. To study clinical features, functional and neuroimaging characteristics of the brain of patients with ischemic brain disease and epileptic seizures. Methods. 772 patients with acute and chronic cerebral ischemia (265 stroke patients with epileptic seizures and 174 patients with seizures on the background of chronic cerebral ischemia, and 203 patients with stroke and 130 patients with chronic cerebral ischemia without seizures) were comprehensively examined. The clinical neurological examination, electroencephalography, magnetic resonance imaging, ultrasound duplex scanning of extra- and i
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Yamamoto, Kazumi, Fumiharu Akai, Toshiki Yoshimine, and Takehiko Yanagihara. "Immunohistochemical investigation of cerebral ischemia after middle cerebral artery occlusion in gerbils." Journal of Neurosurgery 67, no. 3 (1987): 414–20. http://dx.doi.org/10.3171/jns.1987.67.3.0414.

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✓ Progression and recovery of ischemic and postischemic damage after occlusion of the middle cerebral artery and subsequent reperfusion were investigated in the gerbil. This study was performed by immunohistochemical reaction testing for tubulin and creatine kinase BB-isoenzyme to visualize the neuronal structure and by immunohistochemical reaction testing for astroprotein (an astrocyte-specific protein) to visualize reactive astrocytes. The earliest ischemic lesion became visible in the frontoparietal cortex after 7 minutes of ischemia as a laminar loss of the reaction for tubulin involving t
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Duarte, Sinésio Grace, Antônio Dorival Campos, and Benedicto Oscar Colli. "Functional evaluation of temporary focal cerebral ischemia: experimental model." Arquivos de Neuro-Psiquiatria 61, no. 3B (2003): 751–56. http://dx.doi.org/10.1590/s0004-282x2003000500009.

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OBJECTIVE: Despite cerebral ischemia being a frequent clinical pathologic state, the tolerance of neural tissue to oxygen absence and to reperfusion is controversial. This study aims to evaluate the effects of focal cerebral ischemia/reperfusion, by analyzing the mitochondrial respiration. METHOD: Sixty-four adult rats underwent focal cerebral ischemia by middle cerebral artery occlusion, during 15, 30 and 60 minutes, followed by 10 minutes or 19 hours of reperfusion. The effects of ischemia were analyzed measuring the O2 consumption by mitochondria in the ischemic and non-ischemic areas. RESU
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Han, Xue Mei, Hong Tao Wei, and Song Yan Liu. "Involvement of Erythropoietin Expression in Acupuncture Preconditioning-Induced Ischemic Tolerance." Advanced Materials Research 554-556 (July 2012): 1650–55. http://dx.doi.org/10.4028/www.scientific.net/amr.554-556.1650.

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Abstract Objective To investigate the expression of erythropoietin (EPO) after acupuncture preconditioning plus focal cerebral ischemia treatment. Methods Rat focal cerebral ischemia model and acupuncture preconditioning model were established. Animals were randomly assigned into different groups: control (focal cerebral ischemia) and acupuncture preconditioning plus focal cerebral ischemia, with 8 rats for each group. The expression of EPO after different treatments was determined by histological examination, immunohistochemistry and in situ hybridization. Results The mRNA and protein express
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Dong, Chao, Jiawei Li, Ming Zhao, et al. "Pharmacological Effect of Panax notoginseng Saponins on Cerebral Ischemia in Animal Models." BioMed Research International 2022 (August 4, 2022): 1–12. http://dx.doi.org/10.1155/2022/4281483.

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Panax notoginseng saponins (PNS), bioactive compounds, are commonly used to treat ischemic heart and cerebral diseases in China and other Asian countries. Most previous studies of PNS have focused on the mechanisms underlying their treatment of ischemic cardiovascular diseases but not cerebral ischemic diseases. This study sought to explore the pharmacological mechanisms underlying the effectiveness of PNS in treating cerebral ischemic diseases. Different experimental cerebral ischemia models (including middle cerebral artery occlusion (MCAO) and the blockade of four arteries in rats, collagen
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Zeng, Xian-Si, Wen-Shuo Geng, Lei Chen, and Jin-Jing Jia. "Thioredoxin as a Therapeutic Target in Cerebral Ischemia." Current Pharmaceutical Design 24, no. 25 (2018): 2986–92. http://dx.doi.org/10.2174/1381612824666180820143853.

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Abstract (sommario):
Background: Cerebral ischemia is a common cause of disability and death. Ischemic brain injury results from complex pathological processes, including oxidative stress, inflammation, and apoptosis. Thioredoxin( Trx) is an important multifunctional protein, which regulates cellular redox status. Increasing studies have demonstrated that Trx provides a neuroprotective role against cerebral ischemia-induced injury. Methods: A systematic search of PMC and the PubMed Database was conducted to summarize the protective effects of Trx against cerebral ischemia. Results: This article reviews the underst
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Rehni, Ashish K., Pradeep Bhateja, and Nirmal Singh. "Diethyl dithiocarbamic acid, a possible nuclear factor kappa B inhibitor, attenuates ischemic postconditioning-induced attenuation of cerebral ischemia–reperfusion injury in mice." Canadian Journal of Physiology and Pharmacology 87, no. 1 (2009): 63–68. http://dx.doi.org/10.1139/y08-100.

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Abstract (sommario):
The present study was designed to pharmacologically investigate the possible role of nuclear factor kappa B (NF-κB) in the reversal of global cerebral injury induced by ischemia and reperfusion after ischemic postconditioning. Bilateral carotid artery occlusion for 17 min followed by reperfusion for 24 h was employed to produce ischemia- and reperfusion-induced cerebral injury in mice. Cerebral infarct size was measured by using triphenyltetrazolium chloride staining. Memory was evaluated using the Morris water maze test. The rotarod test was employed to assess motor incoordination. Bilateral
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Tesi sul tema "Cerebral ischemia"

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Keasey, Matthew P. "MicroRNAs in Cerebral Ischemia." Thesis, University of Bristol, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526014.

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Molnar, Maria. "Hyperglycemia in Experimental Cerebral Ischemia." Doctoral thesis, Uppsala universitet, Anestesiologi och intensivvård, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-247763.

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Abstract (sommario):
Cerebral ischemia is a life-threatening condition associated with a substantial morbidity and mortality. Hyperglycemia, a common coexisting phenomenon in both stroke and cardiac arrest (CA), may further aggravate ischemic brain injury. To date, the therapeutic possibilities are lim-ited and the search for new treatment modalities is warranted. One aspect of such a research could be to better understand the cerebral pathogenesis induced by hyperglycemic ischemia-reperfusion. We investigated the combination of ischemia and hyperglycemia in two experimental models of stroke and CA. The aims were
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Christensen, Thomas. "Experimental focal cerebral ischemia : pathophysiology, metabolism and pharmacology of the ischemic penumbra /." Copenhagen, 2007. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=016143698&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Thorén, Anna. "Astrocyte metabolism following focal cerebral ischemia /." Göteborg : Institute of Neuroscience and Physiology, The Sahlgrenska Academy, Göteborg University, 2006. http://hdl.handle.net/2077/744.

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Thomas, Sunu Samuel. "Murine models of cerebral ischemia, development of a mouse model of global cerebral ischemia; response of GluR2 knockout mice in a model of permanent focal cerebral ischemia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0026/MQ50439.pdf.

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Li, Yan. "Inhibitory synpatic transmission in striatal neurons after transient cerebral ischemia." Connect to resource online, 2009. http://hdl.handle.net/1805/2021.

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Thesis (Ph.D.)--Indiana University, 2009.<br>Title from screen (viewed on December 1, 2009). Department of Anatomy and Cell Biology, Indiana University-Purdue University Indianapolis (IUPUI). Advisor(s): Zao C. Xu, Feng C. Zhou, Charles R. Yang, Theodore R. Cummins. Includes vitae. Includes bibliographical references (leaves 115-135).
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Karelina, Ekaterina. "MECHANISMS OF SOCIAL NEUROPROTECTION AFTER CEREBRAL ISCHEMIA." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1274922479.

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Adhami, Faisal. "Differential Adult and Neonatal Response to Cerebral Ischemia-Hypoxia." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1196054266.

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Edrissi, Hamidreza. "Blood Brain Barrier Dysfunction in Chronic Cerebral Ischemia." Thesis, Université d'Ottawa / University of Ottawa, 2015. http://hdl.handle.net/10393/32531.

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Cerebral small vessel pathology is now known to be associated with the development of cognitive impairment and mild motor impairments such as gait disturbance in a variety of neurodegenerative diseases. This dissertation explores the hypothesis that blood brain barrier dysfunction is an early event in cerebral ischemia and contributes to the development of cerebral small vessel disease (CSVD). A common rodent model of CSVD is permanent bilateral common carotid artery occlusion in the rat. This model was used to study several aspects of the progression of CSVD including the timecourse of blood
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Ng, Kit-ying. "Neuroprotective effects of adiponectin in focal cerebral ischemia." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B39634371.

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Libri sul tema "Cerebral ischemia"

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Walz, Wolfgang. Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1385/0896035409.

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Hacke, Werner, Herman J. Gelmers, Michael Hennerici, and Günter Krämer. Cerebral Ischemia. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75548-4.

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Walz, Wolfgang, ed. Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5.

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Maier, Carolina M., and Gary K. Steinberg. Hypothermia and Cerebral Ischemia. Humana Press, 2003. http://dx.doi.org/10.1385/1592596533.

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Hartmann, Alexander, and Wolfgang Kuschinsky, eds. Cerebral Ischemia and Hemorheology. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71787-1.

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Hartmann, Alexander, and Wolfgang Kuschinsky, eds. Cerebral Ischemia and Calcium. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-85863-5.

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Hartmann, Alexander, Wolfgang Kuschinsky, and Siegfried Hoyer, eds. Cerebral Ischemia and Dementia. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76208-6.

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1943-, Hartmann A., Kuschinsky Wolfgang 1944-, and Hoyer S. 1933-, eds. Cerebral ischemia and dementia. Springer-Verlag, 1991.

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A, Schurr, and Rigor Benjamin M, eds. Cerebral ischemia and resuscitation. CRC Press, 1990.

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A, Hartmann, and Kuschinsky W, eds. Cerebral ischemia and hemorheology. Springer-Verlag, 1987.

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Capitoli di libri sul tema "Cerebral ischemia"

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Iadecola, Costantino. "Mechanisms of Cerebral Ischemic Damage." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_1.

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Kondo, Yoichi. "Activated and Phagocytic Microglia." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_10.

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Irwin, Anna, and Wolfgang Walz. "Spreading Depression Waves as Mediators of Secondary Injury and of Protective Mechanisms." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_2.

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Kimelberg, Harold K. "Cell Swelling in Cerebral Ischemia." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_3.

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Morley, Paul, Joseph S. Tauskela, and Antoine M. Hakim. "Calcium Overload." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_4.

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Chan, Pak H. "Oxygen Radicals." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_5.

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Bruce-Keller, Annadora J., and Mark P. Mattson. "Mechanisms of Neuroprotective Cytokines." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_6.

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Nowak, Thaddeus S., and Marika Kiessling. "Reprogramming of Gene Expression After Ischemia." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_7.

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Abe, Koji. "Neurons." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_8.

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Jabs, Ronald, Lane K. Bekar, and Wolfgang Walz. "Reactive Astrogliosis in the Injured and Postischemic Brain." In Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5_9.

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Atti di convegni sul tema "Cerebral ischemia"

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Kust, Sergei, Yury Obukhov, Mikhail Sinkin, and Irina Okuneva. "A New Approach for Early Diagnosis of Delayed Cerebral Ischemia After Subarachnoid Hemorrhage." In 2025 27th International Conference on Digital Signal Processing and its Applications (DSPA). IEEE, 2025. https://doi.org/10.1109/dspa64310.2025.10977891.

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Fisher, M., and R. Francis. "ALTERED COAGULATION IN CEREBRAL ISCHEMIA PATIENTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644345.

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We investigated coagulation changes in a group of patients with cerebral ischemia, ranging from transient ischemic attacks to cerebral infarction. Patients were studied acutely (within 72 hours of onset of ischemia) and again approximately 2 months following the initial examination. We evaluated platelet activation, fibrin generation, and fibrinolysis by measuring plasma beta-thromboglobulin (BTG), fibrinopeptide A (FPA), and fibrinopeptide B-beta 1-42 (FPB), respectively. We compared measurements in cerebral ischemia patients with a group of age -and sex-matched neurological inpatients withou
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Khodanovich, M. Yu, and A. A. Kisel. "Animal models of cerebral ischemia." In NEW OPERATIONAL TECHNOLOGIES (NEWOT’2015): Proceedings of the 5th International Scientific Conference «New Operational Technologies». AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4936032.

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Lee, Gija, Seokkeun Choi, Sungwook Kang, et al. "Changes in Extracellular Glutamate Release on Repetitive Transient Occlusion in Global Ischemia Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206602.

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During the operation, surgeons in neurosurgical area usually performed the multiple temporary occlusions of parental artery which may induce the neuronal damage. It is generally thought that neuronal damage by cerebral ischemia is associated with extracellular concentrations of the excitatory amino acids. In this experiment, we measured the dynamics of extracellular glutamate release in 11 vessel occlusion (VO) model during repeated within short interval. Changes in cerebral blood flow were monitored by laser-Doppler flowmetry simultaneously with cortical glutamate level measured by amperometr
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Chalabov, Sh I., and N. K. Klichkhanov. "ANTIOXIDANT PROPERTIES OF SILYMARIN IN CEREBRAL ISCHEMIA." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-279.

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The effect of silymarin on oxidative stress in incomplete cerebral ischemia in rats was studied. Ischemia leads to the development of oxidative stress, which is manifested by an increase in the content of lipid and protein peroxidation products and a decrease in the antioxidant system. At the same time, diet with silymarin prevents the development of oxidative stress during cerebral ischemia in rats.
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Musienko, Julia I., and Natalia I. Nechipurenko. "Infrared laser hemotherap in cerebral ischemia modeling." In European Conference on Biomedical Optics. OSA, 2003. http://dx.doi.org/10.1364/ecbo.2003.5142_200.

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Musienko, Julia I., and Natalia I. Nechipurenko. "Infrared laser hemotherapy in cerebral ischemia modeling." In European Conference on Biomedical Optics 2003, edited by Rudolf W. Steiner. SPIE, 2003. http://dx.doi.org/10.1117/12.501113.

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Palaniswamy, Sangeetha, Venkatapura Ramesh, Rita Christopher, and Dhananjaya Bhat. "A0022 Effect of Remote Ischemic Preconditioning on Cerebral Vasospasm and Biomarkers of Cerebral Ischemia in Aneurysmal Subarachnoid Hemorrhage." In 20th Annual Conference of Indian Society of Neuroanaesthesiology and Critical Care (ISNACC). Thieme Medical and Scientific Publishers Private Ltd., 2019. http://dx.doi.org/10.1055/s-0039-1684129.

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Ignatieva, Olga I., and Natalia G. Tokareva. "Cognitive impairments in patients with chronic cerebral ischemia." In Innovations in Medical Science and Education. Dela Press Publishing House, 2022. http://dx.doi.org/10.56199/dpcsms.utii3716.

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Abstract (sommario):
Cognitive impairments, such as memory and intelligence, attention and mental performance, usually suffer in diffuse brain damage, in particular in chronic cerebral ischemia. However, they can also occur in focal brain lesions, such as cerebral infarcts, reaching the degree of dementia. The aim of the study was to investigate the peculiarities of non-dementia cognitive disorders in patients with the consequences of cerebral infarction. We examined 30 patients with chronic cerebral ischemia with cerebral infarction at the age of 58-66 years (M=62.4) and a similar comparison group was chosen. The
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Soltanian-Zadeh, Hamid, Rabih Hammoud, Michael A. Jacobs, et al. "Tissue characterization in cerebral ischemia using multiparameter MRI." In Medical Imaging 2001, edited by Chin-Tu Chen and Anne V. Clough. SPIE, 2001. http://dx.doi.org/10.1117/12.428162.

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Rapporti di organizzazioni sul tema "Cerebral ischemia"

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Oeltgen, Peter R. Neuroprotective Effects of Opioid-Like Hibernation Factors in Cerebral Ischemia. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada424443.

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TU, LANG, Yimiao Luo, Xinyi Xu, Huiling Xiong, Sihan Hu, and Xiao Ma. Constructing evidence for the treatment of cerebral ischemia-reperfusion injury with ligustrazine: a preclinical meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.6.0002.

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Wu, Xiaoqi, Maoxia Fan, Yaobo Pan, and Dona Guo. Quality of Evidence Supporting the Effects of Ginkgo Terpene Lactone Preparations in Ischemic Stroke: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.9.0124.

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Review question / Objective: 2.2.1 Type of studies SRs/MAs of Randomized Controlled Trials (RCTs) of GTLP for IS in any language. 2.2.2 Type of Participants Included patients were diagnosed with IS according to international or national standards, regardless of race, age, gender, time of onset, and source of cases. 2.2.3 Type of Intervention The intervention method in the control group was routine treatment, and the intervention method in the intervention group was GTLP treatment or GTLP combined with the treatment of the control group. 2.2.4 Types of outcomes Conclusions at least need to incl
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He, Miao, Sen Wang, Jing You, Jing Shi, Yanbing Yin, and Weirong Li. Neuroprotective effects of resveratrol against cerebral ischemia/reperfusion injury through anti-oxidant and anti-inflammatory mechanisms：A Systematic Review and Meta-Analysis in Rodents. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0059.

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Xu, Fangyuan, Qiqi Yang, Wei Huang, and Zhenzhen Liu. The protective effect of acupuncture at Baihui acupoint (DU 20) for cerebral ischemia-reperfusion injury in rat models: a protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.3.0114.

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Yu, Beibei, Yongfeng Zhang, and Shouping Gong. Effects of miRNA-modified exosomes alleviate cerebral ischemic reperfusion injury in Pre-clinical Studies: A Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0062.

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Abstract (sommario):
Review question / Objective: The purpose of this study was to investigate the effect of miRNA-modified exosomes in alleviating cerebral ischemic reperfusion injury compared with the non-treatment group. The research object is an animal model of middle cerebral artery occlusion. The research method is a controlled study. The primary outcome of this study was infarct volume, and the secondary outcome was neurobehavioral performance. Main outcome(s): The primary outcome of this study was Infarct volumes，which was measured by 2,3,5-triphenyltetranzolium chloride (TTC) staining. And it was calculat
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Huang, Wei, Dahong Yang, Danyang Fan, Chao Hou, and Wanqian Liu. Prognostic value of net water uptake in acute ischemic stroke: a protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.12.0077.

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Review question / Objective: The purpose of this protocol is to present a transparent and clear methodology for performing a systematic review and meta-analysis of the available literature aimed to answer the following question: among patients with acute ischemic stroke due to large vessel occlusion, is net water uptake (NWU), as measured in CT images, associated with (i) cerebrovascular complications, including malignant cerebral edema, secondary intracerebral hemorrhage, and (ii) post-stroke functional outcome as measured by the modified Rankin Scale. Condition being studied: Currently, the
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Wang, Kai, Zhiguo Lv, Peng Xu, et al. Relevance of dynamic cerebral autoregulation after acute ischemic stroke with prognosis: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.1.0056.

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Guan, Peiyu, Dingkun Gui, and Youhua Xu. Evaluation on the efficacy and safety of Panax notoginseng saponins in the treatment of stroke among elderly people - A systematic review and meta-analysis of 206 randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.3.0042.

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Abstract (sommario):
Review question / Objective: Population: The symptoms of the population were in line with the relevant diagnostic criteria for stroke both at home and abroad, and were confirmed as ischemic stroke by medical imaging tests such as MRI or CT.The subject groups were older adults with an average age of over 60, regardless of gender or race. Intervention: Intervention involves single use of PNS or combined use of PNS and WM or PNS and Treatment as usual (TAU). Comparator: All the patients in the controlled group underwent conventional routine treatment to improve their cerebral blood supply and dru
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Du, Rui. New Insights into the Therapeutic Potential of Traditional Chinese Medicine and Western Medicine Integration for Coronary Heart Disease Complicated by Cerebral Ischemic Stroke: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2025. https://doi.org/10.37766/inplasy2025.1.0112.

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