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Letteratura scientifica selezionata sul tema "Energy and glucose homeostasis"

Autore: Grafiati

Pubblicato: 28 settembre 2024

Ultima modifica: 31 luglio 2025

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Articoli di riviste sul tema "Energy and glucose homeostasis"

1

Lam, Carol K. L., Madhu Chari, and Tony K. T. Lam. "CNS Regulation of Glucose Homeostasis." Physiology 24, no. 3 (2009): 159–70. http://dx.doi.org/10.1152/physiol.00003.2009.

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The past decade has hosted a remarkable surge in research dedicated to the central control of homeostatic mechanisms. Evidence indicates that the brain, in particular the hypothalamus, directly senses hormones and nutrients to initiate behavioral and metabolic responses to control energy and nutrient homeostasis. Diabetes is chiefly characterized by hyperglycemia due to impaired glucose homeostatic regulation, and a primary therapeutic goal is to lower plasma glucose levels. As such, in this review, we highlight the role of the hypothalamus in the regulation of glucose homeostasis in particula

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Assistant, Professor Farida Rozy. "Comparison of Insulin and Glucagon in the Regulation of Blood Glucose Levels." ISRG Journal of Arts Humanities & Social Sciences (ISRGJAHSS) III, no. III (2025): 147–52. https://doi.org/10.5281/zenodo.15454908.

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<em>Maintaining blood glucose levels is of vital importance, as glucose serves as the primary energy source for body cells. Therefore, to ensure adequate energy supply, body tissues and cells—particularly brain cells—rely on glucose as a principal energy substrate. The objectives of this study are to examine and compare the roles of insulin and glucagon in regulating blood glucose levels, explore their metabolic impacts, identify the mechanisms of action of these two hormones, and investigate how they interact to maintain glucose homeostasis. Furthermore, the study assesses complic

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Pattaranit, Ratchada, and Hugo Antonius van den Berg. "Mathematical models of energy homeostasis." Journal of The Royal Society Interface 5, no. 27 (2008): 1119–35. http://dx.doi.org/10.1098/rsif.2008.0216.

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Diabetes and obesity present a mounting global challenge. Clinicians are increasingly turning to mechanism-based mathematical models for a quantitative definition of physiological defects such as insulin resistance, glucose intolerance and elevated obesity set points, and for predictions of the likely outcomes of therapeutic interventions. However, a very large range of such models is available, making a judicious choice difficult. To better inform this choice, here we present the most important models published to date in a uniform format, discussing similarities and differences in terms of t

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Marty, Nell, Michel Dallaporta, and Bernard Thorens. "Brain Glucose Sensing, Counterregulation, and Energy Homeostasis." Physiology 22, no. 4 (2007): 241–51. http://dx.doi.org/10.1152/physiol.00010.2007.

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Neuronal circuits in the central nervous system play a critical role in orchestrating the control of glucose and energy homeostasis. Glucose, beside being a nutrient, is also a signal detected by several glucose-sensing units that are located at different anatomical sites and converge to the hypothalamus to cooperate with leptin and insulin in controlling the melanocortin pathway.

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López-Gambero, A. J., F. Martínez, K. Salazar, M. Cifuentes, and F. Nualart. "Brain Glucose-Sensing Mechanism and Energy Homeostasis." Molecular Neurobiology 56, no. 2 (2018): 769–96. http://dx.doi.org/10.1007/s12035-018-1099-4.

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Soty, Maud, Amandine Gautier-Stein, Fabienne Rajas, and Gilles Mithieux. "Gut-Brain Glucose Signaling in Energy Homeostasis." Cell Metabolism 25, no. 6 (2017): 1231–42. http://dx.doi.org/10.1016/j.cmet.2017.04.032.

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Wang, Yan, Markey C. McNutt, Serena Banfi, et al. "Hepatic ANGPTL3 regulates adipose tissue energy homeostasis." Proceedings of the National Academy of Sciences 112, no. 37 (2015): 11630–35. http://dx.doi.org/10.1073/pnas.1515374112.

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Angiopoietin-like protein 3 (ANGPTL3) is a circulating inhibitor of lipoprotein and endothelial lipase whose physiological function has remained obscure. Here we show that ANGPTL3 plays a major role in promoting uptake of circulating very low density lipoprotein-triglycerides (VLDL-TGs) into white adipose tissue (WAT) rather than oxidative tissues (skeletal muscle, heart brown adipose tissue) in the fed state. This conclusion emerged from studies of Angptl3−/− mice. Whereas feeding increased VLDL-TG uptake into WAT eightfold in wild-type mice, no increase occurred in fed Angptl3−/− animals. De

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Seo, J., E. S. Fortuno, J. M. Suh, et al. "Atf4 Regulates Obesity, Glucose Homeostasis, and Energy Expenditure." Diabetes 58, no. 11 (2009): 2565–73. http://dx.doi.org/10.2337/db09-0335.

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Giridharan, NV. "Glucose & energy homeostasis: Lessons from animal studies." Indian Journal of Medical Research 148, no. 5 (2018): 659. http://dx.doi.org/10.4103/ijmr.ijmr_1737_18.

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Pepino, Marta Y., and Christina Bourne. "Non-nutritive sweeteners, energy balance, and glucose homeostasis." Current Opinion in Clinical Nutrition and Metabolic Care 14, no. 4 (2011): 391–95. http://dx.doi.org/10.1097/mco.0b013e3283468e7e.

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Tesi sul tema "Energy and glucose homeostasis"

1

Forsyth, Robert J. "The contribution of astrocyte glycogen to brain energy homeostasis." Thesis, University of Newcastle Upon Tyne, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361387.

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Burke, Luke Kennedy. "Neurocircuitry underlying serotonin's effects on energy and glucose homeostasis." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708592.

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Matthäus, Dörte. "The role of CADM1 in energy and glucose homeostasis." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2014. http://dx.doi.org/10.18452/16905.

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Abstract (sommario):

Mehr als 300 Millionen Menschen sind weltweit von Diabetes betroffen, die Mehrheit davon leidet an Typ-2-Diabetes. Typ-2-Diabetes ist durch eine Insulinresistenz charakterisiert, welche meistens durch Übergewicht und Adipositas verursacht wird. Diese Insulinresistenz kann zunächst durch eine erhöhte pankreatische Insulinsekretion kompensiert werden, jedoch können langfristig die pankreatischen beta-Zellen den erhöhten Insulinbedarf nicht mehr decken. Dies verursacht einen starken Anstieg der Blutglucosespiegel und stellt den Beginn der Typ-2-Diabetes Erkrankung dar. Neben genetischen Veränderu

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Wang, Xun. "IRF3 is a Critical Regulator of Adipose Glucose and Energy Homeostasis." Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10537.

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Obesity is associated with a state of chronic inflammation, which is believed to contribute to insulin resistance. We previously identified interferon regulatory factor 3 (IRF3) as an anti-adipogenic transcription factor with high expression in adipocytes. Because IRF3 is known to drive expression of pro-inflammatory genes in immune cells, we hypothesized that it may also promote inflammation and insulin resistance in adipocytes. Consistent with our expectations, we found that the expression of inflammatory genes in adipocytes was induced by IRF3 overexpression, while knockdown of IRF3 had the

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Hall, Jessica Ann. "Thyroid Hormone and Insulin Metabolic Actions on Energy and Glucose Homeostasis." Thesis, Harvard University, 2014. http://dissertations.umi.com/gsas.harvard:11663.

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Faced with an environment of constantly changing nutrient availability, mammals have adapted complex homeostatic mechanisms to maintain energy balance. Deviations from this balance are largely corrected through a concerted, multi-organ effort that integrates hormonal signals with transcriptional regulatory networks. When these relationships are altered, as with over-nutrition and insulin resistance, metabolic disease ensues. Here, I present data concerning two distinct transcriptional pathways--one for thyroid hormone (TH) and one for insulin--that confer hormone responsiveness on metabolic ge

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Rahman, S. A. "Investigating the role of gut hormones in energy and glucose homeostasis." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1417078/.

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Obesity is associated with type-2 diabetes mellitus. Gut hormones are peptides secreted in response to nutrient intake that act to regulate food intake, energy and glucose homeostasis. Thus, alterations in gut hormone abundance and/or signalling can contribute to the development of the obese and T2DM phenotype. The incretin hormones glucagon-like peptide-1 and glucose-dependent insulinotropic hormone augment glucose-mediated insulin secretion. Peptide YY is released from the gut post-prandially and acts primarily as a satiety signal. Recently studies have highlighted a role for PYY in regulati

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Stump, Madeliene. "The role of brain PPAR[gamma] in regulation of energy balance and glucose homeostasis." Diss., University of Iowa, 2017. https://ir.uiowa.edu/etd/6000.

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The Peroxisome Proliferator-Activated Receptor gamma (PPARγ), a master regulator of adipogenesis, has been shown to influence energy balance through its actions in the brain rather than in the adipose tissue alone. Deletion of PPARγ in mouse brain results in resistance to weight gain in response to high fat diet. Activation of PPARγ leads to change in the firing pattern of melanocortin system neurons (POMC and AgRP), which are critical for energy homeostasis. To determine the effects of modulation of brain PPARγ on food intake and energy expenditure we generated a novel transgenic mouse model

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Aatsinki, S. M. (Sanna-Mari). "Regulation of hepatic glucose homeostasis and Cytochrome P450 enzymes by energy-sensing coactivator PGC-1α". Doctoral thesis, Oulun yliopisto, 2015. http://urn.fi/urn:isbn:9789526208053.

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Abstract Peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) is a master regulator of energy metabolism and mitochondrial biology in high-energy cell types and tissues. The regulation of PGC-1α is versatile, and both transcriptional and post-transcriptional mechanisms play major roles. External stimuli affect PGC-1α-regulation which in turn adapts cellular signals to meet them. For example, conditions like fasting and diabetes mellitus (DM) are known to activate PGC-1α expression in the liver, resulting in enhanced de novo glucose production, gluconeogenesis. In the present st

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Eftychidis, Vasileios. "Elucidating the principal role of cholecystokinin neurons of the ventromedial hypothalamic nucleus in energy homeostasis." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:906a0aa6-847a-43b8-a527-458252aca825.

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Abstract (sommario):

The central nervous system (CNS) has a crucial role in the maintenance of energy homeostasis by orchestrating a plethora of signals from peripheral organs about the state of energy stores and the current energy intake needed to match energy expenditure. These signals converge into the hypothalamic regions and its complex local circuitry. CNS-derived cholecystokinin (CCK) is acting at central level to modulate energy balance by regulating the neuronal activity of hypothalamic neuronal populations that regulate food intake, energy storage and consumption. Moreover, our recent published work iden

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Birkenfeld, Andreas L. [Verfasser]. "The role of natriuretic peptides in the regulation of energy metabolism, lipid- and glucose homeostasis / Andreas L. Birkenfeld." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2013. http://d-nb.info/1035182424/34.

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Libri sul tema "Energy and glucose homeostasis"

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Mladen, Vranic, Efendić Suad, and Hollenberg Charles H. 1930-, eds. Fuel homeostasis and the nervous system. Plenum Press, 1991.

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institutet, Karolinska, ed. Food deprivation and glucose homeostasis in hemorrhagic stress. [s.n.], 1987.

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1950-, Pagliassotti Michael J., Davis Stephen N. 1955-, and Cherrington Alan 1946-, eds. The role of the liver in maintaining glucose homeostasis. Landes, 1994.

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Polakof, Sergio. Brain glucosensing: Physiological implications. Nova Science Publishers, 2010.

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Sarabia, Vivian E. Calcium homeostasis and regulation of glucose uptake in human skeletal muscle cells in culture. National Library of Canada, 1990.

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Hodakoski, Cindy Marie. P-REX2 PH Domain Inhibition of PTEN Regulates Transformation, Insulin Signaling, and Glucose Homeostasis. [publisher not identified], 2012.

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Miller, Janette Brand. The new glucose revolution pocket guide to sugar & energy. Marlowe, 2004.

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Gema, Frühbeck, and Nutrition Society (Great Britain), eds. Peptides in energy balance and obesity. CABI Pub. in association with the Nutrition Society, 2009.

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Muromt͡sev, V. A. Medit͡sina v XXI veke: Ot drevneĭshikh tradit͡siĭ do vysokikh tekhnologiĭ. Izd-vo "Intan", 1998.

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Pool, Ontario Assessment Instrument, ed. Energy and the living cell: Draft. Minister of Education, Ontario, 1989.

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Capitoli di libri sul tema "Energy and glucose homeostasis"

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Kirchner, Henriette, Matthias Tschöp, and Jenny Tong. "GOAT and the Regulation of Energy and Glucose Homeostasis." In Ghrelin in Health and Disease. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-903-7_7.

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Kamstra, Kaj, and Alexander Tups. "Neuroendocrine Interactions in the Control of Glucose- and Energy Homeostasis." In Physiological Consequences of Brain Insulin Action. CRC Press, 2022. http://dx.doi.org/10.1201/9781003079927-5.

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Mounien, Lourdes, and Bernard Thorens. "Central Glucose Sensing and Control of Food Intake and Energy Homeostasis." In Metabolic Syndrome. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9780470910016.ch2.

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Guillaume, Maeva, Alexandra Montagner, Coralie Fontaine, Françoise Lenfant, Jean-François Arnal, and Pierre Gourdy. "Nuclear and Membrane Actions of Estrogen Receptor Alpha: Contribution to the Regulation of Energy and Glucose Homeostasis." In Sex and Gender Factors Affecting Metabolic Homeostasis, Diabetes and Obesity. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-70178-3_19.

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Alsahli, Mazen, and John E. Gerich. "Normal Glucose Homeostasis." In Principles of Diabetes Mellitus. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20797-1_2-1.

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Alsahli, Mazen, Muhammad Z. Shrayyef, and John E. Gerich. "Normal Glucose Homeostasis." In Principles of Diabetes Mellitus. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-20797-1_2-2.

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Gerich, John E., Steven D. Wittlin, and Christian Meyer. "Normal Glucose Homeostasis." In Principles of Diabetes Mellitus. Springer US, 2004. http://dx.doi.org/10.1007/978-1-4757-6260-0_2.

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Alsahli, Mazen, Muhammad Z. Shrayyef, and John E. Gerich. "Normal Glucose Homeostasis." In Principles of Diabetes Mellitus. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-18741-9_2.

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Shrayyef, Muhammad Z., and John E. Gerich. "Normal Glucose Homeostasis." In Principles of Diabetes Mellitus. Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-09841-8_2.

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Ferrannini, Ele, and Marta Seghieri. "Overview of Glucose Homeostasis." In Endocrinology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-45015-5_1.

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Atti di convegni sul tema "Energy and glucose homeostasis"

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Imad, BELKACEM. "Identifying Type 1 Diabetes from Daily Continuous Glucose Monitoring profiles Using Machine Learning." In 2024 International Conference on Electrical, Computer and Energy Technologies (ICECET). IEEE, 2024. http://dx.doi.org/10.1109/icecet61485.2024.10698385.

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Nair, Sruthi, Harshada Deshpande, Sagar Dave, Tasleem Anis, Aarya Prajapat, and Kartik Hanumante. "Mind Over Matter: Monitoring Blood Glucose Level with AI-Inspired Health Meter." In 2024 IEEE International Conference on Smart Power Control and Renewable Energy (ICSPCRE). IEEE, 2024. http://dx.doi.org/10.1109/icspcre62303.2024.10675099.

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R, Ilayaraja, Sujith R, Ganapathy V. S, and Helen R. "Enhancing Blood Glucose Monitoring Through Integrated Defected Ground Structure and Radio Frequency Techniques." In 2024 International Conference on Power, Energy, Control and Transmission Systems (ICPECTS). IEEE, 2024. https://doi.org/10.1109/icpects62210.2024.10780145.

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Devi, M. Kiruthiga, R. Deepika, K. Pooja, Akshey Ram M, and N. Ramya. "Expression of Concern for: Real Time Non-Invasive Continuous Glucose Monitoring Using AI." In 2020 International Conference on Power, Energy, Control and Transmission Systems (ICPECTS). IEEE, 2020. http://dx.doi.org/10.1109/icpects49113.2020.10702867.

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Mamadalieva, Ș., Ș. Botirov, and U. Iusupova. "The relationship of cognitive functions and glucose in experimental hypothyroidism." In "Fiziologia şi sănătatea", congresul fiziologilor. Bulletin of the Academy of Sciences of Moldova. Medical Sciences, 2025. https://doi.org/10.52692/cfzl2024.33.

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Thyroid hormones are one of the important factors for brain development and normal functioning. In thyroid dysfunction, regardless of age, defects in the body's cognitive functions have been found. This study examines the relationship between impaired cognitive function associated with the presence of thyroid dysfunction and its changes in glucose homeostasis. For this purpose, laboratory rats with a weight of 180-220 g were diagnosed with hypothyroidism, their ability to find their way in a special maze was studied, and the function of the brain was evaluated. It was found that the average ti

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Gronda, Edoardo, Massimo Iacoviello, and Arduino Arduini. "Sodium glucose co-transporter inhibitors put cardiovascular medicine at a crossroads – Cardiorenal interaction and clinical implications." In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.215g.

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Heart and kidney have essential homeostatic role in circulation balance to cope with variation of body biology needs, however the two organs physiology is designed to satisfy opposing demands. The heart has to provide with oxygen and nutrition of different organs and apparatus based on intercurrent demand without any predeﬁned schedule. In contrast, the kidneys have to adapt the body’s ﬂuid and electrolyte content to cope with changes in the internal and external environment. The heart and kidney function confront different physiological needs, namely the energy the body requires versus the bo

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Ahmed, Sumaya, and Nasser Rizk. "The Expression of Bile Acid Receptor TGR5 in Adipose Tissue in Diet-Induced Obese Mice." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0212.

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Bile acids are significant physiological factors for digestion, solubilization, absorption, toxic metabolites and xenobiotics. In addition, bile acids are responsible of signal transduction as well as metabolic regulation that activate several receptors such as farnesoid X receptor (FXR) and the membrane G-protein receptor 5 (TGR5). Activation of TGR5 by bile acids is associated with prevention of obesity as well as ameliorating the resistance to insulin via increasing energy expenditure. The objective of this research is to investigate TGR5 gene expression level in different fat depots includ

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Belmon, Anchana P., and Jeraldin Auxillia. "An Unprecedented PSO-PID Optimized Glucose Homeostasis." In 2020 International Conference on Smart Technologies in Computing, Electrical and Electronics (ICSTCEE). IEEE, 2020. http://dx.doi.org/10.1109/icstcee49637.2020.9277306.

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Kim, Jaeyeon, Gerald M. Saidel, John P. Kirwan, and Marco E. Cabrera. "Computational Model of Glucose Homeostasis During Exercise." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.260736.

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Kim, Jaeyeon, Gerald M. Saidel, John P. Kirwan, and Marco E. Cabrera. "Computational Model of Glucose Homeostasis During Exercise." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4397397.

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Rapporti di organizzazioni sul tema "Energy and glucose homeostasis"

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Puigserver, Pere. Maintenance of Glucose Homeostasis through Acetylation of the Metabolic Transcriptional Coactivator PGC-1alpha. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada467976.

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Puigserver, Pere. Maintenance of Glucose Homeostasis Through Acetylation of the Metabolic Transcriptional Coactivator PGC1-alpha. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada551301.

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Sosa Munguía, Paulina del Carmen, Verónica Ajelet Vargaz Guadarrama, Marcial Sánchez Tecuatl, Mario Garcia Carrasco, Francesco Moccia, and Roberto Berra-Romani. Diabetes mellitus alters intracellular calcium homeostasis in vascular endothelial cells: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0104.

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Review question / Objective: What are the effects of diabetes mellitus on the calcium homeostasis in vascular endothelial cells? -To describe the effects of diabetes on the mechanisms that regulate intracellular calcium; -To describe other molecules/mechanisms that alters intracellular Ca2+ homeostasis. Condition being studied: Diabetes mellitus is a pathology with a high incidence in the population, characterized by an increase in blood glucose. People with diabetes are 2-4 times more likely to suffer from a cardiovascular complication, such as total or partial loss of sight, myocardial infar

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frydman, judith. Mechanism and function of the chaperonin from Methanococcus maripaludis: implications for archaeal protein homeostasis and energy production. Office of Scientific and Technical Information (OSTI), 2018. http://dx.doi.org/10.2172/1429063.

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Chen, Jiankun, Yingming Gu, Lihong Yin, et al. Network meta-analysis of curative efficacy of different acupuncture methods on obesity combined with insulin resistance. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0075.

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Review question / Objective: Population：Patients diagnosed as obesity with insulin resistance. Obesity reference: Consensus of experts on the Prevention and treatment of adult obesity in China in 2011 and Consensus of Chinese experts on medical nutrition therapy for overweight/obesity in 2016 were developed by the Obesity Group of Chinese Society of Endocrinology(CSE); BMI≥28. IR reference: According to the Expert opinions on insulin resistance evaluation published by Chinese Diabetes Society, HOMA-IR≥2.68 is regarded as the standard for the diagnosis of IR. Regardless of age, gender and cours

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Corscadden, Louise, and Anjali Singh. Metabolism And Measurable Metabolic Parameters. ConductScience, 2022. http://dx.doi.org/10.55157/me20221213.

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Metabolism is the sum of chemical reactions involved in sustaining the life of organisms.[1] It constantly provides your body with the energy to perform essential functions. The process is categorized into two groups:[2] Catabolism: It’s the process of breaking down molecules to obtain energy. For example, converting glucose to pyruvate by cellular respiration. Anabolism: It’s the process of synthesis of compounds required to run the metabolic process of the organisms. For example, carbohydrates, proteins, lipids, and nucleic acids.[2] Metabolism is affected by a range of factors, such as age,

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Suksom, Daroonwan, Chaicharn Deerochanawong, Tanaka, Hirofumi, and Witid Mitranun. The comparison effects of aerobic continuous training and aerobic interval training on glycemic control and endothelial function in type 2 diabetes mellitus. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.71.

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The objective of this study was to compare the effects of continuous aerobic exercise training (CON) and interval aerobic exercise training (INT) on glycemic control and endothelium-dependent vasodilatation in patients with type 2 diabetes. A total of 43 participants with type 2 diabetes aged 50-70 years were stratified in parallel-group study and randomly allocated to the sedentary control, continuous aerobic training (CON), and interval aerobic training ( INT) groups. Exercise training programs were designed to yield the same energy expenditure/exercise session and included walking on treadm

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Cahaner, Avigdor, Sacit F. Bilgili, Orna Halevy, Roger J. Lien, and Kellye S. Joiner. effects of enhanced hypertrophy, reduced oxygen supply and heat load on breast meat yield and quality in broilers. United States Department of Agriculture, 2014. http://dx.doi.org/10.32747/2014.7699855.bard.

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Original objectivesThe objectives of this project were to evaluate the growth performance, meat yield and quality attributes of broiler strains widely differing in their genetic potential under normal temperature vs. warm temperature (short and long-term) conditions. Strain differences in breast muscle accretion rate, metabolic responses under heat load and, gross and histopathological changes in breast muscle under thermal load was also to be characterized. BackgroundTremendous genetic progress has been made in broiler chicken growth rate and meat yield since the 1950s. Higher growth rate is

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Gothilf, Yoav, Roger Cone, Berta Levavi-Sivan, and Sheenan Harpaz. Genetic manipulations of MC4R for increased growth and feed efficiency in fish. United States Department of Agriculture, 2016. http://dx.doi.org/10.32747/2016.7600043.bard.

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Abstract (sommario):

The hypothalamic melanocortin system plays a central role in the regulation of food consumption and energy homeostasis in mammals. Accordingly, our working hypothesis in this project was that genetic editing of the mc4r gene, encoding Melanocortin Receptor 4 (MC4R), will enhance food consumption, feed efficiency and growth in fish. To test this hypothesis and to assess the utility of mc4r editing for the enhancement of feed efficiency and growth in fish, the following objectives were set: Test the effect of the mc4r-null allele on feeding behavior, growth, metabolism and survival in zebrafish.

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Boisclair, Yves R., and Arieh Gertler. Development and Use of Leptin Receptor Antagonists to Increase Appetite and Adaptive Metabolism in Ruminants. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7697120.bard.

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Abstract (sommario):

Objectives The original project had 2 major objectives: (1) To determine the effects of centrally administered leptin antagonist on appetite and adaptive metabolism in the sheep; (2) To develop and prepare second-generation leptin antagonists combining high binding affinity and prolonged in vivo half-life. Background Periods of suboptimal nutrition or exaggerated metabolic activity demands lead to a state of chronic energy insufficiency. Ruminants remain productive for a surprisingly long period of time under these circumstances by evoking adaptations sparing available energy and nutrients. Th

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