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Tesi sul tema "Eotaxin"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 28 gennaio 2023

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1

Pope, Samuel M. "Specific Role of Eotaxin-1 and Eotaxin-2 in Allergic Pulmonary Eosinophilia." Cincinnati, Ohio : University of Cincinnati, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=ucin1098472372.

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2

Benarafa, Charaf. "Equine eotaxin : cloning, expression and biological activity." Thesis, Royal Veterinary College (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394942.

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3

Watanabe, Kimiko. "Eotaxin -1, -2 and -3 generation by different cell types." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404880.

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4

Mariano, Nadia Sabrina. "Estudo da resposta inflamatoria pulmonar alergica em ratos expostos a enterotoxina estafilococica do tipo A (SEA)." [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308954.

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Orientadores: Edson Antunes, Ivani Aparecida de Souza<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-14T02:35:58Z (GMT). No. of bitstreams: 1 Mariano_NadiaSabrina_M.pdf: 621386 bytes, checksum: b01cd4d186fa15a5e8ea3c7657df604a (MD5) Previous issue date: 2009<br>Resumo: O Staphylococcus aureus é um tipo de bactéria gram-positiva que produz e secreta uma série de enterotoxinas com propriedade imunomoduladoras. Entretanto, pouco é conhecido sobre os mecanismos envolvidos na exacerbação do influxo celular observ

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5

Bodman, Lee Graham. "The effect of glycosylation on the biological activity of eotaxin and related chemokines." Thesis, Imperial College London, 2004. http://hdl.handle.net/10044/1/11889.

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6

Ghaffar, Omar. "Constitutive and cytokine-stimulated expression of eotaxin by human airway smooth muscle cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0024/MQ50776.pdf.

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7

Menzies-Gow, Andrew Neil. "The roles of interleukin-5 and eotaxin in oesinophil recruitment, maturation and activation." Thesis, Imperial College London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.429161.

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8

Kirchem, Antje. "Investigation of the signalling pathways coupled to the eotaxin receptor CCR3 in human eosinophils." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406094.

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9

Konikoff, Michael R. "Non-Invasive Biomarkers of Eosinophilic Esophagitis: Blood Eosinophil Level, Eosinophil-Derived Neurotoxin, and Eotaxin-3." Cincinnati, Ohio : University of Cincinnati, 2006. http://www.ohiolink.edu/etd/view.cgi?acc_num=ucin1148043525.

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Thesis (M.S.)--University of Cincinnati, 2006.<br>Advisor: Dr. James E. Heubi. Title from electronic thesis title page (viewed June 3, 2009). Includes abstract. Keywords: Eosinophilic esophagitis; biomarker; eosinophil; eosinophil-derived neurotoxin; eotaxin-3. Includes bibliographical references.

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10

Lieschke, Simone [Verfasser]. "Untersuchung der Rolle von Eotaxin-1 nach ischämischem Schlaganfall im Mausmodell: Neuroprotektion, Neuroregeneration oder destruktiv-proinflammatorische Rolle? / Simone Lieschke." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2021. http://d-nb.info/1232492825/34.

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11

Waddell, Amanda B. "Role of macrophages and eosinophils in inflammatory bowel diseases." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1327949608.

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12

Stenin, Igor [Verfasser]. "Die Freisetzung von IL-4, IL-13, IL-33, sST2 und Eotaxin 3 nach nasaler Allergenprovokation bei Probanden mit allergischer Rhinitis / Igor Stenin." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2016. http://d-nb.info/1100688846/34.

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13

Kobayashi, Yoshiki. "Physiological level of 15-deoxy-Δ[12,14]-prostaglandin J2 prime eotaxin-induced chemotaxis on human eosinophils through peroxisome proliferator-activated receptor-γ ligation". Kyoto University, 2007. http://hdl.handle.net/2433/135911.

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14

Carvalho, Gerson da Silva. "Caracterizaçāo das citocinas na doença de Machado Joseph." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2016. http://hdl.handle.net/10183/150989.

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A Doença de Machado Joseph(DMJ) é uma doença genética autossômica dominante de início na vida adulta que afeta a coordenação motora e cursa com sintomas neurodegenerativos. É causada por uma expansão da repetição CAG no gene ATXN3. Há várias hipóteses a respeito da sua fisiopatogenia, e uma delas envolve a resposta inflamatória. O objetivo deste estudo foi descrever as concentrações séricas das citocinas em indivíduos sintomáticos, assintomáticos e compará-los com os controles saudáveis. Após a confirmação molecular dos pacientes e controles pareados por sexo e idade, os indivíduos foram convi

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15

Su, Yung-Chang University of New South Wales &amp Garvan Institute of Medical Research St Vincent's Clinical School UNSW. "Immune regulation in mouse models of allergic asthma." Awarded by:University of New South Wales & Garvan Institute of Medical Research. St. Vincent's Clinical School, 2006. http://handle.unsw.edu.au/1959.4/26978.

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Allergic asthma is an immunological disease, mediated by CD4+ Th2 cells, and its prevalence has increased over recent decades. Features of allergic asthma include airway hyperresponsiveness (AHR), airway eosinophilia, excessive airway mucus production, and increased IgE and Th2 cytokine levels. Airway remodeling with pulmonary fibrosis is noted in the progress of asthma. In this thesis, a murine model of allergic asthma was used to investigate the effect of cyclophosphamide (CY) on asthma and the involvement of regulatory T cells (Treg), and the role of Granulocyte-macrophage colony stimulatin

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16

Carmo, Lívia Andressa Silva do. "Avaliação ultraestrutural da mobilização do CD63 em eosinófilos humanos estimulados com eotaxina ou TNF-\03B1." reponame:Repositório Institucional da FIOCRUZ, 2014. https://www.arca.fiocruz.br/handle/icict/12865.

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Made available in DSpace on 2016-02-26T13:34:42Z (GMT). No. of bitstreams: 2 livia_carmo_ioc_mest_2014.pdf: 120790006 bytes, checksum: 2a0bcc88a34d66489c438b53fd76cf18 (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2016-01-13<br>Os eosinófilos são leucócitos que estão envolvidos em respostas inflamatórias, alérgicas e imunoreguladoras, através da secreção de proteínas catiônicas e citocinas armazenadas em seus grânulos específicos. Os mecanismos de secreção podem ocorrer por: (i) desgranulação por piecemeal (PMD), caraterizada pelo tra

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17

Lintomen, Leticia. "Estudo da inibição do oxido nitrico sobre funções de eosinofilos humanos estimulados com eotaxina e RANTES in vitro." [s.n.], 2008. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308929.

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Orientador: Edson Arantes<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-11T19:45:11Z (GMT). No. of bitstreams: 1 Lintomen_Leticia_D.pdf: 1121941 bytes, checksum: 2c92a8ddfce83c01ed6c71e524096e0b (MD5) Previous issue date: 2008<br>Resumo: Os eosinófilos participam da patogênese de várias doenças inflamatórias, incluindo infecções parasíticas e doenças alérgicas. Dentre as doenças alérgicas, a eosinofilia tem presença marcante na asma. Atualmente, a asma afeta 300 milhões de indivíduos no mundo, sendo considerada u

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18

Costa, Gislaine Gomes da. "Avaliação da habilidade quimiotaxica e da adesão de eosinofilos de pacientes com rinite alergica : efeito da eotaxina e interleucina-5." [s.n.], 2004. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308952.

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Orientador: Edson Antunes<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-04T02:40:18Z (GMT). No. of bitstreams: 1 Costa_GislaineGomesda_M.pdf: 3654162 bytes, checksum: ae2e21b58ce18e882aab17b0529a6576 (MD5) Previous issue date: 2004<br>Resumo: Neste estudo, investigamos se a IL-5, a eotaxina e a dexametasona modulam a migração e adesão de eosinófilos obtidos de indivíduos sadios e de pacientes com rinite alérgica. O ensaio de quimiotaxia e de adesão foi realizado com eosinófilos de sangue periférico usando-s

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19

Krisiukėnienė, Algirda. "Rūkymo sąlygoti imuninio atsako ypatumai sergant astma." Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2009. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2009~D_20090604_110354-67472.

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Astma-lėtinė kvėpavimo takų liga, pasireiškianti dusulio ar kosulio epizodais bei padidėjusiu bronchų reaktyvumu. Eksperimentiniai bei klinikiniai tyrimai parodė, kad lėtinis neinfekcinis kvėpavimo takų uždegimas- svarbiausia astmos patogenezės grandis, lemianti šios ligos klinikinę eigą, sunkumą bei skiriamo gydymo efektyvumą. Nėra aišku, ar alerginė bei nealerginė astma skiriasi ne tik klinikiniu pasireiškimu, bet ir patomorfologiniais bei patofiziologiniai kvėpavimo takų pakitimais. Rūkymas- svarbus veiksnys, sukeliantis reikšmingus pokyčius kvėpavimo takų gleivinėje. Tačiau kaip rūkymas įt

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20

Castro, Mirian Cabral Moreira de. "Perfil de expressão gênica de il-5, gm-csf, tgf-1, eotaxina na poliposenasossinusal eosinofílica pós-cirurgia endoscópica e efeito da mitomicina c tópica." Universidade Federal de Minas Gerais, 2010. http://hdl.handle.net/1843/BUOS-8GRNKX.

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Nasosinusal polyposis is a degeneration of the mucosa of the nose and paranasal sinuses with the participation of several inflammatory cytokines. Objective: To compare the differences in the expression of inflammatory genes in eosinophilic nasosinusal polyposis prior and after the topical application of Mitomycin C in the maxillary sinus, and to relate it to their expression in normal tissue. Casuistic and Method: twenty patients with nasosinusal polyposis and six patients with pituitary adenoma were selected and underwent endoscopic surgical treatment. Maxillary sinus mucosa was biopsied at s

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21

Barthel, Thomas [Verfasser], and Christoph [Akademischer Betreuer] Garlichs. "Einfluß von Statinen auf die Expression von TGF-ß1, VAP-1, Eotaxin3 und MIG als inflammatorische Trigger humaner hochgradig stenosierter und verkalkter Aortenklappen / Thomas Barthel. Betreuer: Christoph Garlichs." Erlangen : Universitätsbibliothek der Universität Erlangen-Nürnberg, 2013. http://d-nb.info/1034425714/34.

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22

Hollande, Clémence. "Rôle de dipeptidyl peptidase-4 dans la régulation du trafic leucocytaire au cours du carcinome hépatocellulaire." Thesis, Paris 6, 2017. http://www.theses.fr/2017PA066446/document.

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La modification post-traductionnelle des chimiokines par la dipeptidyl peptidase-4 (DPP4 ou CD26) régule négativement le trafic des lymphocytes, et son inhibition améliore la migration des lymphocytes T et l'immunité anti-tumorale en préservant la forme fonctionnelle de CXCL10. En étendant ces résultats initiaux aux humains et à un modèle préclinique de carcinome hépatocellulaire, nous avons découvert un nouveau mécanisme par lequel l'inhibition de DPP4 améliore les réponses anti-tumorales par le recrutement des éosinophiles. Plus précisément, l'administration d'inhibiteurs de DPP4 (DPP4i) con

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23

Manns, Johanna [Verfasser]. "NNY-Eotaxin-CCL11 : Untersuchungen zur Auswirkung der N-terminalen Modifikation des Chemokins Eotaxin-CCL11 bezüglich seiner Rezeptorspezifität und seiner Stabilität / vorgelegt von Johanna Manns." 2006. http://d-nb.info/983425787/34.

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24

Stangenberg, Stefanie. "Rhinoviren stimulieren die Rantes- und Eotaxin-Produktion in nasalen Fibroblasten." 2006. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=016696067&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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25

Pethe, Wolfram. "Zelluläre Quellen und Bedeutung von Eotaxin-2 bei der chronisch polypösen Sinusitis /." 2006. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=015026846&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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26

"Intracellular signal transduction mechanisms regulating apoptosis and eotaxin release of human eosinophils." 2001. http://library.cuhk.edu.hk/record=b6073372.

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Zhang Jiping.<br>Thesis (Ph.D.)--Chinese University of Hong Kong, 2001.<br>Includes bibliographical references (p. 157-179).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Mode of access: World Wide Web.<br>Abstracts in English and Chinese.

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27

LEE, HSI-MING, and 李錫銘. "Suppressive effects of formoterol and salmeterol on eotaxin-1 in bronchial epithelial cells." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/uks939.

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碩士<br>嘉南藥理科技大學<br>生物科技系曁研究所<br>97<br>Eotaxin-1 (CCL11), an eosinophil-specific C-C chemokine, is a potent chemoattractant for mobilization of eosinophils into airways after allergic stimulation. Eotaxin-1 could recruit eosinophils into inflammatory sites, and play a role in the pathogenesis of asthma. Formoterol and salmeterol are two inhaled long acting β2adrenoceptor agonists, widely used for the local treatment of asthma. However, little is known about their effects on the eotaxin-1 expression of bronchial epithelial cells. BEAS-2B cells were stimulated by adding IL-4 with or without 2h pre

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28

Essen, Eddy, and 張一旋. "Investigation on the mechanism of production of eotaxin in mice infected with Angiostrongylus cantonensis." Thesis, 2004. http://ndltd.ncl.edu.tw/handle/79923329774267268607.

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博士<br>高雄醫學大學<br>醫學研究所博士班<br>92<br>There is evidence showing that eosinophils play an important role in non-permissive host reactions against Angiostrongylus cantonensis after infection. Eotaxin is the strongest mediator involved in eosinophil chemotaxis and chemokinesis, which selectively recruits eosinophils. In our study, all eotaxin concentrations weekly in the cerebrospinal fluid (CSF) of the infected mice were significantly higher than those in the serum and got the peak level at third weeks. The increases in eosinophils in CSF paralleled the kinetics of the change in eotaxin levels in th

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29

Spautz, Barbara Veronika. "Vergleichende Untersuchung der TNF-a-induzierten [TNF-alpha-induzierten] Eotaxin-CCL11-Expression durch primäre Fibroblasten." 2008. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=017646056&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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30

Li, Chao Yi, and 李昭儀. "Studies on the Relationship between the CC Chemokine Eotaxin-1 and the Progression of Osteoarthritis." Thesis, 2002. http://ndltd.ncl.edu.tw/handle/36317093020888327016.

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碩士<br>台北醫學院<br>生物醫學技術研究所<br>90<br>We studied on the relationship between CC chemokine Eotaxin-1 and the progression of osteoarthritis. The concentrations of RANTES, MCP-1a and Eotaxin-1 in patients’ plasma with osteoarthritis (OA) are higher than those in normal plasma. Both the concentrations of RANTES and MCP-1a were decreased during injection of hyaluronan three times in patients with OA, while the concentration of Eotaxin-1 was significantly decreased at first time then it reversed. By reverse transcription-polymerase chain reaction (RT-PCR) and enzyme-linked immunosorbat assay (ELISA), we

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31

Chen, Hong-Kong, and 陳杭港. "Studies of the molecular mechanisms of chemokine eotaxin-1 on the metalloproteinases expression in human chondrocyte." Thesis, 2004. http://ndltd.ncl.edu.tw/handle/16466985317383963137.

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碩士<br>臺北醫學大學<br>生物醫學技術研究所<br>92<br>Osteoarthritis (OA) is characterized by loss of the functional integrity of articular cartilage due to an imbalance between catabolic and anabolic chondrocyte activity. Degradation of the collagenous extracellular matrix by metalloproteases (MMPs) plays an important role in the pathogenesis of osteoarthritis (OA). Our previous studies suggested that IL-1β、TNF-α could increase the expression of eotaxin-1 in human primary culture chondrocyte. Recently, we found that CCR3 receptor was a potential receptor for eotaxin-1 binding, and might be involved i

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32

Liu, Keniji, and 劉賢一. "Expression level of Eotaxin, MCP-1, RANTES, CCR5, CCR3 and CCR2 of Taiwanese and Aboriginal Taiwanese." Thesis, 2002. http://ndltd.ncl.edu.tw/handle/75852654725438979912.

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Jan, Ya-Yu, and 詹雅羽. "The Mechanisms of Eotaxin-2-induced toll-like receptor 4 expression in human coronary arterial endothelial cells." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/34k6zv.

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碩士<br>臺北醫學大學<br>醫學檢驗暨生物技術學系所<br>102<br>Objective: Eotaxin-2 interacts with toll-like receptor 4 (TLR4) and induces inflammatory responses in human coronary arterial endothelial cells (HCAECs), which plays an important role in atherogenesis. The objective of this study was to investigate the mechanisms involving eotaxin-2-induced TLR4 expression in HCAECs. Methods and results: Stimulation via eotaxin-2 significantly increased TLR4 mRNA level in HCAECs by real time polymerase chain reaction (RT-PCR). In addition, on eotaxin-2 treatment also increased TLR4 protein expression. An actinomycin D ch

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34

Chao, Pin-Zhir, and 趙品植. "Levels of RANTES, Eotaxin in the patients with chronic rhinosinusitis and its correlation with severity of the disease." Thesis, 2004. http://ndltd.ncl.edu.tw/handle/nc9gu9.

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碩士<br>臺北醫學大學<br>醫學研究所<br>93<br>Chronic rhinosinusitis and nasal polyps mainly develop in the ethmoid sinus, maxillary sinus and middle turbinate area, often in relation to inflammatory and allergic conditions. Their exact etiology and pathogenesis are still under debate. Histologically, inflammatory cell infiltrations of nasal mucosa predominated by eosinophils are typical for such disease, and may be due to the chemotactic activity of chemokines specific for eosinophils. This findings suggest that nasal polyp is an inflammatory growth that is controlled by the local environment and the immuno

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35

Chu, Yu-Te, and 朱育德. "The changes of serum eotaxin, eosinophil cationic protein, and eosinophil count while treating pediatric asthmatic patients with steroids." Thesis, 2006. http://ndltd.ncl.edu.tw/handle/93255409129338112410.

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碩士<br>高雄醫學大學<br>醫學研究所碩士班<br>94<br>Background: Increased serum level of eotaxin is related to asthma severity in adult study. There is little data about pediatric asthma patients with regards to the effects of oral and inhaled corticosteroids on serum eotaxin, eosinophil cationic protein (ECP) concentrations and eosinophil counts. Methods: We investigated prospectively the changes of serum level of eotaxin, eosinophil counts, and ECP after oral steroid for one week and then inhaled corticosteroids with a long–acting β2 agonist (Seretide) treatment for 2 months in the pediatric population. The d

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36

潘潔宜. "Studies on the possible signaling pathways of eotaxin-1 induced matrix metalloproteinase-3 (MMP-3) expression in human chondrocyte." Thesis, 2003. http://ndltd.ncl.edu.tw/handle/78225054534827588023.

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Karsten, Claudia. "Untersuchungen zur Potenz von respiratorischen Zellen im Hinblick auf die Expression von Eotaxin und Exotaxin-3 bei labortechnischer Simulation einer proinflammatorischen Entzündung." 2004. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=014189918&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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38

Knott, Michelle Louise. "Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri." Thesis, 2010. http://hdl.handle.net/2440/67240.

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Parasitic helminth infections are a significant problem worldwide. Some helminth species are becoming resistant to current therapies and new forms of treatment and/or vaccines are required. Nippostrongylus brasiliensis is a tissue-invasive parasitic helminth that infects rodents, and the lifecycle of this parasite is similar to that of the human hookworms. The aims of this study were to investigate primary and secondary immune responses to N. brasiliensis, focusing on the pre-lung phase of infection. The roles of cytokines, chemokines, signalling pathways and leukocytes such as eosinophils wer

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Mwanthi, Muithi. "PAK1's regulation of eosinophil migration and implications for asthmatic inflammation." Thesis, 2013. http://hdl.handle.net/1805/3786.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>More than 300 million people world-wide suffer from breathlessness, wheezing, chest tightness, and coughing characteristic of chronic bronchial asthma, the global incidence of which is on the rise. Allergen-sensitization and challenge elicits pulmonary expression of chemoattractants that promote a chronic eosinophil-rich infiltrate. Eosinophils are increasingly recognized as important myeloid effectors in chronic inflammation characteristic of asthma, although few eosinophil molecular signaling pathways have successfully been targete

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