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Letteratura scientifica selezionata sul tema "GM-CSF"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "GM-CSF"

1

Sands, Bruce E. "GM-CSF." Inflammatory Bowel Diseases 12 (April 2006): S12. http://dx.doi.org/10.1097/00054725-200604002-00024.

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2

Ratto, Alessandra, Claudio Petterino, Tullio Florio, and Federica Barbieri. "Goat anti-human GM-CSF recognizes canine GM-CSF." Veterinary Clinical Pathology 41, no. 1 (2012): 3–4. http://dx.doi.org/10.1111/j.1939-165x.2012.00407.x.

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3

&NA;. "DT388-GM-CSF." Inpharma Weekly &NA;, no. 1152 (1998): 7. http://dx.doi.org/10.2165/00128413-199811520-00012.

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4

Senzer, N., C. Bedell, and J. Nemunaitis. "OncoVEX(GM-CSF)." Drugs of the Future 35, no. 6 (2010): 449. http://dx.doi.org/10.1358/dof.2010.035.06.1500437.

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5

Senzer, N., C. Bedell, and J. Nemunaitis. "OncoVEX(GM-CSF)." Drugs of the Future 35, no. 6 (2010): 449. http://dx.doi.org/10.1358/dof.2010.35.6.1500437.

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6

McNiece, I., R. Andrews, M. Stewart, S. Clark, T. Boone, and P. Quesenberry. "Action of interleukin-3, G-CSF, and GM-CSF on highly enriched human hematopoietic progenitor cells: synergistic interaction of GM-CSF plus G-CSF." Blood 74, no. 1 (1989): 110–14. http://dx.doi.org/10.1182/blood.v74.1.110.110.

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Abstract (sommario):

Abstract Purified preparations of recombinant human granulocyte-macrophage colony-stimulating factor (GM-CSF), granulocyte CSF (G-CSF), and interleukin 3 (IL-3 or multi-CSF) alone and in combination, have been compared for their stimulatory effects on human granulocyte-macrophage colony forming cells (GM-CFC). In cultures of unseparated normal human bone marrow, the combinations of G-CSF plus IL-3 and GM-CSF plus IL-3 stimulated additive numbers of GM colonies, while GM-CSF plus G-CSF stimulated greater than additive numbers of GM colonies, compared with the sum of the colony formation obtaine

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7

McNiece, I., R. Andrews, M. Stewart, S. Clark, T. Boone, and P. Quesenberry. "Action of interleukin-3, G-CSF, and GM-CSF on highly enriched human hematopoietic progenitor cells: synergistic interaction of GM-CSF plus G-CSF." Blood 74, no. 1 (1989): 110–14. http://dx.doi.org/10.1182/blood.v74.1.110.bloodjournal741110.

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Abstract (sommario):

Purified preparations of recombinant human granulocyte-macrophage colony-stimulating factor (GM-CSF), granulocyte CSF (G-CSF), and interleukin 3 (IL-3 or multi-CSF) alone and in combination, have been compared for their stimulatory effects on human granulocyte-macrophage colony forming cells (GM-CFC). In cultures of unseparated normal human bone marrow, the combinations of G-CSF plus IL-3 and GM-CSF plus IL-3 stimulated additive numbers of GM colonies, while GM-CSF plus G-CSF stimulated greater than additive numbers of GM colonies, compared with the sum of the colony formation obtained with ea

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8

Reed, Jacquelyn A., Machiko Ikegami, Eli R. Cianciolo, et al. "Aerosolized GM-CSF ameliorates pulmonary alveolar proteinosis in GM-CSF-deficient mice." American Journal of Physiology-Lung Cellular and Molecular Physiology 276, no. 4 (1999): L556—L563. http://dx.doi.org/10.1152/ajplung.1999.276.4.l556.

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Surfactant proteins and phospholipids accumulate in the alveolar spaces and lung tissues of mice deficient in granulocyte-macrophage colony-stimulating factor (GM-CSF), with pathological findings resembling the histology seen in the human disease pulmonary alveolar proteinosis (PAP). Previous metabolic studies in GM-CSF-deficient [GM(−/−)] mice indicated that defects in surfactant clearance cause the surfactant accumulation in PAP. In the present study, GM(−/−) mice were treated daily or weekly with recombinant mouse GM-CSF by aerosol inhalation or intraperitoneal injection for 4–5 wk. Lung hi

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9

Tazawa, Ryushi, and Koh Nakata. "GM-CSF Therapy for Pulmonary Alveolar Proteinosis." Nihon Naika Gakkai Zasshi 99, no. 7 (2010): 1623–27. http://dx.doi.org/10.2169/naika.99.1623.

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10

Frossard, Jean Louis, Ashok K. Saluja, Nicolas Mach, et al. "In vivo evidence for the role of GM-CSF as a mediator in acute pancreatitis-associated lung injury." American Journal of Physiology-Lung Cellular and Molecular Physiology 283, no. 3 (2002): L541—L548. http://dx.doi.org/10.1152/ajplung.00413.2001.

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Severe pancreatitis is frequently associated with acute lung injury (ALI) and the respiratory distress syndrome. The role of granulocyte-macrophage colony-stimulating factor (GM-CSF) in mediating the ALI associated with secretagogue-induced experimental pancreatitis was evaluated with GM-CSF knockout mice (GM-CSF −/−). Pancreatitis was induced by hourly (12×) intraperitoneal injection of a supramaximally stimulating dose of the cholecystokinin analog caerulein. The resulting pancreatitis was similar in GM-CSF-sufficient (GM-CSF +/+) control animals and GM-CSF −/− mice. Lung injury, quantitated

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Tesi sul tema "GM-CSF"

1

Bailie, Karen Elizabeth Margaret. "G-CSF and GM-CSF : effects on neonatal neutrophils." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.482043.

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2

Chevalier, Anne Sophie. "Utilisation thérapeutique du G-CSF et du GM-CSF en hématologie." Paris 5, 1993. http://www.theses.fr/1993PA05P248.

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Hallsworth, Matthew Pearce. "GM-CSF and eosinophil survival in asthma." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341883.

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4

Bernstone, Laura. "Characterisation of HIV-1 infection and M-CSF and GM-CSF macrophages." Thesis, University of Oxford, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572833.

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Macrophages are a natural target cell for HIV-1 infection, and they contribute to the development of disease as they are important for transmission, dissemination and persistence of the virus in an infected patient. Macrophages are less well-studied than T cells and cell lines in relation to HIV-1 infection, yet macrophages are highly specialised and key aspects of the HIV-1 life cycle in these cells are already known to differ compared to other cell types. HIV-1 entry into macrophages has been suggested to occur by macropinocytosis, however the entry route in these cells has not been fully ch

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5

Lin, Tony Wei Ting. "The role of GM-CSF/G-CSF & BKLF in the development of atherosclerosis." Thesis, The University of Sydney, 2013. http://hdl.handle.net/2123/10407.

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The studies described in this thesis are aimed to investigate the role of the pro-inflammatory cytokine GM-CSF/G-CSF, transcriptional factor KLF3 and psychological stress in KLF3 deficiency mice on neointimal formation, a common feature of atherosclerosis with the aid of perivascular collar model on murine model. The present study supports an important role for GM/CSF-G/CSF and KLF in atherosclerosis. There is now overwhelming evidence that the monocyte/macrophage has a crucial role in the initiation and progression of atherosclerotic plaque and this has led to the view that the recruitment an

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6

Mirabella, Fabio. "Regulation of the human IL-3/GM-CSF locus." Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.487744.

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Abstract (sommario):

The human Interleukin-3 (IL-3) and Granulocyte-Macrophage Colony-Stimulating-Factor (GM-CSF) genes are two closely linked cytokine genes that are located within a conserved cytokine gene cluster. They are expressed in an inducible tissue-specific manner in a variety of haemopoietic cell types, and they contribute to the regulation of inflammatory responses arid haemopoiesis. For this reason their expression is strictly regulated. The experiments in this thesis describe an investigation into the factors and DNA elements involved in the control and regulation of these genes during T cell develop

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7

Grant, Olivia M. "GM-CSF Stress-Induced Priming of the Dendritic Cell." Ohio Dominican University Honors Theses / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=oduhonors1449522036.

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8

Adkins, Karissa Kathleen 1971. "Glucocorticoid regulation of GM-CSF in bronchial epithelial cells." Diss., The University of Arizona, 1998. http://hdl.handle.net/10150/282844.

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Inflammation plays a central role in the pathogenesis of asthma. Glucocorticoids are first line antiinflammatory therapy in the treatment of asthma and are effective inhibitors of inflammatory cytokines. Clinical data demonstrate that granulocyte-macrophage colony-stimulating factor (GM-CSF) production by airway epithelial cells may be an important target of inhaled glucocorticoid therapy. In this study, the regulatory mechanisms of GM-CSF expression by interleukin-1β (IL-1β) and the synthetic glucocorticoid dexamethasone (DEX) were examined in the BEAS-2B human bronchial epithelial cell line.

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9

Aziz, Khalil Abdul. "Influence of GM-CSF and G-CSF on the mutual interactions between platelets and neutrophils." Thesis, University of Liverpool, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241473.

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10

Osborne, Cameron Stuart. "Transcriptional regulation of the GM-CSF gene in T lymphocytes /." Title page, contents and summary only, 1996. http://web4.library.adelaide.edu.au/theses/09PH/09pho81.pdf.

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Libri sul tema "GM-CSF"

1

M, Marty, ed. Manual of GM-CSF. Blackwell Science, 1996.

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2

Weber, Boris. Mobilisierung von hämatopoetischen Stammzellen mit GM-CSF und G-CSF bei Patienten nach Polychemotherapie. [s.n.], 1993.

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3

Howard, Scarffe J., Royal Society of Medicine Services (Great Britain), Sandoz Pharmaceuticals, and Schering-Plough Corporation, eds. Breakthrough in cytokine therapy: An overview of GM-CSF. Royal Society of Medicine Services, 1991.

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4

van, Furth Ralph, ed. Hemopoietic growth factors and mononuclear phagocytes. New York, 1993.

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5

Gregory, Bock, and Goode Jamie, eds. The molecular basis of cellular defence mechanisms. Wiley, 1997.

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6

Gorin, N. C. Molgramostim Gm-csf: Possibilities And Perspectives (International Congress & Symposium). Royal Society of Medicine Press Ltd, 1992.

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7

Wang, Xuan-Ding. Analysis of GM-CSF and GM-CSF/IL-3/IL-5 receptor common beta chain in patients with pulmonary alveolar proteinosis. 1999.

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8

Stockdreher, Karin. Regulation von Aktivations- und Adhäsionsantigenen auf Lymphozyten und Monozyten durch GM-CSF und G-CSF in vivo. 1996.

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9

Breisgau, Universität Freiburg im, ed. Untersuchungen zu GM-CSF, TNF-[alpha], IL-1 und Genexpressionsstudien bei JMML. 2002.

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10

Rothchild, Alissa. Antimicrobial Roles for iNKT Cells and GM-CSF in Mycobacterium Tuberculosis Infection. 2014.

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Capitoli di libri sul tema "GM-CSF"

1

Baum, H. "GM-CSF." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_1306-1.

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2

Baum, H. "GM-CSF." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_1306.

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3

Williams, William V. "GM-CSF Antagonists." In New Drugs for Asthma, Allergy and COPD. KARGER, 2001. http://dx.doi.org/10.1159/000062175.

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4

Dranoff, Glenn, and Kenneth F. May. "GM-CSF and Whole Cells." In Cancer Therapeutic Targets. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4419-0717-2_37.

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5

Dranoff, Glenn, and Kenneth F. May. "GM-CSF and Whole Cells." In Cancer Therapeutic Targets. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6613-0_37-2.

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6

Hartung, Thomas, Sonja von Aulock, and Albrecht Wendel. "Growth Factors G-CSF and GM-CSF: Clinical Options." In Multiple Organ Failure. Springer New York, 2000. http://dx.doi.org/10.1007/978-1-4612-1222-5_64.

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7

Link, H. "Therapie mit den hämatopoetischen Wachstumsfaktoren G-CSF und GM-CSF." In Kompendium Internistische Onkologie. Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/3-540-31303-6_124.

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8

Würfel, Wolfgang. "G-CSF and GM-CSF: Clinical Applications in Reproductive Medicine." In In Vitro Fertilization. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-43011-9_62.

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9

Vellenga, E. "The Expression and Regulation of G-CSF and GM-CSF." In Acute Leukemias V. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-78907-6_20.

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10

Salajegheh, Ali. "Granulocyte-Macrophage and Granulocyte Colony Stimulating Factor (GM-CSF and G-CSF)." In Angiogenesis in Health, Disease and Malignancy. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-28140-7_20.

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Atti di convegni sul tema "GM-CSF"

1

Zimlich, William C., Francesca Mariani, Bernhard Muellinger, et al. "Aerosol Characterization of Nebulized GM-CSF." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5987.

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2

Tazawa, R., T. Ueda, M. Abe, et al. "Antibody Against Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF) and Inhaled GM-CSF for Pulmonary Alveolar Proteinosis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3067.

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3

Bobrova, L. A., and M. Y. Zemskova. "STUDY OF THE INTERACTION BETWEEN GRANULOCYTE-MACROPHAGE COLONY-STIMULATING FACTOR AND S100P PROTEIN ON THE MONOCYTIC LEUKEMIA CELL LINE THP-1." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-221.

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Abstract (sommario):

Granulocyte-macrophage colony-stimulating factor (GM-CSF) is a proinflammatory cytokine that regulates the differentiation of monocytes into macrophages; it can also participate in carcinogenesis. S100P is a calcium-binding protein that interacts with the RAGE receptor and assists in the processes of inflammation, proliferation, and differentiation of cells. We have found that GM-CSF binds to S100P. The biological activity of the GM-CSF/S100P complex was investigated in our research.

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4

Nakata, K., K. Ohashi, R. Tazawa, et al. "GM-CSF Inhalation Promotes the Clearance of GM-CSF Autoantibody in the Lung of Autoimmune Pulmonary Alveolar Proteinosis." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3033.

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5

Debeuf, N., C. Bosteels, K. F. A. Van Damme, et al. "Loss of GM-CSF-dependent instruction of alveolar macrophages in COVID-19 provides a rationale for inhaled GM-CSF treatment." In ERS Lung Science Conference 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/23120541.lsc-2023.137.

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6

Tazawa, R., Y. Inoue, T. Takada, et al. "Aerosolized GM-CSF Therapy of Pulmonary Alveolar Proteinosis (PAP)." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3031.

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7

Wu, H., T. Suzuki, BC Trapnell, and FX McCormack. "Alveolar Macrophage Activation in KGF Treated Mice Is Associated with Early Release of GM-CSF, Followed by GM-CSF Dependent STAT5 Phosphorylation." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3238.

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8

Chan, Jamie S., Ross Vlahos, David Haylock, Sussie Nilsson, Ivan Bertoncello, and Gary P. Anderson. "Macrophage Progenitor Recruitment And Functional Specification Is Controlled By A M-CSF/ GM-CSF/ Osteopontin Axis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3781.

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9

Khasawneh, M., D. C. Patel, and A. Ataya. "Persistent Eosinophilia Due to Subcutaneous GM-CSF Therapy in aPAP." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a4949.

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10

Suzuki, Takuji, Takuro Sakagami, J. C. van der Loo, Brenna Carey, Claudia Chalk, and Bruce Trapnell. "A Novel Cell Line (mAM-hGM-R) For Measuring The Bioactivity Of GM-CSF And Neutralizing Capacity Of GM-CSF Autoantibodies In Human Serum." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2983.

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Rapporti di organizzazioni sul tema "GM-CSF"

1

Emens, Leisha A., and Elizabeth M. Jaffee. Enhancement of an Allogeneic GM-CSF-Secreting Breast Cancer Vaccine by Immunomodulatory Doses of Cyclophosphamide and Doxorubicin. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada417167.

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Hansen, Peter J., and Zvi Roth. Use of Oocyte and Embryo Survival Factors to Enhance Fertility of Heat-stressed Dairy Cattle. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7697105.bard.

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The overall goal was to identify survival factors that can improve pregnancy success following insemination or embryo transfer in lactating dairy cows exposed to heat stress. First, we demonstrated that oocytes are actually damaged by elevated temperature in the summer. Then we tested two thermoprotective molecules for their effect on oocyte damage caused by heat shock. One molecule, ceramide was not thermoprptective. Another, insulin-like growth factor-1 (IGF) reduced the effects of heat shock on oocyte apoptosis and oocyte cleavage when added during maturation. We also used lactating cows ex

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