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1

Roehr, Bob. "Scott Hitt." BMJ 335, no. 7630 (November 29, 2007): 1160. http://dx.doi.org/10.1136/bmj.39408.718958.be.

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2

Jy, Wenche, Wei Wei Mao, Lawrence Horstman, Peter Valant, and Yeon Ahn. "A Flow Cytometric Assay of Platelet Activation Marker P-Selectin (CD62P) Distinguishes Heparin-induced Thrombocytopenia (HIT) from HIT with Thrombosis (HITT)." Thrombosis and Haemostasis 82, no. 10 (1999): 1255–59. http://dx.doi.org/10.1055/s-0037-1614371.

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Abstract (sommario):
SummaryHeparin induced thrombocytopenia (HIT) is a well-known complication of heparin administration but usually resolves upon discontinuation without sequelae. However, a small proportion of HIT patients develop thrombosis associated with HIT, designated as HITT, which is often life-threatening and may lead to gangrene and amputations. Existing laboratory methods of confirming HIT/HITT do not distinguish between HIT and HITT. We report a flow cytometric assay of platelet activation marker CD62P to distinguish the effects of addition of HIT vs. HITT plasma to normal blood. Briefly, normal whol
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3

Kondo, Kimi, D. Johnson, and Eric Wannamaker. "Heparin-Induced Thrombocytopenia and Thrombosis: Preventing your Thrombolysis Practice from Taking a HITT." Seminars in Interventional Radiology 34, no. 04 (December 2017): 409–14. http://dx.doi.org/10.1055/s-0037-1608864.

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Abstract (sommario):
AbstractHeparin-induced thrombocytopenia and thrombosis (HITT) is an under-recognized cause of deep venous thrombosis treatment failure and of complications during catheter-directed thrombolysis. After a review of HITT pathophysiology, diagnosis, and management, three different cases are presented in this article. Each case highlights subtleties and challenges of HITT diagnosis and management. An example of a practical approach to the diagnosis of HITT is presented.
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4

Donie, Donie, Yanuar Kiram, Hermanzoni Hermanzoni, and Eval Edmizal. "The Effectiveness of Footwork Exercises with the HIIT Method in Developing VO2max and Anaerobic Capacity." AL-ISHLAH: Jurnal Pendidikan 13, no. 2 (August 14, 2021): 998–1005. http://dx.doi.org/10.35445/alishlah.v13i2.803.

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The purpose of this study was to determine the development of physiological aspects of increasing aerobic and anaerobic performance in students who become badminton athletes through optimizing footwork exercises using the high-intensity interval method (HITT). This research will provide a solution for trainers in combining and optimizing footwork exercises as a technique in badminton combined with the principles of interval training to increase badminton athletes' aerobic and anaerobic capacity. This study used an experimental approach by giving footwork training treatment (HITT) to 30 Padang
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5

Sabnani, Indu, Ajaz Khan, and Patricia Tsang. "Pattern of Platelet Recovery in Predicting Outcome in Heparin-Induced Thrombocytopenia and Thrombosis." Blood 108, no. 11 (November 16, 2006): 4085. http://dx.doi.org/10.1182/blood.v108.11.4085.4085.

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Abstract (sommario):
Abstract Heparin-induced thrombocytopenia and thrombosis (HITT), a severe immune-mediated drug reaction, is often underdiagnosed. Early management of HITT with alternative anticoagulants can result in a favorable outcome. However, the morbidity and mortality associated with HITT remain high, posing challenges for patient management. We describe the clinical profile and pattern of platelet count recovery in a cohort of 34 patients diagnosed with HITT. This retrospective study consists of 164 patients screened for HITT over 18 months at our institution based on positive platelet factor 4 (PF4) I
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6

Arepally, Gowthami M., and Irene M. Mayer. "Antibodies from patients with heparin-induced thrombocytopenia stimulate monocytic cells to express tissue factor and secrete interleukin-8." Blood 98, no. 4 (August 15, 2001): 1252–54. http://dx.doi.org/10.1182/blood.v98.4.1252.

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Abstract (sommario):
Thrombosis is a life-threatening complication that occurs in a subset of patients with heparin-induced thrombocytopenia (HITT). The pathogenic mechanisms underlying the variable occurrence of thrombosis in HITT is poorly understood. It was hypothesized that monocyte activation leading to tissue factor expression may play a role in promoting a thrombogenic state in HITT. This study demonstrates that a human platelet factor 4 (PF4)/heparin-specific murine monoclonal antibody (KKO) binds to peripheral blood-derived human monocytes in a PF4-dependent manner. KKO and antibodies from patients with H
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7

Gade, Kristine, Jack Melson, Alex Jepsen, Surabhi Palkimas, Bethany Horton, and Hillary S. Maitland. "A Single Institution's Adherence to Heparin-Induced Thrombocytopenia and Thrombosis Testing Guidelines." Blood 132, Supplement 1 (November 29, 2018): 4720. http://dx.doi.org/10.1182/blood-2018-99-117132.

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Abstract (sommario):
Abstract Introduction: What was once an under-recognized clinical entity, heparin induced thrombocytopenia and thrombosis (HITT) has become a well-recognized and frequently tested disease. HITT is a serious and potentially fatal antibody mediated drug reaction to platelet factor 4 and early identification and treatment is essential. The 4T score is well-validated to guide appropriate testing of HITT where a low-probability score of 0-3 equates to a 99.8% negative predictive value for HITT2,3. In an effort to promote cost-conscious care and efficient use of healthcare resources, the American So
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8

Madhavan, Sangeetha, James W. Stinear, and Neeta Kanekar. "Effects of a Single Session of High Intensity Interval Treadmill Training on Corticomotor Excitability following Stroke: Implications for Therapy." Neural Plasticity 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/1686414.

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Abstract (sommario):
Objective. High intensity interval treadmill training (HIITT) has been gaining popularity for gait rehabilitation after stroke. In this study, we examined the changes in excitability of the lower limb motor cortical representation (M1) in chronic stroke survivors following a single session of HIITT. We also determined whether exercise-induced changes in excitability could be modulated by transcranial direct current stimulation (tDCS) enhanced with a paretic ankle skill acquisition task.Methods. Eleven individuals with chronic stroke participated in two 40-minute treadmill-training sessions: HI
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9

Shatskov, A. "Hitt. ninink- and Lat. nītor." Indo-European Linguistics and Classical Philology XXIII (June 2019): 1131–40. http://dx.doi.org/10.30842/ielcp230690152385.

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10

Lee, S. K., R. Willinsky, and K. terBrugge. "Dural Sinus Thrombosis Complicated with Heparin Induced Thrombocytopenia and Thrombosis (HITT)." Interventional Neuroradiology 8, no. 1 (March 2002): 77–80. http://dx.doi.org/10.1177/159101990200800114.

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Abstract (sommario):
A case of dural sinus thrombosis complicated with heparin induced thrombocytopenia and thrombosis (HITT) is presented. Interventional neuroradiologic procedures have potential risks of this devastating complication that is related with heparin. Clinical features and pathophysiologic mechanisms of HITT are discussed.
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11

McGowan, Kelly E., Joy Makari, Artemis Diamantouros, Claudia Bucci, Peter Rempel, Rita Selby, and William Geerts. "Reducing the hospital burden of heparin-induced thrombocytopenia: impact of an avoid-heparin program." Blood 127, no. 16 (April 21, 2016): 1954–59. http://dx.doi.org/10.1182/blood-2015-07-660001.

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Abstract (sommario):
Key Points Use of LMWH is associated with a lower risk of HIT and HITT compared with use of UFH. The Avoid-Heparin Initiative resulted in a dramatic reduction in the burden of suspected HIT, adjudicated HIT, HITT, and associated costs.
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12

Arepally, G. M., S. Kamei, K. S. Park, K. Kamei, Z. Q. Li, W. Liu, D. L. Siegel, W. Kisiel, D. B. Cines, and M. Poncz. "Characterization of a murine monoclonal antibody that mimics heparin-induced thrombocytopenia antibodies." Blood 95, no. 5 (March 1, 2000): 1533–40. http://dx.doi.org/10.1182/blood.v95.5.1533.005k01_1533_1540.

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Abstract (sommario):
Antibodies to PF4/heparin can be demonstrated in almost all patients with heparin-induced thrombocytopenia/thrombosis (HIT/HITT) and in some persons exposed to heparin who do not have clinical manifestations. The role of anti-PF4/heparin antibodies in the pathogenesis of HIT/HITT has been difficult to establish because the antibodies found in serum are generally polyclonal and polyspecific. To circumvent this problem, we developed a murine monoclonal antibody (mAb) to human (h) PF4/heparin complexes. A monoclonal IgG2bκ antibody (designated KKO) was identified that bound specifically to hPF4/h
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13

Arepally, Gowthami, Steven E. McKenzie, Xiao-Ming Jiang, Mortimer Poncz та Douglas B. Cines. "FcγRIIA H/R131 Polymorphism, Subclass-Specific IgG Anti-Heparin/Platelet Factor 4 Antibodies and Clinical Course in Patients With Heparin-Induced Thrombocytopenia and Thrombosis". Blood 89, № 2 (15 січня 1997): 370–75. http://dx.doi.org/10.1182/blood.v89.2.370.

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Abstract (sommario):
Abstract The explanation why only a subset of patients with heparin-induced thrombocytopenia (HIT) develop clinically apparent thromboses (HITT) remains uncertain. It has been proposed that platelet activation induced by cross-linking of FcγRIIA by anti-heparin/platelet factor 4 (PF4) antibodies is central to the pathogenesis of thrombosis. The observation that a common functional polymorphism of FcγRIIA, involving either an arginine (R) or histidine (H) at amino acid 131, may underlie disease susceptibility prompted us to investigate the prevalence of receptor isoforms in patients with HIT an
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14

Parney, Ian F., and David E. Steinke. "Heparin-induced thrombocytopenia and thrombosis following subarachnoid hemorrhage." Journal of Neurosurgery 93, no. 1 (July 2000): 136–39. http://dx.doi.org/10.3171/jns.2000.93.1.0136.

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Abstract (sommario):
U The authors present a case of heparin-induced thrombocytopenia and thrombosis (HITT) that occurred after aneurysmal subarachnoid hemorrhage (SAH), and they review the relevant literature. An immune-mediated syndrome, HITT is characterized by moderate thrombocytopenia and paradoxical vascular thromboses. Although it has been estimated in prospective studies that HITT occurs in between 1 and 3% of patients receiving heparin, it is underrecognized in the neurosurgical literature. In the present case, a 49-year-old woman underwent clipping of a right posterior communicating artery aneurysm after
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15

Bacsi, S., R. De Palma, G. P. Visentin, J. Gorski, and R. H. Aster. "Complexes of Heparin and Platelet Factor 4 Specifically Stimulate T Cells From Patients With Heparin-Induced Thrombocytopenia/Thrombosis." Blood 94, no. 1 (July 1, 1999): 208–15. http://dx.doi.org/10.1182/blood.v94.1.208.413a06_208_215.

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Abstract (sommario):
Heparin-induced thrombocytopenia with thrombosis (HITT) is associated with antibodies specific for complexes consisting of heparin and platelet factor 4 (PF4). Studies in individual patients with HITT have demonstrated immunoglobulin (Ig) class switching from IgM to the IgG or IgA isotypes. This transition is thought to require helper T cells, but no studies of the cellular or molecular basis of this process have yet been reported. To characterize T-cell involvement in HITT, peripheral blood mononuclear cells (PBMC) from two patients with classical HITT obtained shortly after the acute episode
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16

Tessler, Oren, Joshua Vorstenbosch, Sebastien Lalonde, Jonathan Kanevsky та Teanoosh Zadeh. "“Donʼt Let Your Flap Get HITT”". Plastic and Reconstructive Surgery 130 (листопад 2012): 110. http://dx.doi.org/10.1097/01.prs.0000421829.90421.b6.

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17

Roche, Kristina, Brian Harnett, Leona Browne, Stephanie Young, and Rufaro S. Chitsike. "The Incidence of Heparin Induced Thrombocytopenia before and after the Implementation of the Venous Thromboembolism Thromboprophylaxis Protocols within Eastern Health." Blood 134, Supplement_1 (November 13, 2019): 2447. http://dx.doi.org/10.1182/blood-2019-129340.

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Abstract (sommario):
Background: A recognized complication of exposure to heparin is the development of heparin-induced thrombocytopenia (HIT) and heparin-induced thrombocytopenia with thrombosis (HITT). The frequency of HIT in patients exposed to unfractionated heparin (UFH) is higher than those exposed to low-molecular-weight heparin (LMWH). Where the use of unfractionated heparin is not necessary, HIT may be largely preventable by using LMWH rather than UFH on an institution-wide scale. At our institution, UFH was largely substituted by the LMWH for thromboprophylaxis gradually throughout 2014 with the implemen
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18

Sachais, Bruce S., Ann H. Rux, Jillian L. Hinds, and John J. Rux. "Rational Design and Characterization of Platelet Factor 4 Antagonists." Blood 116, no. 21 (November 19, 2010): 724. http://dx.doi.org/10.1182/blood.v116.21.724.724.

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Abstract (sommario):
Abstract Abstract 724 Background: Heparin-induced thrombocytopenia and thrombosis (HITT) is a serious complication of heparin therapy, placing patients at increased risk for thrombosis and death. The chemokine platelet factor 4 (PF4) is central to the pathogenesis of HITT. Tetrameric PF4 forms ultralarge complexes (ULC) with heparin, which, when bound to specific antibodies, lead to thrombocytopenia and thrombosis. We have previously shown that the tetrameric form of PF4 is required for ULC formation. Since a large number of hospitalized patients are exposed to heparin, HITT is a major iatroge
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19

Segna, E., A. R. Bolzoni, C. Baserga, and A. Baj. "Free flap loss caused by heparin-induced thrombocytopenia and thrombosis (HITT): a case report and literature review." Acta Otorhinolaryngologica Italica 36, no. 6 (December 2016): 527–33. http://dx.doi.org/10.14639/0392-100x-1188.

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La trombocitopenia eparino-indotta con trombosi rappresenta una complicanza che può portare a esiti drammatici nella chirurgia ricostruttiva microvascolare, tanto più che il suo riconoscimento non è sempre semplice. In ogni caso di trombosi microvascolare, in corso di terapia eparinica, il sospetto di HITT deve subito insorgere, così da poter intercettare e trattare la catena di eventi che porterebbe alla necrosi del lembo ricostruttivo. Presentiamo un caso che dimostra quanto possa essere difficile la diagnosi di HITT, così come appare negli altri reports reperibili in letteratura internazion
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20

Piovella, Franco, Marisa Barone, Chiara Beltrametti, Chiara Piovella, Andrea M. D’Armini, Federico Capra Marzani, Vittorio Arici, et al. "Efficacy of Fondaparinux in the Treatment of Heparin-Induced Thrombocytopenia with Venous Thromboembolism: Reduction of Thromboembolic Burden, Normalization of Platelet Count and Disappearance of Anti-Platelet Factor 4/Heparin Antibodies." Blood 108, no. 11 (November 16, 2006): 575. http://dx.doi.org/10.1182/blood.v108.11.575.575.

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Abstract (sommario):
Abstract Heparin-induced thrombocytopenia (HIT) and heparin-induced thrombocytopenia with thrombosis (HITT) may develop during anticoagulant treatment in patients submitted to various regimens of unfractionated or low-molecular weight heparins. Several molecules have been studied as alternative anticoagulants in patients with HIT or HITT, including danaparoid, argatroban, lepirudin. Lepirudin requires dosage adjustments in patients with renal insufficiency and has potential for antibody formation. Argatroban requires dosage adjustments in patients with hepatic insufficiency. Argatroban increas
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21

Vucelic, Dragica, Nebojsa Antonijevic, Danijel Galun, Predrag Bulajic, Dragan Basaric, and Miroslav Milicevic. "Heparin-induced thrombocytopenia with iliacofemoropopliteal thrombosis in a patient operated for colorectal carcinoma liver metastases." Acta chirurgica Iugoslavica 58, no. 4 (2011): 93–96. http://dx.doi.org/10.2298/aci1104093v.

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Abstract (sommario):
We report a case of heparin-induced thrombocytopenia thrombosis (HITT) syndrome in a patient prophylactically treated with low molecular weight heparin. A 66-year-old men underwent radiofrequency-assisted partial liver resection for colorectal carcinoma liver metastases a year-and-ahalf after he had been operated for rectal cancer. In the postoperative period, patient was prophilactically treated with reviparin sodium. On the 8th postoperative day, the platelet count decreased by more than 50% without clinical signs of thrombosis. HITT syndrome was suspected on the 19th postoperative day, afte
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22

Pravinkumar, E., and N. R. Webster. "HIT/HITT and alternative anticoagulation: current concepts." British Journal of Anaesthesia 90, no. 5 (May 2003): 676–85. http://dx.doi.org/10.1093/bja/aeg063.

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23

Knight, David J. W., D. Selwyn, and K. Girling. "HIT/HITT and alternative anticoagulation: current concepts." British Journal of Anaesthesia 91, no. 3 (September 2003): 445–46. http://dx.doi.org/10.1093/bja/aeg606.

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24

Saad, R. A. "HIT/HITT and alternative anticoagulation: current concepts." British Journal of Anaesthesia 91, no. 4 (October 2003): 606–7. http://dx.doi.org/10.1093/bja/aeg619.

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25

Sabnani, Indu, Marc Cohen, David A. Baran, Patricia Tsang, Luis H. Arroyo, Margarita Camacho, and Mark J. Zucker. "The Role of Serial Testing for Heparin/PF4 Antibodies in Evolving HITT: A Unique Phenomenon of Delayed Seroconversion with Thrombosis." Blood 110, no. 11 (November 16, 2007): 1309. http://dx.doi.org/10.1182/blood.v110.11.1309.1309.

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Abstract (sommario):
Abstract In the appropriate clinical setting, a negative ELISA test for Hep/PF4 antibodies (Ab) has traditionally been considered sufficient to rule out HIT/HITT. We describe a series of 20 cases of HIT/HITT in which the ELISA at the onset of thrombocytopenia was negative despite high (6/8) “4T’s” scores (Thrombocytopenia, Timing of thrombocytopenia in relation to heparin exposure, Thrombosis, and no oTher explanation of thrombocytopenia) but showed strong seroconversion subsequently. This suggests that the negative predictive value of the ELISA test may be too low to definitively exclude HITT
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26

Piovella, Chiara, John Fanikos, Matthew Labreche, Jay Lin, Samuel Z. Goldhaber, and Steven Baroletti. "Heparin-induced thrombocytopenia (HIT): Clinical and economic outcomes." Thrombosis and Haemostasis 100, no. 12 (2008): 1130–35. http://dx.doi.org/10.1160/th08-05-0312.

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Abstract (sommario):
SummaryHeparin-induced thrombocytopenia (HIT) is an immune-mediated adverse drug reaction that occurs following exposure to unfractionated heparin (UFH) or low-molecular-weight heparin (LMWH).HIT with thrombosis (HITT) can cause devastating venous thromboembolism or arterial clots, prolonged hospitalization, and increased costs. To explore the economic and clinical implications of HIT and HITT, we initiated a single-center patient registry. In this report, we describe patient characteristics, comorbidities, management strategies, clinical outcomes, and costs. We enrolled 349 hospitalized patie
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27

Kellermanns, Franz W., and Tim Barnett. "Commentary: What Were They Thinking? The Role of Family Firm Mental Models on Threat Recognition." Entrepreneurship Theory and Practice 32, no. 6 (November 2008): 999–1006. http://dx.doi.org/10.1111/j.1540-6520.2008.00268.x.

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In this commentary, we extend Sirmon, Arregle, Hitt, and Webb's work in this issue by introducing shared mental models as antecedents of threat of imitability recognition and as moderators of the relationship between threat recognition and strategic action. Specifically, while Sirmon, Arregle, Hitt, and Webb focus on responses to threat recognition, we develop propositions on how shared mental models related to business issues affect the threat recognition process and on how shared mental models related to family issues influence strategic responses to recognized threats. Implications and area
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28

Suh, Jang-Soo, Richard H. Aster, and Gian P. Visentin. "Antibodies From Patients With Heparin-Induced Thrombocytopenia/Thrombosis Recognize Different Epitopes on Heparin: Platelet Factor 4." Blood 91, no. 3 (February 1, 1998): 916–22. http://dx.doi.org/10.1182/blood.v91.3.916.

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Abstract (sommario):
Abstract Antibodies associated with heparin-induced thrombocytopenia/thrombosis (HITT) are now thought to be specific for complexes formed between heparin and platelet factor 4 (PF4), a basic protein found normally in platelet alpha granules. How these antibodies cause thrombocytopenia and, in some patients, thrombosis, is not fully understood, in part because purified antibodies that could be labeled and used as probes to characterize target epitopes have not been available. We developed a novel method for antibody purification involving binding to and elution from PF4 complexed to heparin im
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29

Suh, Jang-Soo, Richard H. Aster, and Gian P. Visentin. "Antibodies From Patients With Heparin-Induced Thrombocytopenia/Thrombosis Recognize Different Epitopes on Heparin: Platelet Factor 4." Blood 91, no. 3 (February 1, 1998): 916–22. http://dx.doi.org/10.1182/blood.v91.3.916.916_916_922.

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Abstract (sommario):
Antibodies associated with heparin-induced thrombocytopenia/thrombosis (HITT) are now thought to be specific for complexes formed between heparin and platelet factor 4 (PF4), a basic protein found normally in platelet alpha granules. How these antibodies cause thrombocytopenia and, in some patients, thrombosis, is not fully understood, in part because purified antibodies that could be labeled and used as probes to characterize target epitopes have not been available. We developed a novel method for antibody purification involving binding to and elution from PF4 complexed to heparin immobilized
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30

Henry Edmunds, L. "Hit, hitt, and desulfatohirudin: Look before you leap." Journal of Thoracic and Cardiovascular Surgery 110, no. 1 (July 1995): 1–3. http://dx.doi.org/10.1016/s0022-5223(05)80002-9.

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31

Demenge, Jonathan, and Bibek Shrestha. "High Impact Tourism Training (HITT) Programme in Nepal." Journal of International and Comparative Education 7, no. 2 (2018): 97–109. http://dx.doi.org/10.14425/jice.2018.7.2.97.

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32

Elliott, Michelle A., Sandra Schulte, Elizabeth A. Plumhoff, Rajiv K. Pruthi, William L. Nichols, Mark R. Litzow, Dennis A. Gastineau, and John A. Heit. "Frequency of Heparin-Induced Thrombocytopenia and Heparin-Dependent IgG Antibodies in Hematopoietic Stem Cell Transplant Recipients." Blood 108, no. 11 (November 16, 2006): 4059. http://dx.doi.org/10.1182/blood.v108.11.4059.4059.

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Abstract (sommario):
Abstract Background Heparin-induced thrombocytopenia (HIT) is an immune-mediated drug reaction in which heparin-dependant IgG antibodies (HIT-IgG) to heparin-platelet factor 4 complexes form with the potential for platelet activation with resultant thrombocytopenia (HIT) and thrombosis (HITT). The frequency of HIT-IgG formation and clinically apparent HIT or HITT varies considerably, according to the patient population (surgical versus medical) and type of heparin (unfractionated [UFH] vs low molecular weight [LMWH]). One large prospective study showed a frequency of HIT-IgG formation of 7.5%
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33

Rauova, Lubica, Mortimer Poncz, Steven E. McKenzie, Michael P. Reilly, Gowthami Arepally, John W. Weisel, Chandrasekaran Nagaswami, Douglas B. Cines, and Bruce S. Sachais. "Ultralarge complexes of PF4 and heparin are central to the pathogenesis of heparin-induced thrombocytopenia." Blood 105, no. 1 (January 1, 2005): 131–38. http://dx.doi.org/10.1182/blood-2004-04-1544.

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Abstract (sommario):
Abstract Heparin-induced thrombocytopenia and thrombosis (HITT) is a severe complication of heparin therapy caused by antibodies to complexes between unfractionated heparin (UFH) and platelet factor 4 (PF4) that form over a narrow molar range of reactants and initiate antibody-induced platelet activation. We observed that UFH and tetrameric PF4 formed ultralarge (> 670 kDa) complexes (ULCs) only over a narrow molar range with an optimal ratio of PF4 to heparin of approximately 1:1. These ULCs were stable and visible by electron microscopy, but they could be dissociated into smaller comp
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34

Ketelhut, Sascha, Eva Kircher, Sebastian R. Ketelhut, Emanuel Wehlan, and Kerstin Ketelhut. "Effectiveness of Multi-activity, High-intensity Interval Training in School-aged Children." International Journal of Sports Medicine 41, no. 04 (January 14, 2020): 227–32. http://dx.doi.org/10.1055/a-1068-9331.

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Abstract (sommario):
AbstractThe present study aimed to evaluate the effectiveness of a school-based multi-activity HIIT on aerobic fitness (AF) and hemodynamic parameters in children. 46 students were randomized into an intervention group (INT) (N=22) and a control group (CON) (N=24). Throughout a 3-month intervention period, both INT and CON participated in the regular physical education classes (PE) twice a week. Only INT received an instructed HIIT during the first 20 min of the PE. In addition to an AF-test, peripheral (pBP) and central (cBP) blood pressure, augmentation pressure (AP), and aortic pulse wave v
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35

Gould, Nadia N., Christine L. Haley, Lu Song, Mervyn A. Sahud, and Jeffrey Dlott. "In Vitro Characteristics of Fondaparinux On HIT/HITT Samples." Blood 114, no. 22 (November 20, 2009): 1319. http://dx.doi.org/10.1182/blood.v114.22.1319.1319.

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Abstract Abstract 1319 Poster Board I-335 Heparin-induced thrombocytopenia (HIT) and HIT-associated thrombosis (HITT) are secondary to immune-mediated IgG antibodies that develop against the molecular complex of platelet factor 4 (PF4)–heparin, occurring with exposure to unfractionated heparin (UFH) and low molecular weight heparins (LMWH). Fondaparinux, a synthetic pentasaccharide that binds antithrombin and selectively inhibits Factor Xa, is indicated for prophylaxis and treatment of thrombosis. Fondaparinux has occasionally been reported to induce production of PF4–heparin antibodies; wheth
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Kong, Jian Pei, Linda Jok, Azlee Bin Ayub, and Rawa Ak Bau. "Worksite weight management program." Nutrition & Food Science 47, no. 4 (July 10, 2017): 490–510. http://dx.doi.org/10.1108/nfs-08-2016-0132.

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Purpose This study aims to pilot test a new multi-component worksite intervention for weight loss in a primary healthcare setting. Design/methodology/approach This randomized trial involved 88 participants (43, 45; intervention, control group). The intervention group enrolled in a 12-week lifestyle program that involved modification of dietary intake by community Registered Dietitian (RDs) and increasing high-intensity interval training (HITT) with motivational interviewing (MI) to support changes. The control group received traditional counselling and weekly aerobic exercise from Medical Offi
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Höfler, Stefan. "Dark Matter." Indo-European Linguistics 3, no. 1 (2015): 24–41. http://dx.doi.org/10.1163/22125892-00301004.

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The assumption of a root *√k̑u̯el ‘dark, black’ offers new possible etymologies for Arm. šaɫax, Gk. πηλός, Toch. B kwele, Hitt. kuu̯aliu-, Gk. κύλα, Lat. culex, and Lat. color, whose derivational background will be dealt with in the course of this paper.
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Smoot, Betsy Rohaly. "An Accidental Cryptologist: The Brief Career of Genevieve Young Hitt." Cryptologia 35, no. 2 (March 31, 2011): 164–75. http://dx.doi.org/10.1080/01611194.2011.558982.

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PATEL, CHIRAG, SALIM SURANI, CHINTHAKA BULATHSINGHALA, BRYAN ANDERSON, AFTAB MAHMOOD, DEANNA YAMAMURA, AHMED HASSAN, and PALLA DE SILVA. "A CASE OF SPONTANEOUS HEPARIN-INDUCED THROMBOCYTOPENIA AND THROMBOSIS (HITT)." Chest 154, no. 4 (October 2018): 308A—309A. http://dx.doi.org/10.1016/j.chest.2018.08.283.

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Souza, Rafael Luiz Mesquita, Felipe José Aidar, Sílvia Schütz, Jymmys Lopes Dos Santos, Nara Michelle Moura Soares, Silvan Silva De Araújo, and Anderson Carlos Marçal. "Effects of high-intensity interval training on health-related physical fitness in children and adolescents: a systematic review." Revista Brasileira de Fisiologia do Exercício 19, no. 6 (December 24, 2020): 519. http://dx.doi.org/10.33233/rbfex.v19i6.4376.

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Introduction: High-intensity interval training (HIIT) can promote improvement in health and physical conditioning and contribute to the increase in cardiorespiratory capacity (VO2max), local muscular endurance, and improved body composition. However, its effects on health-related physical fitness in children and adolescents are still unclear. Objective: The objective of the study was to analyze the effectiveness of HIIT in the health-related physical fitness of children and adolescents. Methods: The present study analyzed articles from databases (SPORTDiscus, Web of Science, Medline via Pubmed
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Souza, Rafael Luiz Mesquita, Felipe José Aidar, Sílvia Schütz, Jymmys Lopes Dos Santos, Nara Michelle Moura Soares, Silvan Silva De Araújo, and Anderson Carlos Marçal. "Effects of high-intensity interval training on health-related physical fitness in children and adolescents: a systematic review." Revista Brasileira de Fisiologia do Exercício 19, no. 6 (December 24, 2020): 519. http://dx.doi.org/10.33233/rbfex.v19i6.4376.

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Abstract (sommario):
Introduction: High-intensity interval training (HIIT) can promote improvement in health and physical conditioning and contribute to the increase in cardiorespiratory capacity (VO2max), local muscular endurance, and improved body composition. However, its effects on health-related physical fitness in children and adolescents are still unclear. Objective: The objective of the study was to analyze the effectiveness of HIIT in the health-related physical fitness of children and adolescents. Methods: The present study analyzed articles from databases (SPORTDiscus, Web of Science, Medline via Pubmed
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Kawakami, K., B. K. Shafer, D. J. Garfinkel, J. N. Strathern, and Y. Nakamura. "Ty element-induced temperature-sensitive mutations of Saccharomyces cerevisiae." Genetics 131, no. 4 (August 1, 1992): 821–32. http://dx.doi.org/10.1093/genetics/131.4.821.

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Abstract Temperature-sensitive mutants of Saccharomyces cerevisiae were isolated by insertional mutagenesis using the HIS3 marked retrotransposon TyH3HIS3. In such mutants, the TyHIS3 insertions are expected to identify loci which encode genes essential for cell growth at high temperatures but dispensable at low temperatures. Five mutations were isolated and named hit for high temperature growth. The hit1-1 mutation was located on chromosome X and conferred the pet phenotype. Two hit2 mutations, hit2-1 and hit2-2, were located on chromosome III and caused the deletion of the PET18 locus which
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Adika, Rohfik, and Subandrio Subandrio. "The Effect Of Electronic Commerce And Brand Awareness On Purchasing Decisions At Shopee Online Shopping." BIMA Journal (Business, Management, & Accounting Journal) 2, no. 1 (May 31, 2021): 53–66. http://dx.doi.org/10.37638/bima.2.1.53-66.

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The impact of the dynamics of business competition in the development of the world of information technology is getting more advanced and rapid from time to time so that purchases via virtual are increasing. The purposee off thiss study was to know the effect off electronic commercee and brand awareness on purchasing decision non Online shopee shopping (Case study on online shopping consumers of muhammadiyah university of bengkulu students).This research uses survey research method with quantitative data analysis. The population in this study were students of the University of Muhammadiyah Ben
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Hussain, Farrukh, Roger Philipp, and Shelley Zieroth. "HITT and stent thrombosis: A “CLINICAL” diagnosis not to be missed." International Journal of Cardiology 133, no. 1 (March 2009): e11-e13. http://dx.doi.org/10.1016/j.ijcard.2007.08.078.

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Kalyan Kunchapudi, S., Greeshma Issac, Saravana Pandian, and Indira Jayakumar. "A case of HITT in a child on extracorporeal Membrane Oxygenation." Egyptian Journal of Critical Care Medicine 6, no. 3 (December 2018): 111–12. http://dx.doi.org/10.1016/j.ejccm.2018.12.009.

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Pathak, Ranjan, Smith Giri, Madan Raj Aryal, Paras Karmacharya, Anthony Donato, and Vijaya R. Bhatt. "Medical and Economic Burden of Heparin-Induced Thrombocytopenia: Data from the National Inpatient Sample 2009-2011." Blood 126, no. 23 (December 3, 2015): 3272. http://dx.doi.org/10.1182/blood.v126.23.3272.3272.

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Abstract Background Although generally safe, heparin use can trigger an immune response in which platelet factor 4-heparin complexes set off an antibody-mediated cascade that can result in heparin-induced thrombocytopenia (HIT). Although older studies report incidences as high as 5% in high-risk subgroups of surgical patients, recent studies report a much lower incidence (0.02% of hospital admissions and <0.1-0.4% among patients exposed to heparin). As hospitals transition to the less immunogenic low molecular weight heparins, reassessment of the overall national burden of HIT would help in
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Nanwa, Natasha, Nicole Mittmann, Sandra Knowles, Rita Selby, Neil Shear, Scott Walker, and William Geerts. "The Direct Medical Costs Associated with Suspected and Confirmed Cases of Heparin-Induced Thrombocytopenia." Blood 110, no. 11 (November 16, 2007): 972. http://dx.doi.org/10.1182/blood.v110.11.972.972.

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Abstract Introduction: Heparin-induced thrombocytopenia (HIT) is a serious adverse reaction associated with heparin exposure. Few studies have assessed the economic burden associated with HIT and none have assessed the costs of suspected HIT. The objective of our study was to identify and quantify the direct medical costs associated with suspected and confirmed cases of HIT from the Canadian hospital perspective. Methods: Over the last 7 years SHSC, a tertiary care adult academic hospital, experienced on average 125 cases of suspected HIT per year. A retrospective burden of illness analysis wa
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Stirrup, Tracey, Diane Williams, Samantha Sharman, Joan Williamson, and Mark Aldersley. "Micro-elimination in prisons: a high-intensity test and treat (HITT) programme." Journal of Hepatology 73 (August 2020): S329. http://dx.doi.org/10.1016/s0168-8278(20)31160-0.

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49

Goti, F., D. Gianom, W. Korte, D. Sege, and M. Decurtins. "Postoperative Heparin- induzierte Thrombopenie mit Thromboembolie (HITT) - Eine seltene Komplikation der Heparintherapie?" Swiss Surgery 7, no. 2 (2001): 0051–56. http://dx.doi.org/10.1024/1023-9332.7.2.51.

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Stannard, Alicja, Jonathan Hudak, Kayla Kowalczyk, Bianca DeLucia, Victoria Lynch, and Jessica Wade. "Validity Of Activity Monitors For Energy Expenditure During Walking, Running, And Hitt." Medicine & Science in Sports & Exercise 53, no. 8S (August 2021): 272. http://dx.doi.org/10.1249/01.mss.0000762252.46129.02.

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