Bibliografie tematiche / HIV - related lung disease

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Letteratura scientifica selezionata sul tema "HIV - related lung disease"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 2 febbraio 2022

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Articoli di riviste sul tema "HIV - related lung disease"

1

Cribbs, Sushma K., Kristina Crothers e Alison Morris. "Pathogenesis of HIV-Related Lung Disease: Immunity, Infection, and Inflammation". Physiological Reviews 100, n. 2 (1 aprile 2020): 603–32. http://dx.doi.org/10.1152/physrev.00039.2018.

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Despite anti-retroviral therapy (ART), human immunodeficiency virus-1 (HIV)-related pulmonary disease continues to be a major cause of morbidity and mortality for people living with HIV (PLWH). The spectrum of lung diseases has changed from acute opportunistic infections resulting in death to chronic lung diseases for those with access to ART. Chronic immune activation and suppression can result in impairment of innate immunity and progressive loss of T cell and B cell functionality with aberrant cytokine and chemokine responses systemically as well as in the lung. HIV can be detected in the lungs of PLWH and has profound effects on cellular immune functions. In addition, HIV-related lung injury and disease can occur secondary to a number of mechanisms including altered pulmonary and systemic inflammatory pathways, viral persistence in the lung, oxidative stress with additive effects of smoke exposure, microbial translocation, and alterations in the lung and gut microbiome. Although ART has had profound effects on systemic viral suppression in HIV, the impact of ART on lung immunology still needs to be fully elucidated. Understanding of the mechanisms by which HIV-related lung diseases continue to occur is critical to the development of new preventive and therapeutic strategies to improve lung health in PLWH.
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2

Ron, Raquel, Ignacio Ruz-Caracuel, Eugenia García e María Luisa Montes-Ramírez. "IgG4-related disease in a patient with HIV infection". BMJ Case Reports 12, n. 4 (aprile 2019): e226809. http://dx.doi.org/10.1136/bcr-2018-226809.

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A 47-year-old HIV-positive man with good immune and virological status presented with chronic multiple enlarged lymph nodes, lung disease and eosinophilia. Radiologic tests showed enlarged cervical, thoracic and axillary lymph nodes, with interstitial lung damage. After several non-specific histologic studies, an elevated serum IgG4 level led us to request immunohistochemistry of a lymph node sample. The test confirmed the diagnosis of IgG4-related disease.
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3

Nguyen, Elizabeth V., Sina A. Gharib, Kristina Crothers, Yu-Hua Chow, David R. Park, David R. Goodlett e Lynn M. Schnapp. "Proteomic landscape of bronchoalveolar lavage fluid in human immunodeficiency virus infection". American Journal of Physiology-Lung Cellular and Molecular Physiology 306, n. 1 (1 gennaio 2014): L35—L42. http://dx.doi.org/10.1152/ajplung.00140.2013.

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The lung is an important reservoir of human immunodeficiency virus (HIV). Individuals infected with HIV are more prone to pulmonary infections and chronic lung disorders. We hypothesized that comprehensively profiling the proteomic landscape of bronchoalveolar lavage fluid (BALF) in patients with HIV would provide insights into how this virus alters the lung milieu and contributes to pathogenesis of HIV-related lung diseases. BALF was obtained from five HIV-negative (HIV−) and six asymptomatic HIV-positive (HIV+) subjects not on antiretroviral therapy. Each sample underwent shotgun proteomic analysis based on HPLC-tandem mass spectrometry. Differentially expressed proteins between the groups were identified using statistical methods based on spectral counting. Mechanisms of disease were explored using functional annotation to identify overlapping and distinct pathways enriched between the BALF proteome of HIV+ and HIV− subjects. We identified a total of 318 unique proteins in BALF of HIV− and HIV+ subjects. Of these, 87 were differentially up- or downregulated between the two groups. Many of these differentially expressed proteins are known to interact with key HIV proteins. Functional analysis of differentially regulated proteins implicated downregulation of immune responses in lungs of HIV+ patients. Combining shotgun proteomic analysis with computational methods demonstrated that the BALF proteome is significantly altered during HIV infection. We found that immunity-related pathways are underrepresented in HIV+ patients. These findings implicate mechanisms whereby HIV invokes local immunosuppression in the lung and increases the susceptibility of HIV+ patients to develop a wide range of infectious and noninfectious pulmonary diseases.
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4

Cowan, N. C., e J. Moxham. "Upper zone cystic lung disease in HIV related Pneumocystis carinii pneumonia". Thorax 48, n. 8 (1 agosto 1993): 869–70. http://dx.doi.org/10.1136/thx.48.8.869.

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5

Powles, T., M. Nelson e M. Bower. "HIV-related lung cancer - a growing concern?" International Journal of STD & AIDS 14, n. 10 (ottobre 2003): 647–51. http://dx.doi.org/10.1258/095646203322387875.

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6

Otani, Kyoko, Dai Inoue, Tomoo Itoh e Yoh Zen. "Transbronchial lung biopsy for the diagnosis of IgG4-related lung disease". Histopathology 73, n. 1 (2 maggio 2018): 49–58. http://dx.doi.org/10.1111/his.13513.

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7

Lipman, Marc, e James Brown. "HIV-related Chronic Obstructive Pulmonary Disease. Are Lung CD4 T Cells Bothered?" American Journal of Respiratory and Critical Care Medicine 190, n. 7 (ottobre 2014): 718–20. http://dx.doi.org/10.1164/rccm.201408-1531ed.

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8

Geraghty, Patrick, Eran Hadas, Boe-Hyun Kim, Abdoulaye J. Dabo, David J. Volsky e Robert Foronjy. "HIV infection model of chronic obstructive pulmonary disease in mice". American Journal of Physiology-Lung Cellular and Molecular Physiology 312, n. 4 (1 aprile 2017): L500—L509. http://dx.doi.org/10.1152/ajplung.00431.2016.

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Cigarette smoke usage is prevalent in human immunodeficiency virus (HIV)-positive patients, and, despite highly active antiretroviral therapy, these individuals develop an accelerated form of chronic obstructive pulmonary disease (COPD). Studies investigating the mechanisms of COPD development in HIV have been limited by the lack of suitable mouse models. Here we describe a model of HIV-induced COPD in wild-type mice using EcoHIV, a chimeric HIV capable of establishing chronic infection in immunocompetent mice. A/J mice were infected with EcoHIV and subjected to whole body cigarette smoke exposure. EcoHIV was detected in alveolar macrophages of mice. Compared with uninfected mice, concomitant EcoHIV infection significantly reduced forced expiratory flow 50%/forced vital capacity and enhanced distal airspace enlargement following cigarette smoke exposure. Lung IL-6, granulocyte-macrophage colony-stimulating factor, neutrophil elastase, cathepsin G, and matrix metalloproteinase-9 expression was significantly enhanced in smoke-exposed EcoHIV-infected mice. These changes coincided with enhanced IκBα, ERK1/2, p38, and STAT3 phosphorylation and lung cell apoptosis. Thus, the EcoHIV smoke exposure mouse model reproduces several of the pathophysiological features of HIV-related COPD in humans, indicating that this murine model can be used to determine key parameters of HIV-related COPD and to test future therapies for this disorder.
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9

Huang, Yvonne J., e Leopoldo N. Segal. "Looking Higher: Is It Prime Time for the Oral–Lung Axis in HIV-related Lung Disease?" American Journal of Respiratory and Critical Care Medicine 201, n. 4 (15 febbraio 2020): 402–3. http://dx.doi.org/10.1164/rccm.201911-2170ed.

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10

Gingo, Matthew R. "The changing landscape of HIV-related lung disease: Non-AIDS lung malignancy as a player in the field". Respirology 19, n. 3 (12 marzo 2014): 300–302. http://dx.doi.org/10.1111/resp.12249.

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Più fonti

Tesi sul tema "HIV - related lung disease"

1

Lemos, Renata Muller Banzato Pinto de. "Proteína C reativa (PCR) em crianças com infecção pelo HIV na ausência de quadro infeccioso concomitante e na vigência de pneumonia aguda". Universidade de São Paulo, 2003. http://www.teses.usp.br/teses/disponiveis/5/5141/tde-23092014-115736/.

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Por serem as manifestações pulmonares de etiologia infecciosas muito freqüentes e potencialmente graves nas crianças com aids, o diagnóstico deve ser precoce para uma rápida e efetiva intervenção terapêutica. A proteína C reativa (PCR), um dos marcadores das provas de fase aguda, tem sido usada na prática clínica como um recurso diagnóstico na diferenciação entre patologias sistêmicas de etiologia viral e bacteriana, bem como na monitorização da eficácia da terapêutica antimicrobiana frente a uma infecção. Apesar da proteína C reativa ser um exame inespecífico e poder estar aumentada em diferentes situações clínicas (infecções sistêmicas, doenças inflamatórias e neoplásicas, isquemias, queimaduras), a infecção bacteriana é a causa mais freqüente para o seu aumento. A PCR eleva-se rapidamente após a injúria tecidual, atingindo valores 10 a 1000 vezes superiores a seu nível basal: em virtude de sua curta meia vida, retorna em pouco tempo aos valores prévios após o fim da agressão. Com o objetivo de encontrar um método laboratorial auxiliar para as infecções pulmonares nas crianças com infecção pelo HIV foi estudada a proteína C reativa, pela técnica de nefelometria, em dois momentos distintos: na ausência de quadro infeccioso concomitante (grupo 1) e na vigência de pneumonia aguda (grupo 2). O grupo 1 envolveu o estudo de 66 crianças com infecção pelo HIV, resultando em 84 amostras de PCR coletadas na ausência de quadro infeccioso concomitante.No grupo 2 foram analisadas 6 crianças com infecção pelo HIV com 9 episódios de pneumonia aguda. As crianças com infecção pelo HIV foram classificadas de acordo com as categorias clínicas e imunológicas da classificação do CDC para infecção pelo HIV em crianças. Dentre as 66 crianças incluídas no grupo 1, 6 pertenciam à categoria N, 11 à categoria A, 27 à categoria B e 22 à categoria C. Das 84 amostras de PCR coletadas no grupo 1, 76 (90,48%) encontravam-se abaixo de 5 mg/l, 7 amostras entre 5 a 20 mg /l e, apenas 1 amostra entre 20 a 40 mg/l (1,15%). No grupo 2, todas as crianças eram pertencentes às categorias B3 (1/6) ou C3 (5/6), refletindo um estágio mais avançado da doença. Das 9 amostras de PCR, 6 apresentavam valores maiores que 40 mg/l, 1 entre 20 e 40 mg/l e as 2 amostras restantes, entre 5 e 20 mg/l. Os dados sugerem portanto que a infecção pelo HIV por si só não é acompanhada de aumento da PCR, bem como não existe relação com a classificação imunológica em que o paciente se encontre. Pacientes com infecção pelo HIV na vigência de pneumonia aguda apresentam níveis aumentados de PCR. Neste estudo, o ponto de corte que diferenciou os grupos 1 e 2 foi PCR = 28,9 mg/l com sensibilidade de 77,8% e especificidade de 100% (IC 95%)
As pulmonary infection is a common and potentially serious condition in HIV-infected children, effectiveness of treatment of this kind of affection depends to a large extent on the promptness of accurate diagnosis. The C-Reactive Protein (CRP), a reasonably well-established acute phase marker, has long been used to differentiate bacterial from viral infections. Despite its lack of specifity, that is, the fact that other conditions like inflammatory diseases, neoplasms, ischemia and burns may also increase CRP levels, Bacterial infections are the most frequent cause of increased CRP found in daily clinical practice. Shortly after any tissular injury, CRP increases considerably, reaching up to 10-1000 times its previous levels. Due to its short half-life, its decrease after the end of the affection is quick as well. This study aimed at evaluating the CRP (assessed by nephelometry) as an auxiliary tool to diagnose pulmonary infection in HIV-infected children. Two groups of patients were considered in this study: group 1 was constituted by 66 HIV-infected children with no clinical signs of concomitant infection (amounting to 84 CRP samples) and group 2 was constituted by 6 HIV-infected children with pneumonia (amounting to 9 CRP samples). All the subjects were assigned to categories according to the pediatric HIV classification system (CDC, 1994). Among the 66 children from group 1, 6 were assigned to categories N, 11 to A, 27 to B and 22 to C. Regarding the levels of CRP in group 1 it was found: 76 samples (90.48%) < 5 mg/l, 7 (8.33%) in the range between 5 and 20 mg/l and 1 sample between 20 and 40 mg/l. In the group 2, all the children were assigned either to category B3 (1/6) or C3 (5/6) and the CRP level distribution was the following: 6 (6/9) > 40 mg/l, 1 (1/6) between 20 and 40 mg/l and 2 (2/6) between 5 and 20 mg/l. These results suggest that 1.HIV infection by itself does not increase the levels of CRP, regardless the immunologic classification of the patient; 2.HIV-infected children with pneumonia present increased levels of CRP 3.In this study, the cut-off point to differentiate groups 1 and 2 was 28,9 mg/l, with sensitivity of 77,8% and specificity of 100% (p < 0.05)
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2

Birch, Jodie. "Telomere dysfunction and senescence in the ageing lung and age-related lung disease". Thesis, University of Newcastle upon Tyne, 2015. http://hdl.handle.net/10443/3927.

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Cellular senescence, the irreversible loss of replicati ve capacity of somatic cells, bas been associated w ith diseases of accelerated lung ageing, including Chroni c Obstructive Pulmonaty Disease (COPD). However, the mechanisms und erl ying senescence of a irway epithelial cell s, parti cula rl y the role of telomere dysfunction in this process, are poorly understood . The aim of this work was to investigate senescence and telomere dysfunction in airway epithelial cells from pati ents with COPD and bronchiectasis, in the ageing murine lung and in the context of cigarette smoke exposure. DNA dam age foci (yH2A.X) and foci associated with telomeres (telomere-associated foci (TA F)), a long with other senescence-associated m arkers, were increased in small airway epith elial cells from patients with COPD, wi thout significant telomere shottening. With age, TAF increased in large and sm all aitway epithelial cells of the murine lung and predicted age-dependent lung emphysema, independen tly oftelomere length . M oreover, fomth generation telomerase-null mi ce showed early-onset emphysema. Exposure to cigarette smoke was found to increase TAF in large and small ai1way epithelial cells of the murine lung and in epith elial cells and fibrobla sts in vitro. Cigarette smoke m ay accelerate telomere dysfunction via reactive oxygen species (ROS) and contribute to Ataxia telangiectasia mutated (ATM)-dependent secretion of pro-inflammatory cytokines interleukin (lL)-6 and IL-8. Inhibition of mechan istic target of rapa myc in complex I (mTORC I ) by rapa mycin alleviated age-assoc iated increases i n TAF in vivo and supressed cigarette smoke-i nduced increases in TAF and in flammatory cytokine release in vitro. Cigarette smoke increases mitochondrial-derived ROS, which is supressed by culturing cell s at low oxygen pressure and by treating cell s w ith rapam ycin. These results suggest that activation of a DN A damage response at telomeres may be induced by oxidative stress from altered mTOR signalling ancl/or dysfu nctional mitochondri a. Telomere dys function could conttibut e to inflamm atory processes and the functional decline that occurs in the ageing lung and in the context of cigarette smoke­ induced accelerated lung ageing.
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3

Beetge, Lijahne. "Themes related to children living with HIV/AIDS". Thesis, Stellenbosch : Stellenbosch University, 2007. http://hdl.handle.net/10019.1/21549.

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Assignment (MA)--University of Stellenbosch, 2007.
ENGLISH ABSTRACT: This review examines themes related to children, especially children under the age of fifteen, living with HIV/Aids. For review purposes themes include defining children living with HIV/Aids as a vulnerable population, the psychological impact of HIV/Aids on children, children’s coping with and perceptions of HIV/Aids. The global HIV/Aids pandemic has caused major worldwide social change. The impact, especially the psychological impact of HIV/Aids on children appears to be vast and at times disastrous in nature. HIV/Aids is estimated to affect the lives of several generations of children and the impact of the pandemic will therefore characterize their communities for decades to come as the numbers of affected and infected children are on the increase. It has been noted by research done that children living with HIV/Aids become impoverished, their insecurities worsen and their psychosocial and psychological problems increase. In addition their educational and work opportunities decline, their nurturing and support systems disintegrate, and child mortality rates rise. The necessity of psychological support for children affected and infected by HIV/Aids is directly linked to the rights of children listed in the United Nations Convention on the Rights of the Child and the South African Constitution. Research reviewed points out that a child’s physical and psychological health is intertwined and therefore a child cannot be physically healthy without also being psychologically secure. Research reviewed also points out that it is of the utmost importance that the voices of children who live with HIV/Aids are heard, their involvement in decision making and planning related to HIV/Aids is allowed and that their needs are met, especially their psychological needs in relation to the pandemic. Overall the literature reviewed in the past four years (2003-2006) highlights the significantly under-investigated, under-researched themes such as psychological aspects related to children living with HIV/Aids, both in South Africa and internationally. These aspects surrounding HIV/Aids, as previously mentioned, also seem to be the least tangible and the most difficult for adults to address or comprehend fully and yet they impinge on all aspects of developing children - hence the identified need in this area for adequate research and psychological support, such as program implementation.
AFRIKAANSE OPSOMMING: Hierdie literatuuroorsig bied ’n ondersoek na die wêreldwye sosiale verandering en uitwerking wat die globale HIV/vigs-pandemie tot gevolg het. Daar word veral gelet op temas wat verwantskap hou met kinders, veral kinders vyftien jaar oud en jonger wat leef met HIV/vigs. Temas sluit in die definiëring van kinders as weerloos, kinders se hantering van HIV/vigs, hulle persepsies van HIV/vigs en hulle sielkundige behoeftes en ander aspekte met betrekking tot HIV/vigs. Die trefkrag van HIV/vigs blyk omvattend en by tye rampspoedig te wees. Na beraming beïnvloed HIV/vigs verskeie geslagte van kinders en die uitwerking van die pandemie sal dus dekades lank nog deel vorm van hulle gemeenskappe soos die aantal kinders wat geïnfekteer en geaffekteer is, toeneem. Navorsing toon dat kinders wat met HIV/vigs leef, verarm. Verder vererger hulle onsekerhede en hulle psigososiale en sielkundige probleme neem toe. Ook neem die kinders se opvoedkundige en werkgeleenthede af, hulle versorgings- en ondersteuningsnetwerke disintegreer en sterftesyfers neem toe. Die noodsaaklikheid van sielkundige ondersteuning vir kinders wat deur HIV/vigs geïnfekteer en geaffekteer is, word direk in verband gebring met die regte van kinders soos gelys in die Verenigde Nasies se Konvensie oor die Regte van die Kind en die Suid-Afrikaanse Grondwet. Die literatuuroorsig toon dat ‘n kind se fisieke en sielkundige gesondheid ten nouste met mekaar verband hou en daarom kan ’n kind nie fisiek gesond wees sonder om ook sielkundig geborge te wees nie. Verder dui navorsing ook op die kardinale belang daarvan dat kinders wat leef met HIV/vigs se stemme gehoor word, hulle betrokkenheid by besluitneming en beplanning verseker word en al hulle behoeftes bevredig word, veral hulle sielkundige behoeftes wat met die pandemie verband hou. Oor die algemeen beklemtoon die literatuuroorsig wat oor die afgelope vier jaar strek (2003-2006) dat daar nie voldoende navorsing gedoen is met betrekking tot temas wat verwantskap hou met kinders en HIV/vigs nie, nie alleen in Suid-Afrika nie, maar ook internasionaal. Hierdie bevindings is kommerwekkend veral omdat Suid-Afrika ‘n land is met onrusbarende groot getalle kinders wat deur HIV/vigs geïnfekteer is. Dit wil ook voorkom of hierdie aspekte wat te make het met kinders en HIV/vigs nie baie duidelik is vir volwassenes nie en ook die moeilikste is om te hanteer of ten volle te verstaan, hoewel dit alle aspekte van die kind se ontwikkeling raak. Vandaar die behoefte op hierdie gebied aan voldoende navorsing en sielkundige ondersteuning, soos program implementering.
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Pitcher, Richard D. "Radiological progression of lung disease in Human Immunodeficiency Virus (HIV)-infected children". Doctoral thesis, University of Cape Town, 2016. http://hdl.handle.net/11427/20351.

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Introduction: There are limited data on the chest X-ray (CXR) abnormalities in human immunodeficiency virus (HIV)-infected children in low- and middle-income countries (LMIC's). Aim: To investigate the evolution of CXR abnormalities in HIV-infected children in LMIC's, to correlate this with the severity of HIV-disease, and to assess the impact of anti-retroviral therapy (ART). Method: A prospective longitudinal study evaluating clinical, immunological and radiographic parameters at regular intervals over a minimum of 24 months. CXR abnormalities were stratified by severity and deemed persistent if present for 6 consecutive months or longer. Univariate and multiple logistic regression analyses assessed associations between radiological and clinical/immunological parameters at enrolment. An ordinal multiple logistic regression model assessed the association of enrolment and time-dependent variables with CXR findings over time.
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Liu, Chi-hang. "HIV/Aids-related stigma and discrimination : the case of Hong Kong /". Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B22330926.

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Rodriguez, Ihsan. "Well-being and Inflammation in Interstitial Lung Disease". The Ohio State University, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1619031719578262.

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Campbell, Joshua David. "Genome-wide characterization of microRNA and gene expression patterns in smoking-related lung disease". Thesis, Boston University, 2012. https://hdl.handle.net/2144/12307.

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Thesis (Ph.D.)--Boston University PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.
Smoking-related lung diseases such as chronic obstructive pulmonary disease (COPD) and lung cancer are significant public health concerns world-wide. High throughput genomic technologies have opened up a new realm of understanding into the complexities of human disease by providing a means by which we can gain considerable amounts of information about a sample. In my research, I examine genome-wide gene expression via microarrays and microRNA expression via small RNA-sequencing (small RNA-Seq) to gain insights into lung disease pathogenesis, assess novel strategies for identifying therapeutics, and develop biomarkers for earlier diagnosis of disease. First, I revealed mechanisms of emphysema progression within individuals by leveraging a unique dataset that contains multiple lung-tissue samples per patient collected from regions with different levels of emphysematous destruction. Pathways involved in immune response and tissue remodeling were enriched among gene expression profiles associated with increasing regional emphysema severity. Using the Connectivity Map, a compound was discovered capable of reversing the gene-expression signature of increasing emphysema severity which can serve as a lead in therapeutic development for COPD. [TRUNCATED]
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Mason, Nicola Anne. "The role of nitric oxide and related mediators in tissue injury and remodelling in pulmonary disease". Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312124.

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Nemuramba, Rathani. "HIV related risk behaviours in South African rural community". Thesis, University of Limpopo (Turfloop Campus), 2010. http://hdl.handle.net/10386/663.

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Thesis (M.A. (Research Psychology)) --University of Limpopo, 2010
This study measures the relationship between the AIDS risk reduction model (ARRM) variables associated with HIV related risk behaviours on learners from a South African rural community. A cross-sectional study was conducted using 308 learners in a Limpopo rural high school to identify HIV risk behaviours. Data were analyzed using binary logistic regression to test the usefulness of ARRM variables in predicting sexual risk. Sexual risk was measured as; (a) vaginal sex without a condom, (b) anal sex without a condom (c) number of sexual partners in the last twelve months and (d) time taken before having sex with a new partner. Two of the ARRM variables, that is perceived susceptibility and sexual response efficacy, were found to be the most important predictors of HIV related risk behaviours. There is an argent need for effective preventive activities in rural areas, especially through school-based interventions. Key words: ARRM HIV HIV prevention Risk behaviors
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Chaves, Alysia Anne. "Mechanisms of AIDS and cocaine related cardiovascular disease". Columbus, Ohio Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1056031201.

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Thesis (Ph.D.)--Ohio State University, 2003.
Title from first page of PDF file. Document formatted into pages; contains xx, 401 p.; also includes graphics (some col.). Includes abstract and vita. Advisor: John A. Bauer, Dept.of Pharmacy. Includes bibliographical references (p. 387-391).
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Più fonti

Libri sul tema "HIV - related lung disease"

1

Studies, National Institute for Occupational Safety and Health Division of Respiratory Disease. Work-related lung disease surveillance report, 1994. Cincinnati, Ohio: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1994.

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2

Studies, National Institute for Occupational Safety and Health Division of Respiratory Disease. Work-related lung disease surveillance report, 1996. Cincinnati, Ohio: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1996.

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3

Studies, National Institute for Occupational Safety and Health Division of Respiratory Disease. Work-related lung disease surveillance report, 1994: World report. [Morgantown, WV]: Division of Respiratory Disease Studies, National Institute for Occupational Safety and Health, U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, 1994.

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4

Studies, National Institute for Occupational Safety and Health Division of Respiratory Disease. Work-related lung disease surveillance report, 1994: World report. [Morgantown, WV]: Division of Respiratory Disease Studies, National Institute for Occupational Safety and Health, U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, 1994.

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5

Work-related lung disease surveillance report, 1994: World report. [Morgantown, WV]: Division of Respiratory Disease Studies, National Institute for Occupational Safety and Health, U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control, 1994.

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6

Commission, Ontario Human Rights. Policy statement on HIV/AIDS-related discrimination. [Toronto]: Ontario, Human Rights Commission, 1990.

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7

Huber, Jeffrey T. HIV/AIDS and HIV/AIDS-related terminology: A means of organizing the body of knowledge. New York: Haworth Press, 1996.

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8

Huber, Jeffrey T. HIV/AIDS and HIV/AIDS-related terminology: A means of organizing the body of knowledge. New York: Harrington Park Press, 1996.

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Huber, Jeffrey T. HIV/AIDS and HIV/AIDS-related terminology: A means of organizing the body of knowledge. New York: Haworth Press, 1996.

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L, Hall Thomas, e Palo Alto Medical Foundation for Health Care, Research, and Education., a cura di. Protocol 6: HIV-related training needs, programs, and costs. Rockville, MD: U.S. Dept. of Health and Human Services, Public Health Service, National Center for Health Services Research and Health Care Technology Assessment, 1989.

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Capitoli di libri sul tema "HIV - related lung disease"

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Inoue, Dai, Yoh Zen, Shoko Matsui, Yuko Waseda, Osamu Matsui e Toshifumi Gabata. "Lung Lesions". In IgG4-Related Disease, 93–98. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54228-5_14.

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Matsubara, Osamu, e Eugene J. Mark. "HHV-8-Related Lung Neoplastic and Nonneoplastic Diseases". In Viruses and the Lung, 177–90. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-40605-8_20.

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Ryu, Jay H., Hiroshi Sekiguchi e Eunhee S. Yi. "IgG4-Related Lung Disease". In Autoimmune (IgG4-related) Pancreatitis and Cholangitis, 199–208. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-6430-4_19.

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Calabrese, Fiorella. "Lung Transplantation-Related Pathology". In Pathology of Lung Disease, 335–52. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-50491-8_16.

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Popper, Helmut. "Smoking-Related Lung Diseases". In Pathology of Lung Disease, 103–19. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-50491-8_7.

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Akgün, Kathleen M., e Kristina Crothers. "Lung Disease in Older Patients with HIV". In Aging and Lung Disease, 227–52. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-727-3_12.

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Matsui, Shoko, Kenji Notohara e Yuko Waseda. "Pathological Findings of IgG4-Related Lung Disease". In IgG4-Related Disease, 163–67. Tokyo: Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54228-5_24.

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Li, Hongjun. "HIV/AIDS Related Urogenital Disease". In Radiology of HIV/AIDS, 689–707. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7823-8_22.

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Sainburg, Robert L., Andrew L. Clark, George E. Billman, Zachary J. Schlader, Toby Mündel, Kevin Milne, Earl G. Noble et al. "HIV-Related Cardiometabolic Complications". In Encyclopedia of Exercise Medicine in Health and Disease, 414. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4276.

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Raj, Satish R., S. R. Wayne Chen, Robert S. Sheldon, Arti N. Shah, Bharat K. Kantharia, Ulrich Salzer, Bodo Grimbacher et al. "Transfusion Related Acute Lung Injury". In Encyclopedia of Molecular Mechanisms of Disease, 2098. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7039.

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Atti di convegni sul tema "HIV - related lung disease"

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Shakhgildyan, V. I., M. S. Yadrikhinskaya, А. А. Orlovsky, О. Y. Shipulina, E. A. Domonova e Е. В. Yarovaya. "CYTOMEGALOVIRUS DNA CONCENTRATION IN BIOLOGICAL SAMPLES AS A KEY TO THE DIAGNOSIS OF CMV PNEUMONIA IN HIV-INFECTED PATIENTS". In Молекулярная диагностика и биобезопасность – 2020. ФБУН Центральный НИИ эпидемиологии Роспотребнадзора, 2020. http://dx.doi.org/10.36233/978-5-9900432-9-9-86.

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Abstract (sommario):
According to examination and follow-up results of 5485 HIV-positive hospitalized patients (3333 of which were diagnosed with AIDS) we have identified the frequency of clinically evident CMV-infection as well as the frequency and character of CMV related lung disease. Statistically significant correlation between viral load, degree of immunosuppression, CMV replication rate and CMV pneumonia development risk has been determined. Qualitative PCR assay for CMV DNA in plasma and respiratory samples was found to have high sensitivity and low specificity for diagnosing CMV-pneumonia. We identified quantitative PCR CMV DNA values in blood cells, plasma, bronchoalveolar lavage, bronchi samples and sputum that confirm the diagnosis of CMV pneumonia with 95% and 99% probability, and exclude CMV related lung damage in HIV patients with 90% and 99% probability.
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Binder, A., A. Dixit, A. H. Sardi, V. Green e C. Swenson. "Salon Related Lung Disease". In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7491.

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Alvarenga Santos, Maria, Susana Clemente, Margarida Felizardo e Sofia Furtado. "Connective tissue disease-related interstitial lung disease and lung cancer". In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2986.

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Riley, L., K. Fredenburg, M. Brantly e A. Ataya. "Smoking-Related Diffuse Cystic Lung Disease". In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6341.

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Patel, H., S. Ie e E. R. Rubio. "Cystic Lung Disease Related to Bevacizumab". In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5773.

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Miller, Elizabeth, e Hyun J. Kim. "IGG4-Related Sclerosing Disease: A Rare Interstitial Lung Disease". In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6454.

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Barczi, E., E. Palmer, A. Kolonics-Farkas, K. Vincze, N. Eszes, A. Bohacs e V. Müller. "Lung Function Decline in Scleroderma-Related Interstitial Lung Disease". In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6830.

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Mikhail, M., A. Avula, N. Narula, S. Acharya, D. Sharma e M. N. Chalhoub. "IgG4-Related Disease Masquerading as Lung Cancer". In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5742.

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Surana, A., e R. Bansal. "IgG4 Related Lung Disease: The Great Masquerader". In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6645.

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Chandra, D., A. Gupta, M. Fitzpatrick, S. Haberlen, M. Neupane, J. K. Leader, L. Kingsley et al. "The Association Between Lung and Coronary Artery Disease in HIV". In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5947.

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Rapporti di organizzazioni sul tema "HIV - related lung disease"

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Li, Zifeng, Xiaolan Fu, Long Yin, Xiaoqiang Hou e Caiyun Chang. Clinical effect of Tripterygium Glycosides in the treatment of connective tissue disease-related interstitial lung disease：Meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, agosto 2021. http://dx.doi.org/10.37766/inplasy2021.8.0028.

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Work-related lung disease surveillance report. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control, National Institute for Occupational Safety and Health, ottobre 1991. http://dx.doi.org/10.26616/nioshpub91113.

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Work-related lung disease surveillance report 1994. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, agosto 1994. http://dx.doi.org/10.26616/nioshpub94120.

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Work-related lung disease surveillance report, 1996. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, gennaio 1996. http://dx.doi.org/10.26616/nioshpub96134.

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Work-related lung disease surveillance report 1999. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, dicembre 1999. http://dx.doi.org/10.26616/nioshpub2000105.

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Work-related lung disease surveillance report, 2002. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, maggio 2003. http://dx.doi.org/10.26616/nioshpub2003111.

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Flavoring-related lung disease: information for healthcare providers. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, maggio 2012. http://dx.doi.org/10.26616/nioshpub2012148.

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Work-related lung disease surveillance report 2007. Volume 1. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, settembre 2008. http://dx.doi.org/10.26616/nioshpub2008143a.

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Flavoring-related lung disease: information for healthcare providers (superseded by 2012-148). U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, novembre 2011. http://dx.doi.org/10.26616/nioshpub2012107.

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Study of the prevalence of chronic, non-specific lung disease and related health problems in the grain handling industry. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control, National Institute for Occupational Safety and Health, ottobre 1986. http://dx.doi.org/10.26616/nioshpub86117.

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