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Articoli di riviste sul tema "Host-virus relationships"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 29 luglio 2024

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1

Lal, Sunil K. "Influenza A Virus: Host–Virus Relationships." Viruses 12, no. 8 (August 9, 2020): 870. http://dx.doi.org/10.3390/v12080870.

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We are in the midst of a pandemic where the infective agent has been identified, but how it causes mild disease in some and fatally severe disease in other infected individuals remains a mystery [...]

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2

Mansour, A., and A. Al-Musa. "Cucumber Vein Yellowing Virus; Host Range and Virus Vector Relationships." Journal of Phytopathology 137, no. 1 (January 1993): 73–78. http://dx.doi.org/10.1111/j.1439-0434.1993.tb01327.x.

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3

López‐Lastra, Marcelo. "Host–virus relationships: a sum of many battles." FEBS Open Bio 12, no. 6 (June 2022): 1094–95. http://dx.doi.org/10.1002/2211-5463.13420.

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4

Dusi, A. N., and D. Peters. "Beet mosaic virus: its Vector and Host Relationships." Journal of Phytopathology 147, no. 5 (May 1999): 293–98. http://dx.doi.org/10.1111/j.1439-0434.1999.tb03833.x.

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5

Xu, Feng, Chen Zhao, Yuhua Li, Jiang Li, Youping Deng, and Tieliu Shi. "Exploring virus relationships based on virus-host protein-protein interaction network." BMC Systems Biology 5, Suppl 3 (2011): S11. http://dx.doi.org/10.1186/1752-0509-5-s3-s11.

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6

MANSOUR, A., and A. AL-MUSA. "Tomato yellow leaf curl virus: host range and virus-vector relationships." Plant Pathology 41, no. 2 (April 1992): 122–25. http://dx.doi.org/10.1111/j.1365-3059.1992.tb02328.x.

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7

Scharninghausen, Jerrold J., Michael Faulde, and Semra Cavaljuga. "Hantavirus host/virus interactions within Southeast Europe." Bosnian Journal of Basic Medical Sciences 4, no. 4 (November 20, 2004): 13–18. http://dx.doi.org/10.17305/bjbms.2004.3353.

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Abstract (sommario):

Viral studies have historically approached their phylogenetic analysis without consideration of the impact of the role the host plays in evolution. Our study examines host/viral interactions through analysis of the phylogenetic relationship between hantavirus genetic sequences and host cytochrome B sequences. Phylogenetic analysis of known Hantavirus genetic sequences were performed using PAUP 3.1.1 (vers. 4.0.0d64). Only sequences available through GENBANK were analyzed. Phylogenetic analysis of hantavirus sequences revealed distinct patterns based upon geographic area. These patterns coincid

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8

Hunziker, Lukas, Mike Recher, Adrian Ciurea, Marianne M. A. Martinic, Bernhard Odermatt, Hans Hengartner, and Rolf M. Zinkernagel. "Antagonistic Variant Virus Prevents Wild-type Virus-induced Lethal Immunopathology." Journal of Experimental Medicine 196, no. 8 (October 21, 2002): 1039–46. http://dx.doi.org/10.1084/jem.20012045.

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Altered peptide ligands (APLs) and their antagonistic or partial agonistic character–influencing T cell activation have mainly been studied in vitro Some studies have shown APLs as a viral escape mechanism from cytotoxic CD8+ T cell responses in vivo. However, whether infection or superinfection with a virus displaying an antagonistic T cell epitope can alter virus–host relationships via inhibiting T cell–mediated immunopathology is unclear. Here, we evaluated a recently described CD4+ T cell escape lymphocytic choriomeningitis virus (LCMV) variant that in vitro displayed antagonistic characte

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9

Brown, J. K. "Host Range and Vector Relationships of Cotton Leaf Crumple Virus." Plant Disease 71, no. 6 (1987): 522. http://dx.doi.org/10.1094/pd-71-0522.

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10

McLeish, Michael, Soledad Sacristán, Aurora Fraile, and Fernando García-Arenal. "Scale dependencies and generalism in host use shape virus prevalence." Proceedings of the Royal Society B: Biological Sciences 284, no. 1869 (December 20, 2017): 20172066. http://dx.doi.org/10.1098/rspb.2017.2066.

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Processes that generate the distribution of pathogens and their interactions with hosts are not insensitive to changes in spatial scale. Spatial scales and species traits are often selected intentionally, based on practical considerations, ignoring biases that the scale and type of observation may introduce. Specifically, these biases might change the interpretation of disease–diversity relationships that are reported as either ‘dilution’ or ‘amplification’ effects. Here, we combine field data of a host–pathogen community with empirical models to test the effects that (i) spatial scale and (ii

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11

Tomaru, Yuji, and Kei Kimura. "Novel Protocol for Estimating Viruses Specifically Infecting the Marine Planktonic Diatoms." Diversity 12, no. 6 (June 4, 2020): 225. http://dx.doi.org/10.3390/d12060225.

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Since their discovery, at least 15 diatom viruses have been isolated and characterised using a culture method with two cycles of extinction dilution. However, the method is time consuming and laborious, and it isolates only the most dominant virus in a water sample. Recent studies have suggested inter-species host specificity of diatom viruses. Here, we describe a new protocol to estimate previously unrecognised host-virus relationships. Host cell cultures after inoculation of natural sediment pore water samples were obtained before complete lysis. The proliferated viral genomes in the host ce

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12

Li, Wanlin, and Nadia Tahiri. "Host–Virus Cophylogenetic Trajectories: Investigating Molecular Relationships between Coronaviruses and Bat Hosts." Viruses 16, no. 7 (July 15, 2024): 1133. http://dx.doi.org/10.3390/v16071133.

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Bats, with their virus tolerance, social behaviors, and mobility, are reservoirs for emerging viruses, including coronaviruses (CoVs) known for genetic flexibility. Studying the cophylogenetic link between bats and CoVs provides vital insights into transmission dynamics and host adaptation. Prior research has yielded valuable insights into phenomena such as host switching, cospeciation, and other dynamics concerning the interaction between CoVs and bats. Nonetheless, a distinct gap exists in the current literature concerning a comparative cophylogenetic analysis focused on elucidating the cont

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13

Masson, Patrick, Chantal Hulo, Edouard de Castro, Rebecca Foulger, Sylvain Poux, Alan Bridge, Jane Lomax, Lydie Bougueleret, Ioannis Xenarios, and Philippe Le Mercier. "An Integrated Ontology Resource to Explore and Study Host-Virus Relationships." PLoS ONE 9, no. 9 (September 18, 2014): e108075. http://dx.doi.org/10.1371/journal.pone.0108075.

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14

Happonen, Lotta Johanna, Peter Redder, Xu Peng, Laila Johanne Reigstad, David Prangishvili, and Sarah Jane Butcher. "Familial Relationships in Hyperthermo- and Acidophilic Archaeal Viruses." Journal of Virology 84, no. 9 (February 17, 2010): 4747–54. http://dx.doi.org/10.1128/jvi.02156-09.

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ABSTRACT Archaea often live in extreme, harsh environments such as acidic hot springs and hypersaline waters. To date, only two icosahedrally symmetric, membrane-containing archaeal viruses, SH1 and Sulfolobus turreted icosahedral virus (STIV), have been described in detail. We report the sequence and three-dimensional structure of a third such virus isolated from a hyperthermoacidophilic crenarchaeon, Sulfolobus strain G4ST-2. Characterization of this new isolate revealed it to be similar to STIV on the levels of genome and structural organization. The genome organization indicates that these

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15

Nagasaki, K., Y. Tomaru, Y. Shirai, Y. Takao, and H. Mizumoto. "Dinoflagellate-infecting viruses." Journal of the Marine Biological Association of the United Kingdom 86, no. 3 (April 10, 2006): 469–74. http://dx.doi.org/10.1017/s0025315406013361.

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Dinoflagellates (Dinophyceae) are considered to be one of the most abundant and diverse groups of phytoplankton; however, the viral impact on dinoflagellates was not studied until recently. This review shows the present information concerning the viruses infecting dinoflagellates and the ecology relationships between the host and the virus. So far, two viruses have been isolated and characterized: a large DNA virus (HcV: Heterocapsa circularisquama virus) and a small RNA virus (HcRNAV: H. circularisquama RNA virus); both of which are infectious to the harmful bloom-forming dinoflagellate H. ci

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16

Acosta-Leal, R., S. Duffy, Z. Xiong, R. W. Hammond, and S. F. Elena. "Advances in Plant Virus Evolution: Translating Evolutionary Insights into Better Disease Management." Phytopathology® 101, no. 10 (October 2011): 1136–48. http://dx.doi.org/10.1094/phyto-01-11-0017.

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Recent studies in plant virus evolution are revealing that genetic structure and behavior of virus and viroid populations can explain important pathogenic properties of these agents, such as host resistance breakdown, disease severity, and host shifting, among others. Genetic variation is essential for the survival of organisms. The exploration of how these subcellular parasites generate and maintain a certain frequency of mutations at the intra- and inter-host levels is revealing novel molecular virus–plant interactions. They emphasize the role of host environment in the dynamic genetic compo

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17

Francis, Magen, Morgan King, and Alyson Kelvin. "Back to the Future for Influenza Preimmunity—Looking Back at Influenza Virus History to Infer the Outcome of Future Infections." Viruses 11, no. 2 (January 30, 2019): 122. http://dx.doi.org/10.3390/v11020122.

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The influenza virus-host interaction is a classic arms race. The recurrent and evolving nature of the influenza virus family allows a single host to be infected several times. Locked in co-evolution, recurrent influenza virus infection elicits continual refinement of the host immune system. Here we give historical context of circulating influenza viruses to understand how the individual immune history is mirrored by the history of influenza virus circulation. Original Antigenic Sin was first proposed as the negative influence of the host’s first influenza virus infection on the next and Imprin

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18

Zheng, Lu-Lu, Chunyan Li, Jie Ping, Yanhong Zhou, Yixue Li, and Pei Hao. "The Domain Landscape of Virus-Host Interactomes." BioMed Research International 2014 (2014): 1–13. http://dx.doi.org/10.1155/2014/867235.

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Viral infections result in millions of deaths in the world today. A thorough analysis of virus-host interactomes may reveal insights into viral infection and pathogenic strategies. In this study, we presented a landscape of virus-host interactomes based on protein domain interaction. Compared to the analysis at protein level, this domain-domain interactome provided a unique abstraction of protein-protein interactome. Through comparisons among DNA, RNA, and retrotranscribing viruses, we identified a core of human domains, that viruses used to hijack the cellular machinery and evade the immune s

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19

da Silva Schreiber, Maicon, and Juliana Fachinetto. "PHYLOGENETIC RELATIONSHIP BETWEEN RABIES VIRUS (RABIES LYSSAVIRUS) VARIANTS FROM TWO DIFFERENT HOST SPECIES." Veterinária e Zootecnia 31 (January 13, 2024): 1–7. http://dx.doi.org/10.35172/rvz.2024.v31.1537.

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Rabies is a fatal zoonotic disease that affects several mammals. Bats are recognized hosts of the rabies virus, and their main food source is the blood of other mammals, particularly cattle. During feeding, bats transmit the virus to cattle, which are victims of the disease, contributing to economic losses and increasing the risk of infection for humans. Based on this affinity in the rabies cycle between bats and cattle, the objective of this study was to analyze the phylogenetic relationships of rabies virus samples in cattle and bats. The G gene of the rabies virus was chosen for this study

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20

Rossi, Chiara, Nicola Zadra, Cristina Fevola, Frauke Ecke, Birger Hörnfeldt, René Kallies, Maria Kazimirova, et al. "Evolutionary Relationships of Ljungan Virus Variants Circulating in Multi-Host Systems across Europe." Viruses 13, no. 7 (July 7, 2021): 1317. http://dx.doi.org/10.3390/v13071317.

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The picornavirus named ‘Ljungan virus’ (LV, species Parechovirus B) has been detected in a dozen small mammal species from across Europe, but detailed information on its genetic diversity and host specificity is lacking. Here, we analyze the evolutionary relationships of LV variants circulating in free-living mammal populations by comparing the phylogenetics of the VP1 region (encoding the capsid protein and associated with LV serotype) and the 3Dpol region (encoding the RNA polymerase) from 24 LV RNA-positive animals and a fragment of the 5′ untranslated region (UTR) sequence (used for defini

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21

Tangherlini, Michael, Cinzia Corinaldesi, Francesca Ape, Silvestro Greco, Teresa Romeo, Franco Andaloro, and Roberto Danovaro. "Ocean Acidification Induces Changes in Virus–Host Relationships in Mediterranean Benthic Ecosystems." Microorganisms 9, no. 4 (April 6, 2021): 769. http://dx.doi.org/10.3390/microorganisms9040769.

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Acidified marine systems represent “natural laboratories”, which provide opportunities to investigate the impacts of ocean acidification on different living components, including microbes. Here, we compared the benthic microbial response in four naturally acidified sites within the Southern Tyrrhenian Sea characterized by different acidification sources (i.e., CO2 emissions at Ischia, mixed gases at Panarea and Basiluzzo and acidified freshwater from karst rocks at Presidiana) and pH values. We investigated prokaryotic abundance, activity and biodiversity, viral abundance and prokaryotic infec

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22

Palmeira, Leonor, Simon Penel, Vincent Lotteau, Chantal Rabourdin-Combe, and Christian Gautier. "PhEVER: a database for the global exploration of virus–host evolutionary relationships." Nucleic Acids Research 39, suppl_1 (November 16, 2010): D569—D575. http://dx.doi.org/10.1093/nar/gkq1013.

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23

Torres, Andrea N., Candace K. Mathiason, and Edward A. Hoover. "Re-examination of feline leukemia virus: host relationships using real-time PCR." Virology 332, no. 1 (February 2005): 272–83. http://dx.doi.org/10.1016/j.virol.2004.10.050.

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24

Alkan, Cigdem, Gautier Brésard, Lise Frézal, Aurélien Richaud, Albane Ruaud, Gaotian Zhang, and Marie-Anne Félix. "Natural variation in infection specificity of Caenorhabditis briggsae isolates by two RNA viruses." PLOS Pathogens 20, no. 6 (June 11, 2024): e1012259. http://dx.doi.org/10.1371/journal.ppat.1012259.

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Antagonistic relationships such as host-virus interactions potentially lead to rapid evolution and specificity in interactions. The Orsay virus is so far the only horizontal virus naturally infecting the nematode C. elegans. In contrast, several related RNA viruses infect its congener C. briggsae, including Santeuil (SANTV) and Le Blanc (LEBV) viruses. Here we focus on the host’s intraspecific variation in sensitivity to these two intestinal viruses. Many temperate-origin C. briggsae strains, including JU1264 and JU1498, are sensitive to both, while many tropical strains, such as AF16, are res

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25

Boromisa, Robert D., and Paul R. Grimstad. "Virus-Vector-Host Relationships of Aedes stimulans and Jamestown Canyon Virus in a Northern Indiana Enzootic Focus." American Journal of Tropical Medicine and Hygiene 35, no. 6 (November 1, 1986): 1285–95. http://dx.doi.org/10.4269/ajtmh.1986.35.1285.

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26

Parikka, Kaarle J., Marc Le Romancer, Nina Wauters, and Stéphan Jacquet. "Deciphering the virus-to-prokaryote ratio (VPR): insights into virus-host relationships in a variety of ecosystems." Biological Reviews 92, no. 2 (April 26, 2016): 1081–100. http://dx.doi.org/10.1111/brv.12271.

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27

Gaunt, Michael W., Amadou A. Sall, Xavier de Lamballerie, Andrew K. I. Falconar, Tatyana I. Dzhivanian, and Ernest A. Gould. "Phylogenetic relationships of flaviviruses correlate with their epidemiology, disease association and biogeography." Journal of General Virology 82, no. 8 (August 1, 2001): 1867–76. http://dx.doi.org/10.1099/0022-1317-82-8-1867.

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Phylogenetic analysis of the Flavivirus genus, using either partial sequences of the non-structural 5 gene or the structural envelope gene, revealed an extensive series of clades defined by their epidemiology and disease associations. These phylogenies identified mosquito-borne, tick-borne and no-known-vector (NKV) virus clades, which could be further subdivided into clades defined by their principal vertebrate host. The mosquito-borne flaviviruses revealed two distinct epidemiological groups: (i) the neurotropic viruses, often associated with encephalitic disease in humans or livestock, corre

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28

Fulhorst, Charles F., Remi N. Charrel, Robert D. Bradley, Mary Louise Milazzo, and Darin S. Carroll. "Natural host relationships and genetic diversity of Whitewater Arroyo virus in southern Texas." American Journal of Tropical Medicine and Hygiene 67, no. 1 (July 1, 2002): 114–18. http://dx.doi.org/10.4269/ajtmh.2002.67.114.

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29

Ammar, E. D., S. Elnagar, A. E. Abul-Ata, and G. H. Sewify. "Vector and Host-Plant Relationships of the Leafhopper-Borne Maize Yellow Stripe Virus." Journal of Phytopathology 126, no. 3 (July 1989): 246–52. http://dx.doi.org/10.1111/j.1439-0434.1989.tb01111.x.

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30

Fayer, Ronald, Palmer Orlandi, and Michael L. Perdue. "Virulence factor activity relationships for hepatitis E and Cryptosporidium." Journal of Water and Health 7, S1 (August 1, 2009): S55—S63. http://dx.doi.org/10.2166/wh.2009.044.

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The hepatitis E virus and Cryptosporidium are waterborne pathogens, each consisting of distinct taxa, genotypes and isolates that infect humans, nonhuman animal species or both. Some are associated with disease, others are not. Factors contributing to disease are extremely complicated, possibly involving differences in one or more traits associated with an organism's taxon, genotype or isolate and its infectious dose, and age or condition, as well as the host's physiology and immune status. Potential virulence factors have not yet been identified for HEV. Putative virulence factors for Cryptos

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31

Walsh, Sarah K., Ryan M. Imrie, Marta Matuszewska, Gavin K. Paterson, Lucy A. Weinert, Jarrod D. Hadfield, Angus Buckling, and Ben Longdon. "The host phylogeny determines viral infectivity and replication across Staphylococcus host species." PLOS Pathogens 19, no. 6 (June 8, 2023): e1011433. http://dx.doi.org/10.1371/journal.ppat.1011433.

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Virus host shifts, where a virus transmits to and infects a novel host species, are a major source of emerging infectious disease. Genetic similarity between eukaryotic host species has been shown to be an important determinant of the outcome of virus host shifts, but it is unclear if this is the case for prokaryotes where anti-virus defences can be transmitted by horizontal gene transfer and evolve rapidly. Here, we measure the susceptibility of 64 strains of Staphylococcaceae bacteria (48 strains of Staphylococcus aureus and 16 non-S. aureus species spanning 2 genera) to the bacteriophage IS

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32

Olson, Victoria A., Kevin L. Karem, Scott K. Smith, Christine M. Hughes, and Inger K. Damon. "Smallpox virus plaque phenotypes: genetic, geographical and case fatality relationships." Journal of General Virology 90, no. 4 (April 1, 2009): 792–98. http://dx.doi.org/10.1099/vir.0.008169-0.

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Smallpox (infection with Orthopoxvirus variola) remains a feared illness more than 25 years after its eradication. Historically, case-fatality rates (CFRs) varied between outbreaks (<1 to ∼40 %), the reasons for which are incompletely understood. The extracellular enveloped virus (EEV) form of orthopoxvirus progeny is hypothesized to disseminate infection. Investigations with the closely related Orthopoxvirus vaccinia have associated increased comet formation (EEV production) with increased mouse mortality (pathogenicity). Other vaccinia virus genetic manipulations which affect EEV producti

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33

Truong Nguyen, Phuoc, Santiago Garcia-Vallvé, and Pere Puigbò. "An Unsupervised Algorithm for Host Identification in Flaviviruses." Life 11, no. 5 (May 14, 2021): 442. http://dx.doi.org/10.3390/life11050442.

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Early characterization of emerging viruses is essential to control their spread, such as the Zika Virus outbreak in 2014. Among other non-viral factors, host information is essential for the surveillance and control of virus spread. Flaviviruses (genus Flavivirus), akin to other viruses, are modulated by high mutation rates and selective forces to adapt their codon usage to that of their hosts. However, a major challenge is the identification of potential hosts for novel viruses. Usually, potential hosts of emerging zoonotic viruses are identified after several confirmed cases. This is ineffic

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34

Chatziandreou, N., N. Stock, D. Young, J. Andrejeva, K. Hagmaier, D. J. McGeoch, and R. E. Randall. "Relationships and host range of human, canine, simian and porcine isolates of simian virus 5 (parainfluenza virus 5)." Journal of General Virology 85, no. 10 (October 1, 2004): 3007–16. http://dx.doi.org/10.1099/vir.0.80200-0.

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Sequence comparison of the V/P and F genes of 13 human, canine, porcine and simian isolates of simian virus 5 (SV5) revealed a surprising lack of sequence variation at both the nucleotide and amino acid levels (0–3 %), even though the viruses were isolated over 30 years and originated from countries around the world. Furthermore, there were no clear distinguishing amino acid or nucleotide differences among the isolates that correlated completely with the species from which they were isolated. In addition, there was no evidence that the ability of the viruses to block interferon signalling by t

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35

Rouse, Barry T., and Scott N. Mueller. "Some vexations that challenge viral immunology." F1000Research 5 (May 27, 2016): 1015. http://dx.doi.org/10.12688/f1000research.8391.1.

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The field of viral immunology seeks to understand mechanisms of virus-host interaction with a view of applying this knowledge to the design of effective vaccines and immunomodulators that control viral infections. This brief review discusses several areas of the field that hold substantial promise for translation, but where further work is critically required to find solutions. We emphasize that our fundamental understanding of virus-host relationships is moving in leaps and bounds, but we lag behind in applying this knowledge to the successful control of many viral infections.

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Smith, Daniel F. Q., and Arturo Casadevall. "On the relationship between Pathogenic Potential and Infective Inoculum." PLOS Pathogens 18, no. 6 (June 13, 2022): e1010484. http://dx.doi.org/10.1371/journal.ppat.1010484.

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Pathogenic Potential (PP) is a mathematical description of an individual microbe, virus, or parasite’s ability to cause disease in a host, given the variables of inoculum, signs of disease, mortality, and in some instances, median survival time of the host. We investigated the relationship between pathogenic potential (PP) and infective inoculum (I) using two pathogenic fungi in the wax moth Galleria mellonella with mortality as the relevant outcome. Our analysis for C. neoformans infection revealed negative exponential relationship between PP and I. Plotting the log(I) versus the Fraction of

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Tscharke, David C., and Geoffrey L. Smith. "A model for vaccinia virus pathogenesis and immunity based on intradermal injection of mouse ear pinnae." Journal of General Virology 80, no. 10 (October 1, 1999): 2751–55. http://dx.doi.org/10.1099/0022-1317-80-10-2751.

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Vaccinia virus (VV) proteins that interfere with the host response to infection are of interest because they provide insight into virus–host relationships and may affect the safety and immunogenicity of recombinant VV (rVV) vaccines. Such vaccines need assessment in animal models and with this aim a model of VV infection based on intradermal injection of BALB/c ear pinnae was developed and characterized. In this model, the outcome of infection is affected by the dose of virus inoculated but virus spread is minimal and the mice suffer no signs of systemic illness. Cellular and humoral immune re

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Kelling, Clayton L., Jane E. Kennedy, Lisa C. Stine, Kristine K. Rump, Prem S. Paul, and James E. Partridge. "Genetic comparison of ovine and bovine pestiviruses." American Journal of Veterinary Research 51, no. 12 (December 1, 1990): 2019–24. http://dx.doi.org/10.2460/ajvr.1990.51.12.2019.

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SUMMARY Viral rna oligonucleotide fingerprinting was used to compare genetic relationships among pestiviruses originating from ovine or bovine host species. Ovine pestiviruses, including reference border disease virus and 2 border disease isolates originating from natural pestivirus infections of sheep, appeared to have a more distant genetic relationship among themselves than with certain bovine pestiviruses. A closer genetic relatedness was evident between border disease virus and 3 noncytopathic bovine pestiviruses, including Draper bovine viral diarrhea virus (bvdv), a bvdv isolate that or

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Davis, Robert F. "Cucumber Mosaic Virus Isolates Seedborne inPhaseolus vulgaris: Serology, Host-Pathogen Relationships, and Seed Transmission." Phytopathology 76, no. 10 (1986): 999. http://dx.doi.org/10.1094/phyto-76-999.

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Nolt, B. L. "Indian Peanut Clump Virus Isolates: Host Range, Symptomatology, Serological Relationships, and Some Physical Properties." Phytopathology 78, no. 3 (1988): 310. http://dx.doi.org/10.1094/phyto-78-310.

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41

Li, You, Zhi Jiang Zeng, and Zi Long Wang. "Phylogenetic analysis of the honeybee Sacbrood virus." Journal of Apicultural Science 60, no. 1 (June 1, 2016): 31–38. http://dx.doi.org/10.1515/jas-2016-0009.

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Abstract Sacbrood virus (SBV) is one of the most common and harmful viruses to honeybees. It causes failure to pupate and death during larval stage, in adult bees it has an influence on their behavior and even shortens their life-span. In this study, we analyzed the phylogenetic relationships among the SBV isolates from all around the world, with from both Apis cerana and Apis mellifera. Phylogenetic trees were constructed based on three types of nucleotide sequences: complete genome sequence, VP1 gene and SB1-2 fragment of SBV. Moreover, genome recombination analysis was performed to assess t

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Brown, J. K. "Transmission, Host Range, and Virus-Vector Relationships of Chino del Tomate Virus, a Whitefly-Transmitted Geminivirus from Sinaloa, Mexico." Plant Disease 72, no. 10 (1988): 866. http://dx.doi.org/10.1094/pd-72-0866.

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Sidorkiewicz, Malgorzata. "Hepatitis C Virus Uses Host Lipids to Its Own Advantage." Metabolites 11, no. 5 (April 27, 2021): 273. http://dx.doi.org/10.3390/metabo11050273.

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Lipids and lipoproteins constitute indispensable components for living not only for humans. In the case of hepatitis C virus (HCV), the option of using the products of our lipid metabolism is “to be, or not to be”. On the other hand, HCV infection, which is the main cause of chronic hepatitis, cirrhosis and hepatocellular carcinoma, exerts a profound influence on lipid and lipoprotein metabolism of the host. The consequences of this alternation are frequently observed as hypolipidemia and hepatic steatosis in chronic hepatitis C (CHC) patients. The clinical relevance of these changes reflects

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Lukashov, Vladimir V., and Jaap Goudsmit. "Evolutionary relationships among Astroviridae." Journal of General Virology 83, no. 6 (June 1, 2002): 1397–405. http://dx.doi.org/10.1099/0022-1317-83-6-1397.

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To study the evolutionary relationships among astroviruses, all available sequences for members of the family Astroviridae were collected. Phylogenetic analysis distinguished two deep-rooted groups: one comprising mammalian astroviruses, with ovine astrovirus being an outlier, and the other comprising avian astroviruses. All virus species as well as serotypes of human astroviruses represented individual lineages within the tree. All human viruses clustered together and separately from non-human viruses, which argue for their common evolutionary origin and against ongoing animal-to-human transm

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Jancovich, James K., Michel Bremont, Jeffrey W. Touchman, and Bertram L. Jacobs. "Evidence for Multiple Recent Host Species Shifts among the Ranaviruses (Family Iridoviridae)." Journal of Virology 84, no. 6 (December 30, 2009): 2636–47. http://dx.doi.org/10.1128/jvi.01991-09.

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ABSTRACT Members of the genus Ranavirus (family Iridoviridae) have been recognized as major viral pathogens of cold-blooded vertebrates. Ranaviruses have been associated with amphibians, fish, and reptiles. At this time, the relationships between ranavirus species are still unclear. Previous studies suggested that ranaviruses from salamanders are more closely related to ranaviruses from fish than they are to ranaviruses from other amphibians, such as frogs. Therefore, to gain a better understanding of the relationships among ranavirus isolates, the genome of epizootic hematopoietic necrosis vi

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Calegario, Renata Faier, Sávio de Siqueira Ferreira, Eduardo Chumbinho de Andrade, and Francisco Murilo Zerbini. "Characterization of Tomato yellow spot virus, a novel tomato-infecting begomovirus in Brazil." Pesquisa Agropecuária Brasileira 42, no. 9 (September 2007): 1335–43. http://dx.doi.org/10.1590/s0100-204x2007000900016.

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The objective of this work was the biological and molecular characterization of a begomovirus detected in São Joaquim de Bicas, Minas Gerais, Brazil, named TGV-[Bi2], by determining its host range, complete nucleotide sequence and phylogenetic relationships with other begomoviruses. Biological characterization consisted of a host range study using either sap inoculation or particle bombardment as inoculation methods. The yellow spot virus can infect plants in Solanaceae and Amaranthaceae, including economically importat crops as sweet pepper, and weeds as Datura stramonium and Nicotiana silves

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Campagnola, Grace, Seth McDonald, Stéphanie Beaucourt, Marco Vignuzzi, and Olve B. Peersen. "Structure-Function Relationships Underlying the Replication Fidelity of Viral RNA-Dependent RNA Polymerases." Journal of Virology 89, no. 1 (October 15, 2014): 275–86. http://dx.doi.org/10.1128/jvi.01574-14.

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ABSTRACTViral RNA-dependent RNA polymerases are considered to be low-fidelity enzymes, providing high mutation rates that allow for the rapid adaptation of RNA viruses to different host cell environments. Fidelity is tuned to provide the proper balance of virus replication rates, pathogenesis, and tissue tropism needed for virus growth. Using our structures of picornaviral polymerase-RNA elongation complexes, we have previously engineered more than a dozen coxsackievirus B3 polymerase mutations that significantly altered virus replication rates andin vivofidelity and also provided a set of sec

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Liang, Xiao-Zhen, Bernett T. K. Lee, and Sek-Man Wong. "Covariation in the Capsid Protein of Hibiscus Chlorotic Ringspot Virus Induced by Serial Passaging in a Host That Restricts Movement Leads to Avirulence in Its Systemic Host." Journal of Virology 76, no. 23 (December 1, 2002): 12320–24. http://dx.doi.org/10.1128/jvi.76.23.12320-12324.2002.

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ABSTRACT Hibiscus chlorotic ringspot virus (HCRSV) from naturally infected Hibiscus rosa-sinensis L. loses virulence in its experimental systemic host Hibiscus cannabinus L. (kenaf) after serial passages in a local lesion host Chenopodium quinoa. Here we report the genetic changes responsible for the loss of virulence at the molecular level. A remarkable covariation of eight site-specific amino acids was found in the HCRSV capsid protein (CP) after serial passages in C. quinoa: Val49→Ile, Ile95→Val, Lys270→Arg, Gly272→Asp, Tyr274→His, Ala311→Asp, Asp334→Ala, and Ala335→Thr. Covariation of at l

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Camp, Jeremy V., Ulas Bagci, Yong-Kyu Chu, Brendan Squier, Mostafa Fraig, Silvia M. Uriarte, Haixun Guo, Daniel J. Mollura, and Colleen B. Jonsson. "Lower Respiratory Tract Infection of the Ferret by 2009 H1N1 Pandemic Influenza A Virus Triggers Biphasic, Systemic, and Local Recruitment of Neutrophils." Journal of Virology 89, no. 17 (June 10, 2015): 8733–48. http://dx.doi.org/10.1128/jvi.00817-15.

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ABSTRACTInfection of the lower respiratory tract by influenza A viruses results in increases in inflammation and immune cell infiltration in the lung. The dynamic relationships among the lung microenvironments, the lung, and systemic host responses during infection remain poorly understood. Here we used extensive systematic histological analysis coupled with live imaging to gain access to these relationships in ferrets infected with the 2009 H1N1 pandemic influenza A virus (H1N1pdm virus). Neutrophil levels rose in the lungs of H1N1pdm virus-infected ferrets 6 h postinfection and became concen

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Murata, Takayuki. "Human Herpesvirus and the Immune Checkpoint PD-1/PD-L1 Pathway: Disorders and Strategies for Survival." Microorganisms 9, no. 4 (April 8, 2021): 778. http://dx.doi.org/10.3390/microorganisms9040778.

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The immune system has evolved as a complex and efficient means of coping with extrinsic materials, such as pathogens and toxins, as well as intrinsic abnormalities, such as cancers. Although rapid and timely activation of the immune system is obviously important, regulated downregulation of the system is almost as significant as activation to prevent runaway immunity, such as allergies and hypercytokinemia. Therefore, the immune checkpoint programmed cell death 1 (PD-1)/programmed cell death ligand 1 (PD-L1) pathway is beneficial for the host. On the other hand, pathogens have evolved to evade

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