Bibliografie tematiche / Insulin metabolism

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Letteratura scientifica selezionata sul tema "Insulin metabolism"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "Insulin metabolism"

Humaira, Thisa, and Rizka Sofia. "Pasien Diabetes Melitus dengan Penatalaksanaan Gigi Tiruan Penuh (GTP)." GALENICAL : Jurnal Kedokteran dan Kesehatan Mahasiswa Malikussaleh 3, no. 2 (2024): 115. http://dx.doi.org/10.29103/jkkmm.v3i2.17359.

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Diabetes mellitus (DM) didefinisikan sebagai suatu penyakit atau gangguan metabolisme kronis dengan multi etiologi yang ditandai dengan tingginya kadar gula darah disertai dengan gangguan metabolism karbohidrat, lipid dan protein sebagai akibat insufisiensi fungsi insuli. Insufisiensi fungsi insulin dapat di sebabkan oleh gangguan atau defisiensi produksi insulin oleh sel – sel beta Langerhans kelenjar beta pancreas atau disebabkan kurang responsifnya sel – sel tubuh terhadap insulin. Penyakit ini dapat menimbulkan banyak komplikasi di berbagai bagian tubuh, ternasuk rongga mulut. Beberapa man

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Ukkola, O. "Ghrelin and insulin metabolism." European Journal of Clinical Investigation 33, no. 3 (2003): 183–85. http://dx.doi.org/10.1046/j.1365-2362.2003.01112.x.

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Duckworth, William C., Frederick G. Hamel, and Daniel E. Peavy. "Hepatic metabolism of insulin." American Journal of Medicine 85, no. 5 (1988): 71–76. http://dx.doi.org/10.1016/0002-9343(88)90399-3.

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Heesom, K. J., M. Harbeck, C. R. Kahn, and R. M. Denton. "Insulin action on metabolism." Diabetologia 40 (September 19, 1997): S3—S9. http://dx.doi.org/10.1007/s001250051388.

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Beardsall, Kathryn, Barbro M. S. Diderholm, and David B. Dunger. "Insulin and carbohydrate metabolism." Best Practice & Research Clinical Endocrinology & Metabolism 22, no. 1 (2008): 41–55. http://dx.doi.org/10.1016/j.beem.2007.10.001.

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Heesom, K. J., M. Harbeck, C. R. Kahn, and R. M. Denton. "Insulin action on metabolism." Diabetologia 40, S3 (1997): B3—B9. http://dx.doi.org/10.1007/bf03168179.

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Harned, Leighton Kahle, and Edward Chin. "PSUN278 Factitious hypoglycemia, diagnostic delay due to insulin assay failure to detect insulin analogues." Journal of the Endocrine Society 6, Supplement_1 (2022): A403. http://dx.doi.org/10.1210/jendso/bvac150.838.

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Abstract Factitious hypoglycemia may be difficult to diagnose clinically. Hypoglycemia due to insulin self-administration is established by the presence of a low c-peptide and elevated plasma insulin levels. Commercial insulin assays often fail to detect insulin analogs and can create confusion among providers investigating causes of hypoglycemia. A 20 year old female with no significant past medical history presented to an Emergency Room (ER) with hypoglycemia. She was treated with a single dose of octreotide 150 mcg, dexamethasone 10 mg PO and started on a D10W drip at 100ml/hr prior to tran

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Hirsch, Irl B., Rattan Juneja, John M. Beals, Caryl J. Antalis, and Eugene E. Wright. "The Evolution of Insulin and How it Informs Therapy and Treatment Choices." Endocrine Reviews 41, no. 5 (2020): 733–55. http://dx.doi.org/10.1210/endrev/bnaa015.

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Abstract Insulin has been available for the treatment of diabetes for almost a century, and the variety of insulin choices today represents many years of discovery and innovation. Insulin has gone from poorly defined extracts of animal pancreata to pure and precisely controlled formulations that can be prescribed and administered with high accuracy and predictability of action. Modifications of the insulin formulation and of the insulin molecule itself have made it possible to approximate the natural endogenous insulin response. Insulin and insulin formulations had to be designed to produce ei

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Piloquet, H., V. Ferchaud-Roucher, F. Duengler, Y. Zair, P. Maugere, and M. Krempf. "Insulin effects on acetate metabolism." American Journal of Physiology-Endocrinology and Metabolism 285, no. 3 (2003): E561—E565. http://dx.doi.org/10.1152/ajpendo.00042.2003.

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Acetate metabolism was studied in patients with insulin resistance. To evaluate the interaction between glucose and acetate metabolism, we measured acetate and glucose turnover with a hyperinsulinemic euglycemic clamp (hot clamp) in obese and diabetic patients with insulin resistance ( n = 8) and in a control group with normal insulin sensitivity ( n = 6). At baseline, acetate turnover and plasma concentrations were similar between the two groups (group means: 4.3 ± 0.4 μmol · kg-1 · min-1 and 128.2 ± 11.1 μmol/l). Acetate concentrations decreased in both groups with hyperinsulinemia but were

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Wu, Xinyue. "Lipid metabolism and insulin sensitivity." Theoretical and Natural Science 46, no. 1 (2024): 23–29. http://dx.doi.org/10.54254/2753-8818/46/20240400.

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Diabetes mellitus is known to be a serious chronic disease that requires great attention because it is the cancer that never dies and affects many people worldwide; its later complications are often numerous and frightening, and some of them may even affect their children as hereditary diseases. The relationship between lipid metabolism and insulin sensitivity in relation to diabetes is therefore even more important in this context. Insulin plays an important role as a key metabolic hormone in regulating glucose intake and maintaining lipid metabolic homeostasis. Insulin resistance interferes

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Tesi sul tema "Insulin metabolism"

Vidal, Alabró Anna. "Estudi de l’activació de la glucocinasa (GKA456V) en fetge perivenós." Doctoral thesis, Universitat de Barcelona, 2011. http://hdl.handle.net/10803/32022.

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La GK és clau en la regulació del metabolisme glucídic tant per la seva funció al pàncrees en què determina la secreció d’insulina, com per la seva funció al fetge on controla les vies d’utilització i d’emmagatzematge de glucosa. Per aquest motiu és una bona diana per a la teràpia de la diabetis, i diverses companyies farmacèutiques estan desenvolupant activadors sintètics de la GK (GKA) pel tractament de la diabetis de tipus 2. Els estudis de GKAs existents avaluen l’activació de la GK a nivell sistèmic i no es coneixen els seus efectes específis sobre la GK al fetge. Per indagar-los, com q

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Collison, Mary Williamson. "Insulin signalling in insulin resistance and cardiovascular disease syndromes." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366184.

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Kershner, David. "Oral Glucose Insulin Secretion Test for Identifying Patients with Insulin Resistance." ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/5634.

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Insulin resistance is an increasing public health issue with the current literature, suggesting reduced sensitivity of insulin leads to adult onset diabetes and associated downstream pathologies that reduce life expectancy. The main objectives of this study were to evaluate the ability of the Oral Glucose Insulin Secretion Test (OGIST) to identify insulin resistance and examine differences in the insulin sensitivity based on gender, age, and ethnicity. This study was supported by the insulin resistance theory which focuses on the reduced ability of insulin to bind to the cellular insulin recep

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Mashhedi, Haider. "Implicating insulin in neoplastic growth and metabolism." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=104681.

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In view of accumulating evidence that links obesity to increased cancer burden, there is interest in delineating the mechanisms by which obesity influences neoplastic growth. Increased insulin levels are commonly associated with obesity or ‘the metabolic syndrome' and thus the insulin receptor has been viewed as a potentially important molecular target for the treatment of certain cancers. To study the effects of insulin signaling attenuation on the growth of the experimental insulin-responsive mouse 4T1 breast cancer cells in vivo, we compared the effects of alloxan-induced insulin deficiency

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Shmueli, Ehoud. "Glucose metabolism and insulin resistance in cirrhosis." Thesis, University of Newcastle Upon Tyne, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308777.

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Laberge, Marie-Kristine. "Nck1 is required for ER stress-induced insulin resistance and regulation of IRS1-dependent insulin signalling." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111950.

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Activation of the Unfolded Protein Response (UPR) following stress in the Endoplasmic Reticulum (ER) is an important mechanism by which obesity results in insulin resistance and type II diabetes. We uncovered a role for the adaptor protein Nck in modulating the UPR. In this study, we report that obese Nck1-/- mice, which show lower levels of UPR in liver and adipose tissue, present improved insulin signalling in these tissues. We established that the effect of Nck1 is cell autonomous by showing that HepG2 cells treated with Nck1 siRNA have reduced ER stress-induced UPR and Insulin Receptor Sub

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Sanderson, Alison Louise. "Regulation of skeletal muscle metabolism." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318615.

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Field, Polly Ann. "The effects of insulin resistance on chylomicron metabolism." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302120.

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DeAngelis, Anthony Michael. "CEACAM1 : a link between insulin and lipid metabolism." Connect to full text in OhioLINK ETD Center, 2009. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=mco1243943993.

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Dissertation (Ph.D.)--University of Toledo, 2009.<br>"In partial fulfillment of the requirements for the degree of Doctor of Philosophy in Biomedical Sciences." Title from title page of PDF document. Bibliography: p. 57-61, p. 20-145.

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Denti, Paolo. "Nonlinear Mixed-Effects Modelling of Glucose-Insulin Metabolism." Doctoral thesis, Università degli studi di Padova, 2009. http://hdl.handle.net/11577/3426016.

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Diabetes mellitus is not only a very serious disease, causing discomfort and pain to millions across the world, but with the aging of the population and the prevalence of a sedentary lifestyle, it is assuming the proportion of a real epidemic, becoming a public health and social emergency. In answer to this call, research on diabetes has been intensely carried out in the past decades and the knowledge and understanding of its etiology have been significantly improved. However, investigation is still ongoing, many important questions are still unanswered, and the causes eliciting the pathogenes

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Più fonti

Libri sul tema "Insulin metabolism"

R, Flatt P., ed. Nutrient regulation of insulin secretion. Portland Press, 1991.

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Markussen, Jan. Human insulin by tryptic transpeptidations of porcine insulin and biosynthetic procursors. MTP Press, 1987.

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European Symposium on Metabolism (8th 2002 Padua, Italy). The metabolic syndrome: Diabetes, obesity, hyperlipidemia & hypertension : proceedings of the 8th European Symposium on Metabolism, held in Padua, Italy, between 2 and 5 October 2002. Edited by Crepaldi Gaetano, Tiengo Antonio, and Avogaro Angelo. Elsevier, 2003.

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Trumbach, Sabine. Glukose-Toleranz und Insulin-Sekretion unter simulierter Schwerelosigkeit. DFLVR, 1988.

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Dorrestijn, Jannette. Signal transduction related to the metabolic action of insulin. University of Leiden, 1998.

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Markolf, Hanefeld, and Leonhardt Wolfgang, eds. The metabolic syndrome: An integrated concept for the diagnosis and therapy of a cluster of diseases of civilisation. G. Fischer, 1997.

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Perus̆ic̆ová, Jindra. Glucose tolerance and secretion of insulin in chronic pancreatitis. Univerzita Karlova, 1990.

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European Symposium on Metabolism (7th 1998 Padova, Italy). Insulin resistance, metabolic diseases, and diabetic complications: Proceedings of the 7th European Symposium on Metabolism, Padova, September 30-October 3, 1998. Edited by Crepaldi Gaetano, Del Prato Stefano, and Tiengo Antonio. Elsevier, 1999.

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Yates, Tony Lomax. Insulin sensitivity functions predicted using deconvolution in a model of glucose metabolism. University of Salford, 2000.

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Gaetano, Crepaldi, Tiengo Antonio, and Manzato E, eds. Diabetes, obesity, and hyperlipidemias, V: The plurimetabolic syndrome : proceedings of the European Symposium on Metabolism, Padova, 24-26 May 1993. Excerpta Medica, 1993.

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Capitoli di libri sul tema "Insulin metabolism"

Larner, J. "Effects of Insulin on Glycogen Metabolism." In Insulin. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74098-5_16.

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Bressler, R., and J. J. Bahl. "Insulin Regulation of Metabolism Relevant to Gluconeogenesis." In Insulin. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74098-5_20.

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Frayn, Keith N., and Fredrik Karpe. "Insulin Action on Lipid Metabolism." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch3.

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Konrad, Daniel, Assaf Rudich, and Amira Klip. "Insulin-Mediated Regulation of Glucose Metabolism." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch2.

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Hileman, Stanley M., and Christian Bjørbaek. "Central Regulation of Peripheral Glucose Metabolism." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch7.

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Greenlund, Laura J. S., and K. Sreekumaran Nair. "The Effect of Insulin on Protein Metabolism." In Insulin Resistance. John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/0470011327.ch4.

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Mathias, Dietger. "Metabolism and Insulin Effect." In Fit and Healthy from 1 to 100 with Nutrition and Exercise. Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-65961-8_67.

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Patnaik, Akash, Jason W. Locasale, and Lewis C. Cantley. "Cancer Cell Metabolism." In Insulin-like Growth Factors and Cancer. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-0598-6_13.

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Guest, Paul C. "Insulin Resistance in Schizophrenia." In Reviews on Biomarker Studies of Metabolic and Metabolism-Related Disorders. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-12668-1_1.

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Carmena, R., J. F. Ascaso, A. Merchante, and F. J. Ampudia. "Insulin Resistance and Lipid Disorders." In Drugs Affecting Lipid Metabolism. Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0311-1_44.

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Atti di convegni sul tema "Insulin metabolism"

Tavares, Wagner, Rosângela Lima da Silva, Francivaldo José da Conceição Mendes, et al. "Effects of physiotherapy programs in the treatment and control of diabetes mellitus: A review." In V Seven International Multidisciplinary Congress. Seven Congress, 2024. http://dx.doi.org/10.56238/sevenvmulti2024-142.

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Diabetes mellitus (DM) is a chronic metabolic disorder characterized by persistent hyperglycemia, resulting from a deficiency in the production or action of the hormone insulin. This hormone is produced by pancreatic beta cells, and is responsible for regulating glucose metabolism, promoting its uptake by tissues. Its characteristics are: chronic hyperglycemia, which is generally accompanied by dyslipidemia; arterial hypertension; and endothelial dysfunction.

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Senhorinha, Gláucia Maria, Arlys Emanuel Mendes da Silva Santos, and Douglas Daniel Dophine. "The role of metabolic syndrome in Alzheimer’s disease." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.319.

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Background: Metabolic syndrome (MS) leads to the deposits formation of insoluble protein aggregates, neuroinflammation, oxidative stress, neuronal insulin resistance, progressive insulin resistance, desensitization and β-amyloid amyloidosis in the brain, besides direct ischemic effects which are closely associated with Alzheimer’s disease (AD).1 Objectives: The present study seeks to understand the role of the metabolic syndrome in the pathophysiology of Alzheimer’s disease and to describe preventive and therapeutic interventions. Methods: PUBMED and Web of Science were the databases used, the

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Rad, Milad Ghiasi, Aditya Immaneni, Megan McCabe, Massimiliano Pierobon, and Juan Cui. "A simulation model of glucose-insulin metabolism and implementation on OSG." In 2017 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2017. http://dx.doi.org/10.1109/bibm.2017.8217939.

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Staal, Odd Martin, Anders Lyngvi Fougner, Steinar Saelid, and Oyvind Stavdahl. "Glucose-insulin metabolism model reduction and parameter selection using sensitivity analysis." In 2019 American Control Conference (ACC). IEEE, 2019. http://dx.doi.org/10.23919/acc.2019.8814949.

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El-fadl, Rihab, Nasser Rizk, Amena Fadel, and Abdelrahman El Gamal. "The Profile of Hepatic Gene Expression of Glucose Metabolism in Mice on High Fat Diet." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0213.

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Obesity is a growing problem worldwide, and recent data indicated that 20% of the populations would be obese. Obesity arises as a multifactorial disease caused by inherited traits that interact with lifestyle factors such as diet and physical activity. The liver plays an essential role in the gluco-regulation via regulating glucose, lipid and protein metabolism. The process of glucose metabolism is controlled by a range of molecular mechanisms and genes which affect the metabolism of the liver during intake of high fat diet (HFD). The objective of this research is to investigate the profile of

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Wardelmann, K., M. Rath, JP Castro, et al. "Central acting Hsp10 regulates mitochondrial function, insulin sensitivity and impacts liver metabolism." In Diabetes Kongress 2021 – 55. Jahrestagung der DDG. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1727356.

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Taher, Zahraaakhan. "Estimation of Thyroid Function Tests in Type 2 Diabetes Mellitus." In 5th International Conference on Biomedical and Health Sciences. Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/biohs2024/paper.1278.

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Background and Objectives: Type 2 diabetes mellitus (T2DM) is an endocrine and metabolic disorder caused by insulin resistance, insulin hormone deficiency, and impaired insulin production form pancreatic beta cells. Thyroid hormones play a key role in glucose metabolism via several mechanisms. This study aimed to estimate the serum thyroid hormone levels in type 2 diabetic patients. Methods: The current case and control study was carried out in Erbil city. Blood specimens were collected from 100 subjects divided into two groups (50 case , 50 control). Each group consisted of (32 females and 18

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Campos, L. M., A. G. Rius, D. Kirovski, J. A. D. R. N. Appuhamy, T. F. V. Bompadre, and M. D. Hanigan. "Mammary gland amino acid affinity in response to different levels of dietary protein and insulin." In 6th EAAP International Symposium on Energy and Protein Metabolism and Nutrition. Wageningen Academic Publishers, 2019. http://dx.doi.org/10.3920/978-90-8686-891-9_120.

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Mohammed, Seenaa, Alaa Mahrath, and Tariq Abd. "Assessment of the Correlation of Lipasin Levels with HOMA-IR Score as an Index of Insulin Resistance in Type 2 Diabetic Iraqi Patients." In 5th International Conference on Biomedical and Health Sciences. Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/biohs2024/paper.1168.

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Lipasin, a hormone found in the liver and adipose tissue, is involved in glucose homeostasis and lipid metabolism. Lipasin levels in plasma are controlled by food and rise after glucose consumption. Because plasma glucose and insulin levels both rise after eating, we wanted to know if the rise in plasma insulin sensitivity, or both, is to blame for the rise in lipasin levels. According to this study, the patient group's lipasin concentration increased significantly (p-value˂ 0.05) in comparison to the control group, and the area under the curve (AUC) for lipasin was 75.75 %. As well, the level

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Mougiakakou, Stavroula G., Aikaterini Prountzou, Dimitra Iliopoulou, Konstantina S. Nikita, Andriani Vazeou, and Christos S. Bartsocas. "Neural Network based Glucose - Insulin Metabolism Models for Children with Type 1 Diabetes." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.260640.

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Rapporti di organizzazioni sul tema "Insulin metabolism"

Gao, Hui, Chen Gong, Shi-chun Shen, et al. A systematic review on the associations between prenatal phthalate exposure and childhood glycolipid metabolism and blood pressure: evidence from epidemiological studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.6.0111.

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Review question / Objective: The present systematic review was performed to obtain a summary of epidemiological evidence on the relationships of in utero exposure to phthalates with childhood glycolipid metabolism and blood pressure. Condition being studied: Childhood cardiovascular risk factors including blood pressure, lipid profile (e.g., triglycerides, total cholesterol, HDL−C, LDL−C) and glucose metabolism (e.g., insulin, insulin resistance, insulin sensitivity, glucose) were the interested outcomes. Eligibility criteria: In brief, epidemiological studies including cohort study, case-cont

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Research, Gratis. Brown Fat Activation: A Future Treatment for Obesity & Diabetes. Gratis Research, 2020. http://dx.doi.org/10.47496/gr.blog.01.

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Brown fat holds a promising therapeutic approach to prevent obesity and type 2 diabetes by its profound effects on body weight reduction, heat generation, increased insulin sensitivity and glucose metabolism regulation

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Boisclair, Yves R., and Arieh Gertler. Development and Use of Leptin Receptor Antagonists to Increase Appetite and Adaptive Metabolism in Ruminants. United States Department of Agriculture, 2012. http://dx.doi.org/10.32747/2012.7697120.bard.

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Objectives The original project had 2 major objectives: (1) To determine the effects of centrally administered leptin antagonist on appetite and adaptive metabolism in the sheep; (2) To develop and prepare second-generation leptin antagonists combining high binding affinity and prolonged in vivo half-life. Background Periods of suboptimal nutrition or exaggerated metabolic activity demands lead to a state of chronic energy insufficiency. Ruminants remain productive for a surprisingly long period of time under these circumstances by evoking adaptations sparing available energy and nutrients. Th

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Lekhanya, Portia Keabetswe, and Kabelo Mokgalaboni. Exploring the effectiveness of vitamin B12 complex and alpha-lipoic acid as a treatment for diabetic neuropathy. Protocol for systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0167.

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Review question / Objective: Does Alpha-Lipoic acid increase the uptake of glucose for better glycaemic control? Does vitamin B12 and Alpha-Lipoic acid improve inflammation? The aim of the study is to explore the effectiveness of Vitamin B12 and Alpha-Lipoic Acid as a possible treatment for diabetic neuropathy with major emphasis on markers of inflammation and glucose metabolism. Condition being studied: Diabetic Neuropathy (DN) is a heterogeneous type of nerve damage associated with diabetes mellitus, the condition most often damages nerves in the legs and feet. It presents both clinically an

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yu, luyou, jinping yang, xi meng, and yanhua lin. Effectiveness of the gut microbiota-bile acid pathway (BAS) in the treatment of Type 2 diabetes: A protocol for systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.7.0117.

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Review question / Objective: To systematically evaluate the efficacy of the intestinal microbiome - bile acid pathway (BAS) in the treatment of T2DM. Condition being studied: Bile acids (BAs), an important component of bile, are also metabolites derived from cholesterol and promote intestinal absorption and transportation of dietary lipids . Studies have shown that bile acid receptor agonists can promote glP-1 secretion and improve glucose metabolism in preclinical mouse models of obesity and insulin resistance , which may become a new therapeutic target for Type 2 diabetes. However, no system

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Meidan, Rina, and Robert Milvae. Regulation of Bovine Corpus Luteum Function. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7604935.bard.

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The main goal of this research plan was to elucidate regulatory mechanisms controlling the development, function of the bovine corpus luteum (CL). The CL contains two different sterodigenic cell types and therefore it was necessary to obtain pure cell population. A system was developed in which granulosa and theca interna cells, isolated from a preovulatory follicle, acquired characteristics typical of large (LL) and small (SL) luteal cells, respectively, as judged by several biochemical and morphological criteria. Experiments were conducted to determine the effects of granulosa cells removal

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McGuire, Mark A., Amichai Arieli, Israel Bruckental, and Dale E. Bauman. Increasing Mammary Protein Synthesis through Endocrine and Nutritional Signals. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7574338.bard.

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Objectives To determine endocrine factors that regulate the partitioning of amino acids by the mammary gland. To evaluate dietary flow and supply of energy and amino acids and their effects on milk protein synthesis and endocrine status. To use primary cultures of cow mammary epithelial cells to examine the role of specific factors on the rates and pattern of milk protein synthesis. Milk protein is an increasingly valuable component of milk but little is known regarding the specific hormonal and nutritional factors controlling milk protein synthesis. The research conducted for this project has

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Butler, Walter R., Uzi Moallem, Amichai Arieli, Robert O. Gilbert, and David Sklan. Peripartum dietary supplementation to enhance fertility in high yielding dairy cows. United States Department of Agriculture, 2007. http://dx.doi.org/10.32747/2007.7587723.bard.

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Objectives of the project: To evaluate the effects of a glucogenic supplement during the peripartum transition period on insulin, hepatic triglyceride accumulation, interval to first ovulation, and progesterone profile in dairy cows. To compare benefits of supplemental fats differing in fatty acid composition and fed prepartum on hepatic triglyceride accumulation, interval to first ovulation, progesterone profile, and uterine prostaglandin production in lactating dairy cows. To assess the differential and carry-over effects of glucogenic and fat supplements fed to peripartum dairy cows on ster

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Chansiripornchai, Piyarat, and Thanaphum Osathanon. In vitro differentiation of mesenchymal stem cells from dental and oral tissues into Islet-like cell cluster. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.93.

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Diabetes mellitus is a complicated metabolic disorder resulting in hyperglycemia and long-term complications e.g. diabetic encephalopathy and neuropathy. Treatments of diabetes and its complications have faced many obstacles. Trend of stem cells (SCs)-based therapy has been proposed as a novel approach. Though, the study using dental SCs in this regard is yet lacking. In this study, human dental pulp SCs (hDPSCs) and human periodontal ligament SCs (hPDLSCs) were employed. The results illustrated the capability of differentiation toward islet-like cells (ILCs) cluster / insulin-producing cells

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Sowjanya, Dr Kaniti, Dr Bongu Srinivas, and Dr Metta Lakshmana Rao. A STUDY ON FIBROSCAN COMPARED TO AST TO PLATELET RATIO INDEX(APRI) FOR ASSESSMENT OF LIVER FIBROSIS WITH NONALCOHOLIC FATTY LIVER DISEASE(NAFLD). World Wide Journals, 2023. http://dx.doi.org/10.36106/ijar/1606016.

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Background : Nonalcoholic fatty liver disease (NAFLD) is increasingly recognized as a cause of chronic liver disease, and often results in the devastating outcomes of decompensated liver cirrhosis and hepatocellular carcinoma and is an important feature of metabolic syndromes and insulin resistance. The assessment of liver brosis is essential for predicting the prognosis and outcome of all forms of chronic liver disease. A liver biopsy is the gold standard for the assessment of liver brosis, but it has its limitations, which include life-threatening complications. Alternative methods of non-

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