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Letteratura scientifica selezionata sul tema "Ischemia"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "Ischemia"

1

Lee, Choong-Hyun, Tae-Kyeong Lee, Dae Won Kim, et al. "Relationship between Neuronal Damage/Death and Astrogliosis in the Cerebral Motor Cortex of Gerbil Models of Mild and Severe Ischemia and Reperfusion Injury." International Journal of Molecular Sciences 23, no. 9 (2022): 5096. http://dx.doi.org/10.3390/ijms23095096.

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Neuronal loss (death) occurs selectively in vulnerable brain regions after ischemic insults. Astrogliosis is accompanied by neuronal death. It can change the molecular expression and morphology of astrocytes following ischemic insults. However, little is known about cerebral ischemia and reperfusion injury that can variously lead to damage of astrocytes according to the degree of ischemic injury, which is related to neuronal damage/death. Thus, the purpose of this study was to examine the relationship between damage to cortical neurons and astrocytes using gerbil models of mild and severe tran

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2

Park, Kwon Moo, Ang Chen, and Joseph V. Bonventre. "Prevention of Kidney Ischemia/Reperfusion-induced Functional Injury and JNK, p38, and MAPK Kinase Activation by Remote Ischemic Pretreatment." Journal of Biological Chemistry 276, no. 15 (2001): 11870–76. http://dx.doi.org/10.1074/jbc.m007518200.

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MAPK activities, including JNK, p38, and ERK, are markedly enhanced after ischemiain vivoand chemical anoxiain vitro. The relative extent of JNK, p38, or ERK activation has been proposed to determine cell fate after injury. A mouse model was established in which prior exposure to ischemia protected against a second ischemic insult imposed 8 or 15 days later. In contrast to what was observed after 30 min of bilateral ischemia, when a second period of ischemia of 30- or 35-min duration was imposed 8 days later, there was no subsequent increase in plasma creatinine, decrease in glomerular filtrat

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3

Lee, H. Thomas, and Charles W. Emala. "Protective effects of renal ischemic preconditioning and adenosine pretreatment: role of A1 and A3receptors." American Journal of Physiology-Renal Physiology 278, no. 3 (2000): F380—F387. http://dx.doi.org/10.1152/ajprenal.2000.278.3.f380.

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Renal ischemia and reperfusion during aortic and renal transplant surgery result in ischemic-reperfusion injury. Ischemic preconditioning and adenosine infusion before ischemia protect against ischemic-reperfusion injury in cardiac and skeletal muscle, but these protective phenomena have not been demonstrated in the kidney. Rats were randomized to sham operation, 45-min renal ischemia, ischemic preconditioning with four cycles of 8-min renal ischemia and 5-min reperfusion followed by 45-min renal ischemia, systemic adenosine pretreatment before 45-min renal ischemia, or pretreatments with sele

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4

Kanazawa, Masato, Tetsuya Takahashi, Masanori Ishikawa, Osamu Onodera, Takayoshi Shimohata, and Gregory J. del Zoppo. "Angiogenesis in the ischemic core: A potential treatment target?" Journal of Cerebral Blood Flow & Metabolism 39, no. 5 (2019): 753–69. http://dx.doi.org/10.1177/0271678x19834158.

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The ischemic penumbra is both a concept in understanding the evolution of cerebral tissue injury outcome of focal ischemia and a potential therapeutic target for ischemic stroke. In this review, we examine the evidence that angiogenesis can contribute to beneficial outcomes following focal ischemia in model systems. Several studies have shown that, following cerebral ischemia, endothelial proliferation and subsequent angiogenesis can be detected beginning four days after cerebral ischemia in the border of the ischemic core, or in the ischemic periphery, in rodent and non-human primate models,

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5

Zhao, Ling, Qiwei Liao, Yueting Zhang, Shufen Tan, Shuqing Li, and Tingyu Ke. "Ischemic Postconditioning Mitigates Retinopathy in Tree Shrews with Diabetic Cerebral Ischemia." Journal of Diabetes Research 2020 (February 12, 2020): 1–10. http://dx.doi.org/10.1155/2020/6286571.

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Ischemic postconditioning (PC) is proved to efficiently protect diabetic patients with acute myocardial infarction from ischemia-reperfusion injury. We aimed to explore the protective roles of ischemic PC on diabetic retinopathy in tree shrews with diabetic cerebral ischemia. A diabetic tree shrew model was established through high-fat diet feeding combined with streptozotocin (STZ) injection, while cortical thrombotic cerebral ischemia was induced photochemically. Tree shrews were divided into the normal control group, sham operation group, diabetes mellitus group, diabetes mellitus+cerebral

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6

Li, Chuanfu, Race L. Kao, Tuanzhu Ha, Jim Kelley, I. William Browder та David L. Williams. "Early activation of IKKβ during in vivo myocardial ischemia". American Journal of Physiology-Heart and Circulatory Physiology 280, № 3 (2001): H1264—H1271. http://dx.doi.org/10.1152/ajpheart.2001.280.3.h1264.

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We have demonstrated that in vitro brief ischemia activates nuclear factor (NF)-κB in rat myocardium. We report in vivo ischemia-reperfusion (I/R)-induced NF-κB activation, IκB kinase -β (IKKβ) activity, and IκBα phosphorylation and degradation in rat myocardium. Rat hearts were subjected to occlusion of the coronary artery for up to 45 min or occlusion for 15 min followed by reperfusion for up to 3 h. Cytoplasmic and nuclear proteins were isolated from ischemic and nonischemic areas of each heart. NF-κB activation was increased in the ischemic area (680%) after 10 min of ischemia and in the n

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7

Zhan, Xinhua, Bradley P. Ander, Glen Jickling, et al. "Brief Focal Cerebral Ischemia That Simulates Transient Ischemic Attacks in Humans Regulates Gene Expression in Rat Peripheral Blood." Journal of Cerebral Blood Flow & Metabolism 30, no. 1 (2009): 110–18. http://dx.doi.org/10.1038/jcbfm.2009.189.

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Blood gene expression profiles of very brief (5 and 10 mins) focal ischemia that simulates transient ischemic attacks in humans were compared with ischemic stroke (120 mins focal ischemia), sham, and naïve controls. The number of significantly regulated genes after 5 and 10 mins of cerebral ischemia was 39 and 160, respectively (fold change ⩾∣1.5∣ and P<0.05). There were 103 genes common to brief focal ischemia and ischemic stroke. Ingenuity pathway analysis showed that genes regulated in the 5 mins group were mainly involved in small molecule biochemistry. Genes regulated in the 10 mins gr

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8

KAKOKI, MASAO, YASUNOBU HIRATA, HIROSHI HAYAKAWA, et al. "Effects of Tetrahydrobiopterin on Endothelial Dysfunction in Rats with Ischemic Acute Renal Failure." Journal of the American Society of Nephrology 11, no. 2 (2000): 301–9. http://dx.doi.org/10.1681/asn.v112301.

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The role of nitric oxide (NO) in ischemic renal injury is still controversial. NO release was measured in rat kidneys subjected to ischemia and reperfusion to determine whether (6R)-5,6,7,8-tetrahydro-L-biopterin (BH4), a cofactor of NO synthase (NOS), reduces ischemic injury. Twenty-four hours after bilateral renal arterial clamp for 45 min, acetylcholine-induced vasorelaxation and NO release were reduced and renal excretory function was impaired in Wistar rats. Administration of BH4 (20 mg/kg, by mouth) before clamping resulted in a marked improvement of those parameters (10-8 M acetylcholin

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9

Kim, Gyung W., Taku Sugawara, and Pak H. Chan. "Involvement of Oxidative Stress and Caspase-3 in Cortical Infarction after Photothrombotic Ischemia in Mice." Journal of Cerebral Blood Flow & Metabolism 20, no. 12 (2000): 1690–701. http://dx.doi.org/10.1097/00004647-200012000-00008.

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Apoptosis-related cell death is linked to oxidative stress and caspases in experimental cerebral ischemia, However, the role of oxidative stress in caspase activation and subsequent apoptotic cell death after cerebral ischemia is unknown, The authors evaluated the role of oxidative stress in ischemic cerebral infarction after photothrombosis and the relation between oxidative stress and caspase-related cell death 6 and 24 hours after ischemia with and without U-74389G, a potent free radical scavenger (10 mg/kg, 30 minutes before and after ischemia induction). Reactive oxygen species, detected

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Rivera, Maria Jesus, Monica Cavia-Saiz, Pilar Muñiz, María A. Risalde, and Angélica Martínez-Delgado. "Ischemic Preconditioning on Secondary Arterial and Venous Ischemia in Pedicled Axial Flaps in Wistar Rats." Journal of Reconstructive Microsurgery Open 10, no. 01 (2025): e19-e31. https://doi.org/10.1055/a-2624-2776.

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Abstract Background Microvascular complications, particularly secondary arterial and venous ischemia, pose significant challenges in reconstructive surgery. This study investigates the potential protective effects of ischemic preconditioning on flap survival, anatomopathological alterations, and immunological responses in pedicled axial flaps subjected to secondary ischemia. Methods Adult male Wistar rats underwent arterial or venous ischemia, with and without ischemic preconditioning. Histological assessments, immunohistochemistry studies, and biochemical analyses were conducted to evaluate t

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Tesi sul tema "Ischemia"

1

Du, Ying. "Ischemic and pharmacological preconditioning of rat myocardium : effects on ischemia-reperfusion injury /." View abstract or full-text, 2005. http://library.ust.hk/cgi/db/thesis.pl?BICH%202005%20DU.

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Christensen, Thomas. "Experimental focal cerebral ischemia : pathophysiology, metabolism and pharmacology of the ischemic penumbra /." Copenhagen, 2007. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=016143698&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Dowden, Jennifer. "Characterizing the neuroprotective efficacy of ischemic preconditioning (ischemic tolerance) : is age an important factor? /." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0019/NQ54834.pdf.

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Harhous, Zeina. "Deciphering the Interlink between STAT3 and MAPKs in Ischemia/Reperfusion and Ischemic Conditioning." Thesis, Lyon, 2019. http://www.theses.fr/2019LYSE1145.

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Abstract (sommario):

Les maladies cardiovasculaires sont une des principales causes de morbidité et de mortalité au monde. La plus courante est l’infarctus du myocarde définit pathologiquement par la mortalité cellulaire dû à une ischémie prolongée d’une partie du ventricule gauche. L'ischémie est caractérisée par un apport sanguin insuffisant causé par une obstruction d’une artère coronaire. La restauration, en clinique, du flux sanguin, appelée reperfusion, est considérée comme la méthode la plus efficace contre les dommages ischémiques. Paradoxalement, cette restauration du flux sanguin est associée à une exace

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Zhang, Jitian. "Nebulized phosphodiesterase 3 inhibitor during warm ischemia attenuates pulmonary ischemia-reperfusion injury." Kyoto University, 2009. http://hdl.handle.net/2433/126442.

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Li, Yan. "Inhibitory synpatic transmission in striatal neurons after transient cerebral ischemia." Connect to resource online, 2009. http://hdl.handle.net/1805/2021.

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Thesis (Ph.D.)--Indiana University, 2009.<br>Title from screen (viewed on December 1, 2009). Department of Anatomy and Cell Biology, Indiana University-Purdue University Indianapolis (IUPUI). Advisor(s): Zao C. Xu, Feng C. Zhou, Charles R. Yang, Theodore R. Cummins. Includes vitae. Includes bibliographical references (leaves 115-135).

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Keasey, Matthew P. "MicroRNAs in Cerebral Ischemia." Thesis, University of Bristol, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526014.

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Lipšic, Erik. "Erythropoietin in cardiac ischemia." [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2006. http://irs.ub.rug.nl/ppn/293076030.

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Aoyama, Akihiro. "Post-ischemic Infusion of Atrial Natriuretic Peptide Attenuates Warm Ischemia-Reperfusion Injury in Rat Lung." Kyoto University, 2011. http://hdl.handle.net/2433/142544.

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Hultgren, Rebecka. "Lower limb ischemia in women /." Stockholm, 2004. http://diss.kib.ki.se/2003/91-7349-798-3.

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Libri sul tema "Ischemia"

1

Walz, Wolfgang. Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1385/0896035409.

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2

Hacke, Werner, Herman J. Gelmers, Michael Hennerici, and Günter Krämer. Cerebral Ischemia. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75548-4.

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3

Gusev, Eugene, and Veronica I. Skvortsova. Brain Ischemia. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9248-2.

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4

v. Arnim, Th, and A. Maseri, eds. Silent Ischemia. Steinkopff, 1987. http://dx.doi.org/10.1007/978-3-662-12997-5.

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Walz, Wolfgang, ed. Cerebral Ischemia. Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-479-5.

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6

Dhalla, Naranjan S., Ian R. Innes, and Robert E. Beamish, eds. Myocardial Ischemia. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2055-5.

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Caplan, Louis R., ed. Brain Ischemia. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2073-5.

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Cokkinos, Dennis V., Constantinos Pantos, Gerd Heusch, and Heinrich Taegtmeyer, eds. Myocardial Ischemia. Springer US, 2006. http://dx.doi.org/10.1007/0-387-28658-6.

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9

Gusev, E. I. Brain ischemia. Kluwer Academic/Plenum Publishers, 2003.

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10

J, Boley Scott, and Brandt Lawrence J, eds. Intestinal ischemia. Saunders, 1992.

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Capitoli di libri sul tema "Ischemia"

1

Hasan, Aliya G., and Joel S. Levine. "Ischemia/Ischemic Colitis." In Textbook of Clinical Gastroenterology and Hepatology. Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118321386.ch55.

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Romano, Stefania, and Luigia Romano. "Ischemia (Acute Mesenteric Ischemia and Ischemic Colitis)." In CT of the Acute Abdomen. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/174_2010_82.

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Roth, Elliot J. "Ischemia." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_2186.

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Roth, Elliot J. "Ischemia." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-56782-2_2186-2.

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Mirzakhani, Hooman, and Ala Nozari. "Ischemia." In Chemistry and Biochemistry of Oxygen Therapeutics. John Wiley & Sons, Ltd, 2011. http://dx.doi.org/10.1002/9781119975427.ch10.

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Cheung, Raymond T. F., and Vladimir Hachinski. "Ischemia." In Molecular Mechanisms of Dementia. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-59259-471-9_17.

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Mehlhorn, Heinz. "Ischemia." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_3974.

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Mehlhorn, Heinz. "Ischemia." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_3974-1.

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Urbach, Horst. "Ischemia." In MRI in Epilepsy. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/174_2012_781.

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Gooch, Jan W. "Ischemia." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14054.

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Atti di convegni sul tema "Ischemia"

1

Makhir, Abdelmalek, My Hachem El Yousfi Alaoui, Abdelilah Jilbab, and Larbi Belarbi. "Advancing Ischemia Detection: Multi-Model Classification of ST and T Changes in Ischemia ECG Signals." In 2024 International Conference on Connected Innovation and Technology (ICCITX). IEEE, 2024. https://doi.org/10.1109/iccitx61791.2024.11071138.

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Shafawi, Hazimah Ahmad, and Sharifalillah Nordin. "Myocardial Ischemia Classification Through ECG Using Deep Learning." In 2024 5th International Conference on Artificial Intelligence and Data Sciences (AiDAS). IEEE, 2024. http://dx.doi.org/10.1109/aidas63860.2024.10730738.

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3

Lee, Gija, Seokkeun Choi, Sungwook Kang, et al. "Changes in Extracellular Glutamate Release on Repetitive Transient Occlusion in Global Ischemia Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206602.

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During the operation, surgeons in neurosurgical area usually performed the multiple temporary occlusions of parental artery which may induce the neuronal damage. It is generally thought that neuronal damage by cerebral ischemia is associated with extracellular concentrations of the excitatory amino acids. In this experiment, we measured the dynamics of extracellular glutamate release in 11 vessel occlusion (VO) model during repeated within short interval. Changes in cerebral blood flow were monitored by laser-Doppler flowmetry simultaneously with cortical glutamate level measured by amperometr

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4

Xia, Cao, Ma Kewei, Han Bing, et al. "Protective effects of ischemic preconditioning on myocardial ischemia reperfusion injury." In 2011 International Conference on Human Health and Biomedical Engineering (HHBE). IEEE, 2011. http://dx.doi.org/10.1109/hhbe.2011.6027906.

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Fisher, M., and R. Francis. "ALTERED COAGULATION IN CEREBRAL ISCHEMIA PATIENTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644345.

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We investigated coagulation changes in a group of patients with cerebral ischemia, ranging from transient ischemic attacks to cerebral infarction. Patients were studied acutely (within 72 hours of onset of ischemia) and again approximately 2 months following the initial examination. We evaluated platelet activation, fibrin generation, and fibrinolysis by measuring plasma beta-thromboglobulin (BTG), fibrinopeptide A (FPA), and fibrinopeptide B-beta 1-42 (FPB), respectively. We compared measurements in cerebral ischemia patients with a group of age -and sex-matched neurological inpatients withou

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Шварц, Владислав Александрович, Марина Станиславовна Кудабаева, Илья Леонидович Губский, Дарья Дмитриевна Наместникова, and Марина Юрьевна Ходанович. "LONG-TERM DYNAMIC OF ISCHEMIC LESION VOLUME AND THE BRAIN HEMISPHERES’ VOLUME IN THE MODEL OF FOCAL ISCHEMIA MODEL IN RAT." In Высокие технологии и инновации в науке: сборник избранных статей Международной научной конференции (Санкт-Петербург, Сентябрь 2020). Crossref, 2020. http://dx.doi.org/10.37539/vt187.2020.52.45.003.

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В исследовании изучалась динамика объема ишемического очага и объемов полушарий мозга у животных с локальной ишемией в течении 2 месяцев после ишемии при помощи ручной сегментации. Были выявлены значимые различия между объемами полушарий на 1, 3, 14, 21, 30, 42 день исследования (p<0,01-0,05),, а также резкий рост объема ишемического поражения в течение 1-3 суток, после чего его объем монотонно уменьшался. Ischemic lesion volume and the brain hemisphere volume long-term changes were evaluated during 2 month after focal ischemia in rats using manual segmentation,. Significant differences wer

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Breton, Michael E., Graham E. Quinn, Sara S. Keene, Janet C. Dahmen, and Alexander J. Brucker. "Analysis of ERG Parameters in Central Retinal Vein Occlusion." In Noninvasive Assessment of the Visual System. Optica Publishing Group, 1988. http://dx.doi.org/10.1364/navs.1988.wa1.

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Effective treatment of the complications resulting from an ischemic condition such as central retinal vein occlusion (CRVO) may depend on the accuracy of early diagnosis. In CRVO, relative ischemia of a significant proportion of retina has been associated with ocular morbidity in the form of rubeosis iridis and neovascular glaucoma (1,2,3). Fluorescein angiography (FA) has been used routinely to determine the degree of retinal ischemia in CRVO as in other occlusive vascular diseases of the retina. However, FA interpretation, based on non-quantitative visual judgements, may lack sufficient sens

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Slotvitsky, M. M., A. K. Berezhnoy, T. O. Sergeeva, et al. "REGISTRATION AND MATHEMATICAL MODELING OF THE NADH PHOTOBLEACHING DYNAMICS IN CARDIAC TISSUE FOR THE INDICATION OF ISCHEMIC DAMAGE." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-160.

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In this study, we propose a new approach to noninvasive detection of cardiac tissue ischemia. The nonlinear dynamics of NADH molecules fluorescence changes under the influence of constant exciting light is used as a source of information. This allows us to obtain a map of ischemic regions in real time, as well as to separate reversible and irreversible stages of ischemic damage.

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La Barck, Anthony J., Jennifer E. Akers, and Thomas L. Merrill. "Tissue Oxygen Transfer During Reperfusion and Post-Conditioning." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53064.

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Heart disease is the leading cause of death in the United States. Ischemic heart disease occurs when coronary blood flow to the heart is reduced, limiting the amount of oxygen and nutrients the heart receives. When blood flow is restored after a percutaneous transluminal coronary intervention (PCI), rapid reperfusion from sudden balloon deflation can cause further injury to oxygen-starved tissue, leading to increased cell injury and cell death. Studies in animal models with ischemic heart disease have shown that reperfusion injury may account for up to 50% of the final infarct size [1]. Post-c

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Toczyl, Samanta, Jörg Reifart, and Paul A. Iaizzo. "Effect of Myocardial Postconditioning with Polyunsaturated Fatty Acids on the Hemodynamics of Reanimated Swine Hearts." In 2024 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2024. http://dx.doi.org/10.1115/dmd2024-1072.

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Abstract Even a small to moderate degree of global cardiac ischemia during a cardiac procedure or surgery can significantly contribute to a patient’s postoperative morbidity and mortality. While various ischemic damage mitigation strategies have been implemented, the efficacy of the intravenous administration of polyunsaturated fatty acids has not been extensively studied. This research evaluates the impact of myocardial pharmacological postconditioning via intravenous infusion of lipid emulsions on the hemodynamic performance during global cardiac ischemia using Visible Heart® Methodologies:

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Rapporti di organizzazioni sul tema "Ischemia"

1

Li, Xiao, Fayang Ling, Wenchuan Qi, et al. Preclinical Evidence of Acupuncture on infarction size of Myocardial ischemia: A Systematic Review and Meta-Analysis of Animal Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.6.0044.

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Review question / Objective: Whether acupuncture is effective for infarction size on myocardial ischemia rat models. Condition being studied: Myocardial ischemia is a typical pathological condition of coronary heart disease (CHD), which has been a global issue with high incidence and mortality. Myocardial infarction caused by myocardial ischemia leads to cardiac dysfunction, and the size of myocardial infarction also determines the recovery and prognosis of cardiac function. Acupuncture, a long history of traditional Chinese medicine, is widely used to treat symptoms like thoracalgia and palpi

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2

Axford, T., J. Dearani, I. Khait, S. Khuri, and C. R. Valeri. On-Line Metabolic Monitoring of Tissue Ischemia. Defense Technical Information Center, 1991. http://dx.doi.org/10.21236/ada360147.

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3

Omaye, Stanley T. Efficacy of Gamma-glutamylcysteine (GGC) in Ischemia-reperfusion Injury. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada568392.

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4

Omaye, Stanley T. Efficacy of Gamma-glutamylcysteine (GGC) in Ischemia-reperfusion Injury. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada579994.

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5

chen, yaru, kai wang, jin yan, and zhenghua zhu. Comparison of the protective effect of ischemic preconditioning and remote ischemic preconditioning against ischemia reperfusion injury after hepatectomy: a systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.7.0007.

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6

Oeltgen, Peter R. Neuroprotective Effects of Opioid-Like Hibernation Factors in Cerebral Ischemia. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada424443.

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7

Merrill, Alfred H., and Jr. Subcellular Signal Transduction Systems in the Cellular Trauma of Ischemia. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada229876.

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8

Zafirovski, Aleksandar, Marija Zafirovska, Dimitrij Kuhelj, and Tadeja Pintar. The impact of biomarkers in early detection of acute mesenteric ischemia. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.12.0087.

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9

Zheng, Jinghui. Quercetin for Myocardial Ischemia/Reperfusion Injury: A Preclinical Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.4.0162.

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10

Wu, Xiaoqi, Maoxia Fan, Yaobo Pan, and Dona Guo. Quality of Evidence Supporting the Effects of Ginkgo Terpene Lactone Preparations in Ischemic Stroke: An Overview of Systematic Reviews and Meta-Analyses. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.9.0124.

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Review question / Objective: 2.2.1 Type of studies SRs/MAs of Randomized Controlled Trials (RCTs) of GTLP for IS in any language. 2.2.2 Type of Participants Included patients were diagnosed with IS according to international or national standards, regardless of race, age, gender, time of onset, and source of cases. 2.2.3 Type of Intervention The intervention method in the control group was routine treatment, and the intervention method in the intervention group was GTLP treatment or GTLP combined with the treatment of the control group. 2.2.4 Types of outcomes Conclusions at least need to incl

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