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Letteratura scientifica selezionata sul tema "Kidney glomerulus Diseases"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 21 febbraio 2023

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Articoli di riviste sul tema "Kidney glomerulus Diseases"

1

Beeman, Scott C., Min Zhang, Lina Gubhaju, Teresa Wu, John F. Bertram, David H. Frakes, Brian R. Cherry, and Kevin M. Bennett. "Measuring glomerular number and size in perfused kidneys using MRI." American Journal of Physiology-Renal Physiology 300, no. 6 (June 2011): F1454—F1457. http://dx.doi.org/10.1152/ajprenal.00044.2011.

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The goal of this work was to nondestructively measure glomerular (and thereby nephron) number in the whole kidney. Variations in the number and size of glomeruli have been linked to many renal and systemic diseases. Here, we develop a robust magnetic resonance imaging (MRI) technique based on injection of cationic ferritin (CF) to produce an accurate measurement of number and size of individual glomeruli. High-field (19 Tesla) gradient-echo MR images of perfused rat kidneys after in vivo intravenous injection of CF showed specific labeling of individual glomeruli with CF throughout the kidney.

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2

KITAMURA, MASANORI. "Renal Transfer of Genetically Engineered Cells." Journal of the American Society of Nephrology 11, suppl 2 (November 2000): S154—S158. http://dx.doi.org/10.1681/asn.v11suppl_2s154.

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Abstract. For many years, ex vivo gene transfer has been used for genetic manipulation of various organs. In the kidney, ex vivo gene transfer was reported using mesangial cells and macrophages. In rats, cultured cells injected into the renal artery are accumulated selectively in the glomerulus. With this approach, it is possible to transfer genetically engineered cells to normal and diseased glomeruli. The transfer of genetically engineered cells to glomeruli can be used for several purposes. With the use of resident glomerular cells engineered in vitro, it is possible to examine how the cell

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3

Wang, Honglian, Jingyi Sheng, Huijun He, Xiaocui Chen, Jinhong Li, Ruizhi Tan, Li Wang, and Hui-Yao Lan. "A simple and highly purified method for isolation of glomeruli from the mouse kidney." American Journal of Physiology-Renal Physiology 317, no. 5 (November 1, 2019): F1217—F1223. http://dx.doi.org/10.1152/ajprenal.00293.2019.

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Highly purified mouse glomeruli are of great value for studying glomerulus-associated kidney diseases. Here, we developed a simple and rapid procedure for mouse glomerular isolation with large quantity and high purity based on the combination of size-selective sieving and differential adhesion techniques, which we termed the “differential adhesion method.” In this method, mouse renal cortices were minced and digested with collagenase. Glomeruli were disassociated from tubules by successive sieving through 105-, 75-, and 40-μm cell strainers. The retained glomeruli-rich preparation on the 40-μm

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4

Yoshida, Yutaka, Masahito Miyamoto, Izumi Taguchi, Bo Xu, Ying Zhang, Eishin Yaoita, Hidehiko Fujinaka, and Tadashi Yamamoto. "Human kidney glomerulus proteome and biomarker discovery of kidney diseases." PROTEOMICS – CLINICAL APPLICATIONS 2, no. 3 (March 2008): 420–27. http://dx.doi.org/10.1002/prca.200780016.

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5

Zhu, D., Y. Kim, M. W. Steffes, T. J. Groppoli, R. J. Butkowski, and S. M. Mauer. "Application of electron microscopic immunocytochemistry to the human kidney: distribution of type IV and type VI collagen in normal human kidney." Journal of Histochemistry & Cytochemistry 42, no. 5 (May 1994): 577–84. http://dx.doi.org/10.1177/42.5.8157929.

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We used immunogold electron microscopic (IEM) techniques with periodate-lysine-paraformaldehyde-fixed and Lowicryl-embedded or cryopreserved tissues to study the distribution of alpha 1(IV) and alpha 3(IV) chains of Types IV and VI collagen in glomerular basement membrane (GBM) and mesangial matrix of glomeruli in normal human kidneys. Monoclonal antibodies to alpha 1(IV) and alpha 3(IV) collagen chains and Type VI collagen could be detected only with cryoultramicrotomy, whereas polyclonal anti-Type IV collagen antibody was detectable in Lowicryl-embedded tissue. Ultrastructural detail was bet

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6

PATRAKKA, JAAKKO, VESA RUOTSALAINEN, ILKKA KETOLA, CHRISTER HOLMBERG, MARKKU HEIKINHEIMO, KARL TRYGGVASON, and HANNU JALANKO. "Expression of Nephrin in Pediatric Kidney Diseases." Journal of the American Society of Nephrology 12, no. 2 (February 2001): 289–96. http://dx.doi.org/10.1681/asn.v122289.

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Abstract. Nephrin is a podocyte cell adhesion protein located at the slit diaphragm area of the kidney glomerulus. Mutations in the nephrin gene (NPHS1) lead to congenital nephrosis, suggesting that nephrin is essential for the glomerular filtration barrier. This prompted this study of the expression of nephrin in acquired pediatric kidney diseases usingin situhybridization and immunohistochemistry.In situhybridization for nephrin mRNA was performed in biopsy samples from patients with proteinuria caused by minimal change nephrosis, focal segmental glomerulosclerosis, and membranous nephropath

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7

SUZUKI, DAISUKE, TOSHIO MIYATA, MASAOMI NANGAKU, HIDEO TAKANO, NOBORU SAOTOME, MASAO TOYODA, YASUO MORI, et al. "Expression of Megsin mRNA, a Novel Mesangium-Predominant Gene, in the Renal Tissues of Various Glomerular Diseases." Journal of the American Society of Nephrology 10, no. 12 (December 1999): 2606–13. http://dx.doi.org/10.1681/asn.v10122606.

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Abstract. Mesangial cells play an important role in maintaining a structure and function of the glomerulus and in the pathogenesis of glomerular diseases. Recently, we discovered a new mesangium-predominant gene termed “megsin.” Megsin is a novel protein that belongs to the serine protease inhibitor (serpin) superfamily. To elucidate the pathophysiologic role of megsin in the kidney, the expression and localization of megsin mRNA in renal tissues of patients with IgA nephropathy (IgA-N), diabetic nephropathy (DN), minimal change nephrotic syndrome (MCNS), membranous nephropathy (MN), and norma

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8

Rodriguez, Patricia Q., Asmundur Oddsson, Lwaki Ebarasi, Bing He, Kjell Hultenby, Annika Wernerson, Christer Betsholtz, Karl Tryggvason, and Jaakko Patrakka. "Knockdown of Tmem234 in zebrafish results in proteinuria." American Journal of Physiology-Renal Physiology 309, no. 11 (December 1, 2015): F955—F966. http://dx.doi.org/10.1152/ajprenal.00525.2014.

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Podocytes are highly specialized epithelial cells located at the outer aspects of the glomerular capillary tuft and critical components of the kidney filtration barrier. To maintain their unique features, podocytes express a number of proteins that are only sparsely found elsewhere in the body. In this study, we have identified four (Tmem234, Znf185, Lrrc49, and Slfn5) new highly podocyte-enriched proteins. The proteins are strongly expressed by podocytes, while other parts of the kidney show only weak or no expression. Tmem234, Slfn5, and Lrrc49 are located in foot processes, whereas Znf185 i

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9

Beck, Karl-Friedrich, and Josef Pfeilschifter. "The Pathophysiology of H2S in Renal Glomerular Diseases." Biomolecules 12, no. 2 (January 26, 2022): 207. http://dx.doi.org/10.3390/biom12020207.

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Abstract (sommario):

Renal glomerular diseases such as glomerulosclerosis and diabetic nephropathy often result in the loss of glomerular function and consequently end-stage renal disease. The glomerulus consists of endothelial cells, mesangial cells and glomerular epithelial cells also referred to as podocytes. A fine-tuned crosstalk between glomerular cells warrants control of growth factor synthesis and of matrix production and degradation, preserving glomerular structure and function. Hydrogen sulfide (H2S) belongs together with nitric oxide (NO) and carbon monoxide (CO) to the group of gasotransmitters. Durin

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10

Lee, Hsi-Chieh, and Ahmad Fauzan Aqil. "Combination of Transfer Learning Methods for Kidney Glomeruli Image Classification." Applied Sciences 12, no. 3 (January 20, 2022): 1040. http://dx.doi.org/10.3390/app12031040.

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The rising global incidence of chronic kidney disease necessitates the development of image categorization of renal glomeruli. COVID-19 has been shown to enter the glomerulus, a tissue structure in the kidney. This study observes the differences between focal-segmental, normal and sclerotic renal glomerular tissue diseases. The splitting and combining of allied and multivariate models was accomplished utilizing a combined technique using existing models. In this study, model combinations are created by using a high-accuracy accuracy-based model to improve other models. This research exhibits e

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Più fonti

Tesi sul tema "Kidney glomerulus Diseases"

1

Wang, Yang. "Murine adriamycin-induced nephropathy : the roles of cell-mediated immunity and CD4+ T-lymphocytes." Thesis, The University of Sydney, 2000. https://hdl.handle.net/2123/27827.

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2

Leung, Chi-kam Joseph, and 梁志錦. "The pathogenesis of IgA nephropathy: the roleof IgA molecule and the nature of IgA receptors." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29744908.

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3

Wootton, Andrew. "The glomerular basement membrane and nephritis /." Title page, contents and abstract only, 1985. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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4

Cavaglieri, Rita de Cássia. "Terapia com células tronco derivadas do líquido amniótico humano na nefropatia crônica experimental: é possível bloquear a progresso da doença renal estabelecida?" Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09052018-101720/.

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Abstract (sommario):

Células tronco mesenquimais (CTm) apresentam potencial para tratamento da doença renal pela possibilidade de promover regeneração tecidual e recuperação funcional, possivelmente por seus efeitos parácrinos. Na última década, o líquido amniótico foi descrito como uma fonte promissora de extração e isolamento de CTm. Alguns estudos mostraram o efeito renoprotetor das CTm derivadas do líquido amniótico (CTmLA) na doença renal aguda e crônica, quando inoculadas precocemente. Entretanto, ainda não foi estudado o efeito da administração de CTmLA em modelo experimental de doença renal crônica (DRC) c

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Brittain, Alison Louise. "Growth Hormone (GH) and the Glomerular Podocyte." Ohio University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1554208861914841.

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Sousa, Mauri Félix de. ""Efeitos renais da haploinsuficiência do gene Pkd1 (Polycystic kidney disease 1) em camundongos"." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-21122005-163447/.

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Abstract (sommario):

Vários estudos mostram que na doença renal policística autossômica dominante os cistos surgem a partir de um mecanismo de "dois-golpes". A patogênese das manifestações não-císticas, contudo, é pouco compreendida. Neste estudo usamos uma linhagem de camundongos endogâmica com uma mutação nula em Pkd1, onde animais heterozigotos apresentam formação cística renal mínima até 40 semanas de idade. O clearance de inulina e o número de glomérulos foram menores em machos Pkd1+/- que Pkd1+/+, enquanto o volume glomerular médio foi maior em heterozigotos. A excreção urinária de NO2/NO3 não diferiu signif

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7

Baboolal, Keshwar. "The renin angiotensin system in experimental renal disease." Thesis, King's College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336469.

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8

Randles, Michael. "Proteomic analyses of kidney glomerular extracellular matrix in health and disease." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/proteomic-analyses-of-kidney-glomerular-extracellular-matrix-in-health-and-disease(a39fe408-db06-4d80-b97b-4e0651bf7bc3).html.

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Abstract (sommario):

Glomerular filtration is a vital physiological process removing waste products from the circulation and this process occurs across the glomerular filtration barrier (GFB). The cells and extracellular matrix (ECM), which form this barrier, are exposed to forces during ultrafiltration and special adaptation is required to withstand these forces. Dysfunction in cellular adhesion machinery or ECM assembly within the GFB causes loss of selective glomerular filtration, however, the mechanisms governing these processes are poorly understood. To this end we sought to characterise the glomerular ECM an

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9

Sheerin, Neil Stephen. "Complement in the pathogenesis of immune mediated glomerular injury." Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.313287.

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Stitt, Erin Maureen. "The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease." Thesis, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/19800.

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The incidence of chronic kidney disease (CKD) is increasing. CKD is characterized by a gradual decrease in renal function leading to end stage renal disease (ESRD). Damage to the glomerular podocytes, is one of the first hallmarks of CKD. We hypothesized that podocyte prostaglandin E2 (PGE2) receptors contribute to the progression of glomerular injury in models of CKD. To test this hypothesis, transgenic mice were generated with either podocyte-specific overexpression or deletion of the PGE2 EP4 receptor (EP4pod+and EP4pod-/- respectively). Mice were next tested in the 5/6 nephrectomy (5/6 Nx)

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Più fonti

Libri sul tema "Kidney glomerulus Diseases"

1

Color atlas of kidney biopsy: Pathology of glomerular diseases. New York: Liss, 1985.

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2

Prabhakar, Sharma S. An update on glomerulopathies: Clinical and treatment aspects. Rijeka, Croatia: InTech, 2011.

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3

Prabhakar, Sharma S. An update on glomerulopathies: Etiology and pathogenesis. Rijeka, Croatia: InTech, 2011.

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4

Glomerulopathies: Cell biology and immunology. Australia: Harwood Academic Press, 1996.

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5

Valaitis, Jonas. Renal glomerular diseases: Atlas of electron microscopy with histopathological bases and immunofluorescence findings : presention of 110 cases of patients undergoing kidney biopsies. Chicago: ACSP Press, 2002.

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6

Jay, Bernstein, and Glassock Richard J, eds. Renal disease: Classification and atlas of glomerular diseases. 2nd ed. New York: Igaku-Shoin, 1995.

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7

Diagnostic electron microscopy: A text/atlas. New York: Igaku-Shoin, 1988.

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8

Membranes, International Symposium on Renal Basement. Progress in basement membrane research: Renal and related aspects in health and disease : proceedings. London: J. Libbey, 1988.

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9

1953-, Sakai T., and Kriz Wilhelm 1936-, eds. The vascular pole of the renal glomerulus of rat. Berlin: Springer, 1998.

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10

Marie-Claire, Gubler, and Sternberg Michel, eds. Progress in basement membrane research: Renal and related aspects in health and disease : proceedings of the IVth International Symposium on Renal Basement Membranes and Related Research held in Paris, 21-25 July 1987. London: Libbey, 1988.

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Capitoli di libri sul tema "Kidney glomerulus Diseases"

1

Gould, Edward R., and Anna Marie Burgner. "Glomerular Disease." In The Kidney, 175–97. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3286-3_12.

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2

Glassock, Richard J. "Other Glomerular Diseases." In Core Concepts in Parenchymal Kidney Disease, 285–89. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8166-9_19.

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3

Grünfeld, Jean-Pierre, Guillaume Bobrie, Jean-Michel Pochet, and Micheline Levy. "Inheritance of Glomerular Diseases." In Inheritance of Kidney and Urinary Tract Diseases, 67–87. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4613-1603-9_3.

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4

Jabor, Antonín, Janka Franeková, and Lenka Hošková. "Estimation of Glomerular Filtration Rate." In Biomarkers in Kidney Disease, 1143–73. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-007-7699-9_33.

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Jabor, Antonín, Janka Franeková, and Lenka Hošková. "Estimation of Glomerular Filtration Rate." In Biomarkers in Kidney Disease, 1–32. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-007-7743-9_33-1.

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6

Smyth, Andrew, and Vesna D. Garovic. "Glomerular Disease in Pregnancy." In Core Concepts in Parenchymal Kidney Disease, 315–28. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8166-9_22.

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7

Uppal, Nupur N., Divya Monga, and Hitesh H. Shah. "Glomerular Disease After Kidney Transplantation." In Glomerulonephritis, 787–808. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49379-4_48.

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Uppal, Nupur N., Divya Monga, and Hitesh H. Shah. "Glomerular Disease After Kidney Transplantation." In Glomerulonephritis, 1–22. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-27334-1_48-1.

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Rayner, Hugh C., Mark E. Thomas, and David V. Milford. "Measuring Kidney Function: How to Use Laboratory Tests to Measure Glomerular Filtration Rate." In Understanding Kidney Diseases, 11–28. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43027-6_2.

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O�Donnell, M. P., Z. A. Massy, C. Guijarro, B. L. Kasiske, Y. Kim, and W. F. Keane. "Isoprenoids, Ras and Proliferative Glomerular Disease." In Lipids and the Kidney, 219–27. Basel: KARGER, 1997. http://dx.doi.org/10.1159/000059840.

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Atti di convegni sul tema "Kidney glomerulus Diseases"

1

Slesareva, E. V., R. V. Ureneva, S. M. Slesarev, and O. V. Lyapeykova. "The pathomorphology of the renal cortex depending of arterial hypertension duration course." In VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-180-183.

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The kidneys autopsy material of persons with arterial hypertension in different duration was examined. Morphometry of the renal corpuscle area and cortical and juxtamedullary nephrons vascular glomeruli was performed. There were 4 groups - a control group (with a normal blood pressure level), and groups with arterial hypertension - the initial stage (group 2), arterial hypertension for 5-10 years (group 3), long-term arterial hypertension - more than 10 years (group 4). It was found that cortical nephrons are distinguished by earlier and more pronounced hyperplasia of the vascular glomerulus,

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2

Никитенко, О. П., Т. В. Стоева, М. В. Федин та А. И. Гоженко. "ЭТИОЛОГИЯ И ОСОБЕННОСТИ ТЕЧЕНИЯ ХБП У ДЕТЕЙ ПРИ ПАТОЛОГИИ МОЧЕВЫДЕЛИТЕЛЬНОЙ СИСТЕМЫ". У International Trends in Science and Technology. RS Global Sp. z O.O., 2021. http://dx.doi.org/10.31435/rsglobal_conf/28022021/7434.

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This article presents data on the development of (chronickidney disease) CKD in children.In particular, with the pathology of the urinary system in children of different age groups.We analyzed the main clinical characteristics and laboratory parameters in children with pathology of the urinary system.The main parameters of the functional state of the kidneys were considered, the glomerular filtration rate (GFR) was calculated using the CKD EPI formula.The chronic pathology of the urinary systemin children was also analyzed.The course of CKD in children with various clinical nosological forms o

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Hunt, Sarah, Yoav Segal, Kevin D. Dorfman, and Victor H. Barocas. "A Model of Glomerular Mesangial Transport in Health and Disease." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14712.

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Transport in the kidney is critically important in normal function and in disease. Solute transport occurs in many locations within the kidney, including convection within the capillaries, filtration across the basement membrane, and convection/diffusion within the mesangium (Figure 1). Models of transport in the kidney have traditionally focused on ultrafiltration [1], which is the predominant pathway for fluid and solutes passing into the urinary space. However, the mesangium is often the site of the first symptoms in disease [2], suggesting that mesangial transport is significant, at least

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4

Nieto-Chaupis, Huber. "Nephrine-Albumin Interaction and the Spontaneous Apparition of Series Capacitors in the Renal Glomerulus as Indicator of Kidney Disease." In 2021 IEEE/ACIS 22nd International Conference on Software Engineering, Artificial Intelligence, Networking and Parallel/Distributed Computing (SNPD). IEEE, 2021. http://dx.doi.org/10.1109/snpd51163.2021.9704915.

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Gasparotto, M., M. Gatto, RA Sinico, G. Moroni, L. Iaccarino, and A. Doria. "PO.5.98 Glomerular activity at second kidney biopsy predicts of end-stage kidney disease in a large multi-centric cohort of patients with active lupus nephritis." In 13th European Lupus Meeting, Stockholm (October 5–8, 2022). Lupus Foundation of America, 2022. http://dx.doi.org/10.1136/lupus-2022-elm2022.123.

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Xun, Liu, Wu Xiaoming, Li Ningshan, and Lou Tanqi. "Application of radial basis function neural network to estimate glomerular filtration rate in Chinese patients with chronic kidney disease." In 2010 International Conference on Computer Application and System Modeling (ICCASM 2010). IEEE, 2010. http://dx.doi.org/10.1109/iccasm.2010.5622616.

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Bilancieri, Giovanna Benichel, Gabriela Beck Dos Santos, Letícia Umetsu Yaginuma, Eliazar Da Silva Santos Júnior, and Eliane Cardozo Silva. "VANTAGENS E DESVANTAGENS NA UTILIZAÇÃO DO QUESTIONÁRIO SCORED PARA TRIAGEM DE DOENÇA RENAL CRÔNICA EM PACIENTES NA ATENÇÃO BÁSICA: UMA REVISÃO DE LITERATURA." In I Congresso Brasileiro de Saúde Pública On-line: Uma abordagem Multiprofissional. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2989.

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Abstract (sommario):

Introdução: A doença renal crônica (DRC) é um grave problema de saúde pública, com alta prevalência, de 3 a 6 milhões de brasileiros, e mortalidade, de 15,7 por 100 mil ao ano. O rastreamento precoce em pacientes com comorbidades que predispõem à DRC, é infrequente no sistema único de saúde, resultando no diagnóstico tardio e prejuízos ao paciente. Pretendendo rastrear a DRC em fase inicial, foi elaborado o questionário SCORED, do inglês Screening for Occult Renal Disease. Constituído por 11 perguntas referentes a dados demográficos e clínicos, com pontuação de 0 a 12, sendo considerado pacien

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Nowinski, Adam, Anna Czyzak-Gradkowska, Damian Korzybski, Luiza Jonczak, Przemyslaw Bielen, Robert Plywaczewski, and Pawel Sliwinski. "Obstructive sleep apnea and the risk of chronic kidney disease – glomerular filtration rate estimations based on serum cystatin C and creatinine concentrations." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2329.

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Hamed, Marwa Ibrahim, Salma Al-Dakhakhny, Hassaan Rathore, and Mohamed Izham Mohamed Ibrahim. "Reno-Protective Effects of Angiotensin Receptor Blockers in Hypertensive Rodent Models: A systematic review." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0126.

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Abstract (sommario):

Background and objective: Essential hypertension is a leading risk factor for chronic kidney disease, yet there is no conclusive evidence that lowering blood pressure alone improves renal outcome measures. Angiotensin-II receptor blockers (ARBs) proposed to have renal-protective effects independent of their antihypertensive effect. This systematic review of animal studies aims to collect available information from the published literature about the ARBs' consequences in murine models and analyze it in a structured way to provide a pre-clinical baseline for future analysis of similar clinical i

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Gyoneva, Lazarina, Mohammad F. Hadi, Yoav Segal, Kevin D. Dorfman, and Victor H. Barocas. "Role of Lateral Interactions in Type IV Collagen Network Mechanics." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14625.

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Abstract (sommario):

The basement membrane is a specialized part of the extra-cellular matrix. It is usually characterized as a scaffold for epithelial cells but in some tissues it serves other, mechanical, roles [1]. The mechanical properties of the basement membrane are mainly determined by one of its main constituents — type IV collagen. Unlike the well-known fibrous type I collagen, collagen IV assembles into planar networks (Fig. 1) [2]. The α 1(IV) and α 2(IV) collagen IV chains assemble into the so-called major chain network, present in all basement membranes. The α 3(IV), α 4(IV), α 5(IV) collagen IV chain

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