Accedi

Bibliografie tematiche / Kidney Pathophysiology / Libri

Segui questo link per vedere altri tipi di pubblicazioni sul tema: Kidney Pathophysiology.

Libri sul tema "Kidney Pathophysiology"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 1 febbraio 2022

Cita una fonte nei formati APA, MLA, Chicago, Harvard e in molti altri stili

Scegli il tipo di fonte:

Vedi i top-50 libri per l'attività di ricerca sul tema "Kidney Pathophysiology".

Accanto a ogni fonte nell'elenco di riferimenti c'è un pulsante "Aggiungi alla bibliografia". Premilo e genereremo automaticamente la citazione bibliografica dell'opera scelta nello stile citazionale di cui hai bisogno: APA, MLA, Harvard, Chicago, Vancouver ecc.

Puoi anche scaricare il testo completo della pubblicazione scientifica nel formato .pdf e leggere online l'abstract (il sommario) dell'opera se è presente nei metadati.

Vedi i libri di molte aree scientifiche e compila una bibliografia corretta.

1

Pathophysiology of renal disease. 2^a ed. New York: McGraw-Hill, 1987.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

2

Leaf, Alexander. Renal pathophysiology. 3^a ed. New York: Oxford University Press, 1985.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

3

M, Denker Bradley, e Rose Burton David 1942-, a cura di. Renal pathophysiology: The essentials. 2^a ed. Philadelphia: Lippincott Williams & Wilkins, 2007.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

4

M, Denker Bradley, a cura di. Renal pathophysiology: The essentials. 3^a ed. Baltimore, MD: Lippincott Williams & Wilkins, 2010.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

5

Fuminori, Sakai, Berliner Robert W. 1915-, Honda Nishio e Ullrich K. J, a cura di. The frontiers of nephrology: Proceedings of the International Forum "The Frontiers of Nephrology", honoring Fuminori Sakai, held in Tokyo, Japan, 24-25 August 1989. Amsterdam: Excerpta Medica, 1990.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

6

G, Rennke Helmut, a cura di. Renal pathophysiology: The essentials. Baltimore: Williams & Wilkins, 1994.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

7

Dixhoorn, Mieneke G. A. van. IgA in experimental kidney disease. [Leiden: University of Leiden, 1998.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

8

Höper, J. Influence of local oxygen deficiency on function and integrity of liver, kidney, and heart. Stuttgart: G. Fischer, 1991.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

9

Eleftheriadis, Theodoros. Vitamin D receptor agonists and kidney diseases. Hauppauge, N.Y: Nova Science Publishers, 2010.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

10

Hikaru, Koide, e Hayashi T, a cura di. Extracellular matrix in the kidney: 6th International Symposium on Basement Membrane, Shizuoka, May 29-June 1, 1993. Basel: Karger, 1994.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

11

Kai-Uwe, Eckardt, Nangaku Masaomi e SpringerLink (Online service), a cura di. Studies on Renal Disorders. Totowa, NJ: Springer Science+Business Media, LLC, 2011.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

12

Brewer, Jonathan Douglas. The coronary cure: A modified Mediterranean diet for the prevention of atherosclerosis, hypertension, obesity, diabetes, arthritis, and kidney stones : molecular mechanisms and practical guidelines : nutrition 101. [United States?]: J.D. Brewer, 2004.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

13

A, Stanton Bruce, a cura di. Renal physiology. 3^a ed. St. Louis: Mosby, 2001.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

14

D, Bianchi Claudio M., a cura di. Kidney, proteins, and drugs: An update : 7th International Symposium of Nephrology at Montecatini, Montecatini Terme, October 14-16, 1991. Basel: Karger, 1993.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

15

Gary, Abuelo J., a cura di. Renal pathophysiology: The essentials. Baltimore: Williams & Wilkins, 1989.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

16

Pathophysiology of Renal Disease. 2^a ed. McGraw-Hill Health Professions Division, 1990.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

17

1920-, Seldin Donald W., e Giebisch Gerhard H, a cura di. The Kidney: Physiology and pathophysiology. New York: Raven Press, 1985.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

18

1920-, Seldin Donald W., e Giebisch Gerhard H, a cura di. The Kidney: Physiology and pathophysiology. 2^a ed. New York: Raven Press, 1992.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

19

A, Shayman James, a cura di. Renal pathophysiology. Philadelphia: J.B. Lippincott, 1995.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

20

Seldin, Donald W. The Kidney: Physiology and Pathophysiology. 2^a ed. Raven Pr, 1992.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

21

W, Seldin Donald, e Giebisch Gerhard H, a cura di. The Kidney: Physiology and pathophysiology. 2^a ed. New York: Raven Press, 1991.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

22

W, Seldin Donald, e Giebisch Gerhard H, a cura di. The kidney: Physiology and pathophysiology. New York: Raven Press, 1985.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

23

Goligorsky, Michael S., Julien Maizel, Radovan Vasko, May M. Rabadi e Brian B. Ratliff. Pathophysiology of acute kidney injury. A cura di Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0221.

Testo completo

Abstract (sommario):

In the intricate maze of proposed mechanisms, modifiers, modulators, and sensitizers for acute kidney injury (AKI) and diverse causes inducing it, this chapter focuses on several common and undisputable strands which do exist.Structurally, the loss of the brush border, desquamation of tubular epithelial cells, and obstruction of the tubular lumen are commonly observed, albeit to various degrees. These morphologic hallmarks of AKI are accompanied by functional defects, most consistently reflected in the decreased glomerular filtration rate and variable degree of reduction in renal blood flow, accompanied by changes in the microcirculation. Although all renal resident cells participate in AKI, the brunt falls on the epithelial and endothelial cells, the fact that underlies the development of tubular epithelial and vascular compromise.This chapter further summarizes the involvement of several cell organelles in AKI: mitochondrial involvement in perturbed energy metabolism, lysosomal involvement in degradation of misfolded proteins and damaged organelles, and peroxisomal involvement in the regulation of oxidative stress and metabolism, all of which become defective. Common molecular pathways are engaged in cellular stress response and their roles in cell death or survival. The diverse families of nephrotoxic medications and the respective mechanisms they induce AKI are discussed. The mechanisms of action of some nephrotoxins are analysed, and also of the preventive therapies of ischaemic or pharmacologic pre-conditioning.An emerging concept of the systemic inflammatory response triggered by AKI, which can potentially aggravate the local injury or tend to facilitate the repair of the kidney, is presented. Rational therapeutic strategies should be based on these well-established pathophysiological hallmarks of AKI.

Gli stili APA, Harvard, Vancouver, ISO e altri

24

S, Cotran Ramzi, a cura di. Renal pathophysiology. 3^a ed. Oxford University Press, 1985.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

25

S, Goligorsky Michael, e Gross Steven S. 1951-, a cura di. Nitric oxide and the kidney: Physiology and pathophysiology. New York: Chapman & Hall, 1997.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

26

Goligorsky, Michael S., e Steven S. Gross. Nitric Oxide and the Kidney: Physiology and Pathophysiology. Springer, 2012.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

27

Goligorsky, Michael S., e Steven S. Gross. Nitric Oxide And The Kidney: Physiology and Pathophysiology. Springer, 1997.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

28

Vishnu, Moorthy A., a cura di. Pathophysiology of kidney disease and hypertension. Philadelphia, PA: Saunders/Elsevier, 2009.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

29

Pathophysiology of Kidney Disease and Hypertension. Elsevier, 2009. http://dx.doi.org/10.1016/c2009-0-36246-7.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

30

Merton, Berlyne Geoffrey, a cura di. The Kidney today: Selected topics in renal science. Basel: Karger, 1992.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

31

Charles, Jennette J., e Heptinstall Robert H. 1920-, a cura di. Heptinstall's pathology of the kidney. 5^a ed. Philadelphia: Lippincott-Raven, 1998.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

32

Handbook of Renal Biopsy Pathology. Springer, 2001.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

33

The Frontiers of Nephrology (International congress series). Elsevier, 1990.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

34

Howie, A. J. Handbook of Renal Biopsy Pathology. Springer, 2001.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

35

Leaf, Alexander, e Leaf. Renal Pathophysiology 2/E. Oxford University Press, 1992.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

36

Leaf, Alexander, e Leaf. Renal Pathophysiology 2/E. Oxford University Press, 1992.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

37

J, Alpern Robert, Herbert Steven C, Seldin Donald W. 1920- e Giebisch Gerhard H, a cura di. Seldin and Giebisch's The kidney: Physiology & pathophysiology. 4^a ed. Amsterdam: Academic, 2008.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

38

J, Alpern Robert, Hebert Steven C, Seldin Donald W. 1920- e Giebisch Gerhard H, a cura di. Seldin and Giebisch's the kidney: Physiology & pathophysiology. 4^a ed. Amsterdam: Elsevier Inc., Academic Press, 2007.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

39

Bellomo, Rinaldo, e John R. Prowle. Pathophysiology of oliguria and acute kidney injury. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0211.

Testo completo

Abstract (sommario):

Oliguria and acute kidney injury (AKI) are common in critically-ill patients with studies reporting AKI affecting more than 50% of critically-ill patients. AKI is independently associated with increased mortality and is a potentially modifiable aspect of critical illness. The pathogenesis of AKI is complex and varies according to aetiology. The most common trigger in ICU patients is sepsis—the pathophysiology of septic AKI is poorly understood and probably involves intrarenal haemodynamic and inflammatory processes. In the setting of septic AKI, the classic acute tubular necrosis described in experimental models does not occur and histological changes are only minor. Activation of neurohormonal mechanisms is also important, particularly in the hepatorenal syndrome, where activation of the remain-angiotensin system appears to play a major role. The treatment of oliguria and AKI in ICU patients has traditionally relied on the administration of intravenous fluids. While such therapy is warranted in patients with a clear history, and physical examination suggestive of intravascular and extravascular volume depletion, its usefulness in other patients (e.g. septic patients) remains controversial. Removal of nephrotoxins, rapid treatment of the triggering factors, and attention to cardiac output and mean arterial pressure remain the cornerstones of the prevention and treatment of AKI in ICU.

Gli stili APA, Harvard, Vancouver, ISO e altri

40

Rennke, Helmut G., e Bradley M. Denker. The Renal Pathophysiology: The Essentials. 2^a ed. Lippincott Williams & Wilkins, 2006.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

41

Heptinstall's Pathology of the Kidney. LWW, 2014.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

42

Obesity And the Kidney. S. Karger AG (Switzerland), 2006.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

43

Heptinstall's Pathology of the Kidney 2 Volume Set. 5^a ed. Lippincott Williams & Wilkins, 1998.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

44

The Kidney: Physiology and Pathophysiology (2-Volume Set). 3^a ed. Lippincott Williams & Wilkins, 2000.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

45

Seldin And Giebischs The Kidney Physiology And Pathophysiology. Academic Press, 2012.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

46

Rath, Thomas, a cura di. Chronic Kidney Disease - from Pathophysiology to Clinical Improvements. InTech, 2018. http://dx.doi.org/10.5772/66239.

Testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

47

Tsai, Ching-Wei, Sanjeev Noel e Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

Testo completo

Abstract (sommario):

Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of macrophages, dendritic cells, natural killer cells, and T and B lymphocytes. During the recovery phase, cell adhesion molecules as well as cytokines and chemokines may be instrumental by directing the migration, differentiation, and proliferation of renal epithelial cells; recent data also suggest a critical role of M2 macrophage and regulatory T cell in the recovery period. Other processes contributing to renal regeneration include renal stem cells and the expression of growth hormones and trophic factors. Subtle deviations in the normal repair process can lead to maladaptive fibrotic kidney disease. Further elucidation of these mechanisms will help discover new therapeutic interventions aimed at limiting the extent of AKI and halting its progression to CKD or ESRD.

Gli stili APA, Harvard, Vancouver, ISO e altri

48

C, Gonick Harvey, e Buckalew Vardaman M, a cura di. Renal tubular disorders: Pathophysiology, diagnosis, and management. New York: Dekker, 1985.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

49

F, Keane William, Hörl Walter H e Kasiske B. L, a cura di. Lipids and the kidney. Basel: Karger, 1997.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

50

David, Humes H., a cura di. Pathophysiology of electrolyte and renal disorders. New York: Churchill Livingstone, 1986.

Cerca il testo completo

Gli stili APA, Harvard, Vancouver, ISO e altri

Offriamo sconti su tutti i piani premium per gli autori le cui opere sono incluse in raccolte letterarie tematiche. Contattaci per ottenere un codice promozionale unico!