Bibliografie tematiche / Leptin receptors

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Letteratura scientifica selezionata sul tema "Leptin receptors"

Autore: Grafiati
Pubblicato: 4 giugno 2021
Ultima modifica: 31 gennaio 2023

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Articoli di riviste sul tema "Leptin receptors"

1

Panić, Anastasija, Sanja Soskić, and Esma Isenović. "Leptin and its mechanism of action." Medicinska istrazivanja 49, no. 3 (2015): 36–41. http://dx.doi.org/10.5937/medist1502036p.

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Abstract (sommario):
Leptin is a hormone produced by the adipose tissue, which has effects on the central nervous system. Leptin is bound to its Ob receptor on hypo-thalamic neurons to inhibit feeding behavior and to increase sympathetically-mediated thermogenesis. In addition to anorexia and thermogenesis, leptin also has direct peripheral and central nervous system-mediated effects on the endocrine, vascular, hematopoietc, immune and musculoskeletal systems. Leptin accomplishes its effects using distributed network of leptin receptors and differential molecular signaling pathways. Leptinemia is increased in obes
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2

Gertler, A., Y. Sandowski, and N. Raver. "Recombinant leptin mutants and leptin binding proteins aimed to block leptin action in vivo." Proceedings of the British Society of Animal Science 2002 (2002): 48. http://dx.doi.org/10.1017/s1752756200007043.

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Abstract (sommario):
Obese protein (OB) also known as leptin serves as a protein signal secreted from adipose tissue and acts on a central nervous system that regulate ingestive behavior and energy balance (Campfield, 2000). The sequence of various leptins from 10 mammalian species was compiled and the 3D structure of human leptin mutant W100E was elucidated (Zhang et al., 1997). We have prepared recombinant leptins of sheep, chicken, cow and pig. Mammalian and chicken leptins are respectively 146 and 145 amino acid containing proteins found in circulation. Leptin in blood is found in both free and bound form; the
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Borodkina, Daria A., Olga V. Gruzdeva, Olga E. Akbasheva, Ekaterina V. Belik, Elena I. Palicheva, and Olga L. Barbarash. "Leptin resistance: unsolved diagnostic issues." Problems of Endocrinology 64, no. 1 (April 9, 2018): 62–66. http://dx.doi.org/10.14341/probl201864162-66.

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Abstract (sommario):
Leptin and its receptors are key regulators of body weight and energy homeostasis. A decrease in tissue sensitivity to leptin leads to the development of obesity, insulin resistance, dyslipidemia, etc. Currently, the phenomenon of leptin resistance is explained by a number of mechanisms, including impairment of gene structure, leptin transport through the blood-brain barrier, and leptin receptor signaling. However, it is not known, a decrease in the number of receptors of which area leads to the development of leptin resistance. No relationship has been found between the basal leptin level in
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Borodkina, Daria A., Olga V. Gruzdeva, Olga E. Akbasheva, Ekaterina V. Belik, Elena I. Palicheva, and Olga L. Barbarash. "Leptin resistance: unsolved diagnostic issues." Problems of Endocrinology 64, no. 1 (April 9, 2018): 62–66. http://dx.doi.org/10.14341/probl8740.

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Abstract (sommario):
Leptin and its receptors are key regulators of body weight and energy homeostasis. A decrease in tissue sensitivity to leptin leads to the development of obesity, insulin resistance, dyslipidemia, etc. Currently, the phenomenon of leptin resistance is explained by a number of mechanisms, including impairment of gene structure, leptin transport through the blood-brain barrier, and leptin receptor signaling. However, it is not known, a decrease in the number of receptors of which area leads to the development of leptin resistance. No relationship has been found between the basal leptin level in
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5

Skibicka, Karolina P., and Harvey J. Grill. "Hindbrain Leptin Stimulation Induces Anorexia and Hyperthermia Mediated by Hindbrain Melanocortin Receptors." Endocrinology 150, no. 4 (September 10, 2008): 1705–11. http://dx.doi.org/10.1210/en.2008-1316.

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Abstract (sommario):
Of the central nervous system receptors that could mediate the energy balance effects of leptin, those of the hypothalamic arcuate nucleus receive the greatest attention. Melanocortin receptors (MC-Rs) contribute to the feeding and energetic effects of hypothalamically delivered leptin. Energy balance effects of leptin are also mediated by extrahypothalamic neurons including the hindbrain nucleus tractus solitarius. Hindbrain leptin receptors play a role in leptin’s anorectic effects, but their contribution to its energetic effects and their functional interaction with melanocortin systems wit
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Pan, Weihong, Hung Hsuchou, Hong Tu, and Abba J. Kastin. "Developmental Changes of Leptin Receptors in Cerebral Microvessels: Unexpected Relation to Leptin Transport." Endocrinology 149, no. 3 (November 26, 2007): 877–85. http://dx.doi.org/10.1210/en.2007-0893.

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Abstract (sommario):
The adipokine leptin participates not only in the regulation of feeding and obesity in adults but also in neonatal development. It crosses the blood-brain barrier (BBB) by receptor-mediated transport. Leptin concentrations in blood differ between neonates and adults. We determined the developmental changes of leptin receptor subtypes in the cerebral microvessels composing the BBB and examined their expected correlation with leptin transport across the BBB. Total RNA was extracted from enriched cerebral microvessels of mice 1, 7, 14, and 60 d of age for real-time RT-PCR analysis of leptin recep
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Harris, Ruth B. S. "Leptin-induced increase in body fat content of rats." American Journal of Physiology-Endocrinology and Metabolism 304, no. 3 (February 1, 2013): E267—E281. http://dx.doi.org/10.1152/ajpendo.00251.2012.

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Abstract (sommario):
We previously reported that peripheral leptin infusions in chronically decrebrate rats, in which the forebrain is neurally isolated from the hindbrain, increased body fat and decreased energy expenditure. Any central leptin response in decerebrate rats would depend upon the hindbrain. Here, we tested whether selective activation of hindbrain leptin receptors increased body fat. Fourth ventricle infusion of 0.6 μg leptin/day for 12 days increased body fat by 13% with no increase in food intake. Third ventricle leptin infusions decreased food intake, body fat, and lean tissue with a maximal resp
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BENOMAR, Yacir, Anne-France ROY, Alain AUBOURG, Jean DJIANE, and Mohammed TAOUIS. "Cross down-regulation of leptin and insulin receptor expression and signalling in a human neuronal cell line." Biochemical Journal 388, no. 3 (June 7, 2005): 929–39. http://dx.doi.org/10.1042/bj20041621.

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Abstract (sommario):
Leptin and insulin are major signals to the hypothalamus to regulate energy homoeostasis and body adiposity. IR (insulin receptors) and leptin receptors (long isoform, ObRb) share a number of signalling cascades, such as JAK2/STAT-3 (Janus kinase 2/signal transduction and activator of transcription 3) and PI3K (phosphoinositide 3-kinase); the cross-talk between IR and ObRb have been described previously in non-neuronal cells. Differentiated human neuroblastoma (SH-SY5Y) cells express endogenous ObR and IR, and respond to leptin and insulin with stimulation of STAT-3 and MAPK (mitogen-activated
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Boroń, Dariusz, Robert Nowakowski, Beniamin Oskar Grabarek, Nikola Zmarzły, and Marcin Opławski. "Expression Pattern of Leptin and Its Receptors in Endometrioid Endometrial Cancer." Journal of Clinical Medicine 10, no. 13 (June 24, 2021): 2787. http://dx.doi.org/10.3390/jcm10132787.

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Abstract (sommario):
The identification of novel molecular markers and the development of cancer treatment strategies are very important as cancer incidence is still very high. Obesity can contribute to cancer progression, including endometrial cancer. Adipocytes secrete leptin, which, when at a high level, is associated with an increased risk of cancer. The aim of this study was to determine the expression profile of leptin-related genes in the endometrial tissue samples and whole blood of patients. The study material included tissue samples and whole blood collected from 30 patients with endometrial cancer and 3
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Wada, Nobuhiro, Satoshi Hirako, Fumiko Takenoya, Haruaki Kageyama, Mai Okabe, and Seiji Shioda. "Leptin and its receptors." Journal of Chemical Neuroanatomy 61-62 (November 2014): 191–99. http://dx.doi.org/10.1016/j.jchemneu.2014.09.002.

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Tesi sul tema "Leptin receptors"

1

Yang, Seung Hak. "Gene expression of leptin and leptin receptors in beef cattle." Kyoto University, 2007. http://hdl.handle.net/2433/136511.

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Abstract (sommario):
Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第13091号<br>農博第1596号<br>新制||農||938(附属図書館)<br>学位論文||H19||N4217(農学部図書室)<br>UT51-2007-H364<br>京都大学大学院農学研究科応用生物科学専攻<br>(主査)教授 矢野 秀雄, 教授 今井 裕, 教授 久米 新一<br>学位規則第4条第1項該当
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Duggal, Priya S. "Leptin action on ovulation and leptin receptors across the rat oestrous cycle /." Title page, contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phd866.pdf.

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Chikani, Gentle P. "LEPTIN RECEPTORS IN CAVEOLAE: REGULATION OF LIPOLYSIS IN 3T3-L1 ADIPOCYTES." UKnowledge, 2004. http://uknowledge.uky.edu/gradschool_theses/382.

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Abstract (sommario):
The present study has tested the hypothesis that leptin receptors are localized in caveolae and that caveolae are involved in the leptin-induced stimulation of lipolysis in 3T3-L1 adipocytes. Leptin, a peptide hormone, is secreted primarily by adipocytes and has been postulated to regulate food intake and energy expenditure via hypothalamic-mediated effects. Exposure to leptin increases the lipolytic activity in 3T3-L1 adipocytes. We isolated caveolae from 3T3-L1 adipocytes using a detergent free sucrose gradient centrifugation method. Leptin receptors were localized in the same gradient fract
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De, Silva Andrea, and mikewood@deakin edu au. "The role of leptin receptors in the development of obesity." Deakin University. School of Health Sciences, 1999. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051125.111246.

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Abstract (sommario):
The focus of this dissertation was leptin and the leptin receptor, and the role of these genes (OB and OB-R) in the development of obesity and type 2 diabetes in humans and Psammomys obesus, a polygenic rodent model of obesity and type 2 diabetes. Studies in humans showed that circulating leptin concentrations were positively associated with adiposity, and independently associated with circulating insulin and triglyceride concentrations. Analysis of two leptin receptor sequence polymorphisms in a Caucasian Australian population and a population of Nauruan males, with very high prevalence rates
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Xu, Jialin, and 徐嘉林. "B lymphocyte development and function in leptin receptor-deficient mice." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B45011096.

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Chikani, Gentle. "Leptin receptors in caveolae regulation of lipolysis in 3T3-L1 adipocytes /." Lexington, Ky. : [University of Kentucky Libraries], 2004. http://lib.uky.edu/ETD/ukynusc2004t00203/thesis.pdf.

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Abstract (sommario):
Thesis (m.s.)--University of Kentucky, 2004.<br>Title from document title page (viewed Jan. 7, 2005). Document formatted into pages; contains 70 p. : ill. Includes abstract and vita. Includes bibliographical references (p. 58-68).
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Mak, Amy, and 麥安美. "Expression and regulation of leptin receptor in human and mouse oviduct." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B45010869.

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Soedling, Helen. "The role of leptin receptors in the endocrine pancreas and nucleus tractus solitarius." Thesis, Imperial College London, 2016. http://hdl.handle.net/10044/1/63933.

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Abstract (sommario):
The highly controlled regulation of pancreatic hormone secretion is vital to keep the body’s glucose concentration at a constant level. Defects in the regulation of glucose levels are involved in several metabolic diseases, including type 2 diabetes and obesity. Leptin is a satiety hormone with important roles in the maintenance of body weight and glucose homeostasis. Mice that lack leptin (ob/ob) or the leptin receptor (db/db) are massively obese and have diabetes symptoms. Leptin has been demonstrated to have an effect on glucose homeostasis that is suggested to be secondary to the obesity t
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9

Håkansson, Ovesjö Marie-Louise. "Leptin targets in the brain regulating body weight : receptors and down-stream mediators of leptin in neurons of the hypothalamus and brainstem /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4178-5/.

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Vaughan, Tamisha Y. "Novel Mechanisms Underlying the Inflammatory Effects of Leptin and Low Dose Endotoxin." Diss., Virginia Tech, 2010. http://hdl.handle.net/10919/28013.

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Abstract (sommario):
Obesity over the last several has become a major health concern in our country as well as the world. Obesity is also one of the risk factors which lead to several inflammatory complications such as diabetes, artherosclerosis, etc. Two leading factors involved in the causes of inflammatory complications include leptin and low dose endotoxin lipopolysaccharide (LPS). However, the mechanism underlying the involvement of these two mediators is not clearly understood. The purpose of this study is to understand the mechanism underlying inflammatory complications caused by leptin and low dose endo
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Libri sul tema "Leptin receptors"

1

Leptin and leptin antagonists. Austin, Tex: Landes Bioscience, 2009.

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2

Oliveira, Maria Nubia Gama. Marcadores de obesidade na avaliação do estado nutricional em adolescente. Edited by Marcia Alessandra Arantes Marques. Bookerfield Editora, 2021. http://dx.doi.org/10.53268/bkf21110300.

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Abstract (sommario):
Este livro é resultado da pesquisa de doutorado em um Centro de Referência de Adolescente. O que se destaca a recomendação da leitura do livro marcadores de obesidade na avalição do estado nutricional em adolescente é o método científico, trazendo à tona, à proposta, à pesquisa científica, à faixa etária de 10 a 19 anos. Estudar e atender adolescente são desafiadores, dado que, neste segmento observa-se uma fase de mudanças biológicas importantes, o consumo de refeições nem sempre satisfatória em nutrientes como vitamina A, E e β─caroteno. Somando-se a tudo isto, o excesso de calorias corrobor
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Capitoli di libri sul tema "Leptin receptors"

1

Cottrell, Elizabeth C., and Julian G. Mercer. "Leptin Receptors." In Handbook of Experimental Pharmacology, 3–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-24716-3_1.

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Dam, Julie, Ralf Jockers, Michèle Guerre-Millo, and Karine Clément. "Leptin Receptors and Mechanism of Action." In Leptin, 15–24. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09915-6_2.

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Considine, Robert V. "Measurement of Circulating Leptin and Soluble Leptin Receptors." In Leptin, 39–43. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09915-6_4.

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Gouilleux, Fabrice. "Erythropoietin, Thrombopoietin and Leptin Receptors." In Hormone Signaling, 145–78. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4757-3600-7_8.

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Lubrano, Cécile, Béatrice Dubern, and Karine Clément. "Obesity-related mutations of leptin and melanocortin receptors." In Insights into Receptor Function and New Drug Development Targets, 35–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/3-540-34447-0_3.

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Zabeau, Lennart, Frank Peelman, and Jan Tavernier. "Leptin and Leptin Receptor." In Encyclopedia of Signaling Molecules, 2839–45. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101679.

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Zabeau, Lennart, Frank Peelman, and Jan Tavernier. "Leptin and Leptin Receptor." In Encyclopedia of Signaling Molecules, 1–6. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101679-1.

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Gorska, E., K. Popko, and M. Wasik. "Leptin Receptor in Childhood Acute Leukemias." In Advances in Experimental Medicine and Biology, 155–61. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-4549-0_20.

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Castracane, V. Daniel, and Michael C. Henson. "Regulation of Leptin and Leptin Receptor in the Human Uterus: Possible Roles in Implantation and Uterine Pathology." In Leptin and Reproduction, 111–15. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0157-2_7.

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Otvos, Laszlo, Marco Cassone, Marianna Terrasi, Sandra Cascio, George D. Mateo, Daniel Knappe, Ralf Hoffmann, Predrag Cudic, John D. Wade, and Eva Surmacz. "Agonists and Partial Antagonists Acting on the Leptin—Leptin Receptor Interface." In Advances in Experimental Medicine and Biology, 497–98. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-73657-0_215.

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Atti di convegni sul tema "Leptin receptors"

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Ahmed, Sumaya, and Nasser Rizk. "The Expression of Bile Acid Receptor TGR5 in Adipose Tissue in Diet-Induced Obese Mice." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0212.

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Abstract (sommario):
Bile acids are significant physiological factors for digestion, solubilization, absorption, toxic metabolites and xenobiotics. In addition, bile acids are responsible of signal transduction as well as metabolic regulation that activate several receptors such as farnesoid X receptor (FXR) and the membrane G-protein receptor 5 (TGR5). Activation of TGR5 by bile acids is associated with prevention of obesity as well as ameliorating the resistance to insulin via increasing energy expenditure. The objective of this research is to investigate TGR5 gene expression level in different fat depots includ
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Zheng, Qiao, Sarah Smith, Jinling Zhu, Erin Downs-Kelly, Stephen D. Hursting, Jeremy N. Rich, Nathan A. Berger, and Ofer Reizes. "Abstract 3327: Leptin receptors are expressed in breast cancer stem cells and leptin promotes their self-renewal and survival in mice." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3327.

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Yom, Cha Kyong, Jun Ho Kim, Hee Sung Kim, and Yong Lai Park. "Abstract B50: Leptin and leptin receptor expression in breast cancer." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 15-19, 2009; Boston, MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/1535-7163.targ-09-b50.

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Bruno, Andreina, Antonella Ballacchino, Liboria Siena, Domenica Russo, Salvatore Gallina, Francesco Lorusso, Giuseppina Chiappara, Mark Gjomarkaj, and Elisabetta Pace. "Impairment of leptin/leptin receptor pathway in nasal epithelium from allergic turbinates." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa905.

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Wazir, U., Sarakbi W. Al, WG Jiang, and K. Mokbel. "Abstract P6-05-11: Leptin and Leptin receptor expression in human breast cancer." In Abstracts: Thirty-Fifth Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 4‐8, 2012; San Antonio, TX. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/0008-5472.sabcs12-p6-05-11.

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Karampela, Irene, Evangelia Kandri, George Skyllas, Evangelia Chrysanthopoulou, Gerasimos-Socrates Christodoulatos, Georgios Antonakos, Evangelos Vogiatzakis, Apostolos Armaganidis, and Maria Dalamaga. "Serum kinetics of total leptin and soluble leptin receptor as prognostic tools in sepsis." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2260.

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Ahasic, Amy, Yang Zhao, Li Su, B. T. Thompson, and David Christiani. "Leptin Receptor Gene (LEPR) Polymorphism And ARDS Susceptibility." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2099.

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Yabushita, Hiromitsu, Keita Iwasaki, Taiki Ueno, and Akihiko Wakatsuki. "Abstract 4738: Clinicopathological roles of adiponectin receptor and leptin receptor in endometrial carcinoma." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4738.

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Llanos, Adana A. M., Baichen Xu, Bo Qin, Wenjin Chen, Marina A. Chekmareva, Lei Cong, Chi-Chen Hong, et al. "Abstract C063: Associations of leptin and leptin receptor protein and gene expression with breast cancer clinicopathologic features." In Abstracts: Eleventh AACR Conference on The Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 2-5, 2018; New Orleans, LA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7755.disp18-c063.

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Gu, Fangyi, Peter Kraft, Megan Rice, and Karin B. Michels. "Abstract A104: Leptin and leptin receptor genes in relation to premenopausal breast cancer incidence and grade in Caucasian women." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a104.

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Rapporti di organizzazioni sul tema "Leptin receptors"

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Boisclair, Yves R., and Arieh Gertler. Development and Use of Leptin Receptor Antagonists to Increase Appetite and Adaptive Metabolism in Ruminants. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7697120.bard.

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Objectives The original project had 2 major objectives: (1) To determine the effects of centrally administered leptin antagonist on appetite and adaptive metabolism in the sheep; (2) To develop and prepare second-generation leptin antagonists combining high binding affinity and prolonged in vivo half-life. Background Periods of suboptimal nutrition or exaggerated metabolic activity demands lead to a state of chronic energy insufficiency. Ruminants remain productive for a surprisingly long period of time under these circumstances by evoking adaptations sparing available energy and nutrients. Th
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Yuan, Qihang. Circulating Leptin Level, Soluble Leptin Receptor Level and Their Gene Polymorphism in Patients with Systemic Lupus Erythematosus: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0137.

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Nahta, Rita. Crosstalk between Leptin Receptor and Igf-Ir in Breast Cancer: A Potential Mediator of Chemoresistance. Fort Belvoir, VA: Defense Technical Information Center, April 2008. http://dx.doi.org/10.21236/ada486433.

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Nahta, Rita. Crosstalk Between Leptin Receptor and IGF-IR in Breast Cancer: A Potential Mediator of Chemoresistance. Fort Belvoir, VA: Defense Technical Information Center, April 2009. http://dx.doi.org/10.21236/ada505199.

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Nahta, Rita. Crosstalk Between Leptin Receptor and IGF-IR in Breast Cancer: A Potential Mediator of Chemoresistance. Fort Belvoir, VA: Defense Technical Information Center, April 2010. http://dx.doi.org/10.21236/ada525623.

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