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Bibliografie tematiche / Lethal irradiation

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Letteratura scientifica selezionata sul tema "Lethal irradiation"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 1 febbraio 2022

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Articoli di riviste sul tema "Lethal irradiation"

1

Huang, Zhentai, Michael Epperly, Simon C. Watkins, Joel S. Greenberger, Valerian E. Kagan, and Hülya Bayır. "Necrostatin-1 rescues mice from lethal irradiation." Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease 1862, no. 4 (2016): 850–56. http://dx.doi.org/10.1016/j.bbadis.2016.01.014.

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2

Talmadge, JE, H. Tribble, R. Pennington, et al. "Protective, restorative, and therapeutic properties of recombinant colony-stimulating factors." Blood 73, no. 8 (1989): 2093–103. http://dx.doi.org/10.1182/blood.v73.8.2093.2093.

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Abstract Pretreatment of mice with recombinant murine (rM) colony-stimulating factor-granulocyte-macrophage (CSF-gm) or recombinant human (rH) CSF-g provides partial protection from the lethal effects of ionizing radiation or the alkylating agent cyclophosphamide (CTX). In addition, these agents can significantly prolong survival if administered following lethal doses of irradiation or CTX. To induce protective activity, cytokines were injected 20 hours before lethal irradiation or CTX administration. To accelerate recovery from lethal irradiation, the cytokines must be administered shortly fo

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3

Talmadge, JE, H. Tribble, R. Pennington, et al. "Protective, restorative, and therapeutic properties of recombinant colony-stimulating factors." Blood 73, no. 8 (1989): 2093–103. http://dx.doi.org/10.1182/blood.v73.8.2093.bloodjournal7382093.

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Abstract (sommario):

Pretreatment of mice with recombinant murine (rM) colony-stimulating factor-granulocyte-macrophage (CSF-gm) or recombinant human (rH) CSF-g provides partial protection from the lethal effects of ionizing radiation or the alkylating agent cyclophosphamide (CTX). In addition, these agents can significantly prolong survival if administered following lethal doses of irradiation or CTX. To induce protective activity, cytokines were injected 20 hours before lethal irradiation or CTX administration. To accelerate recovery from lethal irradiation, the cytokines must be administered shortly following i

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4

Chen, Benny J., Divino Deoliveira, and Nelson Chao. "Insulin-Like Growth Factor 1 Protects against Lethal Irradiation." Blood 112, no. 11 (2008): 3488. http://dx.doi.org/10.1182/blood.v112.11.3488.3488.

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Abstract Ionizing irradiation can cause bone marrow failure leading to death. Effective therapeutic agents capable of promoting or accelerating the recovery of the hematopoietic and/or immune compartment following radiation injury are limited. We and others have previously demonstrated that recombinant human growth hormone promotes hematopoietic and immune recovery following stem cell transplantation and irradiation. Published data suggest that growth hormone elicits its pro-hematopoietic effects via action of insulin-like growth factor 1 (IGF1). Since IGF1 has recently been approved by the Fe

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SALIN, C. "Protection of mouse jejunum against lethal irradiation by." Phytomedicine 8, no. 6 (2001): 413–22. http://dx.doi.org/10.1078/s0944-7113(04)70059-8.

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6

Tiberghien, P., V. Laithier, M. Mabed, et al. "Interleukin-1 administration before lethal irradiation and allogeneic bone marrow transplantation: early transient increase of peripheral granulocytes and successful engraftment with accelerated leukocyte, erythrocyte, and platelet recovery." Blood 81, no. 7 (1993): 1933–39. http://dx.doi.org/10.1182/blood.v81.7.1933.1933.

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Abstract Administration of interleukin-1 beta (IL-1 beta) before a lethal irradiation with or without allogeneic bone marrow transplantation (BMT) protects greater than 90% of the irradiated mice. To approach the mechanisms responsible for the radioprotective effect of IL-1, we examined the effects of IL-1 pretreatment on engraftment and kinetics of peripheral blood, spleen, and marrow cell reconstitution after irradiation and BMT. Although the BMT was not necessary for the survival of the IL-1-pretreated lethally irradiated mice, allogeneic marrow did engraft in these mice as evaluated in the

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Tiberghien, P., V. Laithier, M. Mabed, et al. "Interleukin-1 administration before lethal irradiation and allogeneic bone marrow transplantation: early transient increase of peripheral granulocytes and successful engraftment with accelerated leukocyte, erythrocyte, and platelet recovery." Blood 81, no. 7 (1993): 1933–39. http://dx.doi.org/10.1182/blood.v81.7.1933.bloodjournal8171933.

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Administration of interleukin-1 beta (IL-1 beta) before a lethal irradiation with or without allogeneic bone marrow transplantation (BMT) protects greater than 90% of the irradiated mice. To approach the mechanisms responsible for the radioprotective effect of IL-1, we examined the effects of IL-1 pretreatment on engraftment and kinetics of peripheral blood, spleen, and marrow cell reconstitution after irradiation and BMT. Although the BMT was not necessary for the survival of the IL-1-pretreated lethally irradiated mice, allogeneic marrow did engraft in these mice as evaluated in the spleen a

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8

Montfort, Megan J., Christopher R. Olivares, Jean M. Mulcahy, and William H. Fleming. "Adult blood vessels restore host hematopoiesis following lethal irradiation." Experimental Hematology 30, no. 8 (2002): 950–56. http://dx.doi.org/10.1016/s0301-472x(02)00813-5.

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Lange, Claudia, Bärbel Brunswig-Spickenheier, Heike Cappallo-Obermann, et al. "Radiation Rescue: Mesenchymal Stromal Cells Protect from Lethal Irradiation." PLoS ONE 6, no. 1 (2011): e14486. http://dx.doi.org/10.1371/journal.pone.0014486.

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10

Goel, H. C., Madhu Bala, J. Prasad, S. Singh, P. K. Agrawala, and R. C. Swahney. "Radioprotection byRhodiola imbricatain Mice Against Whole-Body Lethal Irradiation." Journal of Medicinal Food 9, no. 2 (2006): 154–60. http://dx.doi.org/10.1089/jmf.2006.9.154.

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Più fonti

Tesi sul tema "Lethal irradiation"

1

Manickam, Arun Kumar [Verfasser], and Michael [Akademischer Betreuer] Reth. "Development of a new mouse host for (syngeneic, allogeneic and xenogeneic) hematopoietic cell transplantations without lethal irradiation as preconditioning." Freiburg : Universität, 2018. http://d-nb.info/117907520X/34.

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Zerabruk, MA. "Repair of sub-lethal damage following single and split-dose irradiation using 60co-gamma and p(66)Be neutrons." Thesis, Cape Peninsula University of Technology, 2005. http://hdl.handle.net/20.500.11838/1504.

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Thesis (MTech Biomedical Technology)--Cape Peninsula University of Cape Town, 2005<br>In clinical radiotherapy, experiments are performed to determine optimal conditions of the radiation prior to radiotherapy. These experiments focus on the relative biological effectivness(RBE) determination and are predominantly applied in high linear energy transfer (LET) radiations i.e. fast neutrons, as the RBE values for such radiations vary greatly. In general, the RBE of a certain radiation relative to a given reference radiation flCo gamma) varies widely with the energy, dose, dose rate, frac

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3

MITAKE, MALVINA B. "Estudos bioquimico e farmacologico das crotaminas nativa e irradiada com radiacao gamma de sup(60)Co." reponame:Repositório Institucional do IPEN, 2000. http://repositorio.ipen.br:8080/xmlui/handle/123456789/10819.

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Made available in DSpace on 2014-10-09T12:44:21Z (GMT). No. of bitstreams: 0<br>Made available in DSpace on 2014-10-09T14:07:53Z (GMT). No. of bitstreams: 1 06882.pdf: 5110355 bytes, checksum: 668c71c778c1cbf8aaae51261934b794 (MD5)<br>Tese (Doutoramento)<br>IPEN/T<br>Instituto de Pesquisas Energeticas e Nucleares - IPEN/CNEN-SP

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Oyewole-Said, Damilola. "Flagellin-Mediated Irradiation Protection in Mice." 2017. http://scholarworks.gsu.edu/biology_theses/78.

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Bone marrow (BM) transfer from flagellin-treated mice has been reported to improve the survival of lethally-irradiated mice. Although the mechanism for flagellin’s antiviral and antibacterial effects have been elucidated, there remains a gap in knowledge regarding its radioprotective effects. Here, we report that flagellin treatment results in a 5-fold increase in the proliferation of Lin-Sca-1+C-Kit+(LSK) cells, a heterogeneous stem and multipotent cell population in BM, with the most striking increase within the ST-HSC, MPP2 and MPP3 subpopulations. Furthermore, the presence of TLR5 but not

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5

Wang, Kuang-Yih, and 王光毅. "The Mechanism of Post-irradiation Hypertonic Treatment Induced Fixation of Potentially Lethal Damage in Glioma Cells." Thesis, 2006. http://ndltd.ncl.edu.tw/handle/09462504082627892589.

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碩士<br>國立陽明大學<br>放射醫學科學研究所<br>94<br>The molecular mechanism underlying radiation potentially lethal damage (PLD) has been of great interest, yet remains unclear, in radiation biology for more than 30 years. In this study, we applied high-density oligonucleotide microarray and bioinformatics to investigate the hypertonic effect on PLD in U87MG glioma cells. Analysis of the microarray data showed that the expression levels of 210 genes out of 20,173 genes were altered. Classification of these affected 210 genes according to their biological functions indicated that they are involved in metabolism

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Capitoli di libri sul tema "Lethal irradiation"

1

Atsushi, Hashimoto, Sawai Jun, Igarashi Hideo, and Shimizu Masaru. "Pasteurization by Far Infrared Irradiation below Lethal Temperature of Bacteria." In Biochemical Engineering for 2001. Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-68180-9_195.

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2

Berk, L., K. Patrene, and S. Boggs. "Increased Mouse Survival by 16,16-Dimethyl Prostaglandin E2 Pretreatment and/or Bone Marrow Transplantation after Supra-Lethal Whole Body Irradiation." In Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3874-5_58.

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3

Dikomey, Ekkehard, Kerstin Borgmann, Malte Kriegs, Wael Y. Mansour, Cordula Petersen, and Thorsten Rieckmann. "DNA repair after oncological therapy (radiotherapy and chemotherapy)." In Oxford Textbook of Oncology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199656103.003.0009.

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Abstract (sommario):

The lethal effect of ionizing irradiation on tumour cells is mostly determined by the repair of DNA double-strand breaks (DSBs). Cells are able to repair most of the DSBs, but 1% to 3 % are either non- or mis-repaired, which will then give rise to lethal chromosomal aberrations. Cells have evolved complex DSB repair mechanisms with a stringent hierarchy to guarantee the genomic stability. However, in tumour cells both mechanisms as well as hierarchy are often disturbed. This knowledge is important for an understanding of the radiation response of tumours, but—most of all—for the establishment of new and specific targets for therapy.

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Dikomey, Ekkehard, Kerstin Borgmann, Malte Kriegs, Wael Y. Mansour, Cordula Petersen, and Thorsten Rieckmann. "DNA repair after oncological therapy (radiotherapy and chemotherapy)." In Oxford Textbook of Oncology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199656103.003.0009_update_001.

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Abstract (sommario):

The lethal effect of ionizing irradiation on tumour cells is mostly determined by the repair of DNA double-strand breaks (DSBs). Cells are able to repair most of the DSBs, but 1% to 3 % are either non- or mis-repaired, which will then give rise to lethal chromosomal aberrations. Cells have evolved complex DSB repair mechanisms with a stringent hierarchy to guarantee the genomic stability. However, in tumour cells both mechanisms as well as hierarchy are often disturbed. This knowledge is important for an understanding of the radiation response of tumours, but—most of all—for the establishment of new and specific targets for therapy.

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5

Rendon, P., G. Franz, and R. J. Wood. "Assessment of Irradiation Doses for TSL (Thermal Sensitive Lethal) Strain Vienna 42." In Fruit Fly Pests. CRC Press, 2020. http://dx.doi.org/10.1201/9780367812430-31.

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6

DORIE, M. J., GABRIELLA BEDARIDA, and R. F. KALLMAN. "INTERLEUKIN-l (IL-l) PROTECTS AGAINST THE LETHAL EFFECTS OF CYCLOPHOSPHAMIDE (CY) AND LOCALIZED LUNG IRRADIATION." In Radiation Research: A Twentieth-century Perspective. Elsevier, 1991. http://dx.doi.org/10.1016/b978-0-12-168561-4.50583-7.

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Atti di convegni sul tema "Lethal irradiation"

1

Zhang, Hong-wei, Li-li Zhang, Can-bang Zhang, Lin Xu, and Ling-yun Zhou. "Experimental study and theoretical analysis of lethal effects and stimulating effects of laser irradiation on biologically suspended cells." In Photonics and Optoelectronics Meetings 2011. SPIE, 2012. http://dx.doi.org/10.1117/12.918788.

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2

Molthen, RC, Q. Wu, ER Jacobs, JE Moulder, and M. Medhora. "Pulmonary Vascular Injury from Single Exposure, Sub-Lethal Thoracic Irradiation: Response to Mitigation Strategies Targeting the Renin-Angiotensin System." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5567.

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3

Palii, Stela S., та Richard S. Paules. "Abstract B22: Combined disruption of ATM and CHK1 functionalities reveals redundancies in the DNA damage response pathways and results in synthetic growth inhibition following γ-irradiation". У Abstracts: AACR Precision Medicine Series: Synthetic Lethal Approaches to Cancer Vulnerabilities - May 17-20, 2013; Bellevue, WA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.pms-b22.

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4

Kumari, Anita, Orpha Rachel Mott, Ercan Cacan, Susanna F. Greer, and Charlie T. Garnett. "Abstract 636: Sub-lethal irradiation of diverse human carcinoma cells imparts enhanced and sustained expression of important modulators of effector CTL activity." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-636.

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